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How do you know you are not as connected to Earth and sun chronically?

You’ll begin to feel better when you use opiates, weed, cocaine, and caffeine in coffee tea or chocolate. Eventually, you cannot live your life without any of some of them.

Morphine, Nicotine, and Cocaine – are all in caffeine’s chemical family. See the picture below. Ask yourself, why are the USE of these drugs on a massive uptick? Might it be the world’s ability to make dopamine in the eye and body is dropping in a blue-lit, RF/microwaved world we’ve built? A mitochondriac gets that perspective but do you? Do you see why the opiate crisis is massive or why a guy from the West Coast of the USA can convince many humans to drink a lot of coffee and add MCT oil or butter to it? Might this be a sign of a broken light environment or too much technology in your environment?

 

THE BIOCHEMICAL EFFECT OF TOO MUCH BLUE OR nnEMF

The group of chemicals that caffeine belongs to are known as alkaloids and they are the most addictive substances known to man. At various points in history, Morphine (Opium) and Nicotine (Tobacco) were socially sanctioned for daily use, but this trend reversed when the terrible side effects of habitual usage emerged in the 20th century. The 20th century brought with it industrialization and brought man from outside to inside to work in factories to manufacture things. Might it have been that migration, which blocked man from the sun informative light to photosynthetically make dopamine, been further worsened because technology and the electric power gird zapped more dopamine from mankind and then we defaulted to other bio-molecules with hexagonal rings that absorb light to replace the lost dopamine? I think you know my answer already.

 

 

 

Caffeine is no less addictive for many people, and the long-term effects on health are becoming increasingly apparent as average daily usage soars. It is no panacea for a low heteroplasmy rate either. Key conditions that caffeine significantly aggravates in my clinical experience are: stress response of all kinds, all muscle pain, most digestive complaints, liver metabolism, high blood pressure, PMS, insomnia, fatigue, weight loss AND weight gain, arthritis, blood sugar imbalances, skin conditions, and many mood disorders.

Is caffeine like sugar in the electrical dance between flower and bee?

Yep……

Alkaloids are found primarily in plants and are especially common in certain families of flowering plants. Flowers coming from the Earth assume the net negative charge of the Earth as an anode and therefore have net negative electric charge.  This negative charge draws bees to them for pollination. Bees and most insects are positively charged in nature, and this is why they emit light like the sun.  Recall that the sun is a cathode ray.  This is why the sun and Earth have opposite changes and why when the sun hits the Earth free electrons are given off the surface of Earth just like any anode does and humans take advantage of this by having sweat glands in their feet to absorb those free electrons.  If one wear rubber shoes all the time or clothing over their skin and never gets this electrical stimulus will they be driven to drink more coffee/tea with caffeine?  MY ANSWER IS YES.  The more disconnected you are to nature the lower your electrical potential is.  This is called your redox potential.  The lower it is the more you will seek the caffeine family of drugs to make up  the deficit.

 

 

BACK TO THE FLOWER BEE ANALOGY:

 

 

Flowers grow because of sunlight and photosynthesis and normally have a -30V electric field. The function of alkaloids in plants is poorly understood but I have a sense it is related to flowers having the ability to alter their charges for many biologic reasons tied to how they sense their environments. In some plants, the concentration of alkaloids increases just prior to seed formation and then drops off when the seed is ripe, suggesting that alkaloids may play a role in this process. Seed production and germination are heavily tied to solar cycles of seasons and varying power density. Alkaloids may also protect some plants from destruction by certain insect species. This suggests to me that they may affect the electric charge variance in flowers to lure or defend against insects and may interact in the physiochemical process of generation of the DC electric current present in plants.

This may BETTER explain how they affect neurologic function in humans by altering the charges associated with the DC electric current in man. It is now well known that morphine has major effects on wound healing when used chronically. the more opiates you use the less well-wound healing works. Generally, an alkaloid contains at least one nitrogen atom in an amine-type structure—i.e., one derived from ammonia of the urea cycle at Kreb’s bicycle by replacing hydrogen atoms with hydrogen-carbon groups called hydrocarbons. This or another nitrogen atom can be active as a base in acid-base reactions. All acid-base reactions are based on redox chemistry on Earth.

The pH has massive effects on the size of the exclusion zone in water created by cells at Kreb’s bicycle. This is likely how these drugs affect the nervous system and act as chemical proxies for a LACK of dopamine = lowered DC electric charge from sunlight/grounding. This is also why many alkaloids possess local anesthetic properties as well (cocaine).

Local anesthetics work by lowering the pH of the environment around nerves to disrupt action potentials. Their molecular structures, however, are not as precise in their actions as dopamine and we get variable results from them because of these chemical changes. Dopamine is a biologic amine and it is made from tyrosine an aromatic amino acid. I spoke about this in my Vermont 2017 and 2018 talks. You can find the 2018 talk available on Patreon now.

 

 

Amines that are isolated from plants are known as alkaloids. This is why these drugs are dopamine analogs in animals. Their ring structure absorbs UV light to varying degrees compared to dopamine’s ability to absorb a specific amount of light because of tyrosine’s ring. When you marry a microwaved/RF environment that is blue lit chronically, It creates a lack of DHA in the short and long loops of the retina and in the skin. With a chronic lack of DHA in cell membranes in the SCN, you create the perfect storm to create the low dopamine state. If it lasts long enough you’ll get a serious neuro-degenerative disease like AMD and Parkinson’s disease.

 

 

Your Vitamin A cycle in the retina and brain is broken and this altered retinol structure destroys all photoreceptors for light. Dopamine photoreceptor is tyrosine. Melanopsin is our blue light detector. It is destroyed by this process and when these things happen this leads to CNS inflammation and lowered melatonin levels. The lowered melatonin levels cause further mitochondrial breakdown because melatonin is a protector antioxidant for mtDNA. When melatonin is destroyed so is dopamine because both are made from aromatic amino acids in the retina in AM sunlight when IR-A and UV-A are present diurnally. So when you reach for your morning smoke or coffee you have announced to the world and to yourself your dopamine and melatonin levels are destroyed. If you do this chronically and think you’re bulletproof, you start using nootropics and opiates and THC and CBD oil to increases your defective DC electric current. Your sex steroid hormones will also drop and your hormone panel becomes flat lined.

 

 

The need for chronic use of these chemicals is a sign that you’re getting worse, not better because you need larger dopamine hits as your sleep worsens. It leads to a complete uncoupling of ubquitination from melatonin cycles in the brain, skin, gut and lungs. Blue light frequency destroys DHA in the retina further slowing the SCN clock in relation to the peripheral organ clocks. As melatonin drops so will DHA in the short and long loops of the eye and every cell membrane in your body. This means you need more DHA and less coffee and chocolate.

 

 

Few people in this low dopamine state will understand it even when I explain it in extreme detail. This blog is evidence of this. This means your SCN no longer is running quicker than the peripheral clocks in front of every gene in your tissues below the central retinal pathways and it and it destroys the timing mechanism built into all circadian controllers.

 

 

What does that mean to a Black Swan mitochondriac? Mitochondrial diseases are coming your way fast and furious the more you “feel the need” to use these chemicals. DHA loss destroys the optical relationship between light bending in our gravitational field to uncouple light cycles from the cell cycle and metabolism. This is why blue light destroys DHA and melatonin levels in humans quickly. These chemicals or a dietary change is not going to fix this clock speed mismatch between the SCN and the organ clocks, but solar light can because it is what makes dopamine and melatonin in your eye every morning if you allow it happen. Most of you DON’T  understand these things, much less do them in your tech world, so you rely on guys to sell you coffee, drugs, or stories how you can be diseased proof with some supplement. My advice is simple. ‘SUN’plements over all supplements is the mitochondriac perspective.  You must live more connected to the sun and Earth and you won’t have a ‘biochemical need’ for things to supplement your DC electric current deficits.

CITES:

My blog series on my website.

 

You need to realize how the sun makes active D3.  The hack is in creating your solar callus first to increase the skin’s ability to make cathelicidin.  1,25-Dihydroxyvitamin D3, the active form of vitamin D, not the storage version D25 OH,  is a major regulator of the expression of the CATIONIC antimicrobial peptide cathelicidin, not only in monocytes but also in epidermal keratinocytes. The involvement of cathelicidin in wound healing and skin diseases as diverse as psoriasis, rosacea, and atopic dermatitis.  This means the hack is learning how to create your solar callus is the key in creating new opportunities for the use of vitamin D in your own life, subtracted from dermatology dogma.

 

 

For example, Rosacea is caused is a lack of UV light and a serious dose of melanopsin dysfunction from blue lit and nnEMF.  Proper UV light assimilation comes from ideal red light exposure at the correct time of the day.

 

 

Increased expression of cathelicidin antimicrobial peptide (CAMP) is related to the pathogenesis of rosacea. CAMP plays a crucial role in antimicrobial defenses, such as the killing of mycobacteria. CAMP gene expression is regulated by vitamin D-dependent (VDR) and vitamin D-independent (C/EBPα) transcription factors. VDR-dependent CAMP expression is sufficient during the summer months in Nordic countries, but insufficient during Nordic winters, due to low ultraviolet (UV) levels.

 

 

It also can be excerbated by excessive blue light, RF, or microwave exposure of the skin in the modern world. Different parts of the spectrum have different effects on the melanopsin and retinol weak covalent bond. All of these other frequencies are capable of altering the bond in ways medicine does not account for in the skin or eye.

 

 

These changes can also occur due to light stressed environments in strong UV places as well. Historically, the high latitude living Celts are and this may have helped them overcome this geographical disadvantage of deficient CAMP production during the winter through an as-yet undefined acquired mutation that activates the alternative vitamin D-independent CAMP promoter C/EBPα.

 

 

C/EBPα is the downstream transcription factor of Toll-like receptor (TLR)-mediated innate immune reactions and endoplasmic reticulum (ER) stress responses. This is why I believe most autoimmune conditions are always linked to a chronic lack of key solar frequencies in the immune system at some level. At the molecular level, all clinical trigger factors for rosacea can be regarded as ER stressors. UV light can repair this by augmenting autophagy and apoptosis by controlling sulfation and electrophiles in blood plasma.

 

 

The best mitohack is contained in the cite below.

A mitohack I have done myself is here: One can use reptile light to improve you live in a poor quantum yield region for UV light once you learn how to build your solar callus. The mechanism of Finsen’s UV treatment of lupus vulgaris by UVA/B is critical- and ER stress-mediated upregulation of CAMP expression happens from this light alone. The two pictures above show the use of the Finsen lamp. You need to know what you are doing when you do this and most people in the public will not have the understanding without deep reading.

 

 

 

Rosacea could therefore be described as an epigenetic modification in people born into a bad light quantum yield world like the Celts’ were. In essence biology’s epigenome gave you “inborn Finsen lamp” in your skin if you know how to use it properly. If your skin disease persists then it tells you that your light environment is toxic to your quantum yield in some way that you do not realize.

One last bit of wisdom about the skin: Vitiligo is also improved by full spectrum sunlight as the picture of this patients shows below. I bet the King of Pop would have liked that information. He spent 50 years of his life in blue light and around electrified instruments and tried to fix his skin with plastic surgery with disasterous results. When you know better, you do better. When you don’t, you might die early.

 

 

 SUMMARY:

When your skin is suboptimal this is your body telling you to look to the light environment you chose to live under.  This choice leads to melanopsin dysfunction in the skin.  The result is simple.  Your life manifest in one picture (below) and very few realize it, much less believe it.  When you only have mostly blue light RF and microwaves around your skin these are the diseases you’ll likely get.  Skin already filters sunlight via its own optical window and it does react well when terrestrial sunlight is filtered by man made design.

I am here to show you what you are allowing to happen to your brain retina and skin in your current location because of the portion of light subtracted from sunlight in the visible spectrum that skews your perspective of reality. Our sun only emits light that makes up 1 billionth of the total electromagnetic spectrum of light in the visible spectrum. Your cells emit even a smaller part of the spectrum and focus its light in the blue spectrum of light. This light has massive neurologic optical effects on your skin, brain, and eyes. So when I tell you, your cell phone is capable of causing brain damage is it a PARADOX or REALITY? Everything unfolds in its proper time / order, in a cause / effect universe. Limited human perspective, fails to see situations from the appropriate casual level. Is it a great idea to explain away ‘paradox’ ??? Understand the effect of light on cells is the basis of quantum biology. It is a new field in medicine that is part of quantum mechanics. So far the basis of quantum mechanics says that is a pipe dream to believe that cause and effect is an absolute truth in reality because in the quantum mechanical experiments over the last 80 years, the data reveal that cause and effect might really be an illusion built around probability. This offends most humans common sense, but it is a very true statement. I like embracing paradox to understand things I presently do not comprehend or that I am blind too because of what I was taught to believe.

 

 

It opens my mind to accept the things I don’t or can’t see. This lack of vision, of how using an even more restrictive part of the electromagnetic spectrum in tech screens really is fueling our Dunning Kruger effect. This is why no one can fathom that our cell phones, laptops, and TV’s can cause these skin diseases and we continue to allow the technology manufacturer to control our behavior and our thinking by using these filtered lights.

Embracing discomfort and paradox forces me to look where no one has looked before. It raises the point, how long should we continue to hold onto the belief paradigm of cause and effect? I’m convinced that all of nature’s best solutions are often the ones that are counterintuitive – that challenge conventional thinking – and end in breakthroughs. It is always easier to do things the same old way…why change? To fight this, keep your dissatisfaction index high and break with tradition. Don’t be too quick to accept the way things are being done now with respect to light in any aspect of modern life.

You must question whether there’s a better way of understanding your present situation. Very often you will find that once you make this break from the usual way – and incidentally, this is probably the hardest thing to do—and start on a new track your horizon of new thoughts immediately broadens. New ideas flow in like water when you live in nature under the power of the visible spectrum of the sun. Always keep your interests broad with respect to light – don’t let your mind be stunted by a limited view.

 

 

A lack of full spectrum solar exposure during the day, or getting man’s light at night is the most common reason for disease epidemics today, and in my opinion, the and most overlooked issue in all of medicine these days. When blue light, RF, or microwaves affects melanopsin adenosine biology is altered.  This is how adenosine rises and it is when melanopsin receptors are being recycled.  Proper ocular melatonin cycling requires that these two frequencies (UV/IR) be present in the AM to stimulate the regeneration processes in the eye during daytime. This quantized process also requires ABSENCE of blue/green light between 400-560nm post sunset!!!! When these things are off the result always = INFLAMMATION = too many protons (deuterium) and/or not enough electrons at the mitochondrial cellular level = lowered melatonin levels in the eye, brain, blood plasma.  The same is now true in the skin and subcutaneous fat.  That is how leptin resistance in a tissue occurs.  Melanopsin dysfunction turns retinol into a bomb and that bomb ruins the aromatic rings in melanopsin, leptin, and melatonin to ruin their ability to communicate with the hypothalamus to give accurate information about energy balance because information quanta is LOST.

CITES:

https://www.patreon.com/posts/13077291

 

The light bulb became reality in 1874. The power grid was born in 1893. But when did the history of electromagnetic hypersensitivity show up in humans? It showed up as soon as humans invented the telegraph. Back in the mid to late 1800’s no one walked around naked making enough Vitamin D (70ng/dl for EHS from sun). In fact, they were heavily clothed which lowered their redox and Vitamin D levels. When you added in the Telegraph we began to see SICKNESS manifest and no one seems to know it. nnEMF was a telegraph issue as well. How do we know this? French Physician, Dr. Ernest Onimus, treated Telegraph Operators in Paris during the 1870s, who were continuously exposed to electric and alien magnetic fields, as suffering from heart palpitations, dizziness, insomnia, poor eyesight, headaches, exhaustion, depression, memory loss & mental illness. Black Swans know this story. If you don’t you might want to consider becoming one. Most people think nothing could possibly happen prior to the advent of the light bulb. They are not wise or deep thinkers. The Carrington event happened in 1859 and killed several telegraph operators via jump conduction. That is what a CME can do to via a conductor/insulator. Just wait til 5G. That is what it can do as well. None of you know what is coming……Black Swans do.

 

 

 

Rockefeller’s story is hard enough to swallow but J.P. Morgan was far worse when he “Morganized” the Electric Power industry. Morgan was financially stronger than the US federal government.

 

 

VIDEO

Being a black swan is really waging war on what created this paradigm and that paradigm was built from the history of Rockefeller, J.P Morgan, And Carnagie.

Vision without execution leads to hallucination was the mantra of Edison.  Little did anyone know if that vision is powered by anything invented by Edison or Tesla and funded by J.P. Morgan would lead to the human disease epidemics of the modern world.  One family who did understand the implications of the collateral damage was the Rockefellers.

They made products to sell into the sickness industry these industrialists created for humanity.  This was smart capitalism and ruthless human behavior.  The Rockefeller family became staples of the democratic party in the USA soon after the Republicans ruined the Standard oil trusts in the first years of the 20th century.

 

 

How did Morgan play his role in this story?  He brought an end to the railroad wars and failing companies on Wall Street by buying and connecting them to create the first superhighway to connect large cities together.  You can see the idea at the 6:30 part of the video below.

VIDEO

J.P. Morgan found his industry to build when he found Edison.  What Junius did for banking, J.P did for the electric power industry at the turn of the century.  At the 8:30 time slot you can see how Junius Morgan warned his son about getting into business that was too risky, but J.P. Morgan was far more aggressive than his dad.

He did…….and he died from the 4000 sq feet of electric lines installed in his house on 5th Ave in NYC.  That is the city I grew up in.  ConEd is the power company.  This story is close to my heart because I learned about it at the Museum History.

J.P. Morgan wanted to kill the kerosene lamps of Rockefeller and make them electric and put him out of business.

Rockefeller made sure his family would make money on the big risky bet of J.P. Morgan.  Morgan thought hitting the kerosense lanp would hurt Rockefeller but he did not realize that Rockefeller process of making kerosene made a waste product called gasoline.  Gasoline was found to work well in combustible engines and Rockefeller simply took his waste product and sold it to Henry Ford cheaply to power the assembly lines of the industrial economy.  Electric light had no effect on the Rockefeller wealth……..until the Rockefellers found out how to make money off of people who got ill from electric power.  That is when the Rockefellers got involved in medicine and funded Big Pharma and the American Cancer Society and pushed the federal government into the Food and drug regulation racket.

Then they made a ton of money off of electric power.  At the turn of the century cancer was a very rare diseases.  Today it is a dominat killer of humans and Rockefeller and J.P. Morgan’s business still reap the rewards on until this day.

Funny thing about the Morgan’s, Everyone who lived in that house in 5th Ave got electrosensitive disease tied to mitochondrial damage.  It seems the people in the Rockefeller foundation doing research made that connection early on too.   Rockefeller actually fueled the marketing campaign that electricty was dangerous to health because scientist he was funding were finding out electric power did have biologic effects in the Early 1900’s.

In the spring of 1852, an illness struck which was to become more common as his life progressed. Rheumatic fever left him in so much pain that he could not walk, and his father Junius sent him to the Azores to recover under sunlight and in nature.

He convalesced there for almost a year, then returned to the English High School in Boston to resume his studies. After he graduated, his father sent him to Bellerive, a school in the high altitude Swiss village of La Tour-de-Peilz, where he gained fluency in French quickly.  I often wonder how the UV light there helped him live to 75 considering the risks he took in his 5th Ave mansion with Edison’s lights.   His father then sent him to the University of Göttingen in order to improve his German. He attained a passable level of German within six months and also a degree in art history, then traveled back to London via Wiesbaden, with his formal education complete.  He loved collecting art and half of his net worth was in paintings when he died.

Morgan backed Edison with 83 million dollars because he believed he had no competition.  It turns out Tesla was in Edison’s employ and already had built the AC motor that used higher voltage Alternating current.  Edison was a DC supporter.  Morgan misplayed his hand.  Tesla quit and found George Westinghouse to fund his ideas.

J.P. Morgan realized he bet on the wrong horse.  And the electric power wars began.

J.P. Morgan used his influence on Wall Street during the 1929 depression to gain control of Tesla patents.  Westinghouse was going bankrupt and told Tesla he could not pay his royalties because of the crash……so Tesla signed his patents to Westinghouse and alleviated the financial presure of the depression to continue the AC power grid build out.  The build out was quite expensive and Westinghouse eventually lost all of his patents to J.P. Morgan in a Wall Street power play.

J.P. Morgan built General Electric with this blue print.   After this stunt the US power grid became an AC grid.  The Electric current wars did Edison in because of stunts he back on prison executions and the electrocution of an elephant.  After this, J.P. Morgan became convinced he had to bury DC electric power and get Tesla’s patents and that is what he did.

VIDEO

In 1892 Morgan arranged the merger of Edison General Electric and Thomson-Houston Electric Company to form General Electric. He also played important roles in the formation of the United States Steel Corporation, International Harvester, J.P. Morgan Bank and AT&T. At the height of Morgan’s career during the early twentieth century, he and his partners had financial investments in many large corporations and had significant influence over the nation’s high finance and United States Congress members. He directed the banking coalition that stopped the Panic of 1907.

This panic is how we got the US FEDERAL reserve bank.  It is also how we got tied up with the money of the Rothschild’s in Europe.

The Federal Treasury was nearly out of gold in 1895, at the depths of the Panic of 1893. J.P. Morgan had put forward a plan for the federal government to buy gold from his and European banks his father built,  but it was declined in favor of a plan to sell bonds directly to the general public to overcome the crisis.

J. P. Morgan, sure there was not enough time to implement such a plan, demanded and eventually obtained a meeting with democratic president  Grover Cleveland where he claimed the government could default that day if they didn’t do something.

Morgan came up with a plan to use an old civil war statute that allowed Morgan and the Rothschilds to sell gold directly to the U.S. Treasury, 3.5 million ounces, to restore the treasury surplus, in exchange for a 30-year bond issue.  That is where the modern 30 year treasury bond of the US government comes from folks.

The episode saved the Treasury, but hurt Cleveland’s standing with the agrarian wing of the Democratic Party, and became an issue in the election of 1896 when banks came under a withering attack from William Jennings Bryan. Rockefeller, Morgan, and Carnagie with the Wall Street bankers donated heavily to Republican William McKinley, who was elected in 1896 and re-elected in 1900 (20 million dollars in 1896 is worth billions today in influence).

Rockefeller, Morgan, and Carnagie demanded that McKinley put their arch enemy on the ticket as Vice President because in those days the office had no power to reach their businesses.  That plan back fired when McKinley was shot and killed by a factory worker.  Teddy Roosevelt became president and he went after Morgan and Rockefeller as soon as he became president.

Just how powerful was Morgan?    In December 1912, one year before his death,  Morgan testified before the Pujo Committee, a subcommittee of the House Banking and Currency committee. The committee ultimately concluded that a small number of financial leaders was exercising considerable control over many industries. The partners of J.P. Morgan & Co. and directors of First National and National City Bank controlled aggregate resources of $22.245 billion, which Louis Brandeis, later a U.S. Supreme Court Justice, compared to the value of all the property in the twenty-two states west of the Mississippi River in 1912.   That is ridiculous power.

In today’s dollars that is 60 trillion dollars worth of value.  That is a big stick folks.

Morgan made some big mistakes……so his Dad was correct about electricity.

Edison vs Tesla power wars is a classic error by Morgan, but he still won when he pushed Westinghouse out of business using financial chicanery.

Morgan did not always invest well, as several failures demonstrated.

In 1900, the inventor Nikola Tesla convinced Morgan he could build a trans-Atlantic wireless communication system (eventually sited at Wardenclyffe) that would outperform the short range radio wave-based wireless telegraph system then being demonstrated by Guglielmo Marconi in Europe. Morgan agreed to give Tesla $150,000 (equivalent to $4,412,400 in 2017) to build the system in return for a 51% control of the patents. Almost as soon as the contract was signed Tesla decided to scale up the facility to include his ideas of terrestrial wireless power transmission to make what he thought was a more competitive system.

Morgan considered Tesla’s changes, and requests for the additional amounts of money to build it, a breach of contract and refused to fund the changes. With no additional investment capital available, the project at Wardenclyffe, Long Island was abandoned in 1906, and never became operational.  This was the forebearer of the cell phone folks.   The base of this tower is still present in NY if you ever visit it.

J.P. Morgan made sure Bell Labs tied to ATT got the ideas from Tesla to make wireless communication possible today. J.P. Morgan banks where the biggest lobbyists for the 1996 FCC law making banks and investors in telecommunications immune from lawsuits if it ever became possible that wireless electric transmissions were deemed dangerous to humans in 1996.  This act is the biggest mistake in US government history in my opinion.

J.P. Morgan’s banks to this day are active in every Federal auction of the electromagnetic spectrum of light the FCC does when network power is sold to the telecom industry.

Just how far reaching is this story…….you know the USDA guideline and the story on cholesterol?  That is also a story tied to Rockefeller, Morgan, and the Rothschild’s fortunes to this day.

 

 

Truth bomb Alert.

81 years-That’s how long we’ve known dietary cholesterol has negligible impact on serum cholesterol.
Not until the 2015 guidelines did the USDA finally admit “cholesterol is not a nutrient of concern”

Are you waiting for them to correct the rest of their mistakes.

In 1937 Columbia University biochemists David Rittenberg & Rudolph Schoenheimer demonstrated that dietary cholesterol had very little if any effect on blood cholesterol. It as never refuted, it didn’t prevent/stop decades of fraudulent Anti-Cholesterol egg-&-butter-bashing that the TItans of business made sure the real truth would never be found out to benefot Big Pharma and cost the federal govermment billions of dollars in prescription drugs.  https://t.co/Cys6qKGVxV

If you shout the lie for half a century or more and then whisper the retraction or omit it altogether.  That lesson was learned from the Rockefeller and the House of Morgan play book folks.  If you look back to see who funded the low fat diet craze you’ll see it was these two families who spent a ton of money in Washington DC to make Ancel Keys ideas policy and law of the land of American medicine using the USDA guidelines.  The Rockefeller and Carangie Foundations to this day are printing money using these techniques of payback.

Both Political Parties in the American political system are behind this scheme and only an outsider threatens it.  Today, that outsider was elected by the people in the 2016 election.  You might be shocked to know that the Rockefeller, Morgan’s, Carnagie’s and the Rothschild control the media companies of the US.  They also own controlling shares of most of the technology companies of the USA.  They are all targets of the current administration.  It might give you a new perspective on truly what is going on behind the scenes in US politics and business today.  The story is very similar to the story laid out in the September 2018 webinar I gave to my members.

The Biophysics of Fluoride also has also has a link to the Rockefeller empire post Standard oil when he built the business of big Pharma from his chemical empire.  His chemical companies came from his oil businesses. Fluoride was a waste chemical like gasoline was and the Rockefeller foundation found a way to get rid of it via dentistry and drugs.  https://t.co/h1DxqvbzoR

Their tentacles are in everything that will get you ill in this country.

WHAT IS THE LATEST PART OF THE PLAYBOOK?

How do you go about undoing decades of manipulation and lies by a media, a ruling class, and celebrity class that is compromised to its core?

You construct an alternative channel to communicate with the public directly.

Then you work with “volunteer propagandists” of good standing to legitimize and publicize ideas and memes — denuding the incumbent rival of its power to set the narratives in medicine. We live in this brave new world now………..and now you know why my blog exists.

 

 

CITES:

http://ushealthmagz.com/2018/07/01/how-rockefeller-founded-big-pharma-and-waged-war-on-natural-cures/

Supplement makers and sellers now use many of the same tactics the Rockefellers used since the break up of Standard oil and few of you realize it.  Maybe now you’ll understand my disdain for pill pushers.

https://www.youtube.com/watch?v=AUpRroefWPk

Cymatics explains life inside a cell in pictures…….it shows how light coming out of a point source like an electric socket can be turned into sound by machines which control the information and energy to make things inside of cell using matter to shape life. In the video, the machine create the sizes and shapes on the speakers. Inside of you, your proteins, transform the light of the sun into sizes and shapes of matter inside of cells. This process is affected by grounding.

 

 

The science of grounding: The sun is a cathode ray who’s light hit earth which acts as the anode. Since it is the third anode from the sun this sets up its harmonic that determines basic morphogenetic process via photo acoustic cymatics.

When a cathode ray hits an anode free electrons are liberated from the anode. This is why humans have sweat glands on their hands and feet. The human breast is also a modified human sweat gland for electron transfer between mother and infants mitochondria.

Those free electrons are liberated according to the sunlight barcode Fraunhofer lines which vary.

 

 

This his how light is used photoelectrically and photo acoustically to power life give it information to organize cells and to drive body plan build out via cymatics by changing light to sound wave which can be controlled magnetically by the Earth magnetic field.

 

 

How does magnetism control matter? With light waves that changed to sound once the light hits a protein in a cell and creates a morphologic pattern. A cell goes even further…….it can alter that pattern by using harmonics of the photoacoustic wave. This is the key to understanding morphogenesis in living systems.

You must hire experts who know the basics of how life organizes to maintain health using magnetic fields to control heat release from mitochondria and photoacoustic waves.

 

 

Grounding is useless if you do not get blue light exposure and nnEMF correct first.

 

 

Today’s lesson on why certain areas are better than others for living systems with mitochondrial damage: An environment with a higher magnetic field strength offers human tissues a massive upgrade because nnEMF and blue light are not as effective as destroying the morphology of a cell because of the higher flux in the Yucatan (3.4 vs 0.4 milligauss) due to the crust being closer to the magnetic dynamo at the surface of the Earth in this area. Then there is the benefit of the Karst effect on the water trapped in the crust for the last 65 million years to create DDW in the cenote system where it normally would not be located based upon the latitude of the crater.

 

 

The science of grounding: the sun is a cathode ray who’s light hit earth which acts as the anode. Since it is the third anode from the sun this sets up its harmonic that determines basic morphogenetic process via photoacoustic cymatics. When a cathode ray hits an anode free electrons are liberated from the anode. This is why humans have sweat glands on their hands and feet. The human breast is also a modified human sweat gland for electron transfer between mother and infants mitochondria. Those free electrons are liberated according to the sunlight barcode Fraunhofer lines which vary. This is how light is used photoelectrically and photoacoustically to power life give it information to organize cells and to drive body plan to build out via cymatics by changing the light to sound wave which can be controlled magnetically by the Earth magnetic field. You must hire experts who know the basics of how life organizes to maintain health.

That magnetic flux in the Yucatan stabilizes stress cellular architecture and it also allows the ATPase to spin faster than it does in areas with a lowered gauss meter reading.

When you move to a new location you still have manufactured blue light to deal with in your eye and skin.

Removal of the blue component of light significantly decreases retinal damage from mitochondria. People forget that “retina” sits in front of the main circadian clock, called the SCN. Chronopathology deals with the subject of disrupted timing in vital biological processes and helps to identify different phases of deviation from the norm for better health.

 

 

A narrow mind will be the most harmful thing you’ll ever own.
The modern world can’t see the obvious health solution because they can’t see the real problem. What one should do when one sees a situation we do not like, we should change it. If we perceive that we can not change it then we must begin to perceive it in a new way to solve it. This new data on melanopsin is such an issue. The light you live with it is the cause of modern demise.

You need to cover your eyes with a good pair of RaOptics glasses and I would cover most of my skin at work too. Then take frequent breaks to get outside where you can de-cloth and get some sun to win.

For those who did not see my webinar on the chiral heat effect and how it links to this post: In all cases, raising the temperature, invokes thermal vibrational and entropic effects. This tends to preferentially stabilize H+ over Deuterium bonds in mitochondria which used to be bacteria. Mother Nature knew exactly what she was doing when she made our stolen bacteria at our core innovate uncoupling proteins and haplotype variations. The more heat you liberate the more deuterium you excrete and the faster ATP is made because the spin rate of the ATPase increases and the TCA cycle performs like a Ferrari and not a Nissan Sentra.

SUMMARY:

Latitude, altitude, and population density are the keys to UV and O2. As altitude increases protons diminish in the atmosphere. At night time as magnetic flux increases there is less positive charge (protons) in the atmosphere and with sunshine in the daytime there is more positive charge in the atmosphere. The sun’s light is a cathode ray. When it hits the Earth in daytime, the Earth acts as an anode in this cosmologic circuit and this allows for more evaporation of water on the planet’s surface to create more protons to dissipate in the atmosphere. Simultaneously the evaporation effect will liberate more electrons from Earth’s surface that our tensegrity system was built to be connected with to Earth’s surface via our limbs.  this is why humans have sweat glands on their palms and feet because we are designed to collect them as an accessory energy source for our massive brains. 

We collect and harvest this energy to store or use to do physiologic work to lower the resistance of our inner mitochondrial membrane in our brain and heart. This reduces the electrical resistance of mitochondria and this helps stimulate autophagy and not apoptosis in those organs.

During daytime, the electric field of Earth is higher than it is at night when light is absent.  Magnetic fields, however, are higher at night, and this is likely why sleep is linked to a loss of the DC electric current in diurnal animals because it supports ATPase spin rates.  https://www.youtube.com/watch?v=fHi61JtVhDw

CITES:

1. https://news.osu.edu/landmark-study-proves-that-magnets-can-control-heat-and-sound/

2. https://pubs.acs.org/doi/abs/10.1021/ja9530376

 

In the 19th century, Dr. E. Babbit, M.D. proved that colored light was capable of healing through the effect on the autonomic nerve fibers in the skin and via the nerves from the eye to the brain. Dr. Spitler proved in the 1930’s that psychiatric illnesses could be cured or improved by using a visual colored monochromatic light source. It is now known that there are at least four effects from light. These are:

1. The optic nerve to the pituitary gland, temporal lobe, and occipital lobe of the brain. This information affects the conscious part of the brain without interpretation.

2. A second nerve bundle from the retina to the hypothalamus, which is a major control area for both the sympathetic and parasympathetic nerves. Dr. Fritz Hollwich, M.D. has shown that color affects neurotransmitter and hormone levels in the brain and spinal cord, which in turn affect the rest of metabolism and biochemistry. No one in the modern nutrition world seems aware of his work in blinded and sighted humans. (Vermont 2017 video)

3. This path goes from the retina to the midbrain, and then to the superior cervical ganglion to the brainstem and then to the pineal gland. This area controls, among other things, our circadian (sleep/wake) cycle. This effect is mediated by melanopsin and retinol in humans in the eye, skin, subcutaneous fat mass, and the arterioles of the skin.

4. The last is a direct effect of the light upon particles that travel in the lymph, blood, and nerves. Researchers at the University of Vienna found that albumin is one of the particles able to be charged by sunlight.  It turns out albumin is made up of a large portion of aromatic amino acids that all absorb UV light in the blood plasma.  It also turns out the pH has a massive effect on how albumincan absorb UV light in the blood plasam.  The charges added to albumin are radically different if it is done by artificial light with blue light present.  After albumin has its topologic charge altered, it is then able to deliver this charge to tissues at distant locations (tissues) in the body.   Here is the original paper on how albumin works in the blood.  The higher one’s albumin ratio is the better they do long term.  This is especially true in most cancers.

Thanks to it, light at specific frequencies in the visible spectrum can act as a powerful tool to stimulate the biochemistry of the brain through the visual system by way of the retinal-hypothalamus brain connection.

INSULIN IS NOT JUST A PANCREATIC BASED HORMONE.

In 1922, Banting and Best found that insulin was the pancreatic hormone that regulates carbohydrate metabolism.  What many people do not know about the insulin story is that ophthalmologist, in 1950 Fritz Hollwich, conducted functional tests of carbohydrate control on blind subjects based upon the methods described by Staub and Traugott.  Their method involved a double alimentary glucose tolerance test given within 60-90 minutes.  They found in healthy people the second dose of grape sugar, which was given when the blood sugar was already falling had no effect or a slight effect on plasma glucose levels.

In his first run of experiments on ten blind subjects using the above methodology, Hollwich obtained negative results deviating from what earlier experiments showed in sighted people.  Hollwich demonstrated that light via the eye had an unknown effect on insulin physiology.  His work was confirmed in 1953 by Fuchs et al, and von Schumann (1953) and by Wassner in 1954.  Hollwich repeated his own experiments with larger numbers of blind patients in 1963, and again with the help of Diekhues in 1967.

After these finding in Europe, the insulin tolerance test of Radoslav became the gold standard. These experiments showed in all cases that in blind patients who received the insulin tolerance test, the blood sugar levels dropped far below the physiologic threshold they expected compared to sighted patients.  Hollwich was the first person in the world who showed that the results of both tests indicated a connection between blindness and a dysfunction of the hypophyseal portion of blood glucose regulation.  This finding is still not well known in modern diabetic research and diabetics with cataracts should be EXPECTED to have substantially different plasma glucose changes than patients without eye disease.

This is also true for diabetics with AMD.  Again none of this is found in the modern literature of diabetes because eye surgery has changed massively since Hollwich’s time.

Hollwich and Diekhues, in 1967 and 1971, did an amazing set of experiments on cataracts patients tested before and after eye surgery.  They showed a significant rise in blood sugar from 87.5mg to 98.5 % post surgery under a constant dietary regimen and light environment.  This variance in the postop Staub-Traugott tolerance test seen in the blind state before surgery and in the state of restored sight post-op can, with the food and light environment controlled served as definitive proof that ambient light’s entry into the eye has a massive influence on the regulation of glucose balance in humans.

Funny how you never hear any food gurus mention these experiments huh?  

This is pretty convenient for Big Pharma too who create drugs just that act on the gut hormone.  None of their solutions deal with the eye or skin.  This is why treating diabetics with just oral or IM injections fails to reverse anything.  I have a sense Big Pharma knows that.  

 

 

We now know, because of Hollwich that insulin is a solar hormone and has a diurnal rhythm independent from glucose intake. Those studies were done in 1974 and 1975 with radioimmune assays by Jarrett in 1974, Lakatuna et al in 1974 and Lestradet et al in 1974, Reinberg et al in 1974, and Thum in 1975. Thum’s paper in 1975, in particular, provides the possibility of assessing the precise means of light experiments with normal-sighted and blind subjects that should be done. None have because of the food guru perspective. Most are not even aware of this work in nutrition research because they only see what they want to see. What happens in the eye and skin when you eat is more important than what you eat. How is that for a truth bomb?

 

 

Most of the modern nutrition studies touted in the LCHF and paleo groups have no ambient light controls for the eye and Hollwich definitive proved in the 1960’s that Banting and Best initial observations of insulin’s effect is radically altered by ambient light via the eye or skin. We can add the skin to Hollwich’s work because in December 2017 we found it is in the skin and subcutaneous fat.

 

 

This is why most of the modern beliefs around carbohydrate metabolism are horribly flawed. I’ve been waiting 4 years for some of the gurus in here to go read his work and realize how much really is not known about ambient light’s effect via the eye and skin now that we found out melanopsin is in both tissues and melanopsin system radically effects the diurnal rhythms of insulin without ANY FOOD in the alimentary tract. Type 2 diabetes (T2D) is not purely a metabolic story tied to food, as you’ve been lead to believe. It has more to do with light via the eye and skin because of Hollwich’s experiments in the blind versus sighted humans from the 1960’s. It has been seriously upgraded by the news we found on melanopsin in the last 8 months. This was the topic of my Vermont 2018 talk. Most people believe diabetes is tied to food and a gut problem. It is not. When the circadian mechanism is off the eneterocytes do not turn over every 24-48 hours and this allows deuterium to enter the liver and this is what really causes diabetes problems most are familiar with. The process, however, begins with blue light exposure in the eye and skin, and this ruins the peripheral clock mechanism in the gut and liver as the picture below shows.

 

 

?The straw that breaks the camel’s back is epigenetic = fake blue light = melanopsin and Vitamin A problem = low melatonin and leads to poor mitochondrial DNA = problem in the eye and skin = a BLUE LIGHT HAZARD in the peripheral gut clocks in the enterocytes that spreads to the liver rapidly. That is really what diabetes is to a mitochondriac. That is what I showed in Vermont 2018.

 

 

?Eat SEASONALLY, but crap LIGHT environment = T2D.

?‍The wise N=1 ➡️ Reasonable seasonal carbs in the AM in summer, but get lots of sun with skin in the game + keep technology in check past 6 PM in any season. Your HbA1c never gets above 4.6 and you won’t need meds or physicians.

Artificial blue light  stimulates the anterior hypothalamus via the central reitnal pathways, by activating the PVN.  Normally sunlight with blue light and red in the AM can help activate the parasympathetic nervous system while stimulating the anterior pituitary hormones.  Subtracting out the red light and adding the blue is our modern problem.   This means that all colors in the bluish spectrum – from blue/green through blue to violet at 400nm is a problem for the gut because of melanopsin and the Vitamin A link to melatonin function.  Blue light via the eye or skin activates digestion and stimulate insulin secretion in the gut without the need for any food in the gut because of how melanopsin lowers melatonin by altering retinol function to ruin photoreceptor function that controls the human circadian mechanism.

VIDEO 1

VIDEO 2

CITES:

Hollwich, F.: The Influence of Ocular Light Perception on Metabolism in Man and in Animal. Berlin, 1985.

https://medicalxpress.com/news/2018-06-diabetes-results-breakdown-epigenetic.html

http://www.jbc.org/content/171/2/767.full.pdf

http://www.biotopics.co.uk/as/insulinproteinstructure.html

Szent-Gyorgyi, A.: Introduction to a Submolecular Biology. Academic Press: N. Y., 1960.

Wurtman, R.: The Effects of Light on the Human Body. In: Scientific American, July 1975, Vol. 233, Nr. 1, S. 68-79.

Gabel, S.: Information Processing in Rapid Eye Movement  Sleep: Possible Neurophysiological, Neuropsychological, and Clinical Correlates. In.: Journal of Nervous and Mental Disease, 175, 1987, S. 193-200.

Toupin, A.: Photic Avtivation and Experimental Data Concerning Colored Stimuli. In: Neurology (Minneap.), 16, 1966, S. 269

Circadian cycles allow for self organizing features and functions in cells. Effectively these cycles are the musics your cells are addicted too. Every AM you are designed to retune the system with the AM sunlight so the music of life contains to reverberate in you cells continuously without interruption.

 

 

Dr. Franz Halberg coined the term “Circadian Rhythm” in the 1950’s, as a result of his studies in what became Chronobiology.  He found our bodies have a repeating cycle, a rhythm lasting about (circa) a day (dian).  He measured these rhythms using the body’s core temperature and levels of activity and referred to the shorter daily waking and sleeping cycles as Ultradian Rhythms.  Sleep centers use EEGs (ElectroEncephaloGraphy) to record brainwave activity during our sleeping ultradian rhythms.  Dr. Halberg discovered the biological processes for producing essential proteins, which are the building blocks of our bodies, are only produced at the start of each ultradian cycle.  The most important ones are ubiquitin, methionine cycles, and proteins made from tryptophan.  Sunlight tunes them all in unique ways with photoentrainment.

The sun directly via its electromagnetic waves to tightly coil DNA/RNA while all versions nnEMF seem to uncoil DNA to affect its energy and information status.  We gained this idea in data on bacteriophages.  They pack their heads with DNA tightly under great force as the bottom left slide shows.

 

 

The area under curve in bottom right graph is a measure of the energy required to pack DNA inside the head of the phage.
Notice the energy increases greatly as more DNA is packed. High energy is due to DNA stiffness and its negative charge.

It appears circadian disruption uncoils DNA for transcription and this induces an electrical status change.  DNA becomes more positive in charge because electrons are lost to aqueous proteins that the ultradian cycles produce to change physiologic function of cells.  This appears to be the basic mechanisms of how things should operate to structure tissues and organize cells.

A priori optimum development in organisms, from single cell to multicellular organisms, or from skeletal cells to a skeleton or limbs exemplify Fibonacci patterns. These developmental patterns consist of two primary characteristics: (1) a number of the organisms structural arrangements may fall into the series 1, 1, 2, 3, 5, 8,…or (2) logarithmic growth based on the ratio of consecutive Fibonacci numbers (1.618033988…, also known as the golden ratio or φ). The growth patterns observed occur throughout nature in the arrangement of skin pores in tetrapods, the spiral shape of snails and sea shells, and the overall structure of plants. Fibonacci numbers occur in atoms and electrons (Huntley, 1969), the DNA molecule (Wahl, 1988), biological cell division (Spears & Bicknell-Johnson, 1998), models of growth and death (Hoggatt & Lind, 1969), bronchial airway segment bifurcations (Goldenberger, West, Dresselhaus, & Bhargava, 1985), experimental growth of tumor nodules (Prokopchuk, 1981), and many other aspects of human biology (e.g., position of facial features, body proportions).

Does musical composition exhibit this organizing pattern too?  Good music does.

Can we use it to help fine tune us when the melanopsin system is de-tuned by poor light choices?  It turns out we can.

Have you ever heard a smart phone interfere with audio equipment at a party or in your car?  This often happens with incoming calls in a car using satellite radio or music apps.  It sounds like bursts of noise when an active cell phone is near a radio or even an MP3 player or Public Address system. Now ask yourself, “How is it possible for something that doesn’t even have  a radio antenna in them to translate cellular signals into these audible bursts?” The answer is simple and central to understanding our current health problems in our electropolluted environments.

 

 

Cell phones, like nearly all wireless devices, use some form of frequency hopping to better utilize available radio spectrum, reduce interference and use lower power. The wireless carrier frequency is usually in the hundreds of megahertz to gigahertz range and at the low power emitted by cell phones the radio waves should pass harmlessly through us.  This is going to change and get substantially worse in most cities when 5G networks that are already built are allowed to broadcast.

So why does epidemiological evidence show a higher risk associated with exposure to these radio waves? The fact you can hear audible bursts with an audio amplifier that isn’t a radio receiver, or a receiver not even tuned to the same carrier frequency, gets to the crux of the electromagnetic problem mankind is facing now.

The culprit is the interval at which the frequency hopping occurs, and not the megahertz to gigahertz carrier frequency of the transmitted bursts. The frequency hopping interval, I am using ‘interval’ instead of ‘frequency’ to avoid confusion with the carrier frequencies, is in the audible range. Our bodies are sensitive to frequencies we can hear, there are frequencies that heal us by working with our circadian cycles and frequencies that make us ill before they kill us because we decouple these cycles from the proteins they make.

It’s as basic as how music affects us. When musical instruments are out of tune or out of harmony with each other the result is agitating noise. And when we hear such a cacophony of dissonance, we turn the music off or leave the performance.

Today, light waves are disrupting the circadian cycles most, but it appears that we might be able to use music to heal.

Self-organized criticality is also a way of understanding complex systems in nature. A power law is a mathematical relationship often referred to as the 1/f law (after the 1/f electronic noise of transistors) in which the distribution of power density at different frequencies (power spectral density) is inversely proportioned to the frequency. Before venturing further, let’s see what the 1/f distribution can tell us about music. This song happens to be one that entrances me when I write about quantum biology.

Fractal musical time is built around power laws buried in nature and sunlight. It is even in the human heartbeat and EEG.

Why do we love music? Have you ever asked yourself this? One reason we enjoy music lies in its balance of predictability and surprise, so researchers claim. They found a musical pitch (frequency) spectrum following a 1/f power law, which achieves this balance of predictability and surprise; but what about musical rhythm? Musical rhythms, especially those of Western classical music, are highly regular and predictable; are they? Daniel Levitin at McGill University Canada, Parag Chordia at Georgia Institute of Technology Atlanta and Vinod Menon at Stanford University California in the United States decided to put that to the test by analyzing the rhythm spectra in 1 788 movements from 558 compositions of Western classical music in their papers.

The rhythmic content of the compositions was systematically measured by noting the duration of the notes and of the rests, transforming the durations into Hz (cycle per second), plotting the data and finding the spectral exponent in the slope of the line obtained. They went through the works of 40 composers in 16 subgenres and found an overwhelming majority of rhythms following the 1/fa power law with a ranging from ~0.5 to ~1. An exponent of 0 would be pure white noise, completely unpredictable, whereas an exponent of 2 and above would be highly predictable. Notably, classical composers whose compositions are known to exhibit nearly identical 1/f pitch spectra demonstrated distinctive 1/f rhythm spectra: Beethoven’s rhythms were among the most predictable, and Mozart’s among the least, with Haydn in between. This song by Tool, above, was built around nature’s key frequency law. The difference in rhythmic predictability is such as to allow composers to identify their compositions uniquely and to distinguish them from works of their contemporaries. No one has made a song like a cadence in this modern piece. What does it say to you as you listen to it? For me the take home is simple: It is not the melody or voice that commands the story;

it is the ear that deciphers the code contained within. It is very similar to how a mitochondria deciphers the light waves that collide with it.

CITES:

1. Mathematical Models in Biology  By Leah Edelstein-Keshet 1988.

IS SLEEP APNEA PROTECTIVE AND NOT PATHOLOGIC?

I believe the answer is yes.  Why do I say this?

It appears sleep apnea protects defective mitochondria from high oxygen tensions. Want to know more watch my May 2018 webinar to understand the mitochondriac perspective.  I go further into detail in my August 2018 webinar that is being released today to my full members.

This means it is really not a disease……it is a reaction to low NAD+ and high ROS made in mitochondria to control the flow of oxygen just as a carburetor does in a car.

Over-eating is going to stress the ECT………that is ruined by the increased space between the cytochromes…….this implies oxygen levels have to be decreased when autophagy is ruined.  The biggest offender is blue light exposure via the eye and skin to cause this change in the melanopsin and Vitamin A couple.  Blue light exposure decouples Vitamin A and Vitamin D from one another as the slide below shows.

 

 

When ECT flow remains brisk and OSA is present, oncogenic risks is higher. Very few people with apnea seem to know that sleep apnea carries a pre-malignant risk with it because of this mitochondrial connection to defective apoptosis.

 

PREDICTION: This is what I think is going to happen to Jimmy Moore and his fat laden diet married to his high technology appetite. This lifestyle subtracts the sun because of his blue light addiction. This ruins the melanopsin/retinol mechanism that is protective to the metabolic rate variations in uncoupling proteins. These factors then lead to the key change in Kreb’s bicycle slowing both cycles kinetics causing methionine to rise.

 

 

 

When that happens autophagy and apoptosis are broken and these things all follow:

Cancer Changes:

1. that affect signaling of damage (the DNA damage response or DDR),
2. the extremely important WNT pathway that controls aspects of making RNA from DNA and especially proteins for apoptosis versus cell division. (The name WNT comes from pathways wingless in flies and int in mice)
3. enzymes called kinases that are part of vital pathways similar to WNT (MAPK, AKT),
4. the cytokine interferon that signals to the nucleus regulating and responding to making blood vessels, cell movement, defense against invasion, and cell death, and
5. inflammatory cytokines that stop apoptosis (nuclear factor-κB (NF-κB),

 

 

Cancer must inhibit apoptosis to occur. May 2018 webinar has it all laid out. Melanopsin optical signaling interruption in the eye and skin is capable of doing it ALL. The key feature in ALL CANCERS: ECT must be maintained while apoptosis inhibited.

OSA is a phenomenon that protects apoptosis from inactivation by keeping oxygen levels low. This protection scheme goes on until the deuterium entombment in the cell membranes of the PUFA’s of the mitochondria overwhelm Kreb’s bicycle and leads to COX-2 amplification.  This detail was covered in the May 2018 webinar and I went even deeper in the August 2018 webinar.  All cancers need high levels of oxygen to maintain high ECT speeds. The rising levels of methionine cause this.  This occurs because of the chronic slowing kinetics of the TCA and urea cycle eventually ruins the recycling of methionine from homocysteine.  This leads to blood vessels releasing angiogenesis factor to sprout new blood vessele to raise delievery of blood and oxygen.  Once this happens cancer is much more likely in a patient with OSA because it inhibits apoptosis.

This happens because oxygen is highly electronegative on the periodic table and it draws electrons from NADH in cytochrome fast. Another way the electromagnetic force is utilized in cristae.

Moreover,  sleep apnea sufferers struggle with spatial recognition and quantifying correct time.  This links it to a dopamine defect somewhere in the system.  This is also associated with a melanopsin defect in the eye or skin, as a result.  This implies sleep apnea is a protective effect cells use to buffer mitochondrial damage from melanopsin/retinol disruption.

 

 

You pay the price of both, with your choices made daily around the electromagnetic spectrum. You pay a biologic toll because of your choice, or you gain a biologic benefit because of your choice. At the outset, the dopamine levels are the same. After you decide and the results are in, you will see that the environment will add or subtract from your dopamine bank account. The presence of time alteration and sleep apnea are two signs you better change what your are doing. The result determines the reality you get. In this way, it should be clear how the environment dictates results we get.

 

 

AM sunlight balances the blue light of the morning. Blue light power increases during the day while red light remains constant in sunlight.

This implies blue light alerts us and is somehow related to time creation in the human brain.

Blue light opens the optical windows of the skin and eyes via the melanopsin effect in arterioles to other portions of light in our environment. When that is sunlight all is well. When that light is all blue light and no UV or IR light sleep apnea is coming faster to your life.

 

 

What does this all imply? Red light slows time. Cold slows time. Blue light creates and speds time perception up in the brain by altering the circadian clock functions. UV light rebuilds and replenishes time in a complex dance that begins in your eyes and skin because of melanopsins effect on Vitamin A = retinol. Therefore, excessive chronic blue light hazard speeds up time, the time mechanism in clock genes, and can give you mitochondrial diseases like cancer. When you are blue light toxic your life is lived like a blue straggler star. Quick and bright. Youre alert, and get sick quicker, and die sooner.

 

 

AM sunlight is the light switch that creates the ideal level of dopamine and melatonin assuming your melanopsin system is working properly and OPERATIONAL.  The switch is tripped by what happens first in the eye and then the skin by the balance between dopamine and melatonin created by the days first light.  Many papers attribute bipolar dysfunction to the lateral habenula in the brain.  People with bi-polar disorder often have sleep apnea too and have altered time sensation.  This area has a lot of melanin in it and has melanopsin inputs to it as well.  This region of the human receives input from the stria medullaris thalami.  This region links the central retinal pathways and the SKIN to the hypothalamus. The stria medullaris is a part of the epi-thalamus. It is a fiber bundle containing afferent fibers from the septal nuclei, lateral pre-optic-hypothalamic region, and anterior thalamic nuclei that connect to the habenula.  The habenula sends many outputs to several midbrain regions involved in releasing neurotransmitters, such as dopamine, norepinephrine, and serotonin.

 

 

All of these chemicals are made from aromatic amino acids that need to be programmed by AM sunlight.  All three of these neurotransmitters are made initially in the eye and then the skin from sunlight and aromatic amino acids.

Obstructive sleep apnea (OSA) patients show multiple neurobehavioral difficulties, including deficits in attention, psychomotor and executive functioning, memory, and affect (Beebe et al., 2003). However, the principal deficits in the syndrome involve the loss of motor control over the upper airway muscles, which fail to discharge in a timely fashion, when they should dilate the airway during diaphragmatic descent, and a loss of control over autonomic motor activity, with chronic, exaggerated sympathetic discharge from the PVN (Somers et al., 1995), and inappropriate lagged or muted dynamic sympathetic responses to blood pressure or ventilatory challenges.  This puts the problem in the brainstem in the dopamine tracts.

The habenula regulates the activity of midbrain centers, including the dopaminergic ventral tegmental area (VTA).  This area is huge in Parkinson’s disease development from melanopsin uncoupling on the surfaces of the eye and skin.  Phenylalanine and tyrosine can both be converted into these three neurotransmitters easily because both can make dopa. L-DOPA, is synthesized in the brain and kidneys of humans.  Look at the picture above where DOPA is made = tyrosine.

Dopamine is also synthesized in plants and most animals. The brain includes several distinct dopamine pathways, one of which plays a major role in reward-motivated behavior. Most people do not even realize with a dopamine defect, control of blood glucose is lost too. This is why people with apnea often have diabetes.

This becomes a key feature in the melanopsin damage seen in all forms of diabetes.  This one is best understood but researchers have done a poor job realizing how these pathways all begin with light signals in the retina/skin/blood vessels for proper physiologic functioning.  This is why bi-polar disease, diabetes, and parkinson’s disease and SLEEP APNEA stump modern medicine.

 

 

AM sunlight also stimulates POMC which makes beta-endorphin to make us seek AM light.  A lack of beta-endorphin sets up humans for an opiate crisis.  Modern humans have lost this GUIDING solar signal because of how they live their life and what light they live under most commonly.  Most types of rewards increase the level of dopamine in the brain.  In some diseases, like schizophrenia, too much dopamine at the wrong time ruins your ability to maintain cognition in time and space and leads to a crazy life.  This occurs in schizophrenia.  Without solar guidance, many modern humans seek opiate rewards in chemical ways to offset the deficit.  This leads many nights living humans to use many addictive drugs to increase dopamine neuronal activity to offset the loss of sunlight. Other brain dopamine pathways are involved in motor control and in controlling the release of various hormones.  These get damaged by chronic sunlight loss or too much blue light at the wrong time post sunset. It can even be a problem during the day if done chronically by a job and a computer screen or a phone to a teenager.  These pathways and cell groups form a dopamine system which is neuromodulatory and controlled by the circadian system by the melanopsin/retinol interactive controller.  This is the topic of my August 2018 webinar and it is why my Patrons are getting this blog right now at the same time.  Patrons it is time you sign up for your real Black Swan training here:  HYPERLINK

Outside the central nervous system, dopamine functions primarily as a local chemical messenger and it needs the direction of sunlight.  How do we get light signals deep into the body this way?  Hemoglobin in the eye and skin is how it begins.  It is loaded with iron porphyrins which absorb 250 – 600 nm.  The water that surrounds it can absorb all the way to 3100nm.

 

 

This light is ferried to cells to program the dopamine and melatonin release locally ot change the metabolic rate of mtDNA by altering the UCP2 permeability of deuterium. The same is true of melatonin. In blood vessels, dopamine inhibits norepinephrine release and acts as a vasodilator with Nitric oxide (UV-A); melanopsin does the same thing using other frequencies of sunlight. In the kidneys, dopamine increases sodium excretion and urine output; This changes the EZ size in blood plasma. In the pancreas, it reduces insulin production (diabetes link); in the digestive system, it reduces gastrointestinal motility and protects the intestinal mucosa gut barrier (leaky gut link); and in the immune system, it reduces the activity of lymphocytes which is important in autoimmunity.

I said June 2, 2018 in Vermont that is not one human chronic disease not LINKED to the melanopsin/retinol mechanism. Here is the slide I used.

 

 

This post is a dip of the toe in why I might be correct in my reasoning. With the exception of the blood vessels, dopamine in each of these peripheral systems is synthesized locally and exerts its effects near the cells that release it. All of these link back to sunlight to the most fundamental level and no one but Black Swans realize it.

The mitochondriac perspective always explains things we observe over the things modern medicine espouses.

 

 

The more I study the history of intellectuals in medicine, the more they seem like a wrecking crew, dismantling civilization bit by bit — replacing what works in nature with what sounds good and repackaging it as evidence-based therapies.

Nothing replaces this……….and it is free.

 

 

Start using it if you have sleep apnea because that symptom is your colony of mitochondria telling you there is a problem.

CITES:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3930100/

 

Few of us realize the liquid crystals in us create time using light.  Time and space are modes by which we think and experience reality but neither are conditions in which we live on Earth.  We live by swinging on the spiral arm of a galaxy constantly moving through space .

Ever since the derivation of the theory of relativity, physicists have thought they have done reasonably well in understanding time; however, biologists are still waiting for a comprehensive theory of timing in living systems:  they remain waiting for a corpus of “laws” describing how cells and organisms can precisely initiate and terminate processes at specified times because they are focused on the nuclear genome and not the mitochondrial genome.  Time perception is critical to mitochondrial biology but it is not especially important to the nuclear genome because the energy provided by the mitochondrial genome allows time to manifest in living things. This deficiency in biology is particularly acute in developmental biology, where complex mechanisms of various paces and durations must be orchestrated to solve huge developmental problems such as the one faced by the fertilized egg: how to become an organism using energy/information and electric and magnetic field to direct and power morphogenesis.

Animal development is, in fact, nothing but time. From the cell cycle to the beating of the heart, our own lives are composed of a multitude of microscopic and molecular oscillations. For developmental biology, the study of causal relationships implies the examination of two time points: inducing the cause and looking at the effect. In QED, cause and effect is blurred because of the uncertainity principle.  In biology, cause and effect “appear” to be linked because of how time is perceived.  We are generally ignorant of the temporal rules governing this transformation. Light and aromatic amino acids form those foundations.  Whereas the rules of a Swiss watch involve a timing mechanism that uses a uniform unvarying tempo to translate rapid oscillations (seconds) into longer periods of time (hours), the rules of the developmental clock of animals are much more complex because they use light and light is non linear in the violet part of the visible spectrum. The animal clock is made up of a variety of counting mechanisms that follow varied temporal rules at different frequencies and often run in parallel without any “apparent” interaction with each other, because their linkages are via molecular resonance. The goal of the developmental clock is not simply to mark off time, but to integrate and unify the myriad temporal signals received from throughout the organism.

However, the frequencies and amplitudes of most, if not all, developmental clocks are robustly determined within the same species, suggesting that these are genetically encoded devices being linked and controlled by resonance instead of being produced by the system itself, as an “emergent feature.”  I have always believed time is an emergent property of life because time emerges from the complexity of light the force carrier of the electromagnetic force. Today, the “old paradigm of biologic time (Franz Hallberg) is dominant in the published literature, but as Max Panck said, things change in science one funeral at a time.  Morever, if the “belief” is true that time is encoded in our DNA (it’s not), what are the genetic balance wheel, hairspring, and gears, and what are the specialized tools that are needed to elucidate the intricate workings of the genome? There are many possibilities to consider here, but I will discuss two.

 

 

The first one concerns clocks generated by mechanisms in “trans”, generally based on the postulate made in the early 1950s by Alan Turing (mathematician, philosopher, and inventor of the concept of the computer), who argued that the diffusion of substances with activation, repression, and retroactive potentials can generate intrinsic oscillations in space and time. The literature has begun to decipher some of these ideas and more are likely to come in the near future.

The second possibility involves mechanisms in “cis”, using the linearity of our genetic material itself, our chromosomes, to measure time; an admittedly elegant solution, but one that severely lacks both experimental and theoretical support. Chronobiology is stuck on these two options because they do not realize that time is a superposition of many possibilities. This is why they ignore the mitochondrial genome. This genome creates many possibilities when small changes occur to it. When we disrupt it with anesthesia timing is completely altered, we observe it and we measure it, but we do not understand the implications for man or his diseases. This will change with the coming age of quantum biology. The old guard expected some help from the human genome sequencing and related high-throughput technologies, but they got stonewalled because it destroyed constructs that we inherited from Watson and Crick. I reject most of their thesis about biology.

 

 

Modern epigenetics data has blown up much of Darwin’s thesis from 1859 but few biologist will admit it today because it is akin to removing the binding from a book.  All organizing principle are afloat now in molecular genetics because of the new insights of epigenetics.  So is science and life.  Every end is a new beginning in understanding and learning with respect to nature’s wisdom.  Darwin’s supporters today are still trying to hold the theory together but the concept of timing and energy is 100% tied to energy collection and generation and distribution in the cell. They have no Earthly idea that energy and information processing are coupled by light.

There is little doubt that the mechanisms in ‘trans’ will be advantageously studied in this context. Time-lapse proteomics and metabolomics, adapted to minute amounts of material such as cohorts of less than a thousand cells, would certainly show significant recurrences and, perhaps, allow the temporal algorithms underlying developmental clocks to be uncovered.  But trends in protein or metabolic movement can just describe the cellular processes and it won’t explain it.  The explaination will take a mind filled with innovated thoughts to link them back to the fabric of nature’s laws.  Today’s developmental chronobiologists believe that a reductionist approach to clock mechanisms will give us the key answers.  In fact, we just gave three people a Nobel Prize recently for discovering the biologic molecules involved in the circadian mechanism.  For example, the tightly regulated timing in the successive appearance of whiskers on the nose of a rodent would likely be deciphered if we could microdissect the whisker field like a chess grid, and submit each square to full protein content analysis.

On the other hand, the challenge in identifying ‘cis’ processes would be to use several species showing graded variations in clock parameters, such as in the frequencies of the same clock, and to find correlations with the “cis organization” of DNA segments through a large-scale approach. Such correlations, if any, could reflect the existence of linear time-measuring devices, from the mere duration of transcription to more complex processes such as progressive transitions in the chromatin state. However, that is easier to say than to do; a genuine “casse-tête” for bioinformaticians.

 

 

I believe based upon what we know today about epigenetics, it is seriously pretentious to try to extract the fourth dimension, time, out of our DNA/RNA structure when we still lack complete understanding of the other three dimensions of our world. But the human quantum computer does not need complete data sets to jump to conclusions. That is what innovation and imagination are useful for in complex problem solving. This science is the privilege and the difficulty of working with embryos: The spatial construction can be understood only in the light of time. The irony of this idea, is that light is what creates time.

 

 

This concept continues to trip up biologists because they are ignorant of the queerness of the physics of light. Similarly, evolutionary genomics will likely teach us what happened during phylogenetic times, and our ontogenic clocks may be revealed by global comparative analyses.

 

 

Chronomics is on the way, but only the unripe mind will think it is the be all end all in the discussion of what time really is.

Time allows us to see what we have become, even when we miss it. The liquid crystals inside of us build this library. Time is erased for some of us, because we cannot use sunlight and water to build the crystals properly to manage our time. Sometimes we erase people and family, and walk away from the shadows creeping up on us. Sometimes you must give up on people, not because you do not care, because they do not care enough for themself by making the correct choices.

Spiral out…………..

 

So do we glow when sunlight hits the skin? Yes, we do. What kind of light do we glow with? ELF-UV light manifests in our blood plasma = do we gain or lose electrons when this occurs? We lose electrons and give them to other anions in our blood. Sulfated amino acids are added to many proteins and lipids in our blood under the power of sunlight. Sunlight increases sulfhydryl groups which are the major anions in our blood plasma.

 

 

Our tissues glow because when an electron is delocalized light is given off and that light produces change without atomic change because it changes the topologic charge of atoms in our skin proteins because UV light causes a loss of electrons in out tissues.  This mechanism is what causes release nitric oxide from the arterioles in our skin to change the topology or femtochemistry of the surface of the skin when it is irradiated by IRA and UVA light.  The change in femtochemistry results in attochemical changes in deuterium in our blood plasma.

Full spectrum sun contain UVA/B light which is capable of altering the topologic charge of nitric oxide (NO).

UVA light releases the most NO into our blood.  IRA light is how the exclusion zone is built initially in sunlight in our plasma.  Both of these frequencies penetrate below the epidermis to where the arterioles of our skin.

The key thing to comprehend is there is no way to satisfy the octet rule for both atoms in NO because of their valence shell electron state. The structure is a double bond with three non-bonding electrons on N and four on O but really one of the electrons on N is not localized to N but the entire molecule.

Formal charge = electrons in ground state – (non-bonding electrons + 0.5 * bonding electrons)

Formal charge for N = 5 – ( 3 + 4 * 0.5) = 0 (there are two bonds so four electrons are involved in bonding)

Formal charge for O = 6 – (4+ 4 * 0.5) = 0

Sunlight alters that balance of charge on our surfaces.  What does it do?

It creates the triplet state of free radicals because of the effect on electron quantum spin.  Every surface in our body has this mechanism available if the sun is allowed to interact with it.  If the sun is absent this will not occur and it will lead to collateral damages.   Where else is electron spin altered?   MITOCHONDRIA.  This is where the critical ROS and RNS signals are made.  These signals affect a third cycle in us that controls the sulfated amino acids.  This is the sulfated amino acid cycle contains cysteine methionine and homocysteine.  This cycle is altered because of the changes in free radical creation by the light our skin does or does not experience.

Full spectrum solar exposure of the skin increases sulfhydryl groups in the blood plasma.  This increases the anions present in our blood.  Sulfur amino acids are a kind of amino acids which contain sulfhydryl groups, and they play a crucial role in protein structure, metabolism, immunity, and oxidation. Recent studies have demonstrated the oxidation resistance effect of methionine and cysteine, two of the most representative sulfur amino acids, and their metabolites.

Methionine and cysteine are extremely sensitive to almost all forms of reactive oxygen species MADE IN THE MATRIX, which makes them antioxidative and critical redox switchs that tell us about TCA and urea cycle kinetics.  They can both be used to tell us when the matrix is slowed by deuteration.

Moreover, methionine and cysteine are precursors of S-adenosylmethionine, hydrogen sulfide, taurine, homocysteine,  and glutathione. These products are reported to alleviate oxidant stress induced by various oxidants and protect the tissue from the damage. However, the deficiency and excess of methionine and cysteine in our diet can affect the normal growth and longevity of animals.

 

 

It is well known that oxidation from free radicals of the cysteine thiol groups occurs in all stressors in humans.  This results in defective glucocorticoid receptor (GR in pic above)  function and it affect ligand and DNA binding in cells.  This implies that in a 5G world where the toplogy of the skin will be affected, cysteine and methionine hacks will be quite important to understand and use when one gets afflicted with electropollution type diseases like autoimmunity, sepsis, and cancer.   To understand how these sulfated amino acids operate you must understand how the sun works in your skin.  I discussed this in the Vermont 2018 talk now available for purchase to Patrons and the public.

Vitamin D3 supplement pills don’t and can never give you triplet state being described here because you need the light stimulus on the skin to get it.  You also need skin in the game to generate the light pressure on your skin and arterioles to enable the chiral effect.   Sunlight exposure of the skin, works in unison to sulfates the cholesterol anion substrates in the skin and blood from the sulfated amino acids so that cholesterol can become Vitamin D3 via various steps between the skin, kidney and liver under the power of UVA and UVB light.

 

 

KEY POINT ALERT:

UVA light helps in performing this task because of the action of activated NO in the skin.  NO inactivates cytochrome 4 while IRA light allows the ATPase to spin freely with fewer electrons needed from foods/fat due to the action of NO on ECT.  UVA light inhibits or slows electron chain transport flow on the inner mitochondrial membrane.   With this knowledge are you still afraid of black lights indoors?  You should not be.  It might be a great hack for you depending upon your N = 1 context.

 

 

Nitric oxide (nitrogen oxide, nitrogen monoxide) is a molecular, chemical compound with a chemical formula of NO that is a colorless gas under standard conditions. Nitric oxide is a free radical—i.e., its bonding structure includes an unpaired electron with a specific electronic spin state. It is a practically important intermediate in the chemical industry. In addition, some are unavoidably produced during combustion of fossil fuels in power plants and automobile engines, with an excess of NO being created when there is present more air or higher temperatures than needed for efficient and complete combustion of the fuel. More oxygen and higher temperatures in a cell favor H+ bonding.

 

 

They lower deuterium bonding in the skin. This is very important because it is the key mechanism of how and why deuterium is found in the high concentration of the blood plasma in the irradiated skin by the sun. In skin thermal temps are raised most by UV/IR light and oxygen is increased by UV light striking the skin alone.

 

 

Not only does skin make it with the powerful sun but so does the atmosphere in a quantum fashion. It is also produced naturally by the extremely high air temperatures produced along the path of lightning in thunderstorms.

Now stop here.  Did I not tell you in the past on previous blogs that the inner mitochondrial membrane can generate the same voltage as a bolt of lightning in the “What powers life and death blog years ago?”  Yep.

30 million volts is possible.

It was found that NO acts through the stimulation of the soluble guanylate cyclase, which is a heterodimeric enzyme with subsequent formation of cyclic-GMP. Cyclic-GMP activates protein kinase G, which causes reuptake of Ca2+ and lowers the intracellular calcium by the opening of calcium-activated potassium channels. This is highly protective in high nnEMF environments. 5G will block this effect in the skin. 5G will also destroy the covalent bond between melanopsin and Vitamin A.  This will demolish the entire circadian mechanism in all the peripheral clock genes.

This is why 5G risks will ramp up mitochondrial diseases linked to the melanopsin/retinol system.  Melanopsin also uses calcium as a secondary messenger.  This creates another risk of nnEMF in destruction of the melanopsin mechanism.  nnEMF alters calcium flows and this is the signal that makes free radicals in the matrix for proper quantized signaling.

 

 

The fall in concentration of Ca2+ ensures that the myosin light-chain kinase (MLCK) can no longer phosphorylate the myosin molecule, thereby stopping the cross-bridge cycle and leading to relaxation of the smooth muscle cell in our skin arterioles allowing more RBC’s loaded with porphyrins to continue absorbing UVA light to keep this action of NO working properly to keep deuterium inside of our arteries where it belongs.  Deuterium concentration in the blood plasma is how the body normally creates ELF-UV within our blood plasma to activate cell water to become an exclusion zone.  This is a topologic charge effect that must occur endogenously.

Nitric oxide, is also known as the ‘endothelium-derived relaxing factor’, or ‘EDRF’, is biosynthesized endogenously from L-arginine (urea cycle), oxygen, and NADPH by various nitric oxide synthase (NOS) enzymes. Reduction of inorganic nitrate may also serve to make nitric oxide in our gut. The endothelium of blood vessels uses nitric oxide to signal the surrounding smooth muscle to relax, thus resulting in vasodilation and increasing blood flow so we can collect a lot of UV light from skin irradiation. How does hemoglobin fit into this blood UV story?

Two important biological reaction mechanisms of nitric oxide are S-nitrosation of thiols and nitrosylation of transition metal ions. S-nitrosation involves the (reversible) conversion of thiol groups, including cysteine residues in proteins, to form S-nitrosothiols (RSNOs). S-Nitrosation is a mechanism for dynamic, post-translational regulation of most or all major classes of protein in all living things. This is why the ubiquitination series has so many blogs and threw so many new details at you.  They were foundational for this blog.

Sunlight controls protein turnover via ubiquitin marking and nitrogen flow in cells using this mechanism.  Methionine is needed for this step no matter the protein being made by a cell.  The second mechanism, nitrosylation, involves the binding of NO to a transition metal ion like iron or copper in your cells.  In CPC #25 you saw how this process is broken in sepsis.

Methylene blue, IV Vitamin C, and glucocorticoids in low doses can improve the function of sulfated amino acids in people with mitochondrial damage who do not get enough sunlight.  The sulfated amino acids are the critical optical switches in many neurotransmitter and glucocorticoid receptors.  They are also critical in DNA function.  This has been shown in many diseases like sepsis in many papers on PubMed.  Sulfur and iron clusters are found in the mouth of every cytochrome protein. Did you also know that cytochrome C oxidase is also a heme protein that has 4 red light chromophores embedded in its atomic structure?   This is why IRA light exposure is a critical part of solar exposure and control of apoptosis.

In this nitrosylation function, NO is referred to as a nitrosyl ligand. Typical cases involve the nitrosylation of heme proteins like cytochromes, thereby disabling the normal enzymatic activity of the enzyme when it becomes necessary in cold environments where sunlight is poor.  This was the key point I made in the Cold Thermogenesis 6 blog years ago.

Nitrosylated ferrous iron is particularly stable, as the binding of the nitrosyl ligand to ferrous iron (Fe(II)) is very strong. Hemoglobin is a prominent example of a heme protein that may be modified by the NO free radical by both pathways: NO may attach directly to the heme in the nitrosylation reaction, and independently form S-nitrosothiols by S-nitrosation of the thiol moieties. There are other heme proteins you will soon learn about that refine this process even further.

Hemoglobin absorbs light at 280 nm (UVC) and 3 other IR frequencies. Maybe now you can see why they link to NO function via the 42% red light in our sun.  The addition of sunlight and seasonal ketosis keeps the TCA and urea cycle clear of a lot of RNS and ROS from broken kinetics in both cycles due to a loss of control of hydrogen isoforms from cell membrane damage.

The sun is designed to increase the mammalian battery using UV/IR light and cold for humans with bad environments.  The Quantlet was designed with this idea in mind.  The best idea is not to rely on any man made device, but to rely on sunlight with a seasonal ketosis regimen in humans to make metabolic cell water devoid of deuterium in the matrix.  Deuterium needs to be kept in the blood to make UV light in us endogenously that programs the proteins in us because it passes its information and energy to water that surrounds every protein in us.

 

 

 

 CITES:

Dr. Kruse talk June 2, 2018 Shelburne Farms.  

WHAT IS THE QUANTUM MECHANISM FOR EVERY AUTOIMMUNE CONDITION ON EARTH?  

Why did this blog start with a Tryptophan operon lecture?  Tryptophan and Methionine are the only two amino acids that have SINGLE codons in the human DNA code.  This makes both of them unique compared to the other known 500 amino acids.  Our DNA only codes from 20 amino acids.  Methionine has to be obtained from the environment.  This makes it more unique.  What does methionine do  in humans that make it UNIQUE?  Our start codon to begin all protein synthesis always codes for methionine in HUMANS.  This means the methionine cycles are a pretty big controller of something important.    Have you ever wonder what they control and why they exist?  What if I told you methionine and tryptophan were two-time crystals that tick everytime sunlight in a season interacts with us.  One is essential and made only in the environment and the other is made by us and it forms one key chemical that controls our mitochondrial DNA.

You think that might be important?  

What do codons and ubiquitin marking  have in common?  What is a codon?  It is the code for an amino acid to be translated when it is low or needed.  After it is made it can be activated by light, electrons, or by protons.  

Are all amino acid codons the same?   No, they are not.  

In fact, not all 64 codons of the genetic code specify a single amino acid during translation. In fact, one amino acid can be encoded by more than one mRNA codon-triplet. Arginine and leucine are encoded by 6 triplets, isoleucine by 3, methionine and tryptophan by ONLY 1, and all other amino acids by 4 or 2 codons.  So is this why methionine and tryptophan are special?  Why is tryptophan coded only by the bases UGG in DNA?

 Science knows that the common 20 amino acids are coded by several kinds of codons,  except for methionine.  It is coded by AUG.  You know there is something else interesting about methionine, don’t you?    

Methionine is the amino acid a cell sends to the ribosome to begin to signal to start a protein translation process. This means methionine is a gatekeeper for protein translation.  Might it be because every peptide bond in proteins costs us 5 ATP?  I think so.  Methionine action on protein translation appears to be quantized to energy flux in the cell.  This means the light signals from antenna arrays in our cells are what link to the timing of how the methionine codon operates.    

This means the more ELF-UV light released in a cell means more protein translation.  

Since methionine is a sulfated amino acid that is essential it cannot be made by our cells.  It has to come from the environment, so that means methionine is capable of telling the cell something about the environment.  

What is this information that it seeks to “know”?   

If more than one amino acid can decide a start point for proteins, the coding sequence would be interrupted in unwanted locations and this would affect its semiconductive diode abilities.  The exclusivity of methionine tells us that nature wants no electromagnetic waves interrupting this wireless communication process.  The fidelity of this signal must be important for some reason.

We know that there are several exceptions in the standard genetic code for codons but over the 3.8 billion of years of evolution there is only one codon as methionine code for tryptophan.  That is extraordinary fact.  It is as extraordinary as DHA never being replaced in 600 million years of eukaryotic evolution in the nervous system and in cell membranes.  It is so extraordinary that there must be a missing role in biology that has yet to be discovered about why this is true.  This blog is about that reason.

DHA, methionine and trptophan are the key gears that make up the circadian mechanism in every eukaryote on Earth.    It forms the basis of the circadian mechanism in complex life in eukaryotes and it links to deuterium biology in our circulatory system.  The topology of our skin sits between these two areas.  The skin is loaded with electromagnetic antenna arrays.  Our apocrine glands and the melanopsin/retinol arrays are the most important antenna’s man has.   

I posted this very question on my FB page ten days before I spoke in Vermont in June 2018 and was quite surprised one of my misfits got the answer to the question fairly close, and it was clear he was making the connections a black swan mitochondriac needs to optimize health and reverse disease using quantum biologic principles.  I said in Vermont in 2018 all diseases were linked to blue light toxicity right at the beginning of the talk.  That is a big statement.  Here is another big piece of data to back up my current beliefs.  

Luke Marshall remarked that ubiquitin marking increases in a 5G blue-lit,  nnEMF world due to mitochondrial matrix deuterium accumulation.  This subsequently leads to dehydration in Krebs bicycle and no DDW is made.  As a result, the respiratory proteins in the cytochromes spread out causing electron transport to become inefficient, and this cause a loss of effectiveness of autophagy.  If it persists ECT can collapse and fail, and sleep is destroyed and melatonin drops.  If apoptosis is intact it can take out the bad engine if it too is not an efficient process because of the circadian mismatch, then our tissues become overloaded with bad engines.  As a result,  we no longer make DDW in the mitochondrial matrix and we cannot fat burn or use amino acids properly.  The kinetics are what is off and this affects how a time crystal operates with light pulses.   

Tryptophan is one of those amino acids that makes melatonin.  Melatonin is the neurohormone that repairs or replaces mitchondrial DNA.  If tryptophan is not properly activated and programmed by the environment, melatonin will not be created.  If melatonin is not created properly we lose control of the only two self regulatory programs in mitochondria.  Those two programs are apoptosis and autophagy.  This blog is about that amino acid and how it operates in a quantized cell.  You already learned about methionine in this series.  When we can’t make water in the matrix our metabolic pathways are not surrounded by the water they need to operate like a semiconductive diode. Blue light antenna’s in our skin control melatonin levels.  The light antenna in us is blue and it is linked to Vitamin A.  Vitamin A is linked to Vitamin D and both are linked to the DHA receptor.  This is how the peripheral clock genes are linked to melatonin.  As Vitamin A drops in the plasma so does melatonin levels.  Melatonin repairs the peripheral clock gene mechanism in humans.

 

 

As a result,  our proteins become dehydrated and lose their topologic charge, this causes a size and shape changes in many small signaling proteins in mitochondria that control apoptosis and autophagy.  The defective protein in these self regulatory programs  then gets marked for a turnover by ubiquitin. The protein that does this is ubiquitin.  I have written an entire series on this protein.  This protein is also controlled by the peripheral circadian mechanism.

 

 

Now you can see how it fits your surfaces in your eye, skin, lung, and gut to operate.  Typtophan makes melatonin.  With respect to tryptophan or any amino acid, a specific gene will need to be expressed in order to replace the protein, the start point is always methionine. As ubiquitin marking increases so does the costly process of protein synthesis, all protein synthesis begins with start codon methionine. 

 

 

This is a big deal in the gut.  Enterocytes use the peripheral circadian clock genes to get rid of their deuterium laden cells every 24-48 hours to keep functioning normally.  If this process is slowed or absent because circadian timing is off, it leads to the accumulation of lipids in the substance of the liver.  Fatty liver is a circadian disease linked to the inappropriate collection of deuterium in the liver from a malfunctioning portal circulation due to the defects in the sloughing of enterocytes.

 

 

Leaking gut is not what most think it is.   Methionine is a hydrophobic sulfur-based amino acid, cysteine is synthesized from methionine and methionine is recycled from homocysteine.  So if homocysteine is raised you know you have a circadian mismatch present.  It likely goes back to blue light toxicity at a surface.  

 

 

Luke went on to speculate that  “Methionine is hydrophobic but it has the potential to act as a hydrogen bond acceptor. It seems like there is going to be a difference in disulfide and hydrogen bonding with methionine compared to cysteine, which then affects deuterium concentration.”   Luke is a mitochondriac paying attention to my work.  Now that you know methionine is the start codon in the tryptophan operon in the video above let me show you more connections you might not have made yet.  The tryptophan operon links all the way to cytochrome 1 (NAD+/NADH) of mitochondria with damaged autophagy and apoptosis.  Cytochrome 1 efficiency SETS one boundary of the redox potential in the mitochondrion as the picture below shows.  This means it is critical to understand WELL if you are going to get better.

 

 

In mammals, NAD+ is synthesized from four different biosynthetic precursors in two distinct pathways. De novo synthesis (the deamidated pathway) uses the amino acid tryptophan, which is metabolized to form biosynthetic intermediates. These intermediates ultimately generate nicotinamide (the pyridine moiety of NAD+) and then form NAD+. Dietary vitamin B3 compounds, including nicotinic acid, nicotinamide and nicotinamide riboside, serve as NAD+ biosynthetic precursors and are salvaged from the diet (the amidated pathway) to generate cellular NAD+.

NAD+ is where “carbohydrates electrons” usually enter mitochondria at cytochrome number 1.  How many people do you think know that Typtophan can be a “bisexual amino acid”?  Tryptophan is a deep UV fluorophore amino acid (240-300 nm) is the basis for construction of NAD+ which is a 340 nm fluorophore protein?  

240 nm light is DEEP SHORT WAVE UV Light.  Where does that light come from folks?  Look at the picture below?  Can it come from the sun?  

 

 

It cannot.  This means that there is a pathway in the body that MAKES UVC light to program typtophan to make melatonin and NAD+ in some way.

That was the topic of my talk in Vermont.

Tryptophan catabolism in humans it is very difficult to comprehend for most but I will give it a try. 

It turns out, one major pathway results in the formation of acetoacetyl CoA, the precursor of ketone bodies – acetoacetate, 3-hydroxybutyrate, and acetone.  Tryptophan is activated and excited by light from 240-300nm.  This means it is an aromatic amino acid that has a phenol ring.  This is UVC light.   UVC light is noted to be TOXIC to all life by NASA.  This should confuse you.

 

 

People believe UVC sterilizes objects and planets.

Why would a living quantum system use UVC light to do something in their blood?

Did you know humans have 150 ppm of deuterium in their blood?

Did you know ketone bodies are deuterium depleted?

Did you know that they also block the NLP3 inflammasomes?

How is this process related to disease reversals and sunlight?  

Might tryptophan be an optical switch or time crystal that allows the body a way to “know” what season it really is because each season has its own electromagnetic fingerprint?  Yep.

 

 

This raises the question what is an inflammasome?    

Inflammasomes are multi-protein signaling complexes that trigger the activation of inflammatory caspases and the maturation of interleukin-1β. Among various inflammasome complexes, the NLRP3 inflammasome is best characterized and has been linked with various human autoinflammatory and autoimmune diseases.  

If you were fortunate to see my Vermont 2018 talk on how the skin, you’d begin to see how big a deal this really is.  

The NLRP3 inflammasome is activated in the cytosol where the matrix of mitochondria makes deuterium depleted water at Krebs bicycle.  This is where the TCA and urea cycle meet because fumerase adds water to both cycles anionic substrates.  

This is how information from SUNLIGHT is transferred to metabolic pathways to know what season it is, and understand how light and oxygen tensions determine the choices that a cell must make.

 

 

Black Swan Mitochondriacs are people who are highly-driven have what’s called a high “need for achievement.” If these people value something, they go after it with relentlessness.  Nothing else matters but THEIR health.  The better a mitochondriac knows nature they better they can “know” themselves.  As a result, the better their relationship is with the rest of the world.  Any time there is mitochondrial damage oxygen becomes toxic and ROS rises and cells become UNABLE to use the TCA and urea cycle and have to default to the older evolutionary pathways present in all cells before the Cambrian explosion, namely glycolysis and the PPP.  Glucose/fructose in foods isn’t toxic to any human or animal.  It is the damage to their mitochondrial that gives silly talking confident monkeys that sugars might be.  It is a horrible narrative done by poor thinking because the silly talking monkeys got tired of thinking and got lazy with their logic. 

 

 

Knowing about biochemistry and not connecting it to anything else in nature, like the sun’s biophysical levers, in a cell, is akin to buying a guitar that Prince once owned and then expecting that you could play Purple Rain as he did in life.

 

 

The implications of all the biophysics in this blog should be obvious.  When deuterium leeches from cell membranes in us and becomes prominent in places it should not be (matrix),  biochemistry in the (TCA/urea cycle) is no longer cyclical and its KINETICS are ruined.  Kinetics ruins the peripheral clock timing mechanism.  These cycles begin to run like a linear chemical reaction driven by an equilibrium bias and it is slow. This lowers oxygen levels and the cell MUST use glycolysis and the PPP to avoid oxygen toxicity.  This pushes a cell closer toward an equilibrium bias, which is another way a mitochondriac use to describe that journey to illness and eventual death.

 

 

To a mitochondriac, death is the slowest form of death we create by controlling oxygen and light like a biophysical carburetor to keep a cell far from equilibrium.  Now think about what this means for deuterium in Kreb’s bicycle, and what Dr. Doug Wallace has found at the IMJ’s (above bottom right of pic).  IMJ’s do what they do for energy because there are built using the information quanta in sunlight.  It is about as counterintuitive as one can imagine.  But it is what life is built around.  These ideas are the results of deep thinking and what the Quantum Thermodynamics blog series is all about.  These blog ideas can do wonders for any silly talking primate when they pay attention to nature’s directions and not other silly talking primates.

Hopefully, with all these lessons in the QT series, you’re beginning to also understand how the connections of light and oxygen links to the physiologic function of melanopsin, blue light, and the Vitamin A cycle.

 

 

This allows you to see how the pieces fit and understand how they can fall apart because of the action at the NLP3 inflammasome.  When Vitamin A goes awry you lose the circadian mechanism and autoimmune conditions become more likely because you cannot create full spectrum shortwave UVC light using deuterium in your blood plasma.  

If you cannot do this, can you make a large EZ?  NOPE 

If UVC is absent can you make melatonin from tryptophan?  NOPE

You remember tryptophan use UVC to activated huh?  

It is kinda weird that the spectrum of activation of Tryptophan spans UVC and UVB light isn’t it?  

Could this be the way an amino acid that makes serotonin and melatonin to control mtDNA seasonally?  Yep.  

This is black swan mitochondriac stuff here.  This is how the pieces fit and I figured it out only after seeing them fall apart in thousands of my patients over the last 25 years.  

Tryptophan is difficult to make sense out of without understanding the biophysics of UVC and UVB light.  Deuterium makes them inside of you using the light that strikes your skin.  

Do I believe UVC is toxic to life in 2018?   

No, I no longer do and I think NASA and biology are dead wrong about UVC light.  I think some UVC falls to earth and the little that does is critical inside the tropics to work with Lo haplotype and black skin.  Why do I believe this.  Look at the picture of all the aromatic amino acids below……….they all use UVC light to operate. 

 

 

So you might be asking yourself, OK Jack,…….tryptophan making ketone bodies explains how it works in winter when no environmental UVB light is present.  Tryptophan is a seasonal EMF time crystal signaler. 

 

 

It means the phenol rings of this aromatic amino acids (ring above) never gets excited by sunlight, therefore, it forms a ketone body that is deuterium depleted for proper use of fat burning in the TCA cycle when it is cold.  

How does it operate in spring and summer?  

Is this the reason why serotonin is also so misunderstood by clinicians and researchers?  Yes, it is.  Serotonin is a quantized semiconductive protein that can act as a neurotransmitter, a protein, and an optical switch for metabolic pathways because it is only made and excited when a certain light frequency range in the solar spectrum that VARIES as the power density of seasons vary, therefore those frequencies must be present in the environment to program this amino acid.  

 

 

Tryptophan catabolism includes the generation of the glucogenic amino acid alanine, which allows tryptophan to be classified as a ‘glucogenic and ketogenic’ amino acid, hence why I called it bisexual above. It turns out serotonin is excited and made in summer because UVB returns to Earth to cause the presence or UVC light in the blood plasma from the pinch of deuterium in the skin I mentioned in the Vermont 2018 talk.

 

 

This version of serotonin has its version or tryptophan fully excited by short wave UVC light from 240-300nm.  This means the photonic programmed amino acid can act very differently in tissues because all aqueous proteins/lipids respond using a molecular resonance phenomenon.  What is the purpose of photosynthetic reactions in animals?

 

 

It turns water into the fuel that cells need and require to operate their proteins.  Water that is made by the mitochondria and surrounds proteins is a repository of electromagnetic waves.  Sunlight is the key driver of animal photosynthesis in humans.

This is why tryptophan and serotonin confuse the non-mitochondriac.   Now for some hardcore science to show you just how confusing tryptophan is to understand without knowing about the biophysical levers that control it.

TIME CRYSTAL WORK BY THE ELECTROMAGENTIC PULSES OF SUNLIGHT.

They are broken by the blue light and 5G pulses of man’s light and we do not realize it.

 

 

BIOLOGY GEEKS:

The tryptophan catabolic pathway starts with either tryptophan 2,3-dioxygenase or indoleamine 2,3-dioxygenase 1 which open the indole ring to form N-formyl-kynurenine. N -formyl-kynurenine is then converted to kynurenine by the enzyme, arylformamidase. It is also called kynurenine formamidase in the literature.

Kynurenine then undergoes a series of catabolic reactions resulting in complete oxidation to acetoacetyl-CoA. The first is catalyzed by kynurenine hydroxylase (also called kynurenine 3-monooxygenase) forming 3-hydroxykynurenine. The next step in the pathway is catalyzed by kynureninase and produces 3-hydroxyanthranilic acid and the amino acid alanine hence the classification of tryptophan as a ‘glucogenic and ketogenic’ amino acid. The next step in the pathway is the conversion of the 3-hydroxyanthranilic acid to 2-amino-3-carboxymuconate-6-semialdehyde (ACMS) and quinolinic acid by the enzyme 3-hydroxyanthranilate 3,4-dioxygenase. ACMS can be metabolized via the action of the enzyme ACMS decarboxylase followed by 10 other enzymes to acetoacetyl-CoA, the end product of complete tryptophan catabolism.  All the enzymes use proton tunneling to operate.  The light that controls this is beyond the normal red light that controls photosynthesis. 

 

 

 There is so much escitation that ocurs beyond the 250-780nm solar spectrum that life uses in mitochondria. The 1538.5-3100nn range deals with proton movements in enzymes and the matrix.

Through cytochrome c oxidase, IR-A light gives a ‘molecular kick’ to the mitochondria from the 42% of the red light in the sun’s rays that are present from sunrise to sunset; this informs or tells (information quanta) the cell to turn on a large number of antioxidant and energy-boosting genes only when ATP is being made by the combination of IRA and UV light we see in parts of a day.
Water viscosity changes with the red light in sunlight to facilitate the ATPase to work at 100% photothermal efficiency. That boost is greater when UVA and/or UVB light are present because of both of those UV frequencies BOTH slow ECT at different places on the inner mitochondrial membrane from cytochrome 1-4 by a quantum stimulus/design.

 

 

Cytochrome c oxidase has 4 red windows at 620, 680, 760, and 820nm of light. Those are some frequencies to consider. Hemoglobin has a sharp cut off at 600 nm. Both are heme proteins which act as circadian time crystals with radically different optical windows. Cytochrome c oxidase is present in mitochondria and RBC have a ton of hemoglobin in their cells which is HAVE NO mitochondria by design because of these light mechanisms. RBC’s are designed to ferry 280-600 nm (IRA and UVA light) 600nm-1000nm red light would cover the activation of fibroblasts to make collagen to stimulate repairs mechanisms. This means humans have a deep ability to go behind the red region to make energy from light that will re-write the biology books.

 

 

This should alert you to just how many paths are possible to alter tryptophan function.  This explains why serotonin and melatonin, both created from tryptophan can have so many varied functions because this amino acid can be programmed by excited electrons, ground state electrons, or protons that activated to transfer information between 900nm-3100nm light.  

The second notable feature of red light beyond the normal photosynthetic window is the energy buildup in water. This creates order and a separating charge both require energy.  These components in nature have plenty of precedent.  They seem to be the boost that helps begin the initial steps of charge separtion in photosynthesis on Earth.  That initial energy boost comes from red light from the sun. Spectral measurements showed that all wavelengths explored, from 300 to 5000 nm, contribute, with the most effective lying between 1500-3000nm in the infrared region beyond cytochrome c optical window.

This a small example of how cells are capable of using quantum superposition in an aqueous solution, from the mitochondriac perspective.  Conventional biology has no chance of comprehending this because they do not understand how the biophysical levers change water’s physics.  

Why is this BIG to the mitochondriac?  

All diseases and aging are associated with low NAD+ levels at cytochrome 1.  This occurs when blue light interacts with Vitamin A and melanopsin to lower melatonin levels to cause mitochondrial damage and elevates heteroplasmy rates and human disease show up in doctors offices and patients lives.

 

 

Did you get it?  

Guess how tryptophan fits also  fits into this developing story in the matrix??

Aside from its role as an amino acid in protein biosynthesis, tryptophan also serves as a precursor for the synthesis of the neurotransmitters serotonin and melatonin and kynurenine also serves as an important intermediate in the pathway for the synthesis of the nicotinamide adenine dinucleotide co-factors, NAD+ and NADP+.

IT FORMS THE KEY MECHANISM TO IMPROVE REDOX POTENTIAL IN THE MATRIX

NAD+ is the key electron acceptor of cytochrome 1 of the TCA cycle.

NADP+ is the key electron acceptor in the Pentose phosphate pathway.  The TCA cycle is used for biosynthesis when oxygen is plentiful.  The PPP is used for biosynthesis when oxygen levels are poor.  

BOOM!

This is the black swan mitochondriac level of understanding modern medicine needs to help the public.

 

 

I just showed you a lot.  Ask some people you care about to join me on Patreon if you want to help them become a black swan.

CITES:

1.  Amino acid synthesis and metabolism – http://themedicalbiochemistrypage.org/amino-acid-metabolism.php

2.  Urea cycle – http://www.ncbi.nlm.nih.gov/books/NBK27982/

3.  Miles B, Amino acid degradation – https://www.google.com.au/?gws_rd=ssl#q=Miles+Amino+acid+catabolism

4. http://www.soc-bdr.org/rds/authors/unit_tables_conversions_and_genetic_dictionaries/genetic_code_tables/

5. https://www.imperial.ac.uk/news/186732/new-type-photosynthesis-discovered/

6. mdpi.com/2310-2861/3/4/43/pdf