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Purines are a natural substance found in the body. They are also found in many foods such as liver, shellfish, and alcohol. They can also be formed in the body when DNA is broken down. When purines are broken down to uric acid in the blood, the body gets rid of it when you urinate or have a bowel movement.  This removal process allows humans and birds to conserve mitochondrial water.

Parkinson’s disease patients suffer from hypoxia of their pigmented neurons in the brain.  This leads to melanin breakdown and lowered levels of endogenous dopamine.  Uric acid, a metabolite of purine biochemistry protects neurons from melanin degeneration and dopamine loss.  In primates, having a higher level of uric acid is protective of hypoxia in dopamine producing neurons.  It appears evolution knew what it was doing when it altered the uric acid pathways in the big brained primate clade.

Parkinson’s Disease (PD) patients have been found to have significantly lower levels of serum Uric Acid than controls (p = 0.000) in published research.  This fact is not well known in clinical medicine.  In fact, in patients on L-dopa the situation is even more dire and shows us why Big Pharma solutions often run contrary to nature’s recipes.  In patients with PD serum UA levels were mush lower in the groups under L-Dopa + dopamine agonist treatment.

Uric acid (urate) is often referred to as a waste product of purine metabolism (Johnson et al., 2009; Rock et al., 2013) by people who just do not understand its impact in big brained primates. Normally, it circulates at high levels in humans and other hominoids due to mutations in the gene encoding the urate-catabolizing enzyme urate oxidase (UOx) during primate evolution (Wu et al. 1992, Oda et al. 2002).

In our species its circulating concentrations are so high they approach the limits of solubility.  Urate is best known clinically for the pain and damage that results when these limits are exceeded and urate crystallizes. When this occurs in joints it results in gout, a form of inflammatory arthritis triggered by urate crystals. Similarly, when urate (or more typically its acid form, uric acid) crystallizes in the urine then it can cause kidney stones.  People who have gout usually have serious blue light toxicity and melanopsin damage that lead to PD.  In fact, anyone with gout is a serious clue to me the quantum clinician that they have serious risks in their environment for damage to all melanin regions of the brain and skin.

To illustrate this point, I’d like to tell you about one of my Kruse Longevity Farm patients I treated in the last year. This person had many symptoms of undiagnosed mild Parkinson’s Disease. They came to me with many unusual symptoms that functional medicine doctors could not make sense of. This particular patient had went to the Kresser Institute and had 9300 dollars of testing over a 9 month period of time and never received an answer about their neurologic and gut issues he had.

After my nurse performed a review of systems and asked 45 minutes of questions I set down to review all the labs/tests that were ordered by 4 other clinicians. Once I reviewed the history, I noticed two things. The patient complained of a 3 year history of tinnitus, biannual bouts of gout, and a significant ENT history of cerumin accumulation and pale skin. This caused severe ear pain and reddened ear drums and multiple rounds of prescription antibiotics. After the first year, the person changed jobs and worked at night as an IT professional. During the first year of symptoms, gut issues and sleep became hugely disrupted no matter what interventions were done. As sleep worsened so did his cognition and motor abilities. He noticed changes to his emotions as well. He developed a worsening ringing in his ears and began to have balance issues.

When the patient arrived at my facility I asked pointed questions about the patients work environment after looking in their ears. Upon looking in the ears I saw cerumin accumulation on one side compared to the other but there was significant erythema of both ear drums. He was also sensitive to artificial light. I removed the ear wax and noted the area underneath it was pale. I also noted the pinna and external auditory canal were very pale and devoid of pigment. I examined his skin and noticed some uneven melanosis in places.

Immediately I asked about their use of cell phones, blue tooth, and nnEMF. Then I got my answer. The patient told me he used to use his cell phone to the ear 100% of the time but then his ENT physician convinced him to use wireless earbuds because of his ear problems to avoid putting the cell phone to his affected ear. Within 3 months of using the ear pods the patients symptoms of mild tinnitus progressed to mild Meniere’s disease with vertigo hearing loss and balance issues. This got worse when he shifted to night time consulting work. During this time he developed abnormal white skin patches over his torso and legs.

I asked him if he had the ear pods during his visit with me. He did. He told me he used them frequently in his car or when he traveled for business. I had him put them in the ear and we measured the nnEMF they emitted. The picture below shows you what I found. On his labs done at the Kresser Institute I noted three draws where his uric acids levels were off the charts. The patient also had high blood pressure every time he had his uric acid drawn according to the chart review.

This level of nnEMF was unbelievable. The amount of radiation delivered to the ear, ear drum, cochlea, and brainstem. The brain stem is where the vagal nuclei are that control the flow of autonomic information from the brain to the gut. His eye exam showed an abnormal blink reflex, and his pupils hardly reacted to blue LED during direct eye exam, while reacting briskly to red LED during the eye exam. This told me that there was a serious lack of dopamine in the pupillary mechanism. The eye exam revealed a pale retina. There was no change in refraction noted by the patient. When I consolidated all these findings, my advice to them were to eliminate the ear buds, quit the night shift, begin a serious increase of building the solar callus every AM, use of PBM/LLLT to the ears and head, and begin using my survivor’s soup patreon recipe every day. I also got him to wear blue blocking glasses with 550 BI tints anytime he was around blue light or in indoors behind glass. He has used Luciaeyes,com blue blockers for the last 7 months.

The remainder of the patients extensive work up at the Kruse Longevity Center under my care showed no other sigficant abnormalities.  The patient instituted all my protocols and advice the symptoms for the most part improved within 3 weeks.  His Bristol stool score went from a 7 to a 4 in two weeks and all the GI symptoms improved quickly when he eliminated tech use to his ears and used more light from the sun and his red light I recommended.  His tinnitus improved over 6 months.  He grade his tinnitus at 7 out of ten at his initial visit, and at his 6 month follow up he reported it was now a 1 of 10.    His Meniere’s symptoms resolved in 4 months 100%.  The patient was taught how to improve his skin’s melanosis with sensible solar behavioral change and I used my light hygiene protocol to augment this result.  That protocol is devoid of any Big Pharma solutions.  His cognition and sleep improved over 3 months, and his uric acid levels normalized over the last year.  This case illustrates how nnEMF can destroy melanosis and cause abnormal neurologic symptoms, skin changes, and gut issues that lead to many low dopamine symptoms and disease phenotypes.

WHY DID ALL THIS MAKE SENSE TO ME? 

Most people think high uric acid levels are pathologic.  This is not always true.  Many times people with electromagnetic sensitivities have high levels of uric acid to compensate for cellular hypoxia.

Uric acid has been studied in several cardiorespiratory processes that produce hypoxia since this condition leads to increased catabolism of purines. In this sense, uric acid has shown me to be a useful as a prognostic marker of hypoxia from many causes.  This includes electromagnetic hypersensitivity, blue light toxicity,  heart failure, pulmonary thromboembolism, viral illness,  and primary pulmonary hypertension.  A higher uric acid level is a subtle sign of significant mitochondrial heteroplasmy from an unknown source linked to hypoxia.  So is a higher level of homocysteine.  Tissue hypoxia contribute to a sequence of events by leading to the depletion of adenosine triphosphate (ATP) and activation of purine nucleotide degradation to uric acid.

It appears this patients previous care givers did not know about these links.  That is why they missed it.  Uric acid is vilified by functional medicine practitioners because they are ignorant of how evolution has used it in primates.  It carries a negative connotation for many clinicians, but as a neurosurgeon, I am acutely aware that high levels of uric acid also are protective in traumatic brain injury, CNS trauma, and this is best seen in people with Parkinson’s disease.  Uric acid has a significant potential for helping people with Parkinson’s disease and other neurodegenerative diseases that are exploding in this tech driven world.

Despite the known and theoretical adverse effects of higher uric acid levels in medical books, the evolutionary biology and biochemistry of uric acid suggests that its salubrious actions has a deep evolutionary purpose for large brained primates.  Uric acid appears to offset and possibly outweigh its detrimental effects (Álvarez-Lario & Macarrón-Vicente, 2010). Gout maybe a disease Nature gives is because uric acid protects us from cognitive decline and motor degeneration of low dopamine states.  I have also noted uric acid levels being higher in those with depression and bipolar disorder.  I believe this is a protective evolutionary response to a poor environment.  It turns out the uric acid-elevating inactivation of the urate oxidase enzyme in chimpanzees, gorillas, and humans can be attributed to multiple independent mutations in UOx during the speciation of primates (Wu et al. 1992, Oda et al. 2002).  This is unique to our evolutionary family.

This is why many doctors have reported acute gout flare ups in patients with PD who were put on receiving L-dopa in combination with a decarboxylase inhibitor. It appears blockade of decarboxylation leads to major changes in the pattern of L-dopa metabolites.

As such, the Black Swan clinician should reasonably presume that uric acid elevation conveys a critical survival advantage to our ancestors, with respect to their large brains. The discovery that uric acid possesses strong antioxidant properties, with a comparable or greater activity than ascorbate at their physiological concentrations in humans (Ames et al., 1981), suggested potential benefits of protection against oxidative stress. Uric acid decreases neuro-degeneration risks and this is likely why all primates have traded the higher levels of gout to protect their larger brains.

Might this be linked to the Warburg metabolism via glutamine?  Yes.  Glutamine enters the Krebs uric acid cycle. This move generates excess uric acid, and ammonia-N is excreted as uric acid-N. When this occurs cells require large inputs of glycine into the urea acid cycle along with, serine, and threonine, which serve as glycine precursors.  This is why I recommended the use of the survivor soup recipe to this patient.  That soup recipe is a glycine replacement mechanism for those who are tech addicted.  It is also why I recommended something called an Optimal Cocktail that I created at Kruse Longevity Center at Destin.

This is an oral trouche we’ve created with one of my pharmacist Farm member’s to offset low glycine states that appears with low dopamine states by taking advantage of the serine glycine interconversion pathway.

Every mole of uric acid synthesized as the end-product of N metabolism requires one mole of glycine, and the loss of glycine incurs a loss of 12.5 ATP molecules.  This amount of ATP stresses mitochondrial function which is underperforming in a low dopamine disease.  This increase need for ATP is why red light therapy helps the low dopamine state.

Since mitochondrial energy in the form of ATP is required to dispose of excess ammonia, it is apparent that ATP must be available for the urea cycle to properly function in low dopamine states. Thus, the urea cycle has to be closely linked physiologically to the citric acid cycle, which derives one of its nitrogens from the transamination of oxaloacetate to form aspartate and returns fumarate to the cycle while recycling metabolic water.  It was discovered in biochemical research that urea cycle disorders not only disturbe the metabolism of amino acids involved in the urea cycle but it also induce the accumulation of ammonia detoxification, but also interfered with the metabolism of amino acids related to some nervous systems, such as pipecolic acid and N-acetylaspartic acid.

SUMMARY

Is urea and uric acid the same thing?

In contrast, mammals (including humans) produce urea from ammonia; however, they also form some uric acid during the breakdown of their nucleic acids. In this case, uric acid is excreted in urine instead of in feces, as is done in birds and reptiles. Uric acid is a compound similar to purines found in nucleic acids.  Since the end product nitrogen metabolism is ammonia in birds, in which ammonia cannot be synthesized by urea; thus, birds and humans are prone to the accumulation of uric acid.  Birds, like humans have high mitochondrial capacity.  This is no coincidence.  What else does uric acid reserve for birds and humans?  Water.  Metabolic water to be exact.   In birds and reptiles, and in some desert dwelling mammals (such as the kangaroo rat), uric acid also is the end-product of purine metabolism, but it is excreted in feces as a dry mass on purpose to conserve water.. This involves a complex metabolic pathway that is energetically costly in comparison to processing of other nitrogenous wastes such as urea (from the urea cycle) or ammonia, but has the advantages of reducing water loss and preventing dehydration.  Water is the key electromagnetic capacitor in cells that have high mitochondrial capacity.  Recall that mitochondria make metabolic water from metabolism.

Purines are found in high concentration in meat and meat products, especially internal organs such as liver, shellfish, and kidney. In general, plant-based diets are low in purines.  This is another reason why a vegan diet stresses primates with a large brain and massive mitochondrial capacity.

Aside from the crucial roles of purines (adenine and guanine) in DNA and RNA, purines are also significant components in a number of other important biomolecules, such as ATP, GTP, cyclic AMP, NADH, and coenzyme A.

There are many naturally occurring purines. Adenosine is one of the major ones that signals the onset of sleep.  This is why the patient above could not sleep well.  Some common human purines are the nucleobases adenine  and guanine . In DNA, these bases form hydrogen bonds with their complementary pyrimidines, thymine and cytosine, respectively.  You saw those appear in the last blog.   This is called complementary base pairing. In RNA, the complement of adenine is uracil instead of thymine.  It is also a purine.

Other notable purines are hypoxanthine , xanthine, theobromine, caffeine, uric acid  and isoguanine.

Hans Kornberg wrote a paper entitled ‘Krebs and his trinity of cycles’ commenting that every school biology student knows of the Krebs cycle, but few know that Krebs discovered two other cycles in biology. These are (1) the ornithine cycle (urea cycle), (2) the citric acid cycle (tricarboxylic acid or TCA cycle), and (3) the glyoxylate cycle that was described by Krebs and Kornberg. Ironically, Kornberg, codiscoverer of the ‘glyoxylate cycle’, overlooked a fourth Krebs cycle – (4) the uric acid cycle.  This cycle is critical in low dopamine states.  Anytime dopamine levels are lowered by blue light and nnEMF we should expect higher levels of uric acid to protect dopamine stores in pigmented cells in our body.

The patient spent over 20,000 dollars in 3 years and got no answers.  He became a Farm member and with in 6 months he found his answer and got better without any need of a drug.  His cost of membership was much lower than he spent to solve this problem.

CITES:

• Association of serum uric acid levels with the progression of Parkinson’s disease in Chinese patients.Sun CC, Luo FF, Wei L, Lei M, Li GF, Liu ZL, LE WD, Xu PY.Chin Med J (Engl). 2012 Feb;125(4):583-7.PMID: 22490478

     Ames BN, Cathcart R, Schwiers E, Hochstein P.  Uric acid provides an antioxidant defense   in humans against oxidant- and radical-caused aging and cancer: a hypothesis.Proc Natl Acad Sci U S A. 1981 Nov; 78(11):6858-62.

Serum uric acid levels and freezing of gait in Parkinson’s disease.Ou R, Cao B, Wei Q, Hou Y, Xu Y, Song W, Zhao B, Shang H.Neurol Sci. 2017 Jun;38(6):955-960. doi: 10.1007/s10072-017-2871-3. Epub 2017 Mar 1.PMID: 28251464

• Pharmacokinetic-pharmacodynamic modeling of levodopa in patients with advanced Parkinson disease.Adamiak U, Kaldonska M, Klodowska-Duda G, Wyska E, Safranow K, Bialecka M, Gawronska-Szklarz B.Clin Neuropharmacol. 2010 May;33(3):135-41. doi: 10.1097/WNF.0b013e3181d47849.PMID: 20216409 Clinical Trial.
• Four pioneers of L-dopa treatment: Arvid Carlsson, Oleh Hornykiewicz, George Cotzias, and Melvin Yahr.Lees AJ, Tolosa E, Olanow CW.Mov Disord. 2015 Jan;30(1):19-36. doi: 10.1002/mds.26120. Epub 2014 Dec 8.PMID: 25488030 Review.
• Elevated homocysteine levels in levodopa-treated idiopathic Parkinson’s disease: a meta-analysis.Hu XW, Qin SM, Li D, Hu LF, Liu CF.Acta Neurol Scand. 2013 Aug;128(2):73-82. doi: 10.1111/ane.12106. Epub 2013 Feb 21.PMID: 23432663 Review.
• Kornberg H.L. Krebs and his trinity of cycles. Nat. Rev. Mol. Cell Biol. 2000; 1: 225-228

I want you to watch the video above in its entirety before you go on to ask yourself this key question.

Do predatory publishing and social media have something in common that breeds the facade that bad ideas and science can, on the surface contain the aura of truth?

I follow a lot of people on social media who I disagree with because it allows me to see aspects of life I am blinded to by my own biases.  Social media allows me to see things I am blind too because the medium is the message.

When “People” become weak, societies crumble because liberties shrink. Only courageous hearts can endure the bitterness of truth…….

All too often we fail to see the good in a difficult situation. Our minds are wired in such a funny way that we tend to believe just because a door closed it must be a bad thing. I say this because I saw a comment from “progressive author journalist” that said, “Ruth Bader Ginsburg is going to be replaced by a woman who walked through every door that Ginsburg opened for her so she can promptly use her position to shut them all for others behind her.”

When I read the comment above on Twitter and saw how many people liked the comment, I thought to myself, there must be a lot of people on Twitter who think chronically with a closed mind.  These are people who revered RBG in life, but never really understood her message.  Every door was closed to RBG and what did RBG do?  Did she fold like a cheap beach chair or did she do something about it by thinking fiercely about her plight? 

When RBG started law school in 1956, women accounted for less than 3% of the legal profession = CLOSED DOOR

Until the early 1950s, the majority of local school boards and clerical firms had “marriage bar” policies in place against the hiring and retention of married women, so heading off to law school with a family in tow was truly a venture into uncharted waters. This is around the same time when Elinor Ostrom—future Nobel Prize winner in economics—would have been splitting up with her first husband because he saw graduate coursework as incompatible with their life together. This is another example of a CLOSED DOOR.    Did this stop Elinor from creating a new room in the future for women like Ms. Ahern?  What is the common tie here?  In the 1950’s these powerful women did not have social media and that medium was not filtering the way they think into something that create a prison for them so they could not destroy the glass ceilings they faced.

Social media, and specificaly, Ms. Ahern’s tweet, allowed me to realize the basis of our political divide is tied to how people think about ideas when they are unable to process information well. 

History didn’t have to move the direction that it did without some help from thinkers like RBG. Waypavers are willing to be first in line to take chances that others around them are either not willing or not able to. In so doing, they forever alter the expectations of those who come after them. By deciding the path of professional constitutional law was possible for her, Ginsburg made it easier for others to decide they were capable as well.

Ginsburg credited her mother and her undergraduate teachers with enabling her “to take part in the effort to free our daughters and sons to achieve whatever their talents equipped them to accomplish, with no artificial barriers blocking their way” RBG discussed this in her preface of her “In My Own Words”.  She credited her mother with encouraging her to be a lady, meaning, to always be civil; to be independent; and to love libraries and books. It appears this was RBG medium.  That medium bred a different kind of thinking in Justice Ginsburg.  Ginsburg mentioned the ambitious, literary “Jo” from Louisa May Alcott’s Little Women as a favorite find from those early days she spent in the library on page 4 of the book. I suspect if you took a poll of women in business today, you’d find an awful lot of “Jo”s who were also lit on fire by Ms. Alcott.

RBG also credited two of her undergraduate professors, novelist Vladimir Nabokov, and constitutional scholar Robert Cushman, with teaching her the value of choosing words carefully and taking a principled stance.

Justice Ginsburg lived her principles do you think Ms. Ahern does?   Does she value words as an author?  Might that be tied to the medium she trained on?  Do you think that might have some lasting effect on her work because now she is an author and journalist?  Given what RBG said above about words, the door RBG opened, might be fraught with some problems, no?

Don’t judge Ms. Ahern by a tweet, but I want you to ask yourself a question……..might how she saw and learned about RBG on social media define the message she has received and this is now seared into how she acts, speaks, and talks, and will live the rest of her life?  It is quite clear RBG medium was not equivalent to Ms. Ahern’s.  

Why did her tweet about a closed door and RBG go viral on social media?  Might this virus of poor thinking be more dangerous than anything COVID-19 can throw at humanity?

Has social media let a genie out of the bottle that has fostered an anti-wisdom movement that has led to a door that should be bolted shut? 

Why do people believe that this tweet from Ms. Ahern has an aura of truth to it when the life of RBG was all about having a door slammed in her face at every step of her early life?  Wasn’t the action of a slammed door what created a new life for women?    Moreover, those who do believe that Ms. Ahern tweet has some logic to it, how do you think it will affect how they trust Amy Coney Barrett?  How will it affect anyone else in your life who you perceive that closed a door in your face?

It was at that time, I decided I needed to write about this topic in this blog for my patrons to become “agent provocateur” to get you to think about how bad science and poor ideas go viral today and gain traction THEY SHOULD NOT HAVE.  Many of the papers supported by the paradigm on C19 right now have these same traits.  

You don’t believe me? 

Here Fauci explains how and why he will limit who can have in your home, what pets you can have, the end of large events, and the effective dismantlement of cities. This should chill you.

HYPERLINK

This paper shows us how poor science may lead to a tear down of civil liberites.  RBG was a champion of showing this inequity in her legal career.

RBG was deeply concerned about the impact McCarthyism was having on civil liberties in the 1950s. RBG’s engagement with this cause was her first experience with the idea of the lawyer as a defender of constitutional rights, an influence which would carry through into her work with the ACLU’s Women’s Rights Project. The first brief RBG filed with the U.S. Supreme Court as a lawyer was in the 1971 case of Reed v. Reed, in which a mother had sued to be able to administer the estate of her deceased son. Idaho law had given explicit preference to the father since he was male, but the Supreme Court decided in favor of RBG’s argument that the Fourteenth Amendment did protect against gender discrimination in law.

RBG saw this work to advance equality as the “constitutional legacy” of our founding ideals: The founding fathers rebelled against the patriarchal power of kings and the idea that political authority may legitimately rest on birth status. Their culture at their time, she believed, might have held them back from fully perceiving or acting upon ideals of human equality and dignity.  From my reading of RBG life and her legal career this is why she became a leftist.  She was not a progressive or communist in any way.  Today’s leftist have tried to paint her that way to gain a door or access point to put a progressive communist jurist on the SCOTUS.

What really does leftist mean in the world that formed the great mind of RBG?  RBG education ideas were destroyed by her life experiences she faced.  She used those life experiences and chaos to change her world and the world around her.  She realized in law school and in her cases she argued before the SCOTUS that it was the incompleteness of the founding father’s vision of a non-hierarchical liberal democracy that blinded them to the harm that slavery, inequality in rights, protectionism, and monopoly privileges would wind up causing to their government and their principles.  She realized the founding fathers did not hate women, they just were blinded that not including the words women in the Constitution would lead to collateral deficits in the law affecting generations of women.  They were blinded because of their culture and society at the time did not allow them to see all facets of what their words might do 50, 100, 250 years into the future.

Do you think Ms. Ahern’s tweets will age well into the future as RBG ideas have?  Might the medium both used have something to do with this?  

I’m currently deeply concerned that Dr. Fauci has become modern medicine’s Joe MCCarthy.  His nonsense is destroying civil liberites in the name of pseudoscience.  It is being sold to the public under the guise of Big Pharma and vaccines to give it legitimacy.  

In 2020 America, many people speaking their “truth” on social media but they are really telling lies to their audience and the medium is giving them the ability to do so.  There is only the truth and it is not necessarily “yours”.  You are entitled to your opinion, but you’ve never been entitled to your own set of facts,  before social media arrived.  The medium allows you to cherry pick your data sets and this is fueled by your own cognitive biases and the network of people you surround yourself with.  Your circle of influence says a lot more about you than you want to admit.

This tells me this topic is current and important for you to understand if we are ever to trust one another.  It might be incredibly important for you trusting the scientific links I shared with you here in my past, present and future.    

I believe when you believe bad ideas or succumb to poor science it erodes trust.  It might even erode your trust in something that is true and give way to false beliefs.

When I read the tweet, I immediately thought about the video above and how bad science is now created for propaganda to sell things to people that they really don’t need for health. In fact, if they buy it, it might make them unhealthy. Then I thought about social media and bad ideas and how they spread. Is this why so much of what we believe is FALSE.

We’ve all heard the cliche, the medium is the message. If this is true today, what is our mediums doing to our messaging?

Does the medium of communication contain the seed of destruction for the message, ideas, or information?

Where did the cliche come from?  When it comes to understanding new media, one of the best to learn from is Marshall McLuhan. He was a Canadian born in 1911 and he died in 1980, McLuhan had no opportunity to experience the Web the way we know it today, but that didn’t stop him from exerting a huge influence on it.

It was McLuhan who first spoke about technology and communication having the ability to create a “global village.” As an early educator and pioneer of the study of communication and its evolution over time, McLuhan introduced a lot of observations about the impact of new forms of expression and media. Most notably, McLuhan’s expression “The medium is the message” has had a resounding impact not just on Web design but on mass media in general.

The video above shows you how a new medium has been created to sell things to people they do not need.  That is the point of predatory journals.  It gives cover to bad science and bad scientists.  You, my audience need to fully understand the implications of this new medium so you vet your experts and what they push and sell you and make sure they are not using predatory journals to push pseudoscientific ideas in your direction.  You might never know why their bullshit appears genuine.  Here is an example of a “doctor” selling you, the public an idea that was created in a predatory journal as the video above showed.  Who is packing your parachute?

Now that you know that DEFCON 26 outed GcMAF in the video above, how will this temper how you see Dr. Dietrich Klinghadt’s advice on anything in the future? Did he dig deep enough for his tribe to uncovered truth or did he use predatory journals to create profit for himself? Will Ms. Ahern’s tweets give you confidence that her vision of the present and future is built on a strong foundation? Do you like the way she has used words since she is now an author? Has the medium really helped her?

See, outing bullshit and bullshitter’s has a deep purpose to someone on a journey to see the truth……….the medium we use and how we use has a deep message for our audience.

Since the early days of communication, humanity has been captivated by the methods it uses to convey and preserve information. How we communicate with each other defines who we are and constitutes so much of what makes a culture and an individual unique. 

What if our current use of social media is destroying our human identity and destroying truth so that we get further from the answers society really needs?    If the medium is the message, what is the medium telling you, my student Black Swan, today about how we think about our world?

When I read Ms. Ahern’s Tweet I immediately thought to myself, “maybe these people I should spend some time on and try to help?”   The sudden death of RBG really caused some chaos in the mind of Ms. Ahern.  RBG was a liberal thinker.  She was not progressive.  Progressives always seem to deal in hopes rather than in facts…….Ms. Ahern however, is very progressive in her thinking.  I do not even think she realizes that she is anti RBG in her thoughts, but I want you to think about that idea and see if I am right or wrong in telling it.

That is when i decided to pen this blog to teach my tribe that social media is harming truth, the truth’s in Nature’s recipes that guys like Dr. Fauci and Mark Zuckerburg are trying to hide from you.

Less than 24 hours ago I was banned on FB for telling people that the sun, via Vitamin D creation was a great therapeutic idea for COVID19.

I posted two articles from non predatory journals that support the use of the sun and Vitamin D as a preventative cure from COVID 19.  Then I got this:

I had my legs cut out from underneath me.  I was censored.  What did I do.  I fought back using the medium and the pen as my message to my tribe.

If you feel as if your legs have been cut from underneath you, as Ms. Ahern did above in her tweet, instead of wallowing in despair get back up on your feet and touch the door that you feel has closed to you. If a door you value has closed for any reason, you should spend or waste any more time staring at it.  Ms. Ahern is a budding author.  She better think about the path she is on now.  

When you write to help others never compromise to someone else’s standards.  You have two choices in your life.  Live by your own choices or you’ll be forced to live by someone else’s.  Better to write for yourself and your own standards and have no public, than to write for the public and have no self.  The world is not ready for some folks when they show up, but that shouldn’t stop anyone from trying to help mankind.  Some things in life just can’t be bartered over or placed on the sale rack – and your self-worth is at the top of the list.

Be at peace and look for an open door. It’ll be there. Life is not lived through just one door. Then, walk through that new door it into the next phase of your life. A closed door is nothing but a new opportunity for new life experiences. One that might be greater than the life you knew in the old room, with the locked door tied to it.   RBG did it and so can you.   

Above Erin Schaff a NYT photographer, take a picture where you see conservative women, who support Judge Amy Coney Barrett’s nomination to the Supreme Court, pray while touching the doors of the Court as progressive leftist Jacquelyn Booth lies, crying on the ground, mourning the death of Justice Ruth Bader Ginsburg.

What was Ms. Schaff trying to convey to her Twitter audience if the medium is the message?

When the doors begin to close, the mind should begin to open.

Are you victim of life, or are you capable of overcoming your fears by thinking fiercely well?

How smart are you?

Do you find logic in false ideas?

Why does this happen to you?

Faced with a choice between changing one’s mind and proving there is no need to do so, almost everyone gets busy with the proof. The most difficult subjects can be explained to the most slow-witted man if he has not formed any idea of them already; but the simplest thing cannot be made clear to the most intelligent man if he is firmly persuaded that he knows already, without a shadow of doubt, what is laid before him. Does being super wrong about something in the past negate a current idea that is fabulous?

What’s going on here? Why don’t facts change our minds under a technocracy? And why would someone continue to believe a false or inaccurate idea anyway? How do such behaviors serve us?

2020 has shown us all that humans need a reasonably accurate view of the world in order to survive. If your model of reality is wildly different from the actual world, then you struggle to take effective actions each day.

In my opinion, social media acts to destroy real truths by giving every user a perception of the tech companies algorithm which is built around their version of the truth. Their truth is design around separting you from your dollar just by slightly getting you to change your behavior because of what their algorithm does to your brain via a blue lit screen.

How do I use social media?  I train wolves to think.  Help me find some more of them.  Share this with them if you enjoyed this journey through my mind.  

CITES: 

In My Own Words, RBG.  https://www.amazon.com/gp/product/1501145258/ref=as_li_ss_tl?ie=UTF8&linkCode=sl1&tag=lfdigital-20&linkId=033f04d2ec535d134930c041ebc307be

Twitter feed of Ms. Ahern and Schaff

The movie: The Social Dilemma on Netflix.

Is nighttime and DAYTIME technology devices use to blame for the etiology of most diseases in humans? Yes, I believe it is today. WOW. That is a big statement, Uncle Jack. How and Why? Here is a recent slide from a presentation I gave to shock my audience below.

Melanopsin, like the cone photoreceptor, is a PHOTORECEPTOR TOO FOR BLUE LIGHT. ALL OPSINS in humans are bound to retinol, and when the photoreceptors are damaged it is because of the atomic changes in retinol when the weak covalent bound it broken by light out of the normal circadian cycle.

What is the take home? The blue light hazard associated with man made light or the light present in all laboratories globally today generates massive levels of mitochondrial ROS and oxidative stress occurs directly in the outer segments of photoreceptors after blue light irradiation leading to disease. The cites below show that this statement is not hyperbole. It is now PEER reviewed published reality.

If you want to know where melanoma begins read the last cite in listed below. Melanocytes sense blue light and regulate pigmentation through Opsin-3 = melanopsin.

It took 90 years for “medical science ” to rebut the claim that linoleic acid is essential in any human diet, a claim that has led to an incalculable mischief in terms of dietary advice and disease prevalence, In the last two centuries. How many decades will it take for the average physician or your physician to read those papers covering the nutrition debacle? How long will it take to change their advice or their practice of the healing arts?

I believe it might take several hundred years to get people to understand that blue light from man made devices is more deadly than any other stimulus man has created in the last 120 years because our focus in in nuclear DNA and not on the mitochondrial DNA mutations cause by blue light.

In 1998, we found melanopsin in the retina. In 2009 we found neuropsin in the skin and cornea. In 2014, we found melanopsin in the arterioles of the human body. This tells us all we are creatures of light who capture and transform the light of the sun into energy that cells use to create life and health.

In December 2017, we found melanopsin in the skin and subcutaneous fat of humans. This was huge news to those who understand leptin and that leptin the fat hormone is also found within the subcutaneous fat of MAN. The data is telling us why we have an obesity crisis and it has NOTHING to do with food or exercise but it has a lot to do with circadian arrhythmia of light in our eye and skin and subcutaneous fat mass.

The nighttime and daytime light environment has changed dramatically due to modern technology. Increased usage of mobile devices during ANYTIME OF THE DAY can disrupt your circadian clock. PEOPLE forget that the melanopsin receptor is regenerated DURING daytime!!!! So if you are around man-made blue light during the day you are still ruining your melanopsin photoreceptors. The intense light emitted from technology devices in screens has the potential to alter the timed release of factors within the eye, leading to chronic insults and susceptibility for visual damage. What does this mean to melanopsin photoreceptors?

I gave you my answer in the Vermont 2018 video.

Recent findings cited below to suggest a functional role for the circadian clock in mammalian cone photoreceptor development and provide evidence for a continuing role for thyroid hormone (TH) signaling in cone photoreceptor maintenance. These researchers have said their findings may be of relevance in OTHER tissues where human photoreceptors are as well, where the circadian clock could alter tissue function by directly regulating enzyme type 2 iodothyronine deiodinase (Dio2) expression and thus TH signaling.

THAT is WHAT the data I presented in VERMONT 2018 is all about. I hope you view the talk. It is well worth it to further your education. Once you realize and know where melanopsin is, and follow the damage in its wake, you begin to see where mitochondrial disease begins for the very first time in your life. It happens where clinicians least expect it and that is why they always miss it and the public cannot be helped via a prescription pad. That is where the data is now……..it is not in food/exercise choices, it is made in the light choices we make.

With this new information, researchers can begin to ask questions such as, “How else can we change the photoreceptors in humans using light evolution has never used? Are there other factors that can improve photoreceptor function and help extend their viability that we have failed to consider in science and industry? The BLACK SWAN among you now knows this answer. It should be as clear as the nose on your face, because this new data is telling you disease generation is not what your doctors were taught in medical school.  Your light choices form your medical destiny.  

CITES:

https://consultqd.clevelandclinic.org/circadian-rhythms-thyroid-hormone-and-vision-loss/

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4048889/

https://yelling-stop.blogspot.com/2020/09/fat-and-weight-gain-note-to-peter-and.html

https://www.sciencedirect.com/science/article/pii/S2212492619300892

 

• Mondini dysplasia can cause chronic tinnitus. This defect comes from a defect in the Fibonacci sequences during embryogenesis and can lead to defects in melanosis that can profoundly affect hearing and tinnitus generation in the dysplastic brain.
• The Fibonacci numbers form a sequence of numbers defined by a relationship mathematically.  What this means, in English, is that it is a sequence of numbers whose relationship is this: after the first two numbers, each proceeding number is the sum of the previous two numbers. For example 0, 1, 1, 2, 3, 5, 8, 13, 21, 34, 55, 89, 144, 233…..and so on. Quite simple, really.
• Fibonacci numbers are not purely artifact, they are also found in nature in an uncurling fern, the branching of trees, and leaflets of the pineapple. The Fibonacci sequence also describes the “golden spiral,” which is when a “golden rectangle” is subdivided in smaller and smaller golden rectangles —the result being a predictable spiral.
• Fibonacci numbers have an interesting property. When you divide one number in the sequence by the number preceding it, you are left with a number very close to 1.618. This number is called the “golden ratio,” and rectangle whose sides is equal to the golden ratio is known as a “golden rectangle.”
• One example of a biological structure in the mammalian body which is very close to a “golden spiral” is the cochlea.  You can see that spiral below in blue.

• The Mondini malformation and Mondini defect is an abnormality of the inner ear that is associated with sensorineural hearing loss and tinnitus. This deformity was first described in 1791 by Mondini after examining the inner ear of a deaf boy. The Mondini dysplasia describes a cochlea with incomplete partitioning and a reduced number of turns, an enlarged vestibular aqueduct, and a dilated vestibule. A normal cochlea has two and a half turns, a cochlea with Mondini dysplasia has one and a half turns; the basal turn being normally formed with a dilated or cystic apical turn to the cochlear. The hearing loss can deteriorate over time either gradually or in a step-wise fashion and it can lead to debilitating tinnitus as hearing is destroyed. Destroying the ear with deafness operations might actually make the tinnitus worse. Cochlea implants might be a better way to deal with dysplastic auditory cortex.

About one in five people experience tinnitus, the perception of a sound—often described as ringing—that isn’t really there. Tinnitus brain mapping has revealed just how different tinnitus is from normal representations of sounds in the brain.

Perhaps the most remarkable finding from the brain mapping experiments was that activity directly linked to tinnitus was very extensive and spanned a large proportion of the part of the brain that researchers measured from during brain surgery. This tells us that tinnitus is really not a peripheral disease but a sensory processing disorder.

Only a few groups in the world have the expertise and collaborative infrastructure to conduct these neurosurgical experiments. It is possible because patients who require invasive brain mapping in preparation for epilepsy surgery also volunteer to participate in research studies. The University of Iowa has the ability to do this because of their epilepsy program.

It is such a rarity that a person requiring invasive electrode monitoring for epilepsy also has tinnitus. Some people do not have epilepsy but have been found to have other neurovascular abnormalities that might the cause of the tinnitus.

Iowa’s epilepsy team puts a recording platform into the patient’s brain for clinical purposes and they can modify it without changing the risk of the surgery. This allows them to understand functions in the brain in a way that is impossible to do with any other approach.

Iowa researchers contrasted brain activity during periods when tinnitus was relatively stronger and weaker. They found the expected tinnitus-linked brain activity, but they report that the unusual activity extended far beyond circumscribed auditory cortical regions to encompass almost all of the auditory cortex, along with other parts of the brain.

The sheer amount of the brain across which the tinnitus network is present suggests that tinnitus may not simply ‘fill in the gap’ left by hearing damage, but also actively infiltrates beyond this into wider brain systems based on the findings of this paper below.

These new insights should help to inform treatments such as neurofeedback or optogenetic therapies, where patients learn to control their “brainwaves,” or electromagnetic brain stimulation.

A better understanding of the brain patterns associated with tinnitus may also help point toward new photobiomodulation approaches to treatment.

The team included the University of Iowa researchers Hiroyuki Oya, Gander, Sedley, and Howard and Christopher Kovach, Kirill Nourski, and Hiroto Kawasaki, as well as Timothy Griffiths at Newcastle University. The research was supported by grants from the National Institutes of Health and the Wellcome Trust and Medical Research Council in the U.K.

Tinnitus appears to be a sensory processing disorder whose causes are multiple but all lead to a sensory processing disorder in the thalamus and auditory cortex.  Mondini’s dysplasia is one of many things that cause this cortical dysfunction in the auditory part of the brain.

CITES:

https://www.cell.com/current-biology/fulltext/S0960-9822(15)00278-X

DO TINNITUS, PD, and AD all RELATE TO blue light and nnEMF via hypoxia?

In the human ear, there are melanocytes present in the hearing mechanism which are cells that form melanin. Melanin is a UV light-absorbing pigment that can be made from tyrosine or phenylalanine. You can see from the picture above that the action spectra of light from both aromatic amino acids is 220-300nm light which is deep in the UV range. This implies the light frequencies are somehow linked to how and what we hear. Tinnitus is a warning symptom that your nnEMF environment is sub-optimal. The question is for the inquiring mind, is why did evolution put this photochemical in our hearing apparatus?

Might melanin be how we tune into our environment first before sound affects us?

Remember light travels way faster than sound so from a physics standpoint using light for the afferent sensory reflex loop makes a lot of quantum mechanical sense. It just offends your doctor’s common sense. That is a good thing for a Black Swan. We ask questions few people do.

In this system, full-spectrum solar light is critical because it contains the 200-400 nm light tyrosine/phenylalanine that needs to create melanin. Melanocytes make melanin from aromatic amino acids (tyrosine) that all have hexagonal rings of carbon that absorb UV light from 220 nm to 300 nm. For this mechanism to tune properly, you first need AM balanced light in the visible spectrum that has no UV light present. The dose of the blue light present is stimulatory to the pituitary gland but this pro-growth stimulus is always protected and balanced by the 42% of IR-A present early in the AM. The next light that shows up diurnally in solar exposure is UV-A light in the AM. when it shows up varies based upon your latitude. This explains why tinnitus is rare inside the tropics where light is more stable. I expect this will change as people inside the tropics begins to use technology from the US. This will ruin their afferent reflex loops.

Melanosomes in the ear pay attention to UV light’s arrival by having melanin’s electrons become excited by the sun rays. To activate the system the ear pinna and external auditory canal need the full day spectrum solar light to develop naturally. This includes IR-A, UV-A, and UV-B based upon your latitude and altitude.

The human ear effectively “measures” the light radiation environment you allow via your choices and helps fine tune your hearing by reacting with the photon traps in tyrosine and phenylalanine with the formation of melanin as hearing protectors. If the radiation environment is disturbed (by man-made light), limited or no melanin is formed in the pinna or in the external auditory canal.

Without this initial stimulus, human hearing becomes affected and our hearing becomes susceptible to an improper energy transformation.

People forget that when light collides with melanin the electromagnetic wave becomes and electro-mechanical wave or an electromechanical wave called a phonon/soliton. What is the target of this phonon or soliton? Look at the picture below. Is there a pigmented vascular layer in the inner ear? Yes, there is. Why do we have light pigments in our cochlea? And why do we have melanin in our pinna and our ear canal? These are questions Black Swans ask themselves to understand why their hearing is altered.

Those phonons are targeting pigmented cells in the body of the cochlea by a process of chemiexcitation. If solar light is absent and man-made light dominant in your environment the transformation these light waves into mechanical-acoustic signals (phonons/solitons) is disturbed. This is how tinnitus begins quantum mechanically in my opinion. The afferent reflex arc is disrupted by nnEMF light waves sensed on the external ear and in our ear canals.

Melanin is a pigmented polymer with a known role in dermal solar protection. In vertebrates, melanogenesis has been reported in leukocyte population of cells suggesting a potential role in innate immunity. An altered innate immune response is one of the first things ENT surgeons should suspect in people with tinnitus but few do because they do not know about this link.

The presence of melanin in the inner ear was established more than a century ago, but the exact biological function of the pigment in the labyrinth has yet to be determined in medical textbooks. This is why tinnitus remains a mystery for docs and their patients afflicted with it.

Special attention should be drawn to the composition of melanin. Melanin is a biological reservoir for divalent ions like calcium and as an ion exchanger, as well as an intracellular buffering system for calcium. nnEMF alters the resonance frequency of this atom. nnEMF also alters voltage-gated channels that control calcium flow. This explains why melanin and nnEMF are likely linked to the symptom of tinnitus. It should be pointed out to the non learned that melanin is capable of binding ototoxic drugs. Finally, morphological responses of melanocytes happen when local disturbance of Ca++. Below you can see how calcium is quantized to the light that affects the afferent loop in this reflex.

One last link of Ca2+ release to another ear disease is Meniere’s disease which is often linked to tinnitus.  An altered level of melanin or an imbalance of its functioning can create an imbalanced Ca2+ homeostasis in the inner ear.   This has been demonstrated using  endolymphatic hydrops (EH) as an animal model for Meniere’s disease. These models have shown us that there is a  possibility of a receptor-mediated Ca2+ transport across the pigmented epithelial layer, especially the light cells, and the presence of a ‘chemical signal’ as an initiating modulating factor in the disturbance of Ca2+ homeostasis.  It appears light tunes the cochela using calcium as the electromechanical lever.  It is pointed out numerous times in the PEER reviewed literature that melanin is capable of binding calcium and may act as a buffering system in hearing and in balance.

Quantum mechanics rarely extends to molecular medicine. Recently, the pigment melanin was found to be susceptible to chemiexcitation, in which an electron is chemically excited to a high-energy molecular orbital. In invertebrates, chemiexcitation causes bioluminescence; in mammals, a higher-energy process involving melanin transfers energy to RNA and DNA and mtDNA without using photons.  This can create lethal and mutagenic cyclobutane pyrimidine dimers that can cause many diseases. This process is initiated by the free radicals NO and O2-. Their formation can be triggered by nnEMF light and/or inflammation. Several chronic diseases share two properties: inflammation generates these radicals across the tissue, and the diseased cells lie near melanin. I have said for 15 years that that nonquantized chemiexcitation may be an upstream event in numerous human diseases that lead to mitochondrial damage.

DNA AND mtDNA REPAIR IS NOT EQUIVALENT

Nuclear DNA repair of UV pyrimidine dimers in Chinese hamster ovary cells (CHO cells) is very selective with preferential repair of only sequences containing actively transcribed genomic regions. In essence, the literature over the last 30 years has shown very little repair mechanisms of these dimers in the entire mitochondrial genome of any animal tested.  Nuclear DNA repair mechanisms are quite rapid and very accurate.  Direct nuclear DNA action is likely not the mechanism that leads to disease.  Research has shown, even actively transcribed areas in mtDNA do not appear to be repaired in mitochondrial DNA. This data is in agreement with that previously reported in HeLa cells and in yeast  where a lack of repair of pyrimidine dimers in mitochondrial DNA was also observed.

The chemiexcitation path to diseases is a new quantum mechanical idea. It has long been known that photons of ultraviolet radiation from sunlight can be directly absorbed by DNA, where they excite DNA bases. If two excited pyrimidines (thymine or cytosine) are adjacent in RNA, DNA or mtDNA, a double bond forms automatically by the action of light. UV light can do this but it blue light is also quite capable of doing this as well.  When blue light creates ROS and RNS it opens thymine or cytosine to form two single bonds between the bases and makes a cyclobutane pyrimidine dimer (CPD); this [2+2] cycloaddition reaction can only happen if the bases are in an excited state by light.  Sunlight normally can lead to cell death via apoptosis.  It can also stimulate NK cells to take out the altered cell filled with mitochondrial disease and viral loads.  Alien light does not stimulate immunosurveillance in the same “quantized way” that sunlight does and this can lead to cancers or other mitochondrial disease.  This is often associated with co-morbid hypoxia in the cell.   Immunological surveillance is a monitoring process of the immune system to detect and destroy virally infected and neoplastically transformed cells in the body.  This is why altered melanosis is linked to both viral illness and cancers.

The CPD disrupts base pairing and distorts the DNA helix, leading to cell death or – when DNA replicates – a C→T mutation. Mutations in oncogenes and tumor suppressor genes are required for disease generation like cancer. Chemiexcitation instead excites the DNA bases long after light exposure has ended because of how melanin operates with light. In this situation, light radiation serves to activate enzymes that synthesize the radicals NO• and O2•−for hours afterward. These radicals form peroxynitrite, which oxidizes melanin or its monomer DHICA (5,6-dihydroxyindole-2-carboxylic acid) and allows ambient O2 to create a dioxetane on the melanin. The dioextane moiety, is a strained 4-atom ring containing –C–O–O–C–, is unusual in being able to release large amounts of energy in a single reaction, creating long-lived, high-energy, electronically excited triplet states (denoted by * below).

This triplet energy can transfer to RNA/DNA/mtDNA, resulting in a CPD without the involvement of photons. Thereafter, an unrepaired CPD would again result in mutations or cell death. The same radicals are formed during normal cellular inflammation from nnEMF damage, creates the same end product can be created by enzymes like horseradish peroxidase (HRP), prompting the idea that chemiexcitation can occur in internal organs leading to a myriad of diseases that today have no explanation of what causes them. This mechanism is likely why RF and microwave radiation damaged the ears and brains in people stationed in the Cuban embassy a couple of years ago when and Electromagnetic device was aimed at our emabassy to cause damage to US citizens.

Human mitochondria have their own DNA, which is double- stranded, circular and contains —16,569 base pairs. The mitochondrial genome has been sequenced in its entirety and consists of genes that code for 13 subunits of the respiratory chain complexes, 22 tRNAs and two rRNAs. Until recently, the only mitochondrial DNA defect that appeared to be of significance was that of a mutation in one of the mitochondrial ribosomal RNA genes which conferred chloramphenicol resistance. However, within the past 30 years defects in the mitochondrial genome have been described in a number of human diseases. The initial description by Holt et al. of deleted mitochondrial DNA in patients with mitochondrial diseases has been confirmed by  numerous studies which found that mitochondrial deletions occurred in > 90 % of cases with Kearns—Sayre syndrome. Additionally, mitochondrial point mutations have been associated with Leber’s hereditary optic neuropathy, Pearson’s syndrome, autism, T2D, and some cases of chronic progressive external ophthalmoplegia. Accumulations of mutations in mitochondrial DNA have also been reported in the brains of patients with Parkinsonism and Alzheimers disease. These findings suggest that mitochondrial DNA defects are of biological importance in modern medicine.  It makes you wonder why medicine keeps ignoring these links.

What does all this imply?  If you live at a high latitude, with poor sunlight, have too much nnEMF in your life that causes hypoxia, or you live in a large city with many people addicted to technology, you are at risk for hearing loss and tinnitus.  It also might be a precursor symptom to macula degeneration and neurodegeneration in the brain.  Anywhere melanin is located, chemiexcitation can cause mtDNA damage and cause a hum in your ears.

CITES:

Chemical excitation of electrons: A dark path to melanoma.Premi S, Brash DE.DNA Repair (Amst). 2016 Aug;44:169-177. doi: 10.1016/j.dnarep.2016.05.023. Epub 2016 Jun 1.PMID: 27262612

• UV-induced Melanin Chemiexcitation: A New Mode of Melanoma Pathogenesis.Brash DE.Toxicol Pathol. 2016 Jun;44(4):552-4. doi: 10.1177/0192623316632072. Epub 2016 Mar 7.PMID: 26951162
• Role of Melanin Chemiexcitation in Melanoma Progression and Drug Resistance.Premi S.Front Oncol. 2020 Aug 6;10:1305. doi: 10.3389/fonc.2020.01305. eCollection 2020.PMID: 32850409
• Photochemistry. Chemiexcitation of melanin derivatives induces DNA photoproducts long after UV exposure.Premi S, Wallisch S, Mano CM, Weiner AB, Bacchiocchi A, Wakamatsu K, Bechara EJ, Halaban R, Douki T, Brash DE.Science. 2015 Feb 20;347(6224):842-7. doi: 10.1126/science.1256022.PMID: 25700512
• Unanticipated role of melanin in causing carcinogenic cyclobutane pyrimidine dimmers.Premi S, Brash DE.Mol Cell Oncol. 2015 Jun 23;3(1):e1033588. doi: 10.1080/23723556.2015.1033588. eCollection 2016 Jan.PMID: 27308551

Do you know the purpose of the unusual construction of the mitochondrial membranes? Did you know that the inner mitochondrial membrane is the ONLY membrane in humans that is DEVOID of DHA? Do you know why the membranes in the mitochondria are built by Nature to be extremely hydrophobic? Do you know that these membranes can carry voltages close to 30 million volts because of this build-out? Do you know that one of these membranes actually makes water from food? Did you know that the atomic construction of this water also has to be specifically depleted of a certain isotope? Do you know why all these things exist in your mitochondria?

The answer might shock you. Uncle Jack is going to tell you that all these things occurred 650 million years ago to suspend the laws of physics to allow nature to do things not even gods could fathom. Nature figured out how to use nanoscale spaces to suspend physics to build the possibility of life.

Did you know when a thin layer of water is squeezed between two hydrophobic surfaces, the laws of classical physics break down? Well, I have been teaching my members this since my epic April 2016 webinar on what life really is up too. Now you have more data I am on the right track and the paradigm and all my critics remain myopic.

The take-home is this: PHYSICS IS THE SCIENCE OF PROBABILITIES. BIOLOGY IS THE STORY OF THE IMPROBABLE AND BIOLOGY CAN ONLY MAKE SENSE FROM THE PERSPECTIVE THAT THE LIVING STATE IS ONLY PROBABLE USING REACTIONS WHICH ARE STATISTICALLY IMPROBABLE.

So Nature built cells to have the ability to suspend what we should expect to give us the most improbable answer why life really exists and happens. She figured out how to suspend her own laws using things she built. How is that for counterintuitive?

That is why I teach Black Swans how to think. Black Swans are rare. They go deeper to find wisdom than others would. I call these people Black Swan mitochondriacs. Humans that actually take the time, to read, research, and read about science that on the surface seems superfluous, but is foundational to our health on the submolecular quantum level.

Nature does not employ any “balanced protocol” in her usage. She is all about unbalancing the scales to favor the tasks that cells need to accomplish. NEVER FORGET IT.

And anyone who says otherwise will have to explain this new finding.

People who have open minds to accept new data that blows your paradigm of beliefs apart.

Why do living things prefer the lighter isotope of hydrogen than the heavier version of hydrogen called deuterium?

Heavier things make quantum mechanics “less probable”.

In the early 1980s, researchers first noted an unexpected effect when two hydrophobic surfaces were slowly brought together in water. At some point, the two surfaces would suddenly jump into contact—like two magnets being brought together.  No one knew why or how it happened.

Part of this research challenge was to realize that the hydrophobic interaction is unique to liquid water.   No other solvents are capable of this effect.  Living things are most made of water.    Your colony of mitochondria make water.

The smaller an object/atom is, the less strictly it is governed by the laws of classical physics, and the more it is subject to quantum effects. A tiny hydrogen atom is a quantum object—sometimes behaving like a particle, sometimes more like a wave. Deuterium, twice as heavy as hydrogen, is less subject to quantum effects. The consequence is that D2O is less destabilized than H2O when squeezed between two hydrophobic surfaces and the hydrogen bonds between water molecules get broken. It turns out life wants to shapeshift water’s hydrogen bonds with light to create the magic that life uses.

When mitochondria are non optimal they are making less water and they become hypoxic. This changes the delta psi or the electric charge on its membranes and this lowers the redox potential. We can see this effect via our endogenous glutathione levels on testing. This lowers the probability of these quantum nuclear effects in water making illness more likely over health.

This tells us that this effect is important in creating the living state.  This new research work shows how quantum nuclear effects in water become substantial on the nanoscale.  that is the scale your cells operate in.

Nature is capable of a lot more than we believe.

CITES:

“Nuclear Quantum Effects in Hydrophobic Nanoconfinement” by Buddha Ratna Shrestha, Sreekiran Pillai, Adriano Santana, Stephen H. Donaldson Jr., Tod A. Pascal and Himanshu Mishra, 31 July 2019, Journal of Physical Chemistry Letters.
DOI: 10.1021/acs.jpclett.9b01835

 

 

 

When a wise person speaks he understands fully the intent of the words, but what the ignorant person hears is subject to their present set of knowledge. There is an inherent disconnect. There is always a loss of information and energy transfer in this case which is why social media is not the ideal place to explain yourself in order to teach.

This requires the student to have skin in the game to understand things well and it is best done in person. Where misunderstanding dwells, misuse will not be far behind in the under-educated mind. It is far safer and wiser for the quantum biologist to remain on the solid ground of the physics of light and eschew the shifting sands of philosophic extrapolations found in modern physics. The ignorant among us can know things, but the point of life is to understand how all things link back to the fabric of nature and life.

Life also has another surprise for those with natural wisdom: When we talk sense to a fool they often try to label you foolish only because of their own ignorance. If your audience is not wise this can lead to meme creation and control of many other lean minds. The goal is to make the curious wise with nature’s wisdom quickly to overcome the wasteful inertia of a paradigm’s core beliefs. This is why I am allergic to a closed mind who buries curiosity. What is the core message for a curious mind? Just because you don’t understand it, doesn’t mean or imply that the truth is not being delivered to you. Your wisdom myopia does not trump the deep truths buried in nature’s laws. There is a science of light out there to understand, and to bring it to biology is to brighten everyone’s perspectives. Today, biology is in the dark ages of understanding how light works with and within cells and innovates life from abiotic atoms.

This must change if medicine is to advance. An age in science is called dark, not because the light fails to shine, but because people refuse to see what is already published in the literature but still have yet to understand how it links back to life. Their educational myopia allows them to remain in the dark because they continue to misunderstand the implications of this data.

How is fear like a plague when politics gets in the way of people’s lives?

In September 1923, the Great Kanto earthquake devastated large parts of Tokyo, mostly owing to firestorms.

Rumors spread, and were often repeated in the mainstream press, accusing Koreans, a despised and poor minority, of planning to take advantage of the disaster by starting a violent rebellion. Japanese vigilantes, armed with swords, bamboo spears, and even guns, then set upon anyone who sounded or looked Korean. The media always stokes fires to grow.

Up to 6,000 people were murdered as police looked on and sometimes took part. The media was complicit in those murders, but you’ll never read that in a history book.

This was not some uniquely Japanese phenomenon. Mobs massacring unpopular minorities remain all too common. When Hindus started killing Muslims in Delhi recently, the Indian police were as passive, or as culpable, as the Japanese authorities were in 1923.

One need not go far back in European or American history to find similar, or even worse, cases of lynching and mass murder.

The medium is the message.  Social media allows everyone to be the press today, but none of them do the the bidding of ‘We The People’.  The goal of the free press by the founders was to protect the people from the tyranny of government by getting to the truth.  Content is fire today, and social media is the gas that the media is using to try to change society to an agenda their supporters want.  The media no longer is free.  They are chained to those who pay for their ads.  The INDUSTRIAL part of the industrial military complex.

CONGRESS is doing the bidding of the corrupt NAIAD/FDA/CDC by using the pandemic crisis to incite hatred.  That hatred has collateral effects that Senators and Congress people should have anticipated.  They did not.  In the beginning, those non linear collateral effects should have been considered to lead to deadly consequences.

Now they have.

Conspiracy theories can become lethal when stoked by politicians or media.  Both of stocked those fears.  

The panic that often occurs during a health crisis or in the aftermath of a natural disaster can — and has — led to spasms of irrational violence.  

Politicians will always hide behind the idea of intent, when what they should concern themselves with is the ultimate effect of their actions.  

So, as “WE THE PEOPLE“ have seen this unfold maybe now you can see how “Antifa and Black Lives Matter” groups have gone from after thoughts to  current events.  

Who are these groups using to meet their agenda? 

Answer?  The media and  Mr. George Floyd, Mr. Jacob Blake, and Kyle Rittenhouse.  What are the facts we know at this point?    Floyd and Blake  forced his way into a woman’s home and pointed a gun at her stomach. Jacob Blake was wanted for domestic abuse and was terrorizing a woman when the cops were called. In the Me Too era it’s odd how the Left is making martyrs out of violent men who prey on women, don’t you think?

Rittenhouse was working as a lifeguard in Kenosha the day of shooting.  He went to clean vandalism at school after work.  He was not looking to kill people.  He was looking to protect his town from Black Lives Matter and Antifa terrorists.  Moreover,  The FBI has said  he did not carry his rifle across state lines.  The media has been silent on this.  The videos out there clearly show he shot two people in self defense.  That story is going uncovered by the media that supports Mr. Biden and Harris.  Have you asked yourself why this is the case?

You can’t raise money for Kyle Rittenhouse on GoFundMe — but they raised $2 million for this guy below.  You can’t defend Kyle Rittenhouse on Facebook — but you can defend this guy below

The politicians and media who let this genie out of the bottle, now can do little to contain it.  

This is why Mr. Biden and CNN’s Don Lemon are now trying to reign in looters and rioters. 

They are afraid of what pollsters are saying because their fortunes in November are now at risk.  This is the real prize for the politicians.  It is not about science or a pandemic.

We know what they are afraid of now……..and we know the cost they were willing to inflict on “WE THE PEOPLE” to get what they want…………..

 So what are you afraid of today?

For some people it’s heights. For others, it might be fear of illness or failure. For me, it’s wasting time, and people who focus on words over actions.

If you’re not running into the things that scare you, you aren’t really living, are you?

People often shy away from the things that make them nervous, because unless you’re afraid of showering, it seems a little like masochism to force yourself to face unnecessary things that can make you so unhappy. But consistently avoiding things that make us uncomfortable leaves us standing still in a world that is constantly moving. It doesn’t keep us safe; it keeps us behind the eight ball.

What is the evolutionary purpose of fear? Like GPS, our fears are our mind’s way of letting us know that we are at the edge of our comfort zone. Do you stay in that zone, or do you challenge the status quo daily?

It’s the risks we take, the times when something is hard but we march forward anyway, that adds spice to our lives and gives our memoirs the flavor a life needs.

Why should you look at your fears in the eye and smile? Nothing causes more consternation in a group of hypocrites than one honest person. Confidence is what we get when we take fear, face it, and replace it with something more useful.

“Courage is not the absence of fear; it’s having fear, but pushing through it.” This quote falls a little short of the point I want to make in this blog. This is not about supporting the left or right in this elections. Both parties are wings on the same bird. The media is mocking the bird by helping those who are abusing the people.

These ideas are not presented to sway you or scare you. They are being made to stoke your courage to speak up and no longer be silent. If you have been following my Twitter feed lately you’ll see that I am no longer remaining silent because of fear. You should chase after your fears because when your courageous act is complete, when you’ve pushed your deep-seated fears aside and chased after opportunity, at that moment you’ll truly be unstoppable.

“Nothing in life is to be feared, it is only to be understood. Now is the time to understand more, so that we may fear less.” —Marie Curie

It is time for “WE THE PEOPLE” to understand out government is at war with its people now.

Always remember: it wasn’t the rioting, looting, burned out buildings or dead bodies in the streets that made Democrats pump the brakes a little on BLM-

It was the polls finally turning against them. Nothing matters to the liberals but power. Nothing will break the left’s will to resist but another “shocking” victory in November, in my humble opinion.  Your beliefs might vary.

Did you know that melanopsin damage in the skin fat eyes or in your arteries is capable of causing anemia? How could this happen? Recent research has shown that depletion of mito-Super Oxide Dismutase, which acts as free radical in our mitochondria, inhibited succinate dehydrogenase activity leading to succinate accumulation in our colony of mitochondria. This also lowers the amount of light hydrogen isotope created from foodstuffs that is needed to reduce NAD+ to NADH at cytochrome one. The dual act seems to prevent nuclear DNA demethylation and inhibited red blood cell-like maturation from HEL cells. RBC maturation is a process dependent on DNA demethylation.

This appears how anemia of chronic disease occurs.   Anemia of chronic disease refers to having low levels of red blood cells as a result of how the presence of chronic diseases  (autoimmune diseases in which the body’s immune system attacks joints and/or body organs) or other chronic illnesses associated with low mitochondrial energy production.  People forget the first step of heme synthesis begins in the mitochondrial matrix so any level of mitochondrial damage limits heme synthesis for proteins.

Two hundred-billion red blood cells are renewed in our body every single day. These erythrocytes (also known as RBCs) are generated from bi-potent megakaryocyte-erythroid progenitor cells during a maturation process called erythropoiesis. Hypoxia stimulates erythropoiesis. Erythropoiesis is controlled very tightly and defects in the key elements of this step-wise maturation can lead to life-threatening conditions such as severe anemia or myeloproliferative disease. Transcriptional RNA interference-dependent and translational regulations are part of the core mechanisms required for proper erythropoiesis. Recently, an mRNA modification called N6-methyladenosine (m6A) has been found to control the expansion and self-renewal of hematopoietic stem cells.

In recent years, methylation at the N6 position of adenine (m6A) has gained the attention of RNA biologists because it seems control traffic in protein synthesis inside the cell. This implies that hypoxia is one of the key environmental signals to increase protein synthesis, mTOR, and uncontrolled growth in cells. The process of increased protein synthesis is controlled by the process of ubiquitination. Ubiquitination is controlled by the circadian mechanism in the human body. This links the amount of nnEMF, blue light exposure, or a lack of sun light to alterations in protein metabolism.

These are the foundational changes that support cancer generation when there is a chronic hypoxic stress stimulus.  Succinate accumulation in the TCA cycle in the mitochondrial matrix links mitochondrial MnSOD depletion to aberrant nuclear DNA methylation and these processes altered cell fates.  This is how alterations in energy production in mitochondrial causes an altered metabolism and can lead to diseases. 

Succinate accumulation is associated with increased production of reactive oxygen species (ROS) production due to an altered metabolism.  In the brain increased levels of succinate precedes neuronal injury, and plays a critical role in neurologic disease like epilepsy.  Epilepsy is also associated with hypoxia.

The production of ROS/mitochondrial ROS is associated with defects of mitochondria. For example, the production of ROS may be partly due to a defect in mitochondria that leads to altered energy consumption and energy dyshomeostasis.   There are many sources of mitochondrial ROS, including complex I (NADH dehydrogenase), complex II (succinate dehydrogenase, SDH), and complex III (coenzyme Q-cytochrome C reductase) of the electron transport chain (ETC).

Related studies have also indicated that the ROS signaling may be mainly derived from reverse electron transport (RET) to the ETC at complex I. Inhibition of complex I of the ETC induces mitochondrial ROS, oxidative damage, and increased neuronal loss

Messenger RNA is a flexible toolbox that plays a key role in the dynamic regulation of gene expression. RNA modifications variegate the message conveyed by the mRNA. Similar to DNA and histone modifications, mRNA modifications are reversible and play a key role in the regulation of molecular events.

Many people have asked me how chronic exposure to nnEMF or blue light can lead to cancer and a loss of control of this process is the answer to that question.

This is why when my members hire me to become their doctor at my Center in Destin their peripheral blood smear is one of the key things I look for that tell me about possible diseases that will be a problem for them in the future is they do not change the environments they allow.

CITES

1.       Kuppers DA, Arora S, Lim Y, Lim AR, Carter LM, Corrin PD, Plaisier CL, Basom R, Delrow JJ, Wang S, Hansen He H, Torok-Storb B, Hsieh AC, Paddison PJ. 2019. N6-methyladenosine mRNA marking promotes selective translation of regulons required for human erythropoiesis. Nat Commun. 10(1):4596. doi: 10.1038/s41467-019-12518-6.

2.       Zeng et al. 2018. Refined RIP-seq protocol for epitranscriptome analysis with low input materials. PLoS Biol. 16(9):e2006092. doi: 10.1371/journal.pbio.2006092.

3.       Dominissini et al. 2012. Topology of the human and mouse m6A RNA methylomes revealed by m6A-seq. Nature. 485(7397):201-6. doi: 10.1038/nature11112.

4.       Meyer et al. 2012 Comprehensive analysis of mRNA methylation reveals enrichment in 3′ UTRs and near stop codons. Cell. 149(7):1635-46. doi: 10.1016/j.cell.2012.05.003.

Present knowledge:  London, UK, hospital staff COVID19 antibody #seroprevalence is very high, with 34.7% for those with direct patient contact, and 22.6% for those with no patient contact. London’s general population is at 17.5%.

This is how Herd Immunity is achieved…

https://www.cambridge.org/core/services/aop-cambridge-core/content/view/1F3C3898F2D770164E356D8EA3418D6B/S0899823X2000402Xa.pdf/seroprevalence_of_sarscov2_antibodies_in_healthcare_workers_at_a_london_nhs_trust.pdf

The higher percentages are likely due to density with high-touch high viral load, which is expected in hospitals. This is similar to what was seen in Mumbai’s slums, Australian cruises and prisons across the globe.

There is growing evidence that T-cell immunity allows populations to reach herd immunity once only 10-20% are infected with SARS-CoV-2.

This would explain why a highly transmissible virus in densely populated areas peaked at 10-20% infected regardless of lockdowns or masks.

The pervasive misconception is that we have zero immunity against COVID-19. Based on this flawed understanding, epidemiologists projected that herd immunity is not reached until 60-70% are infected.

This is almost certainly wrong.

Of course, the media ignores this research. While antibodies against COVID-19 may only last months, T cell immunity can remain protective for years.

CONSIDER THE LINK OF THE SUN TO T-CELL FUNCTIONING:

Role of vitamin D in immunity during C19 is critical to get right.  Going outside in the sun is more wise than taking a Vitamin D pill.

A. Vitamin D is a major precursor for activation of T-cell (for any disease)

B. Vitamin D controls cytokines storm in COVID that causes blood clot & vascular damage of the organs like kidneys, heart, brain, and ultimately death.

STUDIES:

1. Review @SpringerOpen provides detailed insight on the role of Vitamin D in building immunity by modulating monocytes, dendritic cells, T and B cells.  https://link.springer.com/chapter/10.1007/5584_2018_246

2. Scientists  @uni_copenhagen have discovered that Vitamin D is crucial to activating our immune defenses & w/out sufficient intake of the vitamin, T cells will not be able to react to and fight off serious infections in the body. LINK: https://www.sciencedaily.com/releases/2010/03/100307215534.htm

3. In this ancillary study of a randomized controlled trial, authors found that short term HIGH-dose vitamin D3 significantly reduced CD4 T-cell activation compared to low-dose vitamin D3, providing human evidence that vitamin D can influence cell-mediated immunity. LINK: https://academic.oup.com/jcem/article/101/2/533/2810779

In a study of 23 people who survived SARS in 2003, every single one had memory T cells that recognized the SARS virus 17 years later. (Nature) https://www.nature.com/articles/s41586-020-2550-z

BACK TO THE PRESENT

Moreover, blood samples from all 23 individuals showed “robust cross-reactivity” against SARS-CoV-2.

This can be called crossover immunity.

Crossover immunity is not limited to just people who were infected with SARS years ago though. In the same study, in 37 persons with no history of SARS or COVID-19 (negative serology and/or samples taken before COVID-19), over 50% had SARS-CoV-2 specific T cells.

This is not surprising because there are at least 4 strains of coronaviruses that cause the “common cold”. The above study is not the only one to show this level of cross-reactivity.

In a study from April 2020, in 68 healthy donors never exposed to COVID-19, 34% had T cells that reacted to SARS-CoV-2. https://www.medrxiv.org/content/10.1101/2020.04.17.20061440v1

This finding was confirmed in yet another study published in Cell in June 2020 showing that 40-60% of unexposed individuals had T cell recognition of SARS-CoV-2.

The authors hypothesized that crossover immunity came from “common cold” coronaviruses. https://www.sciencedirect.com/science/article/pii/S0092867420306103?via%3Dihub

Crossover immunity may explain why so many young and middle-aged individuals are asymptomatic even when testing positive for coronavirus. This is why a positive test should not be alarmist with this RNA virus.

It is likely that their T cells recognized the virus and mounted an immune response before even mild symptoms surfaced. What does this mean?  This is how natural herd immunity without a vaccine occurs in Nature.  This is not a narrative that is being pushed in the media or medical circles right now.  You need to understand why.

All those runny noses from the common cold prepared our T cells to fight COVID-19.

Although it has been ominously called the “novel-coronavirus”, SARS-CoV-2 is yet another coronavirus with many similarities in structure to the common cold coronaviruses. Why are the elderly hit so hard by COVID-19 though?

Indeed the strain of coronavirus that we faced in 2020 is more lethal than those in the past, specifically in the elderly and immunocompromised…With an understanding of T cell immunity, it makes sense that the elderly are more affected by COVID-19.

It is well known that persons in advanced age and/or who are immunocompromised lose T cells. https://medicalxpress.com/news/2020-06-cell-immunity-elderly.html

Let’s get back to herd immunity via T cells.

If ~50% of people had T cell immunity prior to SARS-CoV-2, then that leaves 50% of the population susceptible.

In the regions hit hardest by COVID-19, serology studies show new cases and deaths peaked at around 10-20% infected. Adding the 50% who already had T cell immunity from common cold viruses to the newly infected 10-20% equals about 60-70% immunity.

Not coincidentally, 60-70% is the percentage epidemiologists project is necessary for herd immunity with a respiratory virus. It is likely that many of the hardest hit regions of the world (e.g. Lombardy, NYC, Madrid, London, Stockholm) are now at herd immunity.

Lockdowns & mask ordinances (mostly coming after the peak) likely had little effect, with the exception of perhaps prolonging the spread. Sweden is an example of what herd immunity looks like without lockdowns or masks.

Based on serology testing, ~20% of Stockholm was infected by April.

Deaths peaked in Sweden in April.

Today, the pandemic is over in Sweden with zero deaths per day and subsiding new infections. Lockdown advocates will challenge this thesis and point to Indian slums and areas in Peru that reached much higher infection prevalence.

However, malnourishment is rampant in these very poor regions…And it is well known that T cell function is reduced in the malnourished. This research on T cell immunity is largely being ignored by the mainstream media, possibly due to political and pharmaceutical interests.

Hint: Assuming $35 per vaccine dose (Moderna’s price), vaccinating just the USA alone will result in a revenue of ~$10 billion annually. Considering that the coronavirus vaccine industry has the potential to be the biggest profit maker big pharma has ever seen, it is not surprising that we are seeing an overly aggressive push for lockdowns and masks until there is a vaccine—no matter the cost to the public or governments.

A recent paper out in Cell needs to be highlighted.  

COVID-19, hijacks proteins in host cells that serve as master regulators of key cellular processes. These regulators are called kinases.  Sunlight has a massive effect on kinases involved with C-19 and this is why the class of viruses are considered seasonal RNA viruses.  By doing so, the virus is able to rewire the cell’s internal circuitry to promote its own spread and survival when the solar redox is poor to activate the kinase pathways in the body. But the reliance of the virus on host-cell proteins may also prove to be its Achilles’ heel, as these same proteins can be easily targeted with existing drugs that are cheap and off patent.  These are drugs that Big Pharma does not want anyone to know about because they want everyone believing the narrative the vaccine is the ONLY acceptable way out of this pandemic.

THIS IS FALSE

In a study published June 28, 2020, in Cell, the researchers found that when SARS-CoV-2 infects cells, it assumes control over a family of enzymes known as kinases. Under normal circumstances, kinases serve as master regulators of metabolism, growth, movement, repair and other important cellular functions. Kinases work by attaching tiny chemical tags to proteins through a process known as phosphorylation. This is how sunlight sculpts matter in your cells to alter your immunity and keep you safe.  Once attached, these tags act as switches that turn proteins on or off, which keeps the complex machinery of the cell running smoothly.

When a cell is commandeered by SARS-CoV-2, however, these same kinases behave in ways that disrupt normal cell function and transform the host cell into a virus factory. Cell division comes to a halt, inflammation pathways are activated, and the cell even begins to produce tentacle-like structures known as filopodia, which protrude from the cell’s surface and may serve as molecular highways that help the virus spread rapidly to neighboring cells.

This dependence of SARS-CoV-2 on kinases was revealed in experiments in which the researchers counted and catalogued all the proteins found in both infected and uninfected cells. Though they observed no significant differences in the total amount of protein found in each group, the scientists noticed huge disparities in phosphorylation levels – a clear sign that SARS-CoV-2 was changing kinase behavior in infected cells.

This work is being coordinated at UCSF in California. It is being done at UCSF’s Quantitative Biosciences Institute (QBI). Soon after COVID-19 emerged as a threat to global health, QBI Director Nevan Krogan, PhD, assembled a crew comprised of dozens of UCSF scientists representing a broad range of scientific disciplines in an effort to address the pandemic from as many perspectives as possible. The QBI Coronavirus Research Group (QCRG), as the cross-disciplinary team is now known, includes a number of scientists with expertise in kinases, as well as the drugs that can be used to disable them.  These drugs work against cancers.  You might be shocked to learn that hydroxychloroquine is one of these drugs.  You might begin to understand why the media and Big Pharma want to limit its use.  The media number one advertiser is Big Pharma.

For example, Dr Fauci’s NIH branch has placed a blockade in effect on Chlorquine in this cancer paper.  Ask yourself why he would do this?  Check the status on this article….EMBARGO….to be released at a later date…once you find a copy and read it….you understand why it’s been delayed….crooks against any good science that will limit the patent power for a new vaccine that Dr. Fauci and his Gates cabal will benefit from for sure.  It is a good paper to review for a Black Swan. Chloroquine (CQ) a drug closely related to hydroxychloroquine (HCQ) interfers with phosphorylation of extracellular signal-regulated kinases (ERK)1/2 and the ERK-activating kinases mitogen-activating protein/ERK kinase (MEK)1/2.  This decreases TNF which controls all the inflammatory pathways in the body that control mitochondrial autophagy.

At pharmacological concentrations,  CQ and HCQ have significant effects on tissue homeostasis, targeting diverse signaling pathways in mammalian cells. A key target pathway is autophagy, which regulates macromolecule turnover in the cell. It also recycles defective mitochondrial from direct C19 attack.  In addition to affecting cellular metabolism and bioenergetic flow equilibrium as I laid out in my July 2020 lecture here on Patreon, autophagy plays a pivotal role at the interface between inflammation and cancer progression. Chloroquines consequently have critical effects in tissue metabolic activity and importantly, in key functions of the immune system.  This is the major reason why these antibotic drugs work in viral diseases.  Dr. Fauci knew this information in 2005 as seen above.  Do you still trust the CV task force leader?

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7011648/

SUMMARY

Innate cellular immunity involves nonspecific cells like Natural Killer T cells. They are activated anytime we get exposed to a viral pathogen, known to us or not. They are like a sniper that is told to kill anyone that looks like they don’t belong, as in virally infected cells. They are not specific to any virus or family of viruses.

Adaptive cellular immunity mainly involves the TNF pathways that deal with CD4 and CD8 T cells. These are produced anytime you have a particular virus. They are like a Mafia hit man who is contracted to kill someone and everyone related to that person, so a Coronavirus or any of its cousins. More specific than the NK T cells but nonspecific enough to take out infected cells of a family of viruses if the individual has good cellular immune function. They also provide longer lasting immunity than antibodies for most respiratory viruses.

The humoral immune response involves the production of antibodies (Immunoglobulins IgM, IgG) by B cells that are activated by the cellular immune response if the threat is bad enough. Think about these antibodies like an assassin with an individual target, like Jason Bourne getting a photo of his target and going after that specific person. Antibodies are like a lock and key in their response, very specific to that particular virus and even that particular strain of virus. They do not often offer cross reactive defense to other mutations or related viruses in the family.
THIS is why I have been focused on helping myself, my family, and my friends improve the innate and adaptive cellular immune response, antibodies are fine but less predictable and don’t always last for this type of virus. Also the T Cell immunity offers defense against the future viral threats that we may face, I suspect there will be more.

That is a basic immune system lesson to use when you get confused! And remember Sunlight properly shapes life and health, how’s it shaping yours?  Artificial light improperly shapes your immune system and sets your up for mitochondrial damage making a C19 infection more likely.  Is that what you want now?

ADDITIONAL CITES:

https://pubmed.ncbi.nlm.nih.gov/31782148/

https://www.cell.com/cell/fulltext/S0092-8674(20)30811-4