‘The drug war isn’t what you think it is’ – it is very interesting concept when you begin to. realize the Shamans inside the Amazon have always called ayahuasca, medicine.
The video above is on glioblastoma multiformans. It is the most lethal human cancer we know of in centralized medicine. Nothing kills quicker. In this way SV40 has a lot in common with it. It happens to be the one cancer neurosurgeons are expert in, in terms of treatment options. This blog today is about the decentralized treatment for this cancer linked to the jab. I think these options should be added to any GBM case after my visit to the Amazon in Suriname.
^^^^This is me in the Amazon River system with a Pirana net behind me looking for answers.
Why did I believe forests the best decentralized pharmacies on Earth to look for clues to cure incurable diseases like SV40 intercalation?
Why did I chose Suriname? The picture tells you why I chose it.
THEY HAVE THE LARGEST FOREST OF ANY COUNTRY.
93% of this country is a tropical rainforest.
Did you know most of the forests, are home to plants and animals that cannot be found anywhere else in the world? The same is true with the compounds their soils and water table create out of sunlight and magnetic flux in the magma chambers below them.
So what were the clues I went to Suriname with? What was I looking for?
I was looking for confirmation of why Ivermectin was working on people who took the jab who had turbocancers. Did I find what I was looking for?
I think I might have.
This table above was the treasure map I went to Suriname with. I then found 12 tribes who were treating the jab injured who’s story I laid out here.
https://x.com/dralexisjazmyn/status/1890609382422913290
You might want to listen to the ENTIRE podcast I did with her. I tried to tell some of my high profile clients what I was up to, but none of them wanted to listen to me before I left. I think that might change now.
FOR THE JABBED I BELIEVE WE NEED TO FIND UNIQUE NEVER BEFORE USED CBD’s
Why did I believe the Amazon a key player for the jabbed, especially those with SV40?
Dr’s Mary Sherman treatise made some comments about chemicals that had elements that were related to this class of drugs. She did not know that we had an endogenous endocannabinoid system operational in humans.
The Amazon is 55 million years old and it is where THE unique CBDs are created because of its geology. That is why I went to Suriname. They have CBDs that are unique.
You should visit @cannmed or @cannmedevents to learn more about this topic. That is their Twitter handles.
Cannabinoids exhibit some overlapping mechanisms with ivermectin (IVM) and fenbendazole (FenBen) in cancer treatment, particularly through microtubule disruption, apoptosis induction, and metabolic modulation.
Below are key comparisons:
1. Microtubule Disruption (Similar to Fenbendazole)
•Fenbendazole: Binds β-tubulin, disrupting microtubule polymerization, leading to mitotic arrest and cancer cell apoptosis.
•Cannabinoids: Some cannabinoids, such as CBD (cannabidiol) and THC (tetrahydrocannabinol), have been reported to destabilize microtubules:
•CBD disrupts tubulin polymerization, impairing mitotic spindle formation in glioblastoma and breast cancer models.
•THC alters microtubule dynamics, potentially affecting cancer cell division.
•Overlapping Effects: Cannabinoids may enhance the microtubule-disrupting effects of FenBen.
2. p53 Activation & Apoptosis (Similar to Both IVM & FenBen)
•FenBen & IVM: Upregulate p53, increasing apoptosis via BAX/BAK activation. •Cannabinoids:
•CBD & THC upregulate p53, leading to mitochondrial dysfunction and apoptosis.
•Activation of caspase-3 and caspase-9, triggering programmed cell death in multiple cancers.
•Overlapping Effects: Cannabinoids can synergize with FenBen or IVM to enhance apoptosis in cancer cells.
3. Metabolic Disruption & AMPK Activation (Similar to FenBen)
•Fenbendazole: Blocks glucose metabolism in cancer cells, reversing the Warburg effect. •Cannabinoids:
•CBD inhibits glucose uptake via downregulation of GLUT1 transporters.
•AMPK activation by cannabinoids leads to mTOR inhibition, reducing cancer cell growth. •Overlapping Effects: Cannabinoids mimic FenBen’s metabolic disruption, making them potential synergistic agents.
mTOR inhibition was a big topic in the Quantum Engineering Series and especially in the Melanin Renovation blog. You might want to re-read it if you took that jab.
4. Anti-Inflammatory & Immune Modulation (Similar to Ivermectin)
•Ivermectin: Modulates immune responses by shifting T-cell and cytokine activity, reducing cancer immune evasion.
•Cannabinoids:
•CBD reduces inflammatory cytokines (IL-6, TNF-α), potentially lowering tumor-promoting inflammation. It also affect the nrf2 pathway. NRF2 detox pathway only is tapped by having redox power present in the cell.
If your cells lack the net negative charge, they will never experience this pathway. So if you are pale and think using CBD is a panacea you’re a special kind of centralized idiot.
•Enhancement of immune surveillance through interaction with CB2 receptors on immune cells.
•Overlapping Effects: Cannabinoids may amplify IVM’s immune modulation in cancer therapy. Key Cannabinoids With Cancer-Treatment Potential
1. CBD (Cannabidiol): mechanism of action
•Microtubule disruption •p53 activation → apoptosis •AMPK activation → metabolic inhibition •Anti-inflammatory effects (reduces IL-6, TNF-α) alters NRF2 signaling.
2. THC (Tetrahydrocannabinol): mechanism is different but synergistic.
• Microtubule destabilization
• Apoptosis induction via CB1 receptor
• Inhibition of angiogenesis in tumors
3. CBG (Cannabigerol): mechanism is different but also synergistic.
• Inhibits mitochondrial respiration in cancer cells
• Synergizes with chemotherapy
4. THCV (Tetrahydrocannabivarin): mechanism is synergistic
•Reduces tumor cell proliferation
•Modulates AMPK/mTOR pathway Potential Synergistic Treatment Approaches (mTOR again)
•CBD + Fenbendazole: Both disrupt microtubules and glucose metabolism.
•CBD/THC + Ivermectin: Immune modulation + apoptosis enhancement.
•Full-spectrum cannabinoids + Metabolic inhibitors: Combination therapy for aggressive cancers. All of these chemicals help and many of these chemicals have unique chemistry in the Amazon basin and make up most of the magic present in the CBD decentralized pharmacy we need to tap for the jabbed.
My Conclusion: Cannabinoids share several anti-cancer properties with FenBen and Ivermectin, particularly microtubule inhibition, p53 activation, metabolic disruption, and immune modulation. This suggests they could be complementary in cancer treatment, especially in the jabbed. More decentralized research from the forest is needed to explore their combined effects.
We need to map the effects out and some have already begun this.
5. People in places on Earth are already doing this work and I have spent time meeting and talking with them.
Pay attention to IVM/FenBen for cancer as the recent blogs have laid out.
New data is arriving and ties DIRECTLY to my trip to the Amazon.
IVM prevents SV40 promoters from entering the nucleus but blocking importin A/B. This pathway likely has many chemokines that can impact it.
Yes, ivermectin is known to inhibit importin α/β-mediated nuclear transport, which is relevant in the context of SV40 promoters. Dr’sSarah Stewart and Mary Sherman did not know this when they working for DoD and Dr. Ocshner.
The Mechanism:
•Importin α/β Pathway: This transport system is responsible for shuttling proteins with nuclear localization signals (NLS) into the nucleus. Many viruses, including SV40, hijack this pathway to deliver their regulatory proteins (e.g., Large T – antigen) into the nucleus for replication and transcriptional activation.
• Ivermectin acts as an Inhibitor: Ivermectin binds to importin α/β and disrupts its function, thereby preventing nuclear entry of proteins that depend on this transport mechanism. Implications for SV40 Promoters:
•SV40 Promoters: The SV40 early promoter is often used in molecular biology due to its strong transcriptional activity in mammalian cells. However, its transactivation requires the nuclear localization of SV40 Large T antigen, which depends on importin α/β.
• Blocking Nuclear Entry is a KEY: If ivermectin blocks importin α/β, it could prevent SV40 Large T antigen from entering the nucleus, thereby reducing SV40-driven gene expression and viral replication.
Do we already have Experimental Evidence for this? Yep.
•Studies have demonstrated ivermectin’s ability to inhibit nuclear import of viral proteins from various RNA and DNA viruses (e.g., HIV-1, Dengue, and even SARS-CoV-2).
•SV40 Large T antigen is known to require importin α/β for nuclear entry. If ivermectin blocks this pathway, it could theoretically interfere with any SV40-driven transcription or replication in systems using this promoter.
Certainly, here are some key studies that provide evidence on this topic:
1. Ivermectin as an Importin α/β Inhibitor:
• A study by Wagstaff et al. (2012) demonstrated that ivermectin specifically inhibits importin α/β-mediated nuclear import. The researchers found that ivermectin effectively blocked the nuclear import of proteins dependent on the importin α/β pathway, without affecting other nuclear import pathways. This inhibition also correlated with a reduction in the replication of viruses such as HIV-1 and dengue virus, which rely on this pathway for nuclear entry of their proteins. (http://pmc.ncbi.nlm.nih.gov)
2. SV40 Large T Antigen and Importin α/β:
•The SV40 Large T antigen contains a nuclear localization signal (NLS) that is recognized by importin α, facilitating its transport into the nucleus via the importin β pathway. This nuclear import is essential for the Large T antigen’s role in viral replication and cell transformation. (http://en.wikipedia.org)
These studies collectively suggest that ivermectin’s inhibition of the importin α/β pathway could impede the nuclear import of SV40 Large T antigen, potentially affecting SV40 promoter activity and viral replication.
Does Fenbendazole Upregulate p53?
Yes, Fenbendazole (FenBen) has been reported to upregulate p53, a key tumor suppressor protein, in some cancer models.
Mechanism of p53 Upregulation by Fenbendazole:
1. Disruption of Microtubules:
• Fenbendazole binds to tubulin, preventing microtubule polymerization in a manner similar to colchicine or vinblastine.
• This leads to mitotic arrest, which can trigger cell cycle checkpoints and activation of the p53 pathway.
2. Induction of Cellular Stress & DNA Damage Response:
• Microtubule disruption can cause mitotic spindle stress, leading to chromosomal instability. • This activates ATM/ATR kinases, which phosphorylate p53, stabilizing it and increasing its transcriptional activity.
3.Apoptosis and Autophagy Induction:
• Upregulated p53 can activate BAX/BAK pro-apoptotic proteins, leading to mitochondrial damage and caspase-dependent apoptosis.
• Fenbendazole also promotes autophagy, which can contribute to cancer cell death.
There are 3 dominant phases of synchronized metabolic and transcriptional reprogramming when pigmenting the skin to avoid jab diseases. The melanogenic trigger is associated with high MITF levels and rapid glucose uptake by the cell. Blue light exposure stimulates massive glucose mobilization from ACTH from POMC. The transition to a pigmented state is accompanied by increased glucose channelization to anabolic pathways in biochemistry that support melanosome biogenesis. The H+/D optical switch you heard about in the Kruse for Dummies talk is critical in optimizing this step. You can find this talk in my IG biolink.
I talk about how I do this with pulsed light in the Melanin Renovation blog.
4. Inhibition of Glucose Metabolism
(Warburg Effect Reversal):
•Some studies suggest Fenbendazole reduces glucose uptake by cancer cells, similar to metformin. It might be time to consider this drug for all tech abusers.
• This metabolic stress can further activate AMPK pathway which leadsto p53-mediated tumor suppression. Why did the Brazilian tribes get more sick in my story to Dr. Alexis? They used technology that had enriched blue light. None of the other tribes did.
• This metabolic stress can further activate AMPK pathway which leadsto p53-mediated tumor suppression. Why did the Brazilian tribes get more sick in my story to Dr. Alexis? They used technology that had enriched blue light. None of the other tribes did.
Fenbendazole’s Mechanism for Cancer Treatment
1. Microtubule Disruption (Primary Mechanism)
• Fenbendazole binds to β-tubulin, disrupting microtubule formation.
• This prevents proper mitotic spindle formation, leading to G2/M cell cycle arrest.
• Cells stuck in mitotic arrest either undergo apoptosis or senescence.
2. Apoptosis Activation via p53 & BCL-2 Inhibition
•Cancer cells often overexpress BCL-2, an anti-apoptotic protein that prevents programmed cell death.
•Fenbendazole inhibits BCL-2, shifting the balance towards apoptosis.
3. Disrupting Glucose Metabolism in Cancer Cells
•Fenbendazole has been shown to reduce glucose uptake and ATP production, increasing oxidative stress in tumors. This is why PBM might help the jabbed as well. Light induces an abscopal effect on cells
• This effect weakens cancer cells that rely on glycolysis (Warburg effect), making them more sensitive to treatment. PBM is known to lower blood glucose by close to 30% via its abscopal effect.
4. Synergistic Effects with Chemotherapy & Radiation
Exposure to Ultraviolet Radiation in the A band promotes the expression of melatonin receptors in the skin via alpha MSH made from POMC. This occurs via melatonin receptors called MT1 and MT2.
In the lab, pretreatment with melatonin reduced cyclobutane pyrimidine dimers (DNA strand breaks) levels by approximately 40%. Remember, life is not lived or built in a LAB……….Nature is your lab. AM sunlight increases melatonin and melanin. https://www.nature.com/articles/s41598-017-01305-2
• Some studies suggest Fenbendazole enhances the effects of radiation and chemotherapy by:
• Increasing DNA damage accumulation.
• Disrupting repair pathways (e.g., via p53 activation).
•Weakening microtubule integrity, making cancer cells more vulnerable to other drugs or light therapy.
Supporting Studies & Evidence
1. Fenbendazole inhibits tumor growth via microtubule disruption and p53 activation
• Study in lung cancer cells showed that Fenbendazole caused mitotic arrest, increased p53, and induced apoptosis.
• (Source: PubMed)
2. Fenbendazole enhances radiation sensitivity by targeting microtubules and p53
• Research demonstrated that combining Fenbendazole with radiation led to increased DNA damage and cell death.
• (Source: PMC)
3. Mechanism of Fenbendazole in disrupting glucose metabolism
• Study found Fenbendazole downregulates GLUT1, reducing glucose uptake in cancer cells.
• (Source: PubMed) Conclusion Fenbendazole upregulates p53 by causing mitotic stress, DNA damage, and metabolic inhibition, leading to cancer cell apoptosis.
Its primary mechanism is microtubule disruption, similar to drugs like Vinblastine or Colchicine used in centralized oncology, but with much lower toxicity.
CAN THESE IDEAS HELP TURBO BREAST CANCER = triple negative cancers?
Stage 4 triple negative breast cancer is exploding in the COVID era in our women. I really worry about our children who are still getting popped with these jabs by pediatricians now.
There are case reports that this type of cancer can be erased with a combo of cannabinoids and psilocybin. Might we be able to find unique genomes of these two drugs to combine together to treat SV40 induced cancers?
I think so.
It might turn out that the centralized drug war isn’t what you think it is. It may fuel the decentral drug wars on all human cancers.
Yes, psilocybin (and its active metabolite psilocin) interacts with several cancer-related pathways, some of which overlap with Fenbendazole, Ivermectin, and Cannabinoids. While psilocybin is primarily studied for its psychedelic and neuroplasticity effects, emerging research suggests it may have anti-cancer properties via serotonin receptor modulation, immune regulation, and metabolic disruption.
1. Serotonin (5-HT) Receptor Activation and p53 Pathway (Similar to FenBen & Cannabinoids)
^^^^Remember where you saw this? The Melanin Renovation blog.
Sunlight increases the catalytic activity of enzyme IDO in this pathway as I already taught you
When this happens your cells begin making more kynurenine and less serotonin and melatonin this leads to ALL CHRONIC DISEASES and INCREASES ALL CAUSE MORTALITY because it destroys melanin renovation internally.
WHILE simulataneously SUNLIGHT decreases the catalytic activity of KAT
Making less kynurenine acid (neuro protective for melanin) and more quinolinic acid (harmful for melanin) from melanin degradation.
A lack of sun causes melanin degradation via hypoxia. Non native EMF cause liberation of Vitamin A from the loose covalent bond of opsins in the non visual photoreceptor system due to alien light and that Vitamin A cause hypoxia by lowering NAD+ in a cell. This is today’s major cause of disruption in this pathway. This slide should jar your memory of my past lessons. If you are jabbed you really better get this lesson.
•Fenbendazole & Cannabinoids: Activate p53, leading to apoptosis.
•Psilocybin/Psilocin:
•Activates 5-HT2A receptors, which have been linked to p53 upregulation in certain cancer models.
•Induces apoptosis in neuroendocrine tumors via serotonin signaling.
•Regulates BCL-2/BAX, modulating mitochondrial-mediated apoptosis. Overlap:
•Psilocybin may enhance p53 activity, similar to FenBen and CBD, leading to cancer cell apoptosis.
2. Metabolic Disruption & AMPK Activation (Similar to FenBen & Cannabinoids)
•Fenbendazole: Blocks glucose metabolism, shifting cancer cells away from glycolysis (Warburg effect).
•Psilocybin: Modulates AMPK/mTOR pathways, which regulate cancer metabolism and cell proliferation. 380 nm light addition here to improve mTOR signaling. This is why the tropics are critical to these riddle. Remember this slide from the Melanin Renovation Rx for Mammals on Patreon?
•Downregulates PI3K/AKT, reducing cancer cell survival.
•Potential to inhibit GLUT1 (glucose transport), limiting cancer energy supply. Overlap:
•Psilocybin could synergize with FenBen, Metformin, or Cannabinoids in starving tumors of glucose. PBM should added here because it lowers blood glucose and insulin signalin..
3. Microtubule Interactions & Cytoskeletal Remodeling (Similar to FenBen)
•Fenbendazole: Binds β-tubulin, causing mitotic arrest and apoptosis.
•Psilocybin/Psilocin:
•Modulates cytoskeletal proteins, leading to structural changes in cancer cells.
•Impacts tubulin polymerization in neuronal cells, suggesting possible effects on cancer cell microtubules.
Overlap:
•Though less studied in cancer, psilocybin’s cytoskeletal effects might interact with FenBen-like mechanisms.
4. Immune Modulation & Anti-Inflammatory Effects (Similar to Ivermectin & Cannabinoids)
•Ivermectin: Modulates T-cell responses, enhances anti-tumor immunity.
•Psilocybin:
•Reduces pro-inflammatory cytokines (IL-6, TNF-α, IL-1β), which drive tumor progression. •Enhances immune checkpoint modulation, possibly affecting PD-1/PD-L1 pathways in cancer immunotherapy.
•Shifts immune response toward anti-cancer cytotoxicity.
Overlap:
•Psilocybin’s immune effects mimic Ivermectin’s immunomodulation, making it a potential adjunct in cancer therapy.
5. Potential Synergies with Ivermectin, FenBen, and Cannabinoids PathwayFenBenIvermectinCannabinoidsPsilocybin p53 Activation Microtubule Disruption
(CBD)
(Possible) AMPK/mTOR Modulation
Metabolic Disruption
Immune Modulation
When I asked the Shamans about how they chose their medicines I found out they were using drugs from the forrest that all had these overlaps.
Psilocybin has overlapping mechanisms with Fenbendazole, Ivermectin, and Cannabinoids in p53 activation, immune modulation, metabolic disruption, and possibly cytoskeletal remodeling. While direct microtubule inhibition is uncertain, serotonin signaling, apoptosis, and immune regulation suggest a potential role in cancer therapy. Would you like specific references, drug interaction studies, or potential combination therapies explored further?
THE REMEDY CAME ON LIKE A DREAM TO ME.
SUMMARY
Catastrophic disclosure was not linked to the information and evidence coming out during the COVID era. Covid narratives were built around but a consolidation around these false narrative by federal agencies who incentivized many groups to support COVID psy-op fear; This is how a false flag operations happened at scale from 2020-2025.
All of you need to follow @JohnBeaudoinSr on Twitter because he has all the death data that holds the truths.
He has the data to support this supposition in great detail. This was catastrophic epoch in human health because it leads the world not having unity and peace, something beautiful, a view of us and other civilizations doing things together, a new world being created, technologically sustainable and peaceful, is about uniting the world under a sort of militaristic global totalitarian system built around surveillence capitalism where everyone is terrified what’s out there for many reasons linked to the narratives furnished and created by federal agencies so thatpeople give up their freedom with out a fight It’s a fear play to circumvent a political revolution if the truth really was known by “We The People.”
Many Federal agencies had a legal duty to inform us but they were told by the DoD and Pentagon to hide behind faulty use of the ICD coding system to lie to us. So far they have gone unpunished. The first step is complete banning of the mRNA platform. This podcast shows you why I undressed Calley Means in December with Danny Jones. Calley calling for “transparency” was a criminal speaking given the hard core data John has.
I’ll stop being a conspiracy theorist when the rich and powerful stop conspiring. We are being ruled by centralized greedy, power-hungry sociopaths who are destroying the planet. They are being led online by Sergey Brin and Anne Wojicicki.
Women who are infertile maybe helped by some of this information in this blog to have children. Why? The elites crafted the COVID era with the mRNA platform. The elites are eugenicists and they favor quick death via abortion for women to rid the planet of future children due to their perverse beliefs. The mechanisms of infertility mimic the disease creation of the turbocancers. All are tied to leptin melanocortin pathways.
The elites like Brin, Gates, Fauci, and GAVI linked companies knew to break the golden centralized rule in pregnancy medicine. Pregnant women are already in a state of immunosuppression so why would you then give a pregnant woman 4 different vaccines during this time? To eliminate NEW life is the short answer.
This is especially one that is experimental and the data showed it caused spontaneous miscarriages. So Gates and Fauci knew that the COVID jab was a covert abortion clinic run at global scale.
When you know better you can do better, and Uncle Jack is digging deep for the remedy for 6 billion of you.
CITES
1. https://www.youtube.com/watch?v=cn5CQGpiJWg
2. https://www.youtube.com/watch?v=KQUSoIJkaWg
3. https://www.youtube.com/watch?v=NN9X04C3G5Y
4. https://www.youtube.com/watch?v=SgCSGN8UR9M
5. https://www.youtube.com/watch?v=tAf3UB2ycCs
6. Single dose- is not BigHarma friendly solution. https://www.nejm.org/doi/full/10.1056/NEJMoa2206443