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THE “CITIZEN CANE”…..
……OF MICROSCOPY
I’m teaching 1st-year grad students about cell migration tomorrow. This is the first movie they will see. An immune cell hunting down a bacterium among red blood cells. Movie credit: David Rogers. #CellBiology pic.twitter.com/qk75C8JFom

— Dylan Burnette (@MAG2ART) November 9, 2018

My Black Swan mitochondriacs who see this film may see evidence of something else on this movie besides a WBC cell hunting down a bacteria.  Anyone want to venture a guess what the author of the tweet missed given my lectures on Patreon?

Post your guesses below.  Have an awesome weekend.

Movie credit: David Rogers. #CellBiology pic.twitter.com/qk75C8JFom

DNA only codes for proteins. When you think about this for a moment, it is quite odd. Life just does not use proteins to get the job of living done. Why don’t we code for other things? Is there a deep reason for this that we missed in basic biology?

Divergent thinking is the center of human creativity and most people believe we are born with it but I do not believe this. I believe we can teach people how to think more broadly by learning to examine things we do not believe or accept to see if they have merit or not. Divergent thinking can be acquired and developed skill when you get “skin in the game”. I now excuse people who sense life with tiny thinking. This brand of expansive thinking is a distinct form of higher-order thinking and it can be taught to all ages of autodidacts. “Divergents” always give themselves permission to see things differently than crowds. Misunderstanding vanish when you train yourself to ask, ‘What else might this imply’?

 

 

Glycine helps us break symmetry in nature by being the ONLY symmetric amino acid.  That idea sounds counterintuitive until you understand what nature is up too.  She hitches all of her chromophores that absorb some parts of the visible spectrum and connects them to proteins that act as tuning forks because the light waves the chromophore absorbs turns into an electromechanical wave of vibration in the protein portion of the photoreceptor.  All amino acid residues, other than glycine, has no symmetry elements.  So when they are strung together via the peptide bond they can never line up perfectly.  They must have some type of staggered alignment.  This topology makes them ideal to turn light waves into vibrational tuning forks using resonance.  Melanopsin is loaded with helical proteins and its chromophore appears to be the Vitamin A that is bound to it.  Vitamin A absorbs light and makes these helices vibrate in the eye so we know when night falls.

The general entanglement of one residue of a single chain into a second residue equivalent is done to satisfy a light waves requirement of a staggering molecule because light’s electric and magnetic components are orthogonal to one another.  Since amino acids are inherently asymmetric to one another, an order begins to appear from chaos.  Accordingly, this nanoscopic atomic arrangement dictates a rotational axis must exist in proteins.  This makes them obtain interesting solid state properties.  The protein axis builds a screw-like pitch angle by translation along the molecules axis.  Since light is only absorbed by the electrons in the chromophore the energy and information in light can be transmuted to other forms of energy without any loss as long as this energy is not thermalized back to the environment.  This is what makes photonic energy transfer appear to operate in the classic world like magic to us.  It is magic, but it is quantum biologic magic at work based upon the arrangement of atoms in precision with the 90-degree angle of a lightwave to make an oscillation possible.

 

 

When you realize what Mother nature is really up to with DNA, you begin to realize she codes for proteins that only operate with vibrational modes within the visible spectrum of light. She abhors things that make her proteins vibrate out of tune. Hence the only configurations for a protein chain compatible with what we call life are that the residues must form helical configurations. This is why all proteins that vibrate are helical. It also explains why DNA assumes this shape too.

It appears Mother Nature selected amino acids that can build corkscrew proteins that work with the corksrew light waves in terrestrial sunlight. This appears to be an exclusive relationship and explains why light outside or isolated from the solar spectrum cause signaling problems and interference in the system’s fidelity. Light is captured by electrons and we do a lot of things with its energy and information quanta before we let it fall back to the ground state. That is life’s major magic trick.

 

 

Watch the video now. The gap between learning and knowledge is called procrastination. Procrastination delays our success. This video shows you the way I traveled 15 years ago. the picture below shows you how light screws into the protein threads of photoreceptors. You need to get on the same page with me.

 

 

Television and all technology screens are capable of making you hungry because they induce leptin resistance via melanopsin dysfunction —  and it’s not only all those Pizza Hut commercials!

People are unaware of how the new melanopsin data allows this process to occur.  When blue light or nnEMF disrupt the WEAK covalent bond between melanopsin and retinol, retinol because free from its normal tight circadian control.  When it is freed of these shackles it destroys ALL mammalian photoreceptors because Vitamin A is the ONLY Vitamin on Earth that emits light.  The light it emits changes the optical signaling of the local environment and this changes how chemicals in the cell at this location oscilate.  The oscillatory pattern is tightly controlled by nature to create the precise signal that is needed to make life and wellness possible.   it always creates a stimulus around photoreceptor light centers that create abnormal signaling.

The emission of light has to be tightly controlled in all biologic reactions.  It turns out freed retinal is the secret in life of how chaos is really controlled in cells.  Free retinal is the other side of the optical story of how leptin resistance gets humans ill.

BLUE LIGHT HAZARD = melanopsin dissociates from retinal and free retinal destroys photoreceptors = destroys optical signaling.   The lower your redox is the more retinal is released.  Here is the the BLH and frequencies laid out.

 

 

 

 

When you look at the table something remarkable about the sun should appear to you. As we approach the UVA and red frequencies in both directions the BLH drops quickly. We also now know that the BLH correlates with the free radicals made in mitochondria too.

Certain electromagnetic stimuli are more apt to release retinal in this way because of the weak covalent bond beside frequencies in the visible spectrum of light because of how electromagnetic waves affect bonds differently. This is why the microwave bands in 1G-5G pose a great threat to the retinal mechanism behind leptin resistance.

 

 

Blue light is the one that is now best identified in the literature.  Now that we know the human bond between melanopsin is a weak covalent bond we know that 1G-5G waveforms can also separate them even more easily via rotation.

Recent data in 2015 show that the Schiff base linking the chromophore retinal to the fractal melanopsin protein is more susceptive to spontaneous cleavage in mammalian melanopsins. In fact, the researchers went out of their way to note that this stability is highly diversified between mammalian species, being particularly unstable for human melanopsin.

This raises a big point.  If the bond is unstable in humans what might this mean for free retinal in tissues.  Is this how leptin resistance begins in tissues?

I think it is.  Why do I say it?

This pool of the retinal-free melanopsin molecules would effectively decrease the number of photoreceptive molecules in ipRGCs. If that was true wouldn’t melatonin levels also drop as a result?

What could cause the presence of the free retinal most frequently in modern life? One possibility is a shortage of retinal supply from retinal-producing enzymes or could it be stimuli from the electromagnetic spectrum that do it?  People forget that terrestrial sunlight on Earth does not equal the entire light spectrum in the universe.  Melanopsin was innovated by evolution based upon the light that fell to Earth from the sun as the picture below shows.  Not even the sunlight that astronauts face in orbit in the ISS is equivalent to the sun we get on the tectonic plates on Earth.  This is why they get diseases that were once rare on Earth.  Today, children are getting the same type of diseases as astronauts because of how man has usurped the electromagnetic spectrum for technology use.

Technology use and abuse cause the capricious cohabitation of the human’s mind. Nature’s harmony makes small things grow. Lack of it makes big things decay. Technology ruins man’s biologic coherence and induces degeneration of the mind/body in ways most do not appreciate.  This is why I created the twitter hashtag #mitochondriacwisdom.  

 

 

Does this coning feature of sunlight have implications for the melanopsin system in humans?   Alternatively, could the change in bonding be due to constant retinal release from the protein via cleavage of covalent bond (Schiff base linkage) with retinal (Vitamin A) by light? The latter possibility would be similar to what occurs in cone photoreceptor cells in the eye, where retinal is known to spontaneously dissociate from visual pigments, resulting in reduced overall cellular photosensitivity by destroying photoreceptors as the link above shows.

They found this via mutagenesis analyses, that this diversified stability is mainly due to parallel amino acid substitutions in extracellular regions. In the 2015 paper they proposed that the “differential stability” of the retinal attachment to the melanopsin fractal chromophore likely contribute to functional tuning of non-visual photoreception in mammals.  This means that rotational changes in the covalent bond alter its optical signaling ability.  This implies the fractal protein changes and this means it change how it can react.  This is precisely how the butterfly effect occurs when the chaos of changes appears to come from an order system but yield an wildly unpredictable result.  This was eloquently laid out in Jim El Khalili’s documentary   This is why all Black swans need to be really concerned with technology and artificial light use.  The 2015 paper is warning us that topologic effects in retinal is enough to cause optical damage to photoreceptors it is bound too.   This fully explains why blue light has been causing humans problems since we invented the light bulb in 1874.

 

 

Carbon atoms in single bonds rotate freely in organic chemistry. Rotation can occur because the heaviest electron density in the σ bond exists along an imaginary line between two carbon nuclei. Rotation does not change this electron distribution; the bond strength remains constant throughout rotation. Because rotation is possible, the molecule can have an infinite number of conformations. Conformations = size and shapes = changes the way matter operates thermodynamically. All of our proteins were designed by nature to work only in terrestrial sunlight.

With manmade light the bonds react differently and they change the conditions of existence of the molecules in us. It turns out blue light and nnEMF release Vitamin A from from the carbons in melanopsin and that freed Vitamin destroys all the chromophores that our proteins contain. the picture below shows many pictures of those varied chromophores in us. When they are damaged, they cannot absorb light to make the protein they are connected to vibrate properly in cell water. This ruins the resultant vibration and coherence. This is leads to disease. Leptin is one such chromophore found in your subcutaneous fat. Once it becomes defective it cannot send its optical information to the hypothalamus and you get ill as a result.

 

 

We’ve believed that melanopsin was only present in the eye since its discovery in humans since 1998. We then discovered it in human blood vessels in 2014.

 

 

Then, in December 2017 we got the shock data it was also in our skin and subcutaneous fat helping explain why nature put leptin, another photoreceptor molecule, in our subcutaneous fat.

 

 

Leptin is designed to take optical data from the skin and skin arteriole surface about day and night and couple that with energy balance information and deliver it to the hypothalamus under the cover of darkness. Free retinol from surface light at the wrong time of the day is what ruins this hormones behavior optically. Once leptin signaling is disrupted by circadian mechanisms, the hypothalamus loses control of all growth and metabolism inputs. This leads to many chronic human maladies such as obesity, diabetes, and metabolic syndrome.

This is how I solved my obesity issue 15 years ago when I was 360 pounds. I realized that when leptin was altered by alien light in my environment it changed the the atomic structure of leptin. This small change lead to massive changes in my body mass. The reason made sense. Mitochondria create heat, which is infrared light. We need this light to activate the DDW our mitochondria make. What happens if the mitochondria in me were being destroyed by light in my operating room? What do the laws of physics tell us about heat and size? small objects in nature have a different surface area compared to their volume inside. This means that smaller people lost heat quicker. This is true in planets as it is in people. This is why Mars magnetic field from its interior dynamo died out sooner than Earth. It is smaller, so it loses heat faster. Humans who have mitochondrial damage because of the damage in their heme proteins in their cytochromes would lose a ton of heat if this would be allowed to continue. What does the body do to offset the loss? It makes you fatter. Why? This changes the relationship of the surface area to your interiot volume and improves your thermodynamics while the engines inside of you get worse by the retinal damage of leptin and your cytochrome proteins. This is also why mammals fatten in winter when the sun power varies and you lose more heat to the environment as the temperature around begins to fall. Eating carbohydrates in a falling solar environment fatten you. The cold causes you to use that fat to warm yoruself until the sun comes back in spring. That is how the forces of nature work inside of you when you are leptin resistant. When I realized this for the first time my life changed forever. This was the answer to my obesity crisis.

 

 

Mitochondrial photoreceptor damage are all defects in optical signaling caused by Vitamin A’s ability to destroy photoreceptors.   This is why the the authors in the article make this statement, about blue light, ”It’s toxic. If you shine blue light on retina, the retinal kills photoreceptor cells as the signaling molecule on the membrane dissolves.”    

That is a definitive unequivocal statement made in this paper.

 

What aggravates this effect in modern humans? The human choice to live indoors with blue light and nnEMF.

Your TV is likely the greatest blue light emitter in your house after Obama changed TV signals from analog to digital in 2009.  But every tech device was digital the day it was created.  The sheer number of both devices is what is accelerating modern humans diseases now.  Nobody sees where the pieces fit until they do.

Blue light, via the photoreceptor melanopsin, disrupts autophagy and mitophagy, as I laid out in my Vermont 2018 video.  This, in turn down-regulate apoptosis (programmed cell death) in cells. Thereby, defective cells, running at a net energy loss, send out a call for more food via the defective leptin photoreceptor.  That defect is cause by free retinol in the tissue and in the blood stream that is not circadian controlled by retinol binding protein — Leptin is loaded with these aromatic rings and the light emitted by the Vitamin A alters the quantum HUMO-LUMO rings and this is what blocks leptin from going from your subcutaneous fat around midnight when it is supposed to be dark to enter the hypothalamus to deliver this photonic and electronic data to this part of the brain.  Without the proper message energy balance and metabolism is lost.  That is what leptin resistance is at the quantum level.  Because of the melanopsin dysfunction, the information and energy transfer the hypothalamus requires can never be satisfied in the mitochondrial matrix of the cells affected by this optical deficits due to their defects having escaped the recycling autophagy program built into cells.  That cycle is controlled by the circadian mechanism which Vitamin A has hijacked.  The antidote to blue light in nature,  is 42% of the red light in sun.  It is augmented by UVA and UVB light.  These parts of the solar spectrum strengthen mitophagy and apoptosis to return physiologic law and order back the the defective tissues.   We would be much better off watching the sunrise and the sunset than watching TV, using a computer, or talking/texting constantly on a cell phone,  especially at night. We’d be more wise to use a geothermal pool, or build a green house, or stare at the flames of a campfire…to stimulate the healing powers inside your colony of mitochondria.  This is the credo of the black swan.  This explains fully how I see Leptin resistance develops in humans.

SUMMARY

Leptin resistance is a photonic process in human biology. So what does blue light and nnEMF lead to give what we’ve learned about melanopsin/retinal links? It ruins the Bazan effect to ruin the long loop to cause liver level leptin resistance. This blocks DHA to be replaced in cell membranes in the liver and CNS/PNS. This causes many communication and memory issues via a broken circadian mechanism via the eye and skin. The pic is about the eye but it was created before you knew melanopsin/retinal was in the subcutaneous fat and skin arterioles. See the pic below = Leptin resistance at liver level = lowered global DHA in liver cell membranes that induce PPARγ-target catalase expression and reduce ROS levels, leading to the inhibition of JAK2/STAT3 = what leptin resistance is inside a cell below the pathway level of Ph.D. or MD understanding.

NOW YOU HAVE THE ENTIRE THESIS.

 

 

 

Can water have a ‘memory’ of its previous solutes, environment or processing?

On the surface, this question sounds like quackery and pseudoscience.  But the Black swan will go deeper than most to examine the evidence for themselves as the video above shows.

Montagnier experiments showed the effect.  But he still has no idea how it happens.  Today I am going to share with you how I think it does happen in your cells.

Water forms crystals that varied depending on the environment they sense.  The electromagnetic radiations in that environment can change the crystalline structure of water at the atomic level.  We know crystals are capable of creating memory.   So, is the question as crazy as it first appears?  What does the DATA say?  All systems may retain a memory of their previous treatment, whether this is due to the formation of stable contamination, or to the production of energetic heterogeneities. It has been shown that the physical properties not only depends on the initial temperature but also on kurtosis; the distribution of the particles’ kinetic energies from the mean value, a property that may depend on its past history. It should not be surprising that water also may retain a memory of its past history.

 

 

Are evolutionary changes stored magnetically in water around DNA?

In the literature it is well known that microwave irradiation gives rise to a memory effect on the surface tension of water that lasts for minutes after the effect of temperature rise alone has ended. Most scientists and the public have no idea these papers exist but Black Swans do. The first LINK below shows this effect in cite one.

This is why 5G concerns Black Swans. Microwaves induces topologic effects on water. This means it can affect its electric abilities in any aqueous system. CELLS are such a system.

 

 

More data to worry about: An extraordinary paper authored by Nobel prize-winning Luc Montagnier has described memory effects in aqueous DNA solutions that the authors propose to depend on interactions with the background electromagnetic field. These effects, if real, require the prior processing and dilution of the solutions and are explained by Montagnier as molecular resonance phenomena with nanostructures derived from the DNA and water.

So how could this happen in reality?  Is there an explanation?   What is the biophysics below the cell level a Black Swan can study?

Might the crystals in water vary in their electric abilities based upon the light around the water?  Yep.  Is water ferroelectric?  Yep.

Ferroelectric materials are characterized by the spontaneous electric polarization that can be reversed by inverting an external electric field. Water molecules are dipolar and thus ferroelectric alignment of water molecules is conceivable when water freezes into special forms of ice.  Spin ice experiments were discussed on my blog years ago.  But now the new data published in early 2018 have raised the bar for the evidence.

Did you know that EZ water (exclusion zone) and ice share a lot of physical similarities?

Generally, ferroelectric materials have high dielectric constant.  The EZ has a dielectric constant of 160 whereas regular tap water dielectric constant is only 78.

So it appears the water made in a mitochondrial matrix creates is a thin ferroelectric crystal.   Ferroelectric materials have a spontaneous dipole moment which can point up or down.  But does this unique ability explain WHY water MIGHT store memory?

Yes it does.

Being a ferroelectric crystal means that they can ALSO be used to store information, just like magnetic bits on a hard disk. The advantage of ferroelectric bits is that they can be written at a low voltage and power.  The brain works at 20 volts.   Magnetic bits in your tech gear require much larger currents to create a magnetic field for switching, and thus more power. The disadvantage of most ferroelectrics is that the aligned dipoles are only stable in fairly large groups, so if you make the crystals smaller, the dipole moment eventually disappears.  When light hits the water, it builds a large EZ ferroelectric crystal that is uniform.

 

 

The common denominator in ferroelectric studies has been size and scale.  This is no surprise to anyone who listened to my April 2016 webinar on what constitutes life.  Water is a huge part of how life happens. In physics research, we know initially small crystals become ferroelectric, whereas larger crystals lose this property.  This is exactly how ice and EZ water are initially built.  So it does appear water made in the mitochondrial matrix is quite special because it can create a memory of things found in it.  This might explain why the Kreb and urea cycle are designed by nature to be in this water.  It appears the memory of the seasons on Earth can be magnetically stored in matrix water by its deuterium content.  Might this be how metabolic rate of dofferent tissues is programmed in morphogenesis?  I think so.

Is our circulatory system important in delivering memory to our cells by using water as its currency?

 

 

So to answer to the initial question this blog posed is: Can water have a memory? The answer appears to be yes. Why? Because water is ferroelectric at the nanoscale. When water is below 1.4 nm inside of a cell some rather bizarre things begin to occur at the quantum scale.

 

 

This means that very ‘small bits’ can be constructed from the crystals in EZ water. Furthermore, when a particular substrate is added to water that is magnetic, and this combination of magnetic and ferroelectric bits brings an extra degree of freedom, allowing each bit to store double the information. Could this be why the mouths of all the cytochrome proteins have iron-sulfur cores? Are they key to how life stores energy at low voltages and creates memory?

Does this imply that memory and consciousness might be a function of how good the water our mitochondria matrix creates is?

Yep.

 

 

CITES:

1. H. Parmar, M. Asada, Y. Kanazaw, Y. Asakuma, C.M. Phan, V. Pareek and G. M. Evans, Influence of microwaves on the water surface tension, Langmuir, 30 (2014) 9875-9879;M. T. Amiri and M. C. Amiri, Comment on “Influence of microwaves on the water surface tension”, Langmuir, 31 (2015) 10931-10932; H. Parmar, M. Asada, Y. Kanazaw, Y. Asakuma, C.M. Phan, V. Pareek and G. M. Evans, Reply to comment on “Influence of microwaves on the water surface tension”, Langmuir, 31 (2015) 10933-10934.

2. Yingfen Wei et al, A rhombohedral ferroelectric phase in epitaxially strained Hf0.5Zr0.5O2 thin films, Nature Materials (2018).

3. https://phys.org/news/2018-08-barrier-material-quirk-telecommunications.html

 

Salt levels control the exclusion zone of CSF. Iodine helps augment this affect via the Grotthuss mechanism. It is the single most key mechanism in the brain. Time for our group to bone up on Dr. Gerald Pollack’s work and how it links to proton spin and information transfer to augment autophagy during sleep. Blue screens and RF/microwaves pulse decrease salt in the CSF.

 

 

Salinity and water have a deep link for energy and information generation and plasma in our cells. Most food gurus have no idea why this is the case.

Artists use lies, to tell the truth about life. Because of belief, you usually find something true about yourself in their work. That small parable of truth creates a human unwillingness to delete that belief. Wisdom is not expecting people to understand what the artist intent was; it was the anticipation of how the ideas are perceived that matters.

 

 

When dealing with living things, one can best feel how primitive today’s bio-physics truly is. Mother nature was undaunted by the huge amount of work she needed to employ by designing cells. Such a task, on the surface, might be indistinguishable from a miracle until you realize she had all the time in the world, all materials one could need, and quantum mechanics. The environment is designed to create a “decoherence stimulus” in the mitochondrial situated in the sea buried within your cell to slightly alter the complex quantum situations built into living cells by Mother Nature. Those slight changes in the exclusion zone of the water plasma surrounding a mitochondrion alter the % heteroplasmy to drive evolutionary trajectory towards the environment. Night and day are different environments. One promotes wakefulness and the other promotes sleep. The EZ is the key change to that environmental change.

 

 

Seawater is the ultimate plasma of life. It is also why every cell has salty water inside of a cell. If the sun shines down on the water and some of it evaporates, the salt is left behind and the water will become saltier. Similarly, if some fresh water is added, such as from rain or melting ice, the salt will be diluted and the water will be less salty.

Salinity does change with depth, but the way it changes depends very much on where one happens to be. That’s because of another property of water, its density. In our cells density can also be augmented or subtracted from because of deuterium.  In the brain and CSF deuterium has to be tightly controlled because of the fast metabolic rates of the TCA and urea cycle in the brain.  If too much deuterium enters the cytosol of neurons cognitive haze and poor sleep result.  Deuterium changes the density of metobolic water.

Density is how “heavy” a parcel of water is. And “heavier” water tends to sink and stay below “lighter” water (have a look at this answer from our archives for a little more on how that works).  In the CSF space this is not good because the lowest level of CSF rests on the pia mater and the surface of the necortex which is the most metabolic active tissues in our body.

The density of seawater depends mostly on how much salt is dissolved in it (more dissolved salt = “heavier” seawater), but temperature also has an another effect on hydrogen isotopes in water. In all cases, raising the temperature, invokes thermal vibrational and entropic effects. This tends to preferentially stabilize H+ over D bonds in aqueous solutions.

 

 

Pressure builds heat (IR) and heat moves things with mass. Deuterium has more mass than H+. We also know heat favors H+ bonding over D bonding in an aqueous heat bath like a cell. Heat flows from hot to cold and comes when electric and magnetic forces are confined to a tight space. This is the advantage that mitochondrion seeks to gain for cells. Heat expands most things in nature, but not all things.  Water happens to be one of those things because of its massive heat capacity.   Life uses water for this key reason, and mitochondria have chosen to release heat to water a mitochondria makes for one specific reason:  because heat shrinks water that is confined in small spaces and breaks nature symmetry by shortening the distance of the respiratory proteins on ECT.  This increases energy efficiency and proton flows in cells favoring H+.  This helps keep deuterium in the blood plasma where it belongs and not in the matrix where heat is liberated and metabolic water is made.

How does nature use this in sea water?

The abrupt change in temperature at a thermocline occurs because of the density of water changes as a function of temperature. The density of water links to proton spin in seawater. Pure water, sans salt, has a maximum density at around 4 degrees C (3.98 if you want to be fussy). Of course, in a lake, the densest water will be found at the bottom, with less dense water overlying it; this point was expanded in Ubiquitination 5 blog to a great degree as it relates to Rayleigh Benard convection. This type of convection needs a small amount of gravity to flow.  This is present on Earth but absent in space.  This is why astronauts are at risk for deuterium damage of their Kreb’s bicycle and sleep impairment with long flights.  These convections in heated water represents the lowest energy state on Earth, which is the way the physical world generally likes to be. This is why energy in water always flows or cycles naturally without any physiologic work needed to be added by a cell. A cell takes advantage of this physical property of water to innovate life.

Now then, if you imagine the sun shining down on a lake, the water at the surface will, of course, begin to warm and change the surface temperature in relation to the deeper cooler water.  Now think about what happens on your skin in UV light.  This heats up the surface quick and it also affects the blood plasma below because UVA light increase NO to make vessels come closer to the surface to be irradiated by sunlight.

This sets up the convection cell naturally I mentioned above. That will make that water less dense in certain places in the arterioles to give us laminar flow in the center of the artery (thus more buoyant), and it will be more turbulent on the surface.  That turbulence help libertate NO in vessels.  When these things occur the less dense water in blood will, therefore, float over the cooler water below it. The cooler water below has more electron density because it is denser and the surface water has less electron density in it.

The hydrogen bonding networks in both densities of water are also different and so is how they work with the photoelectric effect. This tells you their charge and size of their EZ will differ too. This is the critical physical part a mitochondrion is built to sense inside of our cells. In the ocean or lake, it is important to the ecosystem and all things that live in it. Why? Because the warmer the temperature of the water,  the more it EXPANDS to build pressure up in the skin and it loses density and gains buoyancy.  This has a big effect on the deuterium in our blood.  This thermal and hydraulic pressure make deuterium emit a full spectrum of ELF-UV.  This light can change the density of the water content of blood because the EZ has an increased viscosity and an optical window at 270 nm.

This is why water everywhere on Earth will float on the deeper cooler water below.  This happens in the sea and in your blood.   Energy in water columns flows from cool to hot because of convection. It turns out, it is not easy to move heat downward in the water column (pelagic water), and that is what causes a very sharp thermocline we see in oceans especially in the poles and Gulf.  In practice, be sure there is a small amount of heat conduction by the water, as well as mixing by the surface winds, and that will cause some LOCAL “thickening” of the thermoclines when we measure them in lakes. Remember lakes and oceans are the different. The Gulf is different from the ocean too because of salinity and its deuterium content.  This varies with latitude.  Temperature also varies with latitude.

Essentially the same thing happens in the open oceans, but there is the added complication of salt in the water (which also has an effect on the density of seawater), and very substantial wind mixing that tends to make oceanic thermoclines a little less distinct than we see in lakes, rivers, and reservoirs. So, if water has more salt in it, it will tend to be “heavy” and will tend to sink. And often there is an increase in salinity with an increase in depth in polar waters. Life there takes full advantage of this.

Life at the surface of the sea takes advantage of its local environment and your mitochondria take advantage of this version of plasma in your cells in a similar fashion because salinity relates to charge and to energy flows. All these little details your food gurus are clueless about. All that links to time. Why? charge and time are fundamentally linked by our surface topology. ‘Now’ is a local theory of what’s happening presently, cobbled together using bits of news from the sensory receptors all bathed in saline water.

Jack is there any new data to support this position?

VIDEO

More proof that studying nocturnal mammals has no place in the study of human neurologic disease based on new data. Why? How much do you know about information quanta and how it operates the universe and in cells via light waves? If you do not know a think about it you might want to read my Quantum Thermodynamic series on Patreon. Information quanta is a key feature of this blog series. These neurons, unique to humans, deal exclusively in the distribution information quanta over the surface of the human brain called the neocortex.

In the end, science is just a progress report of where we are now in our understanding. It is not our final destination, but it is a data point on the road to understanding. That has been and will continue to be the message of my work.
Where we are today in science, in understanding how the brain really works, is close to where a man was 2,000 years ago in trying to understand how the planets moved in relation to the sun. We are nowhere close to where we should be.
So if we are that far away in our understanding of the brain, how can we begin to make sense of it all? We can learn a lot from the macrocosm of space if we scale it to biology on this planet. Today ’s post does that for you, the black swan, who have never heard of this information before. This post is why you need to be part of the tribe who digs deeper than your current health EXPERTS.
The human brain is surrounded by water called CSF that is generally deuterium depleted. Deuterium has a much higher density than water made of regular protons called protium.
I showed you earlier in my many free blog series on my website how molecular oxygen is delivered from the phytoplankton in the photic zone (surface) of the ocean to the ocean depths using the density of cold water to deliver it there. The denser the water, the more oxygen is dissolved in it. The power of the sun’s photoelectric effect splits electrons from water in phytoplankton, which liberates oxygen and electrons. The same thing happens in melanin in humans. Humans have melanin in their brains and more of it than any other mammal on this planet. Ask yourself why evolution would do this?
The liberated O2, in the ocean, becomes more dissolved in colder water by the laws of nature and chemistry, and then it is distributed all over the oceans by the thermohaline currents.

I have been showing you FOR YEARS how the exact same process that happens on the surface of the earth is fractally designed on your own neocortex of your brain.

 

 

HOW TOPOLOGY OF EARTH SCALES TO YOUR BRAIN

The very same process that works in the thermohaline current works in CSF that surrounds your brain to bring higher oxygen levels to the surface of your brain using QED principles linked to LIGHT, WATER, and MAGNETISM.

 

 

It uses the photoelectric effect for animal photosynthesis using many proteins. Melanin is just one protein. The unusual thing is melanin is present in the human brain in many places when we know the UV of the sun does not and cannot get to the surface of the brain. Why would nature do this?

I believe the answer is because neuromelanin is a key to information quanta transfer. It is critical in all neurologic diseases in humans.

Recall inflammation is a measure of pH in water. pH is a log scale of hydrogen protons and includes deuterium and protium fractions. Moreover, when inflammation is present and is rising for ANY REASON at all in the brain, the result in this surface of the brain’s CSF is to alter the density of water that sits above this cortex. This is what these neurons are doing. No one else will tell you this or be able to explain in detail how the process works by my members and Patrons will.

Did you know that UV light exposure of water increase the charge in water? Did you know CSF is a ultrafiltrate of blood plasma that does get irradiated by sunlight. Might this mechanism be the reason why melanin was put in the brain by Mother Nature? Melanin is a fluorophore protein that absorbs UV light. This paper below published in March of 2018 was a game changer for Black Swans.

 

 

Inflammation makes CSF less dense, and when CSF is less dense, the laws of physics control the action of biochemistry that is possible on our surfaces or deep in our tissues. Deuterium and protium have markedly DIFFERENT density measures. When you know better you do better. Time to join my tribe folks. This helps explain why UV light exposure of the eyes and skin helps improve sleep. It changes how much energy and information can be stored in the water in blood plasma that eventually becomes CSF that surrounds our brain. Salt increases the electric potential of the water in our blood plasma. It really helps if salt is used liberally by the black swan.

 

 

SUMMARY

These sensory receptors only absorb the energy and data of just a part of the environmental story going on around your 5 senses; What you are is what the brain perceives is a partial version of true reality. This is akin to biochemical data that is irreproducible that so many “closet scientist/clinicians” like to spew.  From those senses, our brain fills in the missing parts to create a story we call reality and life…….as we lose cellular charge time speeds up, we lose health, and we age faster and our sleep declines.  The fastest ways to cause this is chronic technology use.  We all need a technology diet more than we need a food diet.

This points out why some people cannot decipher data in biochemical journals well. Their sensory receptors are attuned to the wrong things because their environments are pulling charge from their senses and their brains. This lowers their information assimilation abilities in their neocortex.  Deep truth bombs here in the cite below.

 

 

Here is my key point to you: any set of labs are not good enough tools for the science we need to study to get people well. What we observe is not nature in itself but nature exposed to our method of questioning. Cells seem to use light to know about nature in ways our mind or senses cannot observe. The answers never manifest on labs either.  Labs contain clues for the quantum biologists.  This means you really need to understand light to every understand how we work.

CITE:

http://sciencenewsjournal.com/amount-salt-brain-determines-sleep-cycles/

This blog is part of October 2018 Optimal Klub Webinar

Are all cranial nerve neuralgia’s a manifestation of a brainstem redox deficit?

I think they are.  I think all pain syndromes are linked to a lack of redox caused by an inability to maintain your redox potential in your CNS and PNS at some level.

The cell membranes in the cranial nerve nuclei and brainstem nuclei share a blood supply.  They are located in close proximity and recieive input from the retinal pathways and from the skin surfaces.

Trigeminal neuralgia is a disorder of paroxysmal and severely disabling facial pain and continues to be a real therapeutic challenge to the clinicians. As a neurosurgeon I see this condition often.  Trigeminal neuralgia affects the 5th cranial nerve but this condition can affect other cranial nerves as well.  While the exact cause and pathology of this disorder is uncertain.  I was taught to believe in residency that trigeminal neuralgia is most often caused by irritation of the trigeminal nerve by calcified blood vessels.

Today I believe that more proximal defect is due to an endocannabinoid defect in the brainstem of the nuclei of these cranial nerves cause by alien electromagnetic fields and manufactured light that cause a stress response in the paraventricular nucleus that control vagal tone in the autonomic nervous system.  The damage to the PVN can extend to the cranial nerve nuclei via the blood vessels in the brain and retina where melanopsin exists.

This irritation results from damage due to the change in the blood vessels, the presence of a tumor or other lesions that cause the compression of the trigeminal root. The pain of trigeminal neuralgia is characterized by unilateral pain attacks that start abruptly and last for varying periods of time from minutes to hours. The quality of pain is usually sharp, stabbing, lancinating, and burning. The attacks are initiated by mild stimuli such as light touch of the skin, eating, chewing, washing the face, brushing the teeth, and exposure to wind.

Although antiepileptic drug therapy may be beneficial in the treatment of trigeminal neuralgia, up to one-half of the patients become refractory or intolerant to these medications. At present there are few other effective drugs. In cases of lacking effect after pharmacotherapy, surgical options may be considered. Currently there is growing amount of evidence to suggest that the psychoactive ingredient in cannabis and individual cannabinoids may be effective in alleviating neuropathic pain and hyperalgesia.

Evidence suggests that cannabinoids may prove useful in pain modulation by inhibiting neuronal transmission in pain pathways. Considering the pronounced anti-nociceptive effects produced by cannabinoids, they may be a promising therapeutic approach for the clinical management of trigeminal neuralgia.

BLOOD SUPPLY brings many things to bear in many mitochondrial diseases because since 2014 we know that melanopsin is present in aterial trees of almost every organ.

Anterior and posterior branches of the circle of Willis provide arterial blood to the hypothalamus. The hypothalamus also receives arterial supply from the hypothalamic branches of the superior hypophyseal artery from the carotid system.

The vertebrobasilar arteries supply the posterior two-fifths of the cerebrum, part of the cerebellum, and most areas in the brain stem where the cranial nerve nuclei are located.

The circulatory system of the anterior carotid system and vertebral basilar system have lipid rafts in them that create a charged state that is SHARED.  The shared area is between the hypothalamus where the PVN is located and the cranial nerve nuclei.  So when redox charging is ruined in one the clinician should know that it is the arterial tree that connects the two.

Inside of the arterial tree is melanopsin, dopamine, and the machinery for creation of nitric oxide.

All of these proteins are acted upon by excited electrons that fall back to the their ground state and give the charge up to the lipid rafts in the cell membranes of these areas in the brain.

 

Sunlight provides the redox charge that allows mitophagy and aopotosis to optimize the vessels anatomy using cicradian programming. I have a deep sense this is why melanopsin is present in our vessels.

What happens when this system does not work? People wind up with mitochondrial diseases, a low dopamine state, and lowered melatonin level, lowered DHA, are more likely to clot and sustain an alteration of their endogenous cannabinoid system.

One of the key metabolites from DHA is the key endocannabinoid that maintains the charge of the lipid raft in PVN and brainstem nuclei of the cranial nerves. When this charge transfer does not occur within proper ultradian or circadian cycles, we can see cranial nerve pain syndromes, and migraines manifest. Many times these will be associated with calcified brainstem arteries that cannot liberate nitric oxide (NO). NO is designed to relax microcirculations. It turns out the 7 alpha helices also need electrons to help relax the microcirculation in the brainstem too.

 

 

Almost every person I have seen with these pain syndromes have evidence of circadian cycle disruption when I look for it. I believe this happens in humans because melanopsin signaling is destroyed in those arteries as the paper below shows.

 

 

It turns out light-induced isomerization of melanopsin is up to several tens of femtoseconds faster than the analogue isomerization of invertebrate and vertebrate visual pigments. It also has been revealed that melanopsin’s thermal isomerization is controlled by an energy barrier higher than the barrier of dim-light visual pigments. These properties support the idea of why evolution has favored the use of extreme light sensitivity of melanopsins in many tissues in our body.

What happens to human circadian biology is damaged, with respect to melanopsin biology in our arteries?

Melanopsin/retinol dysfunction occurs when the covalent bond between melanopsin and retinol is disrupted by blue light absorption. This unleashes retinol to become a toxin in the arterial wall and surrounding neurons and this toxic liberation is what lowers melatonin levels locally in those tissues to cause them to lose control of local mitophagy and apoptosis in neurons and vessels. The collateral signaling cascade leads to lower DHA replacement in the cell membranes of humans. It turns out that neurons and arteries of the brain have the highest levels of DHA within them.

Lowering DHA in the eye and skin directly ruins clock maintenance.

This cascade of events also lowers electrophiles in the blood plasma like CO2 and free electrons made from melanin’s ability to split water using visible light. Remember all hormones are derived from cholesterol when T3 and Vitamin A convert it to pregnenolone. Without T3 (AM light) or Vitamin A (retinol from melanopsin), you cannot make the substrates of ALL the hormones humans use. This is how it all works in symphony inside the brainstem too. Progesterone protects arteries from damage. All the hormone panels that also work via circadian biology too as the slide from my Vermont 2017 slide shows. Tyrosine needs sunlight to activate and program the aromatic ring to make T3 and T4.

As a result of the breaking of the melanopsin/retinol weak covalent bond the arteries calcify and they become like lead pipes and can strike the exiting cranial nerves in the cisterns filled with CSF around the brain. When retinol is freed it becomes a neurotoxin in the body and is linked to poor sleep and regeneration. This was covered in my Vermont 2018 talk from the slide below.

 

 

The eye, iris, sclera, RPE, have massive amounts of melanin and I think this is present to provide the opthalmic arteriole bed with a massive factory of free electrons from the action of melanin on water in this arterial tree.  I believe those free electrons are critically lost in most pain syndromes of the head and neck in humans.  A loss of free electrons sets the stage for arterial calcificiation.

Most of you know I think diabetes is a blue light hazard disease.  Few of you know that I believe the disease is linked to melanopsin damage of the arterial tree.  This is why diabetics always have poor vessels that are stiff and do not deliver an adequate amount of blood, oxygen, and electrons to distal tissues.

Vascular calcification in humans can occur in either the intimal or medial layers of the arterial wall. Intimal calcification is associated with athero sclerosis, which is characterized by lipid accumulation, inflammation, fibrosis and development of focal plaques.

Medial calcification is associated with arterio sclerosis, i.e. age- and metabolic disease-related structural changes in the arterial wall which are related to increased arterial stiffness. It has been hypothesized that vascular calcification, either intimal or medial, may directly increase arterial stiffness.   I think both are linked to the amount of melanopsin damage in the system.  This is why the CAC blog was given to you as a warning shot of what to expect in a 5G world.   I think nnEMF will demolish the arterial melanopsin system.

Neurosurgeons are taught that the calcified vessel is the main problem in cranial nerve pain syndromes.  It turns out this is not true as the video shows.  My patient was in scrubs.  It turns out he was a nurse who worked in the ICU at night under blue light and nnEMF.

I beleive this environment destroyed his melanopsin mechanism in his arterial tree in his brainstem and caused a myriad of problems in the lipid rafts of the cell membranes.  This is why understanding the Bazan effect (below) is very important in these diseases.

 

If you can restore the melanopsin/retinol function back to the arterial bed, you can restore the return of NO, melanopsin, and natural cannabinoids to the lipid membrane rafts to increase their redox potential in these vessels. If you can increase their charge potential you can reverse the process without opening the skull, in my opinion (below)

 

 

You can see in the picture below the cranial nerve nuclei are quite close to the PVN in the hypothalamus (grey nuclei below) but they are not directly connected to one another in most cases via tracts. This raises the question, what connects them?

 

 

You can see in the picture above the brainstem is outlined but left blank. If you find the mammillary bodies in red above you’ll see that small tube above it. That is the floor of the third ventricle that connects directly to the brainstem which is outlined. The picture below colors in the brainstem and shows you where all the cranial nerve nuclei are.

Both sets of neurons are directly connected by the circulatory system in the brain. The circle of Willis surrounds this area and it is where the anterior carotid system links to the vertebral basilar system at the posterior floor of the third ventricle. We call this a watershed region in the blood supply. This sits right above the trigeminal ganglion picture below.

 

 

Many of you might get lost with this neuroanatomy lesson but the slide below shows you how physiology of the hypothalamus and pituitary and brainstem couple (yellow boxes below). The circadian control in the eye and skin is very important to these systems because it is linked via the arterial supply of this region because melanpsin is located in these arterial trees. Since it is there it is subject to damage.

 

 

The picture above shows you how they are linked, but does not show you the wireless connection via the RBC’s in the circulatory system of the region.

Life can begin when you gain control of the processes where the environment first meets your tissues. This happens on the surfaces of our of cells where membranes exist. Human membranes are unique because they are loaded with DHA in their lipid rafts.

This change in evolution was done to take massive advantage of the free energy in sunlight to create a massive electric charge across a small membrane in our RBC’s.

 

 

During the day, when the sun is present terrestrial to your eyes and skin the RBC’s in your vessels come closer to the surface to sense the electric and magnetic fields in daytime. It turns out the daytime ionosphere has massive quantities of electric fields and very low magnetic fields. This is due to the presence of sunlight. The RBC cell membranes allow us to harness this free energy from sunlight by using melanin as an intermeidate to create a ton of electrons from splitting water in our arterial tree.

Life begins by creating wireless power transmitters in our surface membranes (surface topology) that work by collecting energy buried in the electric and magnetic resonance of fields in sunlight. They can do this in many ways wirelessly from our environment.

This is how life became supercharged by sunlight and the dynamo in Earth. The technologic problem using this method that evolution had to solve, was that the original surface transmitters in early life forms had a poor range and fidelity to share their information. This kept life simple for 3.8 billion years. The reason for the poor range is due to the inverse square law which states that the intensity of electromagnetic oscillations varies inversely to the distance of the emitting source (S) as the slide below shows.

 

 

The further away our skin/blood is from the point source the worse transmission rates are for this energy source. So how did evolution fix this problem?

Nitric oxide and melanopsin both work in unison to bring blood vessels closer to our skin and retinal surfaces in our circulatory system to create the free electrons from water. This effect is very complex. I laid out that complexity in the Vermont 2018 talk. The slide below shows you some of the changes sunlight induces when terrestrial sunlight hits your skin.

 

Mother Nature built cell membranes in our skin and retina that could absorb light and she placed proteins in their lipid rafts to slow the light down (Vermont 2017) to create a mechanical vibrations after light collide with these things in our cells.

Because of this evolutionary design, human membranes no longer suffer from limited signal creation or amplification.  Their specificity and fidelity are improved compared to bacterial systems.

The inner mitochondrial membrane however has to be controlled differently in humans, because it is the only human membrane that retains its bacterial origin.  It has no DHA, by design.

The outer mitochondrial membrane is loaded with DHA and it is contiguous with the Golgi body and rough endoplasmic reticulum where proteins are made.

Why did Mother Nature do this?

When you harness mechanical vibrations and couple them to piezoelectric transducers you can amplify weak signals from the environment easily.  This is how Mother Nature solved the inverse square law for animal photosynthesis.  The signal transduction pathways built by evolution go way back and are still maintained in melanopsin and the G couling systems today.   This is why human melanopsin resembles innvertebrate opsins.  When evolution uses something for a long time without many changes it would be wise for the Black Swan to discover why this is the case.

Piezoelectric and flexoelectric transducers convert mechanical energy into DC electric current. This ability is amplified in humans because of DHA quantum abilities.

Sunlight, like sound, creates vibration in atoms in the air and in cell membranes and is fully able to transfer energy and information of these oscillations. This makes your skin and cell membranes a universal wireless charging system for sunlight.

That system is fed by the magnetic field and photons of the sun. Sunlight powers up electrons via the photoelectric effect.  Excited electrons fall back to the ground state.   This is how life really powers itself.  It is not really a story of ATP, as modern biology believes.

Photons are released from electrons after they are energized by the sun then fall back to the ground state by giving off a photon to our tissues.

In the blood plasma, the tissue most affected by this action is the lipid rafts in the arteriole walls. I believe this is why nature put melanopsin and dopa carboxylase in arterial walls.

They were put there to make dopamine (time) and tightly control circadian signaling to melanopsin/retinol dysfunction. This is why a Jablonski diagram is so common in life’s blueprint in many tissues. DHA is the lipid that does this most effectively on Earth and this is why DHA has not been replaced one time in evolutionary history in 600 million years.

This is why RBC’s are overloaded with DHA in their cell membranes.

They are loaded with carbonic anhydrase and ascorbate to control protons.  Proton control helps the shape of RBC’s too. Diabetes is associated with hyperglycemia and blue light damage.  Taken together, hyperglycemia in diabetes produced lower RBC ascorbate with increased RBC rigidity, and are more osmotical fragile making them less likely to navigate small capuillary beds.  As melanopsin damage rises RBC ascorbate levels drop and RBC look like echinocytes.  This makes melanopsin a key candidate to drive microvascular angiopathy in diabetes.

 

 

We also know that RBC’s are loaded with hemoglobin that looks almost identical to a chloroplast atomically. And we know hemoglobin’s absorption spectrum for light is strong in the visible spectrum of sunlight in the 250-600 nm range.

In fact, the cut off is strong at 600 nm. All this evidence tells us that RBC’s likely contain the key electric sensor for the sun’s light. If you ask most biologic researchers they would tell you the identity of the electric field sensor is unknown today. If you asked a bio-physicists or a mitochondriac they would tell you the electric sensor resides in the lipid raft of the RBC. When the RBC looks abnormal it is a sign of melanopsin damage. This won’t allow the RBC to get into the capillaries of the brainstem.

 

 

Lipids in cell are well known excellent electrical conductors. This will be an important fact to remember in a 5G world. Fats, like DHA, are loaded with pi electrons and all lipid rafts in cell membranes are associated with massive amounts of DHA and sphingolipids.

The arrangement of the lipid raft also tells us why an RBC is shaped the way it is when sunlight hits them. Charge affects size, shape, and density in anything made of mass. Electron colonies around RBC’s are most dense in the toroid region of the RBC and electrons are less dense in the hollowed-out center portion of the RBC.

 

 

This surface arrangement makes it a perfect sine wave antenna for wave transformation into other forms of energy. Proteins change light into other energy waves. The solar plasma is one such waveform that has an electric component. Bio-physical research has now demonstrated that the detergent-resistant membrane nano-domains, known as lipid rafts, act as the primary sensor to electric field-induced directional cell migration in morphogenesis.

 

 

This is why Robert O.Becker found that RBC had to be present in his limb regeneration experiments on frogs and salamanders. And he believed the reason humans lost the ability to regenerate was that our skin heals too fast that the RBC’s component could not generate a large enough electric current to drive the stem cells in the injured limb.

Isothiocyanates in foods are weak electrophiles in organic chemistry. Carbon dioxide in the air and our blood is also an electrophile. Voltage-gated channels act as electrophiles too to free more electrons up to do physiologic work when they are excited by sunlight.

Electrophiles in food act like the reactions of carbon dioxide in cells. Things with a lot of carbon are nucleophiles (DHA), and nucleophiles tend to attack carbon in lipids, proteins, and carbohydrates in cells. This means that when electrophile compounds are added to a lipid raft moiety they become excellent at delocalizing electrons (freeing them) and fostering non-linear optical signaling by allowing them to move to lipids and proteins in a cell.

 

 

This enhances their redox state as the slide above shows. This is a Jablonski diagram. This is how DHA and water turn sunlight into the DC electric current that Becker found.

To take circadian advantage of this, compounds need large electronic dipole moments, and isothiocyanates have them.

So does the lipids in human cell membranes. The other key way they work in a lipid raft in our cell membranes with DHA is the electron donor and acceptor portions of the raft have to be far away from one another…….and with DHA this occurs because of the 22 carbons and their alternating double bonds.

Those 22 alternating double bonds are critical in making the huge pi-electron clouds that interact with the sun’s light to create massive electric fields that can oscillate with sunlight. Please remember Becker found that the DC electric current vanished at night in all hi animals.

Amines are methylene donors and cyan’s, halogens, and nitro’s are acceptors (DeMartino 1988) in chemistry. This is why nature favored biologic amines in proteins. This is why I kept putting all those pictures up of the aromatic amino acids in the last 2 Vermont Talks to explain to you how animal photosynthesis.

 

 

When they bind to DHA they also make the DHA planar (flat) which also helps the electronic effect of the pi electron clouds to interact with sunlight to capture the sun’s power in these electrons to excite them. Melanin in skin augments the DHA effects.

That stored energy can then be transferred to water and to Tensegrity system in the cell membrane and within the cytoarchitecture of the cell to electrify it.

DHA facilitates the transmission because of the pi electron cloud when flattened acts like a giant wire of electrons that has very low resistance. A semiconducting electric current like this has low resistance compared to resistance in a copper wire or the filament of a light bulb which thermalize and let go of the light back to the environment.

This is how melatonin, melanin, and DHA work together to store energy from the sun for use later without the light ever becoming thermalized. Once it is thermalized you lose it by thermodynamic laws. This keeps cells powered and far from equilibrium in the living state.

People need to gear up on 3D atomic chemistry to get why certain foods work in mitochondrial disease states that all are associated with a low DC electric current. A low DC electric current = poor regeneration = poor healing = melanopsin damage = poor sleep.

The sun’s power is changed from photons to a DC electric current photoelectrically by cell membranes.

 

 

This is why I showed this as the first slide in my Vermont 2018 talk.  Cranial nerve pain syndromes are great diseases I can use to teach/show/educate you just how the complex things you are learning manifest in humans.

This is why I had this as my first slide in Vermont 2018 above.

All diseases = melanopsin defect at some level =  low DC electric current = poor electronic flow across the cell membrane. Remember melanopsin is in those lipid rafts too.   The collateral signaling cascade of melanopsin dysfunction deerves your extreme focus.

CITES:

https://www.ncbi.nlm.nih.gov/books/NBK279126/

 

 

 

Is nighttime and DAYTIME technology device use to blame for the etiology of most diseases in humans? Yes, it is. WOW. That is a big statement. How and Why? Here is a recent slide from a presentation I gave to shock my audience below.

 

 

Melanopsin, like the cone photoreceptor, is a PHOTORECEPTOR TOO FOR BLUE LIGHT. ALL OPSINS in humans are bound to retinol, and when the photoreceptors are damaged it is because of the atomic changes in retinol when the weak covalent bound it broken by light out of the normal circadian cycle.

In 1998, we found melanopsin in the retina. In 2014, we found melanopsin in the arterioles of the human body.

 

In December 2017, we found melanopsin in the skin and subcutaneous fat of humans. This was huge news to those who understand leptin and that leptin the fat hormone is also found within the subcutaneous fat of MAN. The data is telling us why we have an obesity crisis and it has NOTHING to do with food or exercise but it has a lot to do with circadian arhthmia of light in our eye and skin and subcutaneous fat mass.

 

 

 

The nighttime and daytime light environment has changed dramatically due to modern technology. Increased usage of mobile devices during ANYTIME OF THE DAY can disrupt your circadian clock. PEOPLE forget that the melanopsin receptor is regenerated DURING daytime!!!! So if you are around man-made blue light during the day you are still ruining your melanopsin photoreceptors. The intense light emitted from technology devices in screens has the potential to alter the timed release of factors within the eye, leading to chronic insults and susceptibility for visual damage. What does this mean to melanopsin photoreceptors?

 

 

 

I gave you my answer in the Vermont 2018 video, and I’s strongly suggest you view it.

Recent findings cited below suggest a functional role for the circadian clock in mammalian cone photoreceptor development and provide evidence for a continuing role for thyroid hormone (TH) signaling in cone photoreceptor maintenance. These researchers have said their findings may be of relevance in OTHER tissues where human photoreceptors are as well, where the circadian clock could alter tissue function by directly regulating enzyme type 2 iodothyronine deiodinase (Dio2) expression and thus TH signaling.

 

 

THAT is WHAT the data I presented in VERMONT 2018 is all about. Once you realize and know where melanopsin is, and follow the damage in its wake, you begin to see where mitochondrial disease begins for the very first time in your life. That is where the data is now……..it is not in food/exercise choices.

 

 

With this new information, researchers can begin to ask questions such as, “How else can we change the photoreceptors in humans using light evolution has never used? Are there other factors that can improve photoreceptor function and help extend their viability that we have failed to consider in science and industry? The BLACK SWAN among you now know this answer. It is as clear as the nose on your face.

CITES:

https://consultqd.clevelandclinic.org/circadian-rhythms-thyroid-hormone-and-vision-loss/

How do you know you are not as connected to Earth and sun chronically?

You’ll begin to feel better when you use opiates, weed, cocaine, and caffeine in coffee tea or chocolate. Eventually, you cannot live your life without any of some of them.

Morphine, Nicotine, and Cocaine – are all in caffeine’s chemical family. See the picture below. Ask yourself, why are the USE of these drugs on a massive uptick? Might it be the world’s ability to make dopamine in the eye and body is dropping in a blue-lit, RF/microwaved world we’ve built? A mitochondriac gets that perspective but do you? Do you see why the opiate crisis is massive or why a guy from the West Coast of the USA can convince many humans to drink a lot of coffee and add MCT oil or butter to it? Might this be a sign of a broken light environment or too much technology in your environment?

 

THE BIOCHEMICAL EFFECT OF TOO MUCH BLUE OR nnEMF

The group of chemicals that caffeine belongs to are known as alkaloids and they are the most addictive substances known to man. At various points in history, Morphine (Opium) and Nicotine (Tobacco) were socially sanctioned for daily use, but this trend reversed when the terrible side effects of habitual usage emerged in the 20th century. The 20th century brought with it industrialization and brought man from outside to inside to work in factories to manufacture things. Might it have been that migration, which blocked man from the sun informative light to photosynthetically make dopamine, been further worsened because technology and the electric power gird zapped more dopamine from mankind and then we defaulted to other bio-molecules with hexagonal rings that absorb light to replace the lost dopamine? I think you know my answer already.

 

 

 

Caffeine is no less addictive for many people, and the long-term effects on health are becoming increasingly apparent as average daily usage soars. It is no panacea for a low heteroplasmy rate either. Key conditions that caffeine significantly aggravates in my clinical experience are: stress response of all kinds, all muscle pain, most digestive complaints, liver metabolism, high blood pressure, PMS, insomnia, fatigue, weight loss AND weight gain, arthritis, blood sugar imbalances, skin conditions, and many mood disorders.

Is caffeine like sugar in the electrical dance between flower and bee?

Yep……

Alkaloids are found primarily in plants and are especially common in certain families of flowering plants. Flowers coming from the Earth assume the net negative charge of the Earth as an anode and therefore have net negative electric charge.  This negative charge draws bees to them for pollination. Bees and most insects are positively charged in nature, and this is why they emit light like the sun.  Recall that the sun is a cathode ray.  This is why the sun and Earth have opposite changes and why when the sun hits the Earth free electrons are given off the surface of Earth just like any anode does and humans take advantage of this by having sweat glands in their feet to absorb those free electrons.  If one wear rubber shoes all the time or clothing over their skin and never gets this electrical stimulus will they be driven to drink more coffee/tea with caffeine?  MY ANSWER IS YES.  The more disconnected you are to nature the lower your electrical potential is.  This is called your redox potential.  The lower it is the more you will seek the caffeine family of drugs to make up  the deficit.

 

 

BACK TO THE FLOWER BEE ANALOGY:

 

 

Flowers grow because of sunlight and photosynthesis and normally have a -30V electric field. The function of alkaloids in plants is poorly understood but I have a sense it is related to flowers having the ability to alter their charges for many biologic reasons tied to how they sense their environments. In some plants, the concentration of alkaloids increases just prior to seed formation and then drops off when the seed is ripe, suggesting that alkaloids may play a role in this process. Seed production and germination are heavily tied to solar cycles of seasons and varying power density. Alkaloids may also protect some plants from destruction by certain insect species. This suggests to me that they may affect the electric charge variance in flowers to lure or defend against insects and may interact in the physiochemical process of generation of the DC electric current present in plants.

This may BETTER explain how they affect neurologic function in humans by altering the charges associated with the DC electric current in man. It is now well known that morphine has major effects on wound healing when used chronically. the more opiates you use the less well-wound healing works. Generally, an alkaloid contains at least one nitrogen atom in an amine-type structure—i.e., one derived from ammonia of the urea cycle at Kreb’s bicycle by replacing hydrogen atoms with hydrogen-carbon groups called hydrocarbons. This or another nitrogen atom can be active as a base in acid-base reactions. All acid-base reactions are based on redox chemistry on Earth.

The pH has massive effects on the size of the exclusion zone in water created by cells at Kreb’s bicycle. This is likely how these drugs affect the nervous system and act as chemical proxies for a LACK of dopamine = lowered DC electric charge from sunlight/grounding. This is also why many alkaloids possess local anesthetic properties as well (cocaine).

Local anesthetics work by lowering the pH of the environment around nerves to disrupt action potentials. Their molecular structures, however, are not as precise in their actions as dopamine and we get variable results from them because of these chemical changes. Dopamine is a biologic amine and it is made from tyrosine an aromatic amino acid. I spoke about this in my Vermont 2017 and 2018 talks. You can find the 2018 talk available on Patreon now.

 

 

Amines that are isolated from plants are known as alkaloids. This is why these drugs are dopamine analogs in animals. Their ring structure absorbs UV light to varying degrees compared to dopamine’s ability to absorb a specific amount of light because of tyrosine’s ring. When you marry a microwaved/RF environment that is blue lit chronically, It creates a lack of DHA in the short and long loops of the retina and in the skin. With a chronic lack of DHA in cell membranes in the SCN, you create the perfect storm to create the low dopamine state. If it lasts long enough you’ll get a serious neuro-degenerative disease like AMD and Parkinson’s disease.

 

 

Your Vitamin A cycle in the retina and brain is broken and this altered retinol structure destroys all photoreceptors for light. Dopamine photoreceptor is tyrosine. Melanopsin is our blue light detector. It is destroyed by this process and when these things happen this leads to CNS inflammation and lowered melatonin levels. The lowered melatonin levels cause further mitochondrial breakdown because melatonin is a protector antioxidant for mtDNA. When melatonin is destroyed so is dopamine because both are made from aromatic amino acids in the retina in AM sunlight when IR-A and UV-A are present diurnally. So when you reach for your morning smoke or coffee you have announced to the world and to yourself your dopamine and melatonin levels are destroyed. If you do this chronically and think you’re bulletproof, you start using nootropics and opiates and THC and CBD oil to increases your defective DC electric current. Your sex steroid hormones will also drop and your hormone panel becomes flat lined.

 

 

The need for chronic use of these chemicals is a sign that you’re getting worse, not better because you need larger dopamine hits as your sleep worsens. It leads to a complete uncoupling of ubquitination from melatonin cycles in the brain, skin, gut and lungs. Blue light frequency destroys DHA in the retina further slowing the SCN clock in relation to the peripheral organ clocks. As melatonin drops so will DHA in the short and long loops of the eye and every cell membrane in your body. This means you need more DHA and less coffee and chocolate.

 

 

Few people in this low dopamine state will understand it even when I explain it in extreme detail. This blog is evidence of this. This means your SCN no longer is running quicker than the peripheral clocks in front of every gene in your tissues below the central retinal pathways and it and it destroys the timing mechanism built into all circadian controllers.

 

 

What does that mean to a Black Swan mitochondriac? Mitochondrial diseases are coming your way fast and furious the more you “feel the need” to use these chemicals. DHA loss destroys the optical relationship between light bending in our gravitational field to uncouple light cycles from the cell cycle and metabolism. This is why blue light destroys DHA and melatonin levels in humans quickly. These chemicals or a dietary change is not going to fix this clock speed mismatch between the SCN and the organ clocks, but solar light can because it is what makes dopamine and melatonin in your eye every morning if you allow it happen. Most of you DON’T  understand these things, much less do them in your tech world, so you rely on guys to sell you coffee, drugs, or stories how you can be diseased proof with some supplement. My advice is simple. ‘SUN’plements over all supplements is the mitochondriac perspective.  You must live more connected to the sun and Earth and you won’t have a ‘biochemical need’ for things to supplement your DC electric current deficits.

CITES:

My blog series on my website.

 

You need to realize how the sun makes active D3.  The hack is in creating your solar callus first to increase the skin’s ability to make cathelicidin.  1,25-Dihydroxyvitamin D3, the active form of vitamin D, not the storage version D25 OH,  is a major regulator of the expression of the CATIONIC antimicrobial peptide cathelicidin, not only in monocytes but also in epidermal keratinocytes. The involvement of cathelicidin in wound healing and skin diseases as diverse as psoriasis, rosacea, and atopic dermatitis.  This means the hack is learning how to create your solar callus is the key in creating new opportunities for the use of vitamin D in your own life, subtracted from dermatology dogma.

 

 

For example, Rosacea is caused is a lack of UV light and a serious dose of melanopsin dysfunction from blue lit and nnEMF.  Proper UV light assimilation comes from ideal red light exposure at the correct time of the day.

 

 

Increased expression of cathelicidin antimicrobial peptide (CAMP) is related to the pathogenesis of rosacea. CAMP plays a crucial role in antimicrobial defenses, such as the killing of mycobacteria. CAMP gene expression is regulated by vitamin D-dependent (VDR) and vitamin D-independent (C/EBPα) transcription factors. VDR-dependent CAMP expression is sufficient during the summer months in Nordic countries, but insufficient during Nordic winters, due to low ultraviolet (UV) levels.

 

 

It also can be excerbated by excessive blue light, RF, or microwave exposure of the skin in the modern world. Different parts of the spectrum have different effects on the melanopsin and retinol weak covalent bond. All of these other frequencies are capable of altering the bond in ways medicine does not account for in the skin or eye.

 

 

These changes can also occur due to light stressed environments in strong UV places as well. Historically, the high latitude living Celts are and this may have helped them overcome this geographical disadvantage of deficient CAMP production during the winter through an as-yet undefined acquired mutation that activates the alternative vitamin D-independent CAMP promoter C/EBPα.

 

 

C/EBPα is the downstream transcription factor of Toll-like receptor (TLR)-mediated innate immune reactions and endoplasmic reticulum (ER) stress responses. This is why I believe most autoimmune conditions are always linked to a chronic lack of key solar frequencies in the immune system at some level. At the molecular level, all clinical trigger factors for rosacea can be regarded as ER stressors. UV light can repair this by augmenting autophagy and apoptosis by controlling sulfation and electrophiles in blood plasma.

 

 

The best mitohack is contained in the cite below.

A mitohack I have done myself is here: One can use reptile light to improve you live in a poor quantum yield region for UV light once you learn how to build your solar callus. The mechanism of Finsen’s UV treatment of lupus vulgaris by UVA/B is critical- and ER stress-mediated upregulation of CAMP expression happens from this light alone. The two pictures above show the use of the Finsen lamp. You need to know what you are doing when you do this and most people in the public will not have the understanding without deep reading.

 

 

 

Rosacea could therefore be described as an epigenetic modification in people born into a bad light quantum yield world like the Celts’ were. In essence biology’s epigenome gave you “inborn Finsen lamp” in your skin if you know how to use it properly. If your skin disease persists then it tells you that your light environment is toxic to your quantum yield in some way that you do not realize.

One last bit of wisdom about the skin: Vitiligo is also improved by full spectrum sunlight as the picture of this patients shows below. I bet the King of Pop would have liked that information. He spent 50 years of his life in blue light and around electrified instruments and tried to fix his skin with plastic surgery with disasterous results. When you know better, you do better. When you don’t, you might die early.

 

 

 SUMMARY:

When your skin is suboptimal this is your body telling you to look to the light environment you chose to live under.  This choice leads to melanopsin dysfunction in the skin.  The result is simple.  Your life manifest in one picture (below) and very few realize it, much less believe it.  When you only have mostly blue light RF and microwaves around your skin these are the diseases you’ll likely get.  Skin already filters sunlight via its own optical window and it does react well when terrestrial sunlight is filtered by man made design.

I am here to show you what you are allowing to happen to your brain retina and skin in your current location because of the portion of light subtracted from sunlight in the visible spectrum that skews your perspective of reality. Our sun only emits light that makes up 1 billionth of the total electromagnetic spectrum of light in the visible spectrum. Your cells emit even a smaller part of the spectrum and focus its light in the blue spectrum of light. This light has massive neurologic optical effects on your skin, brain, and eyes. So when I tell you, your cell phone is capable of causing brain damage is it a PARADOX or REALITY? Everything unfolds in its proper time / order, in a cause / effect universe. Limited human perspective, fails to see situations from the appropriate casual level. Is it a great idea to explain away ‘paradox’ ??? Understand the effect of light on cells is the basis of quantum biology. It is a new field in medicine that is part of quantum mechanics. So far the basis of quantum mechanics says that is a pipe dream to believe that cause and effect is an absolute truth in reality because in the quantum mechanical experiments over the last 80 years, the data reveal that cause and effect might really be an illusion built around probability. This offends most humans common sense, but it is a very true statement. I like embracing paradox to understand things I presently do not comprehend or that I am blind too because of what I was taught to believe.

 

 

It opens my mind to accept the things I don’t or can’t see. This lack of vision, of how using an even more restrictive part of the electromagnetic spectrum in tech screens really is fueling our Dunning Kruger effect. This is why no one can fathom that our cell phones, laptops, and TV’s can cause these skin diseases and we continue to allow the technology manufacturer to control our behavior and our thinking by using these filtered lights.

Embracing discomfort and paradox forces me to look where no one has looked before. It raises the point, how long should we continue to hold onto the belief paradigm of cause and effect? I’m convinced that all of nature’s best solutions are often the ones that are counterintuitive – that challenge conventional thinking – and end in breakthroughs. It is always easier to do things the same old way…why change? To fight this, keep your dissatisfaction index high and break with tradition. Don’t be too quick to accept the way things are being done now with respect to light in any aspect of modern life.

You must question whether there’s a better way of understanding your present situation. Very often you will find that once you make this break from the usual way – and incidentally, this is probably the hardest thing to do—and start on a new track your horizon of new thoughts immediately broadens. New ideas flow in like water when you live in nature under the power of the visible spectrum of the sun. Always keep your interests broad with respect to light – don’t let your mind be stunted by a limited view.

 

 

A lack of full spectrum solar exposure during the day, or getting man’s light at night is the most common reason for disease epidemics today, and in my opinion, the and most overlooked issue in all of medicine these days. When blue light, RF, or microwaves affects melanopsin adenosine biology is altered.  This is how adenosine rises and it is when melanopsin receptors are being recycled.  Proper ocular melatonin cycling requires that these two frequencies (UV/IR) be present in the AM to stimulate the regeneration processes in the eye during daytime. This quantized process also requires ABSENCE of blue/green light between 400-560nm post sunset!!!! When these things are off the result always = INFLAMMATION = too many protons (deuterium) and/or not enough electrons at the mitochondrial cellular level = lowered melatonin levels in the eye, brain, blood plasma.  The same is now true in the skin and subcutaneous fat.  That is how leptin resistance in a tissue occurs.  Melanopsin dysfunction turns retinol into a bomb and that bomb ruins the aromatic rings in melanopsin, leptin, and melatonin to ruin their ability to communicate with the hypothalamus to give accurate information about energy balance because information quanta is LOST.

CITES:

https://www.patreon.com/posts/13077291