Author: info@kruseatdestin.com

For the first time, graphene has been successfully converted into a unique state of topological insulator. Graphene, a two-dimensional material with a single atomic layer composed only of carbon, has attracted attention as a next-generation electronic device material because of its high thermal and electric conductivity. Carbon, in this form, acts like a metal.

Graphene has other queer properties. It is not sensitive to time. The time-reversal invariant 2D topological insulators are also known as the quantum spin Hall effect (QSHE).  A physical system is time reversal invariant if the underlying laws are not sensitive to the direction of time. There are various accounts of time reversal transformation, resulting in different views on whether or not a given theory in physics is time reversal invariant.

However, graphene is useless for many photonic and electronic applications because it is too conductive and has no band gap.

The band gap is a fundamental property of a semiconductor because it determines its color and conductivity.

COLOR

A painter’s palette is rich with colors, some arising from the band gaps in natural semiconductors.

The mechanism behind the color we perceive in semiconductors can be explained by the band theory that governs color in many metals. Like metals, semiconductors have a reflective surface when polished but do not conduct electricity as effectively. Semiconductors frequently act as insulators and require particular conditions to become conductors. While metals become more resistant to the flow of charge with increasing temperature, semiconductors become conductors only with sufficient thermal energy, performing better as temperature increases.  Water does this as well because it is also a semiconductor.  It becomes more quantum coherent when light strikes it to form coherent domains.

If the substance has a large band gap, such as the 5.4 eV of diamond or the similar value of corundum, then no light in the visible spectrum can be absorbed. These substances transmit all incident light and are colorless in their pure forms. In their powdered forms, or when their structure prevents light from being transmitted, all light is reflected to the observer, and we see them as white. Such “large band gap semiconductors” are excellent insulators and behave like covalently bonded materials.

If a pigment can absorb all wavelengths, we see it as black, just as we see most metals as black in their powdered form. A white pigment absorbs no visible light. As in subtractive color mixing, we see its complementary color when a specific wavelength is absorbed from incident white light.

I mentioned this here: VIDEO

A “medium band gap semiconductor” is a material with a somewhat smaller band gap, such as the compound cadmium sulfide (CdS). This is the pigment cadmium yellow, known as the mineral greenockite (more examples in table).

This change in the band gap size is illustrated using mixed crystals of yellow cadmium sulfide (CdS, Eg = 2.6 eV) and black cadmium selenide (CdSe, Eg = 1 .6 eV), which have the same structure and form a solid-solution series. The photograph above illustrates the yellow-orange-red-black sequence of these mixed crystals as the band-gap energy decreases.

Mixed crystals such as cadmium sulfoselenide (Cd4SSe3) form the painter’s pigment cadmium orange and are also used to color glass and plastic. Mercuric sulfide (HgS) exists in two different crystalline forms. Cinnabar (the pigment vermillion) with Eg = 2.0 eV is a deep red but can transform upon exposure to light in an improperly formulated paint to the black metacinnabar with Eg = 1.6 eV in as little as five years; this has happened in several old paintings.

Another method of manipulating the color of semiconductor materials is by adding impurities. These doped semiconductors have energy levels within the band gap and allow us to tailor the wavelength of emitted light. Some semiconductors contain impurities in their natural state, providing useful insights into these behaviors.

CONDUCTIVITY

A band gap prevents short circuits since the electrons aren’t continuously in the conduction band. A small band gap allows the solid to have a strong enough flow of electrons from the valence to conduction bands to have some conductivity. So graphene would constantly short out, which is why adding graphene to the human system is a problem.

If the band gap is too high, most daylight photons cannot be absorbed; if it is too low, then most photons have much more energy than necessary to excite electrons across the band gap, and the rest is wasted.

A band gap is the distance between the valence band of electrons and the conduction band (see the picture above). Essentially, the band gap represents the minimum energy required to excite an electron to a state in the conduction band where it can participate in conduction.

Graphene’s basic properties show for the first time that electrons in a non-metal appear like a metal, and it can behave like a fluid.  Not having a band gap makes it unique.  Metals tend not to have band gaps, either.

In ordinary, three-dimensional metals, electrons hardly interact with each other. However, graphene’s two-dimensional honeycomb structure acts like an electron superhighway in which all the particles have to travel in the same lane. The electrons in graphene act like massless relativistic objects, some with a positive charge and some with a negative charge. They move at incredible speed — 1/300 of the speed of light — and have been predicted to collide with each other ten trillion times a second at room temperature.  These intense interactions between charge particles have never been observed in an ordinary metal.

Life uses carbon, just not in graphene form. When you have a material that’s one atom thick, it’s going to be really affected by its environment. Life cannot have a detector that is this sensitive because there is no way to control the signal well without large external electric and magnetic fields.

If the graphene is on top of something rough and disordered, it will interfere with how the electrons move in that substance. It’s essential to create graphene with no interference from its environment.

Quantum mechanics describes very small things, like electrons, while relativistic physics, pioneered by Albert Einstein, describes very large and very fast things, like galaxies.

Linking classical hydrodynamics theories with Einstein’s theories of relativity can describe high-energy systems like supernovas and black holes.

But it’s challenging to run an experiment on a black hole. Enter graphene.  When an electric field drove the strongly interacting particles in graphene, they behaved not like individual particles but like a fluid that hydrodynamics could describe.  Instead of watching how a single particle was affected by an electric or thermal force, we could see the conserved energy as it flowed across many particles, like a wave through water.

We discovered physics by studying black holes and string theory, which we see in graphene.  This is the first model system of relativistic hydrodynamics in a metal.  Life is based on low-energy physics systems at a small scale. Quantum mechanics rules that domain.   A small graphene chip can be used to model the fluid-like behavior of other high-energy systems found in galaxies and black holes.  Einstein’s relativity rules high energy systems.

Materials conduct heat in two ways: through vibrations in the atomic structure or lattice and carried by the electrons themselves.  Science has needed to find a clever way to ignore the heat transfer from the lattice and focus only on how much heat the electrons carry.  To do so, the researchers turned to noise. At finite temperatures, the electrons move about randomly:  the higher the temperature, the noisier the electrons. By measuring the temperature of the electrons to three decimal points, researchers were able to measure the thermal conductivity of the electrons precisely.  Converting thermal energy into electric currents and vice versa is notoriously complex with ordinary materials.  But in principle, with a clean graphene sample, there may be no limit to how good a device you could make.

SUMMARY

I have a sense that we might be able to cure people using this TI and high-intensity light in combination, but we will have to test the theory.

The material’s band gap is determined by its molecular structure; semiconductors’ periodic, crystalline atomic structure gives their valence electrons the ability to become conductive at specific temperatures.  Carbon on the periodic table has many atomic crystalline forms.  For example, as a diamond, its molecular arrangement gives it a large band gap and can act like a semiconductor.  Graphene is also a crystalline form of carbon, and it has no band gap and acts like a metal.  All living things are carbon-based, but our atomic arrangement is between diamonds and graphene.

This explains why the atomic structure inside your cells is exact and why the jabs were a problem because of the 51 elements they all contained.

Photosynthesis uses visible light to separate water.  To effectively utilize visible light for water splitting, the typical band gap of the semiconductor should lie within the range of 2.0 to 3.0 eV.

Visible light covers the range of approximately 390-700 nm, or 1.8-3.1 eV.

Viktor Schauberger found in water what modern-day physicists are finding out about graphene. Both are fluids that can act like metals when environmental conditions are just right.  What does this finding really imply to humans who observe nature well? This means that low-energy small-scale systems like cells and high-energy systems like CERN use physics very differently than the low-end system in mitochondria, which is quantum-based.  High-end energy systems on a large scale are relativity-based.  People don’t realize that semiconductors act by paying attention to the local environment……..it happens with graphene, and it happens with DHA.  Water and graphene have something else in common.  They are both semiconductors in specific environments.  Semiconductors are antennae for electromagnetic radiations; the thinner they are, the better the antenna is at receiving electromagnetic signals from outside.

Could a neurotropic virus disrupt the main semiconductor system in our brain to alter it?  We’ve seen Zika and Covid clearly do that.  I believe the mRNA jabs do this as well. DHA and water are where it all begins in the brain.

Energy expenditure is all tied to cerebral blood flow (CBF) and cerebral metabolic oxygen consumption (CMRO2).  Is it possible that a virus could alter the brain, causing microcephaly today?  Yep…….So, shrinking cerebral tissue reduces brain volume, improving CBF to CMRO2.  This saves energy when the environment is not ideal.  Might there be a reason Zika, autism, Alzheimer’s, and ALS have all shown up out of the blue?  Might it be why all the post-COVID complications have shown up as well? I think so.

Might that reason be a lack of energy from mitochondria?  Yep.  When a semiconductor senses electromagnetically, there is not that much energy to draw from it, and it shrinks in kind thermodynamically to adjust.  That saves massive energy.  The modern world has created an ionosphere filled with nnEMF and blue light.   Might these two things be linked in some way no one is making sense of?  Yes, I think so………

CITES
https://www.science.org/doi/10.1126/science.aad0343

This might be the most important video you will watch today.

This excerpts from my 2017 Vermont talk laying out how light/EMF controls everything in biology. Credits to “Video Advice” on YouTube.

We think Homo sapiens came into existence around 200,000-300,000 years ago in equatorial Africa based on varying data. Two hundred thousand years is a long time, and it’s a complex number to fathom. So, let’s convert that considerable, unfathomable number into something we can all understand. How about a single calendar year? To put 200,000 years of human evolution onto a calendar year scale, we first need to determine how long an actual calendar year would be on that scale. Here’s how the math works: One year is 365 days; 365 days is 8,760 hours; 8,760 hours is 525,600 minutes; and 525,600 minutes is 31,536,000 seconds. When we divide 31,536,000 seconds by 200,000 years, we determine that an actual calendar year passes by in 158 seconds, or two minutes and 38 seconds, on our scaled calendar year.

A decade is over in 1,577 seconds, or 26 minutes and 47 seconds. A century is complete in four hours, 22 minutes, 48 seconds. And so on. Now that we understand these numbers, we can put significant events in human history on our calendar year timeline. The first one is easy: Our species came into existence in Africa 200,000 years ago, at midnight on January 1. Humans first migrated out of Africa 70,000- 100,000 years ago—give or take—which is July 1 on our timeline. Let that sink in for a moment: For half of our existence as a species, we lived in, or very near, Africa. Modern humans entered Europe about 40,000 years ago.

That event occurred at midnight on October 20 of our calendar year timeline, when 80 percent of our species’ existence was over. It’s incredible how time relativity works, isn’t it, when you think about how humans and time are related? Archaeologists agree that agriculture joined our technological repertoire about 12,000 years ago in the Middle East. That’s December 10 at 2:24 a.m. on our calendar year timeline. The Industrial Revolution, a decades-long technological transition that ushers in, or at least makes possible, the modern era, occurs in the early afternoon hours of New Year’s Eve. Consider the implications of that fact: 99.9 percent of our species’ existence is over before we enter a technological context that is even remotely close to modern. But if you think about it, the Industrial Revolution is archaic compared to today!

No plastics. No cars. No television. No radio. No telephones, much fewer smartphones. The Industrial Revolution marks the beginning of a remarkably different era for us as a species, yet it occurred during the last 0.1 percent of our time on this planet. Wow! What about the iPhone? It shows up on our timeline at 11:31 p.m. on New Year’s Eve, within the last half-hour of our calendar year timeline.

People like Casey Means want you to believe that using nnEMF to monitor your blood glucose is better than how your brain does it. If you think I am kidding, look at this paper below. It should now be clear that, when examined from this perspective, humankind’s relationship with technology, with change, and with time is in uncharted territory for our CIRCADIAN BIOLOGY. The risks for man so far have exceeded the promise of productivity…………..in my estimation.

SUMMARY

Casey Means thinks a cell phone app is good for monitoring blood glucose and insulin. Look at the pictures to see how FLAWED this idea is. Trotsky’s granddaughter, Nora Volkow, who runs the National Institutes of Drug Abuse, wrote this paper a decade before Casey Means started using a cell phone app to monitor blood glucose.

Pardon me if I think she LEARNED nothing at Stanford University about doing research.

I think she learned to do this from Anne Wojcicki at 23andMe when she was married to Google’s Sergey Brin. Trotsky’s granddaughter, Nora Volkow, who runs the National Institutes of Drug Abuse, wrote this paper a decade before Casey Means started using a cell phone app to monitor blood glucose.

LEVELS technology is PSEUDOSCIENCE, and PEER REVIEWED SCIENCE ABOVE IT SHOWS IT.

 

CITES

1. https://pubmed.ncbi.nlm.nih.gov/21343580/

Every sunrise at the beach is a growing heat like a million blazing suns all focused on my mind. It lights my pilot light to warm my insides to conquer another day.

Chaos answers the question humans have posed for millennia, how did we get here?

In life, as everywhere in the cosmos, there exists a struggle between matter, both biotic and abiotic.   Chaos is entropy in abiotic systems (represented by fear in biotic systems) and order is a zero entropy state of life (represented by curious exploration in biotic systems).

Everything that is abiotic dies in “heat death”.  What we fail to realize today in biology is that biological order comes out of this chaos.  Chaos seems to the biologist to be a world described as we understand a heat engine in which heat is converted into motion only at the price of irreversible waste & useless dissipation.

TIME STAMPING

TIMING CONTROLS HOW ENERGY FLOWS IN CELLS. This is why time stamping by the circadian mechanism is the cornerstone of decentralized health. Biotic atoms must be time stamped to avoid chaos.

What is the human time stamping mechanism?

CLOCK and BMAL1 are positive regulators of circadian gene expression, and PER and CRY are the NEGATIVE FEEDBACK LOOP regulators that operate under day and night cycles. These are the positive and negative feedback arms of the circadian mechanism.

Ilya Prigogine defined dissipative structures and their role in thermodynamic systems far from equilibrium, a discovery that won him the Nobel Prize in Chemistry in 1977. Simply stated any dissiptive structure has to time stamp atoms in some way to avoid the chaos of heat death. In summary, Ilya Prigogine discovered that the importation and dissipation of energy into chemical systems could result in the emergence of new structures, which became known as dissipative structures, due to internal self-reorganization. In his 1955 text, Prigogine drew connections between dissipative structures and the Rayleigh-Bénard instability, and the Turing mechanism. Prigogine’s theorem is germane to these ideas. As such mitochondria should be thought of as time stamping machines at the core of cells.

Ilya Prigogine defined dissipative structures and their role in thermodynamic systems far from equilibrium, a discovery that won him the Nobel Prize in Chemistry in 1977. Simply stated any dissiptive structure has to time stamp atoms in some way to avoid the chaos of heat death. In summary, Ilya Prigogine discovered that the importation and dissipation of energy into chemical systems could result in the emergence of new structures, which became known as dissipative structures, due to internal self-reorganization. In his 1955 text, Prigogine drew connections between dissipative structures and the Rayleigh-Bénard instability, and the Turing mechanism. Prigogine’s theorem is germane to these ideas. As such mitochondria should be thought of as time stamping machines at the core of cells.

SUMMARY

The dynamic, energetic closure of the living system proposed built by Nature in cells gives rise to a number of important consequences. First and foremost, it frees the organism from the immediate constraints of energy conservation — the first law of thermodynamics — as well as the second law of thermodynamics, thus offering a solution to the enigma of the organism posed by Lord Kelvin and Schrödinger.

Because of the atomic organization of cells (AMO physics) there is always energy available within the cellular system. The energy derived from the sun is stored coherently in many places in cells, and ready for use, over all space-time domains. Mitochondrial water production is critical in the blueprint because it stores more light energy to use for TIME STAMPING. This picture shows that relationship CLEARLY

The fidelity of this water creation is the basis of the autonomy of organisms. Organisms are never simply at the mercy of their environments on account of the coherent energy stored. When the environment steals this ability from cells (nnEMF) cells are at the mercy of food and exercise.

More to the point, we don’t have to eat constantly (Leptin Rx), leaving plenty of time for other useful, pleasurable activities (SEX). This is why food is not the top of list of worries.

The other consequences are that the organism is exquisitely sensitive and free from the mechanical constraints of life on Earth; and satisfies, at least, some of the basic conditions for quantum coherence. Water creation by mtDNA provides that as well.

According to Ilya Prigogine, determinism loses its explanatory power in the face of irreversibility and instability in dissipative systems. This is a major departure from the approach of Newton, Einstein and Schrödinger, all of whom expressed their theories in terms of deterministic equations.

Indeterminism is the opposite of determinism and is related to chance. Chance is related to probability. In science, most specifically quantum theory in physics links directly to probability and not cause and effect. Indeterminism is the belief that no event is certain and the entire outcome of anything is probabilistic. Heisenberg’s uncertainty principle and the “Born rule“, proposed by Max Born, are often starting points in support of the indeterministic nature of the universe. Indeterminism is also asserted by Sir Arthur Eddington and Murray Gell-Mann. Indeterminism has been promoted by the French biologist Jacques Monod‘s essay “Chance and Necessity“. Ilya Prigogine argued for indeterminism in complex systems.

At life’s genesis chaos has to gain order. Dissipative structure theory really aims to solve this problem for biology by using physics.

Man has lost his humility with progress.  Humility is simply nature’s disposition that prepares our minds for living on intuition.  Nature’s disruption is what human life should rely upon. This process is controlled by sunlight and should be uncontrolled by man.  Manmade light has usurped this process.  This has allowed our brain to become preoccupied with technological progress which is now leading to biological disruption.

CITES

https://www.youtube.com/watch?v=jtMu-KFyKxM

https://forum.jackkruse.com/threads/mitochondrial-thermodynamics-diy-lesson-thread.27408/

If you are following the series, you will see the story of light stress, which was used in an adaptive fashion by mammals 65 million years ago to survive.  What underpins this effect, however, may shock you.  It is POMC.  It turns out that repeated exposure to low levels of mitochondrial stress, which environmental light induces, and various cytosolic and nuclear responses build resilience against higher levels of stress. This response would have significantly benefited mammals during an extinction-level event. This adaptive response, primarily known as mitohormesis, has been shown to extend health span and/or lifespan in several model organisms (Yun and Finkel, 2014).

What are the consequences here?

Ancient adaptions can lead to modern diseases when the spectrum of light changes again.  Remember, the initial adaptation mammals made was due to a lack of UV light in their environment, and they ran predominantly from 390nm to -3100 nm.

Today, centralized medicine looks at hemochromatosis as a disease when, in reality, it occurred as an epigenetic adaptation for an iron protection strategy that humans used to keep adult males alive to reproduce to allow them to survive in Europe for the last 1000 years.  Today, scientists are beginning to realize that our ‘junk DNA’ seems to be the raw material for constructing new wide-bandgap (WBG) semiconductors that use light to sculpt changes.  This is how our semiconductors transform light into epigenetic information to change the game so survival is guaranteed.   Evolution seems to tell us that in the last 1000 years, biology built a new way to defend against pathogens and events we have recently faced so that we can survive whatever life throws at us. This semiconductive fabrication plant in our bodies (POMC) acts like an evolutionary junkyard that allows us to innovate new novel ways to survive a bad event.

We now know that transgenerational epigenetics in the Viking men of the Northern coastline of Northern Europe was selected for hemochromatosis.  We believe that the COLD, harsh Tundra of the north was mineral deficient. Women with this genetic defect would have fared as better child bearers because they could absorb more iron to birth more children who also carried the hemochromatosis defect into the next generation.  It is also believed that the Viking men might have survived the disease because their Gladiator-type lifestyle was ferocious, and they often faced severe blood loss that might have offset the iron defect.  As Vikings settled in Northern Europe, the mutation grew using the “founders effect” caused by inbreeding due to small population sizes.  The founder effect means that any ‘non-lethal defects’ are highly selected for and carried in the entire population of a people.  It is believed that the ‘Viking defect’ was blended into the populations of Northern and Western Europe over 500 years to solve the recurrent Yersina outbreaks that caused the bubonic plague and almost extinguished humans in Europe.  The chronicity of the infection was an ‘epigenetic signal phenomenon’ that allowed for the selection of the hemochromatosis gene to confer reproductive fitness over longevity. At the same time, the Yersina remained active in the human population.  This remained true for close to 500-1000 years.

This “irony” may now explain why ancient physicians were barbers and bloodletters. We used to believe this practice was ‘quackery,’ but we now know that it was the survival of the wisest in action. Bloodletting had a significant role in conferring more longevity to those with hemochromatosis of European descent. When you bleed, you release a massive dose of vasopressin from the posterior pituitary.

Until the 20th century, bloodletting was standard practice. Then, it was stopped, and hemochromatosis became a modern disease. Canadian physiologist Norman Kasting found that bloodletting also released the hormone vasopressin (ADH) from the posterior pituitary. This release from the hypothalamus reduced their fevers and increased their immune function to act faster to save them. This finding was not causation, but the correlation between bloodletting and fever reduction is massive in human history. Bleeding them down may have helped fight infection when it was present.  When we bleed, vasopressin release is also altered. nnEMF does the same.

AUTOIMMUNITY AND VASOPRESSIN

Abnormal non-circadian release of vasopressin is linked to autoimmune conditions in modern man.

The arginine vasopressin hormone (AVP) of the posterior pituitary increases blood−brain barrier permeability. It also affects voltage gates and water balance in cells.

The immune system (IS) plays a vital role in protecting our body and can recognize its tissues from foreign molecules. The IS comprises lymphoid organs, cellular components, humoral factors, chemokines, and cytokines that respond to antigens that can harm the body (Shinde and Kurhekar, 2018; Verbsky and Routes, 2018).

The IS is highly and tightly regulated by mtDNA signaling and biophoton biology; however, its disruption could induce diverse pathological disorders, such as allergies and asthma; subsequently, when the host’s tolerance is exceeded, the condition becomes an autoimmune disease (Anaya et al., 2016).

nnEMF exposure is critical to BBB function, as Russ Adey and Allen Frey demonstrated in the 1960s.

Most autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus, Hashimoto’s thyroiditis, Crohn’s disease, type 1 diabetes mellitus, and MS are considered chronic illnesses today by centralized medicine. I consider them modern diseases linked to aberrant lighting and nnEMFs.

Some of them can induce neuroinflammatory and neurodegenerative processes in the CNS, which occur when the integrity of the BBB is compromised, as the pictures above show. The BBB is considered a highly selective barrier between the cerebral capillary blood and interstitial fluid of the CNS (Kadry et al., 2020; Schreiner et al., 2022) and helps keep harmful substances from reaching the brain as pathogens, toxins, and some drugs, as well as prevents the entry of IS components (Daneman, 2012; Kadry et al., 2020; Knudsen et al., 2022).

The principal constituent of the BBB is the endothelial cells, which provide protection and structural stability to the blood vessels on tight junctions (the liner sheets pericytes and astrocytes that ensheath the blood vessels and restrict the substances entering the brain or its immune system. The breakdown of the BBB promotes its permeability, permitting the entrance of immune molecules and lymphocytes, inducing autoreactive conditions in the blood-spinal cord barrier and BBB; as a consequence, damage and destruction of the myelin sheath increases, causing neurodegenerative disorders such as Alzheimer’s disease, Parkinson’s disease, ALS, and MS.

Similarly, neuroinflammation promotes the entry of IS components through the BBB and blood-spinal cord barrier into the CNS; therefore, an increase in the barriers’ permeability induces the interaction of pro-inflammatory cytokines such as the interleukins 1β and 17A (IL-1β and IL-17A), and tumor necrosis factor α (TNFα), which activates the downregulation of tight junctions in the endothelial cell barriers. IL-17A has been associated with the loosening of both obstacles, as shown by in vivo and in vitro assays. It is related to the production of reactive oxygen species by nicotinamide adenine dinucleotide phosphate (NADPH) and xanthine oxidase action, which are related to increasing in the endothelial cells’ permeability, causing a decrease in the occluding protein, zonula occludens-1 in in-vitro assays by Arima et al., 2013. The photoswitch I have mentioned before is a critical circadian controller of the ROS mechanism.

In contrast, the hyperactivity of sensitized lymphocytes induces the proliferation and secretion of IL-17 in MS. In addition, under normal conditions, T regulatory (Treg) cells alter and break down the balance in IS responses, which are accompanied by MS (Pot et al., 2011). Immune cells such as T and B lymphocytes, macrophages, and innate immune cells promote the disease’s pathophysiology. Myelin antibodies from B cells induce the loss of the myelin sheath. Furthermore, studies have demonstrated the presence of immunoglobulins IgG and IgM in patients with acute and chronic MS.

THESE DISEASES ARE MULTIFACTORIAL, BUT LIGHT AND POMC DYSREGULATION ALWAYS AT THE CORE

The human hypothalamus is a pivotal governing center for various metabolic processes in the body (Roh et al., 2016; Waterson and Horvath, 2015). Proopiomelanocortin (POMC)-producing neurons in the hypothalamic arcuate nucleus (ARH) are critical in regulating energy and water balance. (below)

POMC neuronal activity is strongly tied to mitochondrial function.  The higher your mitochondrial redox power is on your inner mitochondrial membrane, the more POMC a tissue will express.  More POMC = more semiconductors can be made.   POMC neuronal activity can be upregulated in mice by mitochondrial-derived ROS (Diano et al., 2011). Moreover, mitochondrial dynamics (i.e., fusion and fission) in POMC neurons are essential for maintaining whole-body energy and glucose homeostasis under altered metabolic conditions (Ramírez et al., 2017; Santoro et al., 2017) in all mammals. Despite the evident importance of mitochondria-originated signals in POMC neurons (Mishra et al., 2014), the details of the underlying mechanisms remain largely unknown in centralized medical research.

MITOCHONDRIAC UNDERSTANDING:  SUNLIGHT IS MANDATORY for making water at CCO during the day in mammals. If you do not get enough sun or live at a high latitude inside all day, you need more water to avoid the vasopressin release issued POMC.  If we do not get enough sunlight, we’re dehydrated, and then we lose circadian feedback control of vasopressin, and the entire water cycle in our body goes awry. This facilitates the development of autoimmunity.

This is how lousy clock management leads to epigenetic disease by decreasing mitochondrial redox power.  This is why big pharma is now pushing the use of anti-vasopressin analogs for MS patients.  Understanding POMC is understanding modern human neolithic diseases.  Mammals are creatures sculpted by light.

Recent work in this area shows that POMC neurons exhibit a dimorphic (biphasic) response to mitoribosomal stress in a dose-dependent manner; homozygous deletion of Crif1 was detrimental (i.e., severe light stress), whereas heterozygous disruption was beneficial (i.e., mild light stress).

How does IR-A light work in the sun?  A biphasic dose response has been frequently observed where low levels of red light have a much better effect on stimulating and repairing tissues than higher levels of light. The so-called Arndt-Schulz curve is commonly used to describe this biphasic dose response.  Centralized medicine has no idea how POMC works with light. POMC gets cleaved using the biphasic actions in light.  When cleavage is imprecise, disease results. Now you do, too.

Published research has found that low levels of mitoribosomal stress in POMC neurons induced high metabolic turnover and resistance to obesity through cell-non-autonomous mitochondrial stress signaling between the hypothalamus and adipose tissues.  That stress signal is VUV light emission in hypothalamic neurons in the leptin-melanocortin pathway.  That is how you stay thin.  It would be best to renovate the melanin sheets inside your tissues constantly.

In humans, prolonged or repeated cold exposure without surface-level UV light can increase the mass and activity of brown adipose tissue in the neck and supraclavicular regions, as defined by the uptake of glucose, and can improve glucose homeostasis. This is the off switch for ACTH release from POMC mammals used to survive in high latitude and poorly lit cold areas.

POMC neurons control adipose tissue thermogenesis (Dodd et al., 2015).  I knew 15 years ago that this discovery, as pictured below, was coming.  Brown adipose tissue works its job because of melanin.  Surprise!

Mitochondrial communication via optical transmission is the key to adaptive stress response following mitochondrial perturbation. Recently, small functional peptides encoded within the mtDNA, known as mitochondrial-derived peptides (MDPs), have been identified (Reynolds et al., 2020). MDPs represent a unique class of mitochondrial-encoded signaling factors that respond to mitochondrial stress and promote health/longevity (Galluzzi et al., 2018; Mottis et al., 2019; Quirós et al., 2016; Tan and Finkel, 2020).

Notably, MOTS-c (mitochondrial open reading frame of the 12S rRNA-c) mediates mitonuclear communication by translocating to the nucleus upon metabolic stress and regulating adaptive nuclear gene expression to promote cellular homeostasis (Kim et al., 2018).

 

Light sculpts your life.  VUV light, to be exact, when it comes to autoimmune disease conversion. That tells me that mtDNA biophoton release is where the defect is because terrestrial sunlight does not contain these frequencies.

Vasopressin (AVP) is a crucial hormone regulating water balance and is released during hyperosmolality to limit renal excretion.  It has a long history to explore.   It is produced by adjacent melanin in humans. Arginine-vasopressin (AVP) is a nonapeptide that is synthesized mainly in the supraoptic (SON), paraventricular (PVN), and suprachiasmatic nucleus of the hypothalamus. In addition, AVP is produced in several other brain areas and organs, e.g., the medial amygdala, bed nucleus of stria terminalis (BNST), and the adrenal gland chromaffin cells. Its release is associated with any body stress response, opening the blood barriers to the gut and brain.

SUMMARY

When vasopressin is altered, so is iron biology, which leads to an altered amount of ROS/RNS. Why?

Re-read this blog. It tells you how light and ROS are linked.   https://www.patreon.com/posts/quantum-75-p53-106441242

Why do we say oxygen is “reduced” when iron is oxidized in our body?

We say this because iron has gone from its elemental state with no charge ( Fe0) to its ionic state (Fe3+). Because the iron has lost electrons and become positively charged, it has been oxidized. The oxygen has been reduced because it gained the electrons iron donated to it. The electrons from the iron went to the oxygen. Every oxidation process has to have a corresponding reduction.

Iron exists predominantly in two biologically relevant redox states: ferric iron, the oxidized state (Fe3+), and ferrous iron, the reduced state (Fe2+). Fe2+ is well known to facilitate electron transfer reactions that can lead to the generation of reactive oxygen species.

The involvement of singlet oxygen in biophoton emission has implications for our understanding of many diseases like mast cell disease in the skin that links to immune function. Mast cell dysfunction is related to an absence of 1270 nm light in the skin of mammals. Singlet oxygen is known to liberate this frequency of light, as the picture in this blog shows. People with mast cell disorders do not make enough hydrogen peroxide from their mitochondrial respiration. As a result, a comorbid lack of sunlight containing 1270 nm light and lack of H202 creation in tissues is associated with immune dysfunction in mast cells. There is a lesson here that the photoswitch in our cells is teaching us about immune dysregulation.

People with autoimmunity, mastocytosis, and poor wound healing are always deficient in AM sunlight. Why? This is when we get a lot of NIR light with 1270 nm. Early morning sunlight, 6 AM -9 AM, has a relative irradiance with a higher amount of photons in the visible and NIR spectrum than midday exposure (noon). The picture tells why decentralized medicine always recommends AM sunlight. This sun time = TINA = THERE IS NO ALTERNATIVE.

CITES

G.T. Dodd, S. Decherf, K. Loh, S.E. Simonds, F. Wiede, E. Balland, T.L. Merry, H.Münzberg, Z.Y. Zhang, B.B. Kahn, et al.  Leptin and insulin act on POMC neurons to promote the browning of white fat.

Alvarez, J. I., Cayrol, R., and Prat, A. (2011). Disruption of central nervous system barriers in multiple sclerosis. Biochim. Biophys. Acta BBA – Mol. Basis Dis. 1812, 252–264. doi: 10.1016/j.bbadis.2010.06.017

Anaya, J.-M., Ramirez-Santana, C., Alzate, M. A., Molano-Gonzalez, N., and Rojas-Villarraga, A. (2016). The autoimmune ecology. Front. Immunol. 7:139. doi: 10.3389/fimmu.2016.00139

Arima, Y., Kamimura, D., Sabharwal, L., Yamada, M., Bando, H., Ogura, H., et al. (2013). Regulation of immune cell infiltration into the CNS by regional neural inputs explained by the gate theory. Med. Inflamm. 2013:898165. doi: 10.1155/2013/898165

ASTELLAS Pharma US, Inc. (2005). Vaprisol (conivaptan hydrochloride) injection. Rockville, MD: Department of Health and Human Services. Available online at: https://www.accessdata.fda.gov/drugsatfda_docs/nda/2005/021697s000_Vaprisol_Approv.pdf

Baron, J. L., Madri, J. A., Ruddle, N. H., Hashim, G., and Janeway, C. A. (1993). Surface expression of alpha 4 integrin by CD4 T cells is required for their entry into brain parenchyma. J. Exp. Med. 177, 57–68. doi: 10.1084/jem.177.1.57

Tissue atrophy and inflammation in retina cells in the far periphery of the retina were most predictive of cognitive status, the study found.  This is where the iPRGCs of melanopsin are located. https://lakegenevanews.net/lifestyles/health-med-fit/alzheimers-first-signs-may-appear-in-your-eyes-study-finds/article_91986868-0e1a-5002-80d2-29bd618e20a9.html

Microglial cells declined by 80% in those with cognitive issues, the study found.  POMc is located in microglial cells.

RPE cells are located between photoreceptor cells and the choroid membrane, with the basal side connected to Bruch’s membrane and tip microvilli connected to the outer segment of photoreceptor cells (Figure 1).

Damage to the structure and function of the retinal pigment epithelium leads to a variety of retinopathies, and there is currently no curative therapy for these disorders.  The reason for this belief is because modern medicine has no idea that renovating POMC internally by improving mitochondrial redox power and optimizing hemoglobin delvery is the key to RPE repair.

Light in our environment plays the most dominent role in obesity. The slide below shows how badly the food gurus have missed the boat.

Within the central nervous system, energy homeostasis is largely controlled by a fine balance between orexigenic and anorexigenic neuropeptides in the hypothalamic arcuate nucleus (ARC). Neuropeptide Y (NPY) and Agouti-related protein (AgRP) are coexpressed in neurons of the ARC and are potent orexigenic peptides, whereas proopiomelanocortin (POMC) precursor protein in the ARC is cleaved into potent anorexigenic α-melanocyte-stimulating hormone peptides. NPY/AgRP and POMC neurons in the ARC are considered “first-order” sensory neurons in the control of energy homeostasis (1) and receive, coordinate, and respond to changes in nutrient and hormonal fluctuations associated with changes in metabolic status. NPY acts on Y1 and Y5 receptors in the paraventricular nucleus of the hypothalamus to stimulate feeding, whereas acetylated α-melanocyte-stimulating hormone and AgRP peptides act as agonists and antagonists, respectively, at the melanocortin 4 receptor (MC4R). The POMC and AgRP interaction at the MC4R is collectively known as the melanocortin circuit. The critical importance of the melanocortin system in food intake and energy balance is highlighted by conditional gene ablation experiments. Ablation of POMC neurons in adulthood produced an increase in food intake and body weight and caused glucose intolerance, whereas ablation of AgRP resulted in rapid hypophagia, weight loss, and starvation = anorexia

Where the defect is on the retina and skin determines whether you get obesity or anorexia. We see this in drug addicts, alcoholics, and we see it in anorexics who live inside behind glass and infront of sceens. Both conditions, however, are associated with altered signaling in the leptin melanocortin pathways.

Why did obesity really happen dramatically after 1874 when the power grid was begun? LIGHT. It brought us INSIDE where the PROBLEM was.

When we started plugging in to the AC power all hell broke loose and this is where MOST chronic disease epidemics began. HARD STOP

Governments allowed NGOs like Gates and dermatologists began to advocate blocking the sun while putting screens in front of millions of humans from 1947 on. In 2009 Barry Obama and Biden changed FCC broadcast transmission from analog to digital and this brought LCD and plasma screens. Now LCD screen make up 99% of screens. All screens default setting out of a FACTORY ARE SET to blue frequencies. THIS MAKES YOU MORE COMPLIANT TO GOVERNMENT PROGRAMMING. The evolutionary time line of light makes this abundantly clear. Food gurus like the MEANS SIBLINGS are like propaganda wings of the Federal government who are now weaponizing MKULTRA programming at GLOBAL SCALE. They are paid by a16z who is a Silicon Valley investment group who supports Big Tech where all blue light begins.

THIS WAS THE GOAL OF MKULTRA —-> https://www.youtube.com/watch?v=SiBFtwbyv44

In 2007 Barry Obama said we are also going to remove incandescents which contain some UV and IR. the government knew what they were doing. They knew it make you more susceptible to things coming from Wuhan. They told us that it was to lower energy bills. That was a PR bullshit story that the media sold and then the food gurus were weaponized like legacy media reporters to repeat the liwe so you’d all believe it. AND 99% of you have.

They did it to lower our dopamine levels to make us more responsive to the political moves. This also lowered out melatonin levels simultaneously and this is why all kids now get Rx for melatonin from pediatricians who are complicit in the chronic disease epidemics. When dopamine and melatonin drops SO DOES POMC TRANSLATION. This means melanin never gets made properly. THAT IS WHERE YOUR DISEASE EPIDEMICS COME FROM.

Mental disease, drug addiction and obesity all come from obstruction of this pathway by modern lighting. LOOK AT THE PICK ABOVE. YOU ARE BEING LIED TO IN A BIG WAY by the NGO and Government partners in BigHarma and Big Tech that both sit at the CANTILLON EFFECT. Calley and CASEY MEANS ARE PAID BY THESE PEOPLE and this is why both of them have companies that use WBAN technology which MAKES YOU SICK. They got this idea from the Wojcicki sisters of YT and 23andme and YT and Google’s Sergei Brin. See this here to show the PROOF —–> https://threadreaderapp.com/thread/1826512218088722575.htmlXIA

THE DRUG ADDICT STORY BELOW LINKS TO WHY SOME GET ANOREXIA.

SUMMARY

In the leptin melanocortin system of the retinohypothalmic tracts in the eye the, hormones of the “fed state” such as leptin and insulin, are released in the bloodstream by adipocytes and by the β-cells of the pancreas, respectively, cross the blood-brain barrier to bind to leptin and insulin receptors on the surface of pro-opiomelanocortin (POMC) neurons to promote processing of POMC to the mature hormone α-melanocyte-stimulating hormone (α-MSH), which signals to decrease energy intake.

The three MSH molecules from proopiomelanocortin (POMC) decrease appetite, increase satiety, and increase energy expenditure:

• Alpha-melanocyte stimulating hormone (α-MSH)

  A POMC-derived neuropeptide that binds to melanocortin-4 receptor (MC4R) in the brain to suppress appetite and increase energy expenditure

• Melanocortin-4 receptor (MC4R)

  A receptor expressed by neurons in the paraventricular nucleus (PVN) that suppresses food intake when bound to α-MSH

• POMC neurons

  Neurons in the hypothalamus that produce α-MSH and other peptides that act on MC4R-expressing cells

Leptin and serotonin are signals that promote POMC neuron action, which leads to the production and release of α-MSH. In contrast, ghrelin is an orexigenic hormone that activates AgRP neurons, which antagonize MC4R signaling and increase food intake.

Mutations in the POMC gene have been linked to severe childhood obesity.

STOP LETTING THEM LIE TO YOU.

IT ALL BEGINS WITH LIGHT

CITES

Zarbin M. (2016). Cell-Based Therapy for Degenerative Retinal Disease. Trends Mol. Med.22 (2), 115–134. 10.1016/j.molmed.2015.12.007

Tian X., Cui Z., Liu S., Zhou J., Cui R. (2021). Melanosome Transport and Regulation in Development and Disease. Pharmacol. Ther. 219, 107707. 10.1016/j.pharmthera.2020.107707

https://threadreaderapp.com/thread/1826512218088722575.html

Robert O. Becker, MD, found that some part of matter in our nervous system created a DC electric current, and that current drove all healing and regeneration. That current also broke with convention because it could de-differentiate RBCs to primative forms to create stem cells to regenerate our tissues. The current needed was precise. One-thousandth of a million ampere of current was required to do this. Anything less or more than this was a problem. This was the electromagnetic criticality that mammals needed for well-being. When I met Becker in 2007, I told him that POMC was the source of his work. Light was the source of man’s regenerative current. This idea was more heterodox than his journey into bone regeneration.

The phenomenon of criticality has been postulated to explain the sudden emergence of new properties in a wide range of complex systems, from avalanches to flocks of birds to stock market crashes. I believe it is behind the modern epidemics of autism and neurodegeneration, too. I think it is exacerbated by things that reduce our redox potential, like vaccines and modern lighting. Neuroscientists are now seeking evidence that criticality is at work in the brain’s networks of neurons.

Criticality is linked to how the OPN5 melanopsin receptor works in the human brain. It is the critical opsin that defines modern chronic diseases.

I believe self-organizing criticality theory (SOC) needs to critically look at the work of Ilya Prigogine on how dissipative systems operate.  I believe this is why SOC is stuck in the mud with neuroscientists.  To understand this idea you need to understand how the liberation of Vitamin A from our non-visual photoreceptors begins the process of diseases by destroying all of our photoreceptors.

People often forget that all of the cytochrome proteins in our mitochondria are also heme-based photoreceptors linked to this damage cascade. This is found in every human disease I have studied, from autism and Alzheimer’s to zoonosis.

All mitochondria liberate heat. and melatonin and water  Heat is linked to universality and universality is linked to the fractal design found in nature.

Joseph Fourier made an essential contribution to physics in the nineteenth century when he published his mathematical formulation of how heat propagates. Today, Fourier’s law tells us that heat flows from regions of higher temperature to areas of lower: “that heat flux is proportional to the difference in temperature.” This law is a remarkable insight into universality. Here, we have many disparate macroscopic systems, from solids to gasses to liquids, each containing an inordinate sum of molecules and conforming to a heat conductivity law. To the chemist Ilya Prigogine, Fourier’s description was the “birth of the science of complexity.”

Around the same time, another universality, the first law of thermodynamics, was formalizing in science. It tells us that while “energy cannot be created or destroyed, it can be transferred from one form to another. Energy is forever conserved in the universe.

The critical phenomenon of nature is how light energy is transformed.  It can become mechanical, chemical, or vital energy (DC electric current). Light energy undergoes a continual conversion of itself as it interacts with matter.  The atomic organization of matter is the key to energy transformation and criticality.  This energy attraction occurs throughout space and drives the living force in cells. The energy flow is called kinetic energy, which drives heat into one form of energy or another. Thus, order is maintained in the universe—concerning the total energy in the system, nothing is deranged, and nothing is ever lost. Still, the entire machinery in cells, as complicated as it is, works smoothly and harmoniously.

I believe sleep is designed to get brains back to the SUBcritical state, but electromagnetic signaling from circadian biology is what is required to optimize electromagnetic criticality in neurons.  This electromagnetic criticality is linked to the optimized control of ALL charged particles in the entire system.  This creates the quantum coherent state in cells.  Normally, mitochondria set the system close to the subcritical tone of the system, but light sets off the criticality.

Order Comes Out of Chaos

Everything not alive dies in “heat death”.  Order always arises out of environmental chaos.  It’s a world described as an engine in which heat is converted into motion only at the price of irreversible waste & useless dissipation.

So energy all goes from one conversion to another and tends toward a final state of thermal equilibrium = ‘heat death.’  Life uses cells to slow this “heat death” by slowing the flow of entropy by arranging atoms ideally in cells.  The cell is a dissipative structure that controls the flow of energy so that it can be transformed many more times into useful work.  This is the basis of why critical points are needed in Nature’s biological machines inside of cells.

An isolated system is on an inexorable track toward equilibrium, but many interesting things can happen in between. Life at equilibrium is called rigor mortis.  Time spent in a far-from-equilibrium state is called life.  Health is the slowest form of death we can create with our choices around light.

In health, solar energy is pumped into cells and stored, and entropy decreases. This is the blueprint of how all living dissipative structures operate.  The sun is itself a dissipative structure.  So is the Parker spiral.  It is the heliocentric magnetic sheet that engulfs the entire solar system.  This magnetic sheet in the solar wind affects all living things on Earth via their mitochondria.  This is partly how our brains remain subcritical at the proper times.  Sunlight alters our tissues to induce changes in our charge density.  In animals, electrical information generated in mitochondria is more crucial than genes that form tissues, but few people realize this today in centralized science. It turns out that charge density variation by moving electrons around our tissues is the key to understanding what life is up to when it is connected to the decentralized energy systems of Nature.  That is how super-criticality is found during the living state.

WHAT DOES LIFE LOOK LIKE WHEN IT RESIDES ABOVE OR BELOW THIS CRITICAL POINT?

Diseases are processes associated with chronic energy loss and entropy gain. The causes can be vast, but the endpoint is always the same.

When life is lived below the critical point for any reason, heteroplasmy rates expand in those tissues until energy flow ceases, organ failure takes over, and death is invited to your life.

Earthian life is an open system—doing as much as it can with the solar energy it receives from our aging Sun. The living cell represents an incessant metabolic activity, where thousands of chemical reactions take place simultaneously.

Fasting Factoid:  Is fasting tied to a solar mechanism related to iron hemoglobin?  Before you answer, think about these facts about fasting.  The blood diminishes in volume in proportion to the decrease in the size of the body during a fast so that the relative blood volume remains practically unchanged during a fast. The quality of the blood cells and plasma is not impaired; indeed, an actual rejuvenation of the blood may occur if that blood is allowed to touch the rays of the sun because of how porphyrins work with sunlight.

The old literature on fasting has pointed out that the first effect of a fast is to increase the number of red blood corpuscles.  This increase is an acute phase change only and improves the quantum yield if the blood is allowed to be irradiated by sunlight.  If the fast goes too long, the spleen acts to decrease the RBC’s mass.  When this happens, the astute clinician should expect mitochondria in tissues with high heteroplasmy rates to lose more energy and enlarge as things do when they lose energy (think heart failure or a dying star).  The acute increase of erythrocytes during the early part of the fast should be regarded as an improved flow of energy from mitochondria due to autophagy and a cessation of overeating. This red blood cell mass increase has been repeatedly observed in acute anemia cases throughout 20th- 21st century medicine. The decrease in RBC mass is only observed after the starvation period is reached.  What are the implications of this, and how might we mito-hack this with new devices that could take advantage of this unique aspect of RBC mass?

When humans are sleep deprived or suffering a circadian mismatch, their brains become supercritical, although a good night’s sleep can move them back toward the critical point. It thus appears that brains naturally incline themselves to operate near this critical point,  This points out why sunrise every AM is critical to resetting the critical point in the brain every day. POMC activation is the key to regeneration.

The miracle of our brain’s physiology isn’t that you can see the world as it is. It’s that you can see the world as it isn’t. My job is to explain what you can’t see, but physics has been observed to exist. The absence of evidence in a biology book is not an absence of effect in nature.

The claim that the cortex operates near the critical point is a sweeping one, encompassing optimal information processing, neurological health, and a nearly universal application across species. The need for strong scrutiny is not surprising.

So, why is this view of the critical brain still just a hypothesis? While the evidence in its favor is good, it’s still under discussion.

Early critiques pointed out that proving a network was near the critical point required improved statistical tests. The field responded constructively, and this type of objection is rarely heard these days. More recently, some work has shown that what was previously considered a signature of criticality might also be the result of random processes. Researchers are still investigating that possibility, but many of them have already proposed new criteria for distinguishing between the apparent criticality of random noise and the true criticality of collective interactions among neurons.

Meanwhile, over the past 20 years, research in this area has steadily become more visible. The breadth of methods being used to assess it has also grown. The biggest questions now focus on how operating near the critical point affects cognition and how external inputs can drive a network to move around the critical point. Ideas about criticality have also begun to spread beyond neuroscience.

Citing some of the original papers on criticality in living neural networks, engineers have shown that self-organized networks of atomic switches can be made to operate near the critical point so that they compute many functions optimally. The deep learning community has also begun to study whether operating near the critical point improves artificial neural networks.

SUMMARY

Dreams seem natural when we are in them, but it is when we wake up that they begin to seem strange to us.  Might this be evidence of how a point of criticality operates in the mammalian neural network?  At the critical point of CNS, fluid acts as a single phase of matter. These fluids are both liquid and gas in quality. These fluids are highly compressible at a critical point, and they allow for travel into other dimensions of the mammalian network.  Here are some other significant ideas linked to this story. POMC is required with mtDNA water to create the DC current of Becker. This is the critical point you need to acquire in your journey of wellness.

https://twitter.com/DrJackKruse/status/1680466637827235845

CITES

https://www.jneurosci.org/content/31/1/55.full

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3665287/

https://www.nature.com/articles/nphys1803

https://www.sciencedirect.com/science/article/pii/S0896627319307378

https://www.sciencedirect.com/science/article/pii/S0896627319309596

https://www.sciencedirect.com/science/article/pii/S0753332223000756

Sunlight stimulates T lymphocytes in the skin through a mechanism separate from vitamin D production. Sunlight energizes T cells, which play a central role in human immunity.  T cells, whether helper or killer types of lymphocytes, need to move to do their work, which is to get to the site of an infection and orchestrate a response. The study below shows that sunlight directly activates critical immune cells by increasing their movement.  How do they move?  Light in the sun creates H2O2, which makes cells move.  This shows that free radical signaling has a positive connotation, not a negative one.  This is a magnetochemical signal in the sun’s light.

T cells possess intrinsic sensitivity to blue and UV light. Solar light detection is coupled to the generation of H2O2 and activation of Src kinase and PLC-γ1, leading to elevated intracellular [Ca2+]. Non-visual photosensitivity is greater in activated T cells and enhances T-cell motility.

The majority of T lymphocytes are found in our skin. NK cells come from T lymphocytes. NK cells are cytotoxic lymphocytes that have drawn considerable attention recently as a promising tool for immunotherapy in patients with various refractory hematological malignancies and metastatic solid tumors. I have a sense that the SV40 promotor inactivates T cell motility in turbo cancer formation.

Why?  Only a partial response has been shown in centralized clinical results of experimental protocols, attributed mainly to the relatively low number of NK cells infused and their short in vivo persistence. A significant challenge, therefore, in advancing the clinical applicability of NK cells is to expand ex vivo NK cells that display increased functionality upon in vivo infusion. Different combinations of cytokines have been studied to induce NK cell expansion. Sunlight, with fasting, increases NAD+ in cells and is active during the leptin-melanocortin pathway.  This might be critical for jabbed patients to avoid oncogenesis due to the presence of 60 billion copies of the SV40 promoter per jab.

SV40 Ori’s normally need T antigen to replicate. This is true unless they have ColE1 and F1 origins, as seen in the Pfizer vaccine.

I also think CBD oil needs to be studied in those who took the jab based on Kevin McKernan’s work and our results with people who used it during COVID-19.

WHY?

A lesson on the quantum biology of photosynthesis.

Did you know that we get a 40% higher yield of cannabis plants at 1200 ppm CO2?

Currently, the biosphere is starved of CO2 at 420 ppm.

The architects of our vaccine bioweapons program support lowering CO2 further.

They also want to block the sun, which would keep CO2 emissions lowered.

Might that be because Cannabis and nicotine were an antidote to the designs they employ? Did you know King Charles just announced a plan to ban nicotine products in the UK by 2030? Do you think he knows something?

Cannabis split from Humulus lupulus (hops) 24M years ago.

Why is its photosynthesis optimized for 1200ppm CO2?

ANSWER: It is because of the effect of deuterium on water. Plants grown in D(2)O show a decreased tendency to fractionate carbon-13 during photosynthetic incorporation of carbon dioxide. The isotopic ratio C(13)/C(12) of the tissues of deuterated plants appears to be proportional to the deuterium content of the tissue. This effect was found in specimens of the partially deuterated vascular plant Nicotiana tabacum, cannabis, and in cultures of the fully deuterated alga Chlorella vulgaris.

Another trick BigHarma plays is using an aluminum adjuvant, which allows you to camouflage all the DNA in your shot. Why? Aluminium is a perfect atomic reflector of UV light biophotons, so it blocks the optical signaling from DNA to the ribosomes and mtDNA to stimulate protein translation. This has a massive effect on POMC translation as a dopant contaminant.

When Gardasil was first approved in 2006, Merck assured the FDA in the USA that there was no HPV DNA in the vaccine. However, this was challenged in 2011 when Sin Hang Lee found HPV DNA in a young, sexually naive girl who had never been exposed to the virus but had received Gardasil. This was due to the atomic effects of contaminant atoms. They ruin the semiconductor of cells and destroy optical signaling, making disease generation more likely.

In 2011, the FDA admitted residual DNA in the vaccine but said it was “expected” to be in products manufactured using recombinant technology and that Gardasil posed “no risk to vaccine recipients.”

The FDA claimed that the presence of DNA fragments was not a problem without showing any studies to prove it had been investigated or that it was safe for humans.

Is there a risk that residual DNA fragments in Gardasil can enter host cells and integrate into the genome? I believe there is now good evidence that this is the case. All one needs is a “transfection agent,” and some studies suggest adjuvants in vaccines can act as transfection agents.

In 1985, the FDA set an upper limit of 10 picograms per dose. In 1987, the WHO increased its recommended limit to 100 picograms and then again to 10 nanograms (i.e., 100 times higher)—a limit now adopted by the FDA.

The paper pictured above argues that it’s difficult to quantify the levels in Gardasil jab, though. HPV DNA is tightly bound to the aluminum adjuvant (AAHS) and forms an insoluble precipitate, which gives it incredible substantivity in a cell and makes genomic intercalation more likely.

Most genomic experts can easily detect the HPV L1 gene DNA in the insoluble precipitate and the soluble DNA in the solution using nested PCR with Sanger sequencing for confirmation.

Genomics expert Kevin McKernan, the first to discover residual DNA in Pfizer’s COVID-19 vaccine, taught me this lesson during our collaborations. He agrees with Dr. Lee’s papers above that the FDA’s permissible limit of 10 nanograms is futile because it increases the cancer risk for the compliant.

Kevin has repeatedly pointed out on his X page that the trick the FDA plays with the guidelines is when you ask scientists to measure the residual DNA, you’ll miss most of the DNA because it is all bound up to the aluminum adjuvant. This is why they add Aluminum to the jab to hide the contamination. This is why one should never comply with any jab. All jabs carry this risk now.

The 10-nanogram limit they’ve created is just smoke and mirrors because they have legal protection now. They say if it’s below that, then they don’t care, but here you have something that hides the DNA in aluminum, and they whistle past the graveyard of patients who took their shots.

Aluminum is one problem, but now mRNA vaccines are known to have 55 different atoms present and SV40 promoters, all of which can perform this task. I also believe mRNA technology in food will pose this EXACT risk to jabbed people.

SUMMARY

I have spoken with Dr. Alexis Cowan about how sunlight, specifically UV light, operates the wiring diagram of the mitochondria. You can find that here.  It essentially shuts it down while increasing skin T lymphocytes to become NK to hunt for cancer cells—people who took the jab need to upregulate their own NK T lymphocyte production. Sunlight with UV light appears to be the best way to do this. It should be the key option discussed with patients to avoid oncogenesis.

In decentralized medical research, researchers interested in using exogenous nicotinamide (NAM) supplements to help the jabbed should be considered in those who took jabs known to have the SV40 promotor.  I plan on discussing this further with Nicole Shanahan soon after the election.  We need this study for those who complied and took the jab without knowing their risks. The slide below shows why I think 24/7 exposure to daily 380 nm light is the best plan for the jabbed. It directly helps metabolism, but it actually repairs the circadian clock gene dysfunction—the jabs also CAUSE circadian disruption.

NAM is a form of Vitamin B3 and a potent inhibitor of enzymes that use NAD+ for their activity. Hence, NAM might be directly involved in controlling redox-sensitive enzymes, mitochondrial functions, cell metabolism, energy production, and cell motility.  The studies done on NAM by Josh Rabinowitz and Charles Brenner have not been kind to NAM or their analogs, but in post-jabbed patients, the results may be quite different, and I believe this should be studied.

My perspective on chronic disease creation caused by the jabs is a different perspective for disease modeling than exists today in centralized science. It is far more coherent and discernible to understand when you examine the simplicity of the idea.

Biologic information from the environment must have a connection to its source in cells; that source is mtDNA. The mtDNA then makes a wireless connection using light to the sun and Earth, which is paramount for wellness. Only then can the data from nature remain reliable and pliable for life’s uses through all these physical laws I have given you over 15 years.

Only living things sense and seem to know about their connection to “ex-formation” by the mechanisms built into how mitochondria can feel our connection to Mother Nature. That connection is hard-wired into every cell by its mitochondrial genome and copied and pasted into the maternal germ line of all the genomes of life.

In this way, mothers must remain connected to nature to pass information on accurately. The disease can manifest in children when mothers do this. The child does not need to sense this to get a disease because all mitochondrial DNA comes from our maternal line. This is childhood cancer, depression, and suicides have been on the rise since 1900. No one sees what I see, but they will because of what happened with COVID jabs. This is why the jab architects now weaponize the Means siblings on media. They want to rewrite jab history like Bernays taught the Industrial-military complex.

Mitochondria with high heteroplasmy should never be connected with an excellent nuclear genome if one wants to stay healthy. This is obvious during pregnancy when making a child, as decentralized 16 explains. Leptin resistance in a mother is a sign of avoiding pregnancy; it blocks fecundity. It is also why infertility rates are skyrocketing in the Western world, where technology use is rising, ala MKULTA/Brain Health Initiative. The kids born to these parents are now facing mitochondrial diseases like cancer, diabetes, depression, and suicide at unparalleled rates. 

This is akin to putting an Apple computer into a Windows server. Putting insufficient data from mitochondrial DNA into nuclear genomes that are not coherent leads to all disease categories. That is, conditions of existence and natural selection are working in order. They are the Ying and Yang of epigenetics.

Centralized science of vaccination and light use is now genetically engineering changes to the mitochondrial DNA using excessive blue light and nnEMF to expand their distance, ruining our cell’s data processing ability. In this sense, our modern civilization is genetically engineering us to extinction, and the biological information cells continue to receive will get more and more misunderstood until the genome responds with nonsense code. Nonsense code = Chronic disease illness.

The best example of nuclear genome nonsense coding = is cancer. This is how the expansion of the cytochrome proteins over time can lead our cells to oncogenic change. The implication: Might mutations in mitochondria be needed and required from an evolutionary perspective to get us to change what we are doing to remain healthy? Yes, that is my answer. Thus, the most helpful answer for life today is to stay in direct contact with its “ex-formation” to avoid this situation where the disease has to warn us to change our environment. This is why wild animals migrate. They are in tune with nature because they are appropriately connected to the sun and Earth. They do not live indoors as a man does and take BigHarma jabs. The light that regenerates you is the answer. It is never food first unless you are working for the opposition. When you know better, you simply do better.

THE TAKE AWAY:

Electrons are like glue to light as words are to their meaning. Electrons can catch terrestrial sunlight, like words, and capture meaning to create the world we experience. We need sunlight to produce NK killer cells after SV-induced cancers. We can only hope that our words can capture what we mean, but we know they can’t possibly capture that much joy, grief, or wonder…….or can they? I’ve been telling you ALL for some time to spend less time with food gurus like Calley and Cassie Means and way more time with bio-physics because that is where the treasure is buried. I believe the Means siblings are nefarious in this game as well.

Those are the globalists who are all in on the global vaccination plan I warned about in the Danny Jones podcast.

EXPLAIN centralized PEER REVIEW link to the MOSSAD and the US Federal government LIKE YOU ARE THIRD GRADE

I’ll repeat it… “Peer review” is a method used in centralized science to strip you of individual medical sovereignty.

If you outsource your thinking to centralized experts who are controlled by the BigHarma Gutenberg printing press, you will wind up being chronically wallet-biopsied by the industrial healthcare complex. Do you understand the game yet?

Go to minute 3:33:00

https://open.spotify.com/episode/3F72MA1NwSDgmDOG1X7x15

Remember that the PEER review is run by dual passport holder (UK/Israel) Robert Maxwell. His daughter is in jail protecting the Zionist Federation guys and the Royal Family. Maxwell was a primary actor in the MEGA Group. She worked for Epstein, and the MEGA Group employed Epstein.

Epstein has ties to Israeli intelligence and well-documented ties to influential Israeli politicians and the Mega Group. Those guys are layered links to the MOSSAD and Meyer Lansky.

Ronald Lauder, a cosmetic heir who makes billions blocking women from the sun, is a Mega Group member. He has known Trump for close to 60 years because they are both Wharton Alumni in Pennsylvania. He is a former member of the Reagan administration and a long-time donor to Israeli Prime Minister Benjamin Netanyahu. He is very influential in Israel’s Likud Party. He was a long-time friend of lawyer Roy Cohn. Robert Kennedy Sr. was Cohn’s assistant to Senator Joe McCarthy in the 1950s during Eisenhower’s administration. Cohn’s father ran B’nai B’rith as its President. Roy Cohn was one of the first group of Americans who received the Salk vaccine, and he died of AIDS in 1986. Sadly, no one ever checked him for SV40, but if I were in power, I think I’d look at his remains and sample them for SV40.

Lansky and his friends in MEGA lured Cohn to NYC to work for the accounts of MURDER Inc in 1954 and 1955, Cohn reportedly had deep connections in the 1950-80s to Ira Malnick.

Lansky worked on cryptography from 1969 to 1983. That work linked art sales and money laundering through the IRS and Treasury via infiltration of the Executive branch. This work created the PROMIS software program. It was stolen in a bankruptcy conspiracy by the DOJ and Industrial military complex corporations, Wackenhut. This program allowed Lansky and his associates to follow the Fed, Treasury, and IRS’s actions while also monitoring Israel’s financial movements. This gave Lansky data on all their financial interactions to see how well their programmable money project was progressing. I covered this here: https://kruselongevitycenter.com/djonesqapublic

This code in this software had dual use that allowed the US and Israeli governments to launder money for their intelligence arms. It also allowed Lansky and his syndicate to stay ahead of his partners in the Industrial-military complex by following where money was being moved. This software was critical in setting up the money laundering schemes from cartel smuggling and set the stage for the Iran Contra money laundering schemes that linked the MOSSAD to the Executive Branch. It also linked the drug trade into Mena, Arkansas, where John Gotti and the governor made sure that the project operated undisturbed.

These are the people I think Calley and Casey Means are working for.

CITES

1. https://www.nature.com/articles/srep39479

2.  https://journals.asm.org/doi/10.1128/jvi.68.2.787-796.1994

3. https://anandamide.substack.com/p/sv40-origin-of-replication-in-mammalian

If you understand the science of nnEMF you’d understand why the picture above is an oxymoron. The number one indication for spinal cord stimulation in the USA, according to CMS data, is diabetic peripheral neuropathy (DPM)

The global spinal cord stimulation market size was valued at USD 2.88 billion in 2019 and USD 2.41 billion in 2020 and is projected to grow USD 4.12 billion by 2027, exhibiting a CAGR of 8.0%. North America dominated the global market with a share of 73.96% in 2023.

DO SPINAL CORD STIMULATORS MAKE DECENTRALIZED SENSE FOR CHRONIC PAIN for DPN?

Is “the juice worth the squeeze?  Spinal cord stimulators were conceptualized based on the overly simplistic “gate control theory of pain” proposed by Melzack and Wall. Wall himself later wrote, “The least, and perhaps the best, that can be said for the 1965 paper is that it provoked discussion and experiment.” Is that still true based upon this paper written 10 years ago at the 50th anniversary of this “theory.”

HAS IT REALLY REVOLUTIONIZED OUR UNDERSTANDING OF PAIN, OR WAS IT USED TO BLOCK US FROM THE TRUTH ABOUT HOW SUNLIGHT REDUCES PAIN AND DIABETES?

The 1965 gate control theory of pain describes how non-painful sensations can override and reduce painful sensations. A painful, nociceptive stimulus stimulates primary afferent fibers and travels to the brain via transmission cells. Increasing activity of the transmission cells results in increased perceived pain.

So the answer should be a clear no, but we still do them in centralized medicine and CMS data says the market is close to 3 billion dollars. That is a lot of cash for a theory that still has not been proven.

Many people get stimulators for diabetic peripheral neuropathy.  How many pain specialists or neurosurgeons know about the links between POMC and neuropathy?

Very few. Why? In 1965, two authors, both linked to academic centers with military funding, proposed a new theory about how pain happens.

Should neurosurgeons and pain specialists know that POMC is critical in DPN and chronic pain?  Why aren’t they taught the truth?

POMC PAIN STORY IS A STORY ABOUT LIGHT = MKULTRA STORY

The endogenous opioid system is our body’s endogenous line of defense against noxious stimuli. This system is part of the leptin-melanocortin pathway in mammals.  Endogenous opioid precursors are proopiomelanocortin (POMC), pro-dynorphin (PDYN), and pro-enkephalin (PENK)) which are proteolytically cleaved to produce opioid peptides.

These bind to their cognate opioid receptors and trigger downstream signaling events.  Those ion channels are activated by K+ channels and inhibited by Ca++ channels.  Ironically we know that light and electricity can alter these ion channels.  Blu light and nnEMF were known to cause calcium efflux issues in the 1970s.  When K+ channels are activated or Ca2+ inhibited is usually part of the clinical event one should expect.  These set of circumstances result ultimately in the inhibition of neuronal excitability. Persistent neuropathic pain in DPN = diabetic peripheral neuropathy patients suggests a dysfunction in endogenous opioid-mediated antinociceptive mechanisms.  Blue light exposure is known to drive blood glucose high and drive insulin higher.  See Nora Volkow’s papers from NYU circa 2011.  There is a powerful correlation of blue light exposure to diabetic transformation.  Blue light exposure also seems to destroy POMC translation in mammals.

DID YOU KNOW RED LIGHT BY ITSELF LOWERS BLOOD GLUCOSE?

The interesting thing is no one has done a study yet to show that the combination use of UV and IR light might work to reduce pain in the centralized literature.

But did you know that sunlight also reduces the need for insulin for diabetic mammals? It is true, sunlight reduces insulin resistance in all mammals so far tested.

Previous studies have examined the link between the endogenous opioid system and diabetes, and reported that opioid peptide levels are altered in the central nervous system and plasma of diabetic rodent models as well as of diabetic human patients. Today, we now know the opioid levels linked to POMC translation in the peripheral nervous system (PNS) are altered during diabetes.

In fact, in vivo correction of this deficit can rescue diabetes-induced hyperalgesia and associated behavioral changes, thereby showing the relevance of the dysfunctional POMC-MOR signaling to the increased pain sensitivity observed in diabetes.  So, should DC electric pain stimulators be replaced with light therapy stimulators since POMC responds to UV light?  FYI: Your mitochondria is the UV light generators gone bad in diabetes. This is why all living cells have been shown to liberate ultraweak UV biophotons. Diabetics are no longer capable of this due to their defective mitochondria.

They do not want you to know this because they’d lose 3 billion in generator sales.

GOT IT?

It is well known that UV and IR light reduce blood glucose and decrease pain.  Moreover, it is also well known that UV light raises vitamin D levels, and Vitamin D levels highly correlate with increased pain and higher narcotic use.  Might light stimulators be better than spinal stimulators because the gate theory idea proposed by science linked to the industrial and military complex is entirely flawed?  Better yet, was it really flawed or was it put forth by design by the industrial military complex who paid for Melzack and Walls paper 60 years ago?

Ask TED what he thinks about the military and truth: https://x.com/ted_macie/status/1842629039682384115

SUMMARY

Your cells capture the light you choose to live under. This was the message distilled from MKULTRA program. Your choices are quantized in your melatonin and insulin levels. You are essentially an electromagnet for light. As a result of electromagnetic capture, the thoughts created in your brain via your retina and skin have a frequency associated with them and that frequency resonates with a select few. That resonance determines whom you collect in your lives. They are essentially antennas in your life and they are linked by the light you choose.

The centralized paradigm in power in healthcare wants people to think it’s a type of “lock and key mechanism” but this is factually incorrect. It is more like the electro-magnetic gate control we see in semiconducting light diodes. Melatonin receptors are found to a great degree in the retina, RPE, and the visual pathways of the brain. When light is absent then melatonin activates these “light diode-like” gates. For humans, the absence of light is an optical non-linear signal that indicates that it is the period when redox levels are unstressed by solar radiation. This is how the brain tells time, night from day. Melatonin is an old evolutionary clock that was important before the optical lattice clock that now exists in your visual pathways came along via evolution. Light activation can long before food activation in evolutionary timescales.

The circadian clock in the eye and tissues, however, in humans still controls glucose and insulin metabolism, as the picture above shows. It also is the reason why blue light at night is what is driving insulin resistance and the runaway diabetes we see today. The optical signal of melatonin is a key circadian timing signal that contributes to and is a part of a cascade of other responses that help initiate and maintain sleep when light is not present. Melatonin levels also control the distances between respiratory proteins in our mitochondria. If the level is low anywhere in the system % heteroplasmy rises and the ATPase of that mitochondria begins to spin slower than it should and electron chain transport slows in that tissue and damage occurs that can make that organ disease.  As a result NAD+ drop. This is a result of light not food.

Strangely enough, researchers now know that these receptors are also found in the pancreas, but clinicians have no idea this is what is behind the diabetes epidemic globally. It is related to the amount of deuterium on receptors on alpha and beta cells in the pancreas and a lack of sunlight makes it more likely that you’ll get diabetes. Melatonin receptor density is brisk in pancreatic alpha and beta cells. Melatonin controls the mtDNA in the alpha and beta cells in the pancreas. This is how diabetes manifests from getting too much artificial light at night via our eyes or skin. Most people know beta cells release insulin during the day when we are supposed to eat. Beta cells regulate glucose levels in the blood. During daylight blood pools to the surface to absorb specific frequencies of sunlight while releasing nitric oxide. Researchers also know that when melatonin activates these receptors via the blood plasma, insulin secretion is decreased from the pancreas. This mechanism shows your that diabetes is a photo-electric disease. Your government is behind the epidemic.

CITES

https://www.fortunebusinessinsights.com/industry-reports/spinal-cord-stimulation-market-100313#

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4676495/

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7814083/

https://pubmed.ncbi.nlm.nih.gov/27727191/

https://x.com/DrJackKruse/status/1842010173910708409

The future of maternal fetal medicine is understanding howthe governments MKULTRA programming has ruined the fundamental process.

Did you know the Leptin melanocortin pathway controls the entire physiology of the human placenta? This is why Leptin controls fecundity and fertility. The placenta mtDNA liberates more ultra-weak biophotons, which alter the hydrogen binding networks in the water in blood, and this liquid crystalline structure delivers its payload to the mother brain to begin translation of more POMC deposits in her brain. As water content increases in her circulatory system, so does melanin; this raises her alpha MSH level and increases melanin production in the brain. As a result of the relative rise of UV-powered water in her blood and increased melanin, her brain shrinks. This mimics the process I explained in my recent

Palestra Society talk in El Salvador last month. This phenomenon has allowed me to use cerebral brain volumes on my patients to predict transgenerational risks of the unborn child. This is how the future of maternal-fetal medicine will be practiced in decentralized medicine systems.

Nature always favors the living mavericks in her domain who challenge her rules. She will respond by fighting the maverick all the way, but she respects them and adapts to the environmental pressures the maverick applies. Ultimately, nothing else matters but the pressures placed, which cause her adaptations to occur in cells. They prove that she listens to them much more than Darwin’s ideas in centralized texts.

Morphogenesis develops during the first 5-6 months of pregnancy while the brain remains immature and devoid of development. Suddenly, the brain stops developing in six months, and the child gains more weight than at any other time of its in-utero life. How does this happen? The fetal mitochondria create light spectrum where U light is subtracted with IR light, and blue light is emitted. This fattens the child using melanopsin as its photoswitch. A LACK of endocabbinoidal function turns off the UV 380 nm mTOR photoswitch, and the IR 1280 nm photoswitch is also turned off.

This stunts brain development and fattens the child because there is no POMC translation; it results in a lack of melanin and endogenous endocannabinoid signaling. The green rectangle in the slide below shows you its dynamic effect. As leptin, adiponectin, and endocannabinoids drop, so does mTOR signaling. This means they must affect production of UV biophotons and they have to involved the mix of H+/D levels in the mitochondrial matrix.

Many forget that humans arrest neural development to be able to fit their offspring out of the vaginal canal safely. As neural development halts in humans, the child subcutaneous space needs to fattens up. That will facilitate the child’s passage through a tight canal in the mother, and the fat can be used to mature the brain when the child is born and is put in sunlight. 380 nm light not only matures the child’s brain post natally but also drives regenerative programming everywhere in humans. In fact, endogenous endocannabinoid signaling is critical in regulating decidual senescence and parturition timing.

380 nm light increase cognition in the developing brain. because it is associated with energy flow from matter to be turned on as the slide above shows. Synaptic proteins are upregulated also because of 380 nm because it increases nitric oxide (NO). People forget Nitric oxide (NO) works as a retrograde neurotransmitter in synapses. In brain it does different things than it does outside the CNS. NO allows the brain to increase blood flow to increase energy production and because of this it plays an important role in the fetal intracellular signaling in neurons to regulate the neuronal metabolic status to the dendritic spine growth. Remember as a free radical it controls the magnetochemistry of the fetal brain.

BPA increases deuterium absorption and reduces H+ flow in colony of mtDNA fetal brain. Deuterium in mtDNA is known to cause methylation defects, which causes endocrine dysfunction. Prenatal exposure to BPA at low levels can also affect gene expression in developing brain by turning off growth leading to cognitive decline. Fluoride does the same by stealing electrons that have these light frequencies in the UV range and this limits semiconduction and growth of the immature brain as it develops.

HOW DOES MOM’S PLACENTA FATTEN HER BABY PROPERLY?

Did you know the germinal matrix center in the human brain is found in the area of the brain called the thalamus? The thalmus is where all 5 of our sense target. I told you in the Quantum engineering series that all life has a frequency modulated antenna system built into the thalamus to tell time. Specifically it relates to telling seasonal time. It operates by varying the amount of deuterium in this part of the brain to alter its stiffness. Why?

This systemis built to vary its”flexibility” when it is encumbered by too much mass in the antennae of the system. This is where the Kruse for Dummies lecture comes to into play to explain to you how ATAVISM happens in utero to stop brain growth. It is instructive of why obesity in the adult form is a predictor for nnEMF risk. Humans are designed to fatten when they add deuterium to their thalamus to affect of the organism accounts for timing.

What bends space/time in the universe? The masses associated with the matter in the universe do. As result, physics teaches us that magnetic fields tend to flatten and stiffen the fabric of space-time when masses are added to them and this alters our perception of timing. So adding deuterium to the mitochondria of the developing thalamus stops neurogenesis in utero. The added deuterium alters our perception of the seasons, and this fattens the child in the third trimester. The process also halts melanin by turning off UV biophoton production in the placenta. This limits the neuroplasticity in the neural crest cells in different parts of the CNS of the fetus. If you remember from Becker’s work he said he found that the semiconductive pathway in neurons that controlled regeneration seems to come from just below the myelin level. This area is where POMC is normally found in humans and when UV biophotons are present would cause POMC translation to make melanin to charge separate water to make a ton of electrons that would signal a semiconductive circuit. When you turn off UV biophotons creation from mtDNA in the placenta and in the germinal matrix center you affectively stop brain growth.

Anything that has more mass than the smallest atom used inside of our molecular clocks changes how we experience time. This is AXIOMATIC. Mass bends space/time! As a result, we should expect the Fo head of the ATP to stop spinning as much. In fact, ATP production should slow down. How does this happen in humans. The mother’s placenta make a huge amount of NO to inhibit the fetal ATPase. This means that the magnetic fields created inside the childs developing neurons is effectively arrested. Anytime humans are using the TCA cycle, they are creating the largest magnetic fields in mtDNA.

Now ask yourself what magnetic fields do to space-time based on this picture below. It orders atoms around it to allow them to direct energy flow to create life. This process slows light down in tissues and causes time perception to change. The collapse of the magnetic field by the mother’s placenta halts this process. Centralized OB/GYN has never understood why the human placenta normally enhances physiological ROS production during pregnancy. Now you know why. It is not a bug in the design, it is a design feature to stop the childs brain growth.

What does this concept imply? Time slows as one approaches the speed of light. At the speed of light, time ceases to change because it contains all change.

A mother that does not get enough sun on her abdomen during pregnancy runs the risk of developing preeclampsia (PE). PE pathophysiology result from abnormal placentation due to a defective trophoblastic invasion and an impaired remodeling of uterine spiral arteries, leading to a poor adaptation of utero-placental circulation. Leptin and melanin control this process. This can alter how the brain develops in her child. This is really how autism begins in humans. It is also how many childhood cancers begin and I believe it is where Angelman’s Sundrome really comes from. Altered circadian biology of the mother’s placenta combined with a altered germline in eggs and sperm.

Note on the slide above that matenral levels of Vitamin D links to NO production of the placenta. This is why maternal Vitamin D levels are a huge predictor of problems in decentralized medicine. Mother’s have to use their stored light to turn off the magnetic fields in their child. People have totally forgotten the basics. Nitric Oxide turns off CCO and ATP production as the slide above shows. NO enhances blood flow through the placenta but it turns off energy flow to control the child’s brain growth. Fetal RBC contain mitochondria to make them less able to tell time so the child does not get involved with the mother’s placental signal to light. So having a mitochondria in your RBCs means you are less of a sensor to the SCN in the retinohypothalmic tract. This means the placenta of man controls the timing mechanisms in the baby when it makes sense to turn off brain development.

It implies that the your skin and eye take over this process when you leave the uterus as well.

WHERE DOES THE METHYLATION PROBLEMS COME FROM?

Tetrahydrobiopterin is a cofactor of the three aromatic amino acid hydroxylase enzymes that I have showed you hundred fo times in the slide below. THB is also used in the degradation of amino acid phenylalanine and in the biosynthesis of the neurotransmitters serotonin (5-hydroxytryptamine, 5-HT), melatonin, dopamine, norepinephrine (noradrenaline), epinephrine (adrenaline), and is a cofactor for the production of nitric oxide (NO) by the nitric oxide synthases.

I just told you the placenta controls NO production, didn’t I. Have you forgotten how NO controls methylation production? An altered antioxidant capacity from the mother’s circadian defects alteres her NO bioavailability in her placenta. Without NO can you collapse the ATPAse? If you cannot do this, can you control morphogenesis or organogenesis in the fetus? NOPE.

Altered NO levels leads to methylation problems in the nucleus. Centralized science has forgotten the basics. Why is this true? Altered methyaltion results in part from the reaction of NO with the radical anion superoxide (O2•−), which produces peroxynitrite ONOO-,. This is a powerful pro-oxidant and inflammatory agent. Another mechanism is the progressive inhibition of the placental endothelial nitric oxide synthase (eNOS) by oxidative stress. DIdn’t I tell you over a decade ago in the Holy Trinity blog that eNOS is a key circadian switch in the SCN of all mammals? I did. What happens when you affect it? An altered eNOS results uncoupling viaseveral events such as a depletion of the eNOS substrate L-arginine due to increased arginase activity, an oxidation of the eNOS cofactor tetrahydrobiopterin (BH4). It also implies an alteration in eNOS post-translational modifications (for instance by S-glutathionylation). This is how childhood disease manifest in utero. This is how a child is born with a large amount of heteroplasmy when the system signals are not well controlled by sunlight.

More heteroplasmy = higher disease burden in a fetus even before it has taken a breath.

How big a miss is this decentralized science for every centralized OB/GYN?

Did you know that tetrahydrobiopterin is a cofactor for tryptophan hydroxylase (TPH) for the conversion of L-tryptophan (TRP) to 5-hydroxytryptophan (5-HTP)? Tyrosine hydroxylase (TH) catalyses the conversion of L-tyrosine to L-DOPA (DOPA), which is the precursor for dopamine. You do remember that dopamine and melantonin control ALL RENOVATIONS OF PHOTORECEPTORS RIGHT? How many times have you seen this slide as proof I have been telling you this?

Now what are all those photoreceptors I keep mentioning? Remember how many times you have seen this slide in blogs too. The same message has been being pound into your blue light toxic brains for decades.

The control of THB and NO by the placenta turns out to be critical in melanin production as the slide below shows. This is the real reason why most centralized OB/GYNs have to give women prenatal vitamins with folic acid. WHY?

BH4 can be oxidized by one or two electron reactions, to generate BH4 or BH3 radical and BH2, respectively. Research shows that ascorbic acid (also known as vitamin C) can reduce BH3 radical into BH4 preventing the BH3 radical from reacting with other free radicals like superoxide  and peroxynitrite specifically. Without this recycling process, uncoupling of the endothelial nitric oxide synthase (eNOS) enzyme and reduced bioavailability of the vasodilatornitric oxide occur, creating a form of endothelial dysfunction. This is the only thing centralized OB/GYNs got right. They realized that a lack of folic acid was linked to caudal regression syndromes in fetal medicine. The problem is they still have no idea it links to just about every other disease children get from solar deficit mothers. Ascorbic acid is oxidized to dehydroascorbic acid during this process, although it can be recycled back to ascorbic acid to be reused. Look at the slide above. You’ll see how the placenta turns off the recycling of Vitamin C and now you’ll see why. Glutamate release without Vitamin C turns off neurulation in humans.

Folic acid and its metabolites seem to be particularly important in the recycling of BH4 and NOS coupling. This is why this series has a blog on this. When mothers’ are solar deficient they will need folic acid to prevent many diseases in maternal fetal medicine.

I know you’ve seen this slide below a lot too., but I need to you to connect the lessons I am stacking for you now because centralized medicine is being sculpted by BigHarma to make sure you do not get this lesson via how the curriculums in med school have been hijacked by data that came from MKUTLRA. Blocking this pathway on the top line of the slide below stops brain growth in the fetus!!! It also stops T3 production which is needed for neuron growth and sprouting in humans.

What else is caused by this placental light dysfunction story I am weaving for you?

Phenylalanine hydroxylase (PAH) catalyses the conversion of L-phenylalanine(PHE) to L-tyrosine (TYR). Therefore, a deficiency in tetrahydrobiopterin can cause a toxic buildup of L-phenylalanine, which manifests as the severe neurological issues seen in phenylketonuria. So PKU is a risk factor for women who are solar deficient. I bet your doctor never told you that!

HOW WE CREATE DIABETICS?

The role of BH4 in this enzymatic process is so critical in arresting fetal brain growth that most have forgotten that a deficiency of BH4 in the placenta means – WE SHOULD expect a reduction of nitric oxide production. This is the photoswitch that controls this process. Without BH4 you cannot make NO at all. Recall that Nitric oxide synthase (NOS) catalyses the conversion of a guanidino nitrogen of L-arginine (L-Arg) to nitric oxide (NO).

Why does maternal fetal NO causes gestational diabetes? It should be obvious now. Moreover, it should be obvious why gestational diabetes means the mother and baby are going to be future diabetics. Why do I say this? 30 years of maternal fetal medicine research has pointed to a deficiency of BH4 – and thus, of nitric oxide – as being a core cause of the neurovascular dysfunction that is the hallmark of circulation-related diseases such as diabetes. Anyone who is solar deficient and gets more ALAN is guarranteed to get diabetes because of a chronic lack of NO production. Moreover, why do diabetics get more cancer? NO controls the stem cell depot biology. And a lack of BH4 also causes DNA methyaltion defects which make oncogenesis more likely. EVERYTHING FITS DECENTRALIZED medicine theory here, doesn’t it?

IF YOU BLOCK THE SUN YOU WILL GET CHRONIC DISEASES IN EPIDEMIC FASHION.

I hope youre beginning to understand how atavistic effects predict the future of mankind now as they built a tech world with MKULTA light.

Your mother’s placenta uses light message from her mtDNA to create a defect in the antenna buried in your developing thalamus where all human neurogensis occurs. This optical lattice clock switch in her placental ruins the fidelity of the system in the fetal brain by design so it can no longer tell where the Earth and sun are in relation to one another. It destroys the ultradian ryhythms in the child by design.

FOOD mom eats has zero to do with this mechanism. Her SCN sends this signal to her mtDNA in her placenta. Her SCN is an optical lattice clock; it is not a food clock. . It is a pretty remarkable gadget Nature built in our heads and our placenta.

It appears the simple addition of a neutron to H+ to the antenna system in the fetal thalmus is enought to screw up the connection of the thalmus to the heartbeat of the Earth. Without a proper connection to Earth, the fidelity of the music coming from the system fails. What does this FM station listen to? The Schumann resonace of the Earth. It is 7.83 Hz. That is the human alpha wave which is created in the thalamus. That is what causes all chronic disease in humans. It turns out adding neutrons to H+ is enough to do something to the fabric of space-time in the fetal brain that deforms the magnetic fields in the child’s ATPase to allow a child to emerge from a small pelvic outlet. This shows you how the smallest things make the biggest deal in you. Wild decentralized FACTS, that have been blocked in centralized science, I tell you. Sadly this science was shared with the corporations that were involved with the Industrial military complex in the 1970’s and 1980s.

THE PHYSICS WAS WORKED OUT FOR ME IN THE BASEMENT OF CHARITY HOSPITAL IN 1989.

The spacetime curvature for a charged static spherical body is given by the Reissner–Nordström metric. I’d suggest you carefully look at the next slide many many times. It shows you what Einstein’s special relativity really shows. This was critical for the development of technology to operate wirelessly using satellites to control them above Earth. This is why the GPA in you iphone actually works like your SCN does.

The picture above shows you time slows as one approachs the speed of light. The present moment your living right now reading this expands from a narrow sliver until it encompasses both the past and the future. This means that the idea of past and future is an optical illusion caused by the slowing of light by matter. It also means as light slows you get the idea there is a cause and effect on Earth but there is no real relationship. This is why BigHarma continues to use the idea that RCT is the gold standard for centralized science. The physics of special relativity tell you this is a bullshit story from the jump.

When you use these mathematics, you can feed in the value of whatever charge you want and calculate the time dilation as a function of distance from the charged body. If you do this you’ll discover something rather odd in Einstein’s field equations, namely, the charge reverses the effect of the mass in them.

 

This is why REDOX POTENTIAL is the key to chronic disease reversal and nothing else matters.

READ THAT LAST SENTENCE AGAIN.

 

This message was given to the Industrial military complex Big Pharma partners. They were told to add atoms to their drugs to limit the effect of charge. This helps the military use light to control people by using screen technology. It stole their dopamine reward tracts. I mentioned this pathway above. REVIEW IT.

That is what the target of MKULTRA was focused on after all the experiments where completed from 1953-1964. Those experiments continued until the early 1980’s by the CIA In Reagan’s admiistration. It was given to researchers who linked colleges via the ARPANET. This data was them shared by the Industrial military Microsoft’s Bill Gates and Apple’s Steve Jobs by DARPA in the 1980s. This was done by DARPA but pre- GOOGLE. . All that data was sitting in the basement of Charity Hospital where I did my residency. It all came from Tulane neurology and neurosugery experiments done for MKUTRA and by Sidney Gotlieb. I read them all before it was all destroyed by Huricane Katrina. The basement flooded and almost 160 years of medicine was lost in New Orleans.

How do cells increase their charge?

LIGHT DOES IT. Food has no effect on this.

Read it again.

LOOK MORE CAREFULLY AT THE SLIDE ABOVE AGAIN. It is why I posted it again. Do you remember how many times I have told you in podcasts that redox potential is formed by adding net negative charges in a cell? Melanin is the key way all mammals do it. Human biology relies heavily on this. Do you see now by examining the slide above closely how that charge in light frequencies can offset the bad isotopic variation in some of your tissues to make your clocks work better in the circuit that tells the brain what season it is? This is another universal law not subject to an RCT. BigHarma knows what your doctors do not. And they are not going to tell them either. It is encumbent upon you in getting this lesson and why it is being blocked from centralized MDs. It should explain to you why the Universities in ARPA founded HMOs and changes the way healthcare was delivered in the 1980s to put an Oracle between you and your doctor. That Oracle was a healthcare industrial complex player. The system was engineered to put a hospital CEO and an insurance executive decision tree between you and your doctor before ANYtreatment plan can be APPROVED by the governments agents in this plan. HMOs and the insurance industry were designed to limit the time a doctor can spend with you so you only have time to get a presciption that comes from BigHarma that changes the charge in you so you are more likely to follow the rules built by government. I will remind you that Medicare began post MKULTRA in 1968 under LBJ post JFKs assassination.

HOW WAS MKULTRA, LBJ, THE CIA, JFK, AND ISRAEL ALL LINKED TO THIS STORY?

I laid most of it out in the recent Danny Jones podcast but i saved the critical details part for my members so you can understand how it links to the creation of all modern disease epidemics.

THE MOST EXPLOSIVE SUMMARY

On July 30, 1965, President Lyndon B. Johnson signed into law legislation that established the Medicare and Medicaid programs. This created CMS for the industrial healthcare complex. It established Medicare, a health insurance program for the elderly who could and would be taken out, and Medicaid, a health insurance program for people with limited income to make sure they remained poor and sick to be controlled by the government for decades to come. LBJ signed this law in Indepence, Missouri. Why? This is where Manchurian Candidate Harry S. Truman was from.

This completed the medical tyranny portion of the New Deal Era that began under FDR and was linked to the the Manhattan Project story of how DARPA was created by General Groves. How long ago did the Industrial military complex decide to use medical tyranny as the attack vector of the US Constiution? 1911.

This was when the Flexnor report was launched to create BigHarma from Rockefeller’s break up of Standard Oil by Teddy Roosevelt. I had a lot to say about this in the Danny Jones podcast, but I will have a lot more to say about it in future podcasts once this information in that podcast is married in this summary and understood by the public.

Today, you are being highly preconditioned for compliance by the real findings that came from MKULTRA.

At the same time that CMS was created the ideas to taper the Ponzi scheme of money laundering from the public, the USA Executive Branch and the CIA helped pay off the MOSSAD for the JFK assassination by giving Isreal close to 300 pounds of uranium it needed to become a nuclear power. JFK, prior to his death, was going to send in the Atomic and nuclear regulators of the USA in to make sure Isreal never got an atomic weapon. This infuriated Israeli prime minister David Ben Gurion.

CAREFULLY EXAMINE THE PICTURE ABOVE AND NOTE THE DATE IT WAS TAKEN.

How did this story link JFK to Israel and the MOSSAD?

It began with an intelligence failure of the CIA and John Kerry’s father.

Documents published in Wilson Center from the National Archives now have shed light on a particularly notable intelligence failure: how Washington missed warning signs that the Israelis had a nuclear project underway, but also how the U.S. belatedly realized what the Israelis were doing, and how Eisenhower and his senior advisers reacted to this discovery. Among the new documents are:

• The June 1959 Israel-Norway secret agreement providing for the sale of Norwegian heavy water to Israel (through the United Kingdom), transmitted by Oslo Embassy political officer Richard Kerry (father of former Secretary of State John Kerry).
• Reports about information from a then-covert source — University of Michigan nuclear engineering professor Henry Gomberg — who learned that the Israelis had a secret nuclear reactor project that involved experiments with plutonium.
• A telegram from the U.S. embassy in Tel Aviv reporting on Finance Ministry official Addy Cohen’s statement that “we’ve been misbehaving,” and one by an unidentified official close to Prime Minister David Ben-Gurion that the secrecy surrounding Dimona was unjustifiable and that it was “a stupid mistake on the part of Israel.”
• Reports by U.S. Ambassador Ogden Reid on conversations with Ben-Gurion.
• A State Department message to the embassy in Tel Aviv conveying irritation that the responses of the Israeli government showed a “lack of candor.”
• Messages about a role for the International Atomic Energy Agency in inspecting and safeguarding Dimona. When Eisenhower left office Kennedy assumed this mess but he knew Ben Gurion for over a decade as the picture above shows. Now President Kennedy pressured the government of Prime Minister David Ben-Gurion to prevent a military nuclear program, particularly after stage-managed tours of the Dimona facility for U.S. government scientists in 1961 and 1962 raised suspicions within U.S. intelligence that Israel might be concealing its underlying nuclear aims.  This went on during the Cuban Missle crisis and Kennedy was worried about nuclear proliferation post WW2. Kennedy’s long-run objective, documents now show, was to broaden and institutionalize inspections of Dimona by the International Atomic Energy Agency.

Of all U.S. leaders in the nuclear age, John F. Kennedy was the nonproliferation president. Nuclear proliferation was his “private nightmare,” as Glenn Seaborg, his Atomic Energy Commission chairman, noted many times in interviews. Seaborg discovered Plutonium for the Industrial military complex leaders.

On 30 May 1961, Kennedy met Ben-Gurion in Manhattan to discuss the bilateral relationship and Middle East issues. However, a central (and indeed the first) issue in their meeting was the Israeli nuclear program, about which President Kennedy was most concerned.   According to a draft record of their discussion in the National Archives, which has never been cited, but has now been released for the first time by the Wilson Center, Ben-Gurion spoke “rapidly and in a low voice” and “some words were missed.”  He emphasized the peaceful, economic development-oriented nature of the Israeli nuclear project.

Nevertheless the note taker, Assistant Secretary of State Philips Talbot, believed that he heard Ben-Gurion mention a “pilot” plant to process plutonium for “atomic power” and also say that “there is no intention to develop weapons capacity now.” This information stunned JFK. Ben-Gurion tacitly acknowledged that the Dimona reactor had a military potential.  The final U.S. version of the memcon retained the sentence about plutonium but did not include the language about a “pilot” plant and  “weapons capacity.” Anyone who understands basic nuclear physics (see my Patreon Thorium blog on BTC) knows the only reason to use plutonium as a fuel is because you can make military grade uranium for nuclear weapons.

The differences between the two versions suggest the difficulty of preparing accurate records of meetings. But whatever Ben-Gurion actually said, President Kennedy was never wholly satisfied with the insistence that Dimona was strictly a peaceful project. This really was the main precursor to his death in my estimation. Neither were U.S. intelligence professionals. A recently declassified National Intelligence Estimate on Israel prepared several months after the meeting concluded that “Israel may have decided to undertake a nuclear weapons program. That CIA information was leaked to the mob in 1963 after the Bay of Pigs invasion went south. This information was critical to Santo Trafacante because it allowed him ot gain leverage over Castro and the attempts of the Industrial military complex to kill him versus JFK. Trafacante ensured it was JFK that would pay the ultimate price. He viewed his appointment of RFK Sr as Attorney General as the ultimate act of betrayal to the mob bosses in Chicago, New York, Florida, and New Orleans. In fact, Carlos Marcello was on trial as JFK was shot in Dallas on November 22, 1963 because of an RFK Sr prosecution that Joe Kennedy swore would never happen if the mob swung the swing state of Illinois to JFK in 1960. These details matter big time to understand how things really unfolded.

ISRAEL had massive amounts of agents in the USA in the Jewish mob before Israel was even made a country in 1948. Prior to being a country they had a massive intelligence capability world wide that linked banking, science, and gangsters who were all Jewish.

This was the main reason the Jewish gangsters were eventually made MOSSAD agents after 1948. They fed information to Israel that the Chicago and NY families got on Joe Kennedy and his sons. As a result, Meyer Lansky, Jack Ruby Rubenstein, Carlos Marcello, and Santo Trafacante became involved with the assassination of JFK , along with Nixon, the Bush and Hunt Oil families and Clint Murchison linked to Groves Industrial military complex. Texas was also a swing state and the Oilmen were pissed that Kennedy was catering to Blacks in civil rights and was soft on the Communists in Russia and Cuba.

Santo Trafacante was among the most powerful Mafia bosses in the United States when JFK was a Senator in the 1950s. He headed the Trafficante crime family from 1954 to 1987 and controlled organized criminal operations in Florida and Cuba. He was also a MOSSAD asset for reasons I laid out in the Danny Jones Podcast. He was a double agent for the CIA and MOSSAD in the 1960s due to drug laundering to replace what the Chicago and Florida families lost in the casinos due to the Cuban Revolution of 1959.

Castro enlisted Santo to pay back the CIA for trying to kill him 9 times by joining forces with Traficante to turn Americans into drug zombies. Trafacante was the key link the Italian mob to the Jewish in the USA. This is how the MOSSAD got the information that JFK life was at risk. He also was in charge of monitoring the behavior of Judyth Vary Baker under Dr. Alton Ochsner care on Magazine St in New Orleans. He also linked the Chicago mob the NYC mob of Bonanno crime family. This family is known as the American mafia. They are constructed of 5 families all based in New York. These Families had connections with FDR Jr son as a Senator from NY.

Carlos Marcello was a gangster who was based in New Orleans but tightly linked with Sam Giancana of Chicago. Both worked with Trafacante in Florida and Chicago. He knew Lee Harvey Oswald since Oswald grew up in New Orleans. In 2022 more government documents were released which showed us how US law enforcement was linked to the Italian mob. At least nine files released refer to Chicago circa 1963 and several individuals linked to the Outfit.

Most notably among them are former Chicago police officer and Cook County Deputy Sheriff Richard Cain (see above). Cain had deep connections to Chicago organized crime according to investigators, especially ruthless mob boss Sam “Momo” Giancana. Joe Kennedy, father or JFK, asked for Sam’s help in getting Kennedy elected in 1960 through his connections with labor unions linked to Jimmy Hoffa. Illinois was a He was promised under JFK presidency there would be a laissez faire action plan towards organized cirme. This is why Sam helped Kennedy’s father. It is clear today that JFK or RFK was not given this information. It is also clear that RFK Jr knows this information today.

In 1965, the MOSSAD was paid off when Isreal got uranium from LBJ and CIA as payment for their assistance. Did Israel steal bomb-grade uranium from the United States or were they given it by the USA?

After a 1965 inventory, NUMEC was found to be missing about 100 kilograms of bomb-grade uranium, even after accounting for all processing losses. The close personal and commercial ties to Israel of the plant owners and operators raised suspicions that remained unresolved. The affair of the missing bomb-grade uranium was revived in 1976. The newly formed NRC in 1976 UNDER fellow Warren Commission member Gerald Ford who replaced Nixon in 1974.

The NRC was in the process of writing licensing regulations for commercial fuel firms—of which NUMEC was one—and had heard rumors of possible theft in the 1960s from NUMEC’s Apollo facility. NUMEC = Nuclear Materials and Equipment Corporation. NUMEC is based in Pennsylvania. There is a reason this plant was targeted by LBJ, CIA, and Israel. The partners in the JFK crime syndicate were in control of this location. Arlen Specter the lawyer who came up with the one bullet theory in the Warren Commission was given the Senator postion by the Industrial military complex by using election stealing capabilities in the 1960s. The same ones were used to give the election of JFK over Nixon in Illinois and Texas in the 1960 election.

The NRC asked for a CIA briefing. This was by design because President Ford knew the CIA was involved in the theft to pay Israel off. Duckett startled the NRC group with CIA’s conclusion that the missing uranium was in Israeli bombs. The NRC chairman informed the White House, and President Ford took an iimmediate nterest in the case because Duckett was not supposed to tell the truth. He was not involved in the JFK assassination.

Ford’s Attorney General, Edward Levi, discovered that the Atomic Energy Commission (AEC), the NRC predecessor nuclear licensing agency, had previously convinced the FBI not to open a criminal investigation into the material’s disappearance. Why would he do this? Levi was the first Jewish Attorney General of the United States. He was also a MOSSAD asset.

Levi was a Yale Graduate with ties to Israel. In fact, he was a Sterling Fellow at Yale and that dates his affliation with Israel all the way back to 1864 and B’nai Birth. B’nai Birth is the previous group now called AIPAC. Before AIPAC they were the ADL. What is the history of B’nai Birth? This group was loyal to the Royal Family before the Civil War in the USA. As a Yale graduate he was linked to the Bush oil family who was an early partner of the Industrial military complex General Groves put together from the ONI and OSS before the CIA was formed. Preston Bush, the father of George H Bush, was a banker who worked Brown Harriman in NYC who worked with the families who controlled money in the US Federal Reserve. This was established in 1911.

During Levi’s term as Attorney General, he issued a set of guidelines in 1976 to limit the activities of the FBI. This was done in repsonse to Nixon being forced to resign over wiretapping during Watergate. The CIA was furious that the FBI turned on Nixon and this was one way the DOJ in the executive branch was used via lawfare. These Levi guidelines required the FBI to show evidence of a crime before using secret police techniques like wiretaps or entering someone’s home without warning.

How connected to the criminal cabal was Levi? His guidelines were QUICKLY replaced by new ones issued in 1983 by Ronald Reagan’s Attorney General, William French Smith. Levi urged President Ford to appoint Robert Bork, who was his former student and Solicitor General to the SCOTUS. The real reason Bork was not elevated to the SCOTUS is because many democrats knew that Levi and Bork were linked to the conspiracy that assassinated JFK. That did not stop Levi. He told Ford if Bork is blocked he should nominate fellow Chicagoan John Paul Stevens to the United States Supreme Court, and Ford followed his advice. Did you know that Levi later testified in support of Bork at his confirmation hearing for the SCOTUS? Did you know who else was linked to Levi and Israel?

Serving under Levi at various times in his career, in various high staff positions, were such people as Rudolph Giuliani, Robert Bork, Antonin Scalia, Rex E. Lee, and Arthur Raymond Randolph. That is how close the ties with Israel and the MOSSAD are to the USA. It is also why I told Danny Jones why Donald Trump made the JFK achives public. As president, he found how his friends in banking, Wall Street, and real estate were to the JFK assassination attempt. They are the people behind the warning shots being made right now to DJT. I believe if they wanted him dead, he’d already be dead. I think they think they can control Trump if they make it clear to him during this election cycle that he needs to follow the playbook Levi built out in the 1970s. Joinign forces with RFK Jr is why they went after him at Butler in Penssylvania. It should not surpise anyone of you now why it happened in Pennsylvania. The industrial military control over this state and it politicians is close to air tight.

HOW TIGHT IS PENNSYLVANIA in this story?

I told you that NUMEC is in Pennsylavania. Did you know that Zalman Shapiro the owner of NUMEC attorney was Arlen Specter, the Senator for Pennsynvania? Amazing coincidence or was the something more important to this story the USA government wanted buried forever?

So the chain of evidence went from Ford to Bush to Carter to the CIA or did it?

Carter instructed his national security advisor, Zbigniew Brzezinski, the father of modern day broadcaster Mike on MSNBC, to deal with the NUMEC matter in the context of the impending public release of MUF data. Brzezinski’s staffers John Marcum and Jessica Tuchman posed questions to the CIA about the NUMEC affair. This is when key data magically went missing to President Carter.

WHAT WENT MISSING?

Carter was never told about the links of Israel to NUMEC.

Brzezinski shared the information with 2 staffers but not Carter. The two staffers told Brzezinski that environmental samples taken by the CIA in Israel in 1968 contained highly enriched uranium, whose enrichment level was so high it pointed to the Portsmouth, Ohio, uranium enrichment plant as the source. Portsmouth was where NUMEC obtained uranium stock for its naval fuel products. Why he never told Carter is likely tied to his ideology as Polish and hating Russia and this marries to General Groves ideology linked to Stalin.

During the Apollo affari of 1965 the president of NUMEC, Zalman Shapiro, had frequent contacts with Israeli officials, including a science attaché “thought to be an intelligence officer,” and received unexplained VIP treatment in Israel. None of this information went from Brzezinski to Carter.

In fact, Shapiro was by then known to have had contacts with Israel’s head of military intelligence and the head of its nuclear weapons program. He later acknowledged knowing Binyamin Blumberg, head of Israel’s “bureau of scientific liaison,” which engaged in high-risk intelligence capers. On one strange occasion in 1968, Shapiro hosted an Israeli intelligence foursome at the Apollo plant. One was the Mossad agent who headed the team that spirited former Nazi leader Adolf Eichmann out of Argentina and who later ran Jonathan Pollard’s spying on the United States for Israel.

For those of you who do not know, the Mossad is the Israeli agency that handles foreign intelligence collection and covert action. It is Israel version of the CIA. Another was that agent’s deputy in the Eichmann kidnap, who went on to become head of Shin Bet, Israel’s internal security service. A third was Mossad’s director of technical services. The last was Israel’s science attaché, who had held a senior position in Israel’s nuclear weapons program. More amazing coincidences in this story do not you think?

How did I know all this history and link it as a young man before I became a doctor in New Orleans? The man who i have spoken about in many podcasts who taught me everything I know about fiat banking and fractional reserve banking was named Theodore Sterling of the Sterling National Bank on 57th Street and Madison Ave as I grew up in NYC. He told me this history when he told me how banking really operates in the USA. Read the hyperlink on him you just passed before going further.

What happens when Ford leaves office? How did the story remains toghtley controlled by the industrial military complex?

Everyone involved was linked to the Warren Commission.

After the 1976 election, the Ford White House alerted the incoming Carter administration to the NUMEC affair. In December 1976, according to a July 1977 National Security Council memorandum, then-CIA Director George H.W. Bush briefed President-elect Carter on the case. Congress had been pressing for public disclosure of records of large unexplained losses of bomb-grade material (in government parlance, “material unaccounted for,”or MUF) from the government’s nuclear weapons complex. The Carter White House feared the story was sure to hit the headlines if there were any suggestion of Israeli theft from the NUMEC facility.

And any disclosures about Israel’s bomb program would of course have threatened the Carter administration’s Middle East policies between Israel and Egypt. If you really want to know the truth, I have always believed Israel agreed to this peace deal reluctantly to bury the Apollo affair once and for all. But that affair is the gift that keeps giving. It’s secrecy is linked to the real story of how the MOSSAD was involved in 9/11 Tower knockdown fiasco during the George W. Bush presidency. See how the story all begins to make sense when you really see it laid out as it happened. Today’s history books will never let you see how it is all linked.

MOST OF YOU HAVE NO IDEA HOW NEFARIOUS OR HOW OLD THIS PLAN IS IN THE HISTORY OF THE USA.

CITES

1. Danny Jones podcast

2. https://thebulletin.org/2014/04/did-israel-steal-bomb-grade-uranium-from-the-united-states/

3. https://abc7chicago.com/jfk-files-documents-national-archives-chicago-mob/12576586/

4. https://en.wikipedia.org/wiki/Apollo_affair

https://www.facebook.com/watch/?v=10153124017336716

You’re going to be retooled with knowledge and reschooled with Nature’s wisdom today. Share this decentralized wisdom with the world.

Do we need to know our “WHY“?

I think we do.

Without knowing our why I believe the attainment of Optimal will not happen.

I was getting ready for a medical talk earlier this year and I was jotting some things down about how my surgical judgement has changed from the time I left residency to the present and going over my practice patterns for the talk. It dawned on me the practice of medicine is very much like the practice we use in life to become a more imporved version of ourself. Inevitably, practice isn’t the thing you do once you’re good. It’s the thing you do that makes you good. I was surprised about the crowd of people I was asked to speak to. The mayor was in the audience and so was some the state health department officials. I had no idea who i was speaking too. I thought it was going to be mostly patients and PCP’s in my new referal base for my new job. When I saw that my audience was quite different than I anticipated I scrapped my “neurosurgery talk plan” on minimally invasive spine surgery and decided to talk about how my “what”, neurosurgery, led me into my “why”, my real passion………Quantum biology.

The response I got from yesterday’s talk even astounded me. I think I realized something about myself yesterday. When I speak from the heart I connect people with nature. That connection is the key to getting my flock well. This is the basis of becoming a mitochondriac. I realized this week that I help people find their “why” in healthcare via biophysics so their wellness manifests. Do you need some clarity on how to move your “what’s”, to the key “why’s” that will eventually move your decision making process to focus on only the things that matter in your life to keep you well? Then reach out to me and join my flock of mitochondriacs right now.

SO GIVE ME SOME EXAMPLES DOC

“The antagonistic pleiotropy (AP) theory posits that aging occurs because alleles that are detrimental in older organisms are beneficial to growth early in life and thus are maintained in populations.

Although genes of the insulin signaling pathway likely participate in AP, the insulin‐regulated cellular correlates of AP have not been identified.

Answer: young humans before full myelination in brain needs deuterium to complete growth but once myelination is complete then mitochondria need to deuterium deplete to control growth and metabolism pathways into adult life. This is the control system for the insulin signaling systems built into the placenta. The proxy for the system is salinity measures. The more salinity in the system the more UV light can be captured in the system to drive morphogenesis.

As deuterium fractions in tissues rise as we age, salinity measures lower. This means we need more sunlight to offset the risks of aging. This is why those with insulin resistance have high BUN/creat ratios and why they are more apt to have SIADH, HTN.

Sodium transporters brings Vitamin C into the brain for TCA cycle protons recycling to lower the KIE of deuterium in TCA intermediates as we age to control volume changes in the matrix. This is why SIADH is associated with cognitive decline in TBI and aging.

MORE DECENTRALIZED LESSONS?

Na also brings calcium into bone in cases of osteopenia. Low Na = low Ca and Mg = mitochondrial dysfunction. Low salinity in CSF and the body is a feature of all nnEMF exposure in environments.

The higher nnEMF in your environment the lower salinity becomes in blood and CSF and lower salinity = sleep disorders. Most people with high insulin levels due to artificial light exposure also are afflicted with sleep apnea due to deuterium rise cause by the altered Vitamin A signaling. The slide below makes this case.

BUN rises as salinity decreases because deuterium fractions are increasing. Salt from our eccrine sweat glands helps cool our body because it augments skin vasodilation ( like UVA light does).

As man made blue light exposure of skin rises, dopamine creation in the eye and brain drop and our salinity in CSF drops and obesity manifests easier due to deuterium content in the choroid of the eye and the skin and subcutaneous fat mass due to melanopsin.

The TRP receptors of melanopsin are affected negatively by deuteration by altered salinity.

What are TRP receptors?

Transient receptor potential channels, were first discovered in 1969. They are multimodal ion channels that act as sensors of photic, chemically, or physical toxic stimuli. These channels are widely distributed in various tissues and play a variety of roles. Centralized science is still yet to realize how these channels work with light. Blu elight varies with the time of the day and this system measures these changes accurately to tell time at the quantum scale.

It all makes sense when you understand bio-physics of the kinetic isotope effect (KIE) of deuterium in the mammalian system. Centralized MDs have no clue about this work. Most have no clue why PUFA’s in fake fats are really bad: they increase proton leakage in mitochondrial matrix and allows more deuterium in matrix than should be there and this raised heteroplasmy rates to cause disease.

SUMMARY

How do we link the lessons I have taught you over the years to light?

CLOCK and BMAL1 are positive regulators of circadian gene expression in humans, and PER and CRY are the negative regulators that operate under day and night cycles.

Blue light and nnEMF liberate Vitamin A and when it is free in the system it sets off a chain of events. None fo them are good for the non visual photo receptors. For examples, it becomes an aldehyde that destroys the small molecule modulators of the mammalian circadian mechanism shown in the picture above. Once the molecular clock goes awry in a tissue with poor light signaling the implications for many neolithic diseases spiral out of control. What are some of the Vitamin A proteins involved in this downward spiral?

They are called retinoic acid receptor-related orphan nuclear receptors or RORs for short. The RORs have several isoforms too called RORα-γ. These proteins are also under the transcriptional control of CLOCK/BMAL1 heterodimers. This is bad news for the circadian control of PER.

SIRT1 regulates the activity of BMAL1 and CLOCK, two circadian transcription factors, which target NAMPT, an enzyme that synthesizes NAD+ in cytochrome 1. And in a curious feed-forward mechanism, CLOCK and BMAL1 enhance SIRT1 expression… genetic deletion of any of these players induces insulin resistance in humans. This is how chronic blue light exposure causes diabetes. Food does not cause it. LIGHT DOES. Food exacerbates the situation only because carbohydrates make 50% less water at CCO compared to fats.

Sunlight is a NATURAL calcium channel blocker that alters the firing rate of voltage-gated channels on cells. SUNLIGHT in the AM increases the amount of PER protein that is made in the cytoplasm to get to a critical concentration where it enters the nucleus. This alters the free radical signal in mitochondria. Recall all free radicals have one unpaired electron and this makes them magnetochemicals that contol timing decisions in the cell. They act as a logic gate to chose the proper metabolic pathway to use.

This is done in a few ways with sunlight. AM sunlight increases PER, and later in the AM UVA light shows up = nitric oxide (NO) release = lowers BP and controls stem cell depots and the blue light in sun balanced by red light also stimulates melanopsin relaxation of the arterioles in our skin as another collateral effect. This one reason why just going outside in the sun and clean air reduces depression and anxiety because sunlight slows breathing and improves oxygenation. When PER is not made by sunlight depression and suicide are more predictible. Indoor living and ALAN lead to depression and suicide because PER is not made by AM sunlight. the picture shows you the effect in the SCN. It is devasting to the signaling in the leptin melanocortin semiconductive pathway.  Smash that blue hyperlink now.

Breathing indoors requires us to use suboptimal air which is not moving or mixing with the wind or interacting with the sun so its charge is lowered. This poor oxygen level = pseudohypoxia = poor PER creation in the AM —> NAD+ drops = higher heteroplasmy = you age faster = more sympathetic activation = more PVN firing = less vagal tone = you get sick quicker indoors. More than 90% of you live inside.

As a result, SIRT 1 lowers with INDOOR living = alters PER cycling in the SCN and every tissue from the cytosol day and night from the nucleus.

Why is this a big deal? When PER one cycle is off so is NAD+ at cytochrome 1 and this causes advanced aging in man due to heteroplasmy because deuterium is let into the mitchondrial matrix where it is not supposed to be!!!

NAD+ is one of the more immediate players in cytochrome 1 that is a huge driver of circadian biology in humans. It is the coenzyme called nicotinamide adenine dinucleotide (NAD+). It participates in a variety of redox reactions in the matrix that help generate DDW. Solar exposure and fasting work with light frequencies to slow ECT flow and this can increase the intracellular NAD/NADH ratio if the light environment is dominated by sunlight. It won’t do this with artificial light. It lowers NAD+. This is what sets off a cascade of circadian events that can destroy tissues because they involve epigenetics and the regulation of growth and metabolism of man. LIGHT DOES THIS. NOT FOOD OR FUELS.

SUN + fasting -> PER cycling is controlled by AM sunlight —> raised NAD+ -> SIRT1 -> BMAL1/CLOCK -> NAMPT -> NAD+ is increased.

You’ve now been retooled with knowledge to be reschooled with wisdom. Share this decentralized wisdom with the world.

CITES

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5418193/

https://forum.jackkruse.com/threads/the-leptin-melanocortin-pathway-and-lyme-risk-ac-power-grid-problem.29476/page-2#post-340068