Productivity measures are destroying patients and doctors

We do not produce anything worthwhile in medicine today. So it raises the interesting question, how does productivity measures relate to being a physician or a patient in 2018?? I do not think they relate to anything worthwhile in any circumstance. This meme was created by consultants from the business world that hospitals have used to usurp power from physicians. It is an apples to oranges comparison. Physicians work with individuals to diagnosis, prevent, treat, and hopefully improve both longevity and quality of life.



When doctors focus on productivity we begin to treat via our Rx pads and hospital algorithms/recipes built by the paradigm to harvest profits and not raise reversals of diseases. Moreover, in the process we’ve stopped educating our patients as we used to do. I changed that when I began blogging ten years ago. In my opinion, this is when we lose our edge our patients get more ill and their visits shorten because we have to trade time for money. Productivity of time leads to a paucity of good care.

According to Wikipedia, “Productivity describes various measures of the efficiency of production. A productivity measure is expressed as the ratio of output to inputs used in a production process, i.e., output per unit of input. Productivity is a crucial factor in production performance of firms and nations.”

Productivity is not a crucial factor to patients or doctors but it is to bean counters who are controlling hospitals and medical decisions in big government.

Hospitals build distractions in now for nurses, doctors, and patients via the electronic medical record. We now talk to screens and not to each other. This also increases the blue light hazard for all involved.

Being in a productive environment comes with its own challenges, such as the exposure of numerous distractions in the healthcare setting. Some of these include the constant influx of messages and emails from the EMR, which will then tempt you to answer them even if it isn’t necessary. Doing so can inhibit your productivity. In these cases, it’s necessary to make your environment conducive to productivity but this can’t always be the solution if your working environment is not in your own control. Today, we’ve lost control of the environment in medicine.



The players in healthcare also have lost much of their self discipline. Unfortunately, productivity doesn’t come naturally to some people because others are innately lacking in self-discipline. Without this trait, it will be challenging to create quality output in the space of time that makes it desirable. This is especially true in medicine. If a patient is not all in on their treatment plan, how good can we expect resutls to be. Conversely, if a doctor is treating people using the wrong ideas because evidence base algorithms’ are wrong how good will patients do in this paradigm? For example, a procrastinator MD may produce good results, but if the patient output can’t be produced within the required timeframe, because the patient has no skin in the game, it can seriously hamper productivity.



Physicians work with individual patients. We should strive to tailor care with our patients, and not some external stimulus. Sometimes that stimulus we bring to the table and it is completely counterproductive to the over success.



Productivity implies that we can count patient units. That idea really disrupts the essential “why” question?

If you are unfamiliar with “why,” I highly recommend Simon Sinek’s book Start With Why. Why did we become physicians? Why have people become patients? Why did they choose us? I think about all these things now that I am a month away from opening up a Center that will focus on quantum biology. I think the answer for most physicians includes helping individual patients. We strive to do our best for each patient, but we need to reassess things and ask, are we really doing it? If not, why not?



Where did productivity measures enter medicine?  It began when I was in medical school in the 1980’s.  Most experts believe that Hsaio’s NEJM article, “Estimating Physicians’ Work for a Resource-Based Relative-Value Scale,” led to RVUs (relative value units) which many practice administrators use to measure “productivity.”  Hsaio, a noted economist, wrote in the abstract of that article:

We found that physicians can rate the relative amount of work of the services within their specialty directly, taking into account all the dimensions of work. Moreover, these ratings are highly reproducible, consistent, and therefore probably valid.

This is where we really went off the rails in healthcare in my opinion.  Why?

However, this model has led to gaming the system, and equating RVUs with hard work or productivity.  But many physicians believe that the RVU system provides many wrong incentives, the most important being that shortening visit time leads to more patients per day and thus more money.  I’d never get paid if I wrote this Rx for every patient because the paradigm has no rewards for prevention as a productivity measure, yet it is what patients all want.



I wish physicians could just ignore RVUs and spend appropriate time with each patient. When physicians try to do this, practice administrators work to get physicians to see patients faster.

This leads to great stress for many physicians, and often unhappy patients. Many physicians believe that shorter visits (especially with primary care physicians) lead to more testing and consultations. Functional medicine believes they solve the dilemma by expanding the visit but ordering massive amounts of tests they do nothing for outcome or productivity. They are making the same errors allopathic medicine has made by replacing testing and supplements for the prescription pad.



Productivity implies that seeing more patients each day is a good thing. But likely most patients and physicians will agree that we need to optimize the time with each patient. How many patients can we comfortably see in one day and deliver high-quality care? High-quality care does not refer to performance measures, but rather complex multi-dimensional factors that improve the patient experience. For many patients, talking about the things that really matter is both therapeutic and diagnostic. When we shorten our conversation time, and focus on the wrong things we raise the risk of diagnostic errors, while increasing health care costs, and create dissatisfied, confused patients. That is where we are now, because we’ve subtracted nature from medicine.



So please join my personal movement to ban technology productivity from medicine. Technology is ruining medicine for both doctors and patients We are not producing anything worthwhile in medicine right now. We need to be caring for patients who need our full attention in this world set up to harm them from technology and an indoor existence.




How?  It occurs via volitile compounds released from our apocrine glands.

An odor is a volatile chemical compound that humans and other animals perceive via the sense of smell or olfaction.  It turns out our ears are expert on making our noses perk up.  This is how senses work coherently.   Odors are also known as aromas or fragrances and (if they are unpleasant) as reeks, stenches, and stinks. The type of molecule that produces an odor is called an aroma compound or an odorant. These compounds are small, with molecular weights less than 300 Daltons, and are readily dispersed in air due to their high vapor pressure.

The sense of smell can detect odors are extremely low concentrations.

So what makes these volitile chemicals in our ears?  EARWAX does.

Ear wax is not what it seems from a quantum perspective.  It is similar to eyelashes and nose hair, your earwax is a protective device that is often under-appreciated.  It protects your special epithelium and hair cells in your ear that are electromagnetic antennas.  People with little ear wax tend to have more environmental exposure to nnEMF.

Ear wax is not wax.  It is exfoliated skin and it operates using the Auger effect to protect us from electromagnetic waves.  This cerumen substance is a combination of skin cells that have fallen off from inside of the ear, bits of hair and secretions from the ceruminous glands in the outer ear canal.

Did you know that earwax has a circadian basis tied to latitude and mitochondrial biology?  Most people are ignorant of these things.  At Kruse Longevity Center we make it our business to know these things because we begin where allopathic and functional medicine end in their understanding of human biology.

There are two different types of earwax: wet and dry.

A. Dry earwax is usually crumbly and lighter in color from tan to light grey. People that have North-Eastern Asian and Native American ancestry often have this type of earwax.

B. Wet earwax is sticky and yellow to brown in color and can even have an odor. This earwax is normally found in people that are more likely to have European or African ancestry.

The primary purpose of earwax is to protect your ear canal and eardrum from foreign materials and foreign electromagnetic waves.  Whales are mammals that have an exquisite hearing mechanism that uses cerumen because they use echolocation. If I told you we can use ear wax to assess patients for pregnenolone steal syndrome and their blue light hazard would you believe it?  Well, you better, because it can.  And that means there are many more aspects it can tell a Black Swan clinician.

Stress or anxiety can actually increase your earwax production. The glands in the ear that assist secreting wax are a class of glands called the apocrine glands. The human female breast is an apocrine gland.  These glands are the same glands that are responsible for your apocrine glands to create sweat.  This is why a return of sweat creation is part of the Leptin Rx.  They have a larger array of mitochondria that create water and also create your pheromones.  They can create massive odors both good and bad and these act as signals to other animals about your suitability for mating and immune function.

Stressful stimuli activate your PVN in your brainstem and they can make you sweat more (and smell worse to you and your network), stress and other emotional responses (like fear) can also increase your earwax production.  This is why the paper below is not a joke………it is another tool for the Back swan to use in the raod to optimal.




New podcast out for the Thanksgiving week for you to enjoy. In this podcast we go over some classic ideas about how food is an electromagnetic blueprint and we discuss the details linked to freed Vitamin A and how it destroys photoreceptors.

Why is colon cancer exploding in a blue light world?

Why does our skin bleach? Is it tied to hair bleach ladies use for their hair?

Catalase is one of the photoreceptors that Vitamin A destroys in Vitiligo. Artificial blue light and nnEMF cause this stimulus in this autoimmune diseases. We also talk about a lady who is a lawyer in Boston and how her job gives her melasma.

Blue light stimulates melanocytes in humans. Could this be why people are getting ocular melanoma now?

What are the collateral effects of these effects for women now?

In this podcast I lay it out for ladies.

Why do we get fat and depressed?

Why is make up bad?

Is clothing on the breasts a problem too?

Is the AM sun the key to mitochondrial health?

Why are women the key change agents in health?

Why did UV light get a bad reputation in medicine?

Does sunlight really cause skin cancer or protect us from it?




Where do humans live and work in the modern world?  Is it in the sun or not?  Do you use sunglasses?  Then you need to understand the “orange tree” analogy.

Does light dictate microbiome changes or is it food?





People who have uncoupled haplotypes who can use both sunlight and cold to drive their mitochondrial biology will be better adapted for the 5G world that is now being unleashed on us.  This is a very counterintuitive implication of the ideas shared in this interview.  This means people with darker skin and eyes who have uniform levels of melanin, with L0,L1,L2 mitochondrial engines might need to consider inside the Tropic of Cancer and Capricorn to best protect themselves from the Internet of Things because they will be markedly affected by unusual diseases electromechanical hypersensitivity and disorders of their immune system.

Moreover, the use of sunglasses, sunscreens, clothing, and makeup will stress these individuals more leading to second generation mitochondrial diseases that right now remain uncommon.

What are some examples of this predictions?

Lyme, Chronic fatigue, EHS, autoimmunity, ALS, autism, childhood eye and skin diseases, fertility issues, cancer of the RPE in the eye, disorders of the gut will become common.  When these things become more common we should also expect to see more osteopenia and osteoporosis in these people too because as the 5G networks will lower RBC Vitamin C.

Very few see what the Black Swan sees because they do not have a proper perspective of how a quickly changing environment will radically alter how our colony of mitochondria respond.

When vitamin C is lowered by the collateral effects of 5G, bone cells that degrade bone called octeoclasts proliferate, and bone cells that lay down mineral and new bone called osteoblasts are not formed.  The two atoms that allow calcium and apatite to stay bound to bone colllagen lose their electrostatic interactions and this is the main reason why I expect decalcification of bones to be a marker for a 5G environment.  At the same time, these people will quickly calcify their arteries and their production of nitric oxide will drop dramatically cause more eye and skin diseases.

Eating more Vitamin C will not do a thing to help this problem because of the unique perspective I gave you in the November 2018 webinar in the topic.  The oral saturation effect of Vitamin C cannot overcome a bad environment.

Vitamin C does several non-linear things to improve bone physiology

A. It mineralizes the bone and stimulates bone forming cells to grow.

B. Dampens oxidative stress in the colony of mitochondria in your bone

C. Prevents too much degradation of bone by inhibiting bone absorbing cells.

D. Is vital in the unique collagen in bone that allows for bone synthesis

These things all begin with the quantum biologic effects that occur on the eye and skin.  I just came home from Mexico speaking to 250 eye doctors about the collateral effects of these things.  This podcasts deepens the thesis I am laying out here at Patreon.



My Black Swan mitochondriacs who see this film may see evidence of something else on this movie besides a WBC cell hunting down a bacteria.  Anyone want to venture a guess what the author of the tweet missed given my lectures on Patreon?

Post your guesses below.  Have an awesome weekend.

Movie credit: David Rogers. #CellBiology



DNA only codes for proteins. When you think about this for a moment, it is quite odd. Life just does not use proteins to get the job of living done. Why don’t we code for other things? Is there a deep reason for this that we missed in basic biology?

Divergent thinking is the center of human creativity and most people believe we are born with it but I do not believe this. I believe we can teach people how to think more broadly by learning to examine things we do not believe or accept to see if they have merit or not. Divergent thinking can be acquired and developed skill when you get “skin in the game”. I now excuse people who sense life with tiny thinking. This brand of expansive thinking is a distinct form of higher-order thinking and it can be taught to all ages of autodidacts. “Divergents” always give themselves permission to see things differently than crowds. Misunderstanding vanish when you train yourself to ask, ‘What else might this imply’?



Glycine helps us break symmetry in nature by being the ONLY symmetric amino acid.  That idea sounds counterintuitive until you understand what nature is up too.  She hitches all of her chromophores that absorb some parts of the visible spectrum and connects them to proteins that act as tuning forks because the light waves the chromophore absorbs turns into an electromechanical wave of vibration in the protein portion of the photoreceptor.  All amino acid residues, other than glycine, has no symmetry elements.  So when they are strung together via the peptide bond they can never line up perfectly.  They must have some type of staggered alignment.  This topology makes them ideal to turn light waves into vibrational tuning forks using resonance.  Melanopsin is loaded with helical proteins and its chromophore appears to be the Vitamin A that is bound to it.  Vitamin A absorbs light and makes these helices vibrate in the eye so we know when night falls.

The general entanglement of one residue of a single chain into a second residue equivalent is done to satisfy a light waves requirement of a staggering molecule because light’s electric and magnetic components are orthogonal to one another.  Since amino acids are inherently asymmetric to one another, an order begins to appear from chaos.  Accordingly, this nanoscopic atomic arrangement dictates a rotational axis must exist in proteins.  This makes them obtain interesting solid state properties.  The protein axis builds a screw-like pitch angle by translation along the molecules axis.  Since light is only absorbed by the electrons in the chromophore the energy and information in light can be transmuted to other forms of energy without any loss as long as this energy is not thermalized back to the environment.  This is what makes photonic energy transfer appear to operate in the classic world like magic to us.  It is magic, but it is quantum biologic magic at work based upon the arrangement of atoms in precision with the 90-degree angle of a lightwave to make an oscillation possible.



When you realize what Mother nature is really up to with DNA, you begin to realize she codes for proteins that only operate with vibrational modes within the visible spectrum of light. She abhors things that make her proteins vibrate out of tune. Hence the only configurations for a protein chain compatible with what we call life are that the residues must form helical configurations. This is why all proteins that vibrate are helical. It also explains why DNA assumes this shape too.

It appears Mother Nature selected amino acids that can build corkscrew proteins that work with the corksrew light waves in terrestrial sunlight. This appears to be an exclusive relationship and explains why light outside or isolated from the solar spectrum cause signaling problems and interference in the system’s fidelity. Light is captured by electrons and we do a lot of things with its energy and information quanta before we let it fall back to the ground state. That is life’s major magic trick.



Watch the video now. The gap between learning and knowledge is called procrastination. Procrastination delays our success. This video shows you the way I traveled 15 years ago. the picture below shows you how light screws into the protein threads of photoreceptors. You need to get on the same page with me.





Television and all technology screens are capable of making you hungry because they induce leptin resistance via melanopsin dysfunction —  and it’s not only all those Pizza Hut commercials!

People are unaware of how the new melanopsin data allows this process to occur.  When blue light or nnEMF disrupt the WEAK covalent bond between melanopsin and retinol, retinol because free from its normal tight circadian control.  When it is freed of these shackles it destroys ALL mammalian photoreceptors because Vitamin A is the ONLY Vitamin on Earth that emits light.  The light it emits changes the optical signaling of the local environment and this changes how chemicals in the cell at this location oscilate.  The oscillatory pattern is tightly controlled by nature to create the precise signal that is needed to make life and wellness possible.   it always creates a stimulus around photoreceptor light centers that create abnormal signaling.

The emission of light has to be tightly controlled in all biologic reactions.  It turns out freed retinal is the secret in life of how chaos is really controlled in cells.  Free retinal is the other side of the optical story of how leptin resistance gets humans ill.

BLUE LIGHT HAZARD = melanopsin dissociates from retinal and free retinal destroys photoreceptors = destroys optical signaling.   The lower your redox is the more retinal is released.  Here is the the BLH and frequencies laid out.





When you look at the table something remarkable about the sun should appear to you. As we approach the UVA and red frequencies in both directions the BLH drops quickly. We also now know that the BLH correlates with the free radicals made in mitochondria too.

Certain electromagnetic stimuli are more apt to release retinal in this way because of the weak covalent bond beside frequencies in the visible spectrum of light because of how electromagnetic waves affect bonds differently. This is why the microwave bands in 1G-5G pose a great threat to the retinal mechanism behind leptin resistance.



Blue light is the one that is now best identified in the literature.  Now that we know the human bond between melanopsin is a weak covalent bond we know that 1G-5G waveforms can also separate them even more easily via rotation.

Recent data in 2015 show that the Schiff base linking the chromophore retinal to the fractal melanopsin protein is more susceptive to spontaneous cleavage in mammalian melanopsins. In fact, the researchers went out of their way to note that this stability is highly diversified between mammalian species, being particularly unstable for human melanopsin.

This raises a big point.  If the bond is unstable in humans what might this mean for free retinal in tissues.  Is this how leptin resistance begins in tissues?

I think it is.  Why do I say it?

This pool of the retinal-free melanopsin molecules would effectively decrease the number of photoreceptive molecules in ipRGCs. If that was true wouldn’t melatonin levels also drop as a result?

What could cause the presence of the free retinal most frequently in modern life? One possibility is a shortage of retinal supply from retinal-producing enzymes or could it be stimuli from the electromagnetic spectrum that do it?  People forget that terrestrial sunlight on Earth does not equal the entire light spectrum in the universe.  Melanopsin was innovated by evolution based upon the light that fell to Earth from the sun as the picture below shows.  Not even the sunlight that astronauts face in orbit in the ISS is equivalent to the sun we get on the tectonic plates on Earth.  This is why they get diseases that were once rare on Earth.  Today, children are getting the same type of diseases as astronauts because of how man has usurped the electromagnetic spectrum for technology use.

Technology use and abuse cause the capricious cohabitation of the human’s mind. Nature’s harmony makes small things grow. Lack of it makes big things decay. Technology ruins man’s biologic coherence and induces degeneration of the mind/body in ways most do not appreciate.  This is why I created the twitter hashtag #mitochondriacwisdom.  



Does this coning feature of sunlight have implications for the melanopsin system in humans?   Alternatively, could the change in bonding be due to constant retinal release from the protein via cleavage of covalent bond (Schiff base linkage) with retinal (Vitamin A) by light? The latter possibility would be similar to what occurs in cone photoreceptor cells in the eye, where retinal is known to spontaneously dissociate from visual pigments, resulting in reduced overall cellular photosensitivity by destroying photoreceptors as the link above shows.

They found this via mutagenesis analyses, that this diversified stability is mainly due to parallel amino acid substitutions in extracellular regions. In the 2015 paper they proposed that the “differential stability” of the retinal attachment to the melanopsin fractal chromophore likely contribute to functional tuning of non-visual photoreception in mammals.  This means that rotational changes in the covalent bond alter its optical signaling ability.  This implies the fractal protein changes and this means it change how it can react.  This is precisely how the butterfly effect occurs when the chaos of changes appears to come from an order system but yield an wildly unpredictable result.  This was eloquently laid out in Jim El Khalili’s documentary   This is why all Black swans need to be really concerned with technology and artificial light use.  The 2015 paper is warning us that topologic effects in retinal is enough to cause optical damage to photoreceptors it is bound too.   This fully explains why blue light has been causing humans problems since we invented the light bulb in 1874.



Carbon atoms in single bonds rotate freely in organic chemistry. Rotation can occur because the heaviest electron density in the σ bond exists along an imaginary line between two carbon nuclei. Rotation does not change this electron distribution; the bond strength remains constant throughout rotation. Because rotation is possible, the molecule can have an infinite number of conformations. Conformations = size and shapes = changes the way matter operates thermodynamically. All of our proteins were designed by nature to work only in terrestrial sunlight.

With manmade light the bonds react differently and they change the conditions of existence of the molecules in us. It turns out blue light and nnEMF release Vitamin A from from the carbons in melanopsin and that freed Vitamin destroys all the chromophores that our proteins contain. the picture below shows many pictures of those varied chromophores in us. When they are damaged, they cannot absorb light to make the protein they are connected to vibrate properly in cell water. This ruins the resultant vibration and coherence. This is leads to disease. Leptin is one such chromophore found in your subcutaneous fat. Once it becomes defective it cannot send its optical information to the hypothalamus and you get ill as a result.



We’ve believed that melanopsin was only present in the eye since its discovery in humans since 1998. We then discovered it in human blood vessels in 2014.



Then, in December 2017 we got the shock data it was also in our skin and subcutaneous fat helping explain why nature put leptin, another photoreceptor molecule, in our subcutaneous fat.



Leptin is designed to take optical data from the skin and skin arteriole surface about day and night and couple that with energy balance information and deliver it to the hypothalamus under the cover of darkness. Free retinol from surface light at the wrong time of the day is what ruins this hormones behavior optically. Once leptin signaling is disrupted by circadian mechanisms, the hypothalamus loses control of all growth and metabolism inputs. This leads to many chronic human maladies such as obesity, diabetes, and metabolic syndrome.

This is how I solved my obesity issue 15 years ago when I was 360 pounds. I realized that when leptin was altered by alien light in my environment it changed the the atomic structure of leptin. This small change lead to massive changes in my body mass. The reason made sense. Mitochondria create heat, which is infrared light. We need this light to activate the DDW our mitochondria make. What happens if the mitochondria in me were being destroyed by light in my operating room? What do the laws of physics tell us about heat and size? small objects in nature have a different surface area compared to their volume inside. This means that smaller people lost heat quicker. This is true in planets as it is in people. This is why Mars magnetic field from its interior dynamo died out sooner than Earth. It is smaller, so it loses heat faster. Humans who have mitochondrial damage because of the damage in their heme proteins in their cytochromes would lose a ton of heat if this would be allowed to continue. What does the body do to offset the loss? It makes you fatter. Why? This changes the relationship of the surface area to your interiot volume and improves your thermodynamics while the engines inside of you get worse by the retinal damage of leptin and your cytochrome proteins. This is also why mammals fatten in winter when the sun power varies and you lose more heat to the environment as the temperature around begins to fall. Eating carbohydrates in a falling solar environment fatten you. The cold causes you to use that fat to warm yoruself until the sun comes back in spring. That is how the forces of nature work inside of you when you are leptin resistant. When I realized this for the first time my life changed forever. This was the answer to my obesity crisis.



Mitochondrial photoreceptor damage are all defects in optical signaling caused by Vitamin A’s ability to destroy photoreceptors.   This is why the the authors in the article make this statement, about blue light, ”It’s toxic. If you shine blue light on retina, the retinal kills photoreceptor cells as the signaling molecule on the membrane dissolves.”    

That is a definitive unequivocal statement made in this paper.


What aggravates this effect in modern humans? The human choice to live indoors with blue light and nnEMF.

Your TV is likely the greatest blue light emitter in your house after Obama changed TV signals from analog to digital in 2009.  But every tech device was digital the day it was created.  The sheer number of both devices is what is accelerating modern humans diseases now.  Nobody sees where the pieces fit until they do.

Blue light, via the photoreceptor melanopsin, disrupts autophagy and mitophagy, as I laid out in my Vermont 2018 video.  This, in turn down-regulate apoptosis (programmed cell death) in cells. Thereby, defective cells, running at a net energy loss, send out a call for more food via the defective leptin photoreceptor.  That defect is cause by free retinol in the tissue and in the blood stream that is not circadian controlled by retinol binding protein — Leptin is loaded with these aromatic rings and the light emitted by the Vitamin A alters the quantum HUMO-LUMO rings and this is what blocks leptin from going from your subcutaneous fat around midnight when it is supposed to be dark to enter the hypothalamus to deliver this photonic and electronic data to this part of the brain.  Without the proper message energy balance and metabolism is lost.  That is what leptin resistance is at the quantum level.  Because of the melanopsin dysfunction, the information and energy transfer the hypothalamus requires can never be satisfied in the mitochondrial matrix of the cells affected by this optical deficits due to their defects having escaped the recycling autophagy program built into cells.  That cycle is controlled by the circadian mechanism which Vitamin A has hijacked.  The antidote to blue light in nature,  is 42% of the red light in sun.  It is augmented by UVA and UVB light.  These parts of the solar spectrum strengthen mitophagy and apoptosis to return physiologic law and order back the the defective tissues.   We would be much better off watching the sunrise and the sunset than watching TV, using a computer, or talking/texting constantly on a cell phone,  especially at night. We’d be more wise to use a geothermal pool, or build a green house, or stare at the flames of a campfire…to stimulate the healing powers inside your colony of mitochondria.  This is the credo of the black swan.  This explains fully how I see Leptin resistance develops in humans.


Leptin resistance is a photonic process in human biology. So what does blue light and nnEMF lead to give what we’ve learned about melanopsin/retinal links? It ruins the Bazan effect to ruin the long loop to cause liver level leptin resistance. This blocks DHA to be replaced in cell membranes in the liver and CNS/PNS. This causes many communication and memory issues via a broken circadian mechanism via the eye and skin. The pic is about the eye but it was created before you knew melanopsin/retinal was in the subcutaneous fat and skin arterioles. See the pic below = Leptin resistance at liver level = lowered global DHA in liver cell membranes that induce PPARγ-target catalase expression and reduce ROS levels, leading to the inhibition of JAK2/STAT3 = what leptin resistance is inside a cell below the pathway level of Ph.D. or MD understanding.




The predictions I made 5 years ago about blue light and nnEMF will now be tested on humans. If you live in California pay attention to the news now in your local area. Why? I think the first people who will know something is amiss in our environments are ER physicians and ICU nurses and hospitalist doctors who begin to see infections that devolve into sepsis rather quickly without much warning. 
Sepsis mortality is remarkably high and likely will skyrocket in a blue lit 5G world because of how the immune system is controlled by Vitamin A.  This disease process will be very pronounced complication in American cities with 5G.  The onset will be acute and it maybe teethered to other normally inconsequential diseases.  The situation in these cases will turn dire quickly. The latest surviving sepsis campaign guidelines in 2018 has told clinicians that sepsis related mortality is around 15% while septic shock is associated with 40% in-hospital mortality and in poor countries, the mortality goes even higher up to 60%.  I expect that number to flip in a 5G city.  Why?  Hospitals are loaded with light and technology that increase the liberation of Vitamin A from melanopsin.  In those hospitals more technology is used in ICU’s and operating rooms so length of stay and length of surgery will be key indicators of who is at risk.  When you layer this with redox status of the patient pre-sepsis it will become obvious who that at risk people will be to clinicians eventually when they begin to understand the links to why this is happening.
Thousands of interventions have been tried over decades failed to improve sepsis survival in healthcare. Even drugs that were able to reduce mortality in one study, failed to do so in another study.
Moreover, my expectation is that in these patients, clinicians will find that most antibiotics, antivirals, and antifungal medications will be impotent for the patients situation.
For the observant family member or healthcare professional this should be a sign to you that the underlying cause  of the infective process might not be what the blood cultures reveal, it maybe the action of free retinal in the tissues causing destruction of optical signaling in the immune systems cells.
It will appear to the nurses who are taking care of your family that the drugs normally used are having no effect.  At the same time there will be tell tale signs that melanopsin dysfunction is the real cause of the septic state. 
1.  Quick onset of decline
2. Extremely low Vitamin D level
3. Very abnormal BUN/CREATINE ratios
4. Very abnormal hormone panels.  These will never be drawn in a hospital setting until physicians learn why it is happening.  Vitamin A and T3 are cofactors that convert cholesterol to most of the hormones under the pwer of the visible spectrum of TERRESTRIAL sunlight.  If Vitamin A running wild no sex steroid hormones can be made.  DHEA-S will usually be low.
5. Procalcitonin will skyrocket in these cases.  This protein is a biomarker that exhibits greater specificity than other proinflammatory markers (eg, cytokines from melanopsin dysfunction).  It will be one of the best tests in identifying this acute destructive Vitamin A pathology in patients with sepsis that seem to confound the physicians at the bedside.  I believe it can and will be used in the diagnosis of these technology induced infections.
6. B12, thiamine, Vitamin C and folate levels will be extremely low because glucose metabolism is all that the matrix has left in acute mitochondrial poisoning.  That effectively is what melanopsin dysfunction is. 
7. Anemia onset will be rapid and the RBC count will have changes in the RDW and MCV/MCHC.  Hemolytic anemia will be more common than we see in more chronic Warburg shifted diseases. 
8. Rapid onset of capillary and vascular damage will occur seemingly out of no where because Vitamin C formation from glucose goes to zero because Kreb’s bicycle cannot operate any longer in these states.  The timescale of the decline helps explain the effects.
The 5G cocktail that will show remarkable benefit within 24-48 hours will be an intravenous mixture of vitamin C, thiamine and stress dose hydrocortisone.
Rivers of ink have been spillied in the critical care literature on sepsis.  the same is true about oxygen delivery and fluid responsiveness as key therapies in these cases.  5G sepsis will require even faster metabolic resusitation because of how Vitamin A destroys photoreceptors so rapidly that decipher optical comminications in metabolic pathways.  Heme is the key chromophore to understand.  Heme proteins are one of the oldest photoreceptors in all living things.
Restoring oxygneation and intravascular volumes are clearly important in all cases of sepsis but the rapidity of mitochondrial destruction and redox power is the key to understanding why the treatment of 5G sepsis HAS to be thought of differently.  Acute critical care teaching has cause physicians and nurses to focus on easily observable phenomena at the bedside in these cases, but that focus has led healthcare professional to ignore something of greater importance: metabolic resuscitation of acutely dying colonies of mitochondria in a patient with nnEMF toxicity.  Effectively, that is what 5G sepsis looks like and is.  It presents as acute rapid mitochondrial failure in organs.  The critical care team has to learn the hard lesson that their treatments can deliver all the oxygen we want to the hypoxic tissues, but if the colonies of mitochondria are failing in the tissues, it just won’t work.
The early use of Vitamin C will protect blood vessels and RBCs from melanopsin damage and acute proton dislocation in the mitochondrial matrix by UCP2 dysfunction and to improve their ability of RBC’s which will acutely lose their Vitamin C levels in melanopsin dysfunction due to their photoreceptor (heme and aromatic amino acids) damage which alters the ability of RBC’s to deform their size and shape to allow them to navigate damaged vascular beds to improve oxygenation.
Sepsis from electromagnetic pollution is a state of acute mitochondrial failure on a large scale in many organs and it will be linked to many stressors of the PVN.
Sepsis in one of the clinical situation we see in humans where glucose based metabolism exists exclusively.  This is not a common situation.  Beta-oxidation and the urea cycle are damage severely in this case.  The damage is greater than we see in cancer.
In humans, Vitamin C is created from glucose shunting.  When glucose is the only fuel you can use to remain alive, it is not too difficult to see why Vitmain C drops like a lead baloon.   In animals that synthesize vitamin C natural (not humans), synthesis is normally downregulated exactly in fasting or low-carbohydrate conditions, or when glycogen is otherwise low.  Notably, this is not the case in hibernation, where the reverse is true.
Sepsis is a unique clinical situation in humans.  Humans do not need exogenous Vitamin C much at all because they are omnivores and can live off of fat and proteins without any carbohydrates at all.  This is what separates them on the primate tree, but this has massive implications for a 5G world.  Why?
Blue light and nnEMF environments increase glucose in the blood by increasing the AMPk pathways (Volkow and Frey).  This means under nnEMF humans have to use glucose and cannot burn fat or protein well.  
What are the implications of this?  
Since the literature clearly shows that vitamin C synthesis is downregulated when food or carbohydrates are low suggests the following possibilities for humans in an altered EMF environment. First, it suggests that vitamin C might be more necessary in a glucose based metabolism than in a ketotic one.Sepsis is one of those situations.
Second, it suggests that there are compensatory mechanisms that come into play when vitamin C is low that are also triggered by low-carbohydrate conditions, and therefore, vitamin C requirements are lower in low-carbohydrate conditions.  People who crave carbohydrates at home are sure to have melanopsin damage somewhere in their system when you understand the clinicial implications of this blog.   This means that people who are 100% carnivorous may never need vitamin C because their matrix has very little chance of being destroyed by the deuterium in carbohydrates.  UCP-2  is another chromophore that can be destroyed by free Vitamin A to let deuterium into Kreb’s bicycle.
Third, it suggests that high levels of vitamin C, when a 100% carnivore,  might be quite detrimental under low-carbohydrate conditions.  I can tell you I have seen this effect in functional medicine patients who were given Meyer’s cocktails and got violently ill when the prescriber did not understand that the person was very ketogenic to begin with on a nutritional basis.
What is the point of this dance, Jack?
When your mitochondria is running on solely on glucose, Vitamin C become critically important in the kinetic flux of protons in the matrix.  This is the context non-Black Swan clinicians RARELY comprehend.  The more blue light and 5G tech you use, the more Vitamin C your system needs to remain well.  This is why sepsis is important to understand.  In a septic state, from melanopsin dysfunction, you will be OBLIGATE user of vitamin C.
In 1975, this was shown in a paper by Newton and Mann.  They showed that diabetics appeared to mimic a localize chronic form of scurvy.
Why is this data important especially because it was in diabetics?  Diabetes is destruction of heme in the cytochrome proteins of the matrix?  Is shows you biochemistry is completely light dependent.
What happens on the surface determines how it can operate in cells below.  It appears our essential micronutrients are very dynamic in different light environments in reference to nnEMF and retinal. Your skin’s intereaction with incident light is involved in the biochemical reactions happening inside of your matrix all the time.  You have no ability to control this process.  It seems Mother Nature set the system up like this for a reason (Quantum Zeno effect).
How much of ANY micronutrient is linked to the incident EMF your skin and subcutaneous fat senses from your environment.  Since your skin is the largest organ in your body, and melanopsin/retinal are in your skin coupled to leptin, this begin to shows us how metabolism is controlled by the light we live under.  We have the ability to alter our needs significantly depending on the light your surfaces sense.  This is why this slide showed up in the Vermont 2018 talk.  
Vitamin C can be spared by something that takes over one of its functions, or by something that increases its effectiveness.  Iodine is one such substance for humans via the Grotthuss mechanism, for example.
Human cells are expert in creating glutathione from cysteine if redox power is decent.  In sepsis it is not.  Therefore, in this situation,  glutathione offers no Vitamin C buffering because sepsis is a situation when your entire colony is failing.  Mitochondrial functioning has to be half way decent to get this effect.
Ketosis helps create glutathione in humans but it requires an intact redox state.
Sepsis is a condition where humans face the worse state of insulin resistance one could fathom.  In this state, Vitamin C can save their life when it is threatened.  It turns out, Linus Pauling might have been correct about Vitamin C in a blue-lit nnEMF world we’ve created.  He was clearly wrong in the world he lived in…….dominated by the sun.  Why?  because sunlight allows the matrix to make water and CO2 easily because it restores redox power.  Today that is no longer true.  Blue light and nnEMF do the opposite.  It looks like his hypothesis is now the way of all Black Swan’s in training.
I have a sense when 5G hits big time we might have to add uric acid and glutathione and iodine to the sepsis cocktail for survival.  Why uric acid?
Humans and other  primates share is a loss of function of the uricase enzyme. Uricase breaks down uric acid, and the result of this mutation is higher uric acid levels in primates. There is some decent data this helped the apes live longer.  It seems evolution used this mutation like she used the loss of vitamin C synthesis in primates, for a selective advantage.  I think that advantage was tied to the loss of hair on man compared to apes.  This allowed their skin to sense more incoming UV light. Hair is an light antenna but too much of it would have limited the UV input at the surface of the skin.
I think apes lost Vitamin C first, then uricase, before humans replaced both with iodine from the marine chain and made the point moot.  In sepsis, this evolutionary dance might be the difference between life and death in a 5G city in a hospital ICU.    
Many people do not know that Vitamin C is a co-factor in the formation of catecholamines and CORTISOL and those chemical all have aromatic amino acids with photon traps inside them.  This makes them targets for retinal knockout.   If you have a low dopamine state……….pay attention right now.  
Intestinal absorption of vitamin C is saturable.  This means taking it orally won’t work!!!  Given the increased metabolic consumption of Vitamin C in critical illness like a 5G sepsis, the only way to replete Vitamin C in this context is intravenously.  The effect of Vitamin C on mitochondrial damage is also born out in what Tanaka found out in 2000 in patients with sepsis.  When IV Vitamin C is used patients needs less IVF’s.  The reason is simple but Tanaka and critical care professionals still do not understand the reason.  Mitochondria end product is water at cytochrome 4.   Redox power at NADH needs to be around -400mV.  Glutathione helps keep this there.  DDW is what can help this situation in 5G blue light toxic states. 
This restores metabolic balance of Kreb’s bicycle.  When this happens the voltage drop in mitochondria from NADH to oxygen is off a cliff and it happens fast.  This is how 5G will discharge our batteries.
Tanaka found patients in the vitamin C group required less fluid resuscitation, had higher urine output, and developed less wound edema.  That is what I call evidence of matrix salvage by improving the proton problem in the matrix by nnEMF.
Thiamine will be found to be very helpful in those with severe lactic acidosis from the mitochondrial damage of free running Vitamin A.  Vitamin A will attack and destroy the heme proteins in the cytochromes just as it did in the circulatory system.
Acute thiamine deficiency decreases the pyruvate flux to the Krebs bicycle so it increases the production of lactate by altering the aerobic metabolism as the pic above shows.  This is the key sign of the fastest Warburg shift a clinician will ever see in their career.  Cells with acute lactic acidosis become unable to use the TCA or urea cycle for any metabolic functions and this is why; Krebs bicycle is demoloshed by all the heme damage in the cytochromes.
The stress dose of hydrocortisone will be important in preventing severe acute adrenal insufficiency because of the acute onset of the pregnenolone steal syndrome and to support rapidly dropping coritsol levels to maintain life.  Cortisol in this case, is the critical hormone that 5G will zap quickly.  Vitamin C also reverses the oxidation of the glucocorticoid receptors so the steroids work.  This is why Vitamin C has to be given with the correct meds in sequence.
The effectiveness of this therapy will be measured best by the PCT clearance.  PCT clearance is measured by the initial PCT drawn in the ER and then we subtract the PCT values at 72 hours divided by the initial PCT value and then we multiply this by 100 to give us a percentage.  
Infections caused by Vitamin A dysfunction from melanopsin dysfunction will be called drug-resistant cases by hospital personal.  Pay attention to these catch phrases.
In these cases, it won’t be that the drug does not work……it will because the mechanism of action is light-based from the liberation of Vitamin A on all photoreceptors and the antibiotic cannot stop the release of retinol from melanopsin fast enough in the skin due to how 5G interacts with the skin’s topology. Just watch and see if I am correct now.
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18.  (repeat of Mann and Newton)