Fact: Blue light/nnEMF dehydrates cells because they stop H2O production from the mitochondrial TCA cycle. Why is this a big deal? Neurons absorb and release water when firing information. When H2O is MIA so are neurological function/capabilities. You lose the ability to sleep and account for time in your molecular clocks when water is absent. This is why children experience time differently than adults. Less water, less sunlight, or more ALAN at night destroy the clock timing mechanism of living things. Few see this recipe in life’s blueprint
You have to remember Vitamin D is a proxy for your time machine mechanism buried inside your mitochondria. Your job is to see Nature’s science, then understand what it means for your longevity when you are sick. This is a huge big deal if you complied and got jabbed. Without electrification of the phospolipids in your membranes you are asking for a jab complication.
Every person has a different efficiency rate of their time machines based on how the mechanism is built to operate. Few see that Vitamin D is just a bystander in how the process works and this is why when your Vitamin D comes back is a very complex answer.
Here is how it operates.
Did you know the molecule of “more time” (melatonin) acts to aggregate melanosomes in cells to organize them magnetically at night when light is absent?
Did you know the molecule of “more time” optimizes another time crystal in mitochondria called tryptophan in NAD+ tell mitochondria where the Earth is in relation to the sun in a calender year to “fact check” seasonal light variations?
The non visual photoreceptor system in the skin integument, like cholesterols/melanin, concern themselves with the powerful UVB sunlight to create vitamin D as a bystander chemical that acts to electrify the membranes of mammals. This allows for carbon fixation as the top line in the slide shows. Different domains of life use timing and genes to change where energy flows. For example, plants have their own particular route of carbon fixation, converting inorganic carbon in carbon dioxide into organic compounds. They use sunlight to do it. Sunlight is turned into a DC electric signal in its membranes to store energy at the electronic level. Carbon is primarily fixed through photosynthesis, but some organisms use chemosynthesis in the absence of sunlight. Mammals do this. They use their livers to replace the sun. The fixation of carbon dioxide into the skeletal muscle glycogen in intact fasted mammals is best explained by electrification of their membranes which leads to secondary deposition of glucose in the muscle supplied primarily by the liver. We know this by isotopic labeling.
THE ELECTRIFICATION OF MAMMALIAN CELL MEMBRANES IS DONE BY SUNLIGHT.
The sun light is turned into a DC electric signal which turns on the biosynethic decision making abilities in cells. The phtonic skin signal informs the liver and kidney what to do with cholesterol and Vitamin D from this point. The wisdom in your body should amaze you.
Did you know the that the molecule of “more time”, melatonin, also controls the only two programs that recycle your mitochondrial engines, autophagy & apoptosis? Go look two pictures above. It is right there on the bottom of the slide. Without melatonin creation by mtDNA, the quality control mechanism in mt DNA manifest in your heteroplasmy rate. That rate becomes your healthspan and your possible longevity in your life. This is why sunlight links to longevity. That is why the slide below exists.
The efficiency of your mitochondrial “time machines” links to ROS/RNS production from mitochondrial metabolism, which in turn, controls all the magnetochemistry that controls how timing inside of cell directs the flow energy in your key metabolic pathways. Timing alone, controls the decision tree in cells that says, “should this tissue use beta-oxidation, glycolysis, gluconeogenesis, the PPP to get the job of living in this environment.
Melanin, also made from UV A,B,C light controls the levels of ROS/RNS that is coming out of the mitochondria in cells. This is a UNIQUE signal (Kruse for Dummies idea). Melatonin, Melanin, and Vitamin D and NO all manifest from UV light’s interactions with matter in your cells.
Darkness at night, due to the absence of light, cools cells so that magnetic effects in melatonin can use second messengers in cAMP and Ca2+ to aggregate melansomes located around your mitochondria to do its magics inside of cells to tell time to control energy flows. You can fixate carbon and nitrogen at night using the energy stored in biomolecules in your liver to run the dark program without light.
For light to have its full biological effect, photons must be absorbed by visual and non visual photoreceptors in the living body. If one or both are missing, there is no effect. ROS/RNS creation in mitochondria and left over oxygen from metabolism begin this timing process.
Superoxide and H202 are a type of ROS made in mitochondria when we are healthy. Melanin keeps both in check. When do we make ROS during the day? When the sun is out. This is when cells also make melanin. It is also when Vitamin D has an opportunity to be made as well.
BUT WHAT IF YOUR VITAMIN D DOES NOT RESPOND TO SUNLIGHT?
WHY BEING IN THE SUN & still having a low D3 level & STILL BEING SICK TELLS ME YOU GOT A VITAMIN A/nnEMF PROBLEM?
You know Vitamin D is yoked to Vitamin A right. I’ve only said it 1000 times in blogs and podcasts. So when you live in the sun and Vitamin D does not rise here is why——-> https://lnkd.in/e6zptMZW
Non native EMF destroys your clock timing mechanism in your cells and you lose the ability to control the flow of energy in cells. Disease results and you lose time and longevity. That is the story of life in a few words.
Now that the story ion timing is complete, you can see that any kind of blue light or non-native EMF basically uncouple vitamin A from melanopsin. That released retinol from the non visual photoreceptors destroys dopamine, melatonin, melanin, and all heme based photoreceptors so that light becomes useless to cells. This is why a low Vitamin D level is possible with a tan integument or a fit looking body. It does not mean your healthy. Without proper timing longevity is impossible. There is a reason this pictured is my pinned Tweet on X.
Blue light and nnEMF becomes a nuclear weapon for all photo receptors. So you’ll be interested in this. “You may not know it, B12 is a photoreceptor in humans. B12 is another non visual photoreceptor in the liver, CNS, and circulatory system.
And guess what happens when you have melanopsin and retinol damage to B12 levels? That’s the reason why vegans have such demolished B12 level and shrinks their brains and destroys their non visual photoreceptors everywhere, including MELANIN. When melanin goes, timing inside a cell is lost. Magnetochemistry in mammals controls how energy flows. It also controls periodicity in the clock genes and makes the job of the SCN easy. When this goes awry the SCN mechanism becomes uber important to control the molecular clocks in the mitochondria of tissues.
Daylight is always associated with higher electric fields and lower magnetic fields and this correlates to mornings having higher ROS production and melanin production. It is not always linked to Vitamin D levels because UVB light is highly variable based on your latitude and light stability. This effect is seen in mitochondria and it helps tan your interior.
THE TIME CRYSTAL BURIED IN CYTOCHROME 1 IS MADE OF TRYPTOPHAN IS NAD+ THAT LINKS TO MELATONIN BIOLOGY
The morning AM ROS burst at cytochrome 1 (NAD+/NADH) links to the PER2 gene in the mammalian clock mechanism to begin to measure entropy in cells. These effects are rooted in the spin selected ROS partitioning between the free radicals created from metabolism, superoxide and H202, known as the radical triad/pair mechanism of quantum mechanics.
SUMMARY
Energy is trapped directly at the electronic level in cells. Energy is stored as vibrational & electronic bond energies in biochemicals (PER/HIF-1), but also in the structure of the system: its membranes, and in gradients, fields and flow patterns, compartments, organelles & cell water and tissues only gain their organization behind their QED magic. That magic is done in how time controls the flow of energy in clock genes. Just wearing sunglasses does this. This is why people who wear sunglasses burn so often. It is an issue of timing and a loss of magnetochemistry.
Clock genes and time crystalline proteins that ticked when they absorbed light in some way were used to coordinate all zip codes inside the Jacquard cards of biochemistry in cells to measure the entropy to control the overall flow of energy in metabolic pathways. Time controls the flow of energy in matter and this allows cells to extract information about where the Earth is in relation to the sun. The sun created the most powerful protein in Earth’s history (melanin) because it can harvest the entire power potential buried in the electromagnetic spectrum. When melanin is heated up it becomes a supreme electrical conductor. When it cools it mimics what a superconductor can do with magnetic flux in light. This allows for electron- electron interactions and for electron-phonon coupling only seen in superconductors. Yes, we have superconductive proteins in us that operate with light, temperature, and precision timing.
Water was the plasma for most of Earth’s history, but nature created melanin to work with water something unique occurred. The binary code of life that builds order from chaos begins by deciphering the code that exists between H+ and deuterium separated from water and plugged into ancient biochemical pathways to sculpt something new.
Ocular and integumentary melatonin levels both end in leptin biology. This is why the leptin receptor sits behind the retina and infront of the hypothalamus that controls energy balance. It also explains why our subcutaneous fat sits just below the skin, and why leptin, the hormone that links to the leptin receptor in the brain visits the brain every night during the dark, in the hypothalamus to adjust the clock timing mechanism in the SCN. Few see Nature’s recipes. Hopefully some of you can start putting the science of the blogs together like a QUILT.
Cellular disorganization is always linked to poor timing and its effects always manifests in disease creation before death manifests; Disease and death are an immutable ledger built as a consequence of how time does or does not flow in our clock timing mechanisms in cells. In this way, we always seem to get the sense that illness comes before death in most living sequences unless we are talking acute trauma. This points out why the information of the organization is as important as energy flux in a cell to maintain wellness. Timing is the MOST critical ingredient in Nature’s recipes.
When timing is off inside of a cell, the energy cost become prohibitive for a cell in a diseased person whose tissues are afflicted with defective mitochondrial DNA. This amplifies the need for precision accuracy, and when that periodicity is absent, this amplifies the creation of new mutations in the mitochondrial genome. This further lowers energy production and this further magneifies timing efficiency and forms a doom loop for the cell. The phenotype in the cell when timing is lost is the alteration of epigenetic signaling mandating for higher turnover. Mitochondria mutations speed up nuclear genome action. When the nuclear genome increases it is for one reason: To fix the atomic arrangement of atoms inside of a cell to recapture the timing mechanism required for living. The nuclear genome is designed by nature to be quite stable and quiescient.
Mitochondrial DNA is more vulnerable to alteration than nuclear DNA, for two reasons.
First, mitochondria are a major source of intracellular reactive oxygen species (ROS). Therefore mitochondrial DNA is designed to be under much stronger oxidative stress than is the nuclear DNA.
Second, mitochondria have a matrix-side negative membrane potential to carry out oxidative phosphorylation. This electric membrane potential concentrates lipophilic cations inside mitochondria up to approximately 1,000-fold.
That electric potential called redox power is created by the trsansformation of sunlight. This is how light controls metabolism. Light > Food always. It is axiomatic.
How do eukaryotic cells make light from matter? It all begins with the fact that oxygen is the terminal electron acceptor for eukaryotes mitochindria. The oxygen molecule O2 displays some very particular features. It is a paramagnetic gas. It is the second most electronegative element in the periodic table. Because of this one should expect that O2 is a very reactive chemical compound. Counterintuitively, however, it is a quite unreactive molecule on Earth. How can I say this? . A testimony to this is the decentralized fact that we live in a planet with a 21% oxygen atmospheric content, and nevertheless life thrives and even depends on it.
The reason why O2 is so much unreactive than we expect this atom to be, in spite of its electronegativity, is the fact that in the fundamental state it assumes a triplet electronic configuration. Triplet electronic configuration occurs when the valence electrons arrange in the O2 molecule has the electronic configuration with the least energy. According to the molecular orbital theory, this arrangement has a triplet character. Triplet character occurs when the two outermost electrons in oxygen have different antibonding orbitals, but with the same spin state.
3^O2 expresses the triplet character of the ground state. This ground state has a biradical chemical character, which renders it quite unreactive to most chemical compounds. This triplet character of oxygen makes ground state O2 a paramagnetic compound, where oxygen gas tends to move to the point of strongest magnetic field. This is why oxygen is drawn to your colonies of mitochondria. Mitochondria create a magnetic field because they have electron movements along its innermitochondrial membrane while having a spinning ATPase on that same membrane. This makes all mitochondrial respiation electromagnetic at the most fundmental level.
Implications of this for cells? The cell cycle is controlled by light creation in cells.
The metabolic rate in our mitochondria drives developmental timing by controlling the cell cycle.
Having light stored at the electronic level is critical to the mystery of morphogenesis.
Back at the origin of life 3.8 billion years ago on Earth electric membranes drove CO2 fixation by converting gases from volcanoes (driven by the solar plasma) into organic chemicals to create growth in the absence of oxygen. This was the first step life took at the ocean floors.
Metabolism has always been spontaneous on Earth. Today, we believe complex life emerged from ancient autotrophic pathways fueled by volcanic gases as carbon and the sun as ultimate energy sources. Variants of these pathways remain in modern autotrophs in the deepest branches of the tree of life. In this way, the DC electric current in membranes preceded all chemistry. The energy metabolism of modern autotrophs resembles the geological interactions of H2 and CO2 gases in hydrothermal vents. Centralized science believes this points to a metabolic origin of biochemistry at the interface of the lithosphere and hydrosphere. Decentralized science believes this points to a quantum thermodynamics at the core of life because the sun drives all these processes. This step is fundamentally why light tops food in all my discussions.
Reactive oxygen species (ROS) are formed during normal metabolic processes. Mitochondrial respiration is spontaneous on Earth. Genes amplify metabolism and in this way, gene products are capable of making more or less light from how they amplify or de-amplify metabolic networks in cells.
ROS are magnetic signal created from excess oxygen in mtDNA. People forget molecular oxygen is the only paramagnetic gas on the periodic table. This makes it a unique magnetic signal used in cellular communication. All free radicals have one unpaired electron to make them magnetic sugnals. All ROS are called singlet oxygen signals that come from mtDNA actions. When electrons are added to a magnetic molecule this is called a reduction type of reaction. When singlet oxygen is reduced by the addition of electrons, the ROS magnetic signal shifts to a lower energy state and as a result emits photon.
The electronic transition of electronically excited species from the singlet or the triplet excited state to the ground state is accompanied by photon emission. This is how light is crerated by life. In this mechanism only a few photons are emitted per second per square centimeter, the photon emission is ultra-weak in nature. But it is these photons that explain how light sculpts life.
Reactive oxygen species are formed during the metabolic processes linked to life-sustaining enzyme-catalyzing reactions. They also can be formed during the response to stress reactions when any life form is exposed to biotic and abiotic stress factors from our environment. When ROS are effectively scavenged by the antioxidant defense system, the oxidative effect of ROS on biomolecules such as lipids, proteins and nucleic acids is fully prevented.
HOW IS ENDOGENOUS LIGHT CREATED?
Cells cannot product light without oxygen or ROS/RNS made in mitochondria. Mitochondria are not only time machones, they are factoried for biophotons. That is the bare minimum and is confirmed in Roeland Van Wijk book on the topic of biophotons. During the process of cellular respiration, electrons are transported through a series of mitochondrial complexes to the terminal electron acceptor, molecular oxygen (O2). In the process of cellular metabolism, the electrons released from the ETC react with O2 to produce superoxide (O−2) radicals. Mitochondrial complexes I, II, and III contribute to the maximum in redox signaling. Superoxide radicals generated at complexes I and III are released into the intermembrane space which comprises 80% of superoxide radicals generated in the mitochondria and remaining 20% are made by mitochondrial matrix. The mitochondrial permeability transition pore in the outer membrane of the mitochondrion allows the passage of superoxide radicals into the cytoplasm where it is dismutated to hydrogen peroxide, a highly diffusible secondary messenger. This reaction is catalyzed by superoxide dismutase located in the mitochondrial matrix (MnSOD) or in the cytosol (by Cu/ZnSOD).
Furthermore, aquaporin 8 serves a channel for the release of hydrogen peroxide from the cell membranes. There is an another major site for the generation of ROS termed as peroxisomes where superoxide and H2O2 are generated through xanthine oxidase in the peroxisomal matrix and membranes. Other sources of ROS include endogenous metabolites such as fatty acids, prostaglandins, and exogenous components including drugs, flavorings, coloring agents, antioxidants, etc. These substances are processed in the smooth endoplasmic reticulum and transformed into free radicals, especially ·OH. Macrophages and leucocytes, as a part of immune response contribute to the formation of free radicals
SO NOW YOU KNOW HOW ROS ARE MADE, HOW DO THEY TRANSFORM MATTER TO EMIT LIGHT?
However, under circumstance, when the formation of ROS exceeds the capacity of antioxidant defense system, biomolecules in cells spins are changed by magnetic forces in mitochondria. The single unpaired electron of ROS oxidizes lipids, proteins in both nuclear genome and mitochondrial nucleic acids. This interaction leads to the formation of high-energy intermediates like singlet oxygen. The decomposition of high-energy intermediates generates the electronically excited species which undergo an electronic transition from either the singlet or the triplet excited state to the singlet ground state. When there is an electronic transition of electron spin light is liberated.
When electron spin is changed, & our biomolecules are changed and the result in matter in our cells is to liberate light.
Singlet oxygen, systematically named dioxygen and dioxidene, is a gaseous chemical with the formula O=O, which is in a quantum state where all electrons are spin paired. It is kinetically unstable at ambient temperature in cells, and its rate of decay is slow.
Singlet oxygen (represented as 1ΔgO2, abbreviated as 1O2 in papers) is not a radical but represents an excited state of O2 in which the spin of one of the unpaired electrons is changed to yield two electrons with opposite spins. The terms ‘singlet oxygen’ and ‘triplet oxygen’ derive from each form’s number of electron spins. The singlet has only one possible arrangement of electron spins with a total quantum spin state of 0, while the triplet state has three possible arrangements of electron spins with a total quantum spin of 1, corresponding to three degenerate states. An excellent way to detect the presence of singlet oxygen in reactions is using steady-state or time-resolved measurements to find its characteristic phosphorescence at around 1270 nm.
When oxygen is reduced by losing an electron, singlet oxygen is created chemically. Singlet oxygen is an reactive oxygen species (ROS). The loss of the electron shifts the biomolecule to a lower energy state and as a result it emits photon. RNS works exactly the same way. This is how mitochondrion make light from matter.
The oxidation of biomolecules occurs by hydrogen abstraction by superoxide anion and hydroxyl radicals or by the cycloaddition of singlet oxygen initiate a cascade of oxidative reactions that lead to the formation of electronically excited species such as triplet excited carbonyl, excited pigments and singlet oxygen. The abstraction of hydrogen is defined by the removal of a hydrogen atom or group from a molecule by a free radical. Hydrogen/deuterium atom abstraction is often confused with deprotonation, which is the removal of a hydrogen atom (i.e., a proton) by a base in an acid-base (proton transfer) reaction.
When deuterium is abstracted out by light cells need to use more UV light to do so because you need more VUV-UVC-UVB-UVA light to abstracted the heavier isotope of hydrogen because of the extra neutron. Generally, the way oocyte selection occurs in mammals is by hydrogen abstraction. The oocytes with the lowest atomic mass are ejected first. during menarche. As time elapses, the eggs with the most deuterium and released last because more light is needed to get that job done. This drains the electronic level of its stored light.
This means the older a pregnancy becomes the more light is needed in the transgenerational process. In this way you can see now why high maternal age and transgenerational epigenetic diseases occur. It is also why more chromosomal abnormalities occur as mammals age. If the UV light is expended abstracting deuterium there is less light left at the elctronic level to drive the cell cycle past the mitosis level. This is a huge problem in infertility, cancer, and in morphogenesis (autism)
The photon emission of these electronically excited species is in the following regions of the spectrum (1) triplet excited carbonyl in the near UVA and blue–green areas (350–550 nm), (2) singlet and triplet excited pigments in the green–red (550–750 nm) and red-near IR (750–1000 nm) areas, respectively and (3) singlet oxygen in the red (634 and 703 nm) and near IR (1270 nm) areas. The understanding of the role of ROS in photon emission allows us to use the spontaneous and stress-induced ultra-weak photon emission as a non-invasive tool for monitoring of the oxidative metabolic processes and the oxidative stress reactions in biological systems in vivo, respectively.
Transcriptional regulation of proteins is done by GLUT expression by ROS in cells.
Since ROS is made from excess dissolved oxygen not used in mitochondria, when oxygen in cells decreases, ROS signaling becomes more unique because of scarcity. This should make you think about my Kruse for Dummies lecture.
Low oxygen triggers signal-transduction pathways involved in both cell death and survival. Anoxia activates proapoptotic BCL-2 proteins and caspases to initiate apoptosis. The adaptive cellular events that occur in response to hypoxia are mediated largely by the transcription factor hypoxia-inducible factor-1 (HIF-1)
During hypoxia, ROS levels increase by design and play an important role in HIF-1α stabilization. HIF-1 consists of two subunits, HIF-1α and HIF-1β. Under normoxic conditions, prolines within the oxygen-dependent degradation domains (ODDs) of HIF-1α are hydroxylated by prolyl-4-hydroxylases (PHDs; Ivan et al. 2001). This hydroxylation mechanism acts as an ubiquitination signal leading to proteasomal degradation of HIF-1α. In the absence of oxygen, HIF-1α ubiquitinylation is inhibited allowing its interaction with HIF-1β to drive transcription of various target genes, including GLUT1. This is the basis of how the Warburg shift occurs in humans.
Stimulation of cellular glucose uptake in mammalian cells is frequently observed during conditions of oxidative stress when ROS and RNS spikes. GLUT1 helps in the transport of glucose, galactose, mannose, glucosamine and ascorbic acid in mammals.
HIF-1 induces the expression of multiple antiapoptotic BCL-2 proteins to promote cell survival. Interestingly hypoxia increases production of mitochondrial reactive oxygen species (ROS), which serve as signaling molecules to activate HIF-1.
Hypoxia-inducible factors (HIFs), are major molecules that respond to hypoxia and elevated temperatures to play important roles in cancer development by participating in multiple processes. HIF1 is linked to circadian clock controls, metabolism, proliferation, and angiogenesis. The Warburg phenomenon reflects a pseudo-hypoxic state that activates HIF-1α. In addition, a product of the Warburg effect, lactate, also induces HIF-1α. However, Warburg proposed that aerobic glycolysis occurs due to a defect in mitochondria. I believe the defect occurs, first, in the circadian mechanism to affect mitochondrial metabolism. Moreover, both HIFs and mitochondrial dysfunction can lead to complex reprogramming of energy metabolism, including reduced mitochondrial oxidative metabolism, increased glucose uptake, and enhanced anaerobic glycolysis
HOW DOES HYPOXIA LINK TO ALTERED TIME STAMPING IN CELLS?
Circadian clocks are endogenous coordinators of the 24-hour rhythm of behavioral and molecular processes in living organisms. For humans, a master clock modulating circadian rhythms is located in the suprachiasmatic nucleus of the hypothalamus and is a pacemaker of the system. In mammals, the circadian clock is comprised of a set of genes, which function as activators—CLOCK and BMAL, which, similarly to HIF, are bHLH-PAS transcription factors.
HIF-1α GENE HAS A TRANSCRIPTION REGULATORY ELEMENT TO ALTER THE CIRCAIDAN CLOCK MECHANISMS. THIS IS HOW DIESESE ARE CAUSED IN THE MODERN WORLD.
Through binding to regulatory elements containing E-boxes (also present in HIF-1α gene) they activate the transcription of repressor protein period (PER) and cryptochrome (CRY). Additionally, HIF can bind to promoter regions of repressor proteins through hypoxia response elements (HRE), causing their transcrioptional upregulation leading to altered cell signaling. This is how the redox shift leads to alien light creation to lead to genetic changes we see in ALL CANCERS.
It is believed in centralized science that mitchondrial ROS oxidizes lipids, proteins and nucleic acids and thus initiate a cascade reactions that leads to the formation of electronically excited species responsible for the photon emission in near UVA, visible and near IR regions of the spectrum. I think this is a simple explanation for what is really going at the atomic level in a cell.
To make it crystal clear how bad the advice centralized medicine is giving patients you just need to review this thread in the context of what is being clearly laid out in this blog. Read this thread below in blue before going on.
COVID AMPLIFIES THIS SCIENCE FOR THE SLEEPING SO THEY WAKE UP WHERE MODERN DISEASES COME FROM
TURBOCANCERS, mRNA, SV40, CIRCADIAN MISMATCH LINKS TO BIOPHOTONS
p53 is an important tumor suppressor gene, found to be mutated or absent in over 50% of all cancers studied. It functions as a sequence-specific DNA-binding transcription factor. In response to double-stranded DNA breaks, p53 is converted from a latent to an active form. This results in increased expression of p53-responsive proteins such as p21 which are required for growth arrest at the G1-to-S phase transition. It also mediates apoptosis via the increased expression of proteins such as Bax. Inactivation of p53, therefore, results in the loss of a cell cycle checkpoint control required for repair of damaged DNA and prevents apoptosis in response to severe DNA damage. In the absence of these responses, oncogenic mutations which may result in tumor progression can accumulate in nuclear DNA and this can happen quickly in people who took the mRNA jab. The damage can accumulate rapidly. From the above, it is clear that the transcriptional activation function of p53 is critical to its role as a tumor suppressor gene. Since genes amplify metabolic networks, p53 clearly clearly has a lot to do with the biophoton spectra that cells emit.
Note the history of the discovery of p53 brings us back to the story of SV40 and the polio vaccine. That is why Pfizer erased it from their plasmid map in the mRNA platform. Simian virus 40 (SV40) large tumor antigen (T antigen) has been shown to inhibit p53-dependent transcription by preventing p53 from binding to its cognate cis element.
Why is p53 called p53?
One must know the history and the discovery of the most studied gene in human history, also known as the guardian of the genome. Why is it considered the guardian of the genome? Aneuploidy refers to the state of unequal chromosome copy numbers and is one of the most prominent genomic aberrations in solid tumors. Most solid tumors are aneuploid, and p53 has been implicated as the guardian of the euploid genome.
How long did the pulse of cancer stay in human cancer data from the Cutter incident of the Polio vaccines?
Bernice Eddy found the SV40 in the Salk vaccine and told the world about it in th emid 1950s’ The NIH and FDA ruined her career over her admission and scrubbed their websites. Most people ran from studying SV40 after this event in centralized science until the Nixon administration. What cancers in humans are linked to SV40 contamination?
In the 1970s, David Lane & Arnie Levine started studying a virus called SV40. Their interest was in a viral gene responsible for transformation, the SV40 oncogene called large T antigen. Lane’s task was to extract the large T antigen protein from cells infected with SV40. He used electrophoresis to separate the protein molecules based on size and charge. Whenever he ran electrophoresis to purify the large T antigen, he always found an unknown protein with a molecular weight of 53 kilodaltons. Initially, others in Lane’s lab thought it was a contaminant or a breakdown product of the large T antigen. As reports of this protein came from other labs too, it was named p53 based on its molecular weight.
In 1979, immunological studies identified the p53 protein due to its immunoreactivity with tumor antisera, suggesting its role as a tumor-associated antigen. Everyone was convinced they had found a new oncogene. The excitement was high! Wait a minute—did I say oncogene? p53 is actually a tumor suppressor gene! This is my favorite plot-twist of the p53 story: its mischaracterization as an oncogene. The initial misclassification was due to the research climate of the time (1980s). Oncogenes were thought to be the key to understanding cancer, and the idea of a tumor suppressor gene was in its infancy.
As mentioned above, p53 was initially found bound to the major oncogenic protein of SV40, so it was understandable at the time to think it must be an oncogene as well. However, some experimental observations did not fit well with the idea that p53 was an oncogene. In 1986, the first tumor suppressor gene, Retinoblastoma gene (RB1), was discovered, confirming Knudson’s Two-Hit hypothesis. In 1989, this two-hit model was applied to p53, specifically in colorectal tumors. It fit this model, as in virtually all cases, both copies of p53 were mutated.
This conclusion was confirmed by subsequent findings that patients with inherited mutations of p53 were predisposed to diverse tumor types. Mice with engineered “knock-outs” of the p53 gene were also tumor-prone. Today, more than 70,000 research papers are published on p53, making it the most studied human gene in history. Mutations in p53 are found in >50% of human cancers. “It’s impossible or very difficult to get a malignant tumor without the activity of p53 being disrupted.” ~Bert Vogelstein, a legendary figure in p53 story who was the first to termed it as a tumor suppressor gene.
New centralized data does not support a role for p53 in aneuploidy surveillance in organotypic cultures. There is strong evidence indicating that the loss of p53 is associated with chromosome instability and poor prognosis in the development of several cancers. See the papers (Donehower et al., 2019; Foijer et al., 2014; Fujiwara et al., 2005; Watson and Elledge, 2017)
When p53 goes awry the majority of tumors show varied TP53 mutations based on the following papers: (Clausen et al., 1998; Muller and Vousden, 2013). Live-cell imaging of Trp53+/+ and Trp53 & mCOs showed that mitotic errors, including lagging chromosomes and multipolar mitoses, occurred frequently in cells lacking p53, consistent with several published studies (Artegiani et al., 2020; Drost et al., 2015).
The bottom line issue for me? Decentralized science must show that when p53 signaling goes awry, it corresponds to a lack of ultraweak -UV biophoton production at the mtDNA level. This is why apoptosis goes awry and this also corresponds to the following variables: Low mtDNA melatonin production, low mtDNA water production, and altered mtDNA CO2 production in most solid tumors. This results in cell cycle arrest and cells begin to migrate to other tissues that have the ability to generate the ultra weak UV biophotons to complete the cell cycle.
For light to have a biological effect, photons must be absorbed by photoreceptors in the living body or there is no effect. ROS/RNS creation in mitochondria and oxygen begin this process.
H202 is a type of ROS made in mitochondria when we are healthy. When do we make ROS during the day? Daylight is associated with higher electric fields and lower magnetic fields and this correlates to mornings having higher ROS production. The morning AM ROS burst links to the PER2 gene in the mammalian clock mechanism. These effects are rooted in the spin selected ROS partitioning between peroxide and H202, known as the radical pair mechanism
When you add hydrogen perioxide to tissue heat is released and this heat is a clue that light is being liberated from the atoms in matter in your cells. I do this in almost every surgery I do but rarely tell people why I am doing it or how it works. My surgical team sees a lot of bubbles released and those bubbles are hydrogen gas being liberated at the same time. But there is more magic below the cell level to help my patients get better. I got the idea from radish roots.
This study below investigates the spontaneous emission of biophotons from radish root cells and the effects of various treatments on this biophysics phenomenon.
Key findings in the paper:
1. Freshly isolated radish root cells exhibit spontaneous biophoton emission at a rate of about 4 counts per second. This is an ultraweak photon release.
2. Addition of hydrogen peroxide (H2O2) to the cells from matter in cells significantly enhances biophoton emission from cells up to a rate of about 500 counts per second. When I read this I said I know how I can improve redox in the surgical site of my patients. This light has a specific spectra in the case of radish roots. This varies in eukaryotes like mammals.
3. The enhancement of biophoton emission by the addition of H2O2 is completely abolished when molecular oxygen is removed by using a glucose/glucose oxidase system or when reactive oxygen species (ROS) are scavenged using reducing agents such as sodium ascorbate and cysteine. This is a big clue glucose is not the real problem in cancer, unfettered light release to alter the nuclear genes is by excessive ROS. This tells you why cancers always are associated with their own brisk blood supply to keep oxygen humming to create light cells are missing to get through mitosis. That light is ultraweak UV light.
4. Spectral analysis of the H2O2-induced biophoton emission shows that biophotons are mainly emitted in the green-red region of the spectrum in radish.
5. The energy for this biophoton luminescence in cells is produced when an excited biological molecule drops to a lower energy state and the majority of the excited biological molecules are reactive oxygen species (ROS) Singlet oxygen, systematically named dioxygen and dioxidene, is a gaseous inorganic chemical with the formula O=O, which is in a quantum state where all electrons are spin paired. It is kinetically unstable at ambient temperature, but the rate of decay is slow.
Singlet oxygen (represented as 1ΔgO2, abbreviated as 1O2) is not a radical and represents an excited state of O2 in which the spin of one of the unpaired electrons is changed to yield two electrons with opposite spins. The terms ‘singlet oxygen’ and ‘triplet oxygen’ derive from each form’s number of electron spins. The singlet has only one possible arrangement of electron spins with a total quantum spin of 0, while the triplet has three possible arrangements of electron spins with a total quantum spin of 1, corresponding to three degenerate states. An excellent way to detect the presence of singlet oxygen is using steady-state or time-resolved measurements of its characteristic phosphorescence at around 1270 nm. Phosphorescence is a process in which energy is absorbed by matter in a cell and is released relatively slowly back into the cell in the form of light. This is in some cases the mechanism used for glow-in-the-dark materials which are “charged” by exposure to light. In mammals the amount of light is absorbed quickly by chromophore proteins to be used for signaling. This is why surgeons do not see what is clearly there during surgery. Plus, humans retina cannot see NIR light at 1270 nm . So how do we know this light is present? We use machines that can detect it.
Electron paramagnetic resonance (EPR) spin-trapping data shows that the formation of singlet oxygen is observed after addition of H2O2 and correlates with the enhancement in biophoton emission.
These findings in the paper below provide direct evidence that singlet oxygen is involved in biophoton emission from radish root cells. The results suggest that the biophotons are generated through a process involving the formation of singlet oxygen, which emits 1270nm light in the NIR range of the spectrum whose production can be enhanced by the addition of hydrogen peroxide.
SUMMARY
We know about how radish roots do it, but what about us when the solar cycle is awry?
Melatonin as a potent antioxidant and anti-inflammatory agent is known for protection of normal tissues against ionizing irradiation. This tells us that is has to be a control switch for ROS creation. Bright light and heat lowers melatonin effects and dark and cold temperatures increase its effects. Melatonin clearly is linked to ROS creation in some novel way. What do we know about sunrise? Light dominates the environment and the electric ield in the plasma called the ionosphere increases. The magnetic field of the gases in that ionosphere increases. 21% of the atmosphere is made of oxygen that is paramagnetic and this causes it to be drawn to tissues with high mitochondrial capacity. lWhen the sun rises, ight (photons) begins transfers energy to matter, causing the electrons to be emitted. What is it about electrons that maybe the clue to this mystery?
If you look back to Becker’s work he warned us about about the negative effects of electropollution. He also taught us about magneto pollution to a lesser degree. He also showed us how strong gauss magnets were able to induce anesthesia in salamanders before surgical procedures. He told us about the effects he and Marino found along electric power lines in Upstate New York in human subjects.
MAMMALIAN IMPLICATIONS OF THIS?
The TCA cycle produces more reducing power with a 22 ATP gain from its reducing power while glycolysis only produces 6 ATP from reducing power and Pentose phosphate group varies in the number of reducing power it produces. This tells us glycolysis and the TCA cycle on a relative basis vary greatly in how much ATP and ROS they produce.
Metabolic rate, when viewed from ATP creation is driving a developmental timing mechanism in a cell. But something must act as an off and on switch.
We know from the video about that 1270nm light equalizes the amount of ATP and ROS they produce. This appears to be the tipping point between day and night to act as an off and on switch in some way.
The implications are vast for many diseases from cancer, healing, autoimmunity, and to mastocytosis. How so?
For close to 15 years I have been giving major clues out to my tribe that magnetic fields are linked to circadain clock biology. How do they link is the biggest mystery in decentralized science. This blog explains the link CLEARLY.
The centralized assumption that I have always rejected has always been that the eye mainly senses light, whose local distribution is transmitted to the brain in a kind of copy by a mosaic of impulses. This is an idea so obvious to abuse by Nature to encode a hidden message in how life really operates. Instead of accepting that assumption, I looked for evidence in the literature of researchers who were wiser and actually thought about attaching electrodes to the animals who made it through the last extinction effect optic nerves so we might be able to eavesdrop on the signals Nature was really sending to message. I found evidence in amphibians where researchers positioned an aluminum hemisphere around a frog’s eye and moved objects attached to small magnets along the inner surface of the sphere by moving a large magnet on its outer side. This effect was eerily similar to the effects found in Becker’s work on regeneration and salamanders. This simple experiment showed me that non visual photoreception was the mysterious behind what Nature was doing. Then I jumped hard and looked for more zebra’s. And I found them in the eye of mammals and wrote the evidence in slides for talks I gave all over the world.
WHAT IS THE ELECTROMAGENTIC INFORMATION CELLS LOSE WHEN OUR EYES GO AWRY?
Think about what I have said about the European Robin eyes and the cryptochrome gene for 20 years now. The answer is buried there.
The development of the radical pair mechanism used for magnetoreception in bird retina has allowed for explanation of the fact that magnetic fields are observed to have an effect on chemical reactions speed in animals that made it through the last extinction event. It turns out the same mechanism found in birds, and their ancestors, theropod dinosaurs is also found in all mammals. The eye speaks to the brain using Nature’s most hidden recipe built in a language already highly organized in atomic physics, clocked and expertly interpreted, instead of transmitting some more or less accurate copy of the distribution of light on the receptors.
This quantum mechanism describes how an external magnetic field can alter chemical yields by interacting with the spin state of a pair of radicals.
The radical pair mechanism is based on the dependence of product yields on
1) the hyperfine interaction involving electron spins and neighboring nuclear spins of atoms and
2) the intensity and orientation of the geomagnetic field.
3) the chemical reaction kinetics in radical pair mechanism (RPM) is related directly to the qualities contained in the magnetic field in question. Overall, the spin evolution between singlet and triplet states in ROS/RNS radicals are affected by the magnetic field, is the key feature for radical pair mechanism.
4) Early proponents & biophysicists missed the magnetochemical effect in cells and focused on light chromophores.
This told my decentralized mind that one needs to know the general scheme of chemical reactions involving radical pairs generated from singlet and triplet precursors; one needs to understand the spin dynamics of the radical pairs; and the magnetic field dependence of product yields caused by the radical pair mechanism. It also told me I needed to understand solar cycles and disease patterns better. This told me I needed to look carefully at the eye of birds where the RPM mechanism was discovered in the mid 1970s. So that is what I did. If you go back and look at the Cold Thermogenesis #6 blog post you’ll see what I said there about the eye and spin dynamics. It was all there early.
During summer solstices did you know that pipes corrode faster than normal? Do you know why this happens? It is the same reason bipolar patients symptoms get worse during the summer solstice: Magnetic field strength is at its strongest. Did you know the Pandemic of 1918 also was linked to solar dynamics and magnetic strength? The majority of pandemic influenza outbreaks since 1700 CE were associated with minima and maxima of sun spot numbers linked to the 11 year solar cycle. In fact, seventy-four percent of influenza pandemics and epidemics (26/35 events) since 1700 occurred at or within one year of the peak or trough in sunspot numbers, increasing to 89 percent (31/35) within two years. This links magnetic field strength to immune function.
Chemical reactions in our immune system that involve radical intermediates are influenced greatly by magnetic fields. These fields act to alter their rate, yield, or product distribution. These effects have been studied extensively in liquids, solids, and constrained media such as micelles. None of these are well studied in centralized medicine. They will be well studied in El Salvador’s new decentralized system. It maybe the earliest detection system of disease we have today.
CONSIDER THIS EXAMPLE:
The involvement of singlet oxygen in biophoton emission has implications for our understanding of many diseases like mast cell disease in the skin that links to immune function. Mast cell dysfunction is linked to an absence of 1270 nm light in skin of mammals. Singlet oxygen is known to liberate this frequency of light as the picture below shows. People with mast cell disorders do not make enough hydrogen peroxide from their mitochondrial respiration. As a result, with a comorbid lack of sunlight containing 1270 nm light and lack of H202 creation in tissues is asosciated with immune dysfunction in mast cells. There is a lesson here that radishes are teaching us about mastocytosis.
People with cancer, autoimmunity, mastocytosis, and poor wound healing always are deficient in AM sunlight. Why? This is when we get a lot of NIR light that has 1270nm light. Early morning 6AM -9AM sunlight has a relative irradiance that has a higher amount of photons in the visible and NIR spectrum compared to midday exposure (noon). The picture tells why you decentralized medicine always recommends AM sunlight. This sun time = TINA = THERE IS NO ALTERNATIVE. You remember that recent blog?
All of you know about the COVID pandemic. Do you know that it began during a weak solar cycle. This was my first evidence that the 2020 Wuhan flu was manufactured because it made no sense based on what I know about immune cell function. What prediction can I make right now about those who took the experimental treatment? 2024-2026 will be deadly for those people. Why?
The 1918–1919 pandemic flu virus happened during during an exceptionally strong solar cycle and caused acute swelling of and bleeding from the lungs from the healtiest people on Earth, and people who were infected typically suffocated within one to two days. Most were men in great metabolic shape. The second wave of the pandemic was responsible for the most deaths, due to an unusually severe hemorrhagic pneumonia. Today, in 2024 we just had 4 massive CMEs where the aurora’s were seen into the tropics, and the people who were the most healthy are dying at unbelievable rates now as evidence by my Tweets over the last 9 months. How did i know in 2020 this was coming. I know history and I know about the RPM mode of transmission to ROS/RNS.
H5N1 is bird flu. Why is the CDC, FDA, and WHO warning today’s COVID jab victims today about bird flu? Because they know they caused the COVID pandemic accidentally via gain of function study in Wuhan and now they are seeing evidence of patients in the experimental group who are beginning to experience similar pathologies to those of the 1918–1919 pandemic. Namely they are EXPECTING MASSIVE acute respiratory distress syndrome to overwhelm high vaccinated populations. This is the collateral effect they fear. The act of blocking your eye or skin from the sun around these coming dates might be a deadily choice for many. Also on these dates I expect we will have serious political and economic news hit to change sentiment rather quickly. Neurologic processing changes this way when ROS/RNS peaks.
HOW DO YOU TREAT IT BEST?
ARTIFICIAL RED PANELS DO NOT EQUAL AM SUNLIGHT EVER.
The radical-pair mechanism explains how a magnetic field can affect reaction kinetics by affecting electron spin dynamics. Most commonly demonstrated in reactions of organic compounds in cells involving their radical intermediates, and a strong magnetic field can speed up a reaction rates by decreasing the frequency of reverse reactions. This is why bipolar patients are acting very bizarre this year. It is also why those with neurodegeneration are having wild swings in their symptoms day to day now. Look at the narratives around Biden’s behavior lately. It is all controlled by the puse of ROS his defective mitochondria are making during this tumultuous time as we revovled around the sun.
Mitochondria act as signaling organelles in low-oxygen conditions. Hypoxia (0.5–3% oxygen) increases mitochondrial ROS that activate transcription of adaptive genes. Anoxia (0–0.5% oxygen) initiates mitochondrial outer membrane permeabilization (MOMP) to activate cell death. You’ve been given an amazing piece of decentralized science today. Use it wisely.
My predictions for my tribe in SOLAR CYCLE 25?
Know your solar history and you choices around the jab. On September 15, 2020, the solar minimum occured between Solar Cycle 24 and 25 – the period when the sun is least active – happened in December 2019 when Wuhan virus escaped. When I reviewed the 13-month period, the smoothed sunspot number fell to 1.8. The only part of the government who told the truth at this time was NOAA and NASA. According to the Solar Cycle 25 Prediction Panel they are praying for weak Solar Cycle 25 because of the lab leak. Early 2024 results however, have showed their guesses were WRONG. They expected peak sunspot activity expected in July of 2025, but within the last month we have had spectacular CME from sunspot activity that hit Earth. This is why so many people with jab injuries, mental illness, clots and cancer are hitting my emergency room like nuts the past 6 months. I expect the July 2024 – July 2025 Solar cycle to be DEADLY.
The Solar wind energy striking the Earth’s magnetosphere affects the entire environment because the pressure on the region increases and the magnetosphere shrinks sometimes four Earth’s radii. This sudden compression causes earthquakes in specific plates. We’ve had a quite a few more earthquakes in El Salvador the first 6 months of 2024. Maximum quake frequency occurs at times of moderately high and fluctuating solar activity. Terrestrial solar flare effects which are the actual coupling mechanisms which trigger quakes appear to be either abrupt accelerations in the earth’s angular velocity or surges of telluric currents in the earth’s crust. It appears even earthquakes are magnetochemical.
Solar Cycle 24 was average in length, at 11 years, and had the 4th-smallest intensity since regular record keeping began with Solar Cycle 1 in 1755. It was also the weakest cycle in 100 years. Solar maximum occurred in April 2014 with sunspots peaking at 114 for the solar cycle, well below average, which is 179.
Solar Cycle 24’s progression was also unusual and why NO PANDEMIC should have hit us at this time. This is why I knew in 2020 that this was a gain of function Lab leak manufactured problem. Now you know why I was so on top of it. in 2024 the Sun’s Northern Hemisphere led the sunspot cycle, peaking over two years ahead of the Southern Hemisphere sunspot peak. This resulted in solar maximum having fewer sunspots than if the two hemispheres were in phase. This has a lot to do with the changing of the magnetic poles in the Northern hemisphere now. The magnetic North Pole is now in the UK. Recall, last summer I visited this area. Now you know why.
Solar Cycle 25 PREDICTIONS for the EXPERIMENTAL GROUP AND THOSE WITH LOW REDOX
For the last eight months of 2023, activity on the sun steadily increased, indicating we transitioned much more rapidly to Solar Cycle 25 in the first 6 months of 2024. More people already sick with diseases will die sooner. Kids with autism will struggle more this year. People who have a framshifted genome will get sicker quicker and die more rapidly than their centralized MDs expect. The more healthy they are, the more likely they will get sick and die. It is only shocking when you DO NOT UNDERSTAND THE BIOPHYSICAL LESSONS IN THIS BLOG.
I have a sense Solar cycle 25 is also going to be be unusual with more strength and higher peak of sunspots than NASA and NOAA expects. In Solar cycle 25 NASA said they expected only 115 sunspots. The first 6 months of 2024 told me otherwise. We are experiencing it right now based on the recent auroras and the weather in the tropics. We are seeing this in the radical changes in President Biden’s behavior and his mental state. Right now El Salvador is getting smoked by harsh weather NOAA/NASA/Bukele did not expected. This is why I am making my predictions to you right now. Right now people are getting more sick and dying who should not. I know why. This blog has those answers. ROS is out of control in living things. Few see what I see.
What was buried in the CT #6 blog? How quickly solar activity rises is an indicator on how strong the solar cycle will be and how diseases states will react. Share this uncommon adivce with your loved ones.
There has been some recent news out about folate levels, sunlight and artificial light. Many people do not know folate is a photosynthetic chemical and it links dark skin (melanin) for natural folate protection.
We have clear evidence (Cite 1) that ultraviolet radiation affects directly in proportion to folate human blood. Seasonal cycles repeat annually and light stability is more common at low latitudes. Therefore, the amount of photosynthetic chemicals we use acts as a photosensitizer to our chromophore proteins. This has big implications inside the tropics. The percentage of low folate values increases in summer by almost 3.5 percent in comparison to winter. Moreover, geneder differences are huge. Overall folate levels are lower in men as compared to women, regardless of seasonality. This makes sense because of who has the children.
Vitamin B9 (Folate) was first extracted from spinach leaves in 1941. The term folate comes from the same root as foliage: green and leafy. Folates are vital: they accept carbon atoms and pass them on as needed as the fundamental basis for proper methylation. This implies that during summer months folate levels should be expected to be at their lowest levels NATURALLY.
Folate biology has a specific and tight seasonal control mechanism linked to light. This is not a food story at all
What if there is no seasonal or light controls in place?
Folate is often given to pregnant women to prevent spinal dysraphisms today. This is something pediatric neurosurgeons deal with and why I know a lot about them. The incidence of these conditions has dropped but it is has done at a COST. Why? but too much folate/folic acid causes cognitive haze and sleep difficulty and may cause neural migration problems in artificially lit environments. This should awaken you what I wrote in Quantum Engineering #45.. In North America, (CAN/USA) folic acid was added to all grains in 1996. This is only a few decades ago and we are seeing the transgenerational effects of this right now in our children’s disease phenotypes.
In general, it does not appear that even large amounts of folic acid taken orally are acutely toxic in adults. However, given the fundamental role of folate levels in synthesizing nucleotides (including RNA and DNA) and in methylation reactions as a methyl donor, high levels may have inadvertent implications for proper methylation of DNA during times of rapid cell division, such as in prenatal development. You’d think the idea that adding folic acid to the food supply might have caused centralized medicine to expect unintended consequences. Few thought about, much less studied it. I have always been worried about this effect since I learned that B12 was a photoreceptor in humans. (slide from Vermont)
This idea has been speculated in research circles as early as 2005, and specifically speculated to be relevant for the increase in autism in 2011. MOST PEOPLE WITH MTHFR DEFECTS ARE SUPERSENSITIVE TO LIGHT VARIATIONS
An early review of potential problems with mass folic acid supplementation of the food supply was undertaken by Lucock and Yates. Here, they noted that a drastic increase in folates could lead to a selection for the previously rare MTHFR genetic substitution of T for C at area 677 (MTHFR C677T), and that if folic acid is supplemented at doses above 400 mcg that unmetabolized folic acid will circulate in the blood supply at a level largely consistent with the excess dose. In 2005, Lucock and Yates noted that high levels of folic acid in the blood does not generally occur as a result of ingesting natural folates and that “no work has been done so far to evaluate the biological and genetic consequences of excess long term exposure” to these circulating folic acids on DNA/RNA biology. After that review, there were two separate findings of unexpected increases in asthma and breathing problems associated with folic acid use.
It now appears that we have clear data that excessive methyl donor transfer has significant epigenetic effects in humans. This work dovetailed with another review questioning the wisdom of mass folic acid supplementation published in 1996. Smith et al. pointed out that by supplementing the food supply; several hundred thousands of persons are exposed to greatly increased levels of folic acid. These authors noted that prior research had shown that expectant mothers with low vitamin B-12 (most vegans/vegetarians) AND high levels of folic acid were associated with offspring having an unexpected increased risk for insulin resistance and disease associated with this condition. This is worrisome when you know that blue light exposure does the same thing and more.
Troen et al. found that some women past childbearing age subjected to high folic acid supplementation may be at risk for reduced immune system functioning causing inflammatory autoimmune conditions to spike. This problem has gotten worse since I first looked into it in 2005.
Did you know mammals exhibit genomic instability under conditions of folate deficiency in the skin. Remmber your skin is a neuroectodermal derivative. A lack of folate adversely effects your skin’s cellular capacity to handle UVR light. This is why so make pale gingers burn so fast. Moreover, did you know that optimizing folate levels in skin is beneficial in preventing or repairing the pro-carcinogenic effects of UVR exposure? Folate restriction by any modern excuse leads to rapid depletion of intracellular reduced folates resulting in S-phase growth arrest. Did you know this leads to ncreased levels of inherent DNA damage, and it causes increased uracil misincorporation into DNA. This is called a frame shift mutation and it is how light can cause transepigenetic signaling. This frameshift mutation will not cause a significant loss in overall cellular viability. It is how mammals adapt to changing seasonal light environments. Folate depleted keratinocytes can be sensitized toward UVR induced apoptosis. This changes the biophoton spectra emission from mtDNA and this displays a diminished capacity to remove DNA breaks. This allows for changes in phenotype. This occurs by photo and oxidative DNA alterations. Your dermatologist likes to call this damage, but Mother Nature uses this for seasonal adaptation in your skin. When this occurs more UVR = more melanin production. This protects your folate stores from degrading. Nature is amazing when you see her recipes. Dermatolgyis dangerous because they do not understand what she is doing on your behalf. Thus, folate deficiency creates a “permissive environment for genomic instability to allow for seasonal change. It is not an early event in the process of skin carcinogenesis or autism. If one abuses the use of folic acid and blue light than you can and will get diseases. The effects of folate restriction, even in severely depleted, growth-arrested keratinocytes, were reversible by repletion with folate foods. In summertime, foods with folate are plentiful in the tropics for this reason. Photosynethesis always has our cells backs.
FOLIC ACID SUPPLEMENTS EXACERBATE MTHFR DEFECTS
Methylenetetrahydrofolate reductase (MTHFR) is an enzyme encoded by the MTHFR gene and has significant implications in the field of decentralized medicine. This enzyme plays a critical role in the folate metabolism pathway, which is integral for processes like DNA synthesis and repair, as well as epigenetic alterations via methylation by light variation. Variants of the MTHFR gene are associated with altered enzyme activity, which can, in turn, affect mtDNA heteroplasmy and disease phenotypes. This happens by alterations created in the spectra of biophotons made via metabolism. You’ll hear more about that soon enough.
Increased consumption of folic acid is prevalent in our modern world, and has negative consequences for MTHFR patients.. The effects of folic acid on the liver, the primary organ for folate metabolism, are now being unfolded. Methylenetetrahydrofolate reductase (MTHFR) provides methyl donors for S-adenosylmethionine (SAM) synthesis and methylation reactions.
New data now suggests that high folic acid consumption reduces MTHFR protein and activity levels, creating a pseudo-MTHFR deficiency in the liver and skin of mammals. This deficiency results in hepatocyte and keritinocyte degeneration in both organs, suggesting a 2-hit mechanism whereby mutant hepatocytes cannot accommodate the lipid disturbances (how fatty acid carbon lenghts are dealt with and how melanin operates in the skin) and altered membrane integrity arising from changes in phospholipid/lipid metabolism. People forget the skin biology needs optimal light to control the lipid rafts in the skin as seasons change. Folic acid destroys this ability. This data has clinical implications for individuals consuming high-dose folic acid supplements, particularly those who are MTHFR deficient.
WHAT ABOUT CYP VARIANTS?
CYP enzymes are heme based and subject to blue light toxicity. They have been identified in all kingdoms of life: animals, plants, fungi, protists, bacteria, archaea, and even in viruses. This is why so many astronauts have viral particles in the blood and space is known to foster cataracts and protein folding issues in the eye. However, they are not omnipresent in all bacteria; So space blood infections vary compared to the ones we see on Earth. More than 50,000 distinct CYP proteins are known to man.
Most CYPs require a protein partner to deliver one or more electrons to reduce the iron (and eventually molecular oxygen). Remember electrons must be excited by sunlight to use the photoelectric effect in the photon traps in aromatic amino acids. Based on the nature of the electron transfer proteins, CYPs can be classified into several groups: I covered this with Rohan during a past Sunday Q & A that lasted 5 hours. Members should listen back to those recorded Q & As to refresh their minds.
CYB5R/cyb5/P450 systems, in which both electrons required by the CYP come from cytochrome b5.
FMN/Fd/P450 systems, originally found in Rhodococcus species, in which an FMN-domain-containing reductase is fused to the CYP. (cytochrome 2 in humans is related to this system.
P450-only systems, which do not require external reducing power. Notable ones include thromboxane synthase (CYP5) for platelets, prostacyclin synthase (CYP8) for PG synthesis, and CYP74A (allene oxide synthase). You should begin to see just how we are beings of light who need solar programming by sunlight and not man-made light.
Anything transferring electrons to proteins = a semiconductive circuit. When you understand the photoelectric effect only works with photons and electrons you begin to see why SNP status are mostly superfluous and generally do not matter when you’re in the sun chronically and you have melanin in your integument and interior properly renovated.
Melanin operates in us to charge separate water into its components of H+ and oxygen and 2 -4 electrons. The level of oxygen dissolved in cells is critical in varying the ultraweak biophoton signature from metaboilsm. Those two electrons liberated from charge separation obviate the need for exogenous and endogenous sources of electrons (grounding/food). SNPs evolved because mammals varying in how they ground and what they eat based on latitude and what photosynethesis can provide. SNPs and SAPs tell vary in how melanin biology is operating. They are like equalzer buttons for music made in cells.
People with a lack of melanin think SNPs matter way more than they do because functional medicine MDs tell them this. This is incorrect. Pale people without enough melanin lack electrons to run their semiconductive circuits. You’d be wise to avoid that level of centralized thinking in the future. Folic acid lowers electrons = lowers your redox power.
How humans operate is specific to humans and not to other mammals. Most of the BH4 and Vitamin C used as cofactors deliver electrons to the biochemicals. BH4 and Vitamin C deliver the exogenous sources. Melanin delivers the endogenous source of electrons. Humans create massive amounts of electrons when POMC is operational in their tissues. Here you see Noether’s thereom show up yet again.
THE SUN IS THE BEST SOLUTION TO THIS PROBLEM
SUNLIGHT reduces all these risks, while modern lighting exacerbates it. Moreover, it appears nature is trying to tell us that the sun raises melanin and melanin is protective. Strong solar cycles in photosynthesis seem to simultaneously lower folate in foods in the summertime for a deep reason. That reason is epigenetic hypermethylation which can lead to sleep apnea and cancer formations later in life due to altered DNA methyl marking. This process also alters how RBC circadian cycles (ferrodoxin) can work within their circadian cycles with the innate immune system and TOLL receptors. Most people with anemias have this intrinsic problem linked to modern behaviors around light.
Folate is destroyed by strong sunlight with both UVA and UVC light. Darkened skin protects the stores we have, but there is now proof that folate levels are designed to be low when the solar radiation is strong in the local environment. These days most people are eating food humans have been engineered and/or genetically altered in some way. Then add in the effect of Artifiical light day and night. This throws off the normal variation of the natural folate cycle during seasons. Today, people in developed countries are getting MASSIVE amounts of folates in the form of folic acid. This is the real reason to avoid grains. Folates are now being ingested in three ways: as natural folates from food, as synthetic folic acid added to processed grains and synthetic from vitamin supplements. All of these idea are counter evolutionary to Nature’s laws.
SUMMARY
Methylation patterns are linked to how light is transformed in a cell. When sunlight is absent for any reason mitochondrial redox drops. When this happens energy transformation drops. As a result of a lack of energy methylation problems shows up in both genomes. Decentralized clinicians know what to look for. Allopathic and function docs would know what to look for because they still have no idea that light controls the enzymatic flux in metabolic pathways.
Loss of mitochondrial redox power causes methylation problems to manifest in your labs without any SAP/SNP issues present in your MTHFR survey or nuclear genome. Very few clinicians know a lack of sunlight causes methylation defects.
It shows up in your labs in a very specific pattern of results as an accumulation of methionine and S-adenosylhomocysteine, with low or low-normal levels of S-adenosylmethionine (SAM) and homocysteine. PBM treatment fixes it. Sunlight is always the best treatment. Supplements not so good.
As a result of the supplementation of folic acid, the circulating level of unmetabolized folic acid, as well as total folates, has greatly increased over the past generation, probably to levels largely unprecedented in human history.
Folic acid has been shown to be able to epigenetically alter the functioning of the genome and to have long term effects on gene expression as I mentioned above.
The Centers for Disease Control Vaccine Safety Datalink data set compared children with autism to control children on several variables. Many people who think the link of vaccines to autism might be shocked to find out that folic acid supplementation during gestation is associated with a serious increased risk for autism. I believe the combination of artificial light and folic acid supplementation are causing neural migration problems. This is why parental melanin levels are important clues. This effect remains even when health-seeking behaviors and other variables are controlled. This is information parents of kids with AUTISM need to know. Autism, asthma, allergy, ectopy, eczema, diabetes T1D, T2D, and MODY, auto-immunity, and spinal abnormalities have their lowest incidence is lowest in equatorial environments and it appears now we know why this is the case. This is decentralized medicine 101 I will bring to El Salvador.
CITES
1. Valencia-Vera E, Aguilera J, Cobos A, Bernabó JL, Pérez-Valero V, Herrera-Ceballos E.. ‘Association between seasonal serum folate levels and ultraviolet radiation’. ‘J Photochem Photobiol B’. 2019 Jan;190:66-71
The circadian system orchestrates the temporal organization of many aspects of physiology, including metabolism, in synchrony with the 24 hr rotation of the Earth. Like the metabolic system, the circadian system is a complex feedback network that involves interactions between the central nervous system and peripheral tissues. Massive amounts of emerging evidence suggests that circadian regulation is critically linked to metabolic homeostasis and that dysregulation of circadian rhythms can contribute to all diseases. Conversely, metabolic signals feed back into the circadian system, modulate by applying what circadian gene expression is based on the light sensing from the environment. This alters behavior and disease phenotype. A loss of circadian periodicity causes metabolic derangement. Light controls food at all levels.
The podcast above was all about the treatment of idiopathic intracranial hypertension that manifests with optic nerve swelling, visual changes due to retinal blood vessel defects, and headache. You might be surprised to know that idiopathic intracranial hypertension (IIH) is also associated with tinnitus. That should stop you dead in your tracks if you are black swan mitochondria considering my recent podcasts and blogs here. You now know that your cochlea is lined with melanin. In fact, every sense organ is between the environment and the brain.
CPC stands for a clinico-pathologic conference that we often see in medical schools that are used for teaching students and doctors.
Today you are going to medical school, albeit, a decentralized medical school lesson how why light trumps food.
What did the centralized podcast lesson above miss? Melanopsin has been found as the dominant opsin in the human brain but it is also found in blood vessels of the brain and is critical in relaxing these blood vessels. What happens when there is melanopsin damage to the vessels of the retina and brain when there is associated melanopsin damage? Do blood vessels relax or do they become stenotic? Might this be what the group of endovascular neurosurgeons missed above?
I think so.
What happens when melanopsin damage occurs? The weak covalent bond between it and vitamin is broken and Vitamin A is liberated. The freed vitamin A destroys all the photoreceptors like heme proteins, nitric oxide, B12, monoamine oxidase, glutathione, and many others. It ruins the ability of cells to use light to signal to and fro. Fidelity of signaling is destroyed.
Vitamin A is the first discovered fat-soluble vitamin and is primarily found in animal products or converted from dietary carotenoids in plant products. It is not a single compound but a group of derivatives including retinol, retinal, retinoic acid (RA), and some carotenoids according to different terminal functional groups. In general, vitamin A refers to retinol, while retinoid refers to a general term that includes vitamin A metabolites and compounds and exhibits vitamin A-like biological activity.
Preformed vitamin A (usually from animal products) and provitamin A (including beta-carotene, usually from plant-derived food) are the two forms of vitamin A in the human diet. After being absorbed in the intestines, these two forms of vitamin A are converted to retinol and then oxidized to form retinal and RA to support the biological functions of vitamin A. The retinyl ester is the storage form of vitamin A in the liver and must be converted to retinol before being utilized, and these vitamin A derivatives are finally metabolized by the CYP26 family enzyme.
The metabolic barrier provided by CYP26 enzymes in various tissues such as the testes likely ensures that RA gradients are regulated by enzyme expression and activity within the tissue and not by circulating concentrations. Therefore, understanding the destruction, activity, and expression of CYP26 enzymes in individual tissues and cell types is critically important for defining the relationship between all trans RA concentrations and biological outcomes within a tissue. This means retinoic acids also control all sexual behavior and fertility as well. Today we have record rates of infertility and transgenerderism. The link both are linked to the light humans live under. Few see this links.
Previous studies have shown that retinoic acid (RA), the bioactive form of vitamin A, is involved in the regulation of various intracellular responses related to biological rhythms. RA is reported to affect the circadian rhythm by binding to RA receptors, such as receptors in the circadian feedback loops in the mammalian suprachiasmatic nucleus.
Many people wrongly believe solar light cycles are tied to Vitamin D and its receptor in the skin, eye, and brain. Few know about vitamin A and how it controls the human photoperiod in our brains where our POMC neurons exist. It is the major player in determining photoperiodicity in the human brain because of how Vitamin A is covalently bonded WEAKLY to melanopsin. When blue light dominates an environment humans lost their photoperiodicity and develop sleep problems.
Photoperiodicity is accounted for by biophysical changes in Vitamin A in the brain. It accurately mirrors the changes in the brain and the body that occur between the seasons when you are a careful observer.
At the equator, light doesn’t vary at any time of the year. As latitude rises light variation increases. Your body responds to every degree of latitude change via Vitamin A biology and POMC translation. Read that again.
When comparing the effects of the short days that occur in winter with the long days that occur in the summer Vitamin A (retinoic acid) swings in massive amounts. Researchers are finally beginning to understand how the brain converts the electromagnetic signal of light, first to an electric message (DC current), and then to a chemical one in the neuron synapse called a neurotransmitter. Vitamin A entangles the brain to the skin with its cousin Vitamin D. It is also critical in ephaptic communication I mentioned it to Dr. Huberman on Twitter and he brought it up during the Rubin podcast. Ephaptic signaling refers to extracellular signals generated by either a single neuron or a population of neurons, and the neurons need not be in physical contact. This likely has a quantum mechanical basis linked to coherence.
SUMMARY
Hypothalamic tanycytes are chemosensitive glial cells that contact the cerebrospinal fluid in the third ventricle and send processes into the hypothalamic parenchyma. Hypothalamic tanycytes sense vitamin A levels in the CSF. Did you know this?
We know that there are big changes between seasonal conditions on the planet even at the equator, and that seems to be how the wild animal controls weight gain and energy balance naturally in the environment. It also explains why obese women with IIH have massive alterations of Vitamin A too. Modern humans tend to gain weight in winter, and this is likely a new phenomenon related to their light environments. This is when they should be losing weight according to Nature’s laws. If you open any newspaper in January in the northern hemisphere, you will see tons of ads for New Year’s resolutions and gym memberships at this time to humans lose weight. This is unusual when you consider that wild animals do the opposite in January. Wild animals tend to get fatter going into the summer, and leaner into the winter when the light cycle is lowest and food is more sparse.
We now know that the human brain can sense retinoids, and regulate whole-body retinoid balance. This is how seasonal; light changes and local light changes sculpt the human ectoderm.
The reason becomes quite obvious when you consider that wild animals live by the dictums of their environment, but modern humans create their own environment via culture and socialization. This creation of their own environments destroys their photoperiodicity and dramatically alters Vitamin A signaling in the brain. Vitamin A is crucial in properly regulating the clocks tied to the hypothalamus that controls appetite, feeding, and energy balance. This is how quantum time is altered.
Today researchers have found that between winter and summer, retinoic acid changes dramatically in all mammals. There is a lot more to this ‘quantum dance’ of Vitamin A too. It appears there is much more powerful retinoic acid signaling during the periods of summer compared to the short days of winter. This implies that Vitamin A levels in the brain must be correctly tied to the seasonal alterations in ‘adiposity’ and depression result. Vitamin D gets all the press in the media, but Vitamin A control in your brain is way more important seasonally because of how it links to the POMC biology of melanin inside your body. It seems counterintuitive until you understand how QED works in the brain.
Larger size = more entropy in the PLL and disc = loss of circadian timing which leads to less energy transformation in the tissue degenerating.
The same thing happens in heart failure. The heart gets larger.
The same thing happens in stars. Star gets larger when they die.
The same thing happens in an ankle sprain. Ankles sprains lead to swelling.
What is the embryological origin of the spinal nerve like the one pictured above?
The ectoderm is also sub-specialized to form the neural ectoderm, which gives rise to the neural tube and neural crest, which subsequently give rise to the brain, spinal cord, and peripheral nerves. It also gives rise to neural crest cells that create melanin in the spinal nerve.
The melanocyte lineage is derived from the neural crest, which has its origins in the neural tube. Following its formation, neural crest cells delaminate from the dorsal-most aspect of the neural tube by a process of epithelial-to-mesenchymal transition. The neural crest delaminates from the developing neural tube and overlying ectoderm early in development. The pigment cells are the only derivative to migrate along the dorso-lateral pathway. This should tell spine surgeons that most diseases of spinal nerves that are associated with sensory findings are telling us precisely where our patients have lost melanin endogenously. This is why I make my cervical and thoracic disc patients remove their shirts to examine their skin. I am looking for signs of melanin destruction, lack of electrical conductivity, and count the number of nevus present in the skin of the affect nerve roots. I am also looking for evidence of melatonin and melanopsin disruption in the same dermatome. I also look for abnormal cholesterol deposits.
Mitochondria are dissipative structures in cells, but not the only ones. They transform energy and create order from the disorder in light energy they use to operate. The water mitochondria create is probably the single most important dissipative structure that life is based upon in cells. So I always look for signs of dehydration in the skin in the dermatome in question. I look at capillary refill and skin turgor.
I also shine a red light on the skin to see the response of the underlying blood vessels. you can see the red light above penetrates deeply into the dermatome.
After that test is done, then I cover the area for a brief time and shine a UV light on the same patch of skin and see how it effects the underlying blood vessels on the surface. If the become very visible, this tells me a a great deal about the NO reserve in this dermatome. There usually is a big difference in the etiology of disc herniations in this test. In acute injury the disc disruption is from biomechanical failure and no from photochemical degradation related to melanin loss. This tells me the status of NO production at this dermatomal level along with the melanin issues present in this nerve distribution.
Veins appear blue/green because these colors have shorter wavelengths that scatter more than red light. This scattering of shorter wavelengths is the same reason the sky is blue or eyes are blue. Your body does not produce blue pigment. Instead, near infrared light enters deep into your skin, hits your blood vessels, and is scattered back to you with blue being most visible and red largely being absorbed. So the color you see is blue, not red. You’ll notice this does not happen when blood vessels are closer to the surface (e.g. veins in your eyes, or skin when you blush)
Doctors and nurses use NIR light to find veins in patients. VIDEO
In difficult cases I will put a tourniquet in the limb in question and make blood pool and then do a prick of the dermatome to check for the HbA1C in that distribution. I call this my melanopsin and melatonin effect. Blue light and nnEMF exposures in dermatomes chronically induce melatonin suppression and this disrupts the circadian-regulated mechanism in the limb. This circadian damage leads to hyperglycemia and hyperinsulinemia in the body part that causes premature aging and higher heteroplasmy. These limbs often have many dangerous looking skin lesion in this.
The C4-5 disc herniation pictured above tell us a story about melanin in the C4-5 neural distribution. There was a lack of melanin in the skin of this dermatome. C4 provides sensation for parts of your neck, shoulders and upper arms. This dermatome is usually covered in humans who wear shirts. Cervical nerve 5 controls the deltoid muscles of your shoulders and your biceps. C5 provides sensation to the upper part of your upper arm down to your elbow.
In the developing embryo, dermatomes arise from somitic mesoderm, which develops from the middle layer of embryonic tissue lateral to the developing neural tube. Dermatomes are arranged with basic segmental pattern in the vertebrate trunk, although some overlap exists with similar areas above and below.
SUMMARY
This Tweeter thought he was being wise ass when he responded in the thread by saying this:
You cannot tell whether a man is clever by his answers, but you do get a great sense whether a man is wise by his questions.
The business of decentralized medicine is to teach patients to live with some uncertainty. It is not to reassure them, but to upset them. It sounds counterintuitive until you understand the perspective. This perspective is built when you watch a great white shark attack. As the shark digs its teeth into the carcass of a dead whale his teeth are replaced and his bite gets sharper. This allows the shark to get to the liver and heart of the dead animal which in turn provides massive benefits to the shark. When you dig your teeth into your own assumptions, your teeth, too become sharper. Your mind allows you to dig deeper into a subject. You become what the world needs simply by helping yourself. Questions open a space in your mind that allow better answers to germinate and connect with other ideas like turning a field over.
The status quo is the opiate of the mind. The status quo is driven by seeking comfort, convenience, and the desire for stability, and a steadfast refusal to embrace the suck that life brings to us. It is brought us by design. We need to learn from our failures. Seeking stability leads to complacency and stagnation. You become a fool when you stop asking questions. Always question before you comply with anyone or anything.
For centralized thinkers, the status quo is a powerful force that stifles innovation, creativity, and critical thinking. The master key of wisdom is a persistent and frequent questioning of the status quo. Our mind opens and awaken by embracing stress because it turns on the light inside of us when everything outside feels dimmed. Become facile asking question where the answer makes a difference to society.
As physicians, it is our job to question the rules in existence and those being enforced on our patients; we are the ones that must ask if they are relevant or outdated, necessary or arbitrary, helpful or oppressive to the truth. Without excellent questions no human progress is possible.
Thank you Crytpoattache for being a douchebag. You stimulated me to create a lesson for my tribe. Always embrace the suck folks. You’ll never know where it will lead you.
What if I was to tell you the two most important biomolecules that do this is dopamine and melatonin, would you believe it? Dopamine derives itself from the breakdown of melanin. Melanin can be made accretively from dopamine as well. This allows life to experience the world as it is not. It provides cells a new lens, a new perspective of what life might be like when additional energy is added to the mix. Ironically, a loss of oxygen is how melanin becomes dopamine. When you turn your attention away from reality, dopamine jumps to action. It allows you to “move beyond the concrete”, to a realm that doesn’t yet exist. It motivates you to pursue, to control, and to possess a universe beyond your immediate grasp.
To make large collections of semiconductive proteins like dopamine and melatonin quantum coherent you need to link them together electrically. Melanin does this for mammals. Melanin in your skin and neuroectoderm conduct DC electricity. This is what links these biomolecules.
Melanin is the master semiconductive protein in mammals. Dopamine can be the child of melanin degradation. But melanin can be made accretively from dopamine as well. It is a bidirectional pathway in mammals. Dopamine can be made many ways by mammals. This neurotransmitter is often referred to as the “reward molecule”. From my vantage point, life in the primate clade is based around the thrill of the chase, the anticipation of something new, and the excitement of getting something that’s novel and unexpected. It is what really gets our molecules buzzing.
IS MITOCHONDRIA: METABOLISM LINKED TO MELANIN RENOVATION ENDOGENOUSLY?
Dopamine and melatonin have to be linked electrically to become coherent. This tells us that there should be a deep tie in mitchondrial metabolism and the electrical coupling of dopamine and melatonin. What is it?
Amano et al. (cite 4) have provided a theoretical framework that demonstrates that resting tremor and other motor behaviors seen in Parkinson’s Disease are actually metabolically energy efficient. They posit that the role of dopamine, which can be a precursor to the formation of neuromelanin, and energy metabolism in the brain is linked and supported this assertion with research finding that “dopamine lesions result in reduced glucose uptake,” showing a preservation of energy, and dopamine is related to glucose metabolism. They conclude, “the loss of dopamine neurons in Parkinson’s Disease is likely to contribute to dysfunctional glucose metabolism.” Ironically none of them have made the link to why red light lowers blood glucose by 27% nor why when melanin is missing in cells endogenously, cells lose their ROS generation power from mitochondrial metabolism.
It appears that ROS and a lack of redlight production in mitochondria maybe the key link in diseases associated with altered glucose metabolism. They also note the growing amount of literature arguing mitochondrial dysfunction is common in Parkinson’s Disease and is causing metabolic dysfunction. They went on to say that they have discovered that there is an inverse relationship between melanin levels and mitochondrial ATP production. In fact, it appeared to them that melanin may hold the primary supply of energy while mitochondria produce supplemental energy, in an opposing, but complimentary, interdependent relationship.
BACK TO SUPERPOSITION OF DOPAMINE. MELATONIN, AND MELANIN
In quantum science, objects such as electrons and photons have wavelike properties that can combine and become what is called superposed. Particles are not the only thing that can be superposed. So can whole atoms. Did you know that this ability in quantum mechanics is not limited to just atoms either.
Complex molecules can be superposed.
Many have heard about many world interpretations verison of quantum mechanics. Very few have heard about many world chemicals that are capable of staying in superposition to deliver different possibilities and outcomes in Nature.
Physicists have now proven any chunk of matter can also occupy two places at once. Physicists call this phenomenon “quantum superposition,” and for decades, they have demonstrated it using small particles. But in recent years, physicists have scaled up their experiments, demonstrating quantum superposition using larger and larger particles.
The double-slit experiment reveals the central puzzles of the decentralized systems in quantum mechanics, putting us ‘up against the paradoxes and mysteries and peculiarities of nature”.
Researchers had long known that light, fired through a sheet with two slits in it, would create an interference pattern, or a series of light and dark fringes, on the wall behind the sheet. But light was understood as a massless wave, not something made of particles, so this wasn’t surprising. However, in a series of famous experiments in the 1920s, physicists showed that electrons fired through thin films or crystals would behave in a similar way, forming patterns like light does on the wall behind the diffracting material.
If electrons were simply particles, and so could occupy only one point in space at a time, they would form two strips, roughly the shape of the slits, on the wall behind the film or crystal. But instead, the electrons hit that wall in complex patterns suggesting the electrons had interfered with themselves . That is a telltale sign of a wave; in some spots, the peaks of the waves coincide, creating brighter regions, while in other spots, the peaks coincide with troughs, so the two cancel each other out and create a dark region. Because physicists already knew that electrons had mass and were definitely particles, the experiment showed that matter acts both as individual particles and as waves.
But it’s one thing to create an interference pattern with electrons. Doing it with giant molecules is a lot trickier. Bigger molecules have less-easily detected waves, because more massive objects have shorter wavelengths that can lead to barely-perceptible interference patterns. And these 2,000-atom particles have wavelengths smaller than the diameter of a single hydrogen atom, so their interference pattern is much less dramatic.
To pull off the double-slit experiment for big things, the researchers built a machine that could fire a beam of molecules (hulking things called “oligo-tetraphenylporphyrins enriched with fluoroalkylsulfanyl chains,” some more than 25,000 times the mass of a simple hydrogen atom) through a series of grates and sheets bearing multiple slits. Recall cells are filled with porphyrins like the two below.
In the experiment in Cite #1, the beam was about 6.5 feet long. That’s big enough that the researchers had to account for factors like gravity and the rotation of the Earth in designing the beam emitter. They also kept the molecules fairly warm for a quantum physics experiment, so they had to account for heat jostling the particles.
When the researchers switched the machine on, the detectors at the far end of the beam revealed an interference pattern like we see with electrons. In fact, the molecules being studied were clearly occupying multiple points in space at once. This means large biomolecules can and do act like electrons do. They do have a superposed ability.
It’s an exciting result, for quantum biology, proving quantum interference at larger scales is possible. This was the first time in history this has been detected.
SUMMARY
The implications of this endogenous ability in chemicals is the basis of how MOLECULAR RESONACE operates. Molecular resonance is a quantum mechanical characteristic of all matter. This ABILITY is inherently BUILT into how REALITY is perceived. It is buried inside of the chemicals that cells have chosen to use through evolutionary timescales.
The most important biomolecules are coded for by DNA. DNA seems to favor biomolecules that have specific semiconductive and optical characteristics in their absorption and emission spectra. The design process of cellular life begins with the DNA code. Centralized biology today is a detailed, disorganized collection of disparate facts. It is like a hoarder’s basement, or a rat’s nest. There is no decentralized connecting design of what is buried in DNA’s code. You can scoop up a bag full of facts and try to make sense of it, but that would be an exercise in futility. True wisdom is fractal and non linear. The design can be complex, with microscopic details, but the overall design is coherent and beautiful. To make large collections of semiconductive proteins quantum coherent you need to link them together electrically.
We do this PHOTOELECTRICALLY. This idea implies that cells have some electric tuning ability built into their protein structure.
It appears the choice is related to the spectrum of light that interacts with them. Possibilities and probabilities for life is made tunable just by changing the incident light photons. That is how these chemicals all operate. This implies that your mitochondrial metabolism creates an adaptable light spectrum during metabolism and it is this light that tunes and controls the chemicals in you to act in different ways.
A dopamine molecule consists of a catechol structure (a benzene ring with two hydroxyl side groups) with one amine group attached via an ethyl chain. The amine part of the ring contains nitrogen. If you draw the two possible Kekulé structures for benzene (pic below), you will know that the real structure of benzene isn’t like either of them. The molecular structure acts like it is capable of being in two states.
The two structures above for benzene’s ring are known as canonical forms, and they can each be thought of as adding some knowledge to the real structure. For example, the bond drawn at the top right of the molecule is neither truly single or double, but somewhere in between. Similarly with all the other bonds. The real structure is somewhere between the two – all the bonds are identical and somewhere between single and double in character.
Benzene two structures also sit in a superposed position. That’s because of the electron delocalization in the benzene ring. The aromatic rings of carbon in benzene are the playground for bio- photons as the picture below shows. Those benzene rings capture the photons and tune it.
As such, dopamine is the simplest possible catecholamine, a family that also includes the neurotransmitters norepinephrine and epinephrine.
If we take the two forms as the picture above shows perhaps the two most important ones, it suggests that there is delocalization of the electrons over the whole structure of the 6 carbon ring. With dopamine above, that electron density is a bit low around the nitrogen atom carrying the positive charge on one canonical form or the other. Any canonical form that you draw that shows nitrogen close to the ring, it follows that another atom must balance that charge. In dopamine that atom is oxygen at the 7 and 10 o’clock positions. Where the charge change occurs changes the absorption and emission spectra of dopamine. Separating negative and positive charges in this fashion is thermodynamically unfavorable. This is how tunability occurs with charge separation in a benzene ring. Light tunes molecules by altering their charges. This is why all the aromatic amino acids have benzene rings in their molecules. This also happens in methylene blue.
HOW DO YOU CREATE YOUR INNER MASTERPIECE? YOUR CHOICES DICTATE THAT PATH
Dopamine controls the process of choice and action. Nothing EXCELLENT ever happens without EXECUTION. No masterpiece has ever been made by INTENTION alone. EXECUTION takes INITIATIVE and INTELLIGENCE. EXECUTION is the antidote to PROCRASTINATION. Ideation without execution leads to deletion of any idea. A poor idea executed accomplishes more than a great idea that stays locked away in a person’s head. This blog is exploring how the small changes in light can affect change in your brain. Actions end superposition. Actions – executions of ideas. EXECUTION calls off the fence of indecision and put us in the valley of decision; it calls us off the bench and onto the field. It does matter how much talent you have as a player in your life, but that talent is useless with inaction because you can never score a goal while you’re on the bench. That is how dopamine is the molecule of more for mammals.
This idea explains to us how dopamine can do all the things it is capable of doing without a lot of modifications we can observe biochemically. Dopamine drives you to seek out things far away, both physical, things you are blinded to, such as love, sex, wisdom, and power. Changing your ultraweal biophoton signature and shining it onto dopamine in certain circuits allows you to put hot sauce on your dinner, think about building rockets to fly to the moon, worshipping a God in the sky, beyind the space and time frames you live in. This chemical appears to have unlimited abiliities to bring to your life endless possibilities over any distance, whether that distance is intellectually or geographical. In your brain’s quantum computer, dopamine has become a single molecule that is the ultimate evolutionary handy tool. It is what moved us past Neanderthals, and began to urge us, through multiple tracts in our brains to move beyond the pleasure of just being, into exploring the cosmos of possibilities that come into focus when we imagine. Every creature on Earth has dopamine and melatonin in their cells, but no creature has more of them then humans. Madness and genius both depend on how dopamine is programmed by light. This tells you dopamine itself is capable of superposition. This idea also underpins this cliche as well. Talent hits targets no one else can hit; but genius hits targets no one else can even see.
I think this idea extends to chiral molecules in biology. DNA is made up of chiral molecules. Matter-wave diffraction patterns can put chiral molecules into superpositions of left- and right-handed forms. Experiments have already shown it and this will enabling new studies to be done of how the two states interact with their environment to give two different outcomes.
Small chiral molecules such as amino acids and sugars are the building blocks of larger molecules, such as proteins and nucleic acids, which are also chiral. A chiral molecule and its mirror image are called enantiomers; one is dextrorotatory (D) and the other is levorotatory (L). This is another way evolution likely happens that is also a break with Darwinism.
Most of you know I think this guy in the Tweet is a Twit when it comes to science. But even a blind squirrel is able to tell time correctly twice a day. Listen carefully what he says about Neanderthals and what I have told you about dopamine and creatitivity in this series already.
Semiconductive quantum dots (QDs) have been widely used for fluorescent labelling in modern experiments. However, their ability to transfer electrons and holes to biomolecules leads to spectral changes and effects on living systems that have yet to be exploited in centralized science. Cells appear to do this easily.
How do I envision how this operates in us?
Quantum Dot-dopamine conjugates can be used to label living cells in a redox-sensitive pattern: under reducing conditions, fluorescence is only seen in the cell periphery and lysosomes. As the cell becomes more oxidized, Quantum Dot labelling appears mostly in the perinuclear region of cells. This would include in or on surrounding mitochondria where biophotons are created metabolically.
With the most-oxidizing cellular conditions, Quantum dot labelling throughout the cell is seen already in experiments. Thee experiments have taught us phototoxicity results from the creation of singlet oxygen, and it can be reduced with antioxidants. Melatonin is the major antioxidant inside the cell created by mitochondria to manage this process. there are many small molecule proteins liberated from mitochondria that could do this. When you comprehend what I am proposing here this picture below should have new meaning. It shows you how reality is perceived and how it can be changed rather easily. This explains how dopamine can build creativity, madness and genius in humans and how its creation can vary in one single life to explain what happened as Jimi Henrdrix, Kurt Cobain, or Jackson Pollack aged in their lives.
The living universe selects for maximum entropy, and minimum waste heat. Melanin was critical in evolution of bringing biological complexity to the interior of mammals.
In this context, the melanin universe in mammals can and should be seen as a self-organizing system that seeks to optimize its energy use and minimize waste heat. This is reflected in the emergence of complex structures and patterns in the universe, from the formation of galaxies and stars to the development of dopamine, and life on Earth.
The universe and cells have much in common. Both are decentralized systems.
The idea that the a cell and the universe are self-organizing systems that seeks to maximize entropy and minimize waste heat has far-reaching implications for our understanding of life and the universe and our place within it. It suggests that the universe is a dynamic and ever-changing system that is constantly seeking to optimize its energy use and maximize its complexity. It also suggests that tissues, collection of cells have the same ability buried within them.
Dopamine narrates your life and it is the main story teller of how life came to be within the primate clade in evolution.
The Melanin Renovation Rx is based on Noether’s theorem, which is the key missing piece. The theory states that every differentiable symmetry of the action of a physical system with conservative forces has a corresponding conservation law. The symmetry in the mammalian physical system is based on UV light. Time symmetry implies energy conservation. So, what happens to mammals when they lose energy? They lose time symmetry. In other words, time flows differently backward and forward. There was a time on early Earth that the TCA cycle only flowed counterclockwise because there was not a lot of oxygen or as much UV light around.
It sounds incompressible and unimportant until I remind you of what you just learned about the TCA cycle in the last few blogs. In modern times, biochemists are familiar with it running forward clockwise with plentiful oxygen and sunlight is pumped into the system every AM, and it rotates counterclockwise when these conditions are not met. The implications of this rotational symmetry change are enormous for you understanding. Why? Your time experience varies depending on the direction of the spin of this cycle in your cells. Sounds crazy doesn’t it?
Theories of criticality in physics have been successfully applied to biology. The mathematical core of these theories rests upon the idea that a “phase transition,” which can be either critical or not, may be described as a point along the line where the intended control parameter runs. For example, the ferromagnetic/paramagnetic transition of iron and oxygen takes place for a precise temperature value, the Curie temperature.
Remember that light and temperature control the entropy flow in your circadian clock mechanism. When you realize this, you might begin to see how people experience time differently in their lives
If you paid close attention to the video above, you’d have heard that energy conservation implies time symmetry. It also follows that anytime there is a violation of time symmetry, the energy conservation of the system has been violated in some fashion. This happens when mtDNA is altered, melanin is lost, or water is not created in cells from metabolism. All of these things change oxygen tensions inside of cells as a result. All these things cause our cells to liberate more light than we should. This light liberation changes the AMO physics in mitochondria. That is what the ROS and RNS signal tells the decentralized clinician. These ideas are all buried in the slide below.
What facts about Nature isn’t in the slide above? Most centralized scientist believe ROS is associated negatively with cell function. It just is not true. ROS is not inherently negative for cell function; instead, it plays a complex role in regulating various cellular QUANTUM processes that centralized biology has not stumbled upon yet. The interplay between ROS, endogenous biophoton creation, and low intensity laser creation inside of cells offers opportunities for the centralized audience to rethink their beliefs and approaches to understanding cell biology. Most of centralized science has no idea that cellular conditions involving elevated ROS production (chronic diseases like diabetes) are associated with increased low-intensity light therapy sensitivity. The reason is quite simple. Diabetic tissues have lost most of the sotred photonic energy at the electronic and vibrational levels in cells. Diabetic cells emit higher amounts of biophotons to their environment in an attempt to raise their own ROS to actually stimulate higher ATP production. The problem is oxygen tensions in diabetics is low, along with biophoton power and intensity so that endogenous light production for regeneration becomes improbable. The detection of biophotons is now well established in the biophysical literature, is also associated with the production of ROS and the cellular redox state. Low intensity light therapy via PBM which mimics what red light from the solar spectrum normally gives the semiconductors in cells. This light also has the potential to induce cellular effects through accelerated ATP production. This paper here: HYPERLINK should blow your socks off. When I read it in 2010 I was stunned.
Humans no longer live in the light this mechanism is designed to work with. This is a big part of why we have chronic disease epidemics that we do today. Few see how light is the driver of the entire process. By the time this blog is done, I think you will have enough data to show you why I am adamant that light trumps food at all the most critical levels in cells.
WHY DOES COLD HELP MAMMALS ALSO CONSERVE TIME?
When the temperature decreases and passes through that point, the magnetic orientation organizes along one direction, and magnetism appears. When the temperature increases through that point, disorder prevails, and magnetism disappears. Magnetism emerges from the system’s atomic-molecular organization. Think of mitochondrions as organelles that create paramagnetic chemicals (free radicals) by altering the atomic lattice of atoms fed into it. Most of the atoms fed into mitochondria are not paramagnetic. Many of the atoms that come from mitochondria have distinct magnetic fingerprints. Those fingerprints emerge from alterations of the AMO physics that are occurring inside the organelle.
Mammals’ criticality begins with UV light because it works with mtDNA, quantum dots, and melanin to create VUV-IR light inside a cell. When the critical amount of these entities goes missing inside mammals, they suffer a loss of healthspan or a loss of time. Their TCA cycle does not spend most of its time spinning in a clockwise fashion. When they get enough AM light, and sunlight during the day, they become robust and antifragile because the TCA cycle is operating as it should. When they do not, they get sick and die sooner than they should.
The TCA cycle highlights this time symmetry idea. The same biochemical pathway is used to create and destroy cells. I have told you many times that everything can and should be thought of as a clock. That is clearly how Nature views the TCA cycle when you view it from this perspective.
Very recently in this series of blogs I told you about an experiment in 2012 that showed us that the laws of physics are not time symmetric. It was buried from most people’s awareness because the experiment occurred at the same time the Higgs boson was discovered. Prior to this 2012 experiment, centralized science believed, that whether you run “a clock” forward or backward, most believed the laws of physics to be the same because of Noether’s theorem. The 2012 experiment showed us otherwise and I became quite happy because it explained to me really what the TCA cycle was doing and why mammals lose light when the TCA cycle is spinning counterclockwise. During this shift in time, we should expect a change in energy based on the relationship of conservation laws to time time symmetry. That was the point of showing you the video above. The TCA cycle is a cell’s difficulty adjustment in how it tells time. I want you to understand that when time is the currency in question, energy has to be expended and it is lost to the environment. When this happens aging manifests. This is truly how time is created in biological systems based on a proof of work mechanism.
This concept of time is fundamental to our way of thinking. It is derived from the more basic concept of the order in which events occur. Given the perspective I am trying to show you, I want you to ask yourself the following questions. Who or what should be in charge of time if putting someone in charge is not allowed in the system? How can you have a reliable clock if there is no central frame of reference? The key here is to understand in your cell, a mitochondria is the time machine that houses this TCA clock. That TCA cycle mimics a difficulty adjustment in the cell. Can I just make energy or do I have to make things I need to operate my cell? Got it? How that clock works is based on how often it senses the sunrise in its environment. I have brought this message to social media for 20 years.
AMO PHYSICS INSIDE OF THE MITOCHONDRIA IS CRITICAL
This criticality of this idea scales and corresponds to the appearance of a “coherent structure” in mammalian cells, that is, space and/or time correlations at all scales, which at the transition point (melanin renovation point) give a “global” aspect to the new physical object. These ideas are pretty relevant to analyzing quantum actions buried within biological organisms.
Melanin is critical in maintaining the AMO physical organization of the mitochondria and the functioning of the TCA cycle. Why? Melanin is only translated from its parent gene when UV light is present in the mammalian environment and or its tissue. This point cannot be magnified enough of how critical this relationship is to light. No other gene is linked to smallest part fo the visible spectrum of the sun and this same frequency of light is linked to every single biophoton cells release. It is a remarkable connection in nature that has gone unnoticed by centralized science.
Biological systems, on the other hand, exhibit criticality across multiple parameters such as genetic variations, environmental changes, and interactions with other organisms. This ongoing criticality allows for flexibility and adaptability in response to changing conditions, ensuring the survival and evolution of the organism.
Furthermore, the concept of criticality in biology extends beyond individual organisms to entire ecosystems. Ecosystems are complex networks of interacting species and environmental factors, and their stability and resilience are maintained through a delicate balance of interactions. When this balance is disrupted, it can lead to catastrophic shifts in the ecosystem, such as species extinctions or ecosystem collapse.
Overall, the concept of criticality in biology highlights the dynamic and adaptive nature of living systems, and underscores the importance of studying and understanding the complex interactions that govern biological processes. By recognizing and studying critical transitions in biological systems, we can better understand how organisms and ecosystems respond to environmental changes and potentially develop strategies for conservation and management.
Circadian control in a tissue is an ideal example of how biology builds its most important criticality. Its efficiency is correlated with mitochondrial redox power between -440meV to -200MeV. Outside this “critical” zone, life has problems. Without the maintenance of this redox zone, the clock within the time machine is altered, and mammalian health metrics fall apart.
I submit to you that both of the pictures above are showing you Nature’s recipe for time buried in living systems. The first picture you are well acquainted with if you are a scientist. The second one is for mathematicians which also shows how light time shifts mammalian longevity.
Life solves this problem by re-inventing time itself. It says no to seconds and yes to mitochondria to create its own version of biological time stamping in cells. All clocks rely on periodic processes, something that we might call a “tick.” Mitochondrial periodicity is linked to AM sunrise and this begins the “tock” that clicks in the TCA cycle. The frequency of your clock — how often it ticks — depends on its use-case with the light choices you make. Your clock is an atomic optical lattice clock.
Most clocks you are familiar with have a fixed frequency. After all, we want to know the time precisely when we are going to a meeting or a class. There are, however, clocks that have a variable frequency. Your circadian clocks are based on the variable frequencies of how the sun changes in a day. Those clocks are called metronomes. A metronome has a variable frequency that you can set before you make it tick. While a metronome keeps its pace constant once it is set. This is why AM sunlight is irreplaceable in humans. It sets the pace for the TCA cycle. This makes the TCA act like a difficulty adjust knob in the clock.
While time causality is essential in biochemical pathways, it is not sufficient to explain biological time keeping. This is why we have molecular clocks. They focus on time causality. The more counterintuitive reality of timing in the mitochondria is that they need unpredictability to work well. Why do we need two ways of handling time? It turns out we need unpredictability for time to flow for our quantum realm in cells. In the physical realm, we observe natural processes to describe the flow of time. We observe a general increase in entropy and call that the arrow of time. Even though the laws of nature seem to be oblivious in regards to the direction of this arrow in most cases, certain things can’t be undone, practically speaking. You can’t unscramble an egg, for example..
Similarly, entropy-increasing functions are required to establish an arrow of time in the quantum realm in mitochondria where subatomic particles are being used for timing. Just like it is practically impossible to unscramble an egg, it is practically impossible to unscramble the epigenetic signatures that control the genome. When timing is off in your mitochondria that is precisely what happens to your DNA and that is what causes genomic shifts in cancer.
In biology, the proof-of-work mechanism is physically linked to what happened directly with the sun that day. It is not just a record of an event — it is the event itself.
Without this controlled increase in entropy in cells, we could go forward and backward in time without consequence. Cells rely upon two sources of unpredictability: biochemical reaction times and the proof-of-work mechanism in sunlight’s energy. Just like nobody can predict what your Twitter feed will look like tomorrow, nobody can predict what the next human offspring will look like in the future either. This is what powers evolutionary change. This is how biological timestamping operates. It is not the world Darwin saw. It is a quantum mechanical process that uses light to create a ledger of time we call life. How is that for a new way to look at yourself?
SUMMARY
In a mitochondria, all we have is subatomic particles which contain data from our environment. It should follow then, that the biological realm is informational in nature, the obvious conclusion is that computation is all we have. If your world is made of data, manipulation of data is all there is. This is why Nature built a mitochondria after endosymbiosis. She needed a time machine to make complex life.
Proof-of-work works in Nature-to-cell setting because it is trustless, and it is trustless because it is disconnected from all superfluous inputs — such as the readings of clocks, food, your family’s opinions, or the narrative of social media. It relies on one thing and one thing only: computation requires work, and in our universe, work requires energy and time. The brilliant thing about cell biology’s proof-of-work mechanism is that it creates its own reality, along with its own space and time.
Einstein proved time is relative, and simultaneity is nonexistent. This fact alone makes all timestamps — especially across large distances like we have in biology — inherently unreliable. This is why the SCN needs to be time stamped every morning by AM sunlight. It is also why your iPhone needs to update its GPS coordinates to work. It also explains fully why timestamps of GPS satellites have to be adjusted constantly, by humans on the surfaces of Earth to make tech gear operate properly. For life, biological timestamping has to be very precise to maintain health. If it is not chronic disease is the result. This is why evolution built the TCA cycle to function in spontaneous fashion. See Nick Lane’s picture below. Nick still has not figured out why Nature did this.
The reason was simple. Life has to be costly in time and not in energy. The TCA cycle is the difficulty-adjustment in cells. It’s evolutionary function is about keeping a constant time in cells, not a constant level of immunity in all tissues or in energy expenditure. We know the TCA cycle varies its energy production in various tissues. Using the TCA cycle spin was an ingenious early step in evolutionary design because longevity has to be costly in time, not energy. Time is the most valuable asset in Nature because it has a fixed quantity. Energy can be sourced in many ways quantum mechanically by cells. if you are understanding this blog well now, Noether’s theorem says that energy conservation is LINKED to time symmetry. This means that every improvement in energy generation in cells over evolution would allow life to create time. Time is the only thing cells cannot make more of. They became able to build complexity because they began to harvest more energy from the environment. That is what the picture above Nick’s head really says, but Nick is yet to realize it. I hope you do now too.
Nick Lane is correct that understanding why the TCA cycle underpins life is a great mystery to be solved. I am giving you my solution in this blog. It has been in my head for 20 years. The TCA is the biological difficulty adjustment life needs because it is essential as some of the amino acids its uses.
Because, without it, the internal clock of mitochondra would tend to go faster and faster as more biochemical pathways join the biological network in cells. It would speed up biosynthesis pathways we use to create offsprings via sex. We would quickly run into the coordination problem that mitochondria was built to solve as evolution’s time machine. Cancer is this coordination problem. This is why light stress increases ATP production. ATP production is telling us something about the NAD+ to oxygen difficulty adjustment in mitochondria. Something is awry in our quantum timing mechanism. This story goes way back in the blogs. Don’t believe me? READ IT. Note the date. The ideas in that blog began in my head in 2000-2004. You likely never saw it this way before today.
By viewing living entities as extended critical transitions, we can see them as dynamic systems that continually renew and adapt their structure. This perspective allows us to understand the complexity of biological processes in a new light, recognizing that they are not just static phenomena, but rather evolving and changing systems. This can help us overcome the challenge of analyzing the intricate and nuanced nature of living matter, providing a framework for studying and understanding biology in a more interconnected way. This is the idea buried deep in the QUILT document. Ultimately, this change in perspective from physics to biology offers new insights and approaches to unraveling the mysteries of life.
There is a very common belief in humans in a divine force or energy that gives life to all living beings has been prevalent throughout human history and has shaped the way we view life and existence. It has provided a framework for understanding the purpose and meaning of life, as well as guiding moral and ethical behavior.
The concept of life as a gift from a higher power has been a source of comfort and hope for many people, offering a sense of purpose and belonging in a vast and often mysterious universe. It has also served as a basis for the belief in an afterlife or continuation of the soul after death, providing solace in the face of mortality.
While the scientific understanding of life has evolved and advanced over time, the religious and spiritual concept of life as a divine gift remains a powerful and enduring belief for many people around the world. It continues to shape our values, beliefs, and behaviors, influencing how we live our lives and how we view the world around us. Ultimately, the concept of life as a sacred and precious gift reminds us to cherish and respect all living beings, and to strive for a more harmonious and interconnected existence.
Some argue that the theory of evolution through natural selection contradicts the second law of thermodynamics, which states that entropy, or disorder, in a closed system will always increase over time. They claim that the complexity and organization seen in living organisms goes against this principle, as it suggests a decrease in entropy rather than an increase.
However, proponents of evolution point out that the Earth is not a closed system, but receives energy from the sun, allowing for the increase in complexity and organization seen in living organisms. They argue that the second law of thermodynamics applies to isolated systems, not open ones like the Earth.
Ultimately, the debate over evolution and thermodynamics continues to be a contentious issue, with both sides presenting compelling arguments. However, many scientists and scholars maintain that there is no inherent contradiction between the two concepts, and that they can be reconciled within the framework of modern scientific understanding.
As yourself this question: Is life a property of matter that does not naturally exist? Might life be a phase of matter that we invented with the help of the environment. On the most fundamental level, all matter that exists is just an arrangement of atoms and their constituent particles, is it not?
This perspective challenges the traditional notion of life as a distinct and separate concept. Instead, it suggests that life is simply a human construct used to categorize certain arrangements of matter. This mindset can lead to a greater appreciation of the interconnectedness and complexity of the universe, blurring the lines between living and non-living entities. It invites us to question our assumptions about what it means to be alive and opens up new possibilities for understanding the nature of existence.
Are mammals/humans, for instance, a bag of special water filled with 26-element human molecules that has been synthesized, by the operation of universe, over the course of the last 13.7 billion years? Or is this something more to this reductionist view point?
Living entities are not “just” processes but something more: they are lasting, extended critical transitions, always transient toward a continually renewed AMO structure. In general, physical processes do not change fundamental symmetries; to the contrary, they are primarily meant to preserve them.
Living organisms exhibit emergent properties (quantum mechanical) that cannot be fully explained or understood by simply breaking them down into their molecular components. The photomolecular effect is one example. These properties arise from the interactions and relationships between molecules and cells, and they give rise to the incredible diversity and complexity seen in the natural world.
Therefore, while it is important to recognize the fundamental chemical and molecular basis of life, it is also crucial to appreciate the complexity and emergent properties that make living organisms more than just the sum of their parts.
From Nature’s view, the world should be seen as only connections, nothing else. Everything is based on probability and nothing is based on cause and effect. This is the perspective of this decentralized quantum biologist.
Prokaryotes release 5000 time more light than eukaryotes. Eukaryotes release more light when they are stressed. Do eukaryotic cells use this excess light in some way we do not yet understand?
Yes, I believe they do. It has to do with biological chip design on the topological insulator chips inside of cells. It is a process of biological photolithography.
Manipulating the flow of light in a material at small scales is a specialty of cells loaded with hydrated carbon-based semiconductors. Why? Doing so allows for complexity development under low-power situations. In a semiconductor, electrons can, in principle, move freely, but an external magnetic field can stop this motion. The circular movement of the ATPase caused by the magnetic field stops conduction, and as such, electrons can only exist in the material if they have very specific energies. The energy level corresponds to the light frequencies that excite these electrons by the photoelectric effect. These energy levels are called Landau levels, and they are characteristics of electrons operating in a magnetic field. These forces are operating right around the mitochondria creating a force field of action that allows for quantum mechanical process life needs. You must open your eyes to new data to see the magic inside of cells.
NATURE’S MAGNETS ARE IONS EJECTED FROM THE CORE OF STARS.
This implies that all biological magnets used in mitochondria and cells come from things that make light. This is axiomatic and critical in showing how physics trumps biochemistry in controlling how life unfolds below the cell level.
Since manganese has a nuclear spin of 5/2 and a nuclear magnetic moment of 3.4687, spectral lines of Mn II show it has a hyperfine structure (HFS). Manganese has one stable isotope with a mass number of 55. The ground electronic configuration of Mn II is 3d5(6S)4s (Sansonetti & Martin 2005). This specific electronic configuration allows cells to create SOD2 from excess oxygen from mtDNA metabolism.
It reveals the queerest aspects of Nature that remain hidden from the biologists and biochemists. Manganese is not a particularly common element on Earth. It is the 12th most common element in the rocks of Earth. However, where it is concentrated reveals the quantum biological story of the evolution of prokaryotes and their lives before oxygen filled the ionosphere. The most incredible abundance of manganese is ferromanganese nodules and crusts along the ocean floor. Marine chemical processes and microorganisms (prokaryotes) capture dissolved manganese in seawater, which is precipitated on the ocean floor. Once captured, the prokaryotes could use the element to create a spectra and a magnet to drive biology using light DIRECTLY from matter.
It should immediately stimulate your neurons to recall that Prokaryotes emit 5000 times more light than eukaryotes. It would help if you remembered that prokaryotes populated our ocean long before eukaryotes ever appeared on Earth. This mechanism is ancient. It is not holistic! It is not alternative medicine. In fact, it is native biology, and it reveals that centralized science and medicine are the real alternative versions of events created by the minds of man in his labs. It is directly related to the organizational plans buried in Nature.
Shannon’s theory of Information has told us that unusual things make excellent information carriers in computing machines. The ground electronic configuration of Mn II is 3d5(6S)4s, a superb information carrier with evolutionary severe weight. Phosgene is a chemical that puts an acetyl group on the aromatic amino acid tyrosine, and this small biochemical change blocks the electronic transition and blocks the flow of information in mitochondria. It blocks the Mn redox cycling between ESR-silent Mn(III) and ESR-active Mn(II) required for superoxide dismutation. Many people do not know that ESR silent Mn(lll) is the source of many of the critical evolutionary porphyrins of life. High spin Mn(lll) has an electronic configuration 3d4(6S)4s. Nature used the D shell electronic configuration of Mn as her signal for SOD2 production. Let that level of complexity sink in while you still think biochemistry is “the main controller” in your cells.
High spin Mn(III) is the archetypal of such non-Kramers ions. Mono and polynuclear Mn(all) are of central importance in biological systems that use heme-based proteins like SOD2, catalase, and photosystem II, while Mn(III) porphyrins phthalocyanines have been used as building blocks in the construction of molecule-based magnets. This has huge implications for how life operates below the cell level. Soon you’ll learn about that level because it is not in your biology books and not in any medical textbooks. That is how vital Turin’s serendipitous finding was for the Ivory Tower. Some of us already knew biology was driven by changing the electronic configuration of ions using light.
For example life is flexible. How does QED happen during mechanical deformations in life? Generally, mechanical deformation stops conduction in semiconductors or topologic insulators like graphene; those type of materials turn into an insulator in this case and consequently the electrons become fixed in their lattice & are bound to Landau levels.
Does like act like electrons since they are linked by the photoelectric effect? Yes. A photonic crystal, like cell water, normally consists of a regular—two dimensional—pattern of holes as one would expect to see in a silicon layer on a chip. Breaking this regularity in water in exactly the right manner will deform the array and consequently lock the photons inside the cell. This is how we create Landau levels for photons.
Implications? Once you slow light down in a crystal, you can steer it to where you need it. So, increasing your metabolism increases the biophoton creation in the mitochondria. Then, cell water captures it and drives it to the biochemical boxcars. In Landau levels, light waves no longer move; they do not flow through the crystal either but stand still. This shows that the deformation of liquid crystalline water can have a similar effect on photons as a magnetic field on electrons. By altering the deformation pattern, research has shown you can establish various types of effective magnetic fields in one material. This can be used to do physiological work. As a result, photons can move through certain parts of the material but not in others. Hence, life seems to have an ability to steer light on a liquid crystalline surface.
Stopping light in its tracks to steer biophotons begins to show us how organs really function on our semiconductive chips built by Nature. The ability to stop light gives cells a new biological ability that mimics on-chip applications. This ability can explain how smells and words can go from sensations to visual imagery in working memory. It likely will explain consciousness, memory, and how holography forms in water.
Slowing light in water was a critical evolutionary step that existed before Nature invented DNA. Ferrodoxins and aromatic amino acids were in our primative oceans. How did it happen?
How do I see a quantum cell using this physics?
Although a magnetic field doesn’t DIRECTLY affect the photons of light directly, a magnet does distort the medium through which light passes and thereby “bend” the light rays indirectly. This is used inside of cells where mitochondria and melanin are in close proximity. Cells take advantage of every option Nature provides it to transform energy. When oxidative phosphorylation is defective, this adjusts the cell’s stress response like the mammalian DAMP program.
Aging isn’t driven by ROS, as doctors are taught to believe. Many now think it is driven by the DAMP Program. I believe sunlight controls all the DAMP molecules. I believe slowing light down in cells is linked to healthspan and aging. Why do I say this? Aging and cancer share some common origins and hallmarks, such as genomic instability. Sunlight exposure creates genomic stability to quiet epigenetic programming and lower protein turnover in cells. Recent advances indicate that damage-associated molecular pattern molecules (DAMPs) such as high mobility group box 1, histones, S100, and heat shock proteins play location-dependent roles inside and outside the cell.
Mitochondria under stress via the DAMP program respond by transforming more ATP to bend light to compensate for the stress. Why does it do this? Light travels through space-time along a geodesic – the shortest possible path between two points on a curved surface. Making more ATP means the ATPase spins faster than 9000 RPM and this increases the magnetic strength in the cell. This magnetic flux is used to make the stressor hormetic. It drives adaptive changes in cells by altering the epigenome.
SUMMARY
When water superconduction is broken in you due to modern life, you must rely on the rapid recycling of ATP from the pathways I mentioned in the EMF-4 blog. This means that glycolysis and the PPP will be the metabolism the cell will have to rely on when there is a proton problem. When you live in a world only powered by ATP, your sleep is the first thing to go south. Initially, it becomes poor before it totally fails, and you get diagnosed with sleep apnea. As it goes on more chronically without proper treatment, you eventually die from right-sided congestive heart disease and pulmonary hypertension. Yes, in total sleep failure, you get a specific kind of heart failure preceded by a fatal heart arrhythmia. This rhythm modern cardiology knows about but still has not made the link to it. I have it because of the loss of water proton conduction.
When your sleep fails, your gene transcription falls off., and your mitochondria are liberating more light than they should and making more ATP than they should. This activates the DAMP program in cells.
The DAMP program provides interaction platforms at molecular levels linked to common hallmarks of aging and cancer. They can act as inducers, sensors, and mediators of stress through individual plasma membrane receptors, intracellular recognition receptors (e.g., advanced glycosylation end product-specific receptors, AIM2-like receptors, RIG-I-like receptors, and NOD1-like receptors, and toll-like receptors), or following endocytic uptake. Thus, the DAMP Hypothesis is novel and complements other theories that explain the features of aging. Aging is not a disease. It is a feature of quantum cell design. DAMPs represent ideal biomarkers of aging and provide an attractive target for interventions in aging and age-associated diseases. DAMP phenotype = a loss of energy at the electronic level in the cell. A loss of energy/information leads to a loss of time in your life.
Chronically pumping in light to cells from the sun allows you to remain in a “zero entropy state” of health. When quantum timing is off, life dies because it has to rely on ATP regeneration and recycling systems (EMF-4) which are only designed to support non-complex life like Archaea and Eubacteria……not a matrix with a complex nervous system in its head.
Cells that use water superconduction live far from equilibrium all the time. Cells are dissipative forms of plasma that allow for exotic physics to occur daily as light and dark act on the cell. A zero entropy state defines what a perfect equilibrium is, in case you are wondering. Thermal energies, particularly at cellular equilibrium, possess no information potential at all to help biochemical reactants meet and react, whereas an excitation or resonance of a specific frequency at a certain temperature where no other excitation of the same energy exists in a system far from equilibrium (a cell) not only has just the requisite information to do the work but the inherent power to do it as well.
This power is built into its coherent cytostructural design and not into its mechanistic reactions found in a modern biochemistry textbook. The living organisms’ cytostructure provides the motive force of attraction using the power of semiconduction between appropriate bio-reactive moieties. In turn, this enables efficient energy transfers to take place simultaneously without time ever being a major player in those reactions. In essence, time can stand still in a zero-entropy system because light is not moving inside your cells due to the physics of ions and matter in cells in how they are atomically organized.
When this system gets any disorder (entropy) placed into it, we call this inflammation, randomness, or chaos increase, and the result is a process called aging. It does appear that order comes with chaos. This chaos is accounted for in a cell by its telomere length becoming shorter. The shorter the telomere, the more molecular chaos is present and, hence, the older the living organism is. This links leptin resistance to aging and shorter telomere lengths. When life is constructed in a quantum fashion, ‘information’ is not something apart from the energy. It is accounted for in functional design and organization.
This might be the most important blog I’ll write when it comes to neurosurgery and the genesis of neurological disease generation and regeneration. It will be a grand unifying theory that explains, childhood cancer, autism, and glioma progression in adults.
Robert Becker proposed on basis of his experimental work that living matter behaves as a semiconductor in a wide range of length scales ranging from brain scale to the scale of the entire body. Direct currents flowing only in the preferred direction would be essential for the functioning of the living manner in this framework. He found these currents in his experiments but could never explain them today, this blog will explore the generation of these currents.
Becker’s injury currents of regeneration were found but no one has explained where the DC comes from. This blog explores the likely source. FERROELECTRIC POTENTIAL OF THE MITOCHONDRIAL CYTOCHROME PROTEINS. Ferroelectricity binds all cells in tissues together so they act quantum coherently.
This explains why Becker found the nerve input (DC electric current) was critical in the current of injury. This bioelectric current is critical. In neurons, water is released every time an action potential is created in a tissue. Mechanical deformation in tissues generates action potentials in an injury situation. This would cause water to move within the nervous system. Water is polarizable by light. Birefringence is the optical property of a material having a refractive index that depends on the polarization and propagation direction of light.
To generate a DC electric current from light there has to be a way to turn solar light into electricity to link cells in a tissue. Nature turned to solid-state physics to do this. She used ferroelectricity to do it.
WHAT IS SOLID-STATE PHYSICS?
Solid-state physics is a branch of physics that deals with practical and theoretical investigations of the properties of solids, such as superconductivity, photoconductivity, and ferromagnetism. In other words, it is that the study of solids, through various methods like crystallography, quantum physics, electromagnetism, and metallurgy. Solid-state physics is the most important branch of condensed matter physics. It helps to investigate how the large-scale properties of solid materials result from their atomic-scale properties. Thus, physics forms a theoretical basis of materials science. This blog will make the case that decentralized biology really is a solid-state physical problem to be solved for centralized medicine.
One of the basic ideas of the mitochondriac theory of living matter is that various currents, even ionic currents, are quantal currents. The first possibility is that they are Josephson currents (Quantum brain blog) associated with Josephson junctions but already this assumption more or less implies also quantal versions of direct currents. The quantum model for nerve pulse assumes the use of WBG semiconductors that all use non-linear optics (NLO) to communicate and transfer information in many ways always using energy stored at the electronic level of cells. This allows atoms to be arranged in such a fashion that light stronger than terrestrial sunlight can be used to target melanin in humans to regenerate and renovate their tissues using endogenous generate DC electric current and quantal ionic currents through the cell membranes where DHA dominates physiological function. These currents will act like Josephson currents, making the situation automatically stationary.
One can criticize this assumption since the Compton length of ions for the ordinary value of the Planck constant is so small that magnetic flux tubes carrying the current through the membrane look rather long in this length scale. Therefore either the Planck constant should be rather large or one should have a non-ohmic quantum counterpart of a direct current in the case of ions and perhaps also protons in the case of neuronal membrane: electronic and perhaps also protonic currents could be still Josephson currents. This would conform to the low dissipation rate. For this reason, cells evolved to be non-equilibrium dissipative structures.
Enough with the fancy science stuff written to satisfy my future critics who’ll read this.
Let’s get to the Rock star in this story, Robert O. Becker.
The notion of tissues containing a direct current is indeed familiar already from the work of Robert Becker and in the following the results related to laser-induced healing, acupuncture, and DC currents are discussed first. The obvious question is whether these direct currents are actually currents and whether they could be universal in living matter. The mitochondriac model for quantal direct currents is proposed and its possible implications for the model of nerve pulse are discussed. Whether the model is consistent with the vacuum extremal property remains an open question.
Below is Becker’s work in cartoon form on bone semiconduction.
The gist of Becker’s idea on regeneration & wound healing is present below. in that picture represent the state of Becker’s work in 1972. The implication here is thus: The DC electric current from the circulatory system loaded with RBC filled with porphyphrins that absorb sunlight and charge up their cells powerfully because they are DEVOID of mitochondria. This charge density increases the Coulomb force and this creates an electric charge density that can be used and transform the matter in an RBC (hemoglobin semiconductive dye) into more primitive cells.
We call this de-differentiation in biology today. This is evidence of time reversal in a biological system. Beckers’s work shows us time can be reversed and light energy can be used to do to regenerate brand-new tissues. This ability remains in humans but it is not as great as it is in the simpler system of the salamander. Nonetheless, his work is eye-opening when you marry it to the idea of Dr. Doug Wallace who says that mitochondrial redox power makes diseases show up and disappear out of the blue as redox varies. So it raises the question for the mitochondriac how does this happen in us precisely?
I believe the answer is ferroelectricity.
Electro-optics are part of non-linear optical communication and ferroelectricity is part of the electro-optical story in solid-state physics.
NON-LINEAR OPTICS is the base communication system inside of cells. Color = frequency in light. Nonlinear optics allows us to change the color of a light beam, to change its shape in space and time, and to create the shortest events ever made by humans. It also allows us to turn light into an electric current. Nature has been at this game a lot longer than Silicon Valley. They just began the game in 1961.
Many people forget hemoglobin in an RBC is a crystal. They really forget that when sunlight hits the water in the circulatory system it takes on a physical state change. Water become liquid crystalline as well = which gains coherent domains which become redox piles of electrons to be moved in the cell system. Those electrons are critical for ferroelectric coupling in the circulatory system for arterial elasticity and cardiovascular health.
Applying a voltage to a crystal changes the refractive indices of the crystal and introduces birefringence to the system. This is what a mitochondrion does inside human cells. Light is capable of changing charges and charges change voltages. We change the voltages on our inner mitochondrial membrane and this changes the colors a mitochondrion can emit. All those colors stimulate different chromophores inside the structure of a mitochondrion to run the metabolism of oxidative phosphorylation and create water, CO2, and heat to reverse the process of photosynthesis. The light show cephalopods are putting on is the optical representation of their metabolism in real-time. It doubles as a communication skill because they can vary their metabolism in the compartments of their cells. They emit these colors to communicate. It replaces their ability to speak. Humans have had 560 million years to upgrade and change their communication skills.
I told Dr. Huberman I liked cephalopods because as a neurosurgeon they allow me to look back and see the earliest organizations of a neural network. Many people do not know that Cephalopods evolved during the Cambrian period (∼530 Ma) very close to when this UV expansion fell to Earth. This also explains how we went from bacteria and archaea so fast into very complex cephalopod animals. The earliest melanin chemicals were critical to this rapid evolution. These animals came from a monoplacophoran-like mollusk in which the conical, external shell was modified into a chambered buoyancy apparatus. During the mid-Palaeozoic (∼416 Ma) cephalopods diverged into nautiloids and the presently dominant coleoids.
The strong linear coupling of the director ň to electric field E in ferroelectric liquid crystals can be utilized to perform high-speed electro-optic switching suitable for device applications.
REGENERATION AND THE DC ELECTRIC CURRENT
Beckers’s work is largely unknown and was buried by the military and Big Pharma.
The implication of his work was toward the idea of human regeneration and renovaBeckers’stion of all disease states. My work is taking up where he left off. He is an unknown giant in decentralized medicine in my opinion.
His best work was done in bone regeneration which in human remains. We do not heal fractures, we regenerate the bone completely. This statement is huge.
The distinction between bone response to stress and to fracture lies in cellular changes which seem to be of great importance. In the case of fracture repair, Becker found that certain cells under appropriate electrical stimulation (direct current in p.p.amp.ranges) would undergo reversion to a more primitive cell type in the human cell line. Becker found that the physical characteristics of the current were KEY in performing this task.
RBCs from the human circulatory system de-differentiated, and this RBC became transformed into a primitive material found in cells and it was subsequently re-differentiated into those cell types needed for the particular tissue repair process required. Becker was able to study some of the electrical parameters of importance in the injury current; the most outstanding was that the effective levels of voltage and current had both upper and lower limits. In other words, voltages or currents above an upper limit were non-productive of cellular changes until cur- rent densities became high enough to produce heating effects. I believe the reason for this is because the current links to the dielectric constant in water making humans have the ideal refractive index to be able to use solar power to regenerate our tissues.
During night and day, our refractive indices change because of human ferroelectric ability in tissues that link all cells into a synctium. This gives us a dark and light mode of plasma possible. It mimics what plants can do in photosynthesis in the dark and light reactions (pic below). Recall from Burr’s work all plants also exhibit a DC electric current when they are injured as well. This tells the mitochondriac mind this is a light-mediated function of all cells.
Life, and, in particular, its amazing diversity spanning more than 21 orders of magnitude in size, is the most complex physical system in the universe. In spite of this, biological systems obey a host of remarkably simple and systematic empirical scaling laws which relate to how organismal features change with size over many orders of magnitude. These include fundamental quantities like metabolic rate (the rate at which energy must be supplied in order to sustain an organism), time scales (such as lifespan and heart rate), and sizes (such as the length of the aorta or height of a tree trunk). It is a remarkable fact that all of these can be expressed as power law relationships with exponents that are simple multiples of 1 (e.g. ~, 3, 3). They appear to be valid for almost all forms of life, whether it be mammalian, avian, reptilian, unicellular, or plant-like. Clearly, the universal character of these “laws” is telling us something important about the way life is organized photonic energy at the electronic and vibrational state of matter in cells and the constraints under which it has evolved.
Becker wrote that he felt he knew why human regeneration was limited to the bone to just bone and fingertips. He theorized that one reason why mammals cannot achieve many kinds of regenerative growth seen in amphibians is the absence of adequate “electrical factors” at the injury site. Acting on that hypothesis, his laboratory initially undertook a study of limb regeneration in the nocturnal white rat. The theoretical current/voltage requirements were small enough so that we could consider bimetallic electrogenic couplings as a power source; similar devices had been used by Professor Stephen Smith in 1967 to restore some measure of limb regeneration in the frog, an amphibian capable of limb regeneration when in the tadpole stage of development but lack this ability in adulthood. I’d remind you at this point frog melanopsin & melanin and human melanin are almost identical homologs. Becker never knew this about the non visual photoreceptors in tissues.
Becker investigated this aspect of Smith’s work and found that Smith’s silver-platinum junctions far exceeded currents approximately five times that injury regeneration required. Becker resolved the problem by putting carbon resistors between silver and platinum electrodes and encasing the system in silicon. When I read this as a resident I knew what this meant. He created his own WBG semiconductor and changed the current using NLO. I do not believe Becker had any idea that he was changing the frequency of light in the diode to alter the DC current downward. It was a remarkable feat that he should have won the Nobel Prize but I do not believe any but myself realized what he did. When I did I realized what cells were doing inside of us all the time. We were filled with Wide Band Gapped semiconductors that were changing sunlight into VUV-IR-A light endogenously and using that light to heal and regenerate us while using ferroelectricity to create the DC electric current. I was astounded at his experimental brilliance.
When Becker made these adjustments guess what happened in that nocturnal white rat? He was able to regenerate all the tissues a rat limb needs to form. He never regenerated the entire limb but he verified that the tissue created was brand new and not just a healed limb. Below was his setup in his 1969 papers.
FROM REGENERATION TO CANCER GENERATION
I briefly discussed this with Huberman and Rick and Huberman was completely unaware of what Becker had found. I explained to him I learned about Becker’s work as a neurosurgery resident in learning about bone healing. It was here I learned bone does not heal it regenerates fully from primitive cells.
The importance of Becker’s observation is two-fold to the mitochondriac. The growth pattern makes it clear that the effect of the electrical energy was to bring about the de-differentiation of a cell population and its subsequent regrowth and re-differentiation, with the expression of multiple genetic pathways (ex., the controlled reading-out, and application of the all-purpose structural information that all cells possess-the process which occurs in its most complete form during the development of an embryo).
As a neurosurgeon, I immediately thought about the cancers I treat and how glioma behave via glioma progression. According to the result of several studies I read back then, the frequencies of brain tumors are of different rates according to countries worldwide. This hinted to me the light environment was critical because of latitude and zipcode effects. It also made me realize nnEMF likely was playing a role in this disease. In addition, these differences were observed among different ethnic groups in the same country. This told me local light factors had to be altering the injury currents in the brain and this led to tumor de-differentiation. This was when I coined the term, “you cannot get well in the same environment you got sick in”. It was because of Becker’s finding I just mentioned. I learned early in my residency that low-grade gliomas could be lived with for some time but with time they always de-differentiated into more lethal tumor types. I realized right away there had to be a link between the sun, gliomas, and the DC electric current. (see pic below)
What was the cause of BRAIN CANCER? Appropriate mtDNA control of apoptosis is important for normal functioning, and unregulated apoptosis has been linked to a variety of disease states. Excess apoptosis is thought to contribute to neurodegenerative diseases, whereas insufficient apoptosis can lead to diseases such as cancer. When UV light is blocked exogenous or endogenously from cells non visual photoreceptors, insuffiencent apoptosis results and this blocks cell cycle progression. I mentioned this in the Tetragrammaton podcast but I do not think Huberman understood the power of my idea.
As a result the cell cannot be removed from the tissue by the immune system and then it self isolates from its embryonic tissue. This changes its bio-electric signature inside the organ in question. It acts like an injury does. It is a stimulus that demands a response. The difference in cancer is the stimulus is ABSENT. Because surrounding ultraweak endogenous UV light is not present, the cell forces itself to remain vital as a solo entity within its embryologic position and this change in bio-electric stimulus leads to oncogenic transformation genetically. The lack of ultraweka-UV light emission causes the isolated cell to want to migrate to tissues who do contain the healing UV light stimulus needed to generate the bio-electric fingerprint to control its growth. Every tissue has its own bio-electric signature from morphogensis. Consider tissues to unique self identifying bio-electric barcodes that determine morphogenetic migration from its embryonic form. If this signature does not by molecular resonance, the migrating cell will resume growing but it will do it in another tissue that does not have its specific and sensitive bio-electric passport. This becomes a metastatic cancer focus. This is why the presence or absence of a tissues ultraweak UV bio-photon emission from their mitochondria is critical in apoptosis control and cancer prevention. This is counterintuitive to the centralized idea present in cancer medicine today.
IMPLICATIONS?
Cancer basicially are cells isolated from Nature and its ability to turn sunlight into a DC electric current. That DC electric connection comes form the sun and links all cells to the whole by the regenerative DC electric current that Becker discovered. When the bioelectric fingerprint isolates the cell from the tissue it is in, it cannot cooperarate coherent quantum mechanically and the cell loses its ability to undergo apoptosis, and it become invisible to the immune system and it reverts to its own survival modes built into using hypoxia and HIF-1. This causes the hijacking of the genome. If the cell is fully participatory in the bio electrical community in a tissue no oncogenesis is possible even if the genetic changes are present. People who think the BRCA mutation is a death sentence from cancer are smooth brainers who deserve what centralized medicine will give them. Oncogensis requires that cells isolate themselves because their is no surrounding ultraweak UV light to get them through the mitosis juncture of the cell cycle. They self isolate by closing their borders and removing their gap junctions. As soon as they do this, the conditions for existence of cancer becomes possible. The overall bioelectric state controls quantum coherence, and that state offers the ultimate control of the genetic state. The Genetic state of the cell is immaterial if the redox potential is controlled by Nature.
I had finally explained why cancer begins and why gliomas have a progression. Within the brain white and grey matter tracts have a very self similar bio-electric passport because they all come from neuroectoderm and the bio-electric power was high because of the mitochondrial capacity in neurons. I realized that the sensitiveity of the frequency match of the bioelectric current had to be controlled by the specific voltages in ferroelectric currents in the brain for low grade gliomas to grow slower and GBM to have very different bio-electric passports that lead to massive growth rapidly. Things began to make sense quantum mechanically to me.
SUMMARY
Becker’s experimental results produced in response to the level of current used (a total of 1 to 3 mJAamp) were extensive enough to warrant the prediction that similar-level bio-electric currents could have profound clinical implications for human disease.
For me Becker’s work, was unravelingthe the control mechanisms of life. I just had to make sense of the experimental data he had. The key was realizing the sun being transformed from cathode ray to terrestrial visible light and being captured by membranes filled with DHA that were connected to the TCA cycle. This caused the forward spin rate. When there was a defect in the bio-electric charge of the membranes, the TCA cycle would spin counterclockwise and this transformed the light emission of the endogenous WBG semiconductors. That change changed the bio-electric passport of the cells using the ferroelectric mechanism. This mechanism was how sunlight was transformed to a charge density on a membrane that leads to a measurable DC electric current in all cells. This creates a UNIQUE bio-electric passport for neuron migration in the developing brain and it creates a unique bio-electric zipcode within the tracts of the CNS. This unifies the how autism and brain cancer are bio-electric diseases.
THE EUREKA MOMENT AT DAVID’S FOOT
At that moment, I believed I could cure many diseases and not just treat them. I therefore now believe that low-level electrical currents and potentials, produced either by direct injection or by semiconductive rectification and induction from an electromagnetic field (melanin in us), have the FULL capability of bringing about very major biological effects of a very basic nature.
Immediately I realize how we went from chimp to human. I realized at the foot of David how every disease should be approached in decentralized fashion. It was 180 degrees from what I was taught in medical school.
The changes Becker found in his experiments appeared to be based upon perturbations produced in pre-existing biological electronic control systems which regulate very basic life functions. I knew immediately that they had to be wide-band gapped semiconductors because the electronic state of water and photosynthesis was 12.06 eV. This meant plants had to use soft Xrays to split water into hydrogen, oxygen, and 2 electrons. I asked myself does nature make mistakes?
I told Huberman that this peculiarity for me was always vexing. How could cells split water if they needed soft X-rays to do so? Becker’s work told me cells were solid state. Then it clicked. We were filled with WBG semiconductors that took the visible spectrum light in eV and added to it with our cellular design. Then splitting water was easy. So I pulled out a chlorophyll molecule and saw Mg sitting inside a nitride cage and I looked up the band gap of MgN. Problem solved. Its band gap was over 7 eV. Becker’s fexperiments explained a lot to my simple brain. They hold MASSIVE promise for a better understanding of ALL of life’s control systems and for clinical application to many mitochondrial diseases.
Becker is a giant to this decentralized mitochondriac. I cannot overstate the impact of his work on me. Becker taught me that just understanding biochemistry was the losing hand centralized medicine plays.
From his work, the biological effects involve basic functions of living material that are under remarkably precise control by mechanisms that have, to date, escaped description in terms of solution biochemistry. This suggests that bio-electromagnetic phenomena are fundamental attributes of living things, ones that must have been present in the first living things. The traditional approach to biogenesis postulates that life began in an aqueous environment, with the development of complex molecules and their subsequent sequestration from the environment by membranous structures. The solid-state approach proposes an origin in complex crystalline structures that possess such properties as semi-conductivity, photoconductivity, and piezoelectricity. All of the reported effects of electromagnetic forces seem to lend support to the latter hypothesis.
With this knowledge, I went from a centralized pusher of drugs to a decentralized physician pushing solid state physics in Nature overnight.
The neural electronic system also seemed to be related to levels of consciousness and biological cycles, and Becker’s work developed the thesis that this system furnishes the linkage mechanism between electromagnetic forces in the environment and biological cyclic behavior. In mammals, I found the electromagnetic induction of melanin from POMC was the key to the human system. It was on the shoulders of Becker, my ideas have gone further. Without his insight, I would be like any other centralized MD. In my opinion, human ferroelectricity in the neural and circulatory systems is another critical link in explaining his experimental findings. There is not a disease we cannot tackle with this wisdom.
The most amazing thing light has built is my species. It astounds me that light is slowed down by atoms in cells on Earth in a specific way that creatures like us do. I am amazed at how creation occurred.
All these thoughts created for you above came from starlight. Sunlight is our remedy in this life. I need to understand it fully before my “pilot light” goes out. Light organizes matter and hides in its electronic state but the type of light, the spectrum of this light from a source, has its own unique ability to sculpt atoms in specific ways to create new things from its inherent organizing ability. It is this ability hidden deep within the light that is the key to understanding life. It provides change without the perception of change. The thing that shows up every morning, which is most familiar, remains the most mysterious force in the Universe. That force organized every life system and every feeling of love that has ever existed. Matter exists to make love to light, to separate its electric and magnetic fields in ways that we just do not understand, and from this dance, light can sculpt a life, make a child, and create a new species. Everything man has known comes from the mysterious moves of our star’s light as it fell to this wet rocky planet. It is sheer amazement to me.
Living systems are nests of abiotic carbon, hydrogen, oxygen, and nitrogen, organized together with traces of a few other elements, yet of a complexity of structure that has hitherto resisted all attempts at complete analysis because we do not understand how light and water bind and weave the process of atoms in things. This makes our protoplasm the most enduring and the most easily destroyed of substances we know; its molecules are constantly programmed electronically by light to break down constantly, yet reorganize under solar power to furnish the power for the manifestations of many vital phenomena. Yet, through its remarkable property of assimilation of light, a power possessed by few other things on earth, it constantly builds up its substance anew from the surrounding medium and it avoids our perception of its recipe.
Now I knew how life operated at its fundamental levels. Now you do too! Below is one of my members, who just did not get the ideas in this blog together in her own life. She just died of cancer this week. I dedicate this blog to her.
CITES
1. Becker, R. 0., and D. G. Murray, “The Electric Control System Regulating Fracture Healing in Amphibians,” Clinical Orthopedics and Related Research, Vol. 73, pp. 169-98, 1970.
2. Frost, Harold M. (ed.), Bone Biodynamics. Boston: Little Brown and Co., 1964.
3. Szent-Gyorgyi, Albert, Introduction to a Submolecular Biology. New York: Academic Press, 1960.
4. Becker, Robert O. Technology Review, December 1972
