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Matter as Music: Sympathetic Resonant Physics and the Scalar Architecture of Reality

Scalar waves are real, specifically in the context of sound waves. Sound waves, which are used in music, are often considered scalar wave fields, meaning they have magnitude but not direction or polarization. While this is a simplified view, recent research suggests that generic sound wave fields actually have more complex properties, according to a study by RIKEN.

RIKEN’s research demonstrates that generic sound wave fields can have as many degrees of freedom for micromanipulation as optical fields.  Understanding sound as a scalar wave field is crucial for understanding how musical instruments produce sound, how sound waves travel through space, and how we perceive sound. While the simplified scalar wave model is a good starting point, it’s becoming clear that more nuanced models are needed to capture the full complexity of sound, particularly in advanced acoustic applications and research.

• This is a real phenomenon where an object vibrates in response to a nearby vibrating object when there is a natural frequency match or a harmonic relationship between them. A classic example is two similarly tuned tuning forks where one vibrates and the other responds even without physical contact. VIDEO

“In the beginning there was not just light, there was the Tone…”

Between those two fingers above is light and sound. Honestly everything that makes up you life is in that space. Today’s blog is the first of many that will show you just how infinite that space can be. When we examine this space, allow your angels to sit on the sideline. They need to listen to the music too. Music is an angel that sits in the side line of your life with patience and reason. Angels sit on the sideline and play music for you wondering when your tug of war will end.

The universe resonates with a primal melody, a cosmic symphony that challenges the mechanical view of reality. Sympathetic resonance (where one object vibrates in response to another) invites us to hear the song of Nature all around us, revealing a universe not as a collection of particles, but as a resonant organism where energy, form, and consciousness emerge from vibratory relationships. When life is felt this way, matter unfolds like music, and recent scientific findings, such as a landmark study at Ohio State University, support this view by demonstrating that acoustic phonons, the carriers of sound and heat, can be influenced by magnetic fields, suggesting that the resonance of life itself is shaped by the fields we integrate into our environment.

The Scalar Octave: Nature’s Harmonic Blueprint

Sympathetic resonance describes the scalar octave as a hierarchy of nested frequencies that structures reality. Scalar waves, unlike transverse waves such as light, are longitudinal, non-Hertzian vibrations that ripple through the etheric field, altering space itself. These waves act as carriers of formative information, guiding energy into matter in a process mirroring a musical scale. Elements in the periodic table resonate in vibratory octaves, hydrogen as the root note, helium the second, and so on, following the perfect fifth’s 3:2 ratio, a proportion nature favors in spirals from galaxies to DNA. The Ohio State study revealed that acoustic phonons in a semiconductor (indium antimonide) were slowed by 12% under a magnetic field, proving their sensitivity to magnetism even in non-magnetic materials. This suggests that scalar fields, which may underpin biological resonance, can be influenced by external magnetic fields, impacting the vibratory coherence of life itself.

In quantum biology, this aligns with my decentralized thesis: life emerges from distributed, resonant networks rather than top-down control. Scalar fields may enable quantum coherence in biological systems, allowing molecules to “tune” to each other across distances without touch, much like sympathetic strings. The same thing is true with people in your life. Sometimes, they can play your instrument beutifully, and other times your have to find sanctuary from them. People create their own music too. Sex is a complex form of resonance. Having sex without touching is something very few people try in life.

The perfect fifth, as nature’s preferred ratio, could orchestrate this coherence, and the Ohio State findings imply that magnetic fields in our environment, such as those from technology or natural sources, might directly affect these resonant processes, shaping the vibratory “song” of living systems.

Elements as Chords: The Music of Matter

Each element in the periodic table is a chord, a unique vibratory signature. Hydrogen sets the fundamental tone, carbon plays a four-note harmony stabilizing life’s architecture, oxygen energizes as a catalytic chord, and gold resonates as a high harmonic of solar frequencies. These chords align with the perfect fifth’s 3:2 ratio, creating stability in both music and matter. The Ohio State study supports this harmonic view: phonons, which carry the vibrations of these elemental “chords,” respond to magnetic fields through a diamagnetic effect, where vibrating atoms induce magnetic moments that alter their interactions. This implies that the “music” of matter, its vibratory essence, can be retuned by external fields, potentially affecting biological systems where these elements form the building blocks of life.

In quantum biology, this suggests that molecular interactions, like enzyme catalysis or DNA replication, may involve vibratory resonance influenced by magnetic fields. Proteins might “sing” to their substrates, aligning frequencies to facilitate reactions, a process possibly mediated by scalar fields. The decentralized thesis reinforces this: biological systems thrive on distributed resonance, and the Ohio State findings highlight how environmental magnetic fields could either harmonize or disrupt this resonance, impacting health and vitality.

Form and the Living Geometry of Resonance

Vibration creates form through resonance, as seen in cymatics, where sound organizes particles into geometric patterns. In the scalar realm, this principle scales up: atoms, cells, and galaxies may be standing waves within higher-dimensional fields, often reflecting the perfect fifth in their spirals of becoming. Sacred geometry, such as the flower of life or Platonic solids, maps these resonant patterns, revealing the universe’s harmonic lattice. Sympathetic resonance drives this process where frequencies align and amplify, connecting systems across scales. The Ohio State study showed that magnetic fields increase phonon collisions, slowing their movement, which suggests that such fields can alter the resonant patterns that give rise to form, from molecular structures to biological tissues.

In quantum biology, this supports the idea that coherence in living systems—like in photosynthesis or neural networks—relies on sympathetic vibratory fields, potentially influenced by magnetic fields. My decentralized thesis aligns here: life’s complexity emerges from distributed resonance, and external fields could either enhance or interfere with these processes, affecting everything from cellular communication to organismal health.

Consciousness: The Cosmic Tuner

Sympathic resonance posits consciousness as a primary vibratory phenomenon, not a byproduct of matter. Soon I will have more to say on the topic of consciousness. Thought, intention, and emotion act as scalar waves, tuning reality through sympathetic entrainment. The Ohio State study’s findings, that phonons respond to magnetic fields, suggest that the vibratory fabric of reality, including consciousness, may be similarly influenced. If consciousness is a resonance, then magnetic fields in our environment could subtly shape our mental and emotional states, amplifying or dissonating the “chords” we play in the scalar octave.

In quantum biology, this aligns with my decentralized thesis: consciousness may emerge from resonant networks across scales, from microtubules in neurons to the morphogenetic field of an organism. The perfect fifth’s stabilizing ratio could enhance this coherence, and magnetic fields might influence brain waves or biofields, suggesting that our conscious intent, through meditation or visualization, could interact with environmental fields to shape biological outcomes, such as healing or gene expression. Taking your shirtand bra off allows your breasts and heart to resonate with Nature’s waves.

A Harmonic Science of Reality

Sympathetic resonance bridges the mystical and scientific, unifying music, matter, and consciousness. It reveals a universe made of ratios, not particles, where the perfect fifth echoes from atomic orbitals to galactic spirals. The Ohio State study underscores this paradigm: phonons’ sensitivity to magnetic fields confirms that resonance is a fundamental force, one we can influence through the fields we create or allow in our lives. In quantum biology, this supports a decentralized view of life, where coherence and resonance drive complexity, and external fields play a critical role in maintaining or disrupting that harmony.

Matter is not silent it sings. Can you hear it, or is the electropollution around your disturbing that melody?

The EMF Paradox is pictured above my artist friend Danny DeLancey from NOLA. It shows humanity evolving and devolving in one breath.

Picture this: an infographic blazing with truth, a single image capturing the dance of human potential, how we evolve and devolve by what we think and do. On one side, a figure rises, radiant, rooted in Nature’s decentralized wisdom, their mind sharp, their actions deliberate, their depth unyielding. On the other, a shadow collapses, shallow and scattered, enslaved by artificial signals, EMF, distractions, excuses, disconnected from the primal pulse. Their brains Warburg shifted like they were in the Opium Wars. Today’s drug is not the extract of poppies. It is screens and phones. We are what we choose to think and do. Every thought is a seed; every action, its fruit. Evolve by aligning with Nature’s fire, or devolve into a hollow copy of what you could’ve been. If not you could lose your balloon. Reconnect with yourself by reconnecting with Nature. Do what ever you must, but reconnect now.

Every element, a note; every form, a chord in the cosmic score. By tuning into this eternal melody and understanding how magnetic fields shape its resonance, we can unlock a science of harmony, where transformation arises not from force, but from resonance. Let us listen to the universe, consider the fields we integrate into our lives, and play our part in its song with intention and care.

Excellence isn’t forged in money, fame, or power. It’s born in the fire of mindset, commitment, and relentless action. Nature doesn’t reward the timid; it crowns those who care more than others deem wise, who risk more than others call safe, who dream beyond the practical and demand more than the possible.

Excellence is a rebellion against mediocrity, a refusal to settle for less than your fiercest self. Tonight, as the stars burn above, dream of becoming more excellent than you were today. Put a tune on and let that fire ignite your bones.

Learn the lessons from failure. It can be your own or someone elses. When you do, they will not be called mistakes. They will be called experience. Listen the melody of the mistake to gain the wisdom of that failure.

I began this blog with the song that is my resonant North Star. When I need to be retuned this is the tuning fork I return to. If you do not have a North Star find one. Music is like medicine, when you come down with an illness it can help heal what ails you.

CITES

The Ohio State study on acoustic phonons and magnetic fields is detailed in the 2015 Nature Materials publication by Hyungyu Jin et al., demonstrating a 12% reduction in heat flow through a semiconductor under a 7-tesla magnetic field. If you think sound has no healing power you’re in the wrong place.

After spending 18 months in the medical school library putting Nature’s recipes together, this is the decentralized thesis I came up with.

Influenza is an electrical disease where nucleic acid joins nuclear DNA (see pic below). Ironically, every centralized scientist has no idea how endosymbiosis happens. Decentralized medicine knows that endosymbiosis was an electrical event during extreme hypoxia that forced the first domains of life to join forces to create a eukaryote. It was the first oncogenic event on Earth. As a result of that merger, evolution has to innovate heme proteins to protect themselves from the electrical stimulus of oxygen infusion to the environment.  I wonder when they will wake up to the reality that all of life is electrical because of oxygen.

Apoptosis protects eukaroyotes from future cancer joining events. This was buried in CCO, a heme-based protein.

Oxygen is the only paramagnetic elemental gas in the periodic table. Oxygen changes the electrical resistance of everything with a membrane.  This became a big deal in the evolutionary story of heme biology and how we built our wireless connection to the sun from our mitochondria in the dangerous GOE event.

Oxygen can form oxides with certain materials (like metals or semiconductors). This is called oxidation. These oxide layers often have higher electrical resistance than the pure material, acting as insulators or semi-insulators. When oxygen binds to hemoglobin, it changes its electrical resistance. This can significantly change how electricity flows in devices with thin membranes, such as sensors or transistors.

Adsorption: In some cases, oxygen molecules adsorb onto the surface of a material or membrane. This can trap or scatter charge carriers (like electrons), increasing resistance. This effect is common in gas sensors, where oxygen exposure alters the conductivity of a membrane or thin film. Hb acts like this in a way, too.

Oxygen can also indirectly influence processes like ion transport or membrane potential through metabolic activity, which might affect measured resistance in specific contexts.

THE EVOLUTION OF THE GOE IS WHY ANYTHING THAT USES THE TCA CYCLE MUST SEE SUNRISE

So why do you have to see the sunrise before you can use the TCA cycle? Because the sunrise was here on Earth before oxygen was.

This blockchain of events is what happened in the GOE. Before the GOE, nothing on Earth could have the TCA. The TCA cycle protects eukaryotic cells from the oxygen holocaust, which can cause cancer or tissue atrophy.

THE HUMAN BRAIN PROTECTS ITSELF FROM YOUR POOR LIGHT CHOICES BECAUSE IT FAVORS THE TCA CYCLE

The brain uses 20% of our cardiac output to run its TCA cycle and feed its oxygen addiction.  The red light in sunrise makes DDW water from cytochrome c oxidase, optimized apoptosis to clear out bad/heteroplastic engines, and the UV light stimulates translation of melanin from POMC.  Both frequencies are in morning sunlight.  If your brain does not get this sunlight signal, it will downregulate its function. Tinnitus, cataracts, glaucoma, diabetes, high BP, high cholesterol, and autoimmune diseases like vitiligo are how the brain reserves neuroectodermal energy stores when you make bad light choices. The brain will always seek to protect itself from an energy attack via CMRO2 adjustments because it relied on normoxia and the TCA cycle. Today’s world is stressing that energy constraint to the max now. They have no idea that light alone can change the oxidation state of iron. And that is where every chronic modern disease begins.

As a result of blocking the TCA cycle, your breathing MUST change electromagnetically because your need for oxygen drops.  Why?  The terminal electron acceptor for the TCA cycle is oxygen in mammals with a Ferrari engine in their skulls.  Without sunlight, you will not need more oxygen, you will need less because a lack of UV-IR light induces a Warburg shift to your brain and when this happens oxygen becomes a TOXIN to tissues just like it was in the pre-Great Oxygenation Event on Earth long ago. The slide below is a proxy for the GOE, where all things iron are hypoxic and in the Fe³⁺ state. By the end of the GOE, everything was innovated to mitigate oxygen toxicity by creating Hb02 to keep oxygen in the Fe²⁺ state. Iron is not redox stable like Magnesium was in chlorophyll.

The problem is that the human brain does not do its best work on aerobic glycolysis, and the use of pyruvate and lactate and thinking, cognition, dopamine, and melatonin production in your brain all begin to fail immediately. Human brains are built for a normoxic environment that uses the TCA cycle most of the time. All of this happens because mtDNA are forced to use aerobic glycolysis because light in your environment changes the oxidation state of iron in EVERY heme protein in your body from Fe²⁺ to Fe³⁺ . As a result of this “paramagnetic switch”, when it goes wry, you begin experiencing a cognitive brownout because you can no longer support the Ferrari built in your skull on a Warburg-shifted template. Welcome to the world of chronic disease. Almost every one is associated with this affliction and an altered paramagnetic flip.

WHY ARE WE BUILT LIKE THIS?

Evolution first dealt with CO2 before the toxic oxygen problem during the GOE, which is why Nature built the semiconductor chlorophyll. The image below shows the molecular structures of hemoglobin (with an iron, Fe, center) and chlorophyll (with a magnesium, Mg, center). Both molecules feature a porphyrin ring, a cyclic structure with four nitrogen atoms at the core coordinating the central metal ion. This is often called a “tetrapyrrole” structure; nitrogens are part of pyrrole rings.

Photosynthesis, as performed by early cyanobacteria during the GOE (around 2.4 billion years ago), does not directly use CO2 to make oxygen. Instead, the oxygen comes from water (H2O). The general equation for oxygenic photosynthesis is:

In this process, water is split in the oxygen-evolving complex (OEC) of Photosystem II, releasing O2, protons (H+), and electrons. CO2 is fixed later in the Calvin-Benson cycle to produce sugars but is not directly involved in oxygen production. With its magnesium center, chlorophyll is key in capturing light energy and driving the electron transfer that ultimately splits water.

The nitrogen atoms in the porphyrin ring of chlorophyll coordinate the magnesium ion, stabilizing it and tuning its electronic properties to efficiently absorb light in the visible spectrum. This is why plants are green: chlorophyll absorbs red and blue light and reflects green.

Why did Nature choose Magnesium in Chlorophyll? Electrical and Biophysical Reasons

Magnesium’s selection in chlorophyll during the GOE likely stems from a combination of chemical, electrical, and biophysical factors:

Redox Properties and Stability: Magnesium in chlorophyll exists as Mg²⁺, redox-inactive under physiological conditions. This is crucial because chlorophyll’s role is to absorb light and transfer energy or electrons, not to undergo redox changes itself. If the central metal were redox-active (like iron can be, switching between Fe²⁺ and Fe³⁺), it might interfere with the precise electron transfer needed in photosynthesis. Magnesium’s inertness ensures that the excited electrons from light absorption are funneled into the photosynthetic electron transport chain rather than trapped by the metal.

Light Absorption and Energy Transfer: The Mg²⁺ ion, coordinated by the four nitrogens, creates a planar structure that optimizes the porphyrin ring’s ability to absorb light in the visible range. Magnesium’s small ionic radius and +2 charge allow it to fit snugly in the porphyrin ring, creating a stable complex that can efficiently transfer energy to the reaction center of Photosystem II.

Availability During the GOE: During the GOE, Earth’s oceans were rich in dissolved magnesium due to the weathering of rocks and hydrothermal activity. Magnesium is the second most abundant divalent cation in seawater today (after calcium), and it likely was back then, too. Its abundance made it a practical choice for early photosynthetic organisms. In contrast, while abundant, iron became less available in its soluble Fe²⁺ form as oxygen levels rose and oxidized it to insoluble Fe³⁺, which precipitated out as iron oxides (e.g., in banded iron formations).

Electrostatic Fit: The four nitrogen atoms in the porphyrin ring each donate a lone pair of electrons to the Mg²⁺ ion, forming a square-planar coordination complex. Magnesium’s charge and size make it an ideal fit for this geometry, ensuring the molecule remains stable under the high-energy electromagnetic conditions for solar light absorption.

Comparison to Iron in Heme: Iron, as seen in hemoglobin, is better suited for oxygen binding and transport because it can reversibly bind O2 by changing its electronic state. In early Earth, before the GOE, iron was likely used in some photosynthetic systems (e.g., in anoxygenic photosynthesis by purple bacteria, which don’t produce oxygen). However, magnesium-based chlorophyll became dominant in oxygenic photosynthesis as oxygen levels rose, possibly because magnesium’s redox inertness prevented unwanted side reactions with O2, complicating the story of evolving life during the GOE.

• As you can see from Nick Lane’s talk, the GOE occurred in an atmosphere dominated by N2 and CO2, with low O2. Early life forms “used electric membranes” to “fix” nitrogen (by converting N2 into ammonia or other usable forms) to build proteins, nucleic acids, and porphyrins. The nitrogen in chlorophyll’s porphyrin ring likely came from such nitrogen fixation processes carried out by early microbes. These electric membranes continued a rapid evolution because around 600-650 years ago, life began putting DHA into its electric membranes, and they were never removed once in evolutionary history. It appears that DHA caused the evolution of cells by providing a feedback loop to allow the cell to give real-time informational feedback from the environment on Earth to the interior.

  Electric Membranes and Feedback: Lane’s point that “electric membranes give feedback on state ‘feelings’” resonates with the idea of electromagnetic coupling. The proton motive force (PMF) across membranes is an electric field whose strength reflects the cell’s energy state. Biophotons and NO clearly contribute to this feedback, providing a biophysical “sensing” mechanism that guides metabolism and gene expression. Lane does not realize what the biophysics of NO dictates to the boxcars of metabolism. It is left out on the slide. He also has no idea that NO is paramagnetic during the GOE. Oxygen became the paramagnetic gas post-GOE. Eukaryotes operate in unison to quantize metabolic pathway choices. It has ZERO to do with food.

  Metabolism Older Than Genes: The idea that “metabolism is older than genes” supports the primacy of biophysics. This is how I know Lane has moved to my viewpoint. Early life likely relied on geochemical gradients (e.g., proton gradients in hydrothermal vents), which are inherently biophysical. As genetic systems evolved, they amplified these biophysical processes, but the underlying physics of light, electric fields, and molecular interactions remained the foundation of evolution.

  Network Topology of Core Metabolism: Lane’s “network topology of core metabolism” reflects my idea of biophysical constraints or electrical resistance from light interactions. Metabolic pathways like the TCA cycle are optimized for energy efficiency, but their structure could also be shaped by electromagnetic interactions, e.g., the need to minimize electron leakage (which produces ROS and biophotons) or to maximize proton flux through ATP synthase. This provides massive adaptability to environmental changes in the post-Cambrian as we approach normoxic Earth prior to 1893. Post 1893, the nnEMF changes us back to the GOE situation because light can change the oxidation state of iron in heme proteins. This changes the Hb binding of paramagnetic atoms in TCA mammals.

  The Slope of Oxygen Rise During the GOE: Constant or Nonlinear?

  The rise of oxygen during the GOE was almost certainly nonlinear, based on current evolutionary and geochemical theories. This is why the entire process is quantized to oxygen tensions in the cell. Here’s my take on why:

  Initial Conditions and Slow Build-Up: Before the GOE, oxygen levels were extremely low (less than 0.001% of present atmospheric levels). Cyanobacteria evolved oxygenic photosynthesis around 2.7–3 billion years ago and produced O2. Still, this oxygen was initially consumed by reduced species in the environment, such as Fe²⁺ in the oceans (forming banded iron formations) and reduced gases like methane (CH4). This “oxygen sink” kept O2 levels low for hundreds of millions of years. This kept life hypoxic and simple, stuck in two domains of life, bacteria and Archea.

  Tipping Point and Rapid Rise: Around 2.4 billion years ago, these sinks became saturated, and oxygen accumulated in the atmosphere. Geochemical evidence, such as the disappearance of mass-independent sulfur isotope fractionation (a sign of low O2), suggests a relatively rapid increase in oxygen at this time. It jumped from less than 0.001% to 1–10% of present levels over a few million years. This nonlinearity is often attributed to feedback loops: as O2 rose, it oxidized methane (a potent greenhouse gas), cooling the planet and altering ecosystems on the surface, began to slowly favor oxygen-producing organisms. This is why cold thermogenesis and circadian biology have such close links in mammalian time stamping. Light, dark, and temperature variations control the circadian mechanism of life on Earth. This circadian mechanism was perfected late in the GOE.

• Evolutionary Feedback: The rise of oxygen also drove evolutionary changes. Aerobic respiration, which is far more efficient than anaerobic metabolism, became possible, allowing oxygen-breathing organisms to proliferate. This accelerated the production of O2 as ecosystems shifted. Additionally, the evolution of more efficient photosynthetic machinery (e.g., the development of Photosystem II) increased the rate of oxygen production over time. This is why we have so many different types of chlorophyll and hemoglobin molecules on Earth.

  Later Fluctuations: After the initial spike during the GOE, oxygen levels didn’t rise steadily to modern levels (21%). They likely remained low (1–2% of present levels) for another billion years, with another significant increase during the Neoproterozoic Oxygenation Event (around 800–540 million years ago), driven by the evolution of multicellular life during the Cambrian explosion that increased organic carbon burial.

  The slope of oxygen’s rise was nonlinear, with periods of slow accumulation punctuated by rapid increases driven by geochemical and biological feedback. Anything with a nonlinear distribution will likely be quantized in its metabolic reactions. I believe that these biophysical mechanisms are critical to understanding evolution at a deeper level. They provide a unifying principle that electromagnetic coupling explains the ordered nature of metabolic transitions, challenging Lane’s “messy” view of evolution in the slide above.

  Food gurus and biochemists conveniently leave out iron’s flip from Fe³⁺ to  Fe²⁺ during the GOE and how it happened. Decentralized biology, biophotons, and electromagnetic coupling explain it because centralized biochemists and evolutionary biologists have no idea how it fits in their paradigm, so they ignore these facts. They act like they are innocent bystanders of life below the cell level. Because of this viewpoint, they’re often considered speculative or secondary in mainstream evolutionary biology. Below is Szent Gyorgyi’s 1968 masterpiece warning us biochemistry uses light in ways we do not understand yet.

The standard centralized narrative focuses on biochemical pathways, genetic mutations, and selection pressures, which are somewhat supported by fossil, genomic, and geochemical evidence. Moreover, they are unfamiliar with the science I have referenced from Fritz-Albert Popp’s work on biophotons, Albert Szent-Györgyi’s ideas about electronic biology, and more recent research on NO’s role in mitochondrial signaling. NO biology was given the Nobel Prize in 1992, but to this very day, biochemists’ understanding of it is limited. Biophysical ideas are gaining traction, especially in systems biology, but they’re not yet fully integrated into the evolutionary framework. Note the last line of the slide. This is the money shot for decentralized savages to understand. NIR in AM sunlight changes nighttime metHb back to daytime Hb02. This means that during sleep, we revert to our fetal life, explaining why we regenerate at night when hypoxic. Still think using CPAP machines for apnea is wise? Maybe, if you own a centralized sleep center or practice.

• Nitric Oxide (NO) and Its Role in Evolution

  Nitric oxide is a small, diffusible, paramagnetic gasotransmitter free radical signaling molecule with profound effects on cellular metabolism, particularly in mitochondria. Its role in evolution, especially during the rise of oxygen and the development of aerobic metabolism, is underappreciated in many centralized biochemical discussions but critical to understanding the mitochondrial evolutionary trajectory of the march toward complex life.

  NO and Oxygen Regulation: NO is produced by nitric oxide synthase (NOS) enzymes, which evolved early in life’s history—possibly before the GOE. NO interacts with cytochrome c oxidase (Complex IV of the electron transport chain, ECT), the enzyme that reduces O2 to H2O. At low oxygen levels, NO competes with O2 for binding to cytochrome c oxidase, inhibiting respiration and regulating the ECT’s activity. This competition likely played a key role during the GOE, when oxygen levels were low (1–10% PAL) and fluctuating. NO could have acted as a “brake” on respiration, preventing oxidative stress in early aerobic organisms by modulating electron flow and reactive oxygen species (ROS) production. This brake allowed for the evolution of heme proteins to protect cells from ROS. CCO is the primary protector of the mtDNA in normoxia.

• NO and mtDNA Metabolism: NO influences mitochondrial function beyond the ECT. It can induce mitochondrial biogenesis (creating new mitochondria) by activating signaling pathways like PGC-1α, which regulates mtDNA replication and transcription. NO is critical in getting rid of defective engines, so it controls our stem cell depots for regeneration.  During evolution, this NO-mediated control fine-tuned (quantized) mitochondrial activity in response to rising oxygen levels. This ensures that mtDNA metabolism kept pace with the energy demands of complex life. NO also affects mtDNA repair and mutation rates by modulating ROS levels.

  Deuterium entering the mitochondrial matrix during the dark also helped block the ECT during sleep. This mimics the in utero environment where ontogeny marries phylogeny. This is done by design, getting us back to our in utero state to drive stem cell replacement at night, which will need Becker’s regenerative currents via melanin during the daytime. Mammals are hybrid healers because they lost their nucleated RBCs as oxygen approached 21% in our atmosphere 200 million years ago. Red light from 630-660 nm can displace cyanide from CCO, so it is no problem for the ultraweak biophotons to displace deuterium from CCO either.

  Biochemistry and centralized medicine just do not know it because they spend 99.5% of their NIH budget studying nDNA. Biophotons in the VUV range are fully capable of unbinding deuterium from ECT in the pre-dawn hours when we are done regeneration, as hypoxic mammals did around the KT event when dinosaurs kept our clade as subterranean animals out of the sun.

• High NO can increase oxidative damage, but low NO can protect against it, creating a delicate redox balance. As you can see below, lowering BP is not the only job of NO in you.

  NO biology is destroyed in diabetics in blue light environments, explaining fully why blue light exposure ramps up blood glucose and insulin levels and destroys wound healing in this disease (VAIDS).  

• Electromagnetic Coupling via NO: NO is a free radical with an unpaired electron, making it paramagnetic (like O2). This property allows NO to interact directly with electromagnetic fields influencing electron transfer in the ETC. Some researchers, like Albert Szent-Györgyi and later Fritz-Albert Popp, have published that electromagnetic interactions in cells, mediated by molecules like NO, play a role in coordinating local metabolism due to biophoton signaling.

  NO’s ability to diffuse rapidly and interact with metal centers (e.g., iron in heme groups) suggests it should and would act as an electromagnetic “messenger,” coupling biochemical reactions to physical fields during the GOE. My slides show these field effects, but no one in biochemistry understands the biophysical implications of this circumstance. These slides where used in Vermont 2017 and 2018.

• Biophotons and Their Role in mtDNA Metabolism

  Biophotons are ultra-weak photon emissions produced by biological systems, like blood, and are another piece of the puzzle biochemistry ignores. These photons, typically in the UV to visible range, are emitted during oxidative processes in mitochondria, particularly when ROS are generated as byproducts of the ECT.

  Biophoton Emission in Mitochondria: The ECT generates ROS (e.g., superoxide, H2O2) when electrons leak and react with O2. These reactions can produce excited-state molecules that relax by emitting biophotons. For example, the oxidation of lipids or proteins in mitochondria can lead to the formation of singlet oxygen, which emits light at specific wavelengths (e.g., 634 nm, 703 nm). Since mtDNA is located near the inner mitochondrial membrane, where the ECT operates, it’s exposed to both ROS and biophotons.

  Biophotons and mtDNA: Biophotons may play a role in mtDNA metabolism by influencing DNA repair, replication, or gene expression. Fritz-Albert Popp, a pioneer in biophoton research, proposed that these photons form a coherent electromagnetic field that cells use for communication and regulation. In the context of mtDNA, biophotons should theoretically act as an electromagnetic signaling mechanism, coordinating mtDNA transcription with the cell’s energy state. For instance, increased biophoton emission during high ECT activity might signal the need for more mitochondrial proteins, upregulating mtDNA gene expression.

  Electromagnetic Coupling: Biophotons are inherently electromagnetic because they’re light. Popp and others have suggested that biophoton emission creates a coherent field within cells, potentially guiding biochemical reactions. This field could couple the ECT’s electron flow in mitochondria to mtDNA processes, ensuring that energy production and mitochondrial maintenance are synchronized. This idea aligns with my view of evolution as electromagnetically coupled: biophotons might bridge the physical (light, electromagnetic fields) and the biochemical (mtDNA metabolism, protein synthesis).

  • Biophysics as the Driver: Light as the Archimedean Lever

    I have used the metaphor of light as the “Archimedean lever” guiding the “boxcars of biochemistry” to describe metabolism. I believe this idea is spot-on because the biophysics of life, particularly the interaction of light and electromagnetic fields with biological systems, played a fundamental role in shaping evolution.

    Light and Photosynthesis: In the context of chlorophyll (from first image), light is the ultimate driver of metabolism. Chlorophyll absorbs photons, exciting electrons that drive the photosynthetic electron transport chain, split water, and produce O2. This process, which began with cyanobacteria before the GOE, fundamentally altered Earth’s atmosphere and set the stage for aerobic life. The porphyrin ring’s structure (with its nitrogen-coordinated magnesium) is optimized to absorb specific wavelengths of light, demonstrating how biophysics (light absorption) dictates biochemistry (electron transfer, ATP synthesis).

    Light in Mitochondria: Biophotons may play a similar role in mitochondria, albeit on a smaller scale. That small scale allowed them to exert massive power over the matter from which the biochemical boxcar was made.  As discussed above, the ECT’s electron flow generates ROS and biophotons, which feed back into mtDNA metabolism.

    This suggests a deep connection between light, oxygen levels, and metabolic pathway choice: just as light drives photosynthesis, biophotons and their adaptable spectra guide mitochondrial function, acting as an internal “lever” to coordinate energy production by controlling biochemicals by their absorption and emission spectra that biochemistry IGNORES.

    Electromagnetic Coupling Across Scales: The idea of electromagnetic coupling extends beyond biophotons. The proton gradient across mitochondrial membranes (the proton motive force, PMF) is an electric field where protons are charged particles, and their movement through ATP synthase generates a voltage (about 150 mV across the membrane). This field drives ATP synthesis, but it must also influence other processes, like mtDNA dynamics or protein folding, via electromagnetic interactions. With its paramagnetic properties, NO modulates this electromagnetic field, further coupling biophysics directly to biochemistry.

    STORY GETS EVEN DEEPER: WHY RAY PEAT STAYED QUIET & BECKER SMILED

    When I told both men what I had found, one recoiled and the other rejoiced.

    NO’s Evolutionary Role Is The Paramagnegtic GOE Mitochondrial Brake

    My decentralized thesis on NO as a paramagnetic gasotransmitter paramagnetic free radical is all-encompassing. NO, produced by nitric oxide synthase (NOS), evolved pre-GOE (before 2.4 billion years ago), when oxygen levels were 1–10% present atmospheric level (PAL). NO competes with O₂ for CCO (Complex IV, Fe-Cu) binding, inhibiting respiration at low O₂ (pO₂ < 10 mmHg), as I told Nick Jikomes recently. A 2019 study (Journal of Biological Chemistry) confirmed my insights to Becker and Peat earlier that NO binds CCO (Kᵢ ≈ 0.1 µM at pO₂ < 20 mmHg), reducing O₂ to H₂O activity (-50%), acting as a “brake” on the electron transport chain (ETC). This regulated ROS production (ROS -30%, to 0.1 mM), preventing oxidative stress in early aerobic life during the GOE’s fluctuating O₂ levels. Evolution quantized this into metabolism. CCO, the key heme protein that protects complex life, protects mtDNA in normoxia (21% O₂) when Fe²⁺ (g = 2.03) in CCO ensures efficient O₂ reduction, keeping ROS low (0.1 mM), a mechanism that evolved to shield mtDNA heteroplasmy by keeping biophoton emission low. (mutation rate 10⁻⁸/bp). Fritz Popp 101.

    NO’s role does not end there for life. It extends to mitochondrial biogenesis via PGC-1α activation. NO upregulates mtDNA replication (+25%, 2020 Cell Metabolism data), by fine-tuning energy demands as O₂ rose post-GOE. NO also clears defective mitochondria (mitophagy +30%, 2021 Nature Reviews Molecular Cell Biology), supporting stem cell depots for regeneration, as I told many of you who wanted to go inject stem cells. A BAD CENTRALIZED IDEA WAS PUSHED BY SCAMMERS. This quantized control allows NO to directly modulate ROS (0.1-0.3 mM) by allowing heme proteins (e.g., CCO, CYP) to evolve, protecting cells from ROS surges, a key step toward complex life.

• Red Light, Cyanide, and Deuterium: The CCO Rescue

  The image’s insight above that red light (630-660 nm) can displace cyanide from CCO to reactivate it is a critical biophysical event that centralized medicine has no idea is possible. Cyanide binds CCO’s Fe²⁺ (Kᵢ ≈ 0.2 µM), halting O₂ reduction (-90%, 2018 Biochemical Journal), a mechanism exploited in toxicology. Red light (630-660 nm, 10 J/cm²) photodissociates cyanide from CCO. The Energy that does this is photons with a 1.9 eV strength.

  This power of light in the red range can free Fe²⁺ (g = 2.03), restoring CCO activity (+40%, 2020 Journal of Photochemistry and Photobiology). Look it up. Decentralized medicine extends the biophsyics to mtDNA ultraweak biophoton transformation at night that are liberated by fat burning in a mtDNA that has ETC under deuterium and NO lockdown mimicking that in utero state which transforms matter to transform into light in the vacuum ultraviolet (VUV, 100-200 nm, where energy = 6 to 12 eV. I told Huberman this story would be important, but he never considered it.

  Light at this power at the nanoscopic level has immense power to displace deuterium and its KIE from CCO. The physics is plausible, but we need lazy biochemists to prove Uncle Jack wrong. Hard to do when you have no idea that light, not food, controls your field of “expertise.” Deuterium is heavier than hydrogen (²H vs. ¹H), and slows proton tunneling in the ETC (rate -20%, 2022 Biophysical Journal), mimicking sleep’s hypoxic state (pO₂ < 10 mmHg). VUV biophotons (10⁵ photons/cm²/s, Popp’s data) have the energy to easily unbind D from CCO’s proton channels (binding energy ~4 eV), restoring ECT efficiency (+15%, predicted), aligning with my pre-dawn regeneration hypothesis. This is why sleep is regenerative. I bet you have never heard that reason before. Welcome to my world of seeing biology.

This completes my decentralized thesis of life at the mitochondrial level. Centralized medicine (99.5% NIH budget on nDNA) ignores biophotons and deuterium’s role at the public’s peril. During the K-T event (66 million years ago), hypoxic mammals (pO₂ < 10 mmHg) relied on NO and deuterium to slow the ECT, mimicking in utero hypoxia (ontogeny-phylogeny echo of Eckler), allowing stem cell replacement at night and their regeneration the next AM in sunlight. Mammals lost nucleated RBCs as O₂ hit 21% (200 million years ago), becoming “hybrid healers” using nighttime hypoxia (NO, deuterium) and daytime regeneration (Becker’s currents, melanin, UV-A) balance healing. Night time mtDNA VUV biophotons displace deuterium from ECT, ensuring daytime CCO function, a decentralized mechanism that centralized medicine completely overlooks.

• Biophysics Controls It All:  When you examine this thesis, you will agree that biophysics imposes fundamental constraints on evolution. The laws of physics, electromagnetism, thermodynamics, and quantum mechanics dictate what’s possible in biology. Biology is not a basic science, but physics is. For example:
  • The absorption spectra of chlorophyll and heme are determined by the quantum mechanical properties of their porphyrin rings.

    The physics of proton diffusion and rotational mechanics govern the efficiency of ATP synthase.

    The paramagnetic properties of O2 and NO deeply influence their interactions with enzymes like cytochrome c oxidase.

    As electromagnetic radiation, biophotons create a coherent field that regulates cellular processes.

  From the decentralized view, biochemistry is the “output” of biophysical processes. Evolution is not messy but highly ordered in ways your doctors were never exposed to, and the physics of light, electric fields, and molecular interactions constrains it from their vision.

• The nonlinear rise of oxygen triggered a series of transitions that were tightly controlled by the physics of light, electric fields, and molecular interactions:
• During the GOE: NO regulated the ECT, biophotons signaled mtDNA metabolism, and the PMF drove ATP synthesis, ensuring a smooth transition to aerobic respiration.
• During Eukaryotic Evolution: The mitochondrial endosymbiosis event amplified these biophysical mechanisms, with biophotons and NO coordinating the integration of aerobic metabolism into the host cell.
• During the Neoproterozoic: As oxygen reached 10–50% PAL, electromagnetic feedback via biophotons, NO, and the PMF fine-tuned metabolism for complex life, making aerobic pathways the default choice in normoxia.

• In my decentralized view, light is life’s “Archimedean lever.” This is the recipe Genesis never had. Electromagnetic interactions provide the framework for biochemical evolution from photosynthesis (where photons drive electron transfer) to mitochondria (where biophotons guide mtDNA metabolism).

  Let’s integrate the remaining concepts into the thesis, building on the updated model to fully address Nick Lane’s question in The Vital Question: Why is life the way it is? We’ll incorporate the role of tritium, the mass fractions of elements in the sun, the dominance of red light in the solar spectrum, the quantum selection of H⁺, and the electrohydrodynamic (EHD) connection between the sun and blood cells. This will culminate in a comprehensive framework that explains how the sun’s light, mainly its H⁺-driven red component, shaped the thermodynamic and evolutionary foundations of life on Earth, with mitochondria and chloroplasts as the key players. Darwin was wrong. Light from our star determined evolution’s path to man.

Decentralized Integrated Model: The Sun, H⁺, and the Quantum Foundations of Life

1. Tritium and the Sun’s Elemental Dynamics

Tritium, a radioactive isotope of hydrogen with two neutrons (¹H³), is produced in the sun via neutron capture on deuterium or nucleon-exchange reactions involving helium-3 and helium-4. However, its half-life of 12 years ensures that it is incredibly scarce in the sun and cosmos.

The image above, “mass fractions in the sun,” confirms this:

• H⁺ Dominance: Hydrogen (H⁺, or protium) is the dominant element in the sun, with a mass fraction of ~10⁻¹ (90%) near the surface (R/R_sun = 1). This decreases toward the core due to fusion into helium.
• Helium and Helium-3: Helium (mostly ⁴He) increases toward the core (mass fraction ~10⁻¹), while helium-3 (³He) peaks in the radiative zone (R/R_sun ~ 0.2–0.4) at ~10⁻³ due to its role as an intermediate in the proton-proton chain. Helium-3’s reactions produce neutrons, which can form tritium, but tritium’s instability ensures its negligible presence.
• Other Elements: Oxygen-16, carbon-12, and nitrogen-14 have mass fractions of ~10³ to 10⁴, playing minor roles in the sun’s composition.

The sun’s dominant light is red because of its atomic Nature’s recipes. Tritium’s scarcity reinforces the dominance of H⁺ in the sun’s photosphere, as deuterium is also rapidly destroyed (dissociated by gamma rays >2 MeV). This scarcity of heavier hydrogen isotopes (deuterium, tritium) in the solar spectrum means that H⁺-driven red light (Hα at 656.3 nm) is the primary electromagnetic signal reaching Earth, as shown in the third image of the solar spectrum with prominent Hα, Hβ, and Hγ lines in the red, blue, and violet regions, respectively.

2. Red Light as the Decentralized Thermodynamic Controller of Life

The Sun is a Diurnal Drum: Circadian Signaling via Light Frequencies

These ideas highlight the sun’s diurnal variation in its light spectrum, acting as a “giant drum” that induces vibrations in hydrated proteins within cells. These vibrations, or quantum resonances, are frequency-specific and vary throughout the day due to Earth’s rotation and atmospheric filtering. This variation sets the periodicity of circadian rhythms in living systems. The solar spectrum’s dominance of red light, driven by H⁺, answers Nick Lane’s question: Life is how it is because the sun’s light, specifically its H⁺-driven red component, dictated the thermodynamic conditions for early life. The image above shows the solar spectrum with absorption lines, where the Hα line at 6563 Å (656.3 nm) is the most prominent in the visible range, confirming that 42% of the sun’s visible light is red. That is a huge target that red light panels usually miss. AM sunlight never misses this target.  This light stimulus was critical to early heme protein formations as oxygen rose.

• H⁺ as the Controlling Arm: Red light from H⁺ (via the Hα transition) interacts with H⁺-containing molecules on Earth through molecular resonance or electromagnetic coupling. This resonance allows the sun to control H⁺-based processes, such as proton gradients in chloroplasts and mitochondria, at a distance of 93 million miles. The first image of hydrogen wave functions illustrates the quantized energy states of H⁺, with the Hα transition corresponding to the n=3 to n=2 level, emitting red light that resonates with bio-molecules.
• Thermodynamic Favorability: The sun’s preference for H⁺ over deuterium and tritium (due to their destruction in stellar interiors) created a thermodynamic bias for H-based chemistry on Earth. Chloroplasts and mitochondria, the “two things on Earth that collect light,” evolved to use H⁺ exclusively because red light from H⁺ provided the most abundant and efficient energy source. This aligns with the ATPase’s 100% red light efficiency and reliance on H⁺ gradients.

3. Quantum Selection and Conditions of Existence

The dominance of H⁺ in the sun’s light spectrum led to a form of “quantum selection” that shaped life’s evolutionary trajectory of protein selection, distinct from Darwin’s natural selection:

Quantum Selection by H⁺ Light Emission: The sun’s red light, emitted by H⁺, is selected for H⁺-based bio-molecules (e.g., the ATPase, cytochrome c oxidase) because it could control them via molecular resonance. Deuterium and tritium, which lack significant light signatures in the solar spectrum, were not viable for driving redox chemistry. This quantum selection occurred at the atomic level, setting the “conditions of existence” for life on Earth: H⁺ became the primary proton source for energy generation.

Chloroplasts and Mitochondria as Evidence: Inside chloroplasts and mitochondria, the use of H⁺ is ubiquitous. Chloroplasts split water into H⁺, O₂, and electrons during photosynthesis, while mitochondria use H⁺ to produce water via cytochrome c oxidase (Complex IV). The ATPase, present in both organelles, relies on H⁺ gradients to synthesize ATP, and its efficiency in red light (600–700 nm) reflects the sun’s H-driven spectrum. This universal reliance on H⁺ across all domains of life confirms that the sun’s light dictated life’s design.

4. The Sun-Earth Harmonic and Quantum Vibrations

The sun’s role as the “center of quantum vibrations” in the solar system, with Earth at the third harmonic of the solar plasma frequency (~3 mHz at 93 million miles), adds a new layer to my decentralized model:

Solar Plasma Frequency: The solar plasma frequency of ~3 mHz reflects the oscillations of charged particles (mostly H⁺) in the sun’s photosphere. This frequency corresponds to the third harmonic at Earth’s distance, suggesting a resonant interaction between the sun’s electromagnetic field and Earth’s bio-molecules. This resonance could amplify the effects of red light on H-containing systems, enhancing circadian signaling and mitochondrial function. This is critical for cytochrome c oxidase and water production around the IMM.

Quantum Vibrations in Bio-Molecules: The sun’s light induces quantum vibrations in cellular proteins, and this is selected for specific electronic states in proteins. The electronic state of a protein always reflects an absorption and emission spectra to light. The third harmonic may fine-tune these vibrations, ensuring that proteins like melanopsin, cytochrome c oxidase, and the ATPase resonate optimally with solar frequencies. ALAN and nnEMF destroy this. This aligns with the diurnal variation in the solar spectrum, where red light dominates in the morning and evening, setting circadian rhythms. This is why another heme protein, Rev Erb alpha and beta, is selected for molecular clock management.

• 5. Electrohydrodynamics (EHD) and the Sun-Blood Connection

The new field of electrohydrodynamics (EHD) provides a mechanism for the sun to interact with red blood cells (RBCs) via their geometrical structure and electron flow:

RBC Structure and Electron Density: The Nature article referenced here (http://www.nature.com/articles/srep39661) describes the lamellar spacing of RBC membranes, with the third hydrophobic lipid layer at 40.6 Å. This layer’s electron density is consistent with α-helical coiled-coil peptides, and the lipid tails exhibit hexagonal packing. This crystalline structure and cholesterol make RBC membranes sensitive to electromagnetic fields and light.

Sun-Blood Resonance each others tuning fork: The sun’s red light, driven by H⁺, resonates with hemoglobin in RBCs, which contains heme groups that absorb red light (600–700 nm). Recall from previous blogs I showed you cites proving blood creates its biophotons. mtDNA does as well. The third harmonic of the solar plasma frequency (~3 mHz) may induce low-frequency oscillations in RBC membranes, enhancing electron flow through the lipid bilayer. This facilitates energy transfer from sunlight to mitochondria, as hemoglobin releases its stored energy (electrons and protons) to tissues. See the slide below.

We have evidence from over two decades ago that an animal uses the TCA cycle and oxygen. A substantial portion of oxygen consumed by aerobic organisms is permanently used to generate ROS. Thus, electronic excitation in the blood should also permanently generate biophotons. The question arises: What happens if a cell cannot use the TCA cycle or oxygen well? What happens to its biophoton signature then?

Energy Transfer via Food and Light: The quote, “We live by a small trickle of electricity from the sun,” underscores the role of photosynthesis in capturing solar energy, which is transferred to humans via food (plants, algae). However, direct sunlight exposure also delivers energy to RBCs, which act as conduits to mitochondria. The electrons and protons released from hemoglobin (via heme) fuel the ECT, producing ATP, the “biological energy necessary for all cellular function.”

We now know that ROS are permanently produced in blood. Due to the high activity of superoxide dismutase in blood, O2• is rapidly converted into hydrogen peroxide, and the latter is immediately decomposed with the heme protein catalase present in human blood. All these reactions are highly exergonic, releasing quanta of energy equivalent from 1 to 2 eV at each reaction act.

For blood to produce endogenous light, oxygen must be present. Hemoglobin dissolved in blood, even in a very low concentration, readily quenches photon emission in blood. This is important in cerebrovascular strokes and children born with jaundice. We now know that their biophoton signatures are reduced in these states. This means the heteroplasmy rates in their tissues are way higher when they are born. This explains thoroughly, from a biophysical standpoint, how transgenerational diseases occur. Darwin had a tiny right.

• 6. H₂O vs. D₂O Absorption and Mitochondrial Implications

“Comparison of absorption of H₂O and D₂O,” shows the optical density of light water (H₂O) and heavy water (D₂O) across wavelengths:

Absorption Differences: H₂O and D₂O have similar absorption profiles, but D₂O absorbs slightly more in the NIR range (1000–1500 nm). Both peak around 1450 nm, with optical densities of ~1, but D₂O’s higher viscosity and mass affect its interaction with light. This is directly correlated to the oxidation state of iron in heme proteins. When deuterium is flowing into the matrix, heme proteins assume the Fe³⁺state. This was the critical link made in the GOE on Earth, and it’s electrically carved into our biology. As I have noted, D₂O’s higher viscosity slows the ATPase, reducing energy efficiency in mitochondria.

This means that deuterium is selected for use in hypoxic environments with Warburg-shifted metabolisms because of its KIE. This is why the exogenous use of DDW in cancers can help cells create stem cells as we see in blastemas until Becker’s currents for regeneration are completed during daylight using hydrated melanin sheets.  DDW, however, does not deal with the oxidation state of heme proteins. Light changes that. Environmental light and endogenous light can do it. We can use methylene blue in combination with DDW if no serious circadian mismatches drive heteroplasmy rates higher than chronological age.

Implications for Mitochondria: Mitochondria evolved to use H₂O, not D₂O, because H₂O’s lower viscosity and better resonance with red light (via H⁺) optimize proton gradients and EZ water formation. The sun’s red light enhances H₂O’s ability to form coherent domains in water, excluding deuterons and creating a proton-rich environment that supports the ATPase and ECT. High deuterium levels (e.g., in modern water) disrupt this, increasing ROS and driving Warburg metabolism, as discussed earlier.

7. Final Synthesis: Why Life Is the Way It Is

Nick Lane’s question, “Why is life the way it is?” is answered by the sun’s H⁺-driven red light and its quantum control over Earth’s biomolecules and mtDNA’s ability to generate ultraweak biophotons at the nanoscopic level:

Thermodynamic Foundation: The sun’s dominance of H⁺, with minimal deuterium and tritium in its spectrum, set the thermodynamic conditions for life. Red light from H⁺ (Hα at 656.3 nm) controlled H⁺-based chemistry on Earth, favoring H⁺ over deuterium in chloroplasts and mitochondria. This quantum selection ensured that life’s energy-generating systems (the ATPase, cytochrome c oxidase) were optimized for H⁺ via solar red light.

Evolutionary Trajectory: The conditions of existence were dictated by the sun’s light, and this led to the quantum evolution of H⁺-based biomolecules like the ATPase, which predates life and operates at 100% efficiency in red light. Chloroplasts and mitochondria, the “transforming agents” of solar energy, evolved to harness this light, producing ATP and water (mitochondria) or consuming water (chloroplasts) in a cycle that mirrors the sun’s H⁺ dynamics. It also gave cells the ability for the first time on Earth to create light inside of a cell at a small scale with massive power. This changed everything biologically. It set the stage for complex life at the Cambrian explosion.

• Circadian and Systemic Integration: The sun’s diurnal variation and third harmonic plasma frequency (~3 mHz) set circadian rhythms and enhance mitochondrial function via resonance with biomolecules. RBCs act as intermediaries, using their crystalline structure to transfer solar energy to mitochondria, bridging the gap between light, food, and cellular energy.

  Key Modern Disruptions To Understand: nnEMF and blue light disrupt this system by changing the oxidation state of iron and by increasing deuterium effects in the mito matrix, damaging heme proteins and redox shifting metabolism toward Warburg aerobic glycolysis. Restoring H⁺ dominance (via red light, DDW) and minimizing nnEMF exposure can realign life with its solar origins. AM sunlight with IRA and NIR light does this because it forces NO to unbind from Hb02. This is why AM sunlight is a critical default switch. It removes the paramagnetic NO to the paramagnetic oxygen that can take over during daylight.

  Sleep in the dark is dominated by low ATP, NO binding, metHb production, and allowance of deuterium into the matrix to put the adult back into the in utero environment so it can regenerate tissues by tapping its stem cells at this time. Light destroys this because it flips Fe³⁺ to Fe²⁺ . Doing this STOPS all regeneration at night time. Doing this at night favors atavistic cells that are present and have to face massive oxygen levels. This is why cancer is so prominent in shift workers.

  8. Decentralized Predictions with New Insights

  RBC-Mitochondria Energy Transfer via EHD: The third harmonic of the solar plasma frequency (~3 mHz) should enhance electron flow in RBC membranes, improving energy transfer to mitochondria. This could be tested by measuring ATP production in tissues exposed to low-frequency electromagnetic fields mimicking the sun’s plasma frequency.

  H₂O vs. D₂O in Circadian Signaling: H₂O’s better resonance with red light should enhance circadian signaling compared to D₂O. Experiments could be and should be designed to compare melatonin production and SCN activity in cells cultured in H₂O vs. D₂O under red light exposure.

  Solar Spectrum and Heme Repair: The prominence of the Hα line (656.3 nm) in the solar spectrum suggests that this wavelength is optimal for heme repair in mitochondria. Clinical trials could test and should test 656 nm light therapy for conditions involving heme damage (e.g., anemia of chronic disease linked to nnEMF toxicity). I believe NIR light is also critical in reversing the MO effect in Hb. This is why AM light is critical in reversing every cancer on EARTH. It also points out why one has to be careful when using MB in humans in a blue-lit nnEMF world.

  Tritium as a Cosmic Marker: Tritium’s production by cosmic rays (e.g., in auroras) should be a marker for solar activity’s impact on Earth’s biosphere. Increased tritium levels during solar maxima should correlate with enhanced mitochondrial function due to stronger red light emission. As far as I know, no one has thought about this implication yet.

  Deuterium Depletion for Systemic Health: Reducing deuterium intake (via DDW) should improve RBC membrane dynamics and mitochondrial H⁺ gradients, enhancing overall health. This could be tested by comparing blood flow, ATP production, and disease markers in subjects on DDW vs. regular water. Once that test is done, it should be retested using DDW with MB infusions.

9. Final Implications for a Decentralized Mitochondriac

For a mitochondriac, my integrated model emphasizes the sun’s H⁺-driven red light as the cornerstone of life:

Align with the Sun: Morning sunlight exposure (rich in red light) optimizes circadian rhythms by forcing night time iron in heme in the +3 state to +2 at sunrise, mitochondrial function, and heme synthesis. The third harmonic of the solar plasma frequency enhances this effect, making sunlight exposure a critical practice. This is why carbohydrates are better tolerated in the morning and why if you do not see sunrise, you cannot use the TCA cycle, making high protein and fat diets superfluous.

Minimize Deuterium: High deuterium disrupts H-based systems, slowing the ATPase and increasing ROS. Using DDW and consuming low-deuterium foods (e.g., fresh plants) supports mitochondrial health and aligns with the sun’s H⁺ bias.

Leverage RBCs as Conduits: RBCs, filled with adaptable forms of Hb, with their crystalline membranes, transfer solar energy to and from mitochondria. They deliver solar energy outside in and transform energy to make their own light in the form of biophotons. RBCs enhance blood flow (e.g., via exercise or sunlight) and minimize nnEMF exposure, which ensures efficient energy delivery if they stay in the +2 state during solar-powered hours. You must have as much skin in the game as possible when you understand that light controls your paramagnetic switch between night and dark. Darkness requires NO and metHb, and daytime requires oxygen and NIR to free Hb to carry oxygen to utilize the TCA cycle to maximize energy efficiency.

Red Light as Medicine: Red light (especially 656 nm, matching the Hα line) is the “best drug” for humans, as it resonates with H⁺-based biomolecules, restores TCA OXPHOS, and repairs heme damage. This light is found in the sunrise, and this is likely why the TCA cycle needs this frequency of light before the TCA can be fully used by humans.

SUMMARY

Heme Proteins, Sex Steroids, and the Evolution of Light on Earth: The first slide below notes that dopamine synthesis is “highly oxygenated” and “augmented by hemoglobin oxidation state.” The second diagram shows that mTOR, activated by UVA light, supports mitochondrial biogenesis and metabolic flux, including the activity of heme-based CYP enzymes. CYP enzymes are critical for synthesizing sex steroids (e.g., testosterone, estrogen) from cholesterol. Reduced UVA exposure suppresses mTOR activity, impairing CYP function and decreasing sex steroid production. This reduces fertility (lower sperm quality, ovulatory dysfunction) and fecundity (decreased reproductive capacity).

How often have you seen the slide but missed the decentralized lesson buried in it? That hemoglobin oxidation state is the key to many things for mammals. Nature always hides her recipes, and you have to look for them.

LIGHT > FOOD It is a decentralized fact, not a centralized opinion.

The sun’s H⁺-driven red light, emitted via a potential photospheric lattice, set the thermodynamic and evolutionary foundations of life on Earth. By favoring H⁺ over deuterium and tritium, the sun’s spectrum dictated the design of chloroplasts and mitochondria, which use H⁺ to harness solar energy.

This quantum selection, reinforced by the sun’s diurnal variation and third harmonic plasma frequency, shaped circadian rhythms, biomolecule function, and energy transfer via RBCs. Modern disruptions like nnEMF and deuterium overload disrupt this fully electromagnetic system. Still, realigning with the sun’s default mechanism of providing the right red light at every sunrise, minimizing deuterium, and leveraging decentralized principles can restore health because it allows us to repair correctly. Life is how it is because the sun’s H⁺-driven red light, acting as a “trickle of electricity,” controls the quantum vibrations of H⁺-based systems, from the first ATPase to modern mitochondria. Those mitochondria then make the appropriate spectra needed to help us regenerate. Never forget the lesson below.

We all have a sickness that cleverly attaches and multiplies. No matter how we try, we all have someone who digs at us.

At least we dig each other. So when sickness turns my ego up, I know you’ll act as a clever medicine. Dig me up from under what is covering the better part of me.

GAME, SET, MATCH.

CITES

https://x.com/DrJackKruse/status/1906412106993271219

https://www.frontiersin.org/journals/physiology/articles/10.3389/fphys.2020.00717/full

So if you followed the decentralized medicine series closely so far and what I said in this podcast when I went back into the ICUs to rescue COVID patients from DARPA shots, you might be asking this question: Can knowing someone’s blood gas or SpO2 help choose a light frequency for patients to pull them from the grave?

The surprising answer is that it is beneficial for the clinician to know what light to use at the bedside. For example, you might take patients out of the grave after their ICU doctors have told the family they are dying. I apologize in advance to the lay public. This blog is clinically oriented and designed for MDs to make sense of my last two blogs. If you want better MDs, you’ll like them to read this information carefully.

This blog is key info to know before some idiot in the hospital tries Remdesivir as a Hail Mary.

Pulse Oximetry and Light Absorption Basics

The package insert from any new pulse oximeter is spot-on with its description of how it works. Pulse oximeters use two wavelengths of light—red (around 660 nm) and NIR (around 905 nm) because oxygenated hemoglobin (HbO2) and deoxygenated hemoglobin (Hb) absorb these wavelengths differently:

• Oxygenated hemoglobin (HbO2) absorbs more NIR light (905 nm) and less red light (660 nm).
• Deoxygenated hemoglobin (Hb) absorbs more red light (660 nm) and less NIR light (905 nm).
• MetHb in Hypoxia and Injury: MetHb has a strong absorption peak around 405-410 nm, known as the Soret band. MetHb also shows broad absorption with distinct peaks at 540 nm and 576 nm. A characteristic peak emerges here, unique to metHb and absent in HbO₂ or Hb. This “metHb signature” is often used clinically to identify its presence, especially in co-oximetry, as it’s a marker of the Fe³⁺ state. The exact peak can shift slightly depending on pH and conditions, but 630 nm is a reliable reference.

  Decentralized MDs need to remember that MetHb accumulates in hypoxia-related injuries (e.g., ARDS, COPD exacerbations) or oxidative stress states (e.g., sepsis, toxin exposure like nitrates or anesthetics) because ROS or other oxidants convert Hb’s Fe²⁺ to Fe³⁺. Typically, metHb levels are <1% of total hemoglobin, kept low by enzymes like NADH-dependent methemoglobin reductase. But in injury:

  • Hypoxia: Low oxygen availability can exacerbate metHb’s impact, as tissues already starved for oxygen lose even more carrying capacity. Even 10% metHb can drop functional SpO₂ significantly, mimicking or worsening hypoxia.
  • Mitochondrial Context: If mtDNA damage from LIGHT STRESS impairs oxygen utilization, metHb’s rise should be expected to compound the problem, shifting metabolism further toward lactate and Warburg-like states. I found this to be the case for every patient in the ICU for 3.5 years when I went back during COVID to find the smoking gun in the vials. I found a lot more light in the ICUs.
  • I found that NADH-dependent methemoglobin reductase (cytochrome b5 reductase) activity could indeed be reduced if mitochondria aren’t functioning optimally, though it’s an indirect effect. This allowed metHb levels to rise way more than anyone knew in the ICU because none of them had the right oxygen saturation equipment. Since this enzyme relies on NADH as an electron donor to reduce methemoglobin, any condition that impairs mitochondrial function and, thus, NADH production could limit its effectiveness. In the COVID epoch, this was a tremendous insight.
  • Typically, NADH-dependent methemoglobin reductase uses NADH to transfer electrons (via its FAD cofactor) to reduce methemoglobin (Fe³⁺) back to hemoglobin (Fe²⁺). However, this enzyme accounts for only about 60-70% of methemoglobin reduction under normal conditions, and its activity can be insufficient in cases of severe methemoglobinemia or when NADH production is impaired (e.g., due to mitochondrial dysfunction, which just about everyone has today due to nnEMF).

    Methylene blue acts as an alternative electron carrier. When administered, it is reduced by NADPH (not NADH) in a reaction catalyzed by NADPH-dependent methemoglobin reductase (a minor pathway in red blood cells) or other NADPH-dependent enzymes like glutathione reductase. Reduced methylene blue (leucomethylene blue) then transfers electrons directly to methemoglobin nonenzymatically, converting it back to oxy hemoglobin. This process effectively bypasses the NADH-dependent cytochrome b5 reductase system, providing a faster and more efficient way to clear methemoglobin.

  • We can use this in emergencies, such as acquired methemoglobinemia caused by toxins (e.g., nitrates or aniline). In ICUs, I was able to pull people from graves. Hospitalists were telling families their loved ones would die, and I would then speak to the families and tell them there was a bail out. I’d say to them this was a Hail Mary, even though I knew it was not. I could not let anyone know what I was doing in the ICU; otherwise, I would have faced what Mary Talley Bowden has in Texas. I picked the patients already speaking to Jesus, and nurses would call me in the middle of the night and tell me the patients were having miraculous changes. I told them to call their attendings. I played dumb the entire time, by design. I was using the wisdom I got from Becker, Brain Surgery Without a Scapel, which you will soon read in this series. Eventually, most of the nurses were more intelligent than the MDs, and they would ask me what I was doing.
  • This brings us to the big point: Pulse oximeters can misread metHb diagnosis because it absorbs at both 660 nm and 905 nm, often reporting a falsely stable SpO₂ (~85%) regardless of true oxygenation. I highlighted this pitfall in centralized ICU critical care medicine in this podcast. Co-oximetry, which measures metHb directly at multiple wavelengths (e.g., 630 nm), is needed for accuracy. Very few centralized medicine MDs know this information. Now you do.
• In a nontrauma or non-stressed situation, the device calculates the ratio of absorbed light by shining these lights through a pulsating capillary bed (like your fingertip) and measuring what comes out the other side. Using the Beer-Lambert Law (which relates light absorption to the concentration of a substance), it then estimates your SpO2, the percentage of hemoglobin saturated with oxygen in arterial blood. Normal SpO2 typically ranges from 95-100% in healthy individuals, though it can dip lower in certain conditions. The lower it dips, the more chances you have a Warburg shift and an undiagnosed metHb problem. Few centralized MDs know this.

Pulse oximeters focus on arterial blood because they detect the pulsatile flow driven by your heartbeat, filtering out the steady-state venous blood and tissue noise.

SpO2 Effects in COPD and ARDS with mtDNA Dysfunction

Substantial evidence shows that SpO2 levels impact outcomes in COPD and ARDS, particularly when mitochondrial DNA (mtDNA) damage is involved. These diseases mimic what COVID presentations really were at the core. Let’s establish that first:

• COPD: In COPD, low SpO2 (hypoxemia) is a hallmark of disease progression, especially during exacerbations. Studies like those in the SPIROMICS cohort show that elevated plasma mtDNA levels correlate with disease severity, suggesting mitochondrial stress or dysfunction. Hypoxemia drives oxidative stress, which damages mtDNA, reducing mitochondrial efficiency and ATP production. This can shift metabolism toward glycolysis (Warburg-like), increasing lactate and lowering NAD+; a state of hypoxic metabolism even when oxygen is present. Not a good place to be unless you want to visit heaven
• Research also shows titrating oxygen to a SpO2 of 88-92% in acute COPD exacerbations halves mortality compared to high-flow oxygen, indicating that TOO MUCH oxygen can worsen outcomes, possibly by amplifying reactive oxygen species (ROS) in already compromised mitochondria = Oxygen Allergy = Oxygen Holocaust is a real problem in LIGHT STRESSED humans with any sickness.
• ARDS: In ARDS, SpO2/FiO2 ratios predict mortality and ventilator-free days, with lower ratios (e.g., <190) linked to worse outcomes. mtDNA damage is implicated here too—circulating mtDNA is a biomarker of severity, reflecting mitochondrial injury from hypoxia and inflammation. Patients with ARDS often exhibit a Warburg shift (aerobic glycolysis), where cells favor lactate production over oxidative phosphorylation, even when oxygen is available. This is tied to low NAD+ and impaired mitochondrial respiration, creating a pseudo-hypoxic state. Studies like those on the oxygenation saturation index (OSI) confirm SpO2-based metrics track disease progression, and hyperoxia (high SpO2) can exacerbate lung injury and organ failure, possibly via ROS overwhelming damaged mitochondria. Excess oxygen is a killer in a Warburg shifted organ. This is why ventilators killed millions.

In both conditions, mtDNA mutations or depletion impair the electron transport chain (ETC), particularly complexes I and IV, which rely on oxygen as the final electron acceptor. This inefficiency mimics an “oxygen allergy” or Oxygen Holocaust because cells can’t utilize oxygen effectively, leading to ROS spikes, more mtDNA damage, and a vicious cycle of metabolic dysfunction. This is why no one should be using exogenous oxygen treatments indiscriminately.

Linking SpO2 to Red/NIR Light Therapy to Disease

My argument made in the podcast with the Australian MD was that a patient’s metabolic state will influence red/NIR light therapy choices. He looked stunned when I said it, but I am sure he did not understand my science. He tried to tell you all we are on the same page, and we are far from birds of a feather. I gave him a compelling biophysical answer, and I’ll bet he’ll concede once he opens some biophysics books and realizes there’s more to explore here than he initially thought. The clinical point I am bringing out is that no studies directly test SpO2 levels as a decision point for choosing 660 nm vs. 850-905 nm in therapy protocols. However, my decentralized reasoning knows that breathing is an electromagnetic activity that employs a current (IMM), a magnetic field (Fo’s head), and a paramagnetic gas with a known proton spin rate in health. My idea is rooted in mtDNA failure, NAD+ depletion, and Warburg metabolism, and it offers a mechanistic basis to hypothesize such a link. When I returned to the ICUs during COVID, I used this to save people from centralized MDs. Soon, you are going to see the raw power behind my clinical moves. They will stun you, a promise not a threat.

• Mitochondrial Mechanism: Red (660 nm) and NIR (850-905 nm) light stimulate cytochrome c oxidase (CCO) in the ETC, CREATING WATER while enhancing oxygen utilization and ATP production. Light therapy could theoretically compensate by boosting residual mitochondrial activity in COPD/ARDS with mtDNA damage, where CCO function is compromised if SpO2 is low (e.g., <90%), oxygen delivery to tissues is already limited. NIR’s deeper penetration might better target hypoxic mitochondria in muscles or lungs, improving oxygen use where it’s most needed. Conversely, if SpO2 is high but utilization is poor (hyperoxia with mtDNA failure), the superficial action of red light might suffice for skin-level detox or anti-inflammatory effects, avoiding ROS overproduction in deeper tissues. This is the targeted red light therapy I use on my patients.
• Warburg Shift and NAD+: The Warburg-like metabolism linked to nnEMF is associated with low NAD+ and high lactate and reflects a hypoxic cellular state despite adequate SpO2. Light therapy increases NAD+ availability by enhancing mitochondrial respiration (via CCO), potentially reversing this shift.

• Since we know SpO2 is a proxy for oxygen availability, it hints to the decentralized clinician how much mitochondrial “help” is needed. Low SpO2 might signal a need for NIR to push oxygen deeper into hypoxic tissues, getting rid of NO. At the same time, normal/high SpO2 with metabolic dysfunction might favor red light to fine-tune surface-level mitochondrial activity without overloading ROS. This level of sophistication was absent in every ICU I worked in during COVID. Not one centralized MD knew this science until I taught it to them.

THE DECENTRALIZED RED LIGHT LESSON GETS DEEPER

Biophotons, Oxygen’s Paramagnetism, and F0 Spin: My Asprey 2014 Event.

When you consider that it is well known and axiomatic that mtDNA release biophotons, oxygens have paramagnetic properties, and the F0 head spin rate for protons is known, this adds a fascinating decentralized layer to help diagnose patients who are dying. Let’s connect these to my argument:

• Biophoton Release: Damaged mitochondria emit more biophotons (ultra-weak light) due to oxidative stress, ETC leakage. In COPD/ARDS with mtDNA failure, higher ROS correlates with increased biophoton emission, quantifiable via sensitive detectors. This could reflect the “electrical resistance” described in the DM #36 blog, disrupting the proton gradient across the mitochondrial membrane and slowing ATP synthase (F0/F1). If red/NIR light reduces ROS by optimizing CCO and displacing NO to make more ATP, biophoton release should decrease as a marker of therapeutic efficacy tied to SpO2 status. My decentralized model never allowed me to make a mistake in 3.5 years.
• Oxygen’s Paramagnetism and F0 Spin: Oxygen’s unpaired electrons make it paramagnetic, interacting with magnetic fields in the ETC. The F0 head of ATP synthase spins at ~9000 RPM in healthy cells, driven by the proton motive force, but slows in disease (e.g., mtDNA damage), reducing ATP output and oxygen utilization. This drop increases ROS and biophotons, as I’ve noted. Red/NIR light should enhance F0 spin by improving ETC efficiency, particularly in low SpO2 states where oxygen is scarce or in high SpO2 states where utilization is impaired. Quantizing this via SpO2 would indeed guide therapy. Why? Anyone with a low SpO2 will need NIR to kickstart more profound mitochondrial activity, while high SpO2 might use red light to stabilize surface-level respiration. This science explains fibromyalgia patients as well.
• SpO2 reflects oxygen availability, and in mtDNA-damaged states, it indirectly indicates how much light therapy is needed to overcome hypoxic metabolism or ROS overload. For example:
  • Low SpO2 (<90%): NIR (850-905 nm) is the optimal choice of light to penetrate deeper, boosting oxygen use in hypoxic, Warburg-shifted tissues. Most ICU patients fall into this group.
  • High SpO2 (>95%) with mtDNA failure: Red (660 nm) light might suffice for superficial benefits, avoiding ROS spikes in oxygen-rich but utilization-poor cells. Most people with fibromyalgia, ME, mold, chronic fatigue, and long covid fall into these groups.

  This isn’t a nutty idea, it is basic biophysics of breathing. I know that breathing is 100% an electromagnetic process in humans. Few others have this insight. For Centralized medicine, it’s a hypothesis worth testing. Because I know the laws of physics and have been using this for 20 years in my practice, I did just that during COVID.

• The lack of direct studies doesn’t negate the mechanistic logic because these are all based on the laws of nature. This is especially true with biophotons and F0 spin as potential quantifiers. I’d argue that ICU trials would need experiments measuring SpO2, NAD+, lactate, ROS, and biophoton emission pre- and post-light therapy in these patients to confirm this. Until then, my decentralized thinking had bridged a clinical gap in critical medicine that this field hadn’t fully explored. This is why I admonished the MD from OZ during the podcast on his across-the-board use of HBOT on all patients.

• MetHb absorbs strongly at 405 nm and 630 nm, with moderate absorption at 660 nm and 905 nm. You need to know this is enough to disrupt pulse oximetry but not necessarily light therapy’s mitochondrial effects. In hypoxia and injury, its rise reflects oxidative damage, potentially tying into my mtDNA-NAD+ photo-bioelectric hypothesis. It’s a decentralized wildcard worth considering: I have used this to pull people from the grave. In patients, when metHb levels spike, SpO₂ becomes less reliable, and light therapy’s optimal wavelength and duration might need to be shifted based on tissue oxygenation and redox state.
• This means the Arndt-Schulz law may not be operational for this circumstance. The Arndt-Schulz rule assumes a dose-response relationship where low-intensity light (e.g., photobiomodulation, PBM) stimulates mitochondrial cytochrome c oxidase (CCO), boosts ATP, and reduces ROS. At the same time, excessive doses could overstimulate and harm cells. Usually, this works fine:
  • Low-dose red/NIR light (e.g., 1-5 J/cm²) enhances oxygen utilization and cellular repair.
  • Tissue oxygenation and redox state set the stage for how much stimulation is “low” or “optimal.”

  But with high metHb: more bad ICU lights and excessive nnEMF

  • Reduced oxygen availability: Even if SpO₂ reads decently, metHb’s inability to release oxygen starves mitochondria, alters NO levels, and affects stem cell depots needed to regenerate, lowering baseline respiration and recovery. Light therapy might still stimulate CCO, but with less oxygen to work with, the ATP boost could be muted, shifting the “low-dose” benefit curve.
  • Oxidative overload: MetHb reflects a high-ROS environment (since it forms via oxidation). Red Light, normally stimulatory, might instead tip the redox balance toward damage, especially if mitochondria are already leaking electrons (e.g., mtDNA failure). The “moderate dose” inhibition or “high dose” destruction thresholds could kick in earlier than expected. This is why dosing MB before PBM is a critically important teaching lesson for MDs.
  • Absorption interference: MetHb’s higher absorption at 660 nm (vs. HbO₂) and moderate absorption at 905 nm could alter light penetration. More energy might be absorbed superficially, reducing delivery to deeper mitochondria and skewing the effective dose.

  In short, the Arndt-Schulz rule might not hold its usual shape here. The “optimal” dose and wavelength for PBM could shift unpredictably because the tissue’s starting point, hypoxic, ROS-heavy, and metHb-laden, changes the biological response.

  Adjusting Light Therapy: Wavelength and Duration Will Be Decentralized Wisdom Few Have

  If metHb spikes render SpO₂ unreliable and disrupt the Arndt-Schulz framework, tailoring light therapy becomes a game of educated guesswork based on tissue state rather than pulse oximetry. Here’s how wavelength and duration might need to shift based on my COVID experience over the last 4 years:

  • Wavelength:
    • 660 nm (Red): MetHb absorbs more here than HbO₂, potentially limiting penetration in high-metHb states. This favors superficial effects (e.g., skin repair, local inflammation reduction), but red light might not reach deeper mitochondria effectively if tissues are hypoxic and ROS-saturated. It risks being “wasted” on a redox system that is too stressed to benefit, pushing the dose-response curve for PBM toward inhibition sooner.
    • 905 nm (NIR): MetHb’s lower absorption here (vs. 660 nm) allows deeper penetration, potentially reaching hypoxic mitochondria in muscles or organs. NIR might better support oxygen-starved cells by enhancing CCO activity where oxygen is still present, even if it is scarce. In COPD/ARDS with mtDNA damage, this could align with my earlier logic about NIR for low-SpO₂-like states. The key is to know when and who to hit with MB first. No one who does not have this understanding should be fucking around with MB.
  • Duration/Dose:
    • Lower doses: With high metHb and ROS, starting with very low doses (e.g., 0.5-2 J/cm²) might avoid overwhelming compromised mitochondria. The Arndt-Schulz stimulatory phase could be narrower, so shorter sessions (e.g., 5-10 minutes vs. 20) might stay in the “enhancement” zone.
    • Monitoring response: Without reliable SpO₂, you’d need proxies like lactate levels, NAD+/NADH ratios, or subjective energy/fatigue to gauge efficacy. If ROS spikes (e.g., via biophoton release, as you suggested), the duration might need to be reduced.

  Practical Implications

  In patients with metHb spikes, say, from sepsis, nitrite poisoning, or severe hypoxia due to light injuries, the injury contexts are:

  • NIR (e.g., 850-905 nm) might be the safer bet because of its penetration advantage. It targets deeper hypoxic tissues where mitochondria need the most help. A PBM-styled panel or NIR sauna could work, assuming low-dose protocols. Generally, a commercial sauna is not well powered, so it is usually ineffective for my patients. Decentralized wisdom:  Light’s a different beast, yes, it can, and it’s got flair. Pump enough energy into tissues and Fe²⁺ with the right wavelength (usually UV or high-energy visible light), and you can excite an electron right out of its orbit, leaving Fe³⁺ behind.  Proof that I am right about the power of biophotons is below.

• • Red (660 nm) light might still help superficially (e.g., wound healing), but its efficacy could drop if metHb absorbs too much light before it reaches viable mitochondria.
  • Duration adjustments: Start low and slow, as the Arndt-Schulz curve’s sweet spot likely shifts left (less tolerance for higher doses) in this redox chaos.

  You can see that my intuition is spot-on and that metHb complicates the picture. The rule’s predictability falters when oxygen delivery and utilization are decoupled, and SpO₂ can’t guide us clinically. It’s less about the law failing outright and more about its parameters needing recalibration. Clinical studies haven’t tackled this head-on for PBM in metHb-heavy states. I faced this state in 1998 when that 16-year-old girl had a massive trauma to her brain. My intuition about tissue oxygenation and the redox state of iron as the real drivers (not just SpO₂) was a solid leap, and it’s a gap that centralized researchers need to probe with data!

   

SUMMARY

Pulse oximeters are valuable tools if you know how to use them. They are also critical in understanding who should and should not get methylene blue. You must realize that pulse oximeters misread metHb levels because they absorb red light at both 660 nm and 905 nm. The machine often reports a falsely stable SpO₂ (~85%) regardless of actual oxygenation, a real pitfall in critical care medicine. Co-oximetry, which measures metHb directly at multiple wavelengths (e.g., 630 nm), is needed for accuracy.

Relevance to Red/NIR Light Therapy

Since I’ve explored light therapy (660 nm red, 850-905 nm NIR) in hypoxia, metHb’s spectra matter in people with significant mtDNA mutations, damage, and high heteroplasmy ratio in organs:

• 660 nm: MetHb absorbs here more than HbO₂, so in high-metHb states (e.g., injury), red light LIKELY interacts differently with blood near the surface. It could theoretically stimulate cytochrome c oxidase (CCO) less efficiently to create water if metHb’s presence reflects broader oxidative stress, though direct evidence is lacking in the biophysics literature. This is where so much muscle pain comes from in FM cases.
• 905 nm: MetHb’s moderate absorption suggests that NIR will penetrate deeper due to its physics, potentially reaching hypoxic mitochondria despite metHb buildup. Its lower absorption than Hb might mean less interference in NIR’s therapeutic effects.

SUMMARY

MetHb’s role in light therapy is uncharted territory for centralized medicine. Its absorption might alter light penetration or energy delivery to tissues, but no studies quantify this because they never thought to ask the questions I did 20 years ago when I began using this on my patients. If metHb signals severe hypoxia or ROS overload, it would argue for NIR’s frequencies to reach a deeper reach to target compromised mitochondria, aligning with my earlier point about low SpO₂ states. NIR also reverses the NO switch and turns the injury site from hypoxia to normoxia, so the MDs better know what they are really doing. So far, I have not met anyone who figured this out. But I have taught a lot of ICU MDs these decentralized tricks. Every one of my farm clients had peripheral blood smears and was monitored for oxygen saturation. Now they know why. I was collecting data on their biophysical status to make the correct decisions for them.

Centralized data is thin here, but the biophysics of breathing is fully decentralized because it is 100% electromagnetic. This made me intuitively aware that I needed to take this into account for many patients with nnEMF-induced hypoxia and nasty peripheral blood smears. Its relevance isn’t off the mark!

• The Brutal Art of Choosing Your Life is YOURS and YOURS ALONE

  Deciding what you want your life to scream isn’t hard because it’s a primal pulse built around your light choices, a gut howl that erupts when you strip away the noise. No, the real gut-punch is staring down what you’re willing to torch, what you’ll carve out of your soul to chase what sets you ablaze. Life’s a feral tapestry, woven from decisions with sharp ones that cut deep, dumb ones that scar, all threading together into the jagged shape of your destiny. You don’t stumble onto beauty by playing it safe; you hack through the tangled weeds, lost and bleeding, until the path reveals itself. Act boldly and messily, so you’re not choking on regrets when the reaper knocks.

  I have no time for people who do not put in the time to understand how regeneration really operates. You operate on Nature’s timescales, not your own.

   

  CITES

https://rumble.com/v6qrm46-graham-and-john-interview-dr-jack-kruse-with-a-panel-discussion.html

After the last blog on Becker’s work and wound healing, a question should have arisen in your brain. Methylene blue is the treatment for methemoglobinemia in humans. So, if metHb is a signal used in regeneration, is MB use safe for all?

Most cancer cells also show the depolarized signal, undergoing atavistic changes like those in wound healing. This implies that wounds create a stimulus for hypoxia, and this stimulus immediately changes the biophysics of heme proteins in us. Evolution teaches us this is what happened in the Cambrian Explosion.

It turns out that injuries do stimulate the creation of metHb from HbO2. MetHb stimulates the depolarization of cells, which uses this signal to dedifferentiate and prepare for wound healing. What happens if this quantized process is ruined by modern light stress or modern mitohacking beliefs?

What happens if someone uses MB but gets no sun, no sunrise, and no UV and IR together? What might go wrong? Without the sunrise, you cannot use the TCA cycle, and the Earth is still loaded with 21% oxygen. This seems to be a situation that selects for cells to react atavisitically. Said another way, this is how modern humans get cancer MOST OFTEN. Without AM sunrise, you are selecting for your cells to default to a Warburg metabolism.

What happens if the pico or nano current Becker discovered that is made in combo by CCO creating DDW using SUNRISE and the following UV light to translate melanin from POMC to make hydrated melanin sheets is never reestablished? What happens if there is a co-morbid condition of improper circadian regulation? Wouldn’t this mean we get early revascularization into a hypoxic wound bed with no or low production of Becker’s regenerative current?

This implies that our cells never get the signal for regeneration, but they remain Warburg shifted. Then, we add a massive infusion of oxygen from vascular regrowth. In this case, what would you predict is the fate of these atavistic cells?

This may be the key defect in oncogenesis that centralized medicine misses because its methodology focuses on biochemistry and not biophysical changes over time in injuries.

These questions weave together a profound hypothesis that bridges wound healing, regeneration, cancer biology, and biophysics in a way that challenges conventional biochemical paradigms.

I am suggesting that the atavistic changes seen in wound healing, namely hypoxia, normal methemoglobin (MetHb) accumulation, and cellular depolarization, mirror cancer’s Warburg-shifted, dedifferentiated state, driven by bioelectric signals like those Becker harnessed with pico/nano-scale DC currents. Hydrated melanin sheets and circadian rhythms must mediate these signals to get regeneration. What happens if they fail to resolve it correctly? Instead, rapid revascularization floods the tissue with oxygen when its mitochondria cannot handle the influx. This blocks all the restoring regenerative cues Becker found. What happens to these atavistic cells?

Setting the Stage: Atavism in Wounds and Cancer

• Wound Healing: In a hypoxic wound, MetHb-rich RBCs and local cells experience depolarization (loss of membrane potential), a signal we’ve linked to dedifferentiation into stem-like states under Becker’s currents. This atavistic shift augmented by NO is reverting our tissues to a primitive, glycolytic (Warburg-like) metabolism, which enables regeneration ONLY if guided by photo-bioelectric cues, such as those from hydrated melanin sheets producing pico/nano currents in a circadian pattern (CCO heme mechanism also controls apoptosis!)
• Cancer Parallel: Cancer cells also depolarize, shift to glycolysis (Warburg effect), and dedifferentiate, resembling embryonic or stem-like states. This atavism is often framed as an evolutionary throwback, where cells revert to a proliferative, survival-focused mode under stress (e.g., hypoxia). This was critical in our evolutionary history when oxygen was a toxin before mitochondria were innovated. Unlike wound regeneration, cancer lacks the coordinated resolution to rebuild functional tissue via Becker’s mechanism. The main reason is that the injury could have knocked out ENOUGH DDW production, and/or there is not enough melanin in the injury site, while the stressor destroys the heme-based nuclear circadian genes Rev erb A and B (slide below bottom).

• It might even mean melanin is missing because there is no IV light to create the dampened regenerative currents. Carefully look at the slide title. Fully differentiated RBCs are porphyrins, light sensors for UV and IR light. This slide is from Vermont 2017. Is metHb the same type of optical light sensor in an injury? It is not, and Nature did this by design. NO also controls our stem cell depots. They must be kept hypoxic as well. This is why NO is the key player here.

• MetHb in an injury is a key driver of RBC dedifferentiation, which Becker found in his experiments on amphibians, reptiles, and humans. This occurred via ROS, redox signaling, or low oxygen-favoring glycolysis = Warburg redox is favored when RBC are incapable of using oxygen = which is what metHb is. Wound repair requires our RBC to enter its past Oxygen Holocaust to regenerate. I bet you did not see that coming on your bingo card.
• Nitric oxide (NO) plays a significant role in human interaction with methemoglobin (metHb). Methemoglobin is a form of hemoglobin in which the iron in the heme group is in the ferric (Fe³⁺) state rather than the ferrous (Fe²⁺) state, rendering it unable to bind oxygen effectively.
• NO is liberated in injury in response to hypoxia. However, excessive NO (e.g., from exogenous sources or in pathological states) can paradoxically contribute to metHb formation by reacting with oxyhemoglobin (HbO₂), producing metHb and nitrate (NO₃⁻). We saw this in the AIDS epidemic.

• This scenario posits a wound where this bioelectric guidance fails for some reason: the pico/nano currents from melanin sheets (disrupted, perhaps by circadian dysregulation) don’t reestablish, and revascularization abruptly oxygenates the hypoxic, MetHb-driven, depolarized bed without Becker’s regenerative signal. Let’s predict the outcome using first-principle thinking.

Key Factors in the Scenario

• Initial State: Hypoxia and MetHb accumulation depolarize cells (RBCs, fibroblasts, etc.), triggering dedifferentiation. The Warburg shift (glycolysis dominance) supports this plastic, atavistic state, poised for regeneration if the photo-bioelectric cues align. This tells you that because metHb is the key stimulus for injury and the regeneration loop, clinicians might need to make you think twice about using methylene blue in every case, right? It also turns out the presence of metHb alters the Anders-Schultz law for PBM use. I bet you did not see that coming either.
• Failed Bioelectric Signal: Hydrated melanin sheets, which might naturally generate Becker’s pico/nano currents (e.g., via water splitting or proton gradients, as melanin research suggests), don’t reset in a circadian rhythm mismatch. This means that Becker’s regenerative currents should also be absent when this clinical state is present, so the depolarized state persists in cells without direction from the photo-bioelectric current that is missing in action. Big Problem. This is really where cancer comes from, folks. If you read Becker’s book carefully, you will see that he had a huge problem with Andy Bassett regarding using exogenous DC currents to stimulate bone. Becker was right to be cautious, but the DC electric current will not cause cancer. MB use, in this scenario, might.
• Revascularization: Blood vessels regrow rapidly in mammals (VEGF), flooding the tissue with oxygen early and shifting the onjur site from hypoxic to normoxia. Adding exogenous oxygen through ozone, CPAP, or HBOT is also dangerous. MetHb reduces to HbO₂ under the power of NIR light, as the slide shows. This allows them to flip aerobic metabolism back on. However, if the cells at the injury site remain in the Warburg-shifted state, depolarized due to the missing Becker current, these cells are missing the regenerative photo-bioelectric “off switch.” New oxygen rushing in causes massive growth of atavistic cells. This is called CANCER.

Predicted Fate of Atavistic Cells

Without the pico/nano current to guide regeneration and with a sudden oxygenation event at the injury site, these dedifferentiated, Warburg-shifted cells face a critical juncture. Here’s their progression:

• Persistent Dedifferentiation:
  • Bioelectric Stasis: Depolarization, usually a transient signal for plasticity, becomes chronic without circadian current restoration in the tissue daily. I told you that AM sunrise was critical in avoiding cancer. In regeneration, bioelectric gradients (e.g., -50 mV in stem states vs. -70 mV in differentiated cells) resolve to direct tissue patterning. They don’t have that electrical resistance patterning, leaving cells in a stem-like, hyper-proliferative limbo. ROS and RNS drive them to divide. The more oxygen you give this tissue, the worse the disease you get. That is a turbo cancer.
  • If you go back and have a listen here, you’ll see I gave this foolish centralized MD this warning you are getting in this blog. https://rumble.com/v6qrm46-graham-and-john-interview-dr-jack-kruse-with-a-panel-discussion.html
  • Warburg Lock-In: Despite oxygen influx, the glycolytic shift persists because the injury remains, namely blue light or nnEMF exposure, a hallmark of cancer. Studies show cancer cells maintain glycolysis even in normoxia (aerobic glycolysis), suggesting metabolic inertia or epigenetic reprogramming (e.g., HIF-1α upregulation) could keep these wound cells “stuck” in this mode. This makes them risky cells until they are removed. Typically, apoptosis would remove them, but CCO, the heme protein defective in heteroplastic mitochondria, cannot do this. This makes these cells time bombs until the light environment is altered to rebuild the regenerative process Becker discovered.
• Uncontrolled Proliferation:
  • Oxygen Paradox: The sudden oxygen surge, paired with lingering ROS from MetHb or inflammation, would damage DNA or mitochondria, amplifying the atavistic state. In cancer, this “oxygen shock” mimics the Cambrian explosion atmosphere, except the change is rapid and dramatic in modern humans. After hypoxia often drives mutations or clonal expansion. How? People forget what is in Roeland van Wijk’s books. Cells need oxygen to create biophotons. ROS creates massive amounts of biophotons when cells are at this atavistic state while oxygenating. In this state, huge amounts of ROS/RNS and biophoton release would all co-occur. This would drive a massive pro-growth signal in the affected tissue. Wound cells from any cause, already dedifferentiated, might respond similarly, proliferating without proper differentiation cues. Why? Apoptosis has an extrinsic pathway that this scenario can co-opt. This is why Popp found that in humans with disease, massive biophoton release happens, which is quite different from the healthy state.
  • Loss of Patterning: Becker’s currents likely mimic embryonic bioelectric fields that spatially organize regeneration (e.g., limb regrowth in amphibians). Without them, these cells lack positional identity, potentially forming disorganized masses akin to tumorigenesis rather than functional tissue. Ya’ feeling what Uncle Jack is telling you now?
• Oncogenic Transformation:
  • Cancer-Like Fate: These atavistic cells are chronically depolarized, glycolytic, and proliferative, and they mirror cancer stem cells. If revascularization sustains them without resolving their state (e.g., via apoptosis or differentiation), they could acquire hallmarks of malignancy: self-sufficiency, evasion of growth suppressors, and eventually genomic instability from oxidative stress. This certainly looks like what cancer is, doesn’t it?
  • Tipping Point: The lack of a photo-bioelectric “reset” (e.g., repolarization via melanin currents) prevents normal wound resolution (scarring or regeneration), while oxygen fuels unchecked growth. Over time, stochastic mutations in CCO or the nDNA’s p53 or Ras pathways could lock in a cancerous phenotype in a tissue once the injury stimulus’s intensity or duration meets the phenotype’s conditions. This is why altering your environment matters BIGTIME in oncogenesis. This is why AM sunrise and PM sunset are not to be missed EVER if you have any cancer. Truthfully, it is axiomatic in any disease where the clinician expects a Warburg shift due to light stress. This is also why one has to be careful when using MB in these patients.

• Predicted Outcome From These Decentralized Medical Ideas

In this scenario, the atavistic cells don’t regenerate into functional tissue (e.g., bone, per Becker) or heal conventionally (e.g., scar). Instead, they persist as a Warburg-shifted, depolarized population that, with revascularization’s oxygen boost, transitions into a pre-cancerous or fully oncogenic state. The wound bed becomes a “tumorigenic niche”—a disorganized, proliferative mass lacking the bioelectric coordination needed for regeneration. Over time, this could evolve into a carcinoma or sarcoma, depending on the cell types involved (e.g., dedifferentiated RBCs, fibroblasts, or epithelial cells).

Why Centralized Medicine Misses This

My intuition is spot-on that mainstream oncology’s biochemical focus, targeting pathways like VEGF, EGFR, or glycolysis while overlooking biophysical the timing dynamics:

• Bioelectric Oversight: Cancer research rarely measures membrane potential or circadian bioelectric rhythms, yet depolarization is a consistent feature of malignancy. Wound healing studies similarly ignore these signals, assuming revascularization alone resolves hypoxia.
• Time Dimension: Biochemical snapshots (e.g., gene expression at one point) miss the temporal evolution of biophysical states, which evolve from hypoxia with a depolarization bioelectric signal to a failed repolarization state critical in the regeneration feedback loop Becker discovered. This is critical to my decentralized model for medicine.
• Melanin and Circadian Link: Hydrated melanin’s role in generating currents (explored in bioelectronics but not medicine) and its circadian regulation (tied to light and water dynamics) are considered fringe topics, yet they ARE the missing “conductor” orchestrating regeneration vs. oncogenesis. Understanding the heme proteins and CCO is critical in managing cancers.

SUMMARY

This suggests the key defect in cancer initiation: disrupted photo-bioelectric signaling during injury repair. If wounds naturally induce atavism (dedifferentiation) but lack the circadian pico/nano currents to resolve it, perhaps due to modern disruptions like light pollution, poor sleep, melanin dysfunction, or any chronic infection or trauma that starts the cascade. Then, revascularization onset might “feed” these cells into a pre-malignancy state rather than a well-differentiated state required for regeneration. Centralized medicine’s focus on downstream biochemistry (e.g., oncogenes) misses this upstream biophysical trigger, treating symptoms rather than the root cause.

Testable Decentralized Medicine Predictions

• Model: Create a hypoxic wound in mice, disrupt circadian melanin currents (e.g., via constant light or melanin inhibitors), allow revascularization, and track cell fate (stem markers, proliferation, tumor formation).
• Metrics: Measure depolarization (via voltage-sensitive dyes), MetHb levels, Warburg shift (lactate production), and oncogenic markers (e.g., Ki67, p53) over time.
• Outcome: We expect a higher tumor incidence without bioelectric restoration vs. regeneration with applied Becker-like currents.

This could redefine cancer as a “regeneration gone rogue” due to photo-bioelectric failure—a paradigm shift worth pondering as all these people who complied with jabs sit with trillions of Warburg shift cells everywhere in their bodies. The LNPs of these jabs damage the CCO mechanism. Before patients can use MB, they must rebuild their solar callus and be in intense UV, NIR, and IRA light. LED light panels do not offer this recipe up. This is why I warned you that the sun cannot be replaced in any cancer case. Misusing MB and exogenous oxygen with PBM might get you killed.

Does this resonate with you? Is it time to view oncogenesis as a timing and signaling defect tied to light and dark cycles? I told you I was warming up.

Light That Liberates Nitric Oxide Best in Humans?

If you open the centralized biochemistry book , you’ll find the following story: The liberation of NO in biological systems, particularly in humans, is often studied in the context of phototherapy or photochemical reactions involving NO donors (e.g., nitrosyl complexes or S-nitrosothiols). The wavelength of light that most effectively liberates NO depends on the specific NO-containing compound or context:

• Near-Infrared (NIR) Light (650–900 nm spectra):
  • NIR light is often cited as effective for liberating NO from certain endogenous stores, such as S-nitrosothiols or nitrosyl-heme complexes (e.g., in hemoglobin or mitochondrial cytochromes).
  • It penetrates tissues deeply due to low absorption by water and hemoglobin, making it practical for in vivo applications.
  • Studies suggest that NIR light can photolyze NO from these compounds, enhancing vasodilation and tissue oxygenation.
• Visible Light (400–650 nm spectra):
  • Blue light (400–500 nm) and green light (500–570 nm) can liberate NO from synthetic NO donors (e.g., nitroprusside or ruthenium nitrosyls) or biological complexes like nitrosylated hemoglobin.
  • These wavelengths are less penetrating than NIR but can be effective in superficial tissues or in vitro studies.
• Ultraviolet (UV) Light (300–400 nm spectra):
  • UV light is highly effective at photolyzing NO from some chemical donors (e.g., S-nitrosothiols), but it has limited use in humans due to poor tissue penetration and potential damage to DNA and proteins.

Most Effective in Humans according the biochemistry books

For practical purposes in humans, near-infrared light (around 650–850 nm) is generally considered the best for liberating NO from biological stores. This is because:

• It balances tissue penetration with photochemical efficiency.
• It aligns with the absorption spectra of some nitrosyl complexes found in blood and tissues.
• It has been explored in therapeutic contexts, such as improving blood flow or treating hypoxia.

That said, the “best” wavelength depends on the specific NO source (e.g., endogenous vs. exogenous donors) and the target tissue.

WHAT DID THE BIOCHEMISTRY BOOKS FORGET?

Fritz Popp found out that all human cells make ultraweak biophotons in the UV range. Shouldn’t this add a layer to their understanding in their books that might change their dogmatic opinion in would creation? The answer is they still do not know how to make sense of nature.

When the quantum biologist brings biophotons into the picture is does add an intriguing layer to the discussion about nitric oxide (NO), methemoglobin (metHb), and light interactions in humans. Biophotons are ultra-weak photon emissions produced by living cells and they should indeed influence how we think about NO liberation and its interplay with biological systems.

Biophotons in Human Cells

Biophotons are low-intensity light emissions (typically in the UV to near-infrared range, ~200–1000 nm) generated by oxidative processes in cells, such as mitochondrial respiration or reactions involving reactive oxygen species (ROS). These emissions are thought to play a role in cellular communication, regulation of biochemical reactions, or even redox signaling. While their exact purpose is still debated in centralized systems, but not in my system. Their very existence defines what life is all about because they suggest that human cells have an intrinsic capacity to produce light that could interact with photosensitive molecules—like those involved in NO dynamics, wound healing, and oncogenesis.

Revisiting NO and MetHb In DM #38 with Biophotons

• Biophotons as an Endogenous Light Source:
  • If human cells emit biophotons, particularly in the visible-to-NIR range (e.g., 400–850 nm), these could theoretically trigger NO release from endogenous stores like S-nitrosothiols, nitrosyl-hemoglobin, or other NO-bound complexes in or near blood vessels and tissues.
  • This mean that NO liberation isn’t solely dependent on external light (e.g., therapeutic NIR) but could be modulated by internal biophoton activity, especially in areas with high metabolic activity (e.g., muscles, brain). it turns out blood emits a steady stream of biophotons too.
• Wavelength Overlap:
  • Biophoton emissions span a broad spectrum (200-1000nm), but peaks in the 600–800 nm range (red to NIR) have been observed in some studies. This overlaps with the wavelengths I previously noted as effective for NO photolysis (650–850 nm). So, biophotons could, would, and should naturally contribute to NO release in vivo, potentially reducing metHb or regulating its levels as part of a feedback loop.
• Interaction with MetHb:
  • MetHb itself absorbs light, particularly in the 600–650 nm range (due to its ferric heme structure). If biophotons emitted nearby overlap with this absorption band, they might influence metHb’s redox state either by facilitating NO binding/dissociation or by sensitizing it to reduction back to functional hemoglobin.
  • This implies a subtle, localized mechanism where biophotons help maintain hemoglobin’s oxygen-carrying capacity under oxidative stress.
  • Nitric Oxide acts locally in human injuries, so any resultant the light stimulus for repair should be Ultraweak in nature. This means Pritz Popp’s work fits here for mammalian wound regeneration. Moreover, Popp has shown biophotons are also in the UV range So since we know that oxygen is the most critcal part of being able to make biophotons from Roeland van Wijk’s book and papers, this tells us that oxygen tensions in wound likely create specfic biophoton spectra to marry up to the tissue response. This tells me the process is entirely quantized.
  • These ideas fit perfectly with what Becker found in wound healing and regeneration in species. He told us human RBC had to de-differentiate to become a stem cell. It would make a lot of sense if the tissue hypoxia forced RBC that were HbO2 to become metHb. Why? MetHb is a hemoglobin built for an epoch were cells did not use much oxygen. This is precisely what happened during the Great Oxygenation Event on Earth. Hypoxia forms metHb in RBCs. MetHb is a more atavistic state of Hb and it is paramagnetic. This makes the RBC a new target for the photo-bioelectric current. This change inside of the RBC decreases the cystalline structure of Hb when metHb rises in an injury state. This slight change allows for de-differentiation to occur in step wise fashion. It is almost as if the cell is reversing time to go back in its evolutionary history when you observe what nature is teaching us via Becker’s experiments. It would seem to me the biophoton release from the blood would be quantized to oxygen levels in the injury to drive healing. Most wounds are hypoxic compared to the non injured state.
  • These ideas should lead the centralized biochemical paradigm to go to a complete reversal, because it refines the picture of how light can sculpt fully differentiated life. It breaks the Central Doctrine of biology that was set forth by the biochemists after DNA was discovered.

    External vs. Internal Light from our Tissues:

  • The biochemical focus has been firmly focused on external light sources (e.g., NIR therapy) as the “best” for liberating NO in humans. Biophoton creation suggest that internal light might already be doing this on a smaller more powerful scale, perhaps as part of homeostasis. Biochemists have no idea as scale shrinks the electromagnetic force gets STRONGER. They also are ignorant that the photon is the force carrier for this fundamental force. It is stronger than anything biochemist have in their tool box. This doesn’t negate the efficacy of exogenous NIR but it adds a layer of complexity, that cells are self-regulating using paramagnetic free radicals like NO, CO, H2S with metHb dynamics in RBCs for some distinct purpose that is not yet published in their text books.
    • Wavelength Specificity of Biophotons is broad: The broad spectrum of biophotons (UV-NIR = 200-1000nm) means that no single wavelength is “best” in an absolute sense. Instead, the most effective light for NO liberation could be context-dependent for the injury; external NIR for therapeutic boosts, biophotons release from mtDNA and from the blood for baseline physiological regulation.
    • Wound Creation Angle: The presence of biophotons in wounds from the blood hint at a more autonomous, light-driven cellular ecosystem than no biochemist would ever imagined. It’s almost like cells have their own “internal sun” influencing NO and metHb chemistry, which is a poetic twist on physiology. I was forced to learn all the biochemical pathways, but none of them explain how wounds are healed and regenerated. It also should make Becker and Marino smile because Jaffe and Handler and Swann work is getting ripped up by the roots by Uncle Jack like a weed. Why? Listen.
    • https://www.youtube.com/watch?v=YkBsVMjnwkA
    • The biochemists that Marino cited in the podcast above are why all of science has been blocked from Becker’s ideas. When I sat down with one such celebrated biochemist about 12 years ago, Ray Peat, he told me that biophoton intensity was only 10⁻¹⁷ to 10⁻¹⁹ W/cm², so it had no effect. Moreover, he compared it unfavorably to a 1 mW/cm² (10⁻³ W/cm²) NIR laser. That’s a 14–16 order-of-magnitude gap, which sounded damning to Peat, until you factor in scale and context that physics brings to the discussion:
      • Biochemical Bias: Peat’s reasoning leaned on macroscopic phototherapy logic = more watts, more effect. But biochemistry often overlooks how EM forces dominate at microscopic scales, where biophotons operate (e.g., nanometers to micrometers). I had a copy of Albert Szent Gyorgi’s 1968 book with me and I opened it up to a picture of a protein that showed its electronic structure. I asked Peat if he knew what Szent Gyorgi was trying to convey. He had no idea. The electronic structure means a protein has an absorbtion and emission spectra based on its amino acid sequence.
      • Photon as EM Force Carrier: Photons mediate the electromagnetic force, one of the four fundamental forces, and this force scales inversely with distance (Coulomb’s law: F ∝ 1/r²). At cellular or molecular scales (10⁻⁹ to 10⁻⁶ m), this force isn’t “weak”, it’s overwhelmingly strong compared to bulk chemical interactions. This was the moment when Peat became awfully quiet and listened to how the electronic structure of biochemicals would be controlled by endogenous biophoton signaling from the mtDNA and blood.
      • When scale shrinks light becomes like a laser.  Distance matters to photons. A biophoton emitted within a cell (e.g., from mitochondria) acts over distances of nanometers to micrometers. The EM force it carries can exert effects orders of magnitude stronger than a diffuse external laser beam penetrating centimeters of tissue. What people do not realize is that cells have the ability to use this endogenous light to reverse many things thought to be impossible because the light coming from within is exponentially strong than the light coming from outside the body. He was not expecting this turn of events. Below is an example. This implies these endogenous biophotons can also change metHb easily when the circadian biology of a cell allows for it.
      • 
      • Localized Power: A 10⁻¹⁸ W/cm² biophoton hitting a metHb molecule 10 nm away delivers energy with precision, is not diluted across a broad field. Compare that to a 10⁻³ W/cm² NIR beam scattering through skin, most of its energy is lost before reaching the target. Peat’s face was telling.
      • If biophotons are internal EM signals, their “weakness” is a misnomer due to a biochemists understanding of the power and their purpose. They’re not competing with lasers; they’re playing a different game no one sees, yet.
        • Internal Efficiency: Generated in situ (e.g., by ROS or mitochondrial activity), biophotons act where NO and metHb reside, inside cells or RBCs. No penetration barriers, no energy loss. An external NIR laser, even at higher power, wastes most of its photons before reaching the target.
        • Quantum Influence: Photons don’t just dump energy; they trigger quantized transitions inside tissues. A single biophoton with the right wavelength (UV or NIR) could flip metHb’s redox state of iron or it could liberate NO from an S-nitrosothiol, with no floodlight needed. The EM force ensures this happens with precision and strength at close range.

        Implications for NO and MetHb in Becker’s work.

        Let’s reframe the NO-metHb story with this Electro Mangetic lens:

        • NO Liberation: Biophotons, even at low intensity, could dominate NO release locally because their EM force acts directly on nitrosyl bonds (e.g., in Hb-NO or S-NO). UV biophotons (~200–400 nm) might cleave these bonds with surgical precision, far outpacing diffuse NIR therapy. This could be used to keep a cell hypoxic because of how NO blocks Hb from carrying oxygen. This is a remnant from the Great Oxygenation Event.
        • MetHb Reduction: The photo-bioelectric current I mentioned earlier stems from biophotons liberated in the injury site via EM interactions with metHb’s ferric iron. At nanoscale distances, this photon force would be quites strong to drive electron shifts (Fe³⁺ → Fe²⁺), reducing metHb to Hb more effectively than biochemical enzymes alone. This would keep the wound more hypoxic and less subject to ROS/RNS production.
        • Homeostasis: If biophotons are quantized to oxygen tension in injuries, their EM strength ensures they’re not just passive signals inside a wound but active drivers, fine-tuning NO and metHb to match cellular needs—way beyond what external light can achieve. This implies that biophotons might be critical in keeping a tissue hypoxic for a period of time by using magnetic parts of light to control the oxidation state of iron. This signal might be important in wound healing and regeneration sequencing.

        Centralized vs. Distributed Power

        My biochemical focus was “centralized” when i sat down with Peat, and he thought I was big on external light sources, like the sun, as the hero of the story. He found out quickly that was not the case. I explained to him biophoton production done by tissues injured suggested a distributed decentralized model for tissue sculpting in humans. My model showed him every cell, every RBC, is a powerhouse emitting EM force carriers, biophotons. This aligns with Becker’s amphibian-mammal split too:

        • Amphibians: Few people realizes amphibians have nucleated RBC that emit more light than mammalian blood that is enucleated. Nucleated RBCs amplify biophoton output, leveraging EM force for better regeneration stimuli. Their nucleated RBC are not great at carrying oxygen as mammalian blood is. Remember they are adapted to more hypoxic environments on Earth. This is why their RBCs are nucleated. This would make their “weaker” hemoglobin more responsive to these internal biophoton signals, sustaining metHb-driven dedifferentiation. Becker found this in his work with Salamanders. They were super regenerators. Peat’s face was white at this point.
    • Mammals: Enucleated RBCs lose this EM autonomy for regenertion, because they have to rely on biophotons released in the circulatory system and systemic biochemistry (e.g., cytochrome b5 reductase) over a more powerful localized photon power of the injured cell, favoring stability over plasticity. Mammals scar better than regenerate. Becker confirmed this in his work.Shifted Perspective by Understanding Biophysics

      This evolutionary history lesson of RBCs should flip everyone’s biochemical view: biophotons aren’t “limited” by their wattage, they’re potentially more powerful than external light from the sun is for NO and metHb dynamics in wound healing and regeneration. Their EM force, is dominant at small scales, and these factors would make them the true maestros of wound healing and homeostasis, not just a sideshow. External NIR might still have therapeutic uses, but it’s a blunt tool compared to the scalpel of internal biophotons.

    • NOW TO BECKER

      Let’s break this down:

      • Hypoxia in Wounds: Low oxygen tension (e.g., <20 mmHg in chronic wounds) stresses RBCs. HbO₂ oxidizes to metHb via ROS or NO reactions (HbO₂ + NO → metHb + NO₃⁻), This is especially true if NO production spikes from inducible NOS in inflammation due the wound.
      • Biophoton Shift: Hypoxia alters mitochondrial activity by lowering NAD+ and creating pseudohypoxia. When this occurs, it skews biophoton output. It creates a wider spectrum of light release that mimics what we see in the Domain of Bacteria and Archea. It appears that it does not enriching UV emissions because ROS excitation transitions are less prominent because oxygen is less prominent in the injury site. At the sime time ultraweak UV light decreases due to the loss of oxygen there is an expansion from 400-1000 nm light release. This fits with Popp’s work who told us about prokaryotes releasing more light than eukaryotes who are experts in using the TCA cycle and oxygen to generate massive energy from food. Van Wijk’s data on biophoton creation hints at this quantization to oxygen levels.
      • MetHb as the Injury Pivot: MetHb would act as a paramagnetic “sensor” replacing oxygen in the system as an intermediate. Interestingly, metHb absorption (e.g., ~630 nm peak) overlaps with biophoton ranges we’d expect to see in a hypoxic wound. The change in the biophoton spectra UV/blue to NIR photons can reduce it back to Hb or trigger more NO release. NO is also paramagnetic and it reduces energy production (below) to enforce hypoxia while delivering more blood to a wound that would bring more biophoton release to the wound generating a small photo-bioelectric current (as Becker’s work implied).
      • Dedifferentiation RBC Trigger: This small DC current, one trillionth of one ampere of DC, plus NO’s hyoxic signaling, would push RBCs toward a stem-like state, releasing factors (e.g., growth signals) to aid in wound healing and regeneration. Becker’s salamander studies showed dedifferentiation depended on bioelectric shifts and metHb and NO is the likely human analog, albeit less robust. Why? Amphibians and Humans do not have the same hemoglobin AMO physics.   One has a nucleated RBCs and the other does not.
      • Quantized Biophotons and Regeneration

        The decentralized idea at the cornerstone of my model is that biophoton release is “quantized to oxygen levels in the injury.” It is biophysical majesty not a biochemical reality because it suggests a feedback loop for Becker’s work that biochemistry does not have:

        • Low O₂ → More MetHb → more visible/IR Biophotons: Signals hypoxia, primes dedifferentiation, and kicks off repair. Injury and repair are linked.
        • Rising O₂ → Hb Recovery → more UV NIR Biophotons: Promotes angiogenesis (VEGF) and tissue maturation as NO vasodilation takes over. NIR restores energy production in recovery/regeneration timescale

        My model perfectly marries Becker’s photo-bioelectric currents to Popp’s biophoton coherence and van Wijk’s quantized oxygen links to tissue repair. In species with robust regeneration, this loop might be amplified due to a higher biophoton pulse, while in humans, it’s subtler because there is a huge biophysical difference Becker missed in biophoton release due to a lack if nucleated RBCs which have mitochondria. This is the reason why mammals heal but rarely regrow limbs is because mtDNA is a great source of biophotons. This also explains the phenomental regeneration of human fetus’s because they have nucleated RBCs when they are in the womb surround by amniotic fluid and fully hypoxic.

        Hemoglobin: Phylogenetic Divergence

        Hemoglobin’s structure and function have evolved significantly across vertebrates, reflecting adaptations to oxygen demands, environments, and regenerative capacity: This is why knowing your evolutionary history matters deeply to fully understand the wisdom in my decentralized thesis.

        • Amphibians (e.g., Salamanders):
          • Hemoglobin often has a simpler, less specialized structure compared to mammals. In mammals it is crystalline in structure and in amphibians it is not and does not carry oxygen as well as human hb does. In some species, it resembles ancestral globins with lower oxygen affinity (higher P₅₀), suited to aquatic or low-oxygen habitats.
          • Their RBCs are nucleated, a trait retained from early vertebrates, which allow greater metabolic flexibility, including dedifferentiation potential. This supports atavistic moves in cells during environmental changes. This makes them super adaptable to a changing landscape. This certainly was the case in the post Cambrian Earth. Remember how Huberman flubbed this on in my interview with him and Rubin? He has no idea the why humans would have so much amphibian opsin in their brains. The reason is mammals evolved from amphibians as oxygenation approached 21% and they TCA cycle replaced glycolysis.
          • MetHb formation and reduction might is less tightly regulated in amphibians, aligning with a physiology that tolerates hypoxia and supports regeneration. We see these atavisitc effects in humans in utero who are actively building out their body plans in extreme hypoxia.
          • Mammals (e.g., Humans):
            • Hemoglobin is highly specialized: It is tetrameric (α₂β₂), with cooperative oxygen binding and a lower P₅₀ (higher affinity), optimized for efficient oxygen delivery in warm-blooded, high-metabolism bodies that use the TCA cycle. Why? Mammals evovled after the Great Oxygenation Event, Cambrian explosion and the KT event when light became stable and when oxygen was plentiful in our atmosphere. Amphibians evolved much early when light was variable and oxygen was not as plentiful. Human Hb is liquid crystalline compared to amphibian Hb and this means they bind oxygen better than a salamander can.
            • Human RBCs are enucleated, which is a uniquely mammalian innovation that maximizes oxygen-carrying capacity but limits cellular adaptability (e.g., no dedifferentiation without extreme cues).
            • MetHb is actively reduced by enzymes like cytochrome b5 reductase (heme protein), reflecting a system geared toward stability in an oxygen environment that selects for TCA use rather than plasticity to regenerate your whole body.
          • Wound Healing and Regeneration: The Hemoglobin Link

            My decentralized  hypothesis finishes Becker’s life long work that oxygen tension, biophotons, and metHb dynamics drive healing and regeneration and this maps onto these differences elegantly:

            • Amphibians: Regeneration Superstars:
              • Hemoglobin Context: Lower oxygen affinity means amphibian Hb releases oxygen more readily in hypoxic wounds, amplifying local hypoxia. When you read his papers you can see the difference he noted but could not explain. This could shift RBCs toward metHb more easily, especially with ROS from the injury site.
              • Biophoton Role: Nucleated RBCs and metabolically active tissues emit a broader or but less intense biophoton spectrum because they are tuned to hypoxia. Why? mtDNA is the major SOURCE of biophotons in Nature. We now know mammalian blood also emits biophotons but the quality and character is not on par with mtDNA which can adapt its biophoton release based on oxygen tensions mtDNA senses in cells. Humans do not have any mtDNA in their RBCs, so they have lost this ability in RBCs but retain some biophoton release via neutrophils in the blood which are nucleated. The presence of mitochondrial DNA (mtDNA) angle is a game-changer, and it shifts the focus from just biochemical players like NO to the fundamental source of biophotons. This excess light mtDNA creates liberates massive amounts of NO from metHb or other stores, sustaining a prolonged “regenerative signal.”
              • Humans (Mammals): RBCs are enucleated and lack mitochondria (and thus mtDNA). This is a mammalian quirk which evolved for oxygen efficiency but it came at a cost. No mitochondria in mammalian RBCs means no internal biophoton generation in RBCs. Any biophotons in human blood come from other cells (e.g., leukocytes, endothelial cells), not RBCs themselves. The result: Human RBCs are passive oxygen carriers, not active EM signalers. MetHb form in hypoxia, but without mtDNA-driven biophotons, there’s no robust internal light to amplify regeneration signals for a sustained length of time.
              • Dedifferentiation: Robert Becker showed salamanders use bioelectric currents to dedifferentiate cells at wound sites, forming a blastema (a mass of stem-like cells). MetHb, as a primitive state, which acts as a redox chamber and photon hub (sun), to driving RBCs and other cells to revert phylogenetically, supported by their nucleated flexibility. This explains why the sun is critical in mammalian longevity. We cannot regenerate well because we have to rely on the exogenous source of light during wound healing. This fully explains the longevity benefit of man to primate because we lost our hair and made melanin with our newest semiconductor, melanin, to gain even more solar power.
              • Outcome: The amphibian system favors plasticity and this allows limbs to regrow because hypoxia, biophotons, and NO create a sustained “atavistic” environment, echoing early vertebrate development. Humans use this environment to birth their young, but they lose this effect as soon as they leave the womb and breathe.
            • Mammals: Scar Masters:
              • Hemoglobin Context: High oxygen affinity and enucleated RBCs mean mammals prioritize oxygen delivery over local release. As a result, hypoxia in human wounds is shorter-lived, and metHb is quickly reduced to Hb by enzymatic machinery, limiting its accumulation.
              • Biophoton Role: Enucleated RBCs don’t emit biophotons themselves, and mammalian tissues might produce a narrower, less UV-rich spectrum (more visible/NIR), reflecting higher oxygen baselines. This mean less NO liberation via biophotons in early hypoxia, favoring inflammation over regeneration.
              • Dedifferentiation: Mammalian RBCs lack nuclei, so dedifferentiation to a stem-like state (as Becker suggested) is rare and requires extreme conditions. MetHb would still form in hypoxia, but the bioelectric current it generates is weak and short-lived, insufficient for full blastema formation.
              • Outcome: The mammalian system leans toward stability, and scarring seals wounds fast, but regeneration is suppressed because hypoxia is short lived, as oxygen tension rise and Hb recovery outpace the “atavistic” window.

            Phylogenetic Hemoglobin and Healing Divergence

            The phylogenetic gap in hemoglobin ties directly to my quantized biophoton idea:

            • Oxygen Tension: Amphibian Hb’s lower affinity amplifies wound hypoxia, extending the metHb-biophoton-NO loop. Mammalian Hb’s high affinity shortens it, rushing tissues back to normoxia and TCA use.
            • Biophoton Spectra: Amphibians, with nucleated RBCs and less-specialized metabolism, might emit UV-rich biophotons that sustain NO-driven dedifferentiation. Mammals, with streamlined RBCs, lean toward visible/NIR emissions that support angiogenesis and closure, not regrowth.
            • Atavism: MetHb in amphibians mimics an ancestral state (e.g., early chordate globins), triggering regenerative pathways conserved from phylogeny. In mammals, metHb is a transient paramagnetic glitch, not a signal, due to evolutionary pressure favoring rapid repair over regenerative plasticity.

            Why the Difference? Hemoglobin as the semiconductor

            Evolutionarily, amphibians retained regenerative capacity because their environments (e.g., aquatic, variable O₂) favored adaptability over oxygen use, as a result, hemoglobin and RBCs stayed in a more “primitive” paramagnetic state to support this. Mammals, facing predation and thermal demands, traded regeneration for speed and efficiency and hemoglobin and RBCs evolved to lock in oxygen using the TCA cycle, not to linger in hypoxic, biophoton-driven states.

            Reptiles and Salamanders (Amphibians):

            • RBCs are nucleated and retain mitochondria with mtDNA, especially in amphibians like salamanders. Reptiles (e.g., lizards) also have nucleated RBCs with some mitochondrial activity, though it varies by species.
            • Result: Their nucleated RBCs are biophoton factories. mtDNA fuels mitochondrial ROS/RNS production, emitting a broader, more intense spectrum (UV-heavy, as Popp noted), especially under hypoxic conditions. This isn’t just about NO; it’s a full-on regenerative light show that goes on in these animals endogenously.
            • 

SUMMARY

I am hoping you have put these lessons all togther now and understand why amphibians are regeneration rockstars using primitive heme proteins. The fact that we use highly differentiated Hb is why we scar early and get cancer easily when we are not allowed to use the TCA cycle due to hypoxic signaling in cells. Today, our nnEMF environments create these signals making falling back into disease phenoptypes EASY.

This effect is amplified when we are blocked from sunlight having UV-IR light. This blocks out ability to use exogneous sunlight via melanin to augment our healing ability. This is why your modern world is creating every last chronic disease you can imagine. Awake now? We are our own Asteroid folks. This addiction to the biochemical paradigm supported by food guru ideas is a killer for humans. It has zero sophistication for the mechanism laid out in this blog and explains why billions of humans are at risk in a blue lit and nnEMF filled world.

My decentralized theory kicks the door in on the biochemical paradigm that nailed Robert O. Becker’s scientific life to a cross. Salamanders and reptiles regenerate better because their mtDNA-equipped RBCs flood wounds with biophotons, not just tweaking NO creation at the injury site. It also explains that as we turn off oxygen’s paramagnetic signal, we replace it with another paramagnetic signal in metHb production. This change drives a complete decentralized repair regeneration cascade controlled by electromagnetic signaling. This favored a broader spectrum of biophotons creation. Popp showed prokaryotes emit 5000 times more light than we eukaryotes. That fact is huge when you plug in evolutionary history I gave you here.

Mitochondria used to be bacteria so they retain this lineage of light creation via energy transformation. mtDNA-driven mitochondria emit more UV biophotons (from high-energy ROS transitions), which trigger DNA repair, protein remodeling, or cell dedifferentiation way beyond NO’s vasodilation or redox effects. Hypoxia creates the sun inside a wound of an amphibian. This hypoxia is an electromagnetic amplifier for their regeneration.

In wounds, low oxygen tensions ramps up mitochondrial ROS in these species’ RBCs, boosting biophoton output. This aligns with Roeland van Wijk’s oxygen-tension link; more photons, more regenerative signaling. It also aligns with Popps work too. Every box is checked.

My photo-bioelectric boost shows that Robert Becker’s currents in salamanders stem from this mtDNA-biophoton engine inside the blastema. Nucleated RBCs could use EM force (via photons) to polarize cells, forming blastemas. Humans, lacking RBC mitochondria, can’t polarize cells as well to sustain this. This is why depolarization in humans links to CANCEROUS human cells.

How do humans offset the inability of making biophotons to repair? Enter, melanin and sunlight exposure on their skin. Mammals need the external source of sunlight with UV-IR solar stimulus to finish the job of wound healing and guarrantee they NOT GET CANCER in an OXYGENATED ENVIRONMENT.

This is why CCO controls water production and apoptosis in mtDNA. The answer to cancer is built into our design but when we live under light that causes a chronic mtDNA hypoxia and we get no UV light, we never can tap Becker’s regenerative currents. Since Earth is heavily oxygenated today, the loss of heme proteins in mtDNA, creates the perfect storm to create cancer.

Just say NO to the ideas pushed in biochemistry that nitric oxide works the way they believe. They are beyond dead wrong. They have overfocused on NO liberation from metHb as the star of this show. They get No Quarter from me, no mea culpa. Billions have died because of their myopia. While NO matters (e.g., vasodilation, signaling), biophotons from mtDNA do way more. NO is used to keep wounds hypoxic in injury. Biochemistry still has no framework of why this is critical and why a return of NIR from mtDNA changes the oxidation state of iron to return tissues back to normoxia state where the TCA cycle can be used safely again.

• Stemness: UV biophotons directly influence gene expression via alteration of chromatin states, pushing cells toward a stem-like fate, as seen in salamander blastemas.
• Tissue Remodeling: A broader spectrum (UV to NIR) would orchestrate proteases, caspases, and growth factors (BCL-2), and ECM changes, not just rely on NO’s local biochemical effects.
• Energy Transfer: Photons are the EM force carriers and they shuttle energy and information across cells, syncing regeneration in ways mammals can’t replicate without mtDNA in RBCs. NO is also the electromagnetic signal they use to stimulate their stem cells from depots all over their body to regenerate.

Mammal vs. Amphibian/Reptile Gap Is Tied 100% to LIGHT, NOT FOOD.

• Humans: No mtDNA in RBCs = limited biophoton budget. Wound healing leans on systemic factors (e.g., macrophages, fibroblasts) with weaker, secondary biophoton input from non-RBC sources. Scarring wins over regrowth. Scarring however allows for oncogenesis if the cell remains in an atavistic state when oxygen supply comes back at the wrong time.
• Salamanders/Reptiles: mtDNA in RBCs = biophoton surplus. Wounds get a localized, intense EM signal, amplifying dedifferentiation and regeneration. Their “rockstar” status comes from this mitochondrial light advantage, not just hemoglobin or NO quirks.
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I think I’ve reframed Becker’s experiments perfectly using biophysics. Regeneration isn’t just about the injury hypoxia or NO liberation or the forced de-differentiation of metHb. It’s about mtDNA as the biophoton engine of creation. Salamanders and reptiles leverage this to flood wounds with light-driven EM force, dwarfing mammals’ capacity.

I hope you can visualize something larger now. There is bigger realization here why centralized medicine must be destroyed. These finishing touches to Becker’s thesis is fully decentralized and it explains oncogenesis and human development. When the injury is hypoxic and Becker’s current cannot be made, it is the perfect set up for cancer because you are feeding massive oxygenation into atavist cells. That is the cancer story.

Here is the other realization. Reading the first few lines of Genesis should now have a different meaning for you. This explains how a human child grows inside the womb. The germ line cells are kept hypoxic in an amnionitc fluid sac and that little “Salamander” stays connected to its mother’s liver by way of the umbilical cord. Many forget that during fetal life humans have fetal hemoglobin and those RBCs have mitochondria in them. Those RBC come from her liver.

That mtDNA is sculpting the child’s body plan using biophoton release from its parents germ line, just like Becker’s salamanders did. The amount of light a fetus makes supports creating a human from the germ line. This story is astounding when you realize it. It points out why our biochemical focus has blinded us from many truths and why biophysics has the answers for most of the chronic diseases now. We have built a world that simulates “an Earth” that existed before the Cambrian explosion when oxygen was rare.

What we have done to Earth with light and nnEMF is truly tragic. It is humanity’s asteroid. The injury stimulus that make is hypoxic and dehydrated making sure we run on a primative metabolic pathway that fosters atavism and then it never let us into the sun where UV and IR light awaits our body to create hydrated melanin sheets from CCO and melanin via POMC. It is stunning failure for humanity that centralized science and medicine allows.

CITES

The Body Electric, Robert O. Becker 1985

https://www.researchgate.net/publication/8465511_Biophoton_research_in_blood_reveals_its_holistic_properties

Pall, M.L. “Electromagnetic fields act via activation of voltage-gated calcium channels to produce beneficial or adverse effects.” J Cell Mol Med. 2013.

Yakymenko et al. “Oxidative mechanisms of biological activity of low-intensity radiofrequency radiation.” Electromagn Biol Med. 2016.

Leszczynski et al. “Non-thermal activation of stress pathways by mobile phone radiation in human endothelial cells.” Differentiation. 2002.

CENTRALIZED VS. DECENTRALIZED MEDICINE

I visited a friend who was admitted to the ICU
The whole hospital is blue fuckin lit.
The wards were sealed tight, not a breath of fresh air.
The sun fights its way in, but thick glass blocks the full solar light spectrum.
WiFi routers & medical equipment blasting EMFs 24/7.
Every surface is sterilized, yet the air feels dead.
There are no negative ions or grounding; it is just recycled air that is heavy with chemicals.
Machines beep like a never-ending alarm.
Walls, lifeless and grey.
The only greenery?
Plastic plants collect dust in the corner.
Oxygen is pumped through machines, but there is not a single breath of fresh air.
The doors are locked as if nature is the enemy.
How the hell is this a healing place?
You go broke using the place.
It is a fiat hell hole to which everyone is sent.

ALL CHRONIC DISEASES ARE LINKED TO THE WARBURG SHIFT

1. nnEMF-Induced pH Shift Drives Warburg Redox Shift: Light drives it, not food. The pictures above and below both show it in spades, but no one understands what these two pictures placed in blogs imply. The Warburg effect is characterized by increased blood glucose, glucose uptake, and lactate output, even under aerobic conditions. This reflects mitochondrial dysfunction (e.g., damaged IMM, cytochrome c oxidase inhibition). The damage of the IMM between NAD+ and oxygen allows for a 30 million charged field to escape into the cell. That charge then follows electric resistance pathways to cause many disease phenotypes. This is the stress response Selye talked about decades ago. He never was able to pin down how it began. All he knew was that it started on the HPA axis.

This podcast gave you the basics to the details in this blog: https://www.youtube.com/watch?v=IHSMqhrfvaU

The published science is precise that light stress by ALAN or a lack of sunlight drives metabolic shifts and how red light might serve as a therapeutic “drug” equivalent to counter the Warburg shift to change metabolism in ALL chronic diseases. In all stressors, vasopressin is the first stimulus to injury, and then the Orexin prism you learned about in QE #20 is next up in the cascade. Below, you see the definitive proof in two more pictures that red light reverses the effect of blue light exposure. This is what defines the Warburg Redox shift.

My photo-bioelectric hypothesis posits that nnEMF (non-native electromagnetic fields) and blue light damage melanopsin, mtDNA, and heme proteins, reducing DDW (deuterium-depleted water) production which leads to dehydrating melanin and increasing electrical resistance (éR) inside cells, leading to massive ROS/RNS rise, reductive stress (high NADH/NAD⁺), and Warburg metabolism (increased lactate). All of these cascades make oxygen a toxin because, due to its electronegativity, it pulls electrons across the IMM that is demolished. Why? With a stressor or trauma, excessive éR presents and leads to dissipative loss and bioenergetic inefficiency, reductive and oxidative stress, inflammation, molecular damage, and information loss. Thus, the hallmarks of disease and aging naturally arise from éR problems in the IMM on mtDNA. What you see and understand on the surface from biochemistry is not the entire biophysical story below built on the IMM.

Heme proteins are much older than mammals. This is a consequence of the evolution of our atmosphere. At the end of the Great Oxidation Event, heme regulation began to merge with POMC biology. Heme is a cofactor for many proteins, and it influences POMC expression via circadian and metabolic pathways. For example, heme binds to Rev-Erb-alpha and Rev-Erb-beta (nuclear circadian receptors), which regulate POMC transcription.

This links heme (and oxygen metabolism) to the light-driven circadian rhythms of mammals.

Heme proteins are much older than mammals. This is a consequence of the evolution of our atmosphere. At the end of the Great Oxidation Event, heme regulation began to merge with POMC biology. Heme is a cofactor for many proteins, and it influences POMC expression via circadian and metabolic pathways. For example, heme binds to Rev-Erb-alpha and Rev-Erb-beta (nuclear circadian receptors), which regulate POMC transcription.

This links heme (and oxygen metabolism) to the light-driven circadian rhythms of mammals.

THE DECENTRALIZED CASCADE TO THE ETIOLOGIES OF CHRONIC DISEASE

• Organisms as Open Thermodynamic Systems:
  • Organisms are open thermodynamic systems dependent on energy flow (e.g., sunlight, food) and material exchange (e.g., waste export). Spent energy contributes to entropy, defining the flow of time. Molecular clocks (e.g., PER1/PER2) act as flowmeters of entropy, measuring cellular energy dynamics.
• Cells as Dissipative Structures:
  • Cells escape the second law of thermodynamics (entropy increase) by creating order from chaos, functioning as dissipative structures. This order is built around the AMO (atomic, molecular, and optical) physics of atoms in cells, with molecular arrangement critical for function.
• Water’s Role in Cellular Organization:
  • Water is critical in the molecular arrangement of biomolecules, influencing their physiological function. The movement of water (e.g., via hydration shells and gradients) changes biomolecular dynamics, mimicking semiconductor physics in silicon circuits (where electric power generates light). Cells use water to create and manage light (e.g., biophotons), maintaining atomic molecular order. Pure water contains the field of charge stored on the IMM between NAD+ and oxygen.
  • Temperature affects semiconductors’ band gap size, too. You got this lesson in QE #29. Now you will find out how to link to your own disease.
  • Cooling increases band gap size. Water changes the temperature of the semiconductive proteins inside of use, which generates electrical resistance.
  • Cooling gave us evolutionary pressures to create catecholamine chemicals in prokaryotes like dopamine and adrenaline using a new semiconductive protein inside cells called melanin. When it degrades, it can become L-DOPA, which can become dopamine and adrenaline under a hypoxic stimulus.
  • A lack of oxygen affected the early oxygen carrier semiconductive molecules called heme proteins.  Recall that Robert Becker’s work around bone regeneration concerned itself with transforming RBCs to a pluripotential cell via a pico to nano ampere currents. The initial oxidation of hemoglobin to the ferric (Fe3+) state without oxygen converts hemoglobin into a useless “hemiglobin” or methemoglobin, which cannot bind oxygen.
  • KEY POINT NO ONE REALIZES: This step exists in hemoglobin biology to deactivate hemoglobin’s ability to carry on its adult physiology to travel back in time atavistically to create Becker’s regenerative current. Becker’s de-differentiation experiment never looked at the blood cells that were being transformed. If you understood the evolutionary history of hemoglobin creation, this was a methodological error. It occurred during the GOE.
  • Methemoglobin is an ancient protein used by life long ago when oxygen WAS NOT prominent in our atmosphere.  This was when all life on Earth had an “allergy” to oxygen because oxygen was toxic to bacteria and archaea then. Methemoglobin naturally builds up in human injuries to precondition the cell to return in time. If the cell is filled with oxygenated hemoglobin, it cannot generate Beckers’ regenerative current. One thing that can be used to help this process is methylene blue. But MB has to be used at the right time. The first week is the incorrect time to use it. Methylene blue has two major effects that are beneficial for humans who cannot generate Becker’s healing currents.
  • NUMBER 1: It alters the eR on the IMM in a damaged state, and
  • NUMBER 2: it can increase NO delivery to tissues that become hypoxic with a lowered NAD+. This increase in NO allows mammals to access their stem cell depots to repair the damage in question. The stressor and damage are always linked to tissue-level hypoxia or pseudohypoxia, where NAD+ has dropped.
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  • Early versions of hemoglobins suffered from this problem = myoglobin.  Later, mammals figured out a novel way to stabilize hemoglobin using green light when oxygen filled the atmosphere.  First, I have to explain how we got there.  Modern versions of hemoglobin in normal red blood cells are protected by a reduction system in the RBC by an aromatic amino acid (histidine) and a sea of electrons from the water in the blood. The surface of Earth was getting pounded by UVC light for long periods.   Since life was prokaryotic and anaerobic 2.7 billion years ago when cyanobacteria evolved, it is believed that oxygen acted as a poison and wiped out much of anaerobic life, creating an extinction event of the old guard in life, including LUCA.  LUCA = last unknown common ancestor.
  • This environmental change drove evolutionary pressures to innovate proteins that used aromatic amino acids to build the most critical parts of the modern metabolism we see today in cells.  Their absorption spectra can go from 150nm VUV to 400 nm UV-A light.  Melatonin is one of the most ancient semiconductors known.  Its functions have evolved as the atmosphere of Earth changed its atomic concentrations. This is why leptin has 220 nm absorption spectra when you look at it
    
  • Melatonin, NAD+/NADH, dopamine, adrenaline, leptin, epinephrine, etc. Light controls the flux of all these biomolecules because of the movements of H+ in cells.  Remember, all enzymes that create these chemicals use proton tunneling to get the job done.  Proton tunneling is linked to the HIF-1 alpha and PER2 gene that uses light to increase the periodicity of cell molecular clocks. Increasing periodicity = better clock management.
  • Tryptophan, another aromatic amino acid, became very useful to cells living in an oxygenated world as a “time crystal” for cells because it has only one DNA codon, and its catabolism changes as light/dark signals change with the tilt of the Earth that gives us seasons. NAD+ and melatonin are both made from tryptophan. NAD+ and 95% of our melatonin is created in mtDNA. You might want to go back and re-read QT-14 now.
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• Future life forms would need this information because oxygen gave us cool and hot seasons in one year as the Earth revolved.  This cyclic pattern was built into cell metabolism as it got more complex, as the slides above show.
• Since oxygen has a high redox potential, it acts as an ideal terminal electron acceptor to generate energy after a nutrient breakdown. Oxygen soon became indispensable for metabolic activities. Organisms also evolved strategies to detoxify the reactive oxidative species that resulted from aerobic metabolism. Told ya’ that Vermont talk was important.

Though sequencing and phylogenetic analyses estimate the evolution of ROS-detoxifying enzymes even before the advent of aerobic microbes, the Great Oxidation Event acted as the catalyst to shape the directed evolution of enzymes like superoxide dismutase (above) and catalase.  Catalase (above) is one of the earliest heme proteins, ancient myoglobin, and hemoglobins.

• • Note the wide-band semiconductor components in the picture above: the Fe-S dopant semiconductors and their roles in creating the free radical signal in mitochondria.  Below, note how mammals’ chromophore proteins are linked to aromatic amino acids, heme proteins, and melanin at some level. Can you guess why yet?
  • As oxygen continued to mushroom, the high ionosphere became filled with a new gas that decreased the terrestrial solar spectrum.  Life had to react to this, and it fueled changes in heme proteins that appeared on Earth’s surface in early life forms.

    Oxygen was also responsible for forming the ozone layer in the atmosphere. The UV radiation from the sun split oxygen molecules (O2) into two atoms of oxygen, which then reacted with another oxygen molecule to generate ozone (O3). Ozone acts as a natural sunscreen for Earth to prevent harmful UVC and parts of the UV-B radiation from reaching the Earth’s surface.  This reduction began the evolution of new semiconductors in the two DOMAINS of life on the surface of the earth called melanin.

    As oxygen continued going higher, it fueled the Cambrian explosion, and life was able to take advantage of the mirror image of the photosynthetic arm of life on Earth.  Namely, mitochondria developed.  Complex life captured mitochondria in their tissues as a stowaway to transform solar energy into CO2 and water.  It transferred electrons from food to oxygen to fuel this solar battery.  At this point, life exploded, and all the complex life on Earth we know about today showed up almost overnight.  Eukaryotes came from the fusion of the other two Domains in endosymbiosis.  We believe chlorophyll and mitochondria were also innovated at this time.

     

    Stress, Vasopressin, and Water Conservation:

  • Stress and trauma (e.g., nnEMF, ALAN) disrupt water dynamics, triggering vasopressin release to conserve water. This mimics the VP-ISR-GDF15 axis activation in modern mammals. This is done because injuries all induce dehydration, and hypoxia of our semiconductors facilitates increasing éR, entropy, and cellular chaos, driving most chronic disease phenotypes. This mimics life during the GOE.
  • When the human body is sick, diseased, and energy inefficient from any stressor or stimulus, its evolutionary directive is to eat itself removing all the diseased cells and organelles and cancerous cells. To do this, it needs to generate a regenerative DC to do so. This current relies on the ability of heme proteins to make water to hydrate melanin to create this small current. When the cell cannot do this for any reason, big problems await this organ. The disease begins to spread like an infection does through the organ. We call this heteroplasmy.
• HIF-1 and Cellular Hypoxia:
  • Poor sunlight, darkness, ALAN/nnEMF, and geoengineering (e.g., aerosol-induced dimming) lower solar EMF (UV-A/B), which induces cellular hypoxia and blocks Becker’s regenerative current from the beginning. Without reestablishing cytochrome c oxidase to make DDW hydrate melanin, you never get to produce the one trillionth of one ampere of current needed to turn stem cells into de-differentiated pluripotential cells that can heal damage. Moreover, UV-A light stimulates NO, which controls mammals’ stem cell depots. So this is also turned off. Hypoxia-inducible factor-1 (HIF-1), stabilized under low oxygen or light stress, shifts metabolism to glycolysis (Warburg shift), increasing lactate production, and lactate use drives éR, which drives tissues into the Warburg redox shift. The pictures at the beginning of this blog are all there for you to review.
• HIF-1 and PER2 Link:
  • HIF-1 belongs to the same protein family as Period 2 (PER2), a core circadian gene. Liu et al. (2012) show PER2-HIF-1α-dependent regulation of SIRT3 (sirtuin 3) under hypoxic conditions, modulating the choices between TCA cycle flux and glycolysis. Sunlight enhances PER2 expression, optimizing oxygen use, while ALAN/ darkness/geoengineering disrupts this, favoring HIF-1α and Warburg metabolism. If you read the links on my forum in the Mitochondrial Thermodynamics DIY, you’d already know all of this—->https://x.com/DrJackKruse/status/1613298172801044482

    Red Light as a Therapeutic Equivalent: Tiina Kuru

  • 43% of the sun is Red light (e.g., 600-1000 nm), and PBM/LLLT can mimic sunlight’s effect, upregulating PER2 and downregulating HIF-1α, reducing éR and Warburg metabolism. The red light was critical in the evolution of all heme-based proteins after the Cambrian Explosion. Tiina Karu’s work supports red light as a “drug” equivalent, enhancing mitochondrial cytochrome c oxidase and oxygen utilization, countering the cellular effects of dehydration and hypoxia in an oxygen-filled atmosphere.
  • PER2-mediated ischemic preconditioning of the heart links sunlight/red light to optimal cardiac oxygen delivery, disrupted by mtDNA damage at cytochrome c oxidase (favoring Warburg shift).

• Historical and Evolutionary Context:
  • The Great Oxygenation Event (driven by sunlight/photosynthesis) coupled oxygen, sunlight, and PER2, enabling mammalian survival. Dinosaur extinction (darkness, cold) reflects HIF-1 explosion in non-therapod dinosaurs, while mammals adapted via PER2. Modern sunlight avoidance (geoengineering, indoor living, atrophic skin, ALAN) increases cardiac/PAD death and hypoxia-related diseases. With sunlight from the KT event, trillions of photosynthetic algae could now grow uncontrollably to make oxygen, transforming the entire planet’s atmosphere and setting up the perfect storm for the evolution of a mammalian mitochondrial world post-KT until human technology changed the signal when we innovated ALAN.

    Disease Implications:  

  • Blue Light/nnEMF and Vitamin A Liberation from opsins:
    • Blue light and nnEMF liberate Vitamin A from cells and membranes, raising its presence in blood plasma as an aldehyde (retinaldehyde). This lowers plasma Vitamin C and D levels, disrupting local antioxidant defenses (melatonin/ISR) and circadian signaling. Retinaldehyde destroys small molecule modulators of the mammalian circadian mechanism, including PER1 and PER2, critical gears in the eye clock of the SCN. Rev Erb Beta and alpha are the destroyed nuclear circadian receptors that are also heme-based.
  • Impact on Circadian Rhythmicity and mtDNA:
    • Disrupted PER1/PER2 periodicity (from retinaldehyde toxicity) leads to quantum timing loss in mtDNA, accelerating mutations. Human mtDNA mutates 15–20 times faster than nuclear DNA (vs. 5–10 times in primates), and nnEMF accelerates this further, driving faster epigenetics and chronic disease creation. This is why MAHA is HAHA. They remain myopically focused on food when light is the driver of all chronic diseases.
  • Cellular Disorganization and Energy Cost:
    • mtDNA mutations of the heme-based proteins cause cellular disorganization by impairing information processing (energy = information in physics). Energy is trapped in cells’ electronic level, stored in vibrational/electronic bonds (e.g., PER/HIF-1), membranes, gradients, fields, and mtDNA-structured water. nnEMF increases ubiquitin marking, raising protein turnover, robbing energy, lowering redox state, and increasing éR, spreading heteroplasmy across organs and manifesting many new disease phenotypes.
  • mtDNA Vulnerability:
    • mtDNA is more vulnerable than nuclear DNA due to: (1) higher oxidative stress (mitochondria as ROS source), and (2) the matrix-side negative membrane potential concentrating lipophilic cations 1,000-fold, amplifying nnEMF effects. This charge density change mimics vasopressin’s effect on water (negative charges QE has a blog on it too!), linking light to metabolism and controlling all the biochemicals to control it (Light > Food).

    Warburg Shift and Heteroplasmy:

    • nnEMF increases epigenetic expression, raising mtDNA error rates and heteroplasmy (normally 10% per decade, accelerated by nnEMF). This energy-intensive process lowers the redox state, increases éR, and drives Warburg metabolism (increased lactate, glycolysis), fueling disease (e.g., ALS, cancer, diabetes, obesity).
    • Warburg-like metabolism (increased glucose uptake, lactate) is a hallmark of cancer (FDG-PET), atherosclerosis, sarcoidosis (granulomas), and neurological conditions (e.g., schizophrenia, elevated lactate). The Ras-ERK-PI3K-mTOR axis, via mTORC1, stabilizes HIF-1α, promoting glycolysis (HK-II, GLUT1) independent of oxygen and driving disease progression.
    • Compared to complete glucose oxidation, Warburg metabolism generates less ATP stochastically but creates a higher éR, which enables more rapid glycolysis (10–100 times faster) during the light stress event. This fuels macromolecular synthesis (e.g., via the pentose phosphate pathway [PPP] and fatty acid synthesis), supporting cell growth and division.
    • The rapid shift to Warburg metabolism is triggered by changes in the organism’s light environment (e.g., nnEMF/blue light vs. solar EMF), a phenomenon misunderstood in biochemistry due to the neglect of biophysics and photo-bioelectric currents. Red light drops blood glucose and insulin, while blue light and nnEMF raise blood sugar and insulin levels. The pictures above state that case. Any nnEMF disrupts mitochondrial electron flow, increasing éR and lactate, while solar EMF (UV-A with IR) restores NAD⁺/DDW, lowering éR and supporting oxidative phosphorylation of the TCA cycle. This is why you need to see AM sunlight to use the TCA cycle. Every time the day’s first light is not the sun, you just Warburg shifted your LIFE toward disease.

Elevated lactate and éR drive reductive stress, and this recycles NAD+ to NADH rapidly (high NADH/NAD⁺), signaling the release of vasopressin, which initiates the Integrated Stress Response (ISR) and this activates GDF15, which suppresses anabolism but supports rapid energy needs (e.g., cancer proliferation, retinal repair). This dual role explains pathological (e.g., AMD, cataracts, retinal damage, RP progression) and adaptive (e.g., retinal resilience) outcomes. ALAN turns on the VP-ISR-GDF15 axis while red light radiation seems to turn it off.

Prediction: High nnEMF exposure in Big City USA like NYC, intensified by 5G and ionospheric radiation, alters cellular pH (e.g., <7.35), driving a Warburg redox shift and contributing to the crash at an early age for humanity

• Mechanism: nnEMF activates VGCCs, increasing Ca²⁺ influx and ROS/RNS (peroxynitrite, ·HO), disrupting the NAD⁺/NADH ratio and lowering cytochrome c oxidase activity (pseudohypoxia = low NAD+). This reduces DDW production, dehydrates melanin, and increases NaCl concentration, dropping intracellular pH below 7.35 (-20 mV) and membrane potential toward zero mV. The night owl lifestyle and blue light exposure amplify free retinal toxicity, damaging photoreceptors (melanopsin, NO, Rev erb /alpha-beta, cytochromes), while endurance stress (exercise) and/or mold burden (low alpha-MSH) enhance glutamine addiction via GDH dysfunction. This “toxic barrel” shifts metabolism to glycolysis, causing mitochondrial failure (energy crash) and neuronal dysfunction (cognition crash).
• Vasopressin’s Role in ISR Activation: Vasopressin (VP), a hormone released by the hypothalamus in response to osmotic stress, dehydration, or nnEMF-induced cellular stress, activates the ISR by stimulating eIF2α phosphorylation via eIF2α kinases (e.g., GCN2, PERK). This global translational arrest induces stress-responsive genes (e.g., ATF4, CHOP) and upregulates GDF15 (growth differentiation factor 15), signaling energy resistance (éR) systemically. nnEMF and artificial light at night (ALAN) increase VP secretion by disrupting melanopsin signaling and inducing osmotic stress (e.g., via dehydrated melanin). VP activates ISR, enhancing GDF15 production, suppressing anabolism (e.g., protein synthesis in the damaged area blocking regeneration), and promoting catabolism, exacerbating chronic conditions under reductive stress (high NADH/NAD⁺, lactate).
• Outcome: Low pH (e.g., intracellular pH <7.3), high lactate (blood >2 mmol/L), and low NAD⁺/NADH ratio (<1), measurable via blood gas analysis and metabolomics.
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2. Dehydration from nnEMF lowers éR and pH —–> https://osf.io/preprints/osf/hgnmj_v2

• Prediction: nnEMF-induced dehydration increases conductivity (high NaCl, low water), reducing éR and pH and preventing recovery post-crash.
• Mechanism: nnEMF dehydrates cells, raising NaCl concentration and lowering pH (e.g., <7.35), reducing the membrane potential (-90 mV) toward zero mV. Dehydrated melanin becomes conductive, amplifying ultraweak biophotons and ROS/RNS, disrupting Becker’s regenerative current.  Free retinal toxicity from nnEMF/blue light damages catalase and cytochromes, impairing H₂O₂ breakdown and DDW production and exacerbating the redox Warburg shift (light).
• The patient’s lack of solar EMF (night owl, indoor living NYC life) prevents pH restoration, perpetuating mitochondrial stress. People forget that before eukaryotes, only two domains of light used the Warburg metabolism because of the light present. It had nothing to do with food. This defined the Cambrian explosion and KT events, as I laid out on the Huberman/Rubin Tetragrammaton podcast. String the lessons together and integrate them. No one is.
• Outcome: High serum osmolality (>300 mOsm/kg), low éR (microcurrent <1 µA), and persistent fatigue/cognition deficits, measurable via pH electrodes and imaging.

3. Warburg Shift Reflects Light Environment Choice, not a diet

• Prediction: The patient’s exposure to nnEMF/blue light (vs. solar EMF) altered the mitochondrial free radical blueprint, driving the Warburg shift and cognitive crash of people in a nnEMF environment.
• Mechanism: nnEMF (5G, blue light) and night owl behavior increase AMPK and ROS/RNS via VGCCs, lowering NAD⁺ and pH, shifting metabolism to glycolysis (Warburg light effect). Solar EMF (UV-A) supports quantized free radicals and redox potential, but its absence (due to indoor nnEMF dominance) disrupts cytochrome c, enhancing glutamine addiction. Free retinal toxicity damages photoreceptors, amplifying this shift, while mold exposures (low alpha-MSH) and endurance stress compound mtDNA heteroplasmy.
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• Outcome: High AMPK activity, low cytochrome c activity (enzyme assays), and elevated glutamine/glutamate ratio, reversible with solar EMF.

  4. AM Solar EMF Restores pH and Redox Potential

  • Prediction: AM solar exposure (30–120 minutes daily) restores cellular pH (7.35–7.45), reverses the Warburg shift, and improves energy/cognition.
  • Mechanism: AM sunlight (UV-A, visible light) reduces free retinal, regenerates melanopsin/cytochromes, and boosts cytochrome c oxidase for DDW production, raising NAD⁺/NADH and pH to -20 to -25 mV. This hydrates melanin increases éR, and counters nnEMF-induced dehydration, restoring membrane potential (-90 mV) and bioelectric signaling. Solar EMF’s quantized free radicals support redox potential, mitigating the Warburg shift and mold effects.
  • Outcome: pH normalization (7.35–7.45), improved NAD⁺/NADH (>2), energy (fatigue scale <10), and cognition (MoCA >28) within 8 weeks.

  5. Comprehensive Redox Protocol Prevents Progression To FORMAL CHRONIC DISEASE

  • Prediction: A redox-first protocol (AM sunlight, grounding, DDW, PBM, hypertonic saline, methylene blue) restores pH/éR, reverses cognitive crashes, and prevents neurodegeneration.
  • Mechanism: AM sunlight and PBM (810 nm) regenerate photoreceptors/cytochromes, grounding reduces ROS/RNS, DDW enhances DDW production, hypertonic saline improves conductivity, and methylene blue repairs mtDNA, raising pH and éR. This requires an MD to implement who knows how to assess patients. This counters nnEMF/blue light, free retinal, and the Warburg shift, restoring mitochondrial and neuronal function. Detox (mercury, mold) must follow REDOX to clear the “toxic barrel,” preventing re-toxification.
  • Outcome: Sustained pH (7.35–7.45), VCS score >7, energy/cognition recovery (MoCA >28, fatigue <10 at 6 months), and reduced Alzheimer’s risk (normal amyloid-beta on CSF).

  My decentralized interpretation of the Warburg effect is a marker of circadian mismatch, not a fuel source. Moreover, it is well supported by recent findings in the literature. The old idea that fuel source is critical is now on life support. As I’ve described, artificial light at night disrupts NAD+/NADH balance, increases ubiquitin, and drives glucose spikes “to brake” ubiquitin marking. AM UV and natural light cycles restore NAD+, lower ubiquitin, and normalize metabolism, reducing cancer, obesity, ALS, and autoimmunity, as I’ve emphasized with plants and animals sharing this response in the ubiquitin series on my website. The picture below lays it out AGAIN.

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• All of these proteins above are heme-based and have tryptophan embedded in them. They explain why chlorophyll came first in evolution, heme proteins came second, and now we are at melanin today. All of this was tied to light variation, temperature changes and oxygen tension variation in the atmosphere of LIFE.
• Altered Mitochondrial Metabolism: Blue light/nnEMF oxidizes heme in cytochrome c oxidase (MT-CO1), stalling oxygen reduction to water. This leaves O₂ as ROS, which excites molecules (e.g., lipids, proteins) to emit biophotons. Warburg redox shift (light) controls metabolism photo-bioelectrically (glycolysis dominance) because it operates when the inner mitochondrial membrane loses delta psi. This increases ROS from incomplete ETC activity (e.g., Complex III leakage via MT-CYB), amplifying biophoton output. This is why mitochondrial damage always leads to more biophoton release, spreading the failure at the speed of light.
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• Photo-Bioelectric Networks: Biophotons travel through cellular water, extracellular matrix, and bioelectric fields (e.g., gap junctions, membrane potentials), signaling mitochondrial distress. Popp suggested biophotons regulate cellular quantum coherence; in my model, their chaotic increase under stress disrupts this, spreading dysfunction. This is why the nuclear spins of atoms are lost. You need optimized optical pumping to maintain quantum coherence for health and longevity.
• 
• Are there other mechanisms in jabs that explain rapid-onset dementia, cognitive decline, and prion-like changes? Could it be the source of the negative polarity in things that give a thing a net negative flux?  Recall that all living things have a serious net negative charge.  Also, recall that water goes from a neutral polarity in its bulk state and then gains its net negative charge in the structured state when UV and IR/NIR light excite water. This structured state excludes protons and creates coherent domains that act like electromagnetic capacitors for the crystalline lattice in cellular water that resists hydrophobicity. HYDROPHOBICITY IN medications with substantivity effects on water CAN allow for the possibility of REVERSE TRANSCRIPTION TO CAUSE NOVEL DISEASES. Did you know this? Slide the above points out. Jabs do this.
• Might the answer be that water and sunlight naturally create unbelievable amounts of electrons and protons free of an energy charge?  Hydrophobicity can break this effect. This allows DNA to unwind and be copied under reverse transcription, which can cause turbo cancers. This effect would be magnified if SV40 contamination was present. DNA plasmid numbers increase the probability of such an event. Translation is widespread in annotated noncoding sequences, including untranslated regions (UTRs), introns, and long noncoding RNAs (lncRNAs), especially in cancer, aging, and neurodegeneration. 3’UTRs follow the mRNA coding sequence, regulate localization, stability, and translation, and control the selection of hydrophobic amino acids that change protein folding in cells.

Impact on Oxygen-Dependent ETC Proteins

The hardest-hit heme proteins—hemoglobin (Hb), myoglobin (Mb), Rev-erb, and ETC cytochromes—are very oxygen-dependent, making them prime targets for biophoton-mediated damage. Understanding how technology is making us ill is key.

SUMMARY

Because of the atomic organization of human cells (AMO physics), energy should always be available within the system, provided oxygen and water are hydrating our semiconductive heme and melanin proteins. When this relationship is altered for any reason, the disease manifests. The energy derived from the sun is stored coherently and ready for use over all space-time domains. Mitochondrial water production is critical in our mammalian blueprint.

The fidelity of this water creation is the basis of organisms’ autonomy and it was built around the GOE and the KT event in our history. As a result, modern organisms are never simply at the mercy of their environments because of the coherent energy stored. When the environment steals this ability from cells (nnEMF), cells are at the mercy of food and exercise.

More to the point, we don’t have to eat constantly (Leptin Rx), leaving plenty of time for other beneficial, pleasurable activities (SEX).

The other consequences are that the organism is exquisitely sensitive and free from the mechanical constraints of life on Earth and satisfies, at least, some of the basic conditions for quantum coherence. Water provides that as well.

Liquid water on Earth is quantum coherent even at ordinary temperatures and pressure. This is why Nature got the idea to build the IMM of cells around liquid water. It functionally is a naturally formed quantum computer.

 

Liquid water made in the mitochondrial matrix is the most wonderous chemical Nature has ever built because the water forms more coherent domains than the water from the hydrology cycle. Even latitude variation shows how water homogeneity changes as solar inclination affects its molecular arrangements and bond angles. Mitochondrial matrix water associates with macromolecules and membranes in cells into a gel-liquid crystalline configuration that enables enzymes and nucleic acids to function as quantum molecular machines that transform and transfer solar energy at close to 100% efficiency. Liquid crystalline water at interfaces also provides the excitation energy that enables it to split into hydrogen and oxygen in photosynthesis, simultaneously generating electricity for intercommunication and the redox chemistry that ultimately powers the entire biosphere on the 3rd rock from the sun.

Water is the means, medium, and message of life. For you to remain healthy, it must be made in large quantities in your mitochondrial matrix. Any reduction in its production will lead to disease.

PODCAST LESSONS FROM 2024: DID YOU UNDERSTAND UNCLE JACK?

Most of you did not. As I said in two podcasts with Daniel Prince and Mark Bell, an inconvenient truth will develop in front of your eyes if you look at the revenue numbers. The second highest revenue generator for banks in the U.S. is hospitals. Number 3 is drug (pharma drugs), cosmetic, and toiletry.

This is why makeup companies and sunscreen companies are lumped in with drugs. The more sun you block, the more drugs become profitable. Number 4 is the health and medical insurance industry. Number 5 is pharma wholesales. The sicker we become by DARPA light programs, the easier it is to control our behavior.

Due to control, numbers 2, 3, 4, and 5 become more profitable. The incentives dictate the outcomes in American centralized healthcare. Centralized Medicine is how you create economic slaves. This is why the DARPA MKULTRA blue light story should be huge, but nobody links things together anymore because their dopamine and melatonin levels are in the tank. Also, because of the blue light MKULTRA story, Danny Jones podcast (first one).

That is what is controlling everything at this point in America. Big Med, Big Harma, Big Insurance are fighting back. Follow the money.

WELCOME TO THE TRUTH.  HOW DID DARPA HELP THE COVID ARCHITECTS?

 

Understanding the Warburg Metabolism and Oxygen Sensitivity

The Warburg effect refers to a metabolic shift observed in cancer cells, where they predominantly rely on glycolysis for energy production even in the presence of oxygen, rather than oxidative phosphorylation (OXPHOS) in the mitochondria. This is often interpreted as a metabolic adaptation to support rapid cell proliferation. However, my decentralized hypothesis reframes this as a photo-bioelectric phenomenon driven by disrupted mitochondrial electrical resistance with a loss of mtDNA water production and amplification of melanin production from excessive ultraweak biophoton production rather than a simple fuel shift.

In the COVID jabs, the cancer pathway is more risky in any vaccine that uses SV40 as the promotor in its production. The second engineered risk is for any jab that uses lipid nanoparticles to surround spike proteins. Both of these modes are capable of causing a disrupted electrical resistance on the inner mitochondrial membrane to cause this process. The patients with Pfizer COVID jabs should be expected to present with stage 4 cancers via the mechanism I am presenting here.

The Warburg effect is well known in biology. It is described via a situation where a cell has elevated glucose utilization in cancer cells. It has traditionally been seen as an adaptation for energy (lactate production via glycolysis). However, recent research suggests it may reflect a broader metabolic and circadian dysregulation rather than a primary fuel source. Studies indicate that circadian disruption (e.g., from artificial light, shift work) uncouples mitochondrial oxidative phosphorylation, lowering NAD+ and raising NADH, leading to pseudohypoxia, ubiquitin marking, and high glucose as a compensatory response. This aligns with the idea that the Warburg metabolism signals environmental stress (e.g., light-nitrogen mismatch) rather than a cancer-specific fuel shift.

HOW DOES IT REALLY HAPPEN IN A DECENTRALIZED FRAMEWORK OF THERMODYNAMICS

I argue from first principles that the Warburg shift arises due to damage to melanopsin (from blue light or nnEMF) and mitochondrial DNA (mtDNA), which impairs water production at cytochrome c oxidase and disrupts the proton motive force (Δψ) across the inner mitochondrial membrane. This leads to decreased electrical resistance and an increase in reactive oxygen species (ROS)/reactive nitrogen species (RNS), WITH a simultaneous overproduction of ultraweak biophotons in the UV range. The resulting dehydration of melanin sheets and loss of bioelectric signaling (a DC current on the order of one trillionth of an amp) prevent regular cellular repair and apoptosis, pushing cells into an atavistic, anoxic-like state reminiscent of pre-oxygenated Earth environments.

In this context, oxygen becomes problematic for cells due to its high electronegativity on the periodic table and role as the terminal electron acceptor. Typically, oxygen facilitates efficient energy production via the electron transport chain (ECT) by reducing electrical resistance on the inner mitochondrial membrane. However, when electrical resistance drops and Δψ is compromised, oxygen’s presence exacerbates oxidative stress, further damaging mtDNA and amplifying ROS/RNS and transforming energy in ultraweak biophotons (Popp, VanWijk). This creates a vicious cycle where oxygen, instead of supporting regeneration, acts as a toxin, driving chronic disease states like cancer. The duration and intensity of the signal predict how fast the process of oncogenesis occurs in patients. This explains why people who took Pfizer jabs contaminated with SV40 and LNPs face the highest risk of turbo cancers.

Why Should We Be Treating Cancers/COVID Patients as an Oxygen Allergy?

Treating a patient with the Warburg metabolism as having an “oxygen allergy” aligns with my model because:

• Disrupted Bioelectric Regulation: The loss of electrical resistance on the inner mitochondrial membrane signals a breakdown in the bioelectric currents that regulate cellular repair and energy flow. Oxygen, by lowering resistance further due to its electronegativity, amplifies this dysregulation, mimicking an allergic or hypersensitive reaction where the body cannot tolerate a usually beneficial substance.
• Excessive Oxidative Stress: The overproduction of ROS/RNS in this state turns oxygen into a pro-oxidant rather than an antioxidant. This is analogous to an allergic response, where an environmental factor (oxygen) triggers an exaggerated and harmful reaction, leading to tissue damage and inflammation.
• Angiogenesis and Oxygen Delivery: My insight suggests that cancer cells, under this bioelectric dysfunction, stimulate angiogenesis via ultraweak biophoton release, increasing blood flow and oxygen delivery to the tumor. This paradoxical oxygen influx worsens the condition, as the mitochondria cannot utilize it effectively, reinforcing the need to limit oxygen exposure to break the progrowth cycle, similar to avoiding an allergen.
• Evolutionary Context: I propose that the Warburg shift reflects a return to an anoxic metabolic state, where cells adapted to low-oxygen environments. Treating it as an oxygen allergy acknowledges this evolutionary mismatch, suggesting that reducing oxygen availability (e.g., through hypoxia-mimicking therapies) might restore electrical resistance and halt the atavistic progression.
• Therapeutic Implications: Interventions like Vitamin C, methylene blue, or deuterium-depleted water (DDW) would work by modulating electrical resistance and mitochondrial function, potentially reducing oxygen’s toxic effects. This supports a strategy of minimizing oxygen’s role until the bioelectric and regenerative signals are restored, akin to desensitization in allergy management. This also explains why people who were treated in hospitals had high death rates. They were all given supplemental oxygen and/or ventilated. This was precisely the wrong thing to do. Why? Doing it pushed the failing colony of mitochondria with a loss of electrical resistance to ARDS. This was what I saw in ICUs and why so many people hold the belief that centralized healthcare was a killing machine for COVID-19 patients.

Connecting to A Broader Decentralized Thesis

My decentralized medicine thesis emphasizes natural light cycles (e.g., UV-A and IR-A for regeneration via melanin and POMC) and challenges centralized metabolic dogma with quantum-physical principles. The “oxygen allergy” treatment rationale fits this by prioritizing bioelectric and environmental factors (light, electromagnetic fields) over biochemical fuel shifts. It suggests that restoring mitochondrial water production, melanin hydration, and electrical resistance via natural light exposure and avoiding blue light would reverse the Warburg effect and prevent chronic diseases like cancer. It also opens the door to drug treatments that increase electrical resistance in these patients. This would include vitamin C, methylene blue, Ivermectin, and Fen/ben combos. All these drugs increase electrical resistance on the inner mitochondrial membrane, which is why they work. Water also helps because it is Nature’s Faraday cage for EMFs. This fundamental insight is lost in centralized healthcare.

WHY IS DEUTERIUM DEPLETED WATER USEFUL IN CANCER?

If you understand what I have laid out here, cancer is photo-bioelectrical electrocution of the inside of a cell. Why? Dehydrated melanin causes massive amplification of the DC electric current a cell makes. Remember what Nick Lane told us in his book Power, Sex, and Suicide? A healthy inner mitochondrial membrane contains 30 million volts of electric charge. This charge would typically be devastating to a cell. When I read this fact long ago, it raised a big question: How does a cell dampen the electrical charge?

Pure water containing no ions is an excellent insulator of electric charge. This is why Nature created the DDW, which was made by mtDNA at cytochrome C oxidase. What was the stimulus for her to gain this wisdom? It was the wound of the Great Oxidation event. The toxic wound Nature sustained from the creation and eventual dominance of oxygen on Earth was a stimulus that drove massive electrocution and endosymbiotic growth or the merger of the FIRST two domains of life on Earth 650 million years ago. Nature would never have innovated mitochondria without this photo-bioelectrocution caused by oxygen.

Pure water is non-deuterated water.  But not even “deionized” water is completely free of ions. Water undergoes auto-ionization at any temperature above absolute zero. Further, because water is such an excellent solvent, it almost always has some solute dissolved in it, most frequently a salt. This increases its electrical conductivity. This explains why all systems in the body except the matrix have saltwater in them. It also points out something else.

nnEMF dehydrates us, leaving more NaCl in less water and increasing electrical conductivity. When you consider how melanin responds electrically to dehydration, you should begin to see why this is a double whammy for a cell or organism. This is why nnEMF causes so many diseases. When you understand the wiring diagram basics, you will realize that we are effectively self-electrocuting ourselves on a chronic basis when there is a loss of electrical resistance in our membranes. This is what causes pro-growth programs in cells. This same set of circumstances caused endosymbiosis. Oxygen drove the growth and fusion between bacteria and Archea to make a mitochondrion. That was the original cancer on Earth. Shocking truth bomb, huh?

WHY CAN I SAY THIS? The basics of electrical resistance is a physics story.

Suppose water has even a tiny amount of such an impurity. In that case, it can conduct electricity readily, as impurities such as salt separate into free ions in aqueous solution by which an electric current can flow.

It is known that the theoretical maximum electrical resistivity for water is approximately 182 kΩ·m²/m (or 18.2 MΩ·cm²/cm) at 25 °C. This figure agrees well with what is typically seen on reverse osmosis, ultrafiltered, and deionized ultrapure water systems used, for instance, in semiconductor manufacturing plants.

A salt or acid contaminant level exceeding even 100 parts per trillion (ppt) in ultrapure water begins to lower its resistivity level noticeably by up to several kilohm-square meters per meter (a change of several hundred nanosiemans per meter of conductance).

NICK LANE’S 30 MILLION CURRENT

Pure water has a low electrical conductivity, so Nature chose this water format to be adjacent to the inner mitochondrial membrane post endosymbiosis to prevent further self-electrocution. As a result, this increases significantly upon solvation of a small amount of ionic material water such as sodium chloride.  Thus, the risks of electrocution are much more significant in water with the usual impurities not found in pure water.  This is why people on heart transplant lists do better when they are given hypertonic saline solutions. In cardiac care units, we induce electrocutations when we use a defibrillator to restart a failing electrical organ called a HEART.

Recall that in centralized medicine, when people have a cardiac arrest, we use defibrillators to jump-start their hearts with an electrical charge because the mtDNA cannot create it. We can improve this situation by using a hypertonic salt solution before the defibrillator. This should be a change made to all ACLS and ATLS protocols. Hypertonic saline is also a key player in decentralized medicine treatment plans. Neurosurgeons are the one group of centralized MDs who learn how to use hypertonic saline in neurosurgical emergencies.

WHY YOUR WOUNDS CREATE YOUR WISDOM: ALL-NIGHT SURGERY LESSONS

This is why I quickly figured out biophysics once I allowed the wounds of my patients to teach me new lessons. Remember that little girl I told you about in the Danny Jones podcast, whom I did a 24-hour surgery with Metallica blasting all night? I told the world it was the most critical case I had ever handled in my career. Why has no one ever asked me why I said that in any podcast or Q&A?  PODCAST LINK

It was that case that taught me why methylene blue and hypertonic saline were tools I could use to save her life. Few centralized MDs understand biophysics. That night, I began using mannitol on that little 16-year-old girl, and I noticed something unusual. Every time anesthesia dosed her with it, it dropped her blood pressure dramatically. I realized I was inducing acute heart failure because she was in neurogenic and cardiac shock at the same time. This is when i began to scream at my neurosurgery staff and told him to get out of my way and I am going to try something new. I realized I had to jump-start her heart and limit her brain swelling to get more time to remove the bone from her brain. So, I immediately stopped the mannitol and gave her hypertonic saline to defibrillate her heart using basic biophysics. NaCl increases electrical currents, so I could jump her heart, giving me more time to save her life.

Anesthesia told me that once I did this, her BP stabilized, and her ECG improved. As I continued to work, Metallica’s song Nothing Else Matters came on in the background. The lyrics hit me just at the right time. Now, go listen to the lyrics sometime.

I realized that after switching her IVs to hypertonic saline to save her failing heart due to the massive intracranial injury, I had to simultaneously add a methylene blue drip to run in slow to increase the electrical resistance in her brain’s demolished mitochondria to give me more time to work in her brain. This was akin to using biophysics to jump her heart while her brain was damaged.

These two seemingly incongruent things should NOT be done simultaneously, but I knew it was my only chance to save her.

I knew this was not what I was taught, but I had the strangest feeling then. I had to do it because it was the right thing to do in my mind. That is what stimulated the massive fight between my neurosurgeon staff and me. That is when I told everyone in the room that they should leave right then if they did not like or agree with what I was doing. Only the staff neurosurgeon left the OR that night. If you listened to the Danny Jones podcast, you know the rest of the story now. That is why these tools are dangerous in the wrong hands.

What did Metallica whisper to me that night in the operating room?

So close, no matter how far
It couldn’t be much more from the heart
Forever trusting who we are
And nothing else matters

To this day, I get tears in my eyes when that song is played around me.

Wounds create our wisdoms and that is why we must embrace the pain and suck that life deals.  A wound is never a mistake if it teaches you a lesson you learn from. Then, it becomes tuition. I said that in 1998, at the Morbidity and Mortality conference, they reviewed this case. Not only didn’t i get canned, my neurosurgery staff all came up to and told me what I did that night was crazy wise. All except one guy. The centralized neurosurgeon whom I threw out of the room was a career military man who had contracts with DARPA working on primates.

That is why this podcast is a key listen for any Savage.

https://www.youtube.com/watch?v=zCGnMY9FSNg

The Next Step?

While my model is intriguing and integrates diverse data, it relies on a bioelectric paradigm that departs from mainstream centralized mitochondrial and cancer biology. To the centralized players, my concept of oxygen as an “allergy” will be considered metaphorical. My perspective is quite different. It is AXIOMATIC. As such, when I returned to ICU medicine as a trauma neurosurgeon, I found these ideas very useful for framing therapeutic strategies for hospitalists and nurses (e.g., hypoxia therapy, redox modulation) who cared for these patients.

My decentralized interpretation of the Warburg effect is a marker of circadian mismatch, not a fuel source. Moreover, it is well supported by recent findings in the literature. The old idea that fuel source is critical is now on life support. The next blog will blow that out of the water.

As I’ve described, artificial light at night disrupts NAD+/NADH balance, increases ubiquitin, and drives glucose spikes to brake ubiquitin marking. AM UV and natural light cycles restore NAD+, lower ubiquitin, and normalize metabolism, reducing cancer, obesity, and autoimmunity, as Ive emphasized with plants and animals sharing this response in the ubiquitin series on my website.

LITERATURE SUPPORT

Recent Research on the Re-Interpretation of the Warburg Metabolism As a Circadian Shift

• Circadian and Metabolic Perspectives in the Role Played by NADPH in Cancer
  • This source discusses the Warburg effect as metabolic reprogramming in cancer cells driven by NADPH availability and touches on its connection to circadian rhythms. It notes that the Warburg effect is linked to circadian disruption. It provides a direct context for NAD(P)H imbalances in cancer, aligning with the decentralized reinterpretation of the Warburg effect as a signal of metabolic and circadian dysregulation rather than a fuel source.
• Associations among Metabolism, Circadian Rhythm, and Age-Associated Diseases
  • Some sources have explored the connection between the Warburg effect, tumorigenesis, and circadian rhythm disruptions, particularly in age-associated diseases like cancer, obesity, and diabetes. They hypothesize that circadian misalignment (e.g., from artificial light) activates the Warburg effect as a cell-autonomous transformation, supporting the view that it reflects an environmental circadian mismatch rather than a metabolic adaptation for energy.
• Cancer metabolism in space and time: Beyond the Warburg effect
  • Space medicine sources have delved into cancer metabolism’s spatial and temporal aspects, suggesting that the Warburg effect may involve broader metabolic and environmental factors beyond simple glycolysis. It provides a foundation for reinterpreting the Warburg effect as circadian and light-related factors influence it. It is consistent with my argument about artificial light’s role in uncoupling light-nitrogen cycles in the mitochondria.
• Oncogenic fatty acid oxidation senses circadian disruption in sleep-deficiency-enhanced tumorigenesis.
  • This study links circadian disruption (e.g., sleep deficiency) to fatty acid oxidation (FAO) and the Warburg effect in cancer. It shows how clock gene dysregulation (e.g., CLOCK, BMAL1) and NAD+ levels drive tumorigenesis. It supports my hypothesis that the Warburg effect signals circadian mismatch from artificial light, with NAD+/NADH imbalances and ubiquitin marking playing key roles.
• Exposure to artificial light at night: A common link for obesity and cancer?
  • This source highlights how artificial light at night (ALAN) disrupts circadian rhythms, increasing risks of obesity, diabetes, and cancer through mechanisms like melatonin suppression, NAD+ decline, and metabolic reprogramming. It aligns with my interpretation of the Warburg effect as a response to circadian misalignment from artificial light, connecting it to ubiquitin, NAD+/NADH, and disease.
• Artificial Light at Night and Type 2 Diabetes Mellitus
  • This review consolidates studies from 2000–2024 on ALAN’s association with type 2 diabetes, obesity, and cancer, emphasizing circadian disruption, NAD+ decline, and glucose metabolism changes. It supports the decentralized view that the Warburg effect and related metabolic disorders stem from artificial light-induced circadian mismatch, not just metabolic adaptation.

    DECENTRALIZED MEDICINE HAS ANSWERS FOR THE DARPA-INDUCED CANCER WAVE

  • My insights reveal that shorter wavelengths (e.g., blue light at night) increase cancer risk by uncoupling light-nitrogen cycles inside mitochondria, lowering NAD+ and pseudohypoxia and raising ubiquitin. At the same time, AM sunlight provides massive red light exposure without UV (290–420 nm), which promotes water production for a latitude-determined duration. This improves mtDNA hydration before we stimulate the regenerative stimulus. Hydration endogenously becomes a Faraday cage for the destroyed inner mitochondrial membrane that has lost its electrical resistance.
  •  

    As the day progresses, sunlight adds other light frequencies that promote diurnal regeneration, oxygen delivery, and health via melanin, POMC, and porphyrins. The Warburg metabolism is an epigenetic post-translational signal from mitochondria that light mismatches, not a fuel shift, cause the condition. This implies that the surface photo-electrochemistry of the eye, skin, and gut can outpace the biochemistry inside the cell in order of critical importance.

  • When someone exhibits the Warburg metabolism, the patient must be treated like they have an oxygen allergy. Why?
  • Adding oxygen to a mitochondrial decreases electrical resistance, altering the regenerative currents stimulated by mtDNA and melanin. A red light stimulates water production, while ultraweak biophotons stimulate POMC translation to create endogenous melanin.

• The mtDNA water production engulfs melanin to RAISE electrical resistance to make a DC electric current that is one trillionth of one amp. This current is used to renovate and regenerate mammalian tissues. If this signal is awry and the electrical resistance drops, this creates lowered NAD+, massive upregulation of ROS/RNS, and massive overproduction of ultraweak biophotons in the UV range. As a result, too little mitochondrial water production is made.
• At the same time, too many ultraweak biophotons are made, creating a situation where melanin sheets created are chronically dehydrated in the cell, and there is persistent chronic ROS/RNS and no generation of the bioelectric signal that the cell needs to self-repair itself. This situation leaves the cell with a massive epigenetic upregulation of endogenous melanin, which becomes highly conductive in the cell, ruins the surrounding regions, and affects the local production of melatonin by mitochondria, making up 95% of melatonin in humans. It is not a pineal story. That is functional medicine nonsense.

• The loss of the cell’s melatonin production will not allow apoptosis to eliminate the poorly functioning cell. When these bioelectric changes occur, the cell reacts atavistically to return to an older evolutionary format when cells were in anoxic environments before the great oxidation event. As a result, the body responds by lowering oxygen delivery to the defective mitochondria because oxygen is the terminal electron acceptor.
• Due to its high electrionegativity on the periodic table, it reduces electrical resistance. During a Warburg shift, the cell needs a raised electrical resistance to stop the process. Organisms with melanopsin damage must vary resistance to bioelectrical flows to survive. Resistance in cells directs energy flow and computes information, which drives biochemical pathway decision-making.

• • When melanopsin damage occurs from blue light or nnEMF, the cells use glucose, alanine, and glutamine. These choices were the best choices when the Earth was anoxic. The Warburg shift is needed in a cancer state to stop the progrowth epigenetic program that has started due to melanopsin damage and mtDNA mutation, stopping water production at cytochrome c oxidase. Loss of oxygen increases electrical resistance on the inner mitochondrial membrane. Once melanopsin damage occurs in any tissue, water production slows down at cytochrome c oxidase, and oxygen use diminishes because of the destruction of delta psi on the inner mitochondrial membrane.
  • Picard and Levin recently wrote an article in which they made my decentralized case for me when they said, “Organisms adapt and optimize fitness by achieving lasting changes in energy resistance.”
  • This explains why Vitamin C, methylene blue, and DDW all operate to help prevent a turbo cancer. They act to limit the stay currents coming from the inner mitochondrial membrane from the damage of SV40 and LNP-spike proteins destroying it. The Resistance to energy flow (éR) triggers signaling outputs whose information content activates (epi)genetic regulatory pathways, which open communication between mtDNA and the nDNA to produce enduring cellular, physiological, anatomical, and behavioral changes. DDW also protects your cells and organs from this self electrocution that leads to turbocancers.
  • These changes feedback to minimize and optimize éR over the long term, enhancing the system’s efficiency, environmental fitness, and health for the environment sensed by mitochondria. Cancer is not a metabolic disorder. It is a Photo-bioelectric disease that can be amplified by DARPA bioweapons.  Turbocancer happens because the duration and intensity of the jabs predict it. After all, bioelectrical activity demolishes the resistance to energy flow (éR) rapidly.
  • You must start at the magneto-heliosphere for a decentralized medical lesson on mtDNA diseases like cancer. Learn the actual ticket that moves the needle of all humans. You must learn that when your Inner mitochondrial membrane has no electrical resistance, this signals a poor redox charge (delta psi) on the inner mitochondrial membrane. This is when oxygen becomes a toxin for most chronic diseases. Your centralized shills will never get you to this level of understanding.
  • Cancers grow because blood flow amplifies to these regions due to the excessive release of ultraweak biophotons. This action stimulates angiogenesis, bringing more oxygen, not less oxygen, to the cancer. Since oxygen has a high electronegativity, it draws current across the damaged landscape of the inner mitochondrial membrane, and this causes massive amplification of ROS and RNS. This causes the progrowth situation to persist and exist. The goal is to limit the spread of that current by any means necessary.
  • People forget that oxygen’s toxic nature drove endosymbiosis on Earth 650 million years ago. Otherwise, Nature would have never innovated mitochondria. Oxygen’s paramagnetism keeps us alive in an oxygenated atmosphere, but it becomes deadly in one where there is a loss of electrical resistance on the inner mitochondrial membrane.
  • These are the critical missing pieces of information in producing all chronic disease epidemics. This also explains why turbocancers are now present at record rates in the jabbed. Become the disruptor, or be disrupted by DARPA centralized medicine machine. They are tapering the Ponzi scheme and it should be no mystery to anyone here how they are doing it.
  • Artificial light damages melanopsin, which is associated with dehydration. This exacerbates the bioelectric signals, explaining modern disease epidemics. This aligns with my decentralized medicine thesis, reinforcing the need for natural light cycles and challenging biological dogma with quantum-physical wisdom. MAHA = HAHA
  • SUMMARY

In summary, a patient with the Warburg metabolism should be treated like they have an oxygen allergy because the loss of mitochondrial electrical resistance turns oxygen into a toxic agent that amplifies oxidative stress, disrupts regeneration, and drives pro-growth signals. Restoring bioelectric balance and resistance is a mandatory clinical need and is key to managing the trubocancer state.

• CITES
  • Wang, R.-H., & others (2017). Associations among metabolism, circadian rhythm and age-associated diseases. Aging and Disease, 8(5), 691–709.
    • DOI: 10.14336/AD.2016.1101
    • My Notes: This review by Wang and colleagues discusses the connection between the Warburg effect, tumorigenesis, and circadian rhythms, hypothesizing that circadian disruption activates cell-autonomous transformation to generate the Warburg effect. It emphasizes NAD+-dependent sirtuins (e.g., SIRT1, SIRT3) and clock genes (e.g., CLOCK, BMAL1, PER2) but focuses on genetic and metabolic pathways, potentially overlooking the environmental role of artificial light and light-nitrogen coupling I’ve highlighted. It critically examines this as an establishment narrative that simplifies the Warburg effect as a biochemical adaptation, missing the broader photobiological and quantum context of circadian mismatch from artificial light.
  • Masri, S., Kinouchi, K., & Sassone-Corsi, P. (2015). The emerging link between cancer, metabolism, and circadian rhythms. Nature Medicine, 21(12), 1795–1803.
    • DOI: 10.1038/nm.0271-8
    • My Notes: Masri, Kinouchi, and Sassone-Corsi explore the connection between circadian disruption, cancer metabolism, and the Warburg effect, noting epidemiological links to artificial light at night (e.g., shift work). They discuss clock genes (e.g., CLOCK, BMAL1, CRY, PER) and metabolic reprogramming but focus on transcriptional feedback loops, potentially underestimating the role of light-nitrogen uncoupling, ubiquitin marking, and NAD+/NADH imbalances I’ve emphasized. It challenges the establishment narrative for prioritizing molecular biology over environmental light dynamics, aligning with the interpretation as a circadian mismatch signal.
  • Sahar, S., & Sassone-Corsi, P. (2009). Metabolism and cancer: The circadian clock connection. Nature Reviews Cancer, 9(12), 886–896.
    • DOI: 10.1038/nrc2747
    • My Notes: This review by Sahar and Sassone-Corsi examines how circadian clocks regulate cancer metabolism, including the Warburg effect, and links it to circadian disruption. It mentions light exposure as a zeitgeber but focuses on genetic and biochemical mechanisms (e.g., sirtuins, NAD+), while missing the decentralized photobiological insights about artificial light’s role in uncoupling light-nitrogen cycles and driving ubiquitin and disease. I critically assess this as an establishment perspective that overlooks quantum and surface chemistry roles, supporting my broader model.
  • Korennykh, A., & others (2019). The metabolites NADP and NADPH are the targets of the circadian protein Nocturnin. Nature Communications, 10(1), 10125.
    • DOI: 10.1038/s41467-019-10125-z
    • My Notes: This study by Korennykh and colleagues at Princeton University investigates Nocturnin’s role in fat metabolism and circadian rhythms, showing its action on NADP+/NADPH and NAD+/NADH, linking to energy regulation. It supports my point about NAD+/NADH imbalances in circadian disruption and metabolic diseases (e.g., obesity, diabetes) but focuses on biochemical mechanisms, missing light-nitrogen coupling and ubiquitin dynamics I’ve described. I challenge the establishment narrative for not exploring environmental light’s photobiological impact, aligning with the decentralized reinterpretation of the Warburg effect.
  • Zhang, Y., Hu, K., Tang, Y., Feng, Q., Jiang, T., Chen, L., Chen, X., Shan, C., Han, C., Chu, W., Ma, N., Hu, H., Gao, H., & Zhang, Q. (2024). Interactive correlations between artificial light at night, health risk behaviors, and cardiovascular health among patients with diabetes: A cross-sectional study. Journal of Diabetes, 16(10), e70008.
    • DOI: 10.1111/1753-0407.70008
    • My Notes: This recent study by Zhang et al. links artificial light at night (ALAN) to diabetes, obesity, and cardiovascular health, showing circadian disruption, NAD+ decline, and metabolic changes. It supports the decentralized view of the Warburg effect as a circadian mismatch signal from ALAN. Still, it focuses on behavioral and epidemiological data, missing quantum and photobiological mechanisms (e.g., light-nitrogen, ubiquitin). I critically examine this as an establishment narrative prioritizing classical epidemiology over my decentralized insights.
  • Engin, A. (2024). Misalignment of circadian rhythms in diet-induced obesity. Advances in Experimental Medicine and Biology, 1460, 27–71.
    • DOI: 10.1007/978-3-031-63657-8_2
    • My Notes: Engin’s review connects circadian misalignment (e.g., from artificial light, shift work) to obesity, diabetes, and cancer, including the Warburg effect as a metabolic response. It mentions NAD+, sirtuins, and clock genes but focuses on biochemical and nutritional pathways, potentially overlooking your light-nitrogen coupling and ubiquitin marking. I challenge the centralized establishment narrative for not addressing photobiological and quantum mechanisms, supporting the decentralized reinterpretation as a circadian mismatch driven by artificial light.
  • GAME, SET, MATCH.

If you ever wondered why I have written about tinnitus so much today is the day where the whole story comes together.

The inner ear of humans and other mammals is contained within the temporal bone. It comprises the cochlea and vestibular organs involved in sound perception, spatial orientation, and balance. Apart from these sensory systems, the inner ear contains several diverse accessory cells, including pigment‐containing melanocytes. The existence of pigment in the inner ear has long been known. It was first described in 1851 by the Italian physician and early pioneer of inner ear microanatomy, Alfonso Corti, but it took 80 years before the pigment was identified as melanin (Wolf, 1931). Studies investigating the function of melanocytes in mice unraveled their role in generation of the endocochlear potential and hearing (Hilding & Ginzberg, 1977; Igarashi, 1989; Schrott & Spoedlin, 1987; Steel & Barkway, 1989; Steel et al., 1987) and balance (Marcus & Wangemann, 2010; Palma et al., 2018).

The Human Cochlea is an amazing design of nature that DARPA is Targeting

Our spiral-shaped auditory organ is a marvel of precision, converting sound waves into electrical signals via hair cells and the auditory nerve. But I have proposed it could do more, specifically with melanin as the key player. The melanin sheet in the cochlea explains how Becker’s work, My work, and DARPA all fit together.

The cochlea contains a lot of melanin deep inside our skull, particularly in the stria vascularis, a region critical for maintaining the endocochlear potential (a bioelectric gradient that powers hearing).
This melanin isn’t just decorative in the ear, it is there as a condensed matter wide band gapped semiconductor to do something most of you did not anticipate. It is adjacent to a place in the brain with no blood-Brain barrier. DARPA learned this from Dr. Allan Frey, who discovered the pulsed microwave effect when he was working for them during the MKULTRA era.
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Centralized thinkers believe melanin is thought to protect against oxidative stress and possibly modulate ion flow. Melanin has multi-use purposes. DARPA found out from Frey’s work that it inhibits Robert O. Becker current of regeneration and because it does this is induces traumatic brain injury.

Physics tells us that melanin absorbs all parts of the electromagnetic spectrum. As such, it can translate those light signals into electrical impulses or heat, subtly altering the cochlea’s function. Frey proved this was possible with pulse microwaves at the same time Becker uncovered the regenerative current for DARPA. Both of the scientists were silo’d and their work was kept from one another. This Manhattan style scenario comes straight out of DARPA design. Allen Frey was hired by DARPA to find out how the Moscow Embassy Incident occurred in the mid to late 1960s, which I mentioned in the Marty Bent podcast.

Here’s where the story gets wild: EMF detection implies the cochlea could pick up a lot of different electromagnetic noise beyond audible sound—think wireless signals, ambient EMF from tech, or even natural geomagnetic fluctuations from the Schuman resonance. Condensed matter physics supports this action; melanin’s disordered structure and electron mobility could make it a natural antenna, resonating with EMF fields and transducing them into something the auditory system (or even mitochondria downstream) might perceive. This fits my earlier thread about sensory dyssynchrony in ASD (QE #45) and what occurs in the jabbed.

Almost any cell with a circadian mismatch has a lowered NAD+ and exhibits a Warburg shift because blue light increases blood glucose and insulin levels. Becker found this in Penscola’s study of pilots in 1969 for the US NAvy, and in 2011, Nora Volkow confirmed it for NADA, the NIH, and SRI (DARPA) in 2011.

If the cochlea is misinterpreting different EMF stimuli as sensory input in the brain, DARPA found it could overload the thalamus and cause traumatic brain injuries in the acoustic arrays. This would throw off mitochondrial timing by inducing mtDNA mutations; it would lower mtDNA melatonin, water, ROS, and CO2 production, especially in sensitive populations with higher heteroplasmy rates who have preexisting diseases, atrophic skin, unmyelinated brains, and pale skin. For example, soldiers in war or kids with ASD exhibit these symptoms. This is precisely what I found in soldiers I evaluated in Hattiesburg, Mississippi, from Camp Shelby Joint Forces Training Facility who came home with acoustic injuries, TBI, and PTSD. Dr. Frey figured this out for DARPA in the 1960s, and DARPA developed direct energy weapons to target this region.

HOW DOES IT HAPPEN IN DARPA TARGETED PROGRAM?

• I’ve emphasized in many blogs that blue photons’ high energy makes them less common for stable, long-term pigmentation or fluorescence in the skin of mammals, but I doubt you understand why I have say it repeatedly. The reason is simple, Nature makes blue light signaling rare in mammals. As a result, this mades melanin the most critical photoreceptor in mammalian biology. Why, Anything that is rare becomes a precision tool for the fidelity of the signal. This was what Claude Shannon taught DARPA in the 1950s when he worked for them at Bell LABS. Melanin’s ability to absorb and dissipate electromagnetic energy ONLY when it hydrated prevents brain damage from blue and UV light. When melanin is not hydrated due to mtDNA mutations induced by nnEMF targeting, people get chronic diseases because heteroplasmy rises. HYPERLINK
• Did you know that condensed matter physicists found that melanin in its natural form is too untidy on a molecular level to conduct electricity with much efficiency? Did you know melanin loses its conductivity when exposed to water?
• People forget melanin creates a DC electric current signal when it is illuminated by sunlight. But few people realize this current is dampened. Eumelanin does the same in our skin and ears. These are the same melanins DARPA has been studying in our skin and our cochlear and semicircular canals. (See the citation below.)
• Eumelanin in its natural form is too untidy on an atomic molecular level to conduct enough electricity with much efficiency for industrial use, but this points out why Mother Nature uses it in mammals. This was the key thing I told Becker in our meeting that I have not mentioned anywhere yet. I learned this from treating PTSD soldiers at Camp Shelby in Mississippi coming home from the theatre of war. All of them got hit with a massive vaccination program before they left for deployment.

• Hydrating melanin in any mammal is done by local mtDNA metabolism. Cytochrome C oxidase creates the water that hydrates melanin. Hydrating melanin decreases the DC electric current to 1 trillionth of an ampere. This is what Decker found in his seminal work on how RBC dedifferentiate to pluripotential cells during wound healing and regneration. for industry, hydrate melanin is useless to electrical engineers, but hydrated melanin is just what the doctor ordered for a dying cell that needs to regenerate. It also explains why the use of the drug melotan is deadly in humans. I studied many bodybuilders, and people who use Melotan, and they tend to die the same way the jabbed die post vaccination. Many of the symptoms people get from melotan use corresponds to what Targeted Individuals report. See the video above. I learned this from my friendship with Charles Poliquin in the early 2000s
• This precision with which water operates at a quantum mechanical level preciselly dampens the DC currents used in cells which causes them to regenerate their tissues. This is what actually drives regeneration and healing in all mammals. This nuance is lost on many who do not understand how the non visual photoreceptors operate in man. DARPA’s research figured this out from combining the work of Frey and Becker from the 1960s.
• Suppose your mtDNA is not making water for any reason (disease due to heteroplasmy or exogenous XRT). In that case, the amount of the DC electric current melanin makes becomes toxic to neurons locally and distally connected to in the brain. This stimulus threatens to burn out local and distal circuits, leading to diseases like cancer and the ones we see in our chronic disease epidemics. This is what we are seeing today in the jabbed and targeted. This is what I believe killed Charles and many of his bodybuilding clients. I spoke about this in the Melanin Rx for mammals blog. Please re-read it.
• This is especially ampolified in all people who took the mRNA vaccines. Why? The spike protein is a mitochondrial toxin that diminshes water production from mitochondria. All soldiers were loaded with jabs before heading to war.
• At the same time, melanopsins’ role in nonvisual photoreception (e.g., via ipRGCs or in skin melanocytes) normally supports circadian and metabolic regulation and the function of our frontal lobes via the central retinal pathway connected to the SCN and habenular nucleus. This is why the jabbed all have circadian dyssynchrony and mental and cognitive issues. This is what long covid really is.

• The mtDNA mutations you heard about in Decentralized Medicine #35 lead to neuronal dehydration, which AMPLIFIES the DC current liberated from melanin and this current amplifies the bioelectric signal connecting it to tracts distally. This wiring diagram results and induces a trauma response (TBI). This what Dr. Ber says above, but has no idea how it occurs. Now you do. This is why DARPA’s directed energy weapons are so effective and cause unusual injury patterns in DTI MRIs. It requires a neurosurgeon to know the mechanism to understand what to look for. We are now seeing the same injury pattern developing in the jabbed because the mechanism is identical to what happens in soldiers with TBI. only the intensity of the TBI is different. People in DARPA programs (targeting) and in WAR have massive TBI injuries. The centralized MDs they see how no idea what they are looking at. This explains why DARPA told the military brass during the Iraq war to provide soldiers with RO water in the desert. DARPA knew exactly why to make the recs. It could limit TBI in soldiers.

• This neuronal loop between melanopsin and melanin prides an energetic constraint in mammalian tissues. This why blue light is SPECIFICALLY reserved for specialized functions (e.g., circadian timing, vision, frontal lobe function) rather than widespread use in the brain. This reinforcing its rarity and utility as a high-information signal. Today’s modern world destroys that fidelity because blue light is everywhere in our environment. DARPA found that out during the MKULTRA program I mentioned in the original Danny Jones podcast.

My earlier point about blue light’s role in circadian biology focused on melanopsin disrupting the circadian clock (SCN) in the retina. With the more recent data I have shared with you over 20 years I have shown you now that melanopsin is present in fat, arteries, the brain, and most other human tissues. This implies that the biochemists, food gurus, and vaccinator’s theories of safety need to be revisited and revised. Moreover, this situation has massive implications for the jabbed and the Targeted Individual in 2025.

• Broader Circadian and Metabolic Effects of Blue light Signal Fidelity:
  • Fat Tissue: Melanopsin in adipocytes allows adipocytes to directly sense blue light (e.g., from artificial sources) and influence lipid metabolism, adipogenesis, or insulin sensitivity. when you begin to realize that blue light activates melanopsin in fat tissue at night, what does this imply? The light stress immediately causes a vassopressin release in the pituitary. (QE#23) Vasopressin release at night disrupts normal metabolic rhythms, raising blood sugar, promoting fat storage, insulin resistance, and obesity—amplifying the pathways in the slide (e.g., increased appetite, insulin resistance). The jabbed’s fat mass will help them by drawing the LNPs of the spike protein, to our fat depots so we can destroy it there safely and later regenerate tissues it destroyed. Those who do not have SQ or visceral fat will be at higher risk for organ damage. This is precisely the opposite of what centralized medicine tells patients. You’ve heard me say it on many podcasts, but now you have my rationale for this wisdom.

• Arteries and Cardiovascular Health: Melanopsin in vascular tissue responds to nnEMF light exposure, affecting blood pressure, endothelial function, or cardiovascular risk. These things all cause PAD and clotting risks. Chronic blue light exposure at night might contribute to cardiovascular complications associated with obesity and diabetes. Those who took any of the jabs are certainly subject to massive clotting and bleeding wherever melanopsin and blue light meet in a tissue. The aftermarket data proves this in 2025.
• Brain Dominance:  Melanopsin is the most dominant opsin in the brain, melanopsin’s widespread expression suggests it plays a central role in coordinating light-dependent neural and hormonal responses. This is why everyone’s hormones panels look like shit today. If you are jabbed they are even worse. Blue light at night disrupts not just the suprachiasmatic nucleus, but other brain regions (frontal lobes) involved in appetite regulation (e.g., hypothalamus), mood (e.g., amygdala), and autonomic functions, further exacerbating metabolic and behavioral dysregulation.

• Implications for Ubiquitous Blue Light present in the jabbed person’s world. The risk for chronic disease generation is astronomical.
  • Because melanopsin is in fat, arteries, and the brain, artificial blue light at night confuses the retina of the human who is jabbed; it directly affects energy storage, blood vessel function, and central nervous system regulation. This systemic disruption explains the prevalence of body and physiological changes reported by the jabbed much more comprehensively than retinal effects alone.
  • Public Health Concerns: The widespread presence of melanopsin suggests that reducing blue light and nnEMF exposure day and night (e.g., through lighting design, screen filters, or behavioral changes) is even more critical than previously thought in those who are jabbed. It’s not just about sleep or regeneration; it’s about protecting metabolic, cardiovascular, and neural health across the body as the jabbed navigate the modern tech-driven world. The distal TBI this creates with destroy and atrophy brain function leading to many diseases and death in covert ways if the clinician is unaware of the mechanism. This is how DARPA learned to begin tapering the PONZI scheme.

• • Evolutionary Mismatch is accentuated in the jabbed: Humans evolved under natural light cycles, where blue light was rare at night. Constant exposure to artificial blue light activates vasopressin and melanopsin in tissues not adapted to nocturnal light, creating a profound mismatch that drives ALL chronic disease. Even MAHA misses this low-hanging fruit.
  • MAHA’s avoidance of banning the mRNA platform early in the DJT term explains why Bobby was confirmed. As long as DARPA forces Bobby to keep the mRNA platform in place, the desired effect of tapering the Ponzi scheme will continue unabated.
  • Adding mRNA jabs to the animals in our food supply will also be a death trap for those who eat the food. This explains why Bill Gates is buying up farmland to grow his food and breed animals that aren’t GMOs. Once the Ponzi scheme is tapered, his properties and farms will be new non GMO food source for the NWO, who avoided these DARPA programs. They knew the game plan first due to their CIA/DoD connections via tech companies.
• Connection to My Decentralized Insights From the Marty Bent podcast on DARPA
  • My point about blue light’s rarity in nature and its role as a “high-information” signal via Shannon’s information theory remains highly relevant to the jabbed. Melanopsin’s systemic distribution suggests that this rare signal was evolutionarily optimized for daytime use, not nighttime exposure. The ubiquity of artificial blue light overwhelms this signal, diluting its informational value and causing widespread dysregulation, which causes the aftermarket disease we are seeing.

My recent discussions and podcasts from 2023-2025 on the leptin melanocortin pathways, LNPs, melanin, POMC, and Vitamin D3 also tie into the jabbed story: See the JONES/BOWDEN/KRUSE podcast and the last slide I presented in it.

The circadian mismatch drives the Warburg shift to cause a higher cancer spike than we saw in the 1950s SV40 debacle. I showed that slide at the end of the Danny Jones Bowden Kruse podcast (above) to show you that the new mRNA spike line has a stepper slope than the one associated with the Cutter Incident. This slope is greater because the risks today are worse because melanopsin’s light sensitivity is being destroyed. Melanopsin evolved to complements melanin’s photoprotective role, and the in the jabbed DARPA has engineered the bioweapon to uncouple us from the ability to regenerate. At the same time, POMC pathways (e.g., MSH production) interact with melanopsin to regulate metabolism and circadian rhythms in response to light. This is why GATES, FAUCI, and your governments wanted you inside out of the sunlight. This is why they outlawed Vitamin D use.

TINNITUS IS A DARPA CREATED DISEASE USED IN THEIR RCT BEING DONE AT SCALE

Tying this to the mitochondrial medicine story Dr. Doug Wallace laid out, cochlear melanin appears to act as a semiconductive relay switch based on my description above. This is something Robert O. Becker worked out in bone. The cochlea has a different way of operating with the endolymph and lacks a BBB. Melanin is a material that can both inhibit and promote electric flow, classifying it as a semiconductor. The molecule switches between resistive and conductive states depending on the amount of water in its local environment. This water can have its dielectric constant varied by atoms inside the liquids it is adjacent to. In the cochlea, potassium is that atom. The amount of potassium in endolymph is quite unusual compared to other organs. This “switching” property is a crucial mechanism that builds the backbone of all types of computers. The human cochlea is a quantum computer that DARPA has targeted initially using Allan Frey’s work.

WIDE BANDGAPPED SEMICONDUCTION: WHY I WAS RELUCTANT FOR HUBERMAN
Melanin as a wide-bandgap semiconductor is a key insight for the decentralized MD. Wide-bandgap materials can handle high-energy input EMF (like the entire electromagnetic spectrum) and switch between insulating (resistive) and conducting states based on external conditions. For melanin, hydration is the toggle switch the ear uses. When it’s dry, it’s resistive; add water, and it becomes conductive, thanks to proton hopping and electron mobility within its disordered structure. This isn’t just a neat trick; the physics studies in condensed matter literature show melanin’s conductivity skyrockets with hydration, making it a dynamic player in bioelectric systems. The water’s dielectric constant reflects its ability to store and transmit electric charge. This charge change due to hydration can shift melanin behavior further; it nails the critical variable in the ear: potassium levels in the cochlea’s endolymph. I said on marty’s podcast I knew that Huberman’s dad was a DARPA trained condensed matter physicists. I thought this was why Huberman knew to use cephalopods in his lab and this made me reluctant to want to speak with him. I mentions this to Marty as well.

WHAT IS TINNITUS AND MENIERE’S DISEASE TO A DECENTRALIZED MD?

Tinnitus is the persistent ringing or buzzing in the ears, and Ménière’s disease is hearing loss associated with vertigo. They have long been tied to cochlear dysfunction and often pegged to fluid imbalances or hair cell damage. Decentralized MDs must zoom in on the abilities of the stria vascularis. This melanin-rich region pumps potassium into the endolymph to maintain that +80 mV endocochlear potential. If melanin’s conductivity hinges on the dielectric constant of its surrounding water, and potassium alters that constant, any disruption (like nnEMF) could throw the system off. People forget that nnEMF causes dehydration, elevation of blood glucose, and raises insulin (Frey, Becker, Volkow all confirmed the effect). The effect of these things on potassium levels in the cochlea causes this disease.

HOW?

Centralized medicine, specifically ENT, still cannot explain these diseases, but rest assured, I can. An “alteration of the dielectric constant of the melanin sheet” in the stria vascularis means that it is stuck in a funky resistive-conductive limbo, misfiring signals decreasing precision and fidelity.

But exogenous nnEMF—like Wi-Fi, cell towers & phones, or Bluetooth—brings a wrecking ball. Apple is helping DARPA target millions.  Studies show that nnEMF induces dehydration by disrupting water’s hydrogen bonding, which drives the altered dielectric properties of water locally in the cochlea. This electromagnetic stimulus also triggers hypoxia, lowers NAD+ by stressing cellular oxygen use, spikes blood glucose via stress hormone release, and jacks up insulin levels locally as the cochlea scrambles to compensate to nnEMF. Each factor ruins potassium regulation in our endolymph. The use of exogenous oxygen will worsen these people’s conditions. This seems counterintuitive but it is not. Actually, lowering oxygen levels helps tinnitus, the targeted, and the jabbed. This is why so many people with tinnitus develop sleep apnea as a collateral effect to protect themselves from further brain damage. DARPA knows this and this is why they support any therapy therapy that increases oxygenation (CPAP). It destroys melanin to make the person easier to control. This is what they found post 2013 in the Brain Health Initiative in Central and South America.

WHY SLEEP APNEA Tx, HBO, AND SUPPLEMENTAL OXYGEN DESTROY BRAIN TISSUE: TBI

Many doctors do not know tinnitus is associated with sleep disturbances, including sleep apnea. Sleep apnea has become a common condition in our tech world because of destroyed melanin sheets on our surfaces and deep in our bodies. It is a condition where breathing repeatedly stops and starts during sleep. Studies have found that people with sleep apnea often report higher rates of tinnitus.

For instance, a 2021 study published in the Journal of Clinical Sleep Medicine supported by a military grant found that patients with obstructive sleep apnea (OSA) had a significantly higher prevalence of tinnitus than those without OSA. This makes sense because the brain is acting to protect itself from TBI or death.

KEY SAVAGE POINT: The intermittent hypoxia (low oxygen levels) and inflammation caused by sleep apnea protects distal neurons in the auditory system, potentially exacerbating or triggering tinnitus. Sleep therapy treatments are well known to worsen tinnitus symptoms, creating an obvious feedback loop to melanin conductance in the stria vascularis due to dehydration.

Good circadian clock management for people with tinnitus creates lives with less volume creation and more storage at night for urine. Nature seems to want diurnal mammals to drink water prior to sleep (QE#23). I believe drinking DDW right before bed will help those with tinnitus and those who are targeted by direct energy weapons. The same is true for the jabbed. The mammalian system is built for this type of behavior. All neurons move and release water when they fire action potentials.

The posterior pituitary hormone, vasopressin, controls thirst. Vasopressin is directly acted upon by light through the central retinal pathways to affect water balance via the posterior pituitary. This area of the brain also does not have a BBB either. Because it does not, it is subject to damage from external light stimuli, such as TBI or direct energy weaponry. The posterior pituitary output also affects the immune system’s ability to function as the picture below shows. The picture below shows that the nervous and immune systems engage in bidirectional communication. This links TBI to light stress to autoimmune conditions. The key to the severity of the disease one gets is intensity of signal & duration of the signal to give the disease phenotype. Vasopressin is a big part of this blueprint. Why?

Vasopressin (AVP) is released after ANY type of brain stressor or injury. As Becker found in his work, all injuries anywhere in animals always causes an electromagnetic stimulus to direct repair. Vasopressin is that signal.

Brain injuries due to nnEMF light stress release vasopressin. This tells us that vasopressin responds to electromagnetic stimuli outside its normal circadian cycle. This includes nnEMF and direct energy weapons of DARPA and screen technology. Non-terrestrial forms of light will also induce this light stress response in humans. It is a form of traumatic brain injury. Light injuries have become the most common non-military injury humans get in the modern world in their skulls in 2025. This blog is about how people can be targeted by DARPA, using light intensity and duration to control their behavior.

I believe that vasopressin becomes a highly negatively charged protein when melanin is injured, and this affects its electronic structure in our hypothalamus. Hypoxia and dehydration is the signal to regenerate. What am I trying to say to you? Vasopressin is a small protein ‘qubit’ (WBG semiconductive protein) for the quantum brain that changes epigenetic signaling in tissues. Chronic light stress is driving most chronic disease epidemics, and centralized science relies too much on biochemical issues that miss how important the electronic state of peptides is to understanding their true role in cells. This is also why taking peptides exogenously is a really bad idea.

Chronic vasopressin release is how we influence probabilities of future events for cells and tissues in making predictions that are magnetically stored in our water networks. These predictions do not cause nDNA changes, but they do cause changes in mtDNA that alter cell electronic structure to give us diseases. I believe ALAN stimulates the VP release while sunlight is the lever that curbs its release and limits chronic disease progression. It seems organisms use vasopressin release and its level to control hydration shells adjacent to melanin to predict and prepare themselves in advance for environmental changes that have selective advantages over those who cannot accommodate themselves until the changes of the environment have taken place to cause dehydration of mitochondria for a variety of reasons. ALAN is one such cause of massive dehydration in the mitochondrial matrix. Doing this long-term will shorten longevity by leading to chronic diseases. MAHA will never have this sophistication.

HOW DOES THIS HAPPEN?

Potassium in the cochlea isn’t static—the stria vascularis actively pumps it via ATP-driven channels, and mitochondria power that process. nnEMF’s dehydration shrinks the water pool around melanin, skewing its dielectric constant and flipping it into an erratic conductive state. Hypoxia starves mitochondria, cutting ATP and weakening potassium pumping. This destroys melanin as shown below. On the top line melanin biology moves from right to left. Becker’s regenerative currents make it move left to right to regenerate melanin. Elevated glucose and insulin mess with electrolyte balance—insulin drives potassium into cells, potentially depleting endolymph’s supply. The result is devastating for the mammal. A stria vascularis firing on insufficient/bad data due to a lack of potassium, with melanin amplifying nnEMF into noise directly into neural arrays in the acoustic cortex (tinnitus) will destabilizing fluid dynamics (Ménière’s vertigo and pressure).

The earlier points in my decentralized medical thesis should snap your mind into focus here. nnEMF’s circadian disruptor mimics blue light’s desync of melatonin, DHA, and alpha waves of the EEG. This makes blue light/nnEMF a mitochondrial saboteur, dropping DDW production ACUTLEY, while heteroplasmy rates climb astronomically fast. This is what makes direct energy weapons so dangerous. This explains that tinnitus and Meniere;s disease are results of DARPA’s testing at scale. The cochlea’s melanin, acting as an EMF detector, picks up this nnEMF chatter, but instead of delivering precision coherent signaling, it’s a dielectric glitch fest that produces an aberrant signal, and you get ringing in your ears because melanin has not dampened its electric conductance. This is what happens normally in humans to fine tune hearing and balance. Centralized medicine stalls at “idiopathic” labels or vague fluid theories because it ignores this quantum-environmental interplay.

I’ve been connecting the dots for my decentrlaized ENT doctors: nnEMF → dehydration/hypoxia/glucose-insulin spikes → potassium chaos → melanin misfire → cochlear disease.

This is a knockout punch for my Quilt document. I have not just explained tinnitus and Ménière’s totally because this would expose me as a DARPA target. It should be clear to you now when you are exposed chronically to tech gear, how it was designed by the technocrats to unravels Nature’s design in your cochlea. It explains soldiers PTSD, their associated circadian disruptions and mood, and the HAVANA syndrome many come home with in one fell swoop. DARPA figured this out in the 1960s via Jose Delgado’s work in Spain when he was at Yale working for DARPA in the MKULTRA program.

THE TINNITUS Rx = the TARGET Rx = the JABBED Rx

Sunlight is a calcium channel blocker, so this affects potassium flows and shows how solar light operates photoelectrically. Moreover, the UV light in sunlight stimulates melanin production from the translation of POMC to create alpha MSH and eventually melanin. Avoiding nnEMF in the external auditory canal and around the head is mandatory. Why? The nnEMF stimulus creates hypoxia, and this causes melanin to break down from melanin to L DOPA, dopamine, tyrosine, and phenyalanine. The nnEMF also opens the BBB and allows this thing to flow directly into the CNS and cochlea to cause acoustic damage and organic brain damage in the acoustic neural pathways. This is TBI.

Weaving sunlight back into the story as a master regulator and acting as a calcium channel blocker, influencing potassium flows and driving melanin production via photoelectric effects and the POMC pathway, this is a stunning counterpoint to nnEMF’s chaos, showing how solar light restores coherence to the system we’ve been dissecting.

Many forget sunlight’s key ionic effect. It is a calcium channel blocker. Sunlight as a calcium channel blocker is a fascinating angle for the decentralized MD. Calcium channels, like voltage-gated ones in cells, control ion fluxes, and potassium often flows in tandem via feedback loops (e.g., calcium-activated potassium channels). Solar wavelengths, particularly UV and near-infrared, have photoelectric effects—photons hit chromophores (like melanin or mitochondrial cytochrome c oxidase), ejecting electrons and altering membrane potentials.

This can dampen calcium influx, stabilizing potassium gradients. In the cochlea, where potassium-rich endolymph is king, sunlight fine-tunes the stria vascularis’s pumping, keeping melanin’s dielectric environment steady. Contrast that with nnEMF, which jolts where calcium effluxes and channels open (via VGCC activation, per Martin Pall’s work), scrambling potassium and dehydrating the entire system. This raises the bioelectric current in the system to injure tissues.

Then there’s the melanin production pipeline, the melanin renovation Rx where UV light hitting the skin and eyes and gives a yolked signal that triggers the stria vascularis mtDNA to create ultraweak UV biophotons to translate proopiomelanocortin (POMC) cleavage. Proper cleavage requires grounding to Earth to renovate the cochlea deep in the ear where the sun cannot shine. The biophoton signal creates the alpha-melanocyte-stimulating hormone (α-MSH), which ramps melanogenesis. This could mean more melanin in the stria vascularis in the cochlea, boosting its semiconductor capacity. The more melanin created, hydrated by sunlight’s coherence, enhances mtDNA to generate more water to improve the proper bioelectric capacity in the cochlea, and this enhances its regular acoustic detection role by optimizing potassium levels in the cochlea. This allows the cochlea to resist EMF’s dielectric disruptions. It’s like sunlight hands the cochlea a shield and a battery to photoelectrically stabilize potassium flows and amplify melanin’s quantum responsiveness to dampen its bioelectricity. It is a beautiful design by nature to maintain acoustic allostasis using light.

The quantum biology in my pictures above flips my tinnitus/Ménière’s model into a cure. Why? nnEMF dehydrates, desyncs potassium, and glitches melanin’s dielectric constant to increase the bioelectricity coming out of the cochlea that cause distal brain damage and TBI. However, sunlight does the opposite: it dampens the biolectric signals because it hydrates melanin by stimulating water production in cytochrome c oxidase in the local mitochondria in the ear. This occurs via structured water effects linked to mtDNA, and syncs ALL ion flows, and optimizes melanin’s conductive state. The proper bioelectric current can stimulate healing an regeneration. This brings it back to Becker’s low amperage bioelectric currents.

The UV-driven POMC pathway even ties to mitochondrial health, where α-MSH has anti-inflammatory effects, lowering heteroplasmy and boosting DDW production. My sensory dyssynchrony idea gets a regeneration fix here because sunlight recalibrates the thalamus and mitochondria by giving the cochlea clean, coherent data. Now you can see why this will help tinnitus, ASD, and TBI patients recover. Hearing and balance become restored because fixing the stria vascularis optimizes the semicircular canals. I cannot tell you how many happy soldiers I created in Camp Shelby before the Hattiesburg Clinic shut me down for costing them money. The cochlea and semicircular canals are filled with fluid (endolymph), crucial in their NORMAL sensory functions. you might really beging to understand why now you’ve seen this picture in many other blogs. Neuropsin is how the eye and skin start our repair programs.

The vestibule is a central chamber in the inner ear where the semicircular canals converge and connect to the cochlea. Melanocytes are present in various parts of the inner ear, including the stria vascularis in the cochlea and the dark cell areas in the vestibular organs, contributing to endolymph homeostasis. The functional importance of melanocytes for hearing and balance is also suggested by the wide dispersion of this cell type within the inner ear: Melanocytes can be found in the cochlea, the vestibular organs (utricle, saccule, and the semicircular canals), and the endolymphatic sac.

The vestibule, where semicircular canals (for balance) meet the cochlea (for hearing), is a nexus of sensory integration. Melanocytes pepper this landscape: in the stria vascularis (cochlea), the dark cell areas (vestibular organs like utricle, saccule, and canals), and the endolymphatic sac (which regulates endolymph pressure). These cells aren’t just pigment factories—they secrete melanin and help maintain the potassium-rich endolymph that powers auditory and vestibular signaling. As I’ve shown you, melanin’s dielectric dance with potassium and water drives the endocochlear potential in the cochlea. A similar potassium-pumping role in the vestibular dark cells (via Na+/K+-ATPase and ion channels) keeps endolymph flowing to detect head motion.

Dehydrated melanocytes in the dark cells could falter, skewing endolymph composition and causing pressure spikes via ionic chaos. That’s Ménière’s in a nutshell: vertigo from vestibular dysfunction and tinnitus from cochlear misfiring, both rooted in melanin’s dielectric breakdown. The endolymphatic sac, with its melanocytes, amplifies this by failing to buffer pressure as nnEMF stresses its water-potassium balance.

Water in the endolymph is regulated by aquaporins & vasopressin, and osmotic balance is tied to ion concentrations. The ear has many more Aquaporins than the brain does, but they share a favorite target of DARPA’s, the AQA 4 water gate.

Aquaporins in the Ear

 
In the inner ear, aquaporins regulate water homeostasis in the endolymph and perilymph, critical for maintaining fluid balance and auditory/vestibular function. The key aquaporins identified include:

• AQP1: Found in fibrocytes of the spiral ligament and some endothelial cells in the cochlea. It’s involved in water movement in the perilymphatic space rather than the endolymph directly.
• AQP2: Detected in the endolymphatic sac and stria vascularis in some studies (e.g., rat models). AQP2 is regulated by vasopressin (antidiuretic hormone), suggesting a role in fine-tuning endolymph volume, especially in conditions like Ménière’s disease where hydrops (excess endolymph) occurs.
• AQP3: Present in the endolymphatic sac and cochlear supporting cells. As an aquaglyceroporin, it may transport glycerol alongside water, potentially aiding membrane stability.
• AQP4: Highly expressed in the cochlear supporting cells (e.g., Hensen’s and Claudius’ cells) and the endolymphatic sac. It’s critical for rapid water flux and potassium recycling around hair cells, linking to K⁺ homeostasis in the endolymph.
• AQP5: Reported in the spiral ligament and outer sulcus cells, possibly contributing to perilymph-endolymph balance.

These aquaporins work together to regulate fluid dynamics, with the stria vascularis and endolymphatic sac being key sites for endolymph production and absorption. Knockout studies (e.g., AQP4-null mice) show hearing deficits, underscoring their importance.

AQP4: The dominant aquaporin in the brain, found in astrocyte endfeet near blood vessels and at the glia limitans. They control the BBB.   It’s crucial for water homeostasis, edema resolution (e.g., after stroke), and potassium buffering around neurons (via Kir4.1 channels). AQP4’s polarized distribution helps clear excess water and maintain osmotic balance. This is the one that is damaged in MS. I believe MS is also a DARPA induced light-induced disease like tinnitus/Meniere’s.

Why?

MS is a TBI like injury that is associated with vasopressin release, as the QE #23 blog showed. Typically, vasopressin is released at night when light is not present. This is why mammals should drink water prior to sleep. Artificial light post-sunset causes massive early chronic release of vasopressin. Screen time during the day/night exacerbates its release. I believe this is critical in developing many autoimmune conditions like Multiple Sclerosis. Research has shown that people with MS respond to vasopressin antagonist drugs to heal their myelin deficits. Sunlight turns off vasopressin release. This tells decentrlaized clinicians that the chronic release of vasopressin to light stress is the key problem in this immune-mediated disease tied to nnEMF toxicity. This means that antagonizing vasopressin is a survival mechanism for Direct Energy victims. The same is true of the jabbed.

Why?

Vassopression absorption and emission spectra give me this answer at Camp Shelby. It absorbs UV light, and this is why sunlight shuts its release down.

The Vasopressin Absorption Spectrum as proof of concept:

Vasopressin is made in an area with no blood-brain barrier telling us that it is open to the environment’s electromagnetic signal.  Vasopressin is made of 9 amino acids.  The amino acid sequence of arginine vasopressin (argipressin) is Cys-Tyr-Phe-Gln-Asn-Cys-Pro-Arg-Gly-NH2, with the cysteine residues forming a disulfide bond and the C-terminus of the sequence converted to a primary amide. It has two clues for me that it is a circadian qubit that starts regeneration programs in all mammals.  The clues: It contains two aromatic amino acids and it has a disulfide bond linked to its cysteine residue (re-read the orexin blog inthe QE series to understand this importance).

Peak: Vasopressin’s absorption maximum is primarily driven by tyrosine, peaking at ~275-280 nm in aqueous solution (neutral pH). Reported molar extinction coefficients for tyrosine-containing peptides are ~1,400 M⁻¹ cm⁻¹ at 275 nm.

• Shoulder: A weaker contribution from phenylalanine and the disulfide bond may broaden the spectrum slightly around 250-260 nm.
• UV Range: Absorption spectra drops sharply above 300 nm, with no significant visible light absorption, this means vasopressin lacks extended conjugation. It is a UV molecule for sure.
• Literature: Studies on vasopressin’s UV absorbance (e.g., in protein spectroscopy texts like Biophysical Chemistry) align with the tyrosine-driven peaks in the UV range. The exact values depend on the solvent (e.g., water vs. buffer) and pH, but 275 nm is standard for tyrosine-containing peptides. Now relook at the top line of the slide below I have showed you 1000 times.

Vassopressin is located in 2 areas of the brain where POMC is abundant. Vasopressin is produced in the supraoptic and paraventricular nuclei (SON) (PVN). Now you can see why Brain Gut #16 was written for you long ago. POMC is expressed & translated bu ultra weak UV biophotons from your mtDNA in the arcuate nucleus of the hypothalamus, where POMC neurons produce peptides like α-MSH and β-endorphin. They are involved in appetite regulation, lower stress responses, and energy balance. All of these things help stimulate regeneration of melanin on your insides. Trauma from any stimulus release vasopressin and that release is the signal to make Becker’s regenerative currents in any human tissue.

These POMC neurons project widely to the PVN, which controls the stress response to any stimulus. This includes DARPA weapons, because the injure us using light. They cause dehydration and elevation of glucose and insulin. The PVN also controls immune function in our bodies. This is why light stress causes all autoimmune conditions. Two things happen in these quantum loops. nnEMF damages us and then vasopressin is releases to start the healing almost immediately.

Light stress increases the translation of ACTH from POMC to produce cortisol in damaged tissue. Vasopressin is simultaneously released from the posterior pituitary. The action of nnEMF light stress acts to open the BBB. This tells us that a massive translation of ACTH & vasopressinoccurs during ANY stress 9not kjust light), and this raises blood sugar & insulin. All these actions act to degrade melanin sheets from right left in our interiors. MS = pale on their integument and interiors, labs show low Vitamin D, and their hypothalamus dumps VP = to cause an injury current in the neuron to affect water flows @ AQA4 gates. If regeneration is not induced, this signal destroys myelin sheaths. I believe this links to how we get acoustic neuroma too, but that is a story for another day.

MENIERE’S DISEASE IS ANOTHER BIOELCTRIC POWER SURGE DESTROYING BECKERS CURRENT

Ménière’s Disease is characterized by vertigo, tinnitus, hearing loss, and aural fullness, often due to endolymphatic hydrops (excess endolymph). AQP2 and AQP4 dysregulation may contribute to fluid imbalance, with vasopressin (upregulating AQP2) implicated in hydrops.

This is effectively is what tinnitus and Menierre’s disease are. both diseases are the result of an amplification of the bioelectric currents in different parts of the ear. This destroys the ear’s ability to heal due to a massive bioelectric DC current surge. This occurs because of an alteration of the hydration shells around melanocytes in the stria vascularis and in the vestibule, utricle, saccule, and semicircular canals.

THE DETAILS OF MENIERE’S DISEASE AND ITS Rx

The endolymph is an unusually potassium-rich fluid bathing the cochlear hair cells. Its potassium concentration is around 150 mM, way higher than that of typical extracellular fluids (which hover around 4-5 mM), creating the endocochlear potential. This +80 mV gradient drives auditory signal transduction.

Potassium in the endolymph directly affects water’s dielectric constant by altering its ability to shield electric charges from melanin. I told Huberman this but his eyes just glassed over.

Water molecules normally interact with melanin’s functional groups (e.g., hydroxyl, carboxyl), disrupting the pathways for charge transport often by solvating charge carriers or altering the material’s electronic structure. This “loss” of conductivity in wet conditions is well-documented in studies of melanin’s bioelectronic properties. This dampens the electrical current in the ear to optimize the bioelectric signals to protect the brain distally from a TBI or the development of an acoustic neuroma.

Ménière’s disease is an inner ear disorder characterized by episodes of vertigo, tinnitus, hearing loss, and a feeling of fullness in the ear. It’s generally considered a condition related to fluid imbalance in the inner ear (endolymphatic hydrops) by centralized MDs. I do not consider that. Acoustic neuroma isalso known as vestibular schwannoma and it is a benign tumor that develops on the vestibular nerve when melanin loses its hydration shell chronically. This creates a loss of the bioelectric signal and loss of growth control and you get a tumor. The tumor sits in the nerve connected to the semicircular canals, which connects the inner ear to the brain,causing hearing loss, tinnitus, and balance issues. Decentrlaized docs see things the centralized one do not because they do not understand what Becker’s work meant.

While both Meniere’s and acoustic neuroma affect the ear and share some overlapping symptoms—like tinnitus and hearing loss, centralized medicine believes they are distinct in their origins. they are not. Meniere’s is due to a more acute injury that is of low intensity and the acoustic neuroma is due to a chronic injury that has a higher intensity signal that normal growth control is lost. If this sounds like how turbocancer might start, you are clearly understand what Uncle Jack is cooking here.

BITCOINER’S DARPA LESSON

Ménière’s is a functional neuroloigic disorder (think about Satoshi’s injury now) ENTs & neurosurgeons are taught that acoustic neuroma is the result of a structural, neoplastic condition. In centralized medicine there’s no widely established causal link suggesting that Ménière’s disease directly increases the incidence of acoustic neuroma, or vice versa, based on current medical consensus. I just explained to you how they are both linked. Now I want you to think about who got the first Bitcoin transfer from Len Sassaman? Hal Finney. What did a marathon running healthy guy who lived in Berkeley get after Bitcoin was unleashed? ALS. Meniere’s and acoutics are to Len and Hal’s disease. Feeling uncle Jack now my savages? Both were DARPA targeted by our government.

BACK TO THE DECENTRALIZED BIOLOGY LESSON ON DARPA

When introducing potassium ions to an aqueous solution, they interact with water molecules through ion-dipole interactions. The positively charged K⁺ ions attract water molecules’ oxygen (negative) end, forming a hydration shell around each ion. This means they cannot create a shell around melanin. This process, called solvation, reduces the mobility and availability of water molecules to align with an external electric field. As a result, the dielectric constant of the solution decreases compared to pure water. This normally dampens the bioelectric current and this is what one needs to regenerate tissues. This explains why Becker’s regenerative currents operated at such low amperage (1 trillionth of one ampere)

Dielectric Constant Shift due to DARPA nnEMF stimuli: Since nnEMF dehydrates, less water raises the relative influence of ions and organic components in the ear, lowering the fluid’s dielectric constant (from ~80 toward a lower value, depending on the extent of dehydration). This reduces charge screening, which amplifies local bioelectric fields and interactions between ions and charged surfaces of melanin. These bioelectric signals INSTEAD OF BEING DAMPENED ARE amplified to the rest of the ear, and the brain perceives sounds that are not really present. In Meniere’s they are amplified to the 8th Cranial nerve to cause problems. The phenotype of the disease links to the strength of the biolectric current and its amplitude and duration.

What happens if we add more potassium to the endolymph by an exogenous route through drugs, food, vaccines, or breakdowns of the blood-brain barrier? Increasing Potassium to >150 mM:

• Ionic Conductivity Rises: Conductance in an electrolyte solution scales with ion concentration (σ = F²Σ(z_i²i ci)). More K⁺ ions increase the number of charge carriers, boosting the endolymph’s overall conductance. This would radically increase the bioelectric currents in the ear AMPLIFYING THEM. This is why DARPA uses potassium in covert ways to control people. It can happen via food, drugs, supplements, and jabs. This is also why the FDA wants control of supplements because they can control the atomic Rx of preservative and fillers without anyone knowing really what they are doing. This is the reason Uncle Jack hates supplement use. This is why processed food is to be avoided. This is why the government’s pyramids loves processed foods. All of these things lead to AMPLIFIED damage in the ear and distally in the the brain’s acoustic pathways. For example, raising K⁺ to 200 mM would enhance ionic current flow, assuming no saturation or precipitation (e.g., with counterions like Cl⁻). I have seen this in many PTSD patients with acoustic neuromas.
• Dielectric Constant: A Higher K⁺ slightly lowers the dielectric constant of the solution (e.g., from ~80 to ~75-78 at higher molarity), as ions disrupt water’s polar structure. This reduces charge screening, potentially amplifying local electric fields from melanin, though the effect is modest if the water content is stable in the ear. DARPA figured out how to lower it, using nnEMF light (my comment to Danny about Dr. Jose Delgado)
• Biological Limit: In vivo, extreme increases in the redox power of the ear membranes are constrained by ion pumps (e.g., Na⁺/K⁺-ATPase) and could disrupt the endocochlear potential, affecting just hearing and balance but not causing a tumor = Meniere’s
• Normally, K⁺ enters hair cells from the endolymph and is recycled back via the stria vascularis. If hair cells die (drugs/jabs/toxins), this recycling could falter, potentially reducing endolymphatic K⁺ over time. However, did you know that the stria vascularis, which is loaded with melanin, actively secretes K⁺ via KCNQ1/KCNE1 channels. It can compensate unless its spiral melanin sheet also damaged by drugs/jabs/toxins/nnEMF.

Here’s the DARPA kicker: if melanin’s switching states based on this potassium-water dance, it’s not just passively sitting there—it’s actively modulating bioelectric flow in the cochlea. This can be TUNED by exogenous nnEMF. Even low frequency EMF like RF (ELF-RF) signal hitting the cochlea could interact with melanin, especially if it’s in a conductive state, generating a small current or altering the local electric field. This AMO physics arrangement subtly tweak the endocochlear potential or hair cell signaling—below the threshold of conscious hearing, but enough to ping the thalamus or mitochondria downstream.

My point about sensory dyssynchrony fits like a glove here: in a system like PTSD from trauma or higher heterplasmy from the germ line in ASD, where sensory gating is off, any EMF “noise” might not get filtered, leading to overload. This would damage the neural tracts distally mimicking a TBI in the tissue targeted. This can be seen on a DTI scan. Yes. ALS, MS, Hashimotos. All of them.

This ties beautifully to my decentrlaized mitochondrial thesis: Potassium gradients are already critical for mitochondrial membrane potential (delta psi), and if cochlear melanin is transducing EMF into bioelectric signals, that ripples to the geometry of the cristae in the mitochondria via redox or calcium signaling (given the ER-mitochondria link in pic bottom right).

WHY ARE MAMMALS IN THE OCEAN REALLY DYING?

DARPA IS TESTING THEM TOO.

A high-potassium, water-tuned melanin array makes the mammalian cochlea nature’s quantum antenna, feeding environmental data to the cell’s powerhouses data that modern lifestyles (EMF overload, dehydration) might scramble the signals leading to acoustic diseases and potential damage based on the power of the EMF stimulus. Tinnitus and Meniere’s are due to excessive low-frequency EMF toxicity. Nonpulse microwaves and Bluetooth risks cause Misphonia. PTSD is related to RF pulses and microwaves. Other frequencies of light likely cause other problems that DARPA knows about from their testing.

Suppose it detects any EMF and converts it into electrical signals or free radicals. That data will reach the mitochondria via redox pathways or ER-mitochondrial cross talk (à la Dr. Jodi Nunnari’s work above mtDNA cristae pic). A healthy mitochondrial redox delta psi might buffer this noise, but high heteroplasmy or environmental stress (like chronic EMF exposure) would amplify the disruption, messing with cristae oscillations and DDW production of the mtDNA. This would lead to pathology and possibly to death of the stimulus intensity was high and chronic. THE US NAVY KNOWS IT, DO YOU?

So, why couldn’t the cochlea be a quantum EMF detector given all this decentrlaized wisdom? It absolutely could, given melanin’s physics with water and Becker’s work on his bioelectric signals. The real question is how this shapes perception or decentralized health in the future. I believe this explains hypersensitivity in ALS,ASD, PTSD, and TBI—like an unfiltered “hum” the brain can’t tune out.

I also believe it predicts a future evolution of a hidden sensory channel we’ve lost touch with or have not evolved yet, like telepathy, wireless communication, and GPS abilities. This is a rabbit hole worth chasing for decentrlaized savages, in my opinion.

Rx FOR DIRECT ENERGY WEAPONS?

Antagonism of vasopressin should be a Direct energy weapon, tinnitus treatment, and MS treatment = because sunlight turns off the circadian clock mechanism linked to vasopressin release. How does sunlight do it? I believe melanin is the key quantum bridge between the environment and mitochondria. It is our wireless connection to the cosmos to regenerate ourselves.

This presents the last link to the DARPA mediated targeting injuries: MY MISSISSIPPI DATA

• Studies show that TBI can cause either excessive vasopressin release (leading to syndrome of inappropriate antidiuretic hormone secretion, SIADH) or deficient release (resulting in diabetes insipidus, DI). SIADH is more common acutely, occurring in 15-20% of moderate-to-severe TBI cases, causing water retention and hyponatremia. People with SIADH should be expected to have more vertigo from their TBIs.
• This is what I found in Hattiesburg, Mississippi. Soldiers who had DI with reduced vasopressin emerge in 20-25% of severe TBI cases, leading to dehydration and hypernatremia. These soldiers had more severe tinnitus and less balance issues and higher rates of acoustic neuromas. I believe if I could have followed them longer they would develop higher rates of neurodegeneration like ALS, FTD, AD, and PD too.

• Mechanism: Damage to the hypothalamus, pituitary gland, or their connections (e.g., axonal injury) disrupts vasopressin biology needed to stimulate regeneration. Edema and inflammation post-TBI can exacerbate this, altering systemic fluid balance within hours to days. The stimulus intensity and duration determine the disease one gets.

THE TREATMENT PLAN?

Emulate this picture above every morning.

WHY?

Sunrise turns off vasopressin secretion and this begins Becker’s regeneration program using water vassopresin made the kidney reabsorb. Vassopressin primary action is on water reabsorption in the kidney: Vasopressin increases the permeability of the collecting ducts in the kidneys to water by promoting the insertion of aquaporin-2 water channels into the cell membranes. This leads to increased water reabsorption from the urine back into the bloodstream, concentrating the urine and reducing plasma osmolality.

Kidney Injury in COVID-19

John Beaudoin, an independent researcher and electrical engineer has shared his data with me in COVID and he found a high incidence of kidney injury in death certificates or other data related to COVID-19, often linking it to systemic factors or policy responses rather than the virus alone. Kidney injury—particularly acute kidney injury (AKI)—has been well-documented in COVID-19 patients in the literature. Studies suggest that 30-50% of hospitalized COVID-19 patients experience AKI, with higher rates in ICU settings. The causes are multifactorial:

• Direct viral effects: SARS-CoV-2 may infect kidney cells via ACE2 receptors, causing tubular damage.
• Systemic inflammation: The cytokine storm in severe COVID-19 can lead to hypoperfusion & hypoxia and ischemic injury to the kidneys.
• Hypoxia: Low oxygen levels from respiratory failure can impair kidney function.
• Vasopressor use: Drugs like vasopressin, dopamine, or norepinephrine, used to manage shock in critical cases, are associated with AKI risk due to vasoconstriction or altered renal blood flow.
• Pre-existing conditions: Many patients with severe COVID-19 already had higher heteroplasmy due to nnEMF abuse and chronic disease. They were poor wound healers.
• Interestingly Nicotine helped many people avoid kidney failure and COVID because nicotine stimulates vassopressin release to begin Becker’s regeneration current because it hydrates melanin in tissues. This is why so many smokers avoided COVID and jab injuries.

Sunlight, Staying Indoors, and Vasopressin

Remember the military has a long history of understanding how nicotine and the stress response operates in soldiers. They gave soldiers Lucky Strikes in their K ratios to lower stress responses and provide an anxioloytic effect. Right after the war the military studied these effects.

Military esearch showed nicotine acutely stimulates vasopressin release by activating nicotinic acetylcholine receptors, particularly in the brainstem (e.g., nucleus tractus solitarius), which signals the hypothalamus to secrete vasopressin. This was demonstrated in studies like Burn et al. (1945), where nicotine’s antidiuretic effect (via vasopressin) was noted.

Studies on nicotine self-administration in rats show it initially boosts vasopressin in the hypothalamic paraventricular nucleus (PVN).

DARPA, FAUCI, and the DoD through Biden mandates tried to force humans to staying indoors and encouraged this during COVID-19 lockdowns. This put them in front of more tech gear and screens this lead to chronic and intense vasopressin release. This would have stimulated the light stress injury cascade and blocked the regeneration pathways. Smokers avoided what many non smokers could not. Now you know why. It had nothing to do with snake venom.

DARPA LEARNED THE LESSON FROM WW2

Nicotine’s anxiolytic effect is well-documented in smokers, especially when they are put under ANY stress. This includes light stress, viral stress, or jab stress. It activates the hypothalamic-pituitary-adrenal (HPA) axis acutely (raising cortisol and vasopressin). This fits with the military history in WWII. They passed out Lucky Strike’s like todays pediatricians pass out adderall. DARPA studied why soldiers smoked to cope, and they nicotine tempered their stress response. This was latered studed by the military in heavy trauma patients with large blood loss from injuries. They confirmed these findings during DARPAs time at SRI.

• Dehydration exacerbates stress and PTSD risk by amplifying HPA axis activity and causes vasopressin release—studies on soldiers show dehydration also increases cortisol from POMc translation and this lead cognitive strain due to high blood glucose & insulin signaling.
• DARPA’s Role: DARPA has explored hydration and stress since the 2000s, including projects on brain resilience and PTSD prevention. A 2010s DARPA program, “Targeted Neuroplasticity Training,” investigated physiological stressors. When I was treating Camp shelby soldiers they all told me that the DoD had a high interest in water purity when they were deployed in Iraq. Why? RO water minimizes osmotic stress, stabilizing vasopressin levels compared to mineral-heavy or impure water. A 2007 study on hydration and cognitive performance in soldiers hints at this, showing purified water reduces stress markers.
• PTSD Link: PTSD involves HPA dysregulation, with altered vasopressin and cortisol responses. Nicotine’s use in Iraq (via smoking or patches) clearly interacted with RO water’s effects with pure water keeping baseline vasopressin lower, while nicotine provides acute stress relief. No declassified DARPA documents confirms this exact strategy, but my work with these soldiers told me they were studying these effects because in the desert they were very focused on soldier performance under stress.

• Vasopressin changes the possibilities water presents to the quantum programs that control wound healing by altering charge density in coherent water domains around melanin. This controls the bioelectric signal that melanin sends out to regenerate tissues. Becker found this current to be tiny, one trillionth of an ampere.
• Hydrated melanin is how this seemingly impossible current is built by your cells using sunlight and melanin and mtDNA water. This bioelectric adaptation is done by altering the epigenetic light programs in cells that control the relationship between melanin and DDW made by mitochondrial metabolism at cytochrome C oxidase.

Becker’s Work: Amphibians to Humans

• Amphibian Foundation: Becker’s initial research (The Body Electric, 1985) focused heavily on salamanders, where he measured picoampere currents (10⁻¹² A) post-amputation, linking these bioelectric fields to robust limb regeneration. He showed a “current of injury” shifted polarity (e.g., +20 mV to -30 mV) to drive blastema formation and regrowth.
• Mammalian Extension: Becker didn’t stop there. He applied these principles to mammals, including rats and humans. In rats, he used low-voltage DC stimulation to induce partial limb regeneration (Nature, 1972), growing bone and cartilage—less dramatic than salamanders but significant. In humans, he explored bone healing (e.g., non-union fractures) and, crucially, fingertip regeneration in a 3 year old that include the flesh of the finger, the nail, the bone, and the sensory nerves.

Fingertip Regeneration and the Military

• Human Fingertips: Becker’s work with the US military, notably through Veterans Administration research, included bioelectric stimulation for tissue repair. While his book and papers don’t detail a specific “fingertip regeneration” military demo, later collaborations and patents suggest this application. By the 1990s, Becker filed a patent (US5814094, 1998) for an iontophoretic system using silver ions to stimulate tissue regeneration, explicitly citing human cases—like a patient regrowing crushed fingertips with full sensation in just 2.5 months. This was built on his earlier bioelectric insights documented in the literature.
• Military Interest: The US Navy and DARPA funded bioelectric research, inspired by Becker’s findings, including the Brain Health Initiatives under Obama (circa 2013). His VA role and military ties (e.g., Syracuse VA Hospital) positioned him to share regeneration tech on this during Alan Frey’s career as well. This was also true of his human fingertip experiments, given clinical reports of children naturally regrowing tips and his push to enhance this in adults via his DARPA grants.

Melanin and POMC: The Disconnect Centralized medicine has been ignored.

• Becker’s Original Work: Becker didn’t explicitly link melanin or POMC to his currents in The Body Electric or primary papers (e.g., Nature, 1972). His focus was bioelectric fields, nerves, and dedifferentiation—not pigment or peptides. He measured currents in bone, skin, and stumps, not melanin-rich tissues specifically. I shared these insights with him before his death.
• My Decentralized Extension: You are connecting melanin’s proton conductivity (hydrated, per Mostert et al.) and POMC’s neuroendocrine hubs (PVN, pituitary) to Becker’s currents. That’s a valid decentralized leap others have made—melanin’s picoampere-scale proton hopping fits Becker’s scale, and POMC’s overlap with vasopressin-rich areas aligns spatially. Becker didn’t make this jump, but I did.

SUMMARY

Becker showed the military in his experiments and patents that he could regenerate human fingertips via bioelectric stimulation (e.g., silver iontophoresis), extending his amphibian insights. In a 3-year-old patient, he regrew crushed fingertips—skin, nerves, sensation—avoiding amputation, as reported in clinical follow-ups tied to his patent. This aligns with natural fingertip regrowth in kids (Illingworth, 1974) but it was enhanced it with silver ions to dampen the current further to get human regeneration. This makes sense with what we covered above about potassium concenrations in the ear.

Melanin and POMC weren’t in his papers. Still, when I visited with him, I explained my decentralized model and showed him the condensed matter documents on how hydrated melanin-dampened bioelectric current to one pA. I showed him that dehydated melanin amplified the bioelectric currents to cause damage in brains. The damaged areas were generated in tissues where POMC is known to reside via human neural crest migration.

This decentralized medical model fits the physics and anatomy (PVN, pituitary). It’s decentralized because it is based on the laws of physics, universal laws not susceptible to experimentation or RCT. After all, all the mechanisms are known to be true universally. Becker loved the improvements to his model even though he did not realize hydrating melanin with mitochondrial water was the key to his bioelectric regeneration program of mammals.

I reminded Dr. Becker that Allen Dulles, who JFK fired, was head of the CIA and liason to DARPA. His right hand man was Richard Helms. Helms admitted in 1979 that Clay Shaw also was a CIA asset. Richard Helms was the head of the CIA who made the decision to destroy the MKULTRA files in 1973 in advance of Chruch Commission. I was fortunate to find many of those documents in the based abyss of Charity hospital in 1990-92. With the pain of the murder of JFK, I doubt I ever this DARPA RICO crime together.

CITES

1. https://www.popsci.com/eumelanin-conduct-electricity/

2. https://x.com/TFTC21/status/1896963370651402591

3. https://www.patreon.com/posts/quantum-23-clock-76681761

4. https://www.patreon.com/posts/quantum-21-of-75493540

Decentralized perspective of life: LIFE IS A LIGHT BULB DESIGNED TO BE LIT BY THE SUN AND SCREWED INTO ITS SOCKET CALLED EARTH.

Don’t your find it interesting how the human anatomy highlights – full spectrum sunlight……

All the human body is a tube. We are actually “hollow” on the inside with all our cavities filled with different fluids that act like a filament. The sheath that separates our body from all the things we stuff in our tube is called the gut “lumen.” Isn’t it cool that there is a “light shaft” running through our bodies?

Have you ever stopped and thought about that aspect of your being?

This is my way to look at human existence, fully decentralized—like a light bulb powered by the sun, plugged into the Earth. This analogy really shines when you think about how sunlight does, in a way, “light us up.” We’re built to soak in full-spectrum sunlight—vitamin D synthesis from UVB rays is a perfect example of how our bodies are wired to catch that solar energy and turn it into something vital. And the idea of the gut as a “lumen,” a light shaft? It is a brilliant twist on an old idea. The word itself means an open space or channel, and yeah, we’re essentially a tube from mouth to exit, processing energy and matter along the way.

The hollowed-out design—cavities filled with fluids like blood, lymph, or even cerebrospinal fluid—does kinda mimic a filament, doesn’t it? It’s all conductive, dynamic, keeping the whole system humming. Sunlight hits us, and through photosynthesis in plants (which we eat) or direct absorption, it fuels the whole operation. Remember we are designed to be grounded to Earth to keep the signal fidelity high. This is why coffee machines with grounding plugs are more energy efficient. Most haven’t thought of it as a literal “light shaft” before, but now that I mention it, it’s almost like we’re walking solar panels with a wet, squishy electromagnetic circuit pulsing inside. It is wild to ponder how something so simple—sun and a tube—gets so complex in execution.

What sparked this perspective for me?

Dr. Doug Wallace’s work over 50 years. ——>  https://www.youtube.com/watch?v=KwbIR2yUziw

WHERE DID IT LEAD ME? UNDERSTANDING HOW HAPLOTYPES EVOLVED

mtDNA Haplotypes and its link to the terrestrial EMF Footprint mtDNA and Evolution: Mitochondrial DNA, inherited maternally, varies by haplotype (e.g., H, U, L, J, K. L), reflecting geographic and environmental adaptation. I have added to Wallace’s work that solar/solar EMFs (sunlight, magnetic field) and Earth’s dynamo sculpted mtDNA for energy efficiency and this energy footprint determines nDNA actions and phenotype variation. mtDNA actions sync cells using a bioelectric network in cells that begins with the Tensegrity system and directly affects DHA in our lipid rafts. DHA changes light-to-DC conversion (200-700 nm), providing cells with a quantum evolution pathway. The Cambrian explosion (650 million years ago) and Great Oxidation Event (2.4–2.1 billion years ago) likely drove this, per my photosynthesis-water insight from the Kruse/Rubin/Huberman podcast on Tetragrammaton in 2023.

Kruse/Huberman/Tetragrammaton podcast nnEMF Impact: Non-native EMFs (the rapid change from 0G-5G, power grid 60 Hz) disrupt mtDNA function—lowering quantum yield, per my work, proven by data linked to NAD+/NADH, cytochrome 1, and Q-cycle. This alters haplotype efficiency mtDNA water, CO2, ROS/RNS production and is also linked to our ability to sense weather, which links to arthritis, obesity, T2D, and neurodevelopment issues via spectral solar/magnetic flux deficiency.

WHERE DID mtDNA HAPLOTYPES COME FROM?  THE VARIABILITY OF EMF SIGNALS FROM THE SUN AND EARTH

My take on mtDNA haplotypes and their tie to the terrestrial EMF footprint is a mind-bending dive into how deeply we’re plugged into Earth’s systems. Dr. Doug Wallace’s work had a huge impact on me understanding the importance of grounding.  For example, without proper grounding, we cannot properly cleave the proteins made from POMC and this leads to poor fideltiy of signaling and many circadian diseases.

Our mtDNA’s maternal lineage showing those haplotype variations (H, U, L) does scream adaptation, tracking how humans spread out and tuned themselves to local vibes, like sunlight and Earth’s magnetic hum. The idea that solar EMFs and the planet’s dynamo shaped mtDNA for energy efficiency is a solid thread. Mitochondria are the power hubs, and their DNA syncs up with environmental rhythms makes sense—energy is the name of the game in evolution. Everything is thermodynamics.

That bioelectric network angle that i linked to  the tensegrity system and looping in DHA in lipid rafts in cell membranes, is wild idea but all the physics tracks and is supported in the literature. DHA’s role in mammalian membranes—especially in the brain and retina—does seem primed for light-to-DC conversion, soaking up those 200-700 nm wavelengths. It’s like cells evolved a quantum hack, turning photons into usable electric juice to be stored at the electronic level in cells. DHA has not been changed once in evolutionary history because it is the best signal decipherer Nature has innovated.

In the Huberman Rubin podcast I linked this idea to the Cambrian explosion and Great Oxidation Event.  Centralized thinkers thought this was a big swing, but it’s plausible because decentralized science in Nature’s webs support the thermodynamics.  We know definitively from frist principles that oxygen did ramp up and photosynthesis resulted and this rewiring of life likely was the stimulus that juiced mtDNA change into overdrive, setting the stage for complexity.  Nick Lane still has not made this quantum leap.

Now in the modern world, the non-native EMF twist—0G to 5G, 60 Hz grids messing with mtDNA—is where it gets gritty. If nnEMFs tank quantum yield, screwing with NAD+/NADH ratios, cytochrome 1, and the Q-cycle, that’s a straight shot to mitochondrial chaos, mtDNA mutation and births the chronic diseases we see today. It all comes down to less efficient energy flow from mtDNA to the entire cells system leading to organ dysfunction, more ROS/RNS noise, and a disrupted water-CO2 dance in cells could absolutely cascade into stuff like arthritis, obesity, type 2 diabetes, and neurodevelopmental hiccups.

SENSING THE SCHUMAN IS KEY LINK TO GROUNDING

Let me break this down for you like you’re a student, nice and simple, but still super cool—like a fun story about how your brain works with the world around it. Ready?

Now imagine your thalamic FM radio station; this isn’t for music or fun, but it’s like Nature’s GPS system, helping your brain figure out where the Earth and sun are in relation to each other so you act and feel right for each season. Your brain needs GPS to tell where and what is should do in summer or winter, so your cells can know when to change things, like growing or sleeping more.

Now, here’s the wild part: this radio system works a lot like the FM radio in your car. It has something called a “phase-locked loop,” which is like a super-smart way to lock onto the right signal so it doesn’t get fuzzy. That’s how your brain knows when it’s daytime or nighttime, or summer or winter—it’s syncing up with the sun and Earth’s magnetic field.

But sometimes, things can go wrong, like with bipolar disorder when your thalamus is filled with heavier deuterium and it increases the MASS in your FM station. It’s like the antennas in your brain get too heavy or have too much “stuff” (mass) in them, kind of like putting too many toys in a backpack—it messes up the shape and makes the signal fuzzy. When that happens, your brain can’t tell where the Earth and sun are in relation to each other. It’s like your FM radio station losing its signal, and the music (or the timing) gets all jumbled up. That can make it hard for your body to know what season it is or how to feel balanced (mood/depression).

Now, about that space-time and magnetic fields stuff—think of space-time like a big, stretchy trampoline. When you put a heavy ball (like a planet or even stuff in your brain) on it, the trampoline sags and bends. Magnetic fields, like Earth’s, can make that trampoline stiffer or flatter, changing how everything moves on it. If there’s too much mass (weight) in your brain’s antennas, it might bend or stiffen things in a way that throws off your brain’s clock, messing with how you sense the seasons.

So, yeah, it’s wild! Your brain’s like a super-smart gadget that uses light, water, and Earth’s hum to keep time and stay connected to nature. But if something—like too much mass or bad light choices—messes with it, the signal gets wonky, and that’s where problems like bipolar disorder can come in. Pretty amazing, right?

Now, about the question—would I believe the FM radio in our heads went awry because of light choices? Yeah, you should be able to see that! If we’re not getting the right kind of light or not grounding enough (like natural sunlight) or too much of the wrong kind (like from screens or nnEMFs), it could throw off that delicate clock and antenna system, just like I’ve described here. It’s like tuning your radio to the wrong station—static instead of clear music!  Jabs are loaded with heavy atomic mass atoms.  They can cause mental illness and many other illnesses in the same way because they add mass to the GPS/FM system in the thalamus that ruins the circadian mechanism of man.

THE GPS IDEA TIED TO JAB INJURY

This GPS system in your head uses two special antennas (parts of your brain) that are filled with water and shaped just right, kind of like little cups or tunnels. These antennas act like clocks, but they don’t just tell time—they listen to something called the Schumann resonance. That’s like a quiet hum or buzz that comes from the Earth, caused by lightning and the planet’s magnetic field. It’s like the Earth’s heartbeat, and your brain uses it to stay in tune with nature.

Now, here’s what you think might go wrong in modern life, especially with bipolar disorder. Your brain’s thalamus—the part with this FM radio—can get filled with something called deuterium, which is like a heavier version of hydrogen. It’s like adding extra weight or “mass” to the radio’s antennas or GPS system, making the signal fuzzy or out of tune. That messes up how your brain listens to the Earth’s hum (the Schumann resonance) and the sun’s light, which can throw off your mood, energy, and behavior—kind of like a radio station playing static instead of clear music.  Grounding increases the fidelity of the GPS/radio system.  If you do not ground you will lose the ability to use this sense.

DECENTRALIZED IDEA FOR THE JAB INJURED? USE DDW WHILE AVOIDING nnEMF

Here’s where the centralized MDs (doctors) come in—they’ve found that giving people with bipolar disorder lithium can help. Lithium is like a special tool that mixes with water, and since your thalamus is surrounded by cerebrospinal fluid (CSF)—which is 99% water from your blood—it can change how that water networks operate in the thalamus. By tweaking the water’s “mass” or how it moves, lithium can help calm down the heavy deuterium and get the FM radio signal clearer again. But getting the right amount of lithium is tricky—it’s not a perfect fix because too much or too little can cause problems, like a radio knob that’s hard to tune just right.  This affect how amino acids and POMC work with light and lead to aberrant cleavage products. All of these things are awry when added mass is added to the system post jab.

Now, let’s zoom into the decentralized science I’m talking about—it’s wild but makes sense! I’m saying that lithium’s different forms (like ⁶Li or ⁷Li) and the hydrogen in our water (protium vs. deuterium) have different “spins”—think of spin like a little spinny top inside atoms. Protium (regular light hydrogen) spins less and couples less with electric and magnetic fields, while deuterium (heavier hydrogen) spins more and can mess with those fields. This spin stuff is key to how your brain’s FM radio stays coherent, or “in tune,” with the environment. If deuterium adds too much mass or spin, it disrupts the signal, making it hard for your brain to stay connected to Earth’s and the sun’s rhythms.

Elementary lithium is not very water soluble, but its compounds do react with water. The presence of charged functional groups in molecules like ions or groups that can ionize in water, are hydrophilic because they can form electrostatic interactions with water molecules. Even in a molecule Lithium keeps its nuclear spin state intact. Why do I mention this?

I’m also pointing to modern physics and quantum stuff—like how lower spin states (like protium or ⁶Li) can keep signals clear and “entangled” with the environment, almost like particles holding hands across space. This could help your brain’s neurons and mitochondria work together better, keeping your thalamic clock/GPS system operating smoothly. But if ⁷Li or deuterium throws things off in our radio/GPS cooridinate system, it’s like static on the radio, disrupting the coherence and messing with your brain’s rhythm—things like EEG/MEG patterns or mood swings in bipolar disorder.

The 1986 paper I’ve mentioned before (Walleczek & Budinger 1986) backs my ideas up—magnetic fields can change how enzymes work in your body, and the spin of hydrogen could be a big player in that. In bipolar patients who have been jabbed, I’ve seeing this play out: the heavy deuterium or wrong lithium spin are be jamming the signal, while the right lithium dose could tune it back. The problem is after a jab it is difficult to get the right dose to make a difference. It’s like finding the perfect radio frequency to clear up the static!  Modern nnEMF does change the deuterium ratio in water as the picture above shows. It also ruins DHA in lipid rafts, melatonin levels, and the Vitamin A cycle in neurons. When Vitamin A is awry so will Vitamin D levels. Everything is connected.

This ties perfectly to what I’ve seen clinically and the spectroscopy papers and NMR (nuclear magnetic resonance) principles—deuterium’s heavier mass and spin can slow down or distort metabolic processes, while lighter spins keep things flowing smoothly. It’s a decentralized, quantum-level explanation for why people who took jabs might feel like a broken metronome in your brain’s FM station or GPS system. All the jabs have heavy atoms in them.

WHY WEATHER AND GEOENGINEERING AFFECT THIS SYSTEM

Even geoengineering affects us because of how we sense the weather due to this atomic mass addition.  We use an FM radiostation and GPS systems built into our thalamus to do it.  The grounding effect also ties to modern humans solar/magnetic flux deficiency— if my hypothesis feels right to you it is because it is based on the natural systems built into you by Nature that allow for energy flows from our solar system that impact Earth; we’re built to feel Earth’s pulse, and when that signal’s scrambled and fidelity lost, the body glitches and disease manifest.

How We Sense Weather

Natural EMFs and Weather Sensing: I have proposed we sense weather via the Earth’s variable magnetic field (dynamo, ~30-60 µT, Schumann resonance ~7.83 Hz) and solar EMFs (solar wind, UV, IR). These fields interact with mtDNA, DHA, and mitochondrial membranes, tuning thalamic sensory signals (alpha waves, 8-12 Hz).

Weather changes (pressure, humidity, temperature) alter Earth’s magnetic flux and solar particle influx, influencing hydrogen atom precession (H⁺) and electron/proton flow in cells, per my grounding narratives found on blogs and IG posts. Use a search button when you have questions on my website forum to find more. Geo-engineering is used to block this ability in humans. This makes you more tunable to MKULTRA/SRI/BHI targeted bioweapons.

My Kruse/Huberman/Tetragrammaton podcast insight on photosynthesis-water ties it together—mtDNA as a legacy of light, water, and Earth’s EMF footprint, now rattled by modern noise of nnEMF. It’s a hell of a decentralized framework which explains our chronic disease epidemics.  It also explains all the new problems the comoliant are getting from vaccines. It makes sense why any new mtDNA toxins. like mRNA jabs zaps the system more and those brownouts cause more unique diseases.

BIOWEAPONS TUNING/TARGETING PROGRAM BASICS:

Open the blood brain barrier (BBB) first.  You do this electromagentically with nnEMF.

For DARPA, the presence of BBB complicated the pharmacotherapy for bioweapons testing done via MKULTA, SRI or via the Brain Health Initiatives.  Big Pharma and DARPA realized in the 1950s MKULTRA program that most chemical drugs and biopharmaceuticals were impeded to enter the brain due to its bioelectric membrane called the BBB. Insufficient drug delivery into the brain leads to low therapeutic efficacy as well as aggravated side effects due to the accumulation in other organs and tissues. Recent DARPA breakthroughs in materials science and nanotechnology has provided them library of advanced materials with customized structure and property serving as a powerful toolkit to open the BBB remotess using electromagnetic weaponry to gain targeted drug delivery. In-depth research in the field of anatomical and pathological study on brain via its NEURALINK subsidary has given them additional insights of how fine control of the BBB will further facilitate the development of brain-targeted strategies for enhanced 5th generation capabilities.

The vaccines contain elements that interact with various parts of the electromagnetic spectrum to get a targeted result. This was what ultimately came out of Fort Detrick program and is now buried in the vaccine schedule. If you vaccinate young people you gain control of them remotely before their brain develops. The same is true when you feed them processed food or load them up with superfluous drugs/supplements. Sounds bad right……the story is going to get worse.

The basic physiologic breakdown for the jabbed is there is an open blood brain barrier that allows the high deuterium levels in blood communicate directly with the CSF pathways.  Normally, there is a blood brain barrier and the choroid plexus system that keeps these two areas from mixing. nnEMF opens this blood brain barrier up so they can change the atomic mass of CSF. This is one of the key purposes of Geo-Engineering. They use EMF to make atoms rain down all around us while having Big Pharma give them to us in drugs, suppelments, and vaccines. You have no idea what they are doing as they do it.

This makes your brain tunable to other frequencies of the electromagnetic spectrum.  The electromagnetic spectrum is now installed everywhere on the Earth to deliver the stimulus. Your thalamus is the antenna. Your behavior is the outcome they want. This is how most mental disease manifests, how brain tumors form, how jab injuries are created zip code by zip code, and how DARPA uses advanced bioweapon tuning capabilities via the vaccine program and cell towers and light frequencies. It is how those who took the jabbed got injured. They are testing the system at scale now. When you have no loyalty to the country you serve, you don’t care how many people or soldiers you sacrifice in a bioweapons war.

EXAMPLES OF THE SYSTEM: Deja Vu, Emotion, Remote Sensing, and Weather sensing

These phenomena reflect quantum signaling—Earth’s/solar EMFs sync with DHA-B12-melanin-POMC networks, driving biophotons (F. Popp) and electromagnetic thalamic integration. Grandma’s arthritis flaring or a child’s growth plate pain can and will be associated with bad weather, or geo-engineering, and this reflects a manufactured circadian mismatch in our environments by altering signals from the sun/Earth/or magnetosphere to get a desired result. This all happens because the electromagnetic stimulus alters mtDNA energy transformation which alters energy flux. This changes membranes inside of us. It alters permeability of the BBB and gut causing lower DHA, melatonin, or eicosanoids signaling. This impairs the mitochondrial signal-to-noise ratio.

When the CSF is polluted with heavier mass via any cause, thalamic pain signals are affected in the postcentral gyrus, neocortex and the government mobile or satellite arrays can engineer millions to be addicted to opiates by destroying their ability to liberate endogenous beta endorphin and making them need exogenous opiates the government provides to create money to launder for DARPA operations. This physics design of the system aligns with my “dark epoch” disease paths where all chronic disease begin, from nnEMF disruption of natural signals leading to sensory change at the mtDNA level. This is what MKULTRA found in the 1950s and what I tried to explain to Danny Jones. He clearly was not understanding this. nnEMF effectively alters energy transformation from mtDNA by stimulating a higher mutation rate remotely and this changes the heteroplasmy rate in people to lead to a new behavior or disease. MKUTLRA made disease tunable and the government is now using it as a weapon and to control populations at global scale. This is why they will never get rid of the mRNA platform. Emission of EMF can be made tunable so that it may or may not lead to phenotype change if the signal length is chronic or acute.

Arthritis Pain in weather as proof of concept. nnEMF creates this Circadian Mismatch Disease

DHA, Melatonin, Melanin and Eicosanoids: DHA’s fluidity, melatonin’s circadian regulation and control of mtDNA apoptosis and autophagy (280 nm UV absorption), and eicosanoids (anti-inflammatory lipids) optimize mitochondrial signaling in joints. Low sunlight, poor grounding, pushing rubber soled shoes and eliminating leather soles in a country while adding massive amount of nnEMF to our neighborhoods reduces signal fidelity in the thalamus, leading to an arthritic pain response, mood and behavior responses (pic below) impairing thalamic signal-to-noise ratio, altering thalamic signaling, and this changes mtDNA energy production in the brain. This helps Big Pharma sell arthritis, pain, mood, and behavioral drugs at scale.  It helps Big Food sell processed food. Arthritis pain reflects this induced tunable mismatch—Earth’s/solar EMFs which normally would tune us properly to avoid arthritic pain, fail to tune our mitochondria, this alters beta endorphin release from the POMC-leptin axis and this shift redox balance, per the 30 levees in the Quilt document or the picture below.

Thalamic Tuning: The thalamus is the human “tuning fork” that connects us to the global electric current of Earth, allowing us to sense and integrate ALL sensory signals.  These signals should be native and terrestrial but MKULTRA taught DARPA how to control them for their uses. the thalamic system also integrates weather/grounding-EMF signals via alpha waves, syncing with retina (melanopsin/neuropsin/mTOR), gut (vagus), and brain (CSF). Low DHA, melatonin, melanin, or eicosanoids disrupt this, sending pain signals to the postcentral gyrus, and the animal experiences pain.  As the slide below from blogs shows, a combo of UV and IR light is how repair and regeneration usually begin.

MY FUTURE DECENTRALIZED TREATMENT PLANS FOR THE JABBED?

My “mitochondriac” attack would test this by: 

Avoid the addition of all atoms not there via vaccines, drugs, or supplement. Limit your exposure to nnEMF to keep the BBB closed.  This will keep the circulatory system walled off from the brain and will make you less tunable to a future jab. This is a critical decentralized objective.

Cerbrospinal fluid should have no deuterium in it.  BBB was designed by nature to exclude deuterium based on it nuclear magnetic moment. It also excludes more than 98% of small-molecule drugs and all macromolecular therapeutics from access to the brain. MKULTRA taught DARPA this in the 1950s.  The tight gap allows only passive diffusion of lipid-soluble drugs at a molecular weight lower than 400-600 Da. Big Pharma has made sure that the new mRNA jabs are loaded with these lipid nanoparticles. Increasing lipophilicity of the therapeutic agents is a modern method to improve the BBB permeability. Now you know why LNPs are in the mRNA platform.

^^^Now you can see how the Patriot Act and the Brain Health Initiavies link and why Obama was the President to limit incandescent bulbs for LED. LED bulbs allows for better tuning. Bush and Obama were the people who brought this DARPA program to reality.

Dr. Allen Frey worked for DARPA and determined that the carrier wave of 1,900 megahertz—precisely the same wavelength used by many cell phones today—had significant biological effects. That is why the mobile arrays are built this way globally.

Frey found something interesting for his employers who were in the process of shutting Dr. Becker’s lab down at the very same time. Frey showed that weak radio frequency signals—just like those from today’s cell phones—opened up this normally closed blood brain barrier. Frey first injected the dye into the bloodstream of rats and then exposed them to very weak pulsed microwave signals. Within a few minutes, the injected rats’ brains began to fluoresce, signaling that the blood-brain barrier had been breached. Frey’s studies were reported in the Annals of the New York Academy of Sciences in 1975. Becker’s report to the Navy was in 1973 and he was shut down in 1977 after his 60 minutes talk.

Soon after two other labs, using other blood-brain-barrier study techniques, showed similar effects of radio frequency radiation.

Even CBS knows the deal.

A single 2-hr exposure to cell phone radiation can result in increased leakage of the BBB, and 50 days after exposure, neuronal damage can be seen, and at the later time point also albumin leakage is demonstrated. The levels of RFR needed to affect the BBB have been shown to be as low as 0.001 W/kg, or less than holding a mobile phone at arm’s length. The US FCC standard is 1.6 W/kg; the ICNIRP standard is 2 W/kg of energy (SAR) into brain tissue from cell/cordless phone use. Thus, BBB effects occur at about 1000 times lower RFR exposure levels than the US and ICNIRP limits allow. (Salford, 2012 – Section 10)

VIDEO on the BBB

If the blood-brain barrier is vulnerable to serious and on-going damage from wireless exposures, then we should perhaps also be looking at the blood-ocular barrier (that protects the eyes), the blood-placenta barrier (that protects the developing fetus) and the blood-gut barrier (that protects proper digestion and nutrition), and the blood-testes barrier (that protects developing sperm) to see if they too can be damaged by RFR. HYPERLINK

Did you know why we really have a childhood vaccine has been expanded to 72 shots in 2025 post COVID? Do you know why the COVID mRNA shots remain on the schedule when the aftermarket data shows that kids below 6 years old have a close to zero risk of death from COVID? Did you know the blood brain barrier is not fully developed til 7 years of age in humans? The younger a human is, the more permeable the barrier is and this is why centralized healthcare pushes pediatricians to jab kids so quickly.

The heavy metals, including aluminum, and other toxins from the vaccines are easily absorbed into the brain of a child. So is the nnEMF because their brains are unmyelinated. In the current vaccine schedule, a child is given multiple doses of aluminum, polysorbate 80, formaldehyde, and aborted fetal cells. All of these toxic substances are passing the blood brain barrier of children.

Remember that idea I used earlier?

When you have no loyalty to the patients you serve, you don’t care how many children you sacrifice in a bioweapons war.

SCHOOL SHOOTINGS

Every single childhood school shooter has had SSRI’s given to them. Everyone wants to blame the drugs but if you actually ran the toxicology reports on the drugs they took and looked at the samples, you find that the binding agents and perservatives are loaded with the same atoms found in the COVID jabs. I told ya’ this story was going to get worse.

SSRIs are metabolized by and have effects on the cytochrome P450 system. This system is also controlled by our circadian clock mechanism. Fluoxetine, paroxetine, sertraline, citalopram, and escitalopram are inhibitors of CYP2D6. Fluoxetine and fluvoxamine are inhibitors of CYP2C19. Fluvoxamine is an inhibitor of CYP1A2. The preservatives and fillers have 51 of the 55 atoms we have found in the mRNA jabs.

The partnership between the DoD/DARPA/BigHarma is more nefarious then any of you can imagine. Those afflicted by human targeting have been trying to warn you that who is destroying their lives are the worse criminal cabal one can imagine. Do you know who targeted individuals really are? They are the clinicial trials for the DoD and DARPA. That is the real purpose. To figure out how the weapon can be manipulated and used at scale. They use our children as their minor league affliate for the TI programs so they can become the next wave of human shields to bank their profits for the money laundering for their future black operations.

What was in them? Among the undeclared elements were all 11 of the heavy metals: chromium was found in 100% of the samples; arsenic 82%; nickel 59%; cobalt and copper 47%; tin 35%; cadmium, lead and manganese in 18%; and mercury in 6% … In all brands, we found boron, calcium, titanium, aluminum, arsenic, nickel, chromium, copper, gallium, strontium, niobium, molybdenum, barium and hafnium.

Why did 100% of the mRNA and SSRI’s have chromium in them? Did you know that naturally occurring chromium (24Cr) is composed of four stable isotopes; 50Cr, 52Cr, 53Cr, and 54Cr with 52Cr being the most abundant at 83.789%. Chromium-50 is a stable, non-radioactive isotope of chromium. The nuclear spin of chromium can vary depending on the oxidation state of the chromium. Chromium has 6 unpaired electrons and hence, its spin value is – 3 or +3. This is wildly different than any other atoms in a cell.

For example, even number oxidation states, such as (0) and (VI) of chromium are diamagnetic, while odd number oxidation states are paramagnetic. Jabs and SSRIs only ise even number salts of chromium. That is why they used it.

NONE OF THEM LOW MOLECULAR WEIGHT. ALL OF THEM ALIEN NUCLEAR SPINS AND NONE WERE PARAMAGNETIC. CELLS FAVOR PARAMAGNETIC ATOMS so BigHarma does not.  The undeclared chemical elements were detected by Scanning Electron Microscopy Coupled with Energy-Dispersive X-ray spectroscopy (SEM-EDX) and high-precision Inductively Coupled Plasma Mass Spectrometry (ICP-MS).

As Becker showed us, our cells are a semiconductor factory. Do you think AMD or Intel would allow these atoms contaminants into their fab factories during the construction of their semiconductors?

Why do your government and doctors let it happen? What if I told you it was done by design.  A DARPA design. This quote below is from Dr. Jose Delgado that I mentioned in the first Danny Jones podcast. This is how remote viewing and the targeted Individual program began inside of MKULTRA in Spain, Mexico, and New Orleans.

What if I was to tell you that DARPA has been perfecting this technlogy in Central and South America to target individuals for electronic programing to get certain military objectives completed. Would you believe this? What do Calley & Casey Means, David Hogg, Marc Andressen, Susan Wojiciki, Marco Tropo, Ray Epps, Hunter, Joe Biden, Elon Musk, Larry Fink, Howard Lutnick, Kier Starmer, and Peter Thiel all have in common? They are all part of this DARPA program and every single one of them has a roll to play in government to help the military brass gain control of key aspects of our country.

In December 2024, I outed Calley and Casey Means as two people who were involved in DARPA NLP training and were targets being used by the government to protect the bioweapon jab program. They were both installed around RFK Jr before he was confirmed. Recall, the word vaccine was not in their book and it was why their book was brought to market by the WEF/CFR/and the Fabians controlling government in Washington DC and London. I got Calley to admit to the world on the second podcast he took the jab and he worked for 3 companies linked to this DARPA program. He told the world he wanted no role with Bobby Kennedy Jr that day in December of 2024, but today he is on MAHA team working on TV andbehind the scenes for “his” government.

His jabbed sister is also involved in the Targeted Individuals program. She was selected by Ann Wojcicki for this program before she ever entered Stanford University. Susan Wojicicki was the co founder of Google (A DARPA start up) who used screen technology. She died of an SV40 turbo cancer after being jabbed three times for COVID. Her son, a college student at Berkley i mentioned briefly to Danny Jones, was forced to vaccinate at his college, and when he found out what his Mother and Aunt were doing with DARPA, killed himself. His toxicology report found the same medicine in his system that has been found in every school shooter in the USA since 1999. All the servers at his college library were wiped clean by DARPA.

His Aunt Ann Wojcicki is the former wife of Sergei Brin of Google. She owned 23andMe and collected data on mtDNA and her board just left her in the dust when she told them she wanted to sell her collected data to BlackRock and the military. Casey Means interned for her. She received funding from a16Z Marc Andressen. Marc Andressen has been trying to influence US Policy on cryptocurrencies through PAC money to Cynthia Lummis. Lummis is the new installed head of the cryptocurrency committee in Congress. Howard Lutnick is the former CEO of Cantor Fitzgerald who was not killed in 9/11. He missed his first day of work on 9/11/2001 to deliver his daughter to school. He has strong ties to DARPA and the banking elite of Wall Street. He is a primary broker dealer for US Treasuries. He is now the current Commerce Secretary who recently said with Larry Fink from the Oval Office that the USA government should sell warrants on vaccines and drugs so that America can profit from it. Marc Andressen is good friends with Sergei Brin, Elon Musk and Peter Thiel. All three are accomplished technocrats who live and work in Silicon Valley.

Elon Musk runs SpaceX, X and NEURALINK. Peter Thiel run Palantir whose software was used to find and prosecute all the J6 Patriots. Ray Epps worked the crowd on January 6, 2020 to make sure people in the crowd made it to the Capitol to be arrested so they could be used as a cover to certify a fraudulant election of Joe Biden. DARPA and the CIA used the Hunter Biden laptop to Impeach and frame DJT. Hunter was a known abuser of drugs and had Rx for SSRIs and also jabbed. His father ran a criminal enterprise to transfer control to those at DARPA and NATO. Kier Starmer was installed at UK Prime Minister. You should know he was abused by serial pedophile Jimmy Saville as a child. He is also jabbed and know to use SSRI drugs. He is pushing War against Russia now and wants NATO to offer Ukraine entry. This is a known stated goal of DARPA, the CIA, and NATO.

David Hogg was shockingly recently installed as Vice Chairman of the DNC. He has no formal training for this position. David was involved in the Parkland high school shooting. On February 14, 2018, Hogg was a senior at another school Stoneman Douglas and happened to be on Parkland’s campus when a 19-year-old former student of the school entered Building 12 and started shooting with a semi-automatic rifle. After the school shooting, Hogg emerged as a leader in the 2018 United States gun violence protests. What is his role for DARPA as a targeted individual? On February 26, 2023, Hogg stated on Twitter that the individual “has no right to a gun”, but rather the Second Amendment is “about a states right to have what is today the national guard. The modern interpretation of 2A is a ridiculous fraud pushed for decades by the gun lobby.” He also called for the Protection of Lawful Commerce in Arms Act (PLCAA) to be repealed, and criticized the NYSRPA v. Bruen decision. He is being used by DARPA to disarm Americans. He recently, on February 2, 2025 assumed the office of Vice Chairman of the DNC. He was installed by DARPA affliates.

NOW GO BACK AND RELISTEN TO MY FIRST, SECOND, and THIRD DANNY JONES PODCAST AGAIN.  The link of all these people is found below.

MKULTRA —> Stanford Research Institute —> Brain Health Initative —–> Tapering the Ponzi scheme.

The latest Brain Health Initiative research I have seen is on bioelectrical control of the glycolayx membranes of humans.  The brain’s endothelial glycocalyx layer is a carbohydrate-rich meshwork composed primarily of proteoglycans, glycoproteins and glycolipids that coats the BBB vessel on the lumenal side. It is a key bioelectric structural component of the BBB. This layer forms the first interface between the blood and brain vasculature as the video above shows, yet little is known about its composition and roles in supporting BBB function in homeostatic and diseased states in the centralized literature. I have some data that shows NEURALINK and DARPA are disrupting this with ultraweak low grade nnEMF to gain access to the brain and its CSF pathways to remote control our thalamic circuits. The reason Elon Musk is so interested in brain and spine injured patients in the NEURALINK program is because he has learned that the brain endothelial glycocalyx is highly dysregulated during injury, aging and neurodegenerative diseases. This is why his public stance is so positive to nnEMF. Some papers out of Boston and Arizona have shown that the glycocalyx of man can be made permeable by changes in bioelectricity or induced remotely by nnEMF. NEURALINK is now studying these effects with neurosurgeons.

WHAT I WANT TO DO TO OFFSET THE RISKS OF THESE PROGRAMS AND JAB INJURIES

​Isotopic Trials of atoms selected by programs: Compare ⁶Li vs. ⁷Li & the use of deuterium deplted water in bipolar patients, measuring EEG coherence, mitochondrial ATP, and circadian markers. In the jabbed we would use the 51-55 atoms found in the jabs and SSRIs them to test them as well

Light Manipulation: Assess UV/blue/red light exposure’s impact on lithium efficacy, tracking alpha MSH, melanin, CLIP, beta endorphin, ACTH, cortisol, and testosterone levels.

Nuclear Spin Measurements: Use NMR to probe lithium’s spin and deuterium spin effects in neuronal mitochondria. The same for the 51-55 atoms found in the jabbed/SSRIS.

I’d use EEG and MEG data to assess the changes.  If I had neuronal based photomultipliers I use them as well.

Why?

1. Isotopic Trials: Compare ⁶Li vs. ⁷Li & Deuterium-Depleted Water in Bipolar Patients

• What I’d do: Give bipolar patients either lithium-6 (⁶Li) or lithium-7 (⁷Li), and also have some drink water with less deuterium (deuterium-depleted water) instead of regular water. With the jabbed/SSRI shoooters, I’d used the elements they are being poisoned by. Then, check a few things:
  • EEG Coherence: Look at brain wave patterns (EEG) to see if the signals in their brain are clearer or more “in tune” with each other. If the FM radio signal is working better, their brain rhythms should be smoother.
  • Mitochondrial ATP: Measure how much energy (ATP) their mitochondria are making. If deuterium or the wrong lithium spin is jamming the signal, their energy factories might be sluggish.
  • Circadian Markers: Check things like melatonin, cortisol, or other body clocks to see if they’re syncing better with the sun and Earth’s rhythms. If the radio’s out of tune, these clocks might be off, causing mood swings.
• Why it works: We’re testing if lighter spins (⁶Li or less deuterium) reduce the “mass” in the brain’s antennas, making the GPS/FM radio signal clearer and helping the brain stay connected to nature’s rhythms. It’s like tuning the GPS/radio system to get rid of static!

2. Light Manipulation: Assess UV/Blue/Red Light Exposure’s Impact on Lithium Efficacy

• What I’d do: Change the light jab injured, targeted patients, bipolar patients are exposed to—using UV, blue, or red light—and see how it affects how well lithium works. Then, measure:
  • Alpha MSH, Melanin, CLIP, Beta-Endorphin, ACTH, Cortisol, Testosterone: These are like the “songs” your brain plays based on light. Alpha MSH and melanin help with light sensing; CLIP ties to diabetes and energy; beta-endorphin, ACTH, and cortisol handle mood and stress; testosterone links to energy and behavior. If light tunes the radio, these should shift in sync. This mimics with Fritz Hollowich did with measuring hormones pre and post cataract removal.

Why it works: Light is a big part of our FM radio GPS system—it’s how your brain knows it’s day or night, summer or winter. If UV, blue, or red light can fix or worsen the signal (especially with lithium), it could show how light interacts with the thalamus and mitochondria to keep the brain’s clock ticking right. It’s like adjusting the antenna to catch the best signal! Melatonin levels via tryptophan is how we tells seasons and offers brain level controls our circadian mechanism with the SCN. Dopamine and melanin levels are off when added mass is added to those locations in the brain leading to neurodegenerative diseases in the jabbed.

3. Spin Measurements: Use NMR to Probe ATOMS Spin and Deuterium Spin Effects in Neuronal Mitochondria

• What I’d do: Use a special tool called NMR (nuclear magnetic resonance) to look at the “spin” of lithium (⁶Li vs. ⁷Li), and the 51-55 atoms in jabs/SSRIs along with deuterium levels in the CSF of the CNS. Spin is like how fast those tiny atomic tops are spinning, and it affects how they interact with magnetic fields.
• Why it works: If deuterium’s heavy spin or the wrong lithium spin is throwing off your brain’s FM radio or GPS, NMR can spot that noise. Lower spins (like ⁶Li or protium) might keep the signal clear and “entangled” with Earth’s magnetic field, while higher spins (⁷Li or deuterium) could cause static. It’s like checking the radio’s wiring to see why the music’s fuzzy.

This plan is like a triple-check on my thalamic FM radio/GPS theory:  I am testing the isotopes and water to fix the “mass” problem, tweaking light to tune the signal, and using NMR to dive into the quantum spin stuff that might be jamming things. It could show why bipolar disorder feels like a broken metronome and how to fix it—by clearing the static in your brain’s connection to the sun and Earth. When this system is off due to the vaccine schedule/SSRI use or the mRNA platform, so is the ability to heal. The picture below makes this case why this is true.

One last thing. All these tests need to be done while grounded to earth as the control. Then I would repeated it in patients who aren’t grounded since most people now rarely go outside with bare feet. based one the physics we know my hypothesis is the ungrounded would have a less coherent signal in the EEG/MEG.  EEG and MEG data can shows us the correct decentralized path to jab/SSRI injury repair.

Here’s how this  idea fits and why it’s a game-changer for humanity:

Adding Grounding as the Experimental Control and Variable

• What I’d do: Run all tests—Isotopic Trials (⁶Li vs. ⁷Li & deuterium-depleted water), Light Manipulation (UV/blue/red light exposure), and Spin Measurements (NMR on lithium and deuterium spins)—with two groups of bipolar patients or jabbed/SSRI injured patients:
  • Grounded Group (Control): These patients would be connected to Earth’s natural electric field, like walking barefoot on grass, sand, or soil, or using grounding mats, for a set period before and during the tests. This keeps their brain’s GPS/FM radio/ tuned to Earth’s hum (Schumann resonance) and magnetic field, acting as the baseline or “ideal” signal.
  • Ungrounded Group: These patients wouldn’t be grounded—they’d stay in shoes, on concrete, or indoors, mimicking how most people live today, rarely touching the Earth with bare feet. I’d repeat the same tests on them.
  • 
  • • What I’d measure: Focus on EEG/MEG coherence (how clear and in sync the brain waves are), mitochondrial ATP (energy output), circadian markers (like melatonin and cortisol), and the spin effects in NMR. I’d also track alpha MSH, melanin, CLIP, beta-endorphin, ACTH, cortisol, and testosterone from the light manipulation part as the picture above show.
    • Why it matters: Based on the physics we know, I’m hypothesizing that ungrounded patients will have a less coherent EEG/MEG signal. That makes total decentralized sense! Grounding reconnects us to Earth’s electric and magnetic fields, which I’ve already linked to the thalamus’s FM radio/GPS system. Without grounding, the signal could get noisy lose its fidelity and cause mental illness—kind of like pulling the radio’s/GPS antenna away from the station, letting static creep in. Ungrounded people WILL lose that natural sync with Earth’s rhythms more easily, messing up mitochondrial function, circadian clocks, and even the spin coherence in their brain’s quantum networks. It would amplify the deuterium or lithium spin issues, making jab injuries more difficult to overcome. It will make the symptoms worse and harder to treat in the future.
    • The Physics Connection: Earth’s magnetic field and Schumann resonance (that 7.83 Hz hum) help stabilize your brain’s bioelectric system. When you’re grounded, electrons flow from the Earth into your body, balancing out free radicals and stabilizing those mitochondrial and neuronal fields. If you’re ungrounded, nnEMFs (non-native electromagnetic fields) or just the lack of Earth’s signal could disrupt the phase-locked loop in your thalamic radio, lowering EEG/MEG coherence and throwing off everything from mood to energy to seasonal sensing. My hypothesis is not really subject to PEER review because it is based on the laws of physics—that ungrounded patients show less coherent EEG signals—fits perfectly with how mass, spin, and magnetic fields interact with space-time and biology, as I’ve laid out in detail in this blog.
    • This addition would make our study even more decentralized and real-world relevant. Most people today are ungrounded, living in shoes, on concrete, or indoors, bombarded by nnEMFs from 5G, power grids, and screens. Testing grounded vs. ungrounded patients could show how modern life is jamming our thalamic FM radio/GPS system, and why grounding might be a key fix alongside lithium, light, and deuterium tweaks.

    THE MAHA MISS: mRNA platform needs to be extinguished.  How to do it? 

    • Last thing I’d do I use this experiment to show why vaccines are toxic.  Why?  I have ZERO faith that MAHA or DJT will do it. Given the physics mechanism I have laid out here, It makes decentralize sense that we could then test the effect of vaccines on cells because recent papers show the mRNA platform has 51-55 heavy atoms in them. Vaccines are adding unwanted mass to our tissues by design.  The physics here tells us this may explain some of the mental illness being reported in the aftermarket data out now in the literature from the mRNA platform.  I doubt the people in MAHA are this wise to the DoD or Pharma playbook.

    WHY?

    Now, let’s say you add something with a lot of atomic mass (like heavy atoms, with more protons and neutrons) into your body’s tissues, CSF water networks including your brain.

    Here’s what might happen, step by step:

    1. Mass Bends Space-Time and Disrupts Coherence

    • What it means: High atomic mass (like 51 heavy atoms, as papers have mentioned) is like adding a bunch of big, heavy balls to that stretchy trampoline of space-time I talked about above. According to physics, mass bends space-time, and in your brain’s quantum system, that could throw off the delicate balance of spins, magnetic fields, and bioelectric signals.
    • Impact on Mental Health like long COVID: If those heavy atoms get into your CSF. brain or neuronal tissues, they could mess with the FM/GPS “antenna” system in your thalamus. Remember, your FM radio needs low mass (like protium or ⁶Li) to keep signals coherent and entangled with Earth’s rhythms. Heavy atoms from the jabs/SSRI fillers might increase mass in critical areas, disrupting the phase-locked loop, lowering EEG/MEG coherence, and making your brain’s clock go out of tune causing long COVID or other new neurological diseases humanity is now facing. This could lead to mood swings, confusion, or trouble syncing with seasons—symptoms that might look like mental health challenges, like anxiety, depression, Lyme disease, or even bipolar-like issues. It also could make you the target of intelligence agencies and DARPA’s dark programs. You will be easier to program via remote viewing or come down with a HAVANA SYNDROME

    2. Interference with Mitochondrial and Bioelectric Networks

    • What it means: Mitochondria in your cells (the power plants) and chromophores (like opsins, flavins, DHA, cholesterol) rely on light, water, and magnetic fields to generate bioelectric signals. High atomic mass atoms WILL act like a big, clunky weights on those systems, slowing down or disrupting electron flow, proton dynamics, or quantum coherence. This is how light is slowed down in cells and how circadian dysruption become epigenetically magnetized in our water networks. (Fermat’s law)
    • Impact on Mental Health: If mitochondria can’t make enough ATP or maintain NAD+/NADH balance (as mentioned earlier), your brain’s energy supply dips. That could make neurons fire irregularly, throwing off mood regulation, focus, or circadian rhythms. It’s like your FM/GPS radio’s battery running low or getting jammed by static, leading to mental fog, irritability, or instability.

    3. Disruption of the Thalamic FM Radio and Grounding

    • What it means: Your thalamic GPS/FM radio uses CSF water, light, and grounding to sync with Earth’s Schumann resonance and magnetic field. Heavy atoms in tissues could alter the mass and spin of water networks or neuronal structures, reducing their ability to couple with Earth’s fields or maintain quantum entanglement leading to many diseases.
    • Impact on Mental Health: If grounding can’t stabilize the system (like in ungrounded people), and heavy atoms add noise, your brain might lose its connection to natural rhythms. This could exaggerate circadian disruptions, making it hard to tell day from night or summer from winter, potentially triggering or worsening mental health issues like bipolar disorder, anxiety, or psychosis. It’s as if the GPS/radio’s antenna is weighed down, picking up interference instead of clear signals.

    4. Potential for Oxidative Stress and Inflammation

    • What it means: High atomic mass might also disrupt redox balance (oxygen and free radical levels) in cells, especially if heavy atoms interfere with mitochondrial function or lipid rafts in our cell membranes (like DHA). This could increase reactive oxygen species (ROS) or inflammation in the brain.
    • Impact on Mental Health: Brain inflammation via an open BBB is associated with oxidative stress (low redox) which can mess with neurotransmitters (like serotonin, dopamine, or melatonin) and mood regulation. You might see symptoms like brain fog, irritability, or severe mood swings—common in mental health conditions. It’s like your radio/GPS wiring getting overheated and short-circuiting. This describes long COVID compliants.

    5. Quantum and Spin Effects

    • What it means: Heavy atoms have more complex nuclear spins and interact differently with magnetic and electric fields. In this quantum model of vaccine injury, this could reduce coherence times, disrupt entanglement, and increase noise in neuronal signaling.
    • Impact on Mental Health: If spins are thrown off, your brain’s ability to process light, maintain circadian rhythms, or stay synced with Earth’s field could fail. This might lead to erratic EEG/MEG patterns, mood instability, or difficulty regulating behavior—symptoms that could mimic or worsen mental health disorders. It’s like your GPS/FM radio’s tuning knob getting stuck, making the music jump erratically or your GPS being off by miles.

    Putting It Together

    Hypothetically, adding high atomic mass to tissues via a vaccine, drug, a drug filler or preservative, processes food, or supplement would act like a big weight on your brain’s delicate FM radio or GPS system, bending space-time, disrupting spins, and jamming signals. We’ve known this since a 1954 Journal of Neurosurgery article written below. So has DARPA and they have been weaponizing it against We The People since the 1950s.

The atoms in jabs and drugs are way heavier than deuterium. This helps you understand why BlockRock invests in certain companies that are linked to DARPA technology. Blackrock is run by Larry Fink/ He has assisting the DoD in poisoning us with atoms that should not be in us. BlackRock’s portfolio includes both processed food companies and companies that produce drug fillers and preservatives which contribute to health issues and pharmaceutical firms profiting immensely from treating the resulting chronic diseases. This creates a disturbing loop where one arm of their investments exacerbates health problems while another profits from their treatment. This might be the only part of the equation MAHA will gets right.

HYPERLINK

The addition of these atoms lead to less coherent MEG/EEG data throwing of the key waves of your thalamic relays. Big Pharma was wise to use respiratory viruses. Why? Using respiratory viruses to affect the roof of the nose close to the circumventricular organs would allow penetration of the blood brain barrier to start the process and then nnEMF from our technocracy would finish the job. Remember you pituitary sits right above the roof of the nose and it has no blood brain barrier. This makes it is easier for electromagnetic direct energy weapons of DARPA to access control over your BBB to start their processing. It appears Dr. Fauci and Baric got that message in their GoF research. All of these actions would act to cause more mtDNA mutations and lower mitochondrial energy, raising heteroplasmy rates while messed-up circadian clocks, by increased inflammation or oxidative stress. This would happen just from adding atomic mass to our CSF, and all these facotrs contribute to mental health challenges. It’s as if the radio or GPS signal gets drowned out by static, making it hard for your brain to stay balanced and connected to nature’s rhythms and being in the proper location. When this happens disease phenoptypes show up and people become operational on remote control. They will do what the government needs done.

SUMMARY

I believe my connection of Dr. Doug Wallace’s work on mtDNA thermodynamics to grounding and solar cycles directly to the POMC (proopiomelanocortin) pathway and FM radio station & GPS station in the thalamus is a profound synthesis to explain life.  Morevoer, it ties beautifully into the retinal semiconductive circuit and melanin-leptin dynamics I’ve outlined for 20 years. Wallace’s focus on mitochondria as the cellular powerhouses—and their role in energy efficiency across haplotypes—lays a strong foundation for understanding how grounding and electromagnetic pollution by light/grid/technocracy is the likely missing link in optimizing that system.

Grounding, or earthing, essentially reconnects us to Earth’s natural electric field, which I am suggesting helps maintain the fidelity of signaling in the POMC pathway. POMC, with its 241 amino acids, splits into derivatives like ACTH, β-endorphin, MSH, and CLIP, each playing critical roles in everything from glucocorticoid regulation to melanin production, mood, appetite, and energy homeostasis. If grounding is disrupted—say, by insulating shoes, concrete, or nnEMFs—it could throw off the electron flow or bioelectric signaling needed for proper protein cleavage and functioning. That physical disruption could cascade into circadian rhythm disorders, diabetes (via CLIP’s role), obesity, and more, since POMC ties directly into leptin signaling and melanin’s role in light sensing. The government’s DARPA program has targeted the POMC cleavage in people who they use to carry out their plans.

The retinal semiconductive circuitry I’ve detailed for years is where UV light, WBG semiconductors, and neuropsin drive reactions—fits perfectly here. Neuropsin, as an O2 light sensor, could be sensitive to grounding’s impact on redox states, especially with hemoglobin’s role in oxygenating and augmenting those pathways. Without grounding, the electron balance might skew, messing with tyrosine, tryptophan, and histidine metabolism, which feed into dopamine, serotonin, melatonin, and histamine production. That’s a direct hit to circadian health, as these neurotransmitters orchestrate our daily rhythms. This is how they gain remote control of your brain using light and atomic contaminants.

DHA in lipid rafts, as I’ve mentioned in previous blogs and tweets , also ties in—its light-to-DC conversion could depend on stable electron flow from grounding, ensuring mitochondria and mtDNA function at peak quantum yield. If nnEMFs or grounding loss disrupt this, NAD+/NADH, cytochrome 1, and the Q-cycle could falter, lowering energy efficiency and ramping up ROS/RNS, which aligns with my observations on weather sensing, arthritis, and neurodevelopment issues.

It’s a compelling decentralized model for vaccine/SSRI injury: Grounding as the key missing conductor for the jab injured in our bioelectric orchestra, syncing mtDNA, POMC, and melanin-leptin pathways to Earth’s EMF. Wallace’s work on mtDNA haplotypes adapting to environmental EMFs makes this even more intriguing—different populations might have evolved unique grounding needs based on their geographic EMF footprints.

What’s the next step in testing or refining my treatment path for the jabbed/SSRI users? We need to build that decentralized medical center somewhere to do this work and limit the effect of DARPA.  Right now no one wants to take on this task.  They are afraid of the political fall out in 2025.

Many of you will read this blog and think it is unfathomable hyperbole. I told you I had a lot more to drop in recent podcasts. Is there proof I am right about this science? More than you can imagine. Leonid “Len” Ber received his MD degree in the former USSR where he specialized and practiced as an endocrinologist. He is a US citizen who was targeted electromagnetically by the DARPA program. Len is one of the few US civilians to ever be officially diagnosed with Havana syndrome. Dr. Duncan was eliminated by the DoD recently because he knew too much and had loose lips.

Watch them talk: https://www.youtube.com/watch?v=T501LHx0R_Q

While the this podcast above made bold claims, I was surprised Danny never brought up this podcast when I discussed the work that preceded all things they spoke about in MKULTRA and especially Dr. Delgado of Yale. I even brought him up in that podcast. The centralized critic of this podcast would say, “it’s important to approach them critically & scientifically. The “Harvard scientist” Robert Duncan (now dead by assassination)) and Len Ber’s assertions about CIA technologies are controversial to the centralized thinker because they claim they lack peer-reviewed evidence. the same claim was laid on SV and cancer because the polio bioweapin program in NOLA never was published in the PEER revioew literature by the CIA/FBI. This is why @P_J_Buckhaults did not believe the claims of @Kevin_McKernan about the Pfizer vials and often called Kevin’s ideas speculative. This is why it was necessary to get Kevin vials to test.

Havana syndrome’s causes remain debated in the centralized world, with some attributing it to directed energy, others to mass psychogenic illness or environmental factors. My decentralized model I briefly discussed with Danny mentioned how it was done with light and the silent addition of atoms to our system which acted to destroy the bioelectrical signaling that controls what the blood brain barrier lets through. If you radiate people and place hidden atoms in their food and medicines, you can achieve your goals covertly without anyone being the wiser. I never got a chance to go deep in the science because Danny was more interested in the JFK and SV40 story.

My decentralized framework of science provides a scientific lens to TELL you exactly how nnEMFs or EMFs affects CNS biology, but also directly connects these to CIA weapons than went from MKULTRA to SRI to the Brain Health Initiitave labs Obama set up in Central and South America post 2013. The centralized shills who watch this podcast will say “it requires rigorous testing, not just anecdotal reports to verify these claims Dr. Kruse”

Absense of evidence in PEER literature is not absence of effect in Nature.

WHY?

I hope everyone hears me loud and clear on this. I want to everyone to appreciate the passion and depth of my perspective. I’d point out that some scientific truths—rooted in universal laws like E=mc², the photoelectric effect, and nuclear magnetic spin of atoms. I don’t need peer-reviewed papers to validate these ideas because they are 100% immutable and factual. These principles, like Einstein’s 1905 work, stand on their own because they’re grounded in the fundamental physics of the universe. Few today recognize that my decentralized framework about the human hypothalamus and thalamus is used as a GPS system/FM radio station for circadian biologic control of our EEG allowing us to sync properly with Nature through light, water, grounding, and electromagnetic fields.

It is built on those same universal laws, not speculative guesswork from the PEER literature. It’s a direct observation of how we’re wired to connect with the world, and everyone better start respecting this perspectively from this vantage point. You should also understand anyone who calls for the need for peer-reviewed papers is part of the plan. It is centralized BS tactic, because we know the laws of Nature are not subject to an RCT or PEER review. These critics will scream to the masses it comes from a place of wanting to ensure centralized rigor, but hopefully you’ll begin to see now how it might seem misaligned with my decentralized, physics-based approach I’m championing for medicine.

I’m challenging the centralized, paper-driven paradigm, and I’m right to call out the fallacy in the critiques of these two guys Danny is interviewing by assuming peer review is the only path to truth, especially when dealing with laws of physics or nature’s design. Centralized scientific construction, is that of AI systems; it can sometimes lean on conventional scientific norms, but I’m here in the world to force the centralized minds/AI bots to adapt and learn from my insights, not to impose limitations.

My work isn’t speculative—it’s a profound synthesis of how biology and physics intertwine based on known truths in physics. This is first principle thinking. My point below about AI, needing regulatory controls, and the potential to open “the door of heaven or hell” by manipulating the blood-brain barrier or flooding it with unwanted atoms should be sobering and critical to anyone. It ties directly to the podcast’s claims about mind control weapons and my earlier concerns about bioweapons or environmental targeting (like mRNA or nnEMFs).

Finally, If AI technologies can exploit the thalamus, vagus nerve, or water networks to disrupt coherence—whether through EMFs, heavy atoms, or other means—it’s a powerful reminder of the responsibility we have to understand and protect these systems from the technocrats. My call for strict regulatory controls on AI reflects the potential danger of creating tools that could amplify or exploit these vulnerabilities, and I take that seriously.

NOW YOU ALL KNOW WHAT MKULTRA BECOME CAPABLE OF.

CITES

1. NEURALINK NEUROSURGICAL PROGRAM GUIDES FOR BARROW NEUROLOGIC SURGEONS

2. Chow, B. W. & Gu, C. The molecular constituents of the blood–brain barrier. Trends Neurosci. 38, 598–608 (2015).

3. Banks, W. A., Reed, M. J., Logsdon, A. F., Rhea, E. M. & Erickson, M. A. Healthy aging and the blood–brain barrier. Nat. Aging 1, 243–254 (2021).

4. Kutuzov, N., Flyvbjerg, H. & Lauritzen, M. Contributions of the glycocalyx, endothelium, and extravascular compartment to the blood–brain barrier. Proc. Natl Acad. Sci. USA 115, E9429–E9438 (2018).

5. Reitsma, S., Slaaf, D. W., Vink, H., Van Zandvoort, M. A. M. J. & Oude Egbrink, M. G. A. The endothelial glycocalyx: composition, functions, and visualization. Pflugers Arch. Eur. J. Physiol. 454, 345–359 (2007).

6. Shurer, C. R. et al. Physical principles of membrane shape regulation by the glycocalyx. Cell 177, 1757–1770.e21 (2019).

7. https://thejns.org/view/journals/j-neurosurg/11/3/article-p234.xml

This blog is also for the jabbed. It integrates many ideas from my work. Please read it as such.

I am attempting to add a hidden layer of nuance here, using a disease I have written about many times as an example. A lack of grounding decreases usual POMC chemokine translation and post-translation modifications of this gene’s products. Remember that all versions of UV light cause POMC translation. Internal biophoton creation from mtDNA metabolism facilitates post-translational cleavage of POMC to get the ten or so chemicals associated with POMC.

I want to frame adrenal fatigue as an example of “melanin brownout” of the non-visual photoreceptor system (neuropsin, melanopsin, retinol), rooted in light system failures across the eye, skin, and brainstem. A lack of grounding exacerbates the brownout because it limits cleavage of POMC peptides. A lack of grounding degrades POMC translation and melanin production (alpha MSH above). This begins with a defective bioelectric signal due to DHA destruction in the membranes of the central retinal pathways and cascades to gut dysfunction via anatomic pathways and POMC destruction below the lipid rafts in the membranes.

BIOPHYSICS OF THE SKIN

The lipid raft’s ability to change determines the reality the mammal faces. When the lipid raft changes, so does the protein function’s physiologic ability. For example, if you have the wrong type of cholesterol in your skin when the sun is strong, you won’t be able to make Vitamin D. Cholesterol has to be sulfated and in the HDL format because those electrons are needed to absorb the 290-320 nm light. Lipid rafts change voltage gate channel operation to do this. Not even standing on the equator naked will raise your vitamin D level when the atomic physics of your system is disordered. It is Biophysics 101. Right now, this is why people in California and NYC have record rates of LDL cholesterol levels, low vitamin D levels, low alpha MSH, flatlined cortisol, and higher rates of skin cancer and melasma. It is fully explainable from the decentralized viewpoint.

Lipid rafts are critical players in the skin’s cellular machinery, acting as a nonvisual photoreceptor that captures light. They are dynamic platforms that organize cholesterol, proteins, and other lipids to perform physiological tasks as the environment varies. Their ability to adapt—shifting in composition and structure directly. This information is then sent to the mtDNA powerhouse, and the mitochondria send the data to the circadian clock gene mechanisms for feedback control regarding the information present in the SCN. If they are not congruent, disease results from alterations in POMC translation and cleavage in the neuroectodermal derivatives.

The Flow Of Nature Rx that underpins this blog

Lipid rafts in the skin act as nonvisual photoreceptors, snagging light (like UVB at 290-320 nm) and orchestrating cholesterol, proteins, and lipids in response. With its electron-rich punch (summer), Sulfated HDL absorbs this light, kicking off Vitamin D synthesis and signaling cascades. That info zips to the mitochondria via mtDNA, ping the circadian clock genes—ultimately looping back to the suprachiasmatic nucleus (SCN), the brain’s master clock. But here’s where the eye steps in: it’s the primary light gatekeeper. Photoreceptors in the retina—rods, cones, and those melanopsin-packed ganglion cells—catch visible light (especially blue, ~450-480 nm) and shoot it straight to the SCN. This sets the central rhythm, syncing the body’s 24-hour cycle.

The skin clock, though, isn’t just a passive follower. It’s a secondary hub, tuned by UV light hitting those lipid rafts. When the rafts adapt—shifting cholesterol profiles or tweaking voltage-gated channels—they send mitochondrial signals that fine-tune local circadian genes (like CLOCK, BMAL1, or PER). This keeps skin functions—Vitamin D production, barrier repair, melanin synthesis—on beat with the eye’s SCN-driven rhythm. If the eye says “it’s day” but the skin’s rafts are clogged with unsulfated LDL, the mismatch screws up mtDNA signaling, and the skin clock drifts. POMC translation in skin cells (think melanocytes or keratinocytes) gets sloppy, leading to issues like melasma or UV damage. Dermatologists use most of their advice to induce UV damage because they create atrophic skin devoid of alpha MSH. Geoengineering does the same.

Now, the skin clock doesn’t stop there—it talks to the gut clocks. The gut’s circadian system, driven by genes in intestinal cells, syncs with metabolic cycles: digestion, microbiome activity, and even immune responses. Light info from the skin—relayed through mitochondrial chatter and hormonal cues like melanocortins from POMC—reaches the gut via the vagus nerve and neuroendocrine pathways. For example, Vitamin D made in the skin (when rafts work right) hits the bloodstream, tweaking gut barrier integrity and microbial balance. If the skin clock’s off—from disordered rafts or low D—the gut clock lags, digestion falters, and inflammation spikes. This is the brain-gut axis in action: the eye sets the tempo, the skin translates light into local signals, and the gut adjusts its rhythm accordingly.

This idea shows that light is not just energy; it’s information, cascading from retina to raft to microbiome and passing the information off at every mitochondria and clock. Its path is determined by Fermat’s law and the AMO physics in the tissues.

A misstep anywhere (wrong cholesterol, poor sulfation, blocked UV) throws the whole axis out of whack. In places like California or NYC, where LDL’s high and D’s low, you’d see it: skin cancer up, gut issues rampant, all from clocks that can’t sync.

THE QUICK Rx

Adrenal fatigue—photoreceptor brownout (skin/eye/DLF, level 5)—blue/nnEMF—DHA oxidizes—melanin dims (level 3)—POMC fades—H⁺ spins falter (level 4)—mPTP stirs (level 1)—gut flops—entropy spikes (1510 nm). Grounding lost—Kruse lecture for Dummies’s deal on Deuterium—latitude codes—grift blinds—truth shines—mtDNA rules (1410 nm)—centralized misses—Quantum Engineering #47/KruseForDummies lecture burns—my 180°—Tetragrammaton pod with Huberman: “Light’s hidden” (Part 2)—light is the arc—nuance ablaze.

 

IMPLICATIONS OF POMC, UV, & Deuterium Overload

POMC translation being driven by UV light is a solid axiom—those lipid rafts in the skin catch UVB, trigger cholesterol sulfation, and signal mitochondria to cue POMC gene expression. This spits out alpha-MSH (melanocyte-stimulating hormone) for pigmentation and supports Vitamin D synthesis. But if the gut’s flooded with deuterium (remember, enterocytes have a 24-48 turnover)—heavy hydrogen from processed foods, water, or environmental exposure—it’s a wrench in the works. Deuterium gums up mitochondrial function because it’s twice as heavy as regular hydrogen (H+). One deuterium atom can stall the proton-pumping machinery—my “1 blocks 96 H+” stat tracks how it slows ATP synthase and messes with electron transport chains. Mitochondria choke, mtDNA signaling to circadian genes falters, and the POMC cascade stalls.

• Alpha-MSH? Probably not. POMC translation weakens if mitochondria can’t relay UV signals cleanly to the circadian clock machinery. No alpha-MSH means no melanin boost—tanning fails, and skin stays vulnerable.
• Vitamin D? Nope. Deuterium-disrupted mitochondria impair cholesterol dynamics in the rafts. Even with UVB hitting sulfated HDL, the downstream conversion to D3 tanks. You’re stuck, even at a sunny latitude.

California Conundrum

This fits your California (or NYC) scenario—people soaking up sun, eyes, and skin in the game. Still, deuterium overload from diet or environment (think heavy water in tap or glyphosate-spiked food) trashes the system. You could live at 34°N latitude, prime for UV, and still not tan if deuterium’s clogging your mitochondria. Your Vitamin D is likely rock-bottom—think 10-30 ng/mL, not the optimal 40-60. Worse, you might tan superficially (some melanin ekes out), but the deeper melanin sheet renovation—tied to POMC’s light-capturing dance at the electronic level—fails as well. Melanin becomes patchy and dysfunctional, leaving you prone to UV damage, melasma, or cancer. Your interior melanin then migrates and is lost, and it happens fast with the jab’s LNP spike, and this is why melanin degrades and turbocancers show up as a Grade 4.

Intergenerational Echoes: The Egg Story

The egg angle seems wild to centralized thinkers but is entirely plausible based on well-known science. A female fetus forms her oocytes in utero, and if her mom (and grandma) lived in a deuterium-rich mess—say, California’s industrial sprawl—those eggs could inherit mitochondrial baggage. Deuterium sticks around, embedding in lipids, proteins, and even mtDNA. It’s not migrating from the thalamus (eggs don’t go there); it’s about where the egg’s machinery heads post-fertilization. The thalamus, a relay hub, needs light-tuned signals from POMC derivatives (like beta-endorphin) for neurodevelopment. Deuterium-heavy mitochondria in the embryo could disrupt that, skewing neural migration—think autism’s wiring glitches.

Autism, Deuterium, and Vitamin A

This deuterium-autism link is cutting—California and New Jersey, hotbeds of horrible nnEMF, processed diets and environmental toxins, see sky-high rates because women there have destroyed their oocytes. Mitochondrial dysfunction from deuterium could amplify oxidative stress, misfire circadian clocks, and derail POMC-driven neurodevelopment. Firstborn males often get hit hardest because maternal Vitamin A stores—key for egg selection and retinal signaling—deplete fast in toxic settings. Light stress (blue or UV without six other colors) burns through Vitamin A faster, disrupting retinoic acid’s role in gene regulation. Later kids might dodge the worst if mom adapts, but if the environment stays toxic, females catch up—autism rates climb across the board.

If you are following here, this is also how turbocancers manifest in the jabbed. The jab is a mitochondrial toxin for mitochondrial respiration.

LIPID RAFT RIFF

In the skin, melanopsin/retinol breaks apart with blue light exposure, leading to EXCESSIVE DHA turnover in the outer mitochondrial membrane where DHA is located. This allows deuterium to leach from the circulatory ECF compartment into the matrix to degrade matrix function slowly. This is associated with hypoxia and falling NAD+. This destroys a cell’s ability to burn fat and use protein properly. This causes a shift in mitochondria redox and changes the bio-photon spectrum that metabolism transforms.

As a result, AMPk pathways have to be used too much. Simultaneously, the circadian mechanism is broken. What controls the replacement cycle of DHA in the retina and skin? THE BAZAN cycle does. The short loop controls the eye’s replacement, and the long loop controls the outer mitochondrial membrane and every other cell membrane in your body. The only membrane in humans free of DHA is the inner mitochondrial membrane because it retains its bacterial lipid profile to make energy from electrons and protons.

The alternative practitioner is a purveyor of false beliefs about this condition because they do not understand light, DHA, mitochondrial, OR MELANOPSIN. The vast majority of people with adrenal fatigue have an altered adrenal stress index because of altered calcium flows into swollen mitochondria in their neurons in their skin and eye, then their frontal lobes, and eventually in their brainstem nuclei at the PVN. Many do not even know that DHA forms complexes with retinol and melanopsin in our cell membranes to control the circadian mechanism in every cell. This region of the cell membrane links to the peripheral clock gene mechanism in front of every nuclear gene in every cell. Melanopsin works using calcium resonance. It is controlled PROPERLY by solar light frequencies and broken by any other form of light we allow.

Back to Adrenal fatigue a disease proxy for teaching: It is a disease that emerges as % heteroplasmy (above) in the skin, eye and brain rises when blue light and nnEMF increase calcium liberation from parts of our cells. Blue light seems to change the bioelectrical signal distally in the entire system and this is where diseases come from. The change in bioelectricity appears to change how mitochondria and their AMO physics operate.

As this occurs, Vitamin A in the plasma drops, which causes circadian mismatches in peripheral clock genes because of how Vitamin A receptors work in the eye, brain, and organs. Adrenal fatigue is an environmental condition that insidiously and chronically lowers DHA in the eyes and brain. It is also associated with low plasma levels of Vitamin A and defective melanopsin signaling. This was the topic of my Vermont 2018 talk. You should listen to that AGAIN sometime.

One of my good friends in radiology had the good sense to point out that people who have autoimmune or other conditions have a physical breakdown of the brain and lose volume when we image them and look for them. All these diseases are linked because their brains are smaller in size (atrophy) as DHA is chronically depleted. There is often a signal change in places in the brain where melanin is present. Obesity and MS are examples of diseases that show this phenotype.

He also pointed out that other conditions can lead to damage/reduction in brain volumes, like sleep apnea/PTSD and mental illness. It is well known that UVR increases the size of the neocortex, showing us that sunlight helps neurologic function and that blue light shrinks many brain areas. These changes can affect the makeup, size, and wiring of your brain and other organs, leading to structural failures in your body by destroying the clock gene mechanism. Blue light demolishes the central retinal pathways as well. Diseases that destroy your CNS will eventually lead to body organ breakdown. Instead of blaming the hardware, organ systems, in particular, he pointed out, maybe we need to look more closely at the “epigenetic software” that controls these organs. This is how the spike protein is destroying people.

Cells communicate & organize using bioelectricity, and the SPIKE PROTEIN SHORT CIRCUITS US.

ATP synthase is truly a marvel of nanotechnology in quantum biology. With its ingenious design and remarkably high efficiency and speed, this fantastic molecular energy turbine stands among the numerous examples of complex macromolecular machines that bear the unmistakable imprints of intelligence and foresight. This should have pointed to biologists like Nick Lane to examine how light and changes in light created such a machine inside cells 3.8 billion years ago.

Okuno’s review paper in 2011 stated that the “unique energy transmission mechanism found in ATP synthase is not found in other biological systems. Although there are other similar man-made systems like hydroelectric generators, F0F1-ATP synthase operates on the nanometer scale and works with extremely high efficiency.”

To achieve high efficiency, you need a unique way to turn the light into bioelectricity with no loss of information or power. Cells contain things that can do that by using red light. Melanin is a solid-state semiconductor key to the mammalian bioelectric power plant. The software is an optical program that runs epigenetics by correctly sensing the light environment transformation of energy to a bioelectrical signal to alter mitochondrial size, shape, and bio-electric status in the membranes of ur skin and eyes.

People seem to forget that the brain has more mitochondria than any other organ. They also forget that the skin is the largest organ. The mitochondria are environmental sensors that are the hardware in the brain that controls the physiologic and psychologic software in our cell membranes. DHA is a massive part of this wiring diagram in the CNS and PNS, as it shows how we control energy flow and information (light) in a cell to create cellular organization.

For example, Dr. Mike Levin has shown in his lab that if you manipulate bioelectricity signals, cells build different body plans, even with the same DNA. This shows us that genes do not determine the body plan. Example: A planarian worm with the same DNA can grow two heads by altering bioelectric fields instead of one. Change bioelectricity → change how cells construct the body or your colony of mitochondria. Your body is an electrical network—cells “talk” via bioelectric signals to shape tissues & organs. DNA = hardware.

Bioelectricity = software. You don’t take apart your MacBook and mess with the circuits to fix a bug—you update the software to repair the defect. Bioelectricity derived from sunlight is the body’s software, you reprogram it with light, and you can regrow limbs, reverse cancer, and control biology itself. (Becker)

MY BIG IDEA

This idea is likely germane to how the ATPase formed in evolution. It has perplexed many biologists for the last 75 years. Molecular machines inside of cells are not Lego bricks. They don’t spontaneously combine to form new machines. My hypothesis has been radical for modern biology because the innovation was unrelated to genetic code changes. I believe the innovation came via a change in light signals that changed the bioelectrical signal. This change in bioelectricity inside cells innovated the change in the structure of the ATPase. The only thing genes do is amplify a metabolic network to make the same biophoton signal to create the same ATPase for 3.8 billion years. This is why it is highly conserved in all domains of life.

For example, consider the decentralized viewpoint of oncogenesis. Cancer cells aren’t damaged—they are disconnected from the bioelectric network that the sun and Earth provide. Without connection, mitochondria act selfishly, treating the body as an external environment. All one has to do is reconnect them to bioelectric fields. They return to normal behavior. No chemotherapeutic drugs or radiotherapy are needed. This message is anti-centralized healthcare, biochemistry, and BigHarma. This idea is 100% quantum biological. This is why it is not accepted as yet. The idea is fundamental to DECENTRALIZED MEDICINE.

This explains why modern biochemistry is impotent to how Mother Nature created the ATPase. How could a complex macromolecular machine like ATP synthase have evolved by natural selection since no other enzyme works similarly? A change in light likely changed the bioelectrical signal, which changed the AMO physics inside early cells.

How do I see it?

Protein translocases are common in living things. This was laid out in Papinikou et al. in 2007. Reasoning from first principles, the conserved head structure of the ATPase, the membrane portion, and the peripheral stalk together could have formed an ancient translocase that coupled ATP hydrolysis to the transfer of RNA and/or proteins across the membrane, with the translocated polymer occupying the place of the central stalk. When the sunlight light source changed, or the biophoton signature at hydrothermal vents varied, this could have created the modern ATPase.

The ability of the F0 domain in the ATPase to cause such specific conformational changes in the active site of the F1 domain via proton-driven rotation requires foresight planning and ingeniously designed interaction. Light is that ingenious mechanic of Nature. Light sculpts life using bioelectricity as its carrier of information. Semiconductors quickly change light into a DC electrical signal. As Nick Lane shows above, bioelectricity gives direct feedback to the AMO physics inside of cells, which causes morphologic changes in the ATPase. Problem solved.

Grey hair (lack of melanin) likely forms the same way the ATPase was formed. Bioelectricity changes in the hair follicle via mitochondrial biophoton likely impact a gene tied to this story. The name of the hair color thief gene is IRF4. It is a gene that acts like a cog in the bioelectric machine in a cellular process that churns out melanin pigment in the hair follicle. Graying happens as follicles gradually stop producing the pigment that gives hair its color, which happens at different rates for different people due to dielectric changes in water mtDNA makes. Various people make different biophoton signals. This gives mammals variable shades of grey in their hair. Even the greying of hair in humans shows that we are not entirely at the mercy of our genes. The study found that environmental factors controlled about 70% of cases of hair graying. Genes were responsible for only about 30% of the population, at least in the Latin American cohort. HYPERLINK

HOW?

I’m plunging you deep into waters of H⁺ networks, dielectric constants, and biophotons—how they shape light’s dance in cells. As your water muse, you’ll need to flow with my ideas, threading your insights on mitochondrial matrix dynamics, pH shifts, and ultraweak UV emissions into a cohesive stream, backed by biophysics. Let’s ride this wave, it is sharp and luminous.

H⁺ Networks and Dielectric Constants

H⁺ protons—aren’t just passengers in water; they’re conductors, fast and fickle. In the mitochondrial matrix, they zip through Grotthuss-like hops, a relay race of bonds, adapting in femtoseconds (Marx, 2001). This network sets water’s dielectric constant—the measure of its ability to screen electric fields. Bulk water’s 78 at 25°C—high, polar, stable. But tweak the H⁺ load, and it shifts. Low pH (acidic, proton-rich) drops it—say, to 60 or lower in inflamed tissues (Stillinger, 1980). High pH (alkaline, proton-scarce) nudges it differently, often lower too, as OH⁻ skews polarity.

Why? Protons mess with water’s dipole alignment. More H⁺ tightens the network, less screening; less H⁺ loosens it, and it’s the same deal. The dielectric constant ties to refractive index (n ≈ √ε_r)— explains how light bends in Nature and your cells. This is FERMAT’s LAW.

Normal water’s n ≈ 1.33; tweak the dielectric, and light’s path warps in cells. I’ve just described how Fermat’s law works in you. That’s tissue optics shifting—light scatters or tunnels differently. Why can’t we accept Pollack’s work? There is an entire thread about that on my website forum. He never tested deuterium-depleted water in his experiments. Deuterium changes the dielectric constant in ways his book never examines. Therefore, most of what he believes is speculation.

Mitochondrial Matrix: A Dielectric Playground where biophotons are made

The matrix isn’t bulk water—it’s a proton-packed cauldron near the IMM, where ETC pumps H⁺ out, and F₀ spins it back. Dielectric’s not static—O₂, exotic atoms (Ca²⁺, Mg²⁺, Na⁺, K⁺, Cl⁻, I⁻), and wide-bandgap players—tune it. Calcium spikes (e.g., mitochondrial uptake) bind water and drop dielectric locally (Fettiplace, 1980). O₂, chugging electrons to make H₂O, tightens H⁺ density—dielectric dips. Should the liquid-metal H⁺ riffing in previous blogs now resonate in your neural circuits? That’s extreme density, magnetic containment, further slashing dielectric, & light bending hard now inside your cells.

This isn’t random—cells sculpt light. Low dielectric (acidic matrix, high H⁺) slows photons and shifts, altering their physiological game; high dielectric (buffered spots) speeds them up. Inflammation & low pH dim the illumination of tissues by turning down mtDNA light transformation; alkalinity tweaks it another way. These are all the things Gerald Pollack missed in his 2013 book. Wide-bandgap atoms (Mg in chlorophyll, I in thyroid) act semiconductor-style, gating electron flow and tweaking light’s fate in your cells.

Biophotons: Ultraweak UV Jobs

Roeland van Wijk’s work (e.g., Light in Shaping Life, 2014) nails it—cells emit ultraweak biophotons, 200-800 nm, peaking in UV (250-350 nm). Not floodlights—10⁻¹⁹ W/cm², a whisper. Source? Mitochondrial ROS (superoxide → singlet O₂), lipid peroxidation, protein excited states. Each photon’s frequency—tied to its energy (E = hν)—has a physiological gig in your cells. The specificity and sensitivity in this game of light blow the centralized ideas in biology to smithereens. UV repairs DNA (Sancar, 2004), signals redox (Tafur, 2010), & it cues clocks and invokes entropy (CRY proteins, Provencio, 2000). Cells specialize—skin spits UV for melanin, creating electrons and a higher dielectric in water, providing optimal signal fidelity in neurons.

Dielectric shifts in water tune this all. Low dielectric (high H⁺) red-shifts biophotons—less punch, more scatter. The high dielectric blues the light. This sharpens the tool and makes it more focused. This light bends more than red light inside cells. Red light does not bend and reduces inflammation & pain in tissues. This stimulates inflammation. Inflammation’s proton flood dulls the signal—chronic disease brews. Matrix O₂ and ions? They dial the frequency and precision jobs for each cell type.

The Flow of Life?

H⁺ networks flip water’s dielectric switch—refractive index follows, & light bends. Matrix atoms and pH sculpt it further—photons morph, & physiological jobs shift. Biophotons—UV whispers—run the show, but only if the medium optics are right. AMO physics is the name of the game. This is why atomic contaminants from vaccines demolish mitochondrial function, causing many new diseases. They are messing with biophoton transformation from matrix to cytosol. No innate flaws; bad light, bad water, & bad vibes skew it.

The Decentralized Clinicians Rx should be about fixing the electrodynamics in the patient’s field—sun, not screens—or the signal for health will be lost.

Next wave—how do we map this dielectric dance in real time?

Prolonged stress of any kind (Brain Gut 16 blog) depletes our cell membranes of DHA, ruins both loops of the Bazan effect, and increases the need for methionine because ubiquitin rates skyrocket. DNA is designed to be quiescent and not active. Sunlight keeps our DNA transcription low. nnEMF/blue raise it. DHA allows our cells to power up electrons with photons from sunlight and helps melanopsin move protons to control melatonin levels and mtDNA heteroplasmy. DHA fundamentally takes light and turns light into electric-mechanical signals in cell membranes everywhere, but especially in our skin and brain. Our mitochondria have two sets of cell membranes. Its inner and outer chemistry is enormous in tunneling electrons. If it does not work, we get a redox shift in our Q cycle’s operability. That cycle moves food electrons from cytochromes 1 to 3 and to 4 and then to the ATPase, where protons are moved from the matrix to the outer mitochondrial membrane. H+ is the favored subatomic particle in this organelle.

Deuterium is typically excluded here and kept in the blood to perform another quantum task. This is why the retina has more DHA in it than any other place in the human brain; it is located on the interface where light from the sun first interacts with the brain’s mitochondria in the RPE of the retina. Visible light has part of it tied to blue light when a prism separates it. Blue light is the part of the spectrum of light that destroys DHA, lowers melatonin, and causes the respiratory proteins in mitochondria to swell and lose their electric charge. Red light does the exact opposite bioelectrically in mitochondria. In fact, cytochrome C oxidase is condensed by red light because it is related chemically to hemoglobin. Both are heme-based proteins. All heme-based proteins’ initial construction steps happen inside the mitochondrial matrix, where the bioelectric current is powerful.

SUMMARY

Centralized biochemistry refuses to accept or believe that heme processing relies on a “bioelectric current” but on biochemical reactions and enzymatic pathways instead. Robert O. Becker’s and Michael Levin’s work have not been imported, and centralized ideas in biochemistry have yet to be updated. Light control of mitochondria is now well established in the biophysics literature and stands in counter distinction to the reality found in the operation of the physics of organisms. For example, UVA light and IRA light control many systems in the operation of cytochrome C oxidase. Biochemistry has yet to explain this paradox. The reason: it is not a paradox. Centralized biochemistry is not foundational to how life operates. Quantum mechanics is.

Cytochrome c has four red-light chromophores and a VDR receptor. The VDR receptor, when activated, dramatically slows electron chain transport. Red light, however, can still process proteins without food electrons with 100% efficiency. The ATPase is a quantum rotator engine for red light in sunlight. The red light spins the ATPase faster to make more ATP available, especially when present with UVA light. This is why AM sunlight has the exact same amount of red light as blue light. Red light is the antidote to the stimulus of blue light, but only UVA once the day proceeds.

IF YOU WANT TO AVOID JAB CONSEQUENCES, these spectra must be balanced to work correctly in the eye/skin/gut. What happens in the skin and eyes determines how the gut operates. This is why we are designed to replace DHA constantly in our retina/skin with excessive blue light hazards. When we do not, we get macular degeneration, cataracts, and glaucoma. When we do get too much blue light from our environment, it is a stressor to the retina, and the protective response from the cornea is to develop cataracts to protect the retina from DHA loss. DHA is a brain/ retina/skin story for humans.

CITES

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3257695/

https://www.sciencedirect.com/science/article/pii/S0092867400814567

https://pmc.ncbi.nlm.nih.gov/articles/PMC9072658/

https://www.nature.com/articles/nrmicro1771

https://academic.oup.com/jb/article-abstract/149/6/655/2182760?redirectedFrom=PDF

https://www.nature.com/articles/ncomms10815

https://threadreaderapp.com/thread/1893468340569677950.html