Very few people know the story of Stevie Nix and her band mate.  She was Alannis Morisette before Alannis was.

Some people confuse intensity for passion and challenge for attraction.  This happens most often when people are not optimized biologically.   I don’t think Stevie or Lindsey ever realized why their love was doomed before it blossomed.  Their light environment was a killer as rock stars.

This leads to us chronically making the same mistakes with the people we choose to love.

All too often we fall in love with the same person, but with a “different face.”

When you get your redox house in order, you’re rebuilding your magnetic power in your colony of mitochondria, so is the human magnet situation why we continue to love people who hurt us?

Have we jumped in too early before our brain mitochondria have caught up to the pituitary glands which control desires?

So ask yourself this question……………Is attraction just flesh and connection linked to soul and spirt?


Is this true?  Are you a buyer or seller of this meme in your own life?

When healing your ailments, you want to make sure you are falling for a healthy partner, and not recreating old dysfunctional patterns of your past.  If you do you will wind up with another bad relationship. You have to fall in love with your best friend.

This means you have to spend time alone finding a friend who is worth your best.  Then you have to give them the best of you as a friend sans the romance.  This takes time and effort.

Every relationship, good or bad,  trains you for the next one.

With healing Black Swans I have seen over time in my practice that one of the most prevalent and under-discussed issue they all have is the powerful and dysfunctional attraction between codependents (“givers”) and pathological narcissists (“takers”).  It shows up on social media forums in many journals on my website.   It has shown up on member events I have put on…….

Real growth calls for  taking ownership of your decision tree.  Thinking is a function of redox power.  So who enters your circle of six wholly depends on your mind.

This exercises in examing how you think about attraction and connection isn’t about blaming yourself. It is about understanding yourself by how you think. Your dating history is more than a pile of wreckage.

It’s a story. You’re the main character and you wrote the novel with your choices. Think about what you have carried from one to the next — both good and bad.  Then ask yourself why you did this?  It usually comes back to how you think about yourself. 

Simply regretting failed relationships leaves you in the backseat. Or even worse, tied up in the trunk — trying to kick out a tail light. Relationships don’t happen to us. We make them happen because of how we think about things and about ourselves.

The vanity of attraction gets people in the most trouble. It really is ludicrous that humans often chose people because of beauty and vanity. It’s akin to picking your meals based on their color instead of their taste.  What really matters to you in life in selecting your circle of six?  Your next mate should only come from your group of best friends.



When you are a slave to vanity, what you seek begins to seek you……..and sadly most get what we are seeking and the relationship ends badly again.

Watch what your attraction asks of you, you just might get it. Will a connection with a best friend do the same?

A healing Black Swan doesn’t fancy the colors of the face, they are always attracted to colors of the mind when they choose their next step.

Who is packing your relationship parachute now? Your old self or the new one your redox is sculpting? If you are using your old paradigm is that wise for your health reversal?



The mind has a powerful way of attracting things that are in harmony with it, good and bad and this is why your circle of six matters deeply to your health. If you do not get this right you will not recover. You will also have a circle of six that is impotent in helping you thrive.

When you are jumping back in to love, play fantasy with your first few choices…… might save you from failure in the future. The most exciting inerpersonal attractions most often occur between two opposites that never meet in reality. These fantasy games of attraction have a deep purpose. Ask yourself this, ‘If somebody never gets enough of you in the beginning, they will always want more.’ This will drown you and harm you longer term. That is how you can use your imagination to win.

You have to make your own relationships. You also have to create the love you want. It’s more than just a euphemism for sex. Good love takes time and effort.

You have to have the right ingredients IN YOU before the recipe works.

Anyone can date. Anyone can have sex. But that doesn’t mean you’re ready for an adult relationship. When you’re healing from an ailment, you’re actually just auditioning for one.

Some of us can love forever, some for six years, some for six months, and others for six hours. It turns out the context of the timing of the attraction matters a great deal given the situation in question. Choose wisely.




If you want to start dating better people, become a better person FIRST.

When we feel stuck in an emotion we are in a toxic relationship. When we haven’t gone through it and dealt with it, it replays over and over like a skipping record. We don’t realize that we can step outside of that experience and have new ones until our redox power returns to power up our ability to realize this fact because we can think well again.

You attract partners who are at your level of maturity and emotional intelligence. So instead of constantly shopping for the person you want to spend your life with, become the person you want to spend your life with.

Relationships grow and die just like plants.  They have a season to them for sure.  It is also true that love can grow, or it can die. It takes more than tips and pointers from any wise book, post, or person to make a relationship last. The Black Swan knows they will run into all kinds of problems, from the kitchen to the bedroom.

So you have to keep observing, communicating, experimenting.  Relationships are the ultimate mitohack in life.  Realize that it is your most important hack too when you are rebuilding your circle of six.




Why are physicians clueless about medicine? They have no idea how hydrogen bonding works in the hydrated proteins coded for in DNA/RNA. If they learned how to make ice cream they would learn very quickly how hydrogen bonds also explain the Mpemba effect. Might this be the key to understanding how to make DDW and measuring it in humans?

I believe it will be.

The Mpemba effect is the observation that WARM water freezes more quickly than cold water.

Water may be one of the most abundant compounds on Earth, but it is also one of more mysterious. For example, like most liquids, it becomes denser as it cools. But unlike them, it reaches a state of maximum density at 4°C and then becomes less dense before it freezes.

In solid form, it is less dense still, which is why standard ice floats on water. That’s one reason why life on Earth has flourished— if ice were denser than water, lakes, and oceans would freeze from the bottom up, almost certainly preventing the kind of chemistry that makes life possible.

Hydrogen bonds are weaker than covalent bonds but stronger than the van der Waals forces that geckos use to climb walls.

Chemists have long known that they are important. For example, water’s boiling point is much higher than other liquids of similar molecules because hydrogen bonds hold it together.

But in recent years, chemists have become increasingly aware of more subtle roles that hydrogen bonds can play but have been unable to pin down exactly why the Mpemba effect exists in water. For example, water molecules inside narrow capillaries form into chains held together by hydrogen bonds. This plays an important role in trees and plants where water evaporation across a leaf membrane effectively pulls a chain of water molecules up from the roots.

The effect is greatest in the arterioles and capillaries of your skin and subcutaneous fat where leptin and melanopsin reside and where nitric oxide is made to alter your microcirculation and blood pressure.

Blood is 93% liquid water by volume. Mitochondrial make all their cell water at cytochrome C oxidase (CCO), which is the 4th cytochrome in our mitochondria.


In liquid water, hydrogen bonds bring water molecules into close contact and when this happens anywhere in NATURE, the natural repulsion between the molecules causes the covalent O-H bonds to stretch and store energy from the environment or system where water exists.  This is really important in the skin where the sun affects the surface or in mitochondria where water is made at CCO and heat is liberated.

But as the liquid warms up, it forces the hydrogen bonds to stretch further apart and the water molecules sit further apart. This allows the covalent molecules to shrink again and give up their energy to the environment. The important point is that this process in which the covalent bonds give up energy is equivalent to cooling.  This has massive implications when you begin to realize that the bond between melanopsin and Vitamin A is a weak covalent bond in diurnal mammals and much stronger in nocturnal mammals.



It is clear that Nature uses the Mpemba effect between the day and night cycle. I wonder when people will finally see the effect of nnEMF energies to this science and how it generations melanopsin dysfunction and leptin resistance? Leptin is also a photoreceptor protein in our subcutaneous fat that is also destroyed and disrupted by melanopsin dysfunction of blue light and nnEMF.




Ref: O: H-O Bond Anomalous Relaxation Resolving Mpemba Paradox

J. R. Errington and P. G. Debenedetti, Relationship between structural order and the anomalies of liquid water, Nature, 409 (2001) 318-321.   (reference 169 from picture in the blog)


Pharmacological treatments for Alzheimer’s disease (AD) have not resulted in desirable clinical efficacy over 100 years. Hydrogen peroxide (H2O2), a reactive and the most stable compound of reactive oxygen species, contributes to oxidative stress in AD patients.  What does elevated H2O2 do to neurons?

IN AD in human more glutamate is released when the environment is loaded with blue light and/or nnEMF. This in turn cascades to deplete Vitamin C.
Where did this concern come from? Brain and eye surgery where light illumination is used.
Concerns have been raised about whether operating microscopes and endo-illumination used during ophthalmic surgeries contribute to retinal damage. Despite the recognition that ascorbic acid (vitamin C) helps to protect the eye from light and the abundance of vitamin C in the retina, artificial aqueous humors used during surgery USUALLY only contain the antioxidant glutathione (GSH below).

Light-induced retinal damage is irreversible and results in permanent visual loss, especially when damage involves the macular area. Ocular tissues are more sensitive and vulnerable to MAN-MADE light than other organs (Sliney, 2002). Although light-induced photoreceptor damage has been well described (Noell et al., 1966Kuwabara and Gorn, 1968), illumination of the ocular fundus by man-made light is inevitable in clinical settings during ocular examinations and surgeries. As a large number of intraocular surgeries, such as corneal and cataract surgeries and vitrectomies, require direct microscopic illumination of ocular tissue, acute retinal damage has been increasingly observed. The damage is thought to result, at least in part, from the effects of this illumination (Arafat et al., 1994Kuhn et al., 1991Michels et al., 1992Kleinmann et al., 2002). What most eye docs fail to realize is this light does not have any UV light in it, yet they continue to blame UV light for damage.  If they used a spectroscope they would see that blue light is the most dominant light in artificial light and is behind the cause of eye damage during surgeries.  Most experiments studying light-induced injury show retinal damage with continuous illumination over several days (Grignolo et al., 1969Malik et al., 1986Edward et al., 1993Organisciak et al., 2003).
Very little UV light penetrates the surface of the globe but all of the blue light from the bulbs we use do penetrate the globe to affect the retina.

Photooxidation of light, in turn then depletes the patient’s eyes of endogenous Vitamin C and it H2O2 directly inhibits formaldehyde dehydrogenase (FDH) in neurons. FDH inactivity and semicarbazide-sensitive amine oxidase (SSAO) disorder resulted in endogenous formaldehyde (FA) accumulation. Unexpectedly, excess FA, in turn, caused acetylcholine (Ach) deficiency by inhibiting choline acetyltransferase (ChAT) activity in vitro and in vivo.

Acetylcholine (ACh) has a crucial role in the peripheral and central nervous systems. The enzyme choline acetyltransferase (ChAT) is responsible for synthesizing ACh from acetyl-CoA and choline in the cytoplasm and the vesicular acetylcholine transporter (VAChT) uptakes the neurotransmitter into synaptic vesicles. Following depolarization, ACh undergoes exocytosis reaching the synaptic cleft, where it can bind its receptors, including muscarinic and nicotinic receptors. ACh present at the synaptic cleft is promptly hydrolyzed by the enzyme acetylcholinesterase (AChE), forming acetate and choline, which is recycled into the presynaptic nerve terminal by the high-affinity choline transporter (CHT1). Cholinergic neurons located in the basal forebrain, including the neurons that form the nucleus basalis of Meynert, are severely lost in Alzheimer’s disease (AD). AD is the most ordinary cause of dementia affecting millions of people worldwide. The hallmarks of the disease are the accumulation of neurofibrillary tangles and amyloid plaques. However, there is no real correlation between levels of cortical plaques and AD-related cognitive impairment.  The plaques correlate with the disease process because a falling mitochondrial redox state links to protein misfolding.  Nevertheless, synaptic loss is the principal correlate of disease progression in AD and loss of cholinergic neurons contributes to memory and attention deficits.

This is why AD brains have low voltage on EEG.  Levels of ACH are linked to voltages on EEG studies.

What else do the eye docs forget to account for during their surgeries?

Free radical scavengers such as glutathione (Winkler and Giblin, 1983), α-tocopherol (vitamin E) (Penn et al., 1987) and ascorbic acid (vitamin C) (Woodford et al., 1983) are thought to serve as components of an endogenous defense system that helps to limit light-induced retinal damage. Vitamin C is also abundant in the human retina, where its concentration in guinea pigs and rats is about 1.6 mM (Heath et al., 1961Woodford et al., 1983). However, intraocular irrigating solutions currently employed during ocular surgery in humans only contain oxidized glutathione as a corneal protectant (Edelhauser et al., 1975Araie, 1986). Importantly, these surgical solutions USUALLY do not contain vitamin C, even though human aqueous humor contains high concentrations of the vitamin.

Does this make any sense?  Could this be why anterior chamber surgery has the side effect profile it does today?

The public does not know these answers because researchers rarely ask these questions in their experiments and never control for the light spectra used in surgery.  How can we learn when we ask the wrong questions in our studies?

How could we measure this if we wanted too?

LDH = Lactate dehydrogenase = lactate biochemistry = the link to Vitamin C physiology of the light stress response.

LDH activity can be measured spectrophotometrically from samples of blood taken every hour during surgery. The activity of LDH can be determined by the rate at which its substrate, pyruvate, is reduced to lactate, as monitored by diminished absorbance of the reduced form of nicotinamide adenine dinucleotide (NADH) at 340 nm at cytochrome 1.   If I can figure it out, why can’t they?

Remember the reduction of lactate implies the additions of electrons to lactate. Guess where the electrons come from in the human retina?

Vitamin C is an electron donor in human biochemistry. This implies that Vitamin C presence or absence allows a stressed cell to choose to use lactate over glucose in metabolism.

The protective effects of vitamin C are not mimicked by glutathione, an agent routinely included in surgical solutions used in eye surgery, because glutathione does not donate electrons as well in the retina (below) in biochemical reactions as Vitamin C does.

It means that ocular surgery using solutions devoid of antioxidants normally found in the retina may be driving diabetic changes in eyes and brains below our perception as surgeons.

How is that for an unintended consequence?

Following extensive light exposure in surgery, it has been found that the level of vitamin C in the retina is diminished (Woodford et al., 1983). Moreover, the fact that diurnal mammals have a normally high concentration of vitamin C in their aqueous humor but nocturnal mammals do not (Koskela et al., 1989) suggests a significant role for vitamin C in protecting diurnal animals from sunlight.  Most eye research is done on nocturnal mammals so this effect magnifies the problem for humans.  It should be clear now that Vitamin C does have a solar component  of action (diunal actions) most never account for in their studies. Interestingly, light-induced retinal damage may be more pronounced in scorbutic than control monkeys (Tso, 1987), again suggesting the importance of vitamin C as a retinal protectant in humans and primates is reenforced.

FACTOID:  What does this imply for 5G and the IoT………..needs that RFID eye chip below to control you. The bar code for the eye is terrestrial sunlight not man made parts of the electromagnetic spectrum used for telecommunications and back lit tech screens all cause these problems in your eyes.

This all points out why blue light and nnEMF are LIKELY causative in the etiology of Alzheimer’s Disease in our modern world.

2G-5G networks create the Fenton free radical in our mitochondria as the pictures in this blog show.

What does this imply for eye surgery and for AD risks longer term?

You’ve got to ask the right questions if you’re going to find the right answers.  On your transition to a Black Swan, you need to ask great questions.  Questions are how you uncover real truths in Nature to learn what you should and unlearn what you must.

Considering the risk of phototoxicity of manmade blue lights via the blue light hazard (above)-, van den Biesen et al. (2000) warned that the safe exposure time of commercial endo-illumination is less than 11 minutes, a time frame that is unrealistic in today’s vitreoretinal surgery. Complicating this is the fact that the detached retina is even more sensitive to light (Zilis et al., 1991). Could this be why these surgeries seem to have a high failure rate and lead to more eye surgery? 

These observations, coupled with the results of the present study, suggest that supplying effective free radical scavengers other than glutathione in surgical solutions may be beneficial for preventing light-induced retinal damage during intraocular surgery, especially vitrectomy, and for maintaining the physiological integrity of the retina for an extended period following surgery. The importance of carefully considering the content of surgical solutions is magnified by the fact that the following vitrectomy the retina is exposed to the ophthalmic solution for 24 hours or more, given the time it takes to replenish the 4.0 ml vitreous cavity with endogenous aqueous humor that is produced at a rate of 3.04 μl/min (Tsukamoto and Larsson, 2004).

Exogenous Vitamin C itself is not appropriate for use in surgical solutions. In the presence of ferrous ions released during bleeding, exogenous vitamin C participates in the Fenton reaction with iron liberated from bleeding (Fisher and Naughton, 2004) and this results in the production of toxic hydroxyl radicals with blue light hazard (435-465nm light).

This thread should be eye opening to any skeptic that blue light or nnEMF can cause disease in man.


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