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Light That Liberates Nitric Oxide Best in Humans?

If you open the centralized biochemistry book , you’ll find the following story: The liberation of NO in biological systems, particularly in humans, is often studied in the context of phototherapy or photochemical reactions involving NO donors (e.g., nitrosyl complexes or S-nitrosothiols). The wavelength of light that most effectively liberates NO depends on the specific NO-containing compound or context:

• Near-Infrared (NIR) Light (650–900 nm spectra):
  • NIR light is often cited as effective for liberating NO from certain endogenous stores, such as S-nitrosothiols or nitrosyl-heme complexes (e.g., in hemoglobin or mitochondrial cytochromes).
  • It penetrates tissues deeply due to low absorption by water and hemoglobin, making it practical for in vivo applications.
  • Studies suggest that NIR light can photolyze NO from these compounds, enhancing vasodilation and tissue oxygenation.
• Visible Light (400–650 nm spectra):
  • Blue light (400–500 nm) and green light (500–570 nm) can liberate NO from synthetic NO donors (e.g., nitroprusside or ruthenium nitrosyls) or biological complexes like nitrosylated hemoglobin.
  • These wavelengths are less penetrating than NIR but can be effective in superficial tissues or in vitro studies.
• Ultraviolet (UV) Light (300–400 nm spectra):
  • UV light is highly effective at photolyzing NO from some chemical donors (e.g., S-nitrosothiols), but it has limited use in humans due to poor tissue penetration and potential damage to DNA and proteins.

Most Effective in Humans according the biochemistry books

For practical purposes in humans, near-infrared light (around 650–850 nm) is generally considered the best for liberating NO from biological stores. This is because:

• It balances tissue penetration with photochemical efficiency.
• It aligns with the absorption spectra of some nitrosyl complexes found in blood and tissues.
• It has been explored in therapeutic contexts, such as improving blood flow or treating hypoxia.

That said, the “best” wavelength depends on the specific NO source (e.g., endogenous vs. exogenous donors) and the target tissue.

WHAT DID THE BIOCHEMISTRY BOOKS FORGET?

Fritz Popp found out that all human cells make ultraweak biophotons in the UV range. Shouldn’t this add a layer to their understanding in their books that might change their dogmatic opinion in would creation? The answer is they still do not know how to make sense of nature.

When the quantum biologist brings biophotons into the picture is does add an intriguing layer to the discussion about nitric oxide (NO), methemoglobin (metHb), and light interactions in humans. Biophotons are ultra-weak photon emissions produced by living cells and they should indeed influence how we think about NO liberation and its interplay with biological systems.

Biophotons in Human Cells

Biophotons are low-intensity light emissions (typically in the UV to near-infrared range, ~200–1000 nm) generated by oxidative processes in cells, such as mitochondrial respiration or reactions involving reactive oxygen species (ROS). These emissions are thought to play a role in cellular communication, regulation of biochemical reactions, or even redox signaling. While their exact purpose is still debated in centralized systems, but not in my system. Their very existence defines what life is all about because they suggest that human cells have an intrinsic capacity to produce light that could interact with photosensitive molecules—like those involved in NO dynamics, wound healing, and oncogenesis.

Revisiting NO and MetHb In DM #38 with Biophotons

• Biophotons as an Endogenous Light Source:
  • If human cells emit biophotons, particularly in the visible-to-NIR range (e.g., 400–850 nm), these could theoretically trigger NO release from endogenous stores like S-nitrosothiols, nitrosyl-hemoglobin, or other NO-bound complexes in or near blood vessels and tissues.
  • This mean that NO liberation isn’t solely dependent on external light (e.g., therapeutic NIR) but could be modulated by internal biophoton activity, especially in areas with high metabolic activity (e.g., muscles, brain). it turns out blood emits a steady stream of biophotons too.
• Wavelength Overlap:
  • Biophoton emissions span a broad spectrum (200-1000nm), but peaks in the 600–800 nm range (red to NIR) have been observed in some studies. This overlaps with the wavelengths I previously noted as effective for NO photolysis (650–850 nm). So, biophotons could, would, and should naturally contribute to NO release in vivo, potentially reducing metHb or regulating its levels as part of a feedback loop.
• Interaction with MetHb:
  • MetHb itself absorbs light, particularly in the 600–650 nm range (due to its ferric heme structure). If biophotons emitted nearby overlap with this absorption band, they might influence metHb’s redox state either by facilitating NO binding/dissociation or by sensitizing it to reduction back to functional hemoglobin.
  • This implies a subtle, localized mechanism where biophotons help maintain hemoglobin’s oxygen-carrying capacity under oxidative stress.
  • Nitric Oxide acts locally in human injuries, so any resultant the light stimulus for repair should be Ultraweak in nature. This means Pritz Popp’s work fits here for mammalian wound regeneration. Moreover, Popp has shown biophotons are also in the UV range So since we know that oxygen is the most critcal part of being able to make biophotons from Roeland van Wijk’s book and papers, this tells us that oxygen tensions in wound likely create specfic biophoton spectra to marry up to the tissue response. This tells me the process is entirely quantized.
  • These ideas fit perfectly with what Becker found in wound healing and regeneration in species. He told us human RBC had to de-differentiate to become a stem cell. It would make a lot of sense if the tissue hypoxia forced RBC that were HbO2 to become metHb. Why? MetHb is a hemoglobin built for an epoch were cells did not use much oxygen. This is precisely what happened during the Great Oxygenation Event on Earth. Hypoxia forms metHb in RBCs. MetHb is a more atavistic state of Hb and it is paramagnetic. This makes the RBC a new target for the photo-bioelectric current. This change inside of the RBC decreases the cystalline structure of Hb when metHb rises in an injury state. This slight change allows for de-differentiation to occur in step wise fashion. It is almost as if the cell is reversing time to go back in its evolutionary history when you observe what nature is teaching us via Becker’s experiments. It would seem to me the biophoton release from the blood would be quantized to oxygen levels in the injury to drive healing. Most wounds are hypoxic compared to the non injured state.
  • These ideas should lead the centralized biochemical paradigm to go to a complete reversal, because it refines the picture of how light can sculpt fully differentiated life. It breaks the Central Doctrine of biology that was set forth by the biochemists after DNA was discovered.

    External vs. Internal Light from our Tissues:

  • The biochemical focus has been firmly focused on external light sources (e.g., NIR therapy) as the “best” for liberating NO in humans. Biophoton creation suggest that internal light might already be doing this on a smaller more powerful scale, perhaps as part of homeostasis. Biochemists have no idea as scale shrinks the electromagnetic force gets STRONGER. They also are ignorant that the photon is the force carrier for this fundamental force. It is stronger than anything biochemist have in their tool box. This doesn’t negate the efficacy of exogenous NIR but it adds a layer of complexity, that cells are self-regulating using paramagnetic free radicals like NO, CO, H2S with metHb dynamics in RBCs for some distinct purpose that is not yet published in their text books.
    • Wavelength Specificity of Biophotons is broad: The broad spectrum of biophotons (UV-NIR = 200-1000nm) means that no single wavelength is “best” in an absolute sense. Instead, the most effective light for NO liberation could be context-dependent for the injury; external NIR for therapeutic boosts, biophotons release from mtDNA and from the blood for baseline physiological regulation.
    • Wound Creation Angle: The presence of biophotons in wounds from the blood hint at a more autonomous, light-driven cellular ecosystem than no biochemist would ever imagined. It’s almost like cells have their own “internal sun” influencing NO and metHb chemistry, which is a poetic twist on physiology. I was forced to learn all the biochemical pathways, but none of them explain how wounds are healed and regenerated. It also should make Becker and Marino smile because Jaffe and Handler and Swann work is getting ripped up by the roots by Uncle Jack like a weed. Why? Listen.
    • https://www.youtube.com/watch?v=YkBsVMjnwkA
    • The biochemists that Marino cited in the podcast above are why all of science has been blocked from Becker’s ideas. When I sat down with one such celebrated biochemist about 12 years ago, Ray Peat, he told me that biophoton intensity was only 10⁻¹⁷ to 10⁻¹⁹ W/cm², so it had no effect. Moreover, he compared it unfavorably to a 1 mW/cm² (10⁻³ W/cm²) NIR laser. That’s a 14–16 order-of-magnitude gap, which sounded damning to Peat, until you factor in scale and context that physics brings to the discussion:
      • Biochemical Bias: Peat’s reasoning leaned on macroscopic phototherapy logic = more watts, more effect. But biochemistry often overlooks how EM forces dominate at microscopic scales, where biophotons operate (e.g., nanometers to micrometers). I had a copy of Albert Szent Gyorgi’s 1968 book with me and I opened it up to a picture of a protein that showed its electronic structure. I asked Peat if he knew what Szent Gyorgi was trying to convey. He had no idea. The electronic structure means a protein has an absorbtion and emission spectra based on its amino acid sequence.
      • Photon as EM Force Carrier: Photons mediate the electromagnetic force, one of the four fundamental forces, and this force scales inversely with distance (Coulomb’s law: F ∝ 1/r²). At cellular or molecular scales (10⁻⁹ to 10⁻⁶ m), this force isn’t “weak”, it’s overwhelmingly strong compared to bulk chemical interactions. This was the moment when Peat became awfully quiet and listened to how the electronic structure of biochemicals would be controlled by endogenous biophoton signaling from the mtDNA and blood.
      • When scale shrinks light becomes like a laser.  Distance matters to photons. A biophoton emitted within a cell (e.g., from mitochondria) acts over distances of nanometers to micrometers. The EM force it carries can exert effects orders of magnitude stronger than a diffuse external laser beam penetrating centimeters of tissue. What people do not realize is that cells have the ability to use this endogenous light to reverse many things thought to be impossible because the light coming from within is exponentially strong than the light coming from outside the body. He was not expecting this turn of events. Below is an example. This implies these endogenous biophotons can also change metHb easily when the circadian biology of a cell allows for it.
      • 
      • Localized Power: A 10⁻¹⁸ W/cm² biophoton hitting a metHb molecule 10 nm away delivers energy with precision, is not diluted across a broad field. Compare that to a 10⁻³ W/cm² NIR beam scattering through skin, most of its energy is lost before reaching the target. Peat’s face was telling.
      • If biophotons are internal EM signals, their “weakness” is a misnomer due to a biochemists understanding of the power and their purpose. They’re not competing with lasers; they’re playing a different game no one sees, yet.
        • Internal Efficiency: Generated in situ (e.g., by ROS or mitochondrial activity), biophotons act where NO and metHb reside, inside cells or RBCs. No penetration barriers, no energy loss. An external NIR laser, even at higher power, wastes most of its photons before reaching the target.
        • Quantum Influence: Photons don’t just dump energy; they trigger quantized transitions inside tissues. A single biophoton with the right wavelength (UV or NIR) could flip metHb’s redox state of iron or it could liberate NO from an S-nitrosothiol, with no floodlight needed. The EM force ensures this happens with precision and strength at close range.

        Implications for NO and MetHb in Becker’s work.

        Let’s reframe the NO-metHb story with this Electro Mangetic lens:

        • NO Liberation: Biophotons, even at low intensity, could dominate NO release locally because their EM force acts directly on nitrosyl bonds (e.g., in Hb-NO or S-NO). UV biophotons (~200–400 nm) might cleave these bonds with surgical precision, far outpacing diffuse NIR therapy. This could be used to keep a cell hypoxic because of how NO blocks Hb from carrying oxygen. This is a remnant from the Great Oxygenation Event.
        • MetHb Reduction: The photo-bioelectric current I mentioned earlier stems from biophotons liberated in the injury site via EM interactions with metHb’s ferric iron. At nanoscale distances, this photon force would be quites strong to drive electron shifts (Fe³⁺ → Fe²⁺), reducing metHb to Hb more effectively than biochemical enzymes alone. This would keep the wound more hypoxic and less subject to ROS/RNS production.
        • Homeostasis: If biophotons are quantized to oxygen tension in injuries, their EM strength ensures they’re not just passive signals inside a wound but active drivers, fine-tuning NO and metHb to match cellular needs—way beyond what external light can achieve. This implies that biophotons might be critical in keeping a tissue hypoxic for a period of time by using magnetic parts of light to control the oxidation state of iron. This signal might be important in wound healing and regeneration sequencing.

        Centralized vs. Distributed Power

        My biochemical focus was “centralized” when i sat down with Peat, and he thought I was big on external light sources, like the sun, as the hero of the story. He found out quickly that was not the case. I explained to him biophoton production done by tissues injured suggested a distributed decentralized model for tissue sculpting in humans. My model showed him every cell, every RBC, is a powerhouse emitting EM force carriers, biophotons. This aligns with Becker’s amphibian-mammal split too:

        • Amphibians: Few people realizes amphibians have nucleated RBC that emit more light than mammalian blood that is enucleated. Nucleated RBCs amplify biophoton output, leveraging EM force for better regeneration stimuli. Their nucleated RBC are not great at carrying oxygen as mammalian blood is. Remember they are adapted to more hypoxic environments on Earth. This is why their RBCs are nucleated. This would make their “weaker” hemoglobin more responsive to these internal biophoton signals, sustaining metHb-driven dedifferentiation. Becker found this in his work with Salamanders. They were super regenerators. Peat’s face was white at this point.
    • Mammals: Enucleated RBCs lose this EM autonomy for regenertion, because they have to rely on biophotons released in the circulatory system and systemic biochemistry (e.g., cytochrome b5 reductase) over a more powerful localized photon power of the injured cell, favoring stability over plasticity. Mammals scar better than regenerate. Becker confirmed this in his work.Shifted Perspective by Understanding Biophysics

      This evolutionary history lesson of RBCs should flip everyone’s biochemical view: biophotons aren’t “limited” by their wattage, they’re potentially more powerful than external light from the sun is for NO and metHb dynamics in wound healing and regeneration. Their EM force, is dominant at small scales, and these factors would make them the true maestros of wound healing and homeostasis, not just a sideshow. External NIR might still have therapeutic uses, but it’s a blunt tool compared to the scalpel of internal biophotons.

    • NOW TO BECKER

      Let’s break this down:

      • Hypoxia in Wounds: Low oxygen tension (e.g., <20 mmHg in chronic wounds) stresses RBCs. HbO₂ oxidizes to metHb via ROS or NO reactions (HbO₂ + NO → metHb + NO₃⁻), This is especially true if NO production spikes from inducible NOS in inflammation due the wound.
      • Biophoton Shift: Hypoxia alters mitochondrial activity by lowering NAD+ and creating pseudohypoxia. When this occurs, it skews biophoton output. It creates a wider spectrum of light release that mimics what we see in the Domain of Bacteria and Archea. It appears that it does not enriching UV emissions because ROS excitation transitions are less prominent because oxygen is less prominent in the injury site. At the sime time ultraweak UV light decreases due to the loss of oxygen there is an expansion from 400-1000 nm light release. This fits with Popp’s work who told us about prokaryotes releasing more light than eukaryotes who are experts in using the TCA cycle and oxygen to generate massive energy from food. Van Wijk’s data on biophoton creation hints at this quantization to oxygen levels.
      • MetHb as the Injury Pivot: MetHb would act as a paramagnetic “sensor” replacing oxygen in the system as an intermediate. Interestingly, metHb absorption (e.g., ~630 nm peak) overlaps with biophoton ranges we’d expect to see in a hypoxic wound. The change in the biophoton spectra UV/blue to NIR photons can reduce it back to Hb or trigger more NO release. NO is also paramagnetic and it reduces energy production (below) to enforce hypoxia while delivering more blood to a wound that would bring more biophoton release to the wound generating a small photo-bioelectric current (as Becker’s work implied).
      • Dedifferentiation RBC Trigger: This small DC current, one trillionth of one ampere of DC, plus NO’s hyoxic signaling, would push RBCs toward a stem-like state, releasing factors (e.g., growth signals) to aid in wound healing and regeneration. Becker’s salamander studies showed dedifferentiation depended on bioelectric shifts and metHb and NO is the likely human analog, albeit less robust. Why? Amphibians and Humans do not have the same hemoglobin AMO physics.   One has a nucleated RBCs and the other does not.
      • Quantized Biophotons and Regeneration

        The decentralized idea at the cornerstone of my model is that biophoton release is “quantized to oxygen levels in the injury.” It is biophysical majesty not a biochemical reality because it suggests a feedback loop for Becker’s work that biochemistry does not have:

        • Low O₂ → More MetHb → more visible/IR Biophotons: Signals hypoxia, primes dedifferentiation, and kicks off repair. Injury and repair are linked.
        • Rising O₂ → Hb Recovery → more UV NIR Biophotons: Promotes angiogenesis (VEGF) and tissue maturation as NO vasodilation takes over. NIR restores energy production in recovery/regeneration timescale

        My model perfectly marries Becker’s photo-bioelectric currents to Popp’s biophoton coherence and van Wijk’s quantized oxygen links to tissue repair. In species with robust regeneration, this loop might be amplified due to a higher biophoton pulse, while in humans, it’s subtler because there is a huge biophysical difference Becker missed in biophoton release due to a lack if nucleated RBCs which have mitochondria. This is the reason why mammals heal but rarely regrow limbs is because mtDNA is a great source of biophotons. This also explains the phenomental regeneration of human fetus’s because they have nucleated RBCs when they are in the womb surround by amniotic fluid and fully hypoxic.

        Hemoglobin: Phylogenetic Divergence

        Hemoglobin’s structure and function have evolved significantly across vertebrates, reflecting adaptations to oxygen demands, environments, and regenerative capacity: This is why knowing your evolutionary history matters deeply to fully understand the wisdom in my decentralized thesis.

        • Amphibians (e.g., Salamanders):
          • Hemoglobin often has a simpler, less specialized structure compared to mammals. In mammals it is crystalline in structure and in amphibians it is not and does not carry oxygen as well as human hb does. In some species, it resembles ancestral globins with lower oxygen affinity (higher P₅₀), suited to aquatic or low-oxygen habitats.
          • Their RBCs are nucleated, a trait retained from early vertebrates, which allow greater metabolic flexibility, including dedifferentiation potential. This supports atavistic moves in cells during environmental changes. This makes them super adaptable to a changing landscape. This certainly was the case in the post Cambrian Earth. Remember how Huberman flubbed this on in my interview with him and Rubin? He has no idea the why humans would have so much amphibian opsin in their brains. The reason is mammals evolved from amphibians as oxygenation approached 21% and they TCA cycle replaced glycolysis.
          • MetHb formation and reduction might is less tightly regulated in amphibians, aligning with a physiology that tolerates hypoxia and supports regeneration. We see these atavisitc effects in humans in utero who are actively building out their body plans in extreme hypoxia.
          • Mammals (e.g., Humans):
            • Hemoglobin is highly specialized: It is tetrameric (α₂β₂), with cooperative oxygen binding and a lower P₅₀ (higher affinity), optimized for efficient oxygen delivery in warm-blooded, high-metabolism bodies that use the TCA cycle. Why? Mammals evovled after the Great Oxygenation Event, Cambrian explosion and the KT event when light became stable and when oxygen was plentiful in our atmosphere. Amphibians evolved much early when light was variable and oxygen was not as plentiful. Human Hb is liquid crystalline compared to amphibian Hb and this means they bind oxygen better than a salamander can.
            • Human RBCs are enucleated, which is a uniquely mammalian innovation that maximizes oxygen-carrying capacity but limits cellular adaptability (e.g., no dedifferentiation without extreme cues).
            • MetHb is actively reduced by enzymes like cytochrome b5 reductase (heme protein), reflecting a system geared toward stability in an oxygen environment that selects for TCA use rather than plasticity to regenerate your whole body.
          • Wound Healing and Regeneration: The Hemoglobin Link

            My decentralized  hypothesis finishes Becker’s life long work that oxygen tension, biophotons, and metHb dynamics drive healing and regeneration and this maps onto these differences elegantly:

            • Amphibians: Regeneration Superstars:
              • Hemoglobin Context: Lower oxygen affinity means amphibian Hb releases oxygen more readily in hypoxic wounds, amplifying local hypoxia. When you read his papers you can see the difference he noted but could not explain. This could shift RBCs toward metHb more easily, especially with ROS from the injury site.
              • Biophoton Role: Nucleated RBCs and metabolically active tissues emit a broader or but less intense biophoton spectrum because they are tuned to hypoxia. Why? mtDNA is the major SOURCE of biophotons in Nature. We now know mammalian blood also emits biophotons but the quality and character is not on par with mtDNA which can adapt its biophoton release based on oxygen tensions mtDNA senses in cells. Humans do not have any mtDNA in their RBCs, so they have lost this ability in RBCs but retain some biophoton release via neutrophils in the blood which are nucleated. The presence of mitochondrial DNA (mtDNA) angle is a game-changer, and it shifts the focus from just biochemical players like NO to the fundamental source of biophotons. This excess light mtDNA creates liberates massive amounts of NO from metHb or other stores, sustaining a prolonged “regenerative signal.”
              • Humans (Mammals): RBCs are enucleated and lack mitochondria (and thus mtDNA). This is a mammalian quirk which evolved for oxygen efficiency but it came at a cost. No mitochondria in mammalian RBCs means no internal biophoton generation in RBCs. Any biophotons in human blood come from other cells (e.g., leukocytes, endothelial cells), not RBCs themselves. The result: Human RBCs are passive oxygen carriers, not active EM signalers. MetHb form in hypoxia, but without mtDNA-driven biophotons, there’s no robust internal light to amplify regeneration signals for a sustained length of time.
              • Dedifferentiation: Robert Becker showed salamanders use bioelectric currents to dedifferentiate cells at wound sites, forming a blastema (a mass of stem-like cells). MetHb, as a primitive state, which acts as a redox chamber and photon hub (sun), to driving RBCs and other cells to revert phylogenetically, supported by their nucleated flexibility. This explains why the sun is critical in mammalian longevity. We cannot regenerate well because we have to rely on the exogenous source of light during wound healing. This fully explains the longevity benefit of man to primate because we lost our hair and made melanin with our newest semiconductor, melanin, to gain even more solar power.
              • Outcome: The amphibian system favors plasticity and this allows limbs to regrow because hypoxia, biophotons, and NO create a sustained “atavistic” environment, echoing early vertebrate development. Humans use this environment to birth their young, but they lose this effect as soon as they leave the womb and breathe.
            • Mammals: Scar Masters:
              • Hemoglobin Context: High oxygen affinity and enucleated RBCs mean mammals prioritize oxygen delivery over local release. As a result, hypoxia in human wounds is shorter-lived, and metHb is quickly reduced to Hb by enzymatic machinery, limiting its accumulation.
              • Biophoton Role: Enucleated RBCs don’t emit biophotons themselves, and mammalian tissues might produce a narrower, less UV-rich spectrum (more visible/NIR), reflecting higher oxygen baselines. This mean less NO liberation via biophotons in early hypoxia, favoring inflammation over regeneration.
              • Dedifferentiation: Mammalian RBCs lack nuclei, so dedifferentiation to a stem-like state (as Becker suggested) is rare and requires extreme conditions. MetHb would still form in hypoxia, but the bioelectric current it generates is weak and short-lived, insufficient for full blastema formation.
              • Outcome: The mammalian system leans toward stability, and scarring seals wounds fast, but regeneration is suppressed because hypoxia is short lived, as oxygen tension rise and Hb recovery outpace the “atavistic” window.

            Phylogenetic Hemoglobin and Healing Divergence

            The phylogenetic gap in hemoglobin ties directly to my quantized biophoton idea:

            • Oxygen Tension: Amphibian Hb’s lower affinity amplifies wound hypoxia, extending the metHb-biophoton-NO loop. Mammalian Hb’s high affinity shortens it, rushing tissues back to normoxia and TCA use.
            • Biophoton Spectra: Amphibians, with nucleated RBCs and less-specialized metabolism, might emit UV-rich biophotons that sustain NO-driven dedifferentiation. Mammals, with streamlined RBCs, lean toward visible/NIR emissions that support angiogenesis and closure, not regrowth.
            • Atavism: MetHb in amphibians mimics an ancestral state (e.g., early chordate globins), triggering regenerative pathways conserved from phylogeny. In mammals, metHb is a transient paramagnetic glitch, not a signal, due to evolutionary pressure favoring rapid repair over regenerative plasticity.

            Why the Difference? Hemoglobin as the semiconductor

            Evolutionarily, amphibians retained regenerative capacity because their environments (e.g., aquatic, variable O₂) favored adaptability over oxygen use, as a result, hemoglobin and RBCs stayed in a more “primitive” paramagnetic state to support this. Mammals, facing predation and thermal demands, traded regeneration for speed and efficiency and hemoglobin and RBCs evolved to lock in oxygen using the TCA cycle, not to linger in hypoxic, biophoton-driven states.

            Reptiles and Salamanders (Amphibians):

            • RBCs are nucleated and retain mitochondria with mtDNA, especially in amphibians like salamanders. Reptiles (e.g., lizards) also have nucleated RBCs with some mitochondrial activity, though it varies by species.
            • Result: Their nucleated RBCs are biophoton factories. mtDNA fuels mitochondrial ROS/RNS production, emitting a broader, more intense spectrum (UV-heavy, as Popp noted), especially under hypoxic conditions. This isn’t just about NO; it’s a full-on regenerative light show that goes on in these animals endogenously.
            • 

SUMMARY

I am hoping you have put these lessons all togther now and understand why amphibians are regeneration rockstars using primitive heme proteins. The fact that we use highly differentiated Hb is why we scar early and get cancer easily when we are not allowed to use the TCA cycle due to hypoxic signaling in cells. Today, our nnEMF environments create these signals making falling back into disease phenoptypes EASY.

This effect is amplified when we are blocked from sunlight having UV-IR light. This blocks out ability to use exogneous sunlight via melanin to augment our healing ability. This is why your modern world is creating every last chronic disease you can imagine. Awake now? We are our own Asteroid folks. This addiction to the biochemical paradigm supported by food guru ideas is a killer for humans. It has zero sophistication for the mechanism laid out in this blog and explains why billions of humans are at risk in a blue lit and nnEMF filled world.

My decentralized theory kicks the door in on the biochemical paradigm that nailed Robert O. Becker’s scientific life to a cross. Salamanders and reptiles regenerate better because their mtDNA-equipped RBCs flood wounds with biophotons, not just tweaking NO creation at the injury site. It also explains that as we turn off oxygen’s paramagnetic signal, we replace it with another paramagnetic signal in metHb production. This change drives a complete decentralized repair regeneration cascade controlled by electromagnetic signaling. This favored a broader spectrum of biophotons creation. Popp showed prokaryotes emit 5000 times more light than we eukaryotes. That fact is huge when you plug in evolutionary history I gave you here.

Mitochondria used to be bacteria so they retain this lineage of light creation via energy transformation. mtDNA-driven mitochondria emit more UV biophotons (from high-energy ROS transitions), which trigger DNA repair, protein remodeling, or cell dedifferentiation way beyond NO’s vasodilation or redox effects. Hypoxia creates the sun inside a wound of an amphibian. This hypoxia is an electromagnetic amplifier for their regeneration.

In wounds, low oxygen tensions ramps up mitochondrial ROS in these species’ RBCs, boosting biophoton output. This aligns with Roeland van Wijk’s oxygen-tension link; more photons, more regenerative signaling. It also aligns with Popps work too. Every box is checked.

My photo-bioelectric boost shows that Robert Becker’s currents in salamanders stem from this mtDNA-biophoton engine inside the blastema. Nucleated RBCs could use EM force (via photons) to polarize cells, forming blastemas. Humans, lacking RBC mitochondria, can’t polarize cells as well to sustain this. This is why depolarization in humans links to CANCEROUS human cells.

How do humans offset the inability of making biophotons to repair? Enter, melanin and sunlight exposure on their skin. Mammals need the external source of sunlight with UV-IR solar stimulus to finish the job of wound healing and guarrantee they NOT GET CANCER in an OXYGENATED ENVIRONMENT.

This is why CCO controls water production and apoptosis in mtDNA. The answer to cancer is built into our design but when we live under light that causes a chronic mtDNA hypoxia and we get no UV light, we never can tap Becker’s regenerative currents. Since Earth is heavily oxygenated today, the loss of heme proteins in mtDNA, creates the perfect storm to create cancer.

Just say NO to the ideas pushed in biochemistry that nitric oxide works the way they believe. They are beyond dead wrong. They have overfocused on NO liberation from metHb as the star of this show. They get No Quarter from me, no mea culpa. Billions have died because of their myopia. While NO matters (e.g., vasodilation, signaling), biophotons from mtDNA do way more. NO is used to keep wounds hypoxic in injury. Biochemistry still has no framework of why this is critical and why a return of NIR from mtDNA changes the oxidation state of iron to return tissues back to normoxia state where the TCA cycle can be used safely again.

• Stemness: UV biophotons directly influence gene expression via alteration of chromatin states, pushing cells toward a stem-like fate, as seen in salamander blastemas.
• Tissue Remodeling: A broader spectrum (UV to NIR) would orchestrate proteases, caspases, and growth factors (BCL-2), and ECM changes, not just rely on NO’s local biochemical effects.
• Energy Transfer: Photons are the EM force carriers and they shuttle energy and information across cells, syncing regeneration in ways mammals can’t replicate without mtDNA in RBCs. NO is also the electromagnetic signal they use to stimulate their stem cells from depots all over their body to regenerate.

Mammal vs. Amphibian/Reptile Gap Is Tied 100% to LIGHT, NOT FOOD.

• Humans: No mtDNA in RBCs = limited biophoton budget. Wound healing leans on systemic factors (e.g., macrophages, fibroblasts) with weaker, secondary biophoton input from non-RBC sources. Scarring wins over regrowth. Scarring however allows for oncogenesis if the cell remains in an atavistic state when oxygen supply comes back at the wrong time.
• Salamanders/Reptiles: mtDNA in RBCs = biophoton surplus. Wounds get a localized, intense EM signal, amplifying dedifferentiation and regeneration. Their “rockstar” status comes from this mitochondrial light advantage, not just hemoglobin or NO quirks.
• 

I think I’ve reframed Becker’s experiments perfectly using biophysics. Regeneration isn’t just about the injury hypoxia or NO liberation or the forced de-differentiation of metHb. It’s about mtDNA as the biophoton engine of creation. Salamanders and reptiles leverage this to flood wounds with light-driven EM force, dwarfing mammals’ capacity.

I hope you can visualize something larger now. There is bigger realization here why centralized medicine must be destroyed. These finishing touches to Becker’s thesis is fully decentralized and it explains oncogenesis and human development. When the injury is hypoxic and Becker’s current cannot be made, it is the perfect set up for cancer because you are feeding massive oxygenation into atavist cells. That is the cancer story.

Here is the other realization. Reading the first few lines of Genesis should now have a different meaning for you. This explains how a human child grows inside the womb. The germ line cells are kept hypoxic in an amnionitc fluid sac and that little “Salamander” stays connected to its mother’s liver by way of the umbilical cord. Many forget that during fetal life humans have fetal hemoglobin and those RBCs have mitochondria in them. Those RBC come from her liver.

That mtDNA is sculpting the child’s body plan using biophoton release from its parents germ line, just like Becker’s salamanders did. The amount of light a fetus makes supports creating a human from the germ line. This story is astounding when you realize it. It points out why our biochemical focus has blinded us from many truths and why biophysics has the answers for most of the chronic diseases now. We have built a world that simulates “an Earth” that existed before the Cambrian explosion when oxygen was rare.

What we have done to Earth with light and nnEMF is truly tragic. It is humanity’s asteroid. The injury stimulus that make is hypoxic and dehydrated making sure we run on a primative metabolic pathway that fosters atavism and then it never let us into the sun where UV and IR light awaits our body to create hydrated melanin sheets from CCO and melanin via POMC. It is stunning failure for humanity that centralized science and medicine allows.

CITES

The Body Electric, Robert O. Becker 1985

https://www.researchgate.net/publication/8465511_Biophoton_research_in_blood_reveals_its_holistic_properties

Pall, M.L. “Electromagnetic fields act via activation of voltage-gated calcium channels to produce beneficial or adverse effects.” J Cell Mol Med. 2013.

Yakymenko et al. “Oxidative mechanisms of biological activity of low-intensity radiofrequency radiation.” Electromagn Biol Med. 2016.

Leszczynski et al. “Non-thermal activation of stress pathways by mobile phone radiation in human endothelial cells.” Differentiation. 2002.

CENTRALIZED VS. DECENTRALIZED MEDICINE

I visited a friend who was admitted to the ICU
The whole hospital is blue fuckin lit.
The wards were sealed tight, not a breath of fresh air.
The sun fights its way in, but thick glass blocks the full solar light spectrum.
WiFi routers & medical equipment blasting EMFs 24/7.
Every surface is sterilized, yet the air feels dead.
There are no negative ions or grounding; it is just recycled air that is heavy with chemicals.
Machines beep like a never-ending alarm.
Walls, lifeless and grey.
The only greenery?
Plastic plants collect dust in the corner.
Oxygen is pumped through machines, but there is not a single breath of fresh air.
The doors are locked as if nature is the enemy.
How the hell is this a healing place?
You go broke using the place.
It is a fiat hell hole to which everyone is sent.

ALL CHRONIC DISEASES ARE LINKED TO THE WARBURG SHIFT

1. nnEMF-Induced pH Shift Drives Warburg Redox Shift: Light drives it, not food. The pictures above and below both show it in spades, but no one understands what these two pictures placed in blogs imply. The Warburg effect is characterized by increased blood glucose, glucose uptake, and lactate output, even under aerobic conditions. This reflects mitochondrial dysfunction (e.g., damaged IMM, cytochrome c oxidase inhibition). The damage of the IMM between NAD+ and oxygen allows for a 30 million charged field to escape into the cell. That charge then follows electric resistance pathways to cause many disease phenotypes. This is the stress response Selye talked about decades ago. He never was able to pin down how it began. All he knew was that it started on the HPA axis.

This podcast gave you the basics to the details in this blog: https://www.youtube.com/watch?v=IHSMqhrfvaU

The published science is precise that light stress by ALAN or a lack of sunlight drives metabolic shifts and how red light might serve as a therapeutic “drug” equivalent to counter the Warburg shift to change metabolism in ALL chronic diseases. In all stressors, vasopressin is the first stimulus to injury, and then the Orexin prism you learned about in QE #20 is next up in the cascade. Below, you see the definitive proof in two more pictures that red light reverses the effect of blue light exposure. This is what defines the Warburg Redox shift.

My photo-bioelectric hypothesis posits that nnEMF (non-native electromagnetic fields) and blue light damage melanopsin, mtDNA, and heme proteins, reducing DDW (deuterium-depleted water) production which leads to dehydrating melanin and increasing electrical resistance (éR) inside cells, leading to massive ROS/RNS rise, reductive stress (high NADH/NAD⁺), and Warburg metabolism (increased lactate). All of these cascades make oxygen a toxin because, due to its electronegativity, it pulls electrons across the IMM that is demolished. Why? With a stressor or trauma, excessive éR presents and leads to dissipative loss and bioenergetic inefficiency, reductive and oxidative stress, inflammation, molecular damage, and information loss. Thus, the hallmarks of disease and aging naturally arise from éR problems in the IMM on mtDNA. What you see and understand on the surface from biochemistry is not the entire biophysical story below built on the IMM.

Heme proteins are much older than mammals. This is a consequence of the evolution of our atmosphere. At the end of the Great Oxidation Event, heme regulation began to merge with POMC biology. Heme is a cofactor for many proteins, and it influences POMC expression via circadian and metabolic pathways. For example, heme binds to Rev-Erb-alpha and Rev-Erb-beta (nuclear circadian receptors), which regulate POMC transcription.

This links heme (and oxygen metabolism) to the light-driven circadian rhythms of mammals.

Heme proteins are much older than mammals. This is a consequence of the evolution of our atmosphere. At the end of the Great Oxidation Event, heme regulation began to merge with POMC biology. Heme is a cofactor for many proteins, and it influences POMC expression via circadian and metabolic pathways. For example, heme binds to Rev-Erb-alpha and Rev-Erb-beta (nuclear circadian receptors), which regulate POMC transcription.

This links heme (and oxygen metabolism) to the light-driven circadian rhythms of mammals.

THE DECENTRALIZED CASCADE TO THE ETIOLOGIES OF CHRONIC DISEASE

• Organisms as Open Thermodynamic Systems:
  • Organisms are open thermodynamic systems dependent on energy flow (e.g., sunlight, food) and material exchange (e.g., waste export). Spent energy contributes to entropy, defining the flow of time. Molecular clocks (e.g., PER1/PER2) act as flowmeters of entropy, measuring cellular energy dynamics.
• Cells as Dissipative Structures:
  • Cells escape the second law of thermodynamics (entropy increase) by creating order from chaos, functioning as dissipative structures. This order is built around the AMO (atomic, molecular, and optical) physics of atoms in cells, with molecular arrangement critical for function.
• Water’s Role in Cellular Organization:
  • Water is critical in the molecular arrangement of biomolecules, influencing their physiological function. The movement of water (e.g., via hydration shells and gradients) changes biomolecular dynamics, mimicking semiconductor physics in silicon circuits (where electric power generates light). Cells use water to create and manage light (e.g., biophotons), maintaining atomic molecular order. Pure water contains the field of charge stored on the IMM between NAD+ and oxygen.
  • Temperature affects semiconductors’ band gap size, too. You got this lesson in QE #29. Now you will find out how to link to your own disease.
  • Cooling increases band gap size. Water changes the temperature of the semiconductive proteins inside of use, which generates electrical resistance.
  • Cooling gave us evolutionary pressures to create catecholamine chemicals in prokaryotes like dopamine and adrenaline using a new semiconductive protein inside cells called melanin. When it degrades, it can become L-DOPA, which can become dopamine and adrenaline under a hypoxic stimulus.
  • A lack of oxygen affected the early oxygen carrier semiconductive molecules called heme proteins.  Recall that Robert Becker’s work around bone regeneration concerned itself with transforming RBCs to a pluripotential cell via a pico to nano ampere currents. The initial oxidation of hemoglobin to the ferric (Fe3+) state without oxygen converts hemoglobin into a useless “hemiglobin” or methemoglobin, which cannot bind oxygen.
  • KEY POINT NO ONE REALIZES: This step exists in hemoglobin biology to deactivate hemoglobin’s ability to carry on its adult physiology to travel back in time atavistically to create Becker’s regenerative current. Becker’s de-differentiation experiment never looked at the blood cells that were being transformed. If you understood the evolutionary history of hemoglobin creation, this was a methodological error. It occurred during the GOE.
  • Methemoglobin is an ancient protein used by life long ago when oxygen WAS NOT prominent in our atmosphere.  This was when all life on Earth had an “allergy” to oxygen because oxygen was toxic to bacteria and archaea then. Methemoglobin naturally builds up in human injuries to precondition the cell to return in time. If the cell is filled with oxygenated hemoglobin, it cannot generate Beckers’ regenerative current. One thing that can be used to help this process is methylene blue. But MB has to be used at the right time. The first week is the incorrect time to use it. Methylene blue has two major effects that are beneficial for humans who cannot generate Becker’s healing currents.
  • NUMBER 1: It alters the eR on the IMM in a damaged state, and
  • NUMBER 2: it can increase NO delivery to tissues that become hypoxic with a lowered NAD+. This increase in NO allows mammals to access their stem cell depots to repair the damage in question. The stressor and damage are always linked to tissue-level hypoxia or pseudohypoxia, where NAD+ has dropped.
  • 
  • Early versions of hemoglobins suffered from this problem = myoglobin.  Later, mammals figured out a novel way to stabilize hemoglobin using green light when oxygen filled the atmosphere.  First, I have to explain how we got there.  Modern versions of hemoglobin in normal red blood cells are protected by a reduction system in the RBC by an aromatic amino acid (histidine) and a sea of electrons from the water in the blood. The surface of Earth was getting pounded by UVC light for long periods.   Since life was prokaryotic and anaerobic 2.7 billion years ago when cyanobacteria evolved, it is believed that oxygen acted as a poison and wiped out much of anaerobic life, creating an extinction event of the old guard in life, including LUCA.  LUCA = last unknown common ancestor.
  • This environmental change drove evolutionary pressures to innovate proteins that used aromatic amino acids to build the most critical parts of the modern metabolism we see today in cells.  Their absorption spectra can go from 150nm VUV to 400 nm UV-A light.  Melatonin is one of the most ancient semiconductors known.  Its functions have evolved as the atmosphere of Earth changed its atomic concentrations. This is why leptin has 220 nm absorption spectra when you look at it
    
  • Melatonin, NAD+/NADH, dopamine, adrenaline, leptin, epinephrine, etc. Light controls the flux of all these biomolecules because of the movements of H+ in cells.  Remember, all enzymes that create these chemicals use proton tunneling to get the job done.  Proton tunneling is linked to the HIF-1 alpha and PER2 gene that uses light to increase the periodicity of cell molecular clocks. Increasing periodicity = better clock management.
  • Tryptophan, another aromatic amino acid, became very useful to cells living in an oxygenated world as a “time crystal” for cells because it has only one DNA codon, and its catabolism changes as light/dark signals change with the tilt of the Earth that gives us seasons. NAD+ and melatonin are both made from tryptophan. NAD+ and 95% of our melatonin is created in mtDNA. You might want to go back and re-read QT-14 now.
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• Future life forms would need this information because oxygen gave us cool and hot seasons in one year as the Earth revolved.  This cyclic pattern was built into cell metabolism as it got more complex, as the slides above show.
• Since oxygen has a high redox potential, it acts as an ideal terminal electron acceptor to generate energy after a nutrient breakdown. Oxygen soon became indispensable for metabolic activities. Organisms also evolved strategies to detoxify the reactive oxidative species that resulted from aerobic metabolism. Told ya’ that Vermont talk was important.

Though sequencing and phylogenetic analyses estimate the evolution of ROS-detoxifying enzymes even before the advent of aerobic microbes, the Great Oxidation Event acted as the catalyst to shape the directed evolution of enzymes like superoxide dismutase (above) and catalase.  Catalase (above) is one of the earliest heme proteins, ancient myoglobin, and hemoglobins.

• • Note the wide-band semiconductor components in the picture above: the Fe-S dopant semiconductors and their roles in creating the free radical signal in mitochondria.  Below, note how mammals’ chromophore proteins are linked to aromatic amino acids, heme proteins, and melanin at some level. Can you guess why yet?
  • As oxygen continued to mushroom, the high ionosphere became filled with a new gas that decreased the terrestrial solar spectrum.  Life had to react to this, and it fueled changes in heme proteins that appeared on Earth’s surface in early life forms.

    Oxygen was also responsible for forming the ozone layer in the atmosphere. The UV radiation from the sun split oxygen molecules (O2) into two atoms of oxygen, which then reacted with another oxygen molecule to generate ozone (O3). Ozone acts as a natural sunscreen for Earth to prevent harmful UVC and parts of the UV-B radiation from reaching the Earth’s surface.  This reduction began the evolution of new semiconductors in the two DOMAINS of life on the surface of the earth called melanin.

    As oxygen continued going higher, it fueled the Cambrian explosion, and life was able to take advantage of the mirror image of the photosynthetic arm of life on Earth.  Namely, mitochondria developed.  Complex life captured mitochondria in their tissues as a stowaway to transform solar energy into CO2 and water.  It transferred electrons from food to oxygen to fuel this solar battery.  At this point, life exploded, and all the complex life on Earth we know about today showed up almost overnight.  Eukaryotes came from the fusion of the other two Domains in endosymbiosis.  We believe chlorophyll and mitochondria were also innovated at this time.

     

    Stress, Vasopressin, and Water Conservation:

  • Stress and trauma (e.g., nnEMF, ALAN) disrupt water dynamics, triggering vasopressin release to conserve water. This mimics the VP-ISR-GDF15 axis activation in modern mammals. This is done because injuries all induce dehydration, and hypoxia of our semiconductors facilitates increasing éR, entropy, and cellular chaos, driving most chronic disease phenotypes. This mimics life during the GOE.
  • When the human body is sick, diseased, and energy inefficient from any stressor or stimulus, its evolutionary directive is to eat itself removing all the diseased cells and organelles and cancerous cells. To do this, it needs to generate a regenerative DC to do so. This current relies on the ability of heme proteins to make water to hydrate melanin to create this small current. When the cell cannot do this for any reason, big problems await this organ. The disease begins to spread like an infection does through the organ. We call this heteroplasmy.
• HIF-1 and Cellular Hypoxia:
  • Poor sunlight, darkness, ALAN/nnEMF, and geoengineering (e.g., aerosol-induced dimming) lower solar EMF (UV-A/B), which induces cellular hypoxia and blocks Becker’s regenerative current from the beginning. Without reestablishing cytochrome c oxidase to make DDW hydrate melanin, you never get to produce the one trillionth of one ampere of current needed to turn stem cells into de-differentiated pluripotential cells that can heal damage. Moreover, UV-A light stimulates NO, which controls mammals’ stem cell depots. So this is also turned off. Hypoxia-inducible factor-1 (HIF-1), stabilized under low oxygen or light stress, shifts metabolism to glycolysis (Warburg shift), increasing lactate production, and lactate use drives éR, which drives tissues into the Warburg redox shift. The pictures at the beginning of this blog are all there for you to review.
• HIF-1 and PER2 Link:
  • HIF-1 belongs to the same protein family as Period 2 (PER2), a core circadian gene. Liu et al. (2012) show PER2-HIF-1α-dependent regulation of SIRT3 (sirtuin 3) under hypoxic conditions, modulating the choices between TCA cycle flux and glycolysis. Sunlight enhances PER2 expression, optimizing oxygen use, while ALAN/ darkness/geoengineering disrupts this, favoring HIF-1α and Warburg metabolism. If you read the links on my forum in the Mitochondrial Thermodynamics DIY, you’d already know all of this—->https://x.com/DrJackKruse/status/1613298172801044482

    Red Light as a Therapeutic Equivalent: Tiina Kuru

  • 43% of the sun is Red light (e.g., 600-1000 nm), and PBM/LLLT can mimic sunlight’s effect, upregulating PER2 and downregulating HIF-1α, reducing éR and Warburg metabolism. The red light was critical in the evolution of all heme-based proteins after the Cambrian Explosion. Tiina Karu’s work supports red light as a “drug” equivalent, enhancing mitochondrial cytochrome c oxidase and oxygen utilization, countering the cellular effects of dehydration and hypoxia in an oxygen-filled atmosphere.
  • PER2-mediated ischemic preconditioning of the heart links sunlight/red light to optimal cardiac oxygen delivery, disrupted by mtDNA damage at cytochrome c oxidase (favoring Warburg shift).

• Historical and Evolutionary Context:
  • The Great Oxygenation Event (driven by sunlight/photosynthesis) coupled oxygen, sunlight, and PER2, enabling mammalian survival. Dinosaur extinction (darkness, cold) reflects HIF-1 explosion in non-therapod dinosaurs, while mammals adapted via PER2. Modern sunlight avoidance (geoengineering, indoor living, atrophic skin, ALAN) increases cardiac/PAD death and hypoxia-related diseases. With sunlight from the KT event, trillions of photosynthetic algae could now grow uncontrollably to make oxygen, transforming the entire planet’s atmosphere and setting up the perfect storm for the evolution of a mammalian mitochondrial world post-KT until human technology changed the signal when we innovated ALAN.

    Disease Implications:  

  • Blue Light/nnEMF and Vitamin A Liberation from opsins:
    • Blue light and nnEMF liberate Vitamin A from cells and membranes, raising its presence in blood plasma as an aldehyde (retinaldehyde). This lowers plasma Vitamin C and D levels, disrupting local antioxidant defenses (melatonin/ISR) and circadian signaling. Retinaldehyde destroys small molecule modulators of the mammalian circadian mechanism, including PER1 and PER2, critical gears in the eye clock of the SCN. Rev Erb Beta and alpha are the destroyed nuclear circadian receptors that are also heme-based.
  • Impact on Circadian Rhythmicity and mtDNA:
    • Disrupted PER1/PER2 periodicity (from retinaldehyde toxicity) leads to quantum timing loss in mtDNA, accelerating mutations. Human mtDNA mutates 15–20 times faster than nuclear DNA (vs. 5–10 times in primates), and nnEMF accelerates this further, driving faster epigenetics and chronic disease creation. This is why MAHA is HAHA. They remain myopically focused on food when light is the driver of all chronic diseases.
  • Cellular Disorganization and Energy Cost:
    • mtDNA mutations of the heme-based proteins cause cellular disorganization by impairing information processing (energy = information in physics). Energy is trapped in cells’ electronic level, stored in vibrational/electronic bonds (e.g., PER/HIF-1), membranes, gradients, fields, and mtDNA-structured water. nnEMF increases ubiquitin marking, raising protein turnover, robbing energy, lowering redox state, and increasing éR, spreading heteroplasmy across organs and manifesting many new disease phenotypes.
  • mtDNA Vulnerability:
    • mtDNA is more vulnerable than nuclear DNA due to: (1) higher oxidative stress (mitochondria as ROS source), and (2) the matrix-side negative membrane potential concentrating lipophilic cations 1,000-fold, amplifying nnEMF effects. This charge density change mimics vasopressin’s effect on water (negative charges QE has a blog on it too!), linking light to metabolism and controlling all the biochemicals to control it (Light > Food).

    Warburg Shift and Heteroplasmy:

    • nnEMF increases epigenetic expression, raising mtDNA error rates and heteroplasmy (normally 10% per decade, accelerated by nnEMF). This energy-intensive process lowers the redox state, increases éR, and drives Warburg metabolism (increased lactate, glycolysis), fueling disease (e.g., ALS, cancer, diabetes, obesity).
    • Warburg-like metabolism (increased glucose uptake, lactate) is a hallmark of cancer (FDG-PET), atherosclerosis, sarcoidosis (granulomas), and neurological conditions (e.g., schizophrenia, elevated lactate). The Ras-ERK-PI3K-mTOR axis, via mTORC1, stabilizes HIF-1α, promoting glycolysis (HK-II, GLUT1) independent of oxygen and driving disease progression.
    • Compared to complete glucose oxidation, Warburg metabolism generates less ATP stochastically but creates a higher éR, which enables more rapid glycolysis (10–100 times faster) during the light stress event. This fuels macromolecular synthesis (e.g., via the pentose phosphate pathway [PPP] and fatty acid synthesis), supporting cell growth and division.
    • The rapid shift to Warburg metabolism is triggered by changes in the organism’s light environment (e.g., nnEMF/blue light vs. solar EMF), a phenomenon misunderstood in biochemistry due to the neglect of biophysics and photo-bioelectric currents. Red light drops blood glucose and insulin, while blue light and nnEMF raise blood sugar and insulin levels. The pictures above state that case. Any nnEMF disrupts mitochondrial electron flow, increasing éR and lactate, while solar EMF (UV-A with IR) restores NAD⁺/DDW, lowering éR and supporting oxidative phosphorylation of the TCA cycle. This is why you need to see AM sunlight to use the TCA cycle. Every time the day’s first light is not the sun, you just Warburg shifted your LIFE toward disease.

Elevated lactate and éR drive reductive stress, and this recycles NAD+ to NADH rapidly (high NADH/NAD⁺), signaling the release of vasopressin, which initiates the Integrated Stress Response (ISR) and this activates GDF15, which suppresses anabolism but supports rapid energy needs (e.g., cancer proliferation, retinal repair). This dual role explains pathological (e.g., AMD, cataracts, retinal damage, RP progression) and adaptive (e.g., retinal resilience) outcomes. ALAN turns on the VP-ISR-GDF15 axis while red light radiation seems to turn it off.

Prediction: High nnEMF exposure in Big City USA like NYC, intensified by 5G and ionospheric radiation, alters cellular pH (e.g., <7.35), driving a Warburg redox shift and contributing to the crash at an early age for humanity

• Mechanism: nnEMF activates VGCCs, increasing Ca²⁺ influx and ROS/RNS (peroxynitrite, ·HO), disrupting the NAD⁺/NADH ratio and lowering cytochrome c oxidase activity (pseudohypoxia = low NAD+). This reduces DDW production, dehydrates melanin, and increases NaCl concentration, dropping intracellular pH below 7.35 (-20 mV) and membrane potential toward zero mV. The night owl lifestyle and blue light exposure amplify free retinal toxicity, damaging photoreceptors (melanopsin, NO, Rev erb /alpha-beta, cytochromes), while endurance stress (exercise) and/or mold burden (low alpha-MSH) enhance glutamine addiction via GDH dysfunction. This “toxic barrel” shifts metabolism to glycolysis, causing mitochondrial failure (energy crash) and neuronal dysfunction (cognition crash).
• Vasopressin’s Role in ISR Activation: Vasopressin (VP), a hormone released by the hypothalamus in response to osmotic stress, dehydration, or nnEMF-induced cellular stress, activates the ISR by stimulating eIF2α phosphorylation via eIF2α kinases (e.g., GCN2, PERK). This global translational arrest induces stress-responsive genes (e.g., ATF4, CHOP) and upregulates GDF15 (growth differentiation factor 15), signaling energy resistance (éR) systemically. nnEMF and artificial light at night (ALAN) increase VP secretion by disrupting melanopsin signaling and inducing osmotic stress (e.g., via dehydrated melanin). VP activates ISR, enhancing GDF15 production, suppressing anabolism (e.g., protein synthesis in the damaged area blocking regeneration), and promoting catabolism, exacerbating chronic conditions under reductive stress (high NADH/NAD⁺, lactate).
• Outcome: Low pH (e.g., intracellular pH <7.3), high lactate (blood >2 mmol/L), and low NAD⁺/NADH ratio (<1), measurable via blood gas analysis and metabolomics.
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• 

2. Dehydration from nnEMF lowers éR and pH —–> https://osf.io/preprints/osf/hgnmj_v2

• Prediction: nnEMF-induced dehydration increases conductivity (high NaCl, low water), reducing éR and pH and preventing recovery post-crash.
• Mechanism: nnEMF dehydrates cells, raising NaCl concentration and lowering pH (e.g., <7.35), reducing the membrane potential (-90 mV) toward zero mV. Dehydrated melanin becomes conductive, amplifying ultraweak biophotons and ROS/RNS, disrupting Becker’s regenerative current.  Free retinal toxicity from nnEMF/blue light damages catalase and cytochromes, impairing H₂O₂ breakdown and DDW production and exacerbating the redox Warburg shift (light).
• The patient’s lack of solar EMF (night owl, indoor living NYC life) prevents pH restoration, perpetuating mitochondrial stress. People forget that before eukaryotes, only two domains of light used the Warburg metabolism because of the light present. It had nothing to do with food. This defined the Cambrian explosion and KT events, as I laid out on the Huberman/Rubin Tetragrammaton podcast. String the lessons together and integrate them. No one is.
• Outcome: High serum osmolality (>300 mOsm/kg), low éR (microcurrent <1 µA), and persistent fatigue/cognition deficits, measurable via pH electrodes and imaging.

3. Warburg Shift Reflects Light Environment Choice, not a diet

• Prediction: The patient’s exposure to nnEMF/blue light (vs. solar EMF) altered the mitochondrial free radical blueprint, driving the Warburg shift and cognitive crash of people in a nnEMF environment.
• Mechanism: nnEMF (5G, blue light) and night owl behavior increase AMPK and ROS/RNS via VGCCs, lowering NAD⁺ and pH, shifting metabolism to glycolysis (Warburg light effect). Solar EMF (UV-A) supports quantized free radicals and redox potential, but its absence (due to indoor nnEMF dominance) disrupts cytochrome c, enhancing glutamine addiction. Free retinal toxicity damages photoreceptors, amplifying this shift, while mold exposures (low alpha-MSH) and endurance stress compound mtDNA heteroplasmy.
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• Outcome: High AMPK activity, low cytochrome c activity (enzyme assays), and elevated glutamine/glutamate ratio, reversible with solar EMF.

  4. AM Solar EMF Restores pH and Redox Potential

  • Prediction: AM solar exposure (30–120 minutes daily) restores cellular pH (7.35–7.45), reverses the Warburg shift, and improves energy/cognition.
  • Mechanism: AM sunlight (UV-A, visible light) reduces free retinal, regenerates melanopsin/cytochromes, and boosts cytochrome c oxidase for DDW production, raising NAD⁺/NADH and pH to -20 to -25 mV. This hydrates melanin increases éR, and counters nnEMF-induced dehydration, restoring membrane potential (-90 mV) and bioelectric signaling. Solar EMF’s quantized free radicals support redox potential, mitigating the Warburg shift and mold effects.
  • Outcome: pH normalization (7.35–7.45), improved NAD⁺/NADH (>2), energy (fatigue scale <10), and cognition (MoCA >28) within 8 weeks.

  5. Comprehensive Redox Protocol Prevents Progression To FORMAL CHRONIC DISEASE

  • Prediction: A redox-first protocol (AM sunlight, grounding, DDW, PBM, hypertonic saline, methylene blue) restores pH/éR, reverses cognitive crashes, and prevents neurodegeneration.
  • Mechanism: AM sunlight and PBM (810 nm) regenerate photoreceptors/cytochromes, grounding reduces ROS/RNS, DDW enhances DDW production, hypertonic saline improves conductivity, and methylene blue repairs mtDNA, raising pH and éR. This requires an MD to implement who knows how to assess patients. This counters nnEMF/blue light, free retinal, and the Warburg shift, restoring mitochondrial and neuronal function. Detox (mercury, mold) must follow REDOX to clear the “toxic barrel,” preventing re-toxification.
  • Outcome: Sustained pH (7.35–7.45), VCS score >7, energy/cognition recovery (MoCA >28, fatigue <10 at 6 months), and reduced Alzheimer’s risk (normal amyloid-beta on CSF).

  My decentralized interpretation of the Warburg effect is a marker of circadian mismatch, not a fuel source. Moreover, it is well supported by recent findings in the literature. The old idea that fuel source is critical is now on life support. As I’ve described, artificial light at night disrupts NAD+/NADH balance, increases ubiquitin, and drives glucose spikes “to brake” ubiquitin marking. AM UV and natural light cycles restore NAD+, lower ubiquitin, and normalize metabolism, reducing cancer, obesity, ALS, and autoimmunity, as I’ve emphasized with plants and animals sharing this response in the ubiquitin series on my website. The picture below lays it out AGAIN.

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• All of these proteins above are heme-based and have tryptophan embedded in them. They explain why chlorophyll came first in evolution, heme proteins came second, and now we are at melanin today. All of this was tied to light variation, temperature changes and oxygen tension variation in the atmosphere of LIFE.
• Altered Mitochondrial Metabolism: Blue light/nnEMF oxidizes heme in cytochrome c oxidase (MT-CO1), stalling oxygen reduction to water. This leaves O₂ as ROS, which excites molecules (e.g., lipids, proteins) to emit biophotons. Warburg redox shift (light) controls metabolism photo-bioelectrically (glycolysis dominance) because it operates when the inner mitochondrial membrane loses delta psi. This increases ROS from incomplete ETC activity (e.g., Complex III leakage via MT-CYB), amplifying biophoton output. This is why mitochondrial damage always leads to more biophoton release, spreading the failure at the speed of light.
• 
• Photo-Bioelectric Networks: Biophotons travel through cellular water, extracellular matrix, and bioelectric fields (e.g., gap junctions, membrane potentials), signaling mitochondrial distress. Popp suggested biophotons regulate cellular quantum coherence; in my model, their chaotic increase under stress disrupts this, spreading dysfunction. This is why the nuclear spins of atoms are lost. You need optimized optical pumping to maintain quantum coherence for health and longevity.
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• Are there other mechanisms in jabs that explain rapid-onset dementia, cognitive decline, and prion-like changes? Could it be the source of the negative polarity in things that give a thing a net negative flux?  Recall that all living things have a serious net negative charge.  Also, recall that water goes from a neutral polarity in its bulk state and then gains its net negative charge in the structured state when UV and IR/NIR light excite water. This structured state excludes protons and creates coherent domains that act like electromagnetic capacitors for the crystalline lattice in cellular water that resists hydrophobicity. HYDROPHOBICITY IN medications with substantivity effects on water CAN allow for the possibility of REVERSE TRANSCRIPTION TO CAUSE NOVEL DISEASES. Did you know this? Slide the above points out. Jabs do this.
• Might the answer be that water and sunlight naturally create unbelievable amounts of electrons and protons free of an energy charge?  Hydrophobicity can break this effect. This allows DNA to unwind and be copied under reverse transcription, which can cause turbo cancers. This effect would be magnified if SV40 contamination was present. DNA plasmid numbers increase the probability of such an event. Translation is widespread in annotated noncoding sequences, including untranslated regions (UTRs), introns, and long noncoding RNAs (lncRNAs), especially in cancer, aging, and neurodegeneration. 3’UTRs follow the mRNA coding sequence, regulate localization, stability, and translation, and control the selection of hydrophobic amino acids that change protein folding in cells.

Impact on Oxygen-Dependent ETC Proteins

The hardest-hit heme proteins—hemoglobin (Hb), myoglobin (Mb), Rev-erb, and ETC cytochromes—are very oxygen-dependent, making them prime targets for biophoton-mediated damage. Understanding how technology is making us ill is key.

SUMMARY

Because of the atomic organization of human cells (AMO physics), energy should always be available within the system, provided oxygen and water are hydrating our semiconductive heme and melanin proteins. When this relationship is altered for any reason, the disease manifests. The energy derived from the sun is stored coherently and ready for use over all space-time domains. Mitochondrial water production is critical in our mammalian blueprint.

The fidelity of this water creation is the basis of organisms’ autonomy and it was built around the GOE and the KT event in our history. As a result, modern organisms are never simply at the mercy of their environments because of the coherent energy stored. When the environment steals this ability from cells (nnEMF), cells are at the mercy of food and exercise.

More to the point, we don’t have to eat constantly (Leptin Rx), leaving plenty of time for other beneficial, pleasurable activities (SEX).

The other consequences are that the organism is exquisitely sensitive and free from the mechanical constraints of life on Earth and satisfies, at least, some of the basic conditions for quantum coherence. Water provides that as well.

Liquid water on Earth is quantum coherent even at ordinary temperatures and pressure. This is why Nature got the idea to build the IMM of cells around liquid water. It functionally is a naturally formed quantum computer.

 

Liquid water made in the mitochondrial matrix is the most wonderous chemical Nature has ever built because the water forms more coherent domains than the water from the hydrology cycle. Even latitude variation shows how water homogeneity changes as solar inclination affects its molecular arrangements and bond angles. Mitochondrial matrix water associates with macromolecules and membranes in cells into a gel-liquid crystalline configuration that enables enzymes and nucleic acids to function as quantum molecular machines that transform and transfer solar energy at close to 100% efficiency. Liquid crystalline water at interfaces also provides the excitation energy that enables it to split into hydrogen and oxygen in photosynthesis, simultaneously generating electricity for intercommunication and the redox chemistry that ultimately powers the entire biosphere on the 3rd rock from the sun.

Water is the means, medium, and message of life. For you to remain healthy, it must be made in large quantities in your mitochondrial matrix. Any reduction in its production will lead to disease.

PODCAST LESSONS FROM 2024: DID YOU UNDERSTAND UNCLE JACK?

Most of you did not. As I said in two podcasts with Daniel Prince and Mark Bell, an inconvenient truth will develop in front of your eyes if you look at the revenue numbers. The second highest revenue generator for banks in the U.S. is hospitals. Number 3 is drug (pharma drugs), cosmetic, and toiletry.

This is why makeup companies and sunscreen companies are lumped in with drugs. The more sun you block, the more drugs become profitable. Number 4 is the health and medical insurance industry. Number 5 is pharma wholesales. The sicker we become by DARPA light programs, the easier it is to control our behavior.

Due to control, numbers 2, 3, 4, and 5 become more profitable. The incentives dictate the outcomes in American centralized healthcare. Centralized Medicine is how you create economic slaves. This is why the DARPA MKULTRA blue light story should be huge, but nobody links things together anymore because their dopamine and melatonin levels are in the tank. Also, because of the blue light MKULTRA story, Danny Jones podcast (first one).

That is what is controlling everything at this point in America. Big Med, Big Harma, Big Insurance are fighting back. Follow the money.

WELCOME TO THE TRUTH.  HOW DID DARPA HELP THE COVID ARCHITECTS?

 

Understanding the Warburg Metabolism and Oxygen Sensitivity

The Warburg effect refers to a metabolic shift observed in cancer cells, where they predominantly rely on glycolysis for energy production even in the presence of oxygen, rather than oxidative phosphorylation (OXPHOS) in the mitochondria. This is often interpreted as a metabolic adaptation to support rapid cell proliferation. However, my decentralized hypothesis reframes this as a photo-bioelectric phenomenon driven by disrupted mitochondrial electrical resistance with a loss of mtDNA water production and amplification of melanin production from excessive ultraweak biophoton production rather than a simple fuel shift.

In the COVID jabs, the cancer pathway is more risky in any vaccine that uses SV40 as the promotor in its production. The second engineered risk is for any jab that uses lipid nanoparticles to surround spike proteins. Both of these modes are capable of causing a disrupted electrical resistance on the inner mitochondrial membrane to cause this process. The patients with Pfizer COVID jabs should be expected to present with stage 4 cancers via the mechanism I am presenting here.

The Warburg effect is well known in biology. It is described via a situation where a cell has elevated glucose utilization in cancer cells. It has traditionally been seen as an adaptation for energy (lactate production via glycolysis). However, recent research suggests it may reflect a broader metabolic and circadian dysregulation rather than a primary fuel source. Studies indicate that circadian disruption (e.g., from artificial light, shift work) uncouples mitochondrial oxidative phosphorylation, lowering NAD+ and raising NADH, leading to pseudohypoxia, ubiquitin marking, and high glucose as a compensatory response. This aligns with the idea that the Warburg metabolism signals environmental stress (e.g., light-nitrogen mismatch) rather than a cancer-specific fuel shift.

HOW DOES IT REALLY HAPPEN IN A DECENTRALIZED FRAMEWORK OF THERMODYNAMICS

I argue from first principles that the Warburg shift arises due to damage to melanopsin (from blue light or nnEMF) and mitochondrial DNA (mtDNA), which impairs water production at cytochrome c oxidase and disrupts the proton motive force (Δψ) across the inner mitochondrial membrane. This leads to decreased electrical resistance and an increase in reactive oxygen species (ROS)/reactive nitrogen species (RNS), WITH a simultaneous overproduction of ultraweak biophotons in the UV range. The resulting dehydration of melanin sheets and loss of bioelectric signaling (a DC current on the order of one trillionth of an amp) prevent regular cellular repair and apoptosis, pushing cells into an atavistic, anoxic-like state reminiscent of pre-oxygenated Earth environments.

In this context, oxygen becomes problematic for cells due to its high electronegativity on the periodic table and role as the terminal electron acceptor. Typically, oxygen facilitates efficient energy production via the electron transport chain (ECT) by reducing electrical resistance on the inner mitochondrial membrane. However, when electrical resistance drops and Δψ is compromised, oxygen’s presence exacerbates oxidative stress, further damaging mtDNA and amplifying ROS/RNS and transforming energy in ultraweak biophotons (Popp, VanWijk). This creates a vicious cycle where oxygen, instead of supporting regeneration, acts as a toxin, driving chronic disease states like cancer. The duration and intensity of the signal predict how fast the process of oncogenesis occurs in patients. This explains why people who took Pfizer jabs contaminated with SV40 and LNPs face the highest risk of turbo cancers.

Why Should We Be Treating Cancers/COVID Patients as an Oxygen Allergy?

Treating a patient with the Warburg metabolism as having an “oxygen allergy” aligns with my model because:

• Disrupted Bioelectric Regulation: The loss of electrical resistance on the inner mitochondrial membrane signals a breakdown in the bioelectric currents that regulate cellular repair and energy flow. Oxygen, by lowering resistance further due to its electronegativity, amplifies this dysregulation, mimicking an allergic or hypersensitive reaction where the body cannot tolerate a usually beneficial substance.
• Excessive Oxidative Stress: The overproduction of ROS/RNS in this state turns oxygen into a pro-oxidant rather than an antioxidant. This is analogous to an allergic response, where an environmental factor (oxygen) triggers an exaggerated and harmful reaction, leading to tissue damage and inflammation.
• Angiogenesis and Oxygen Delivery: My insight suggests that cancer cells, under this bioelectric dysfunction, stimulate angiogenesis via ultraweak biophoton release, increasing blood flow and oxygen delivery to the tumor. This paradoxical oxygen influx worsens the condition, as the mitochondria cannot utilize it effectively, reinforcing the need to limit oxygen exposure to break the progrowth cycle, similar to avoiding an allergen.
• Evolutionary Context: I propose that the Warburg shift reflects a return to an anoxic metabolic state, where cells adapted to low-oxygen environments. Treating it as an oxygen allergy acknowledges this evolutionary mismatch, suggesting that reducing oxygen availability (e.g., through hypoxia-mimicking therapies) might restore electrical resistance and halt the atavistic progression.
• Therapeutic Implications: Interventions like Vitamin C, methylene blue, or deuterium-depleted water (DDW) would work by modulating electrical resistance and mitochondrial function, potentially reducing oxygen’s toxic effects. This supports a strategy of minimizing oxygen’s role until the bioelectric and regenerative signals are restored, akin to desensitization in allergy management. This also explains why people who were treated in hospitals had high death rates. They were all given supplemental oxygen and/or ventilated. This was precisely the wrong thing to do. Why? Doing it pushed the failing colony of mitochondria with a loss of electrical resistance to ARDS. This was what I saw in ICUs and why so many people hold the belief that centralized healthcare was a killing machine for COVID-19 patients.

Connecting to A Broader Decentralized Thesis

My decentralized medicine thesis emphasizes natural light cycles (e.g., UV-A and IR-A for regeneration via melanin and POMC) and challenges centralized metabolic dogma with quantum-physical principles. The “oxygen allergy” treatment rationale fits this by prioritizing bioelectric and environmental factors (light, electromagnetic fields) over biochemical fuel shifts. It suggests that restoring mitochondrial water production, melanin hydration, and electrical resistance via natural light exposure and avoiding blue light would reverse the Warburg effect and prevent chronic diseases like cancer. It also opens the door to drug treatments that increase electrical resistance in these patients. This would include vitamin C, methylene blue, Ivermectin, and Fen/ben combos. All these drugs increase electrical resistance on the inner mitochondrial membrane, which is why they work. Water also helps because it is Nature’s Faraday cage for EMFs. This fundamental insight is lost in centralized healthcare.

WHY IS DEUTERIUM DEPLETED WATER USEFUL IN CANCER?

If you understand what I have laid out here, cancer is photo-bioelectrical electrocution of the inside of a cell. Why? Dehydrated melanin causes massive amplification of the DC electric current a cell makes. Remember what Nick Lane told us in his book Power, Sex, and Suicide? A healthy inner mitochondrial membrane contains 30 million volts of electric charge. This charge would typically be devastating to a cell. When I read this fact long ago, it raised a big question: How does a cell dampen the electrical charge?

Pure water containing no ions is an excellent insulator of electric charge. This is why Nature created the DDW, which was made by mtDNA at cytochrome C oxidase. What was the stimulus for her to gain this wisdom? It was the wound of the Great Oxidation event. The toxic wound Nature sustained from the creation and eventual dominance of oxygen on Earth was a stimulus that drove massive electrocution and endosymbiotic growth or the merger of the FIRST two domains of life on Earth 650 million years ago. Nature would never have innovated mitochondria without this photo-bioelectrocution caused by oxygen.

Pure water is non-deuterated water.  But not even “deionized” water is completely free of ions. Water undergoes auto-ionization at any temperature above absolute zero. Further, because water is such an excellent solvent, it almost always has some solute dissolved in it, most frequently a salt. This increases its electrical conductivity. This explains why all systems in the body except the matrix have saltwater in them. It also points out something else.

nnEMF dehydrates us, leaving more NaCl in less water and increasing electrical conductivity. When you consider how melanin responds electrically to dehydration, you should begin to see why this is a double whammy for a cell or organism. This is why nnEMF causes so many diseases. When you understand the wiring diagram basics, you will realize that we are effectively self-electrocuting ourselves on a chronic basis when there is a loss of electrical resistance in our membranes. This is what causes pro-growth programs in cells. This same set of circumstances caused endosymbiosis. Oxygen drove the growth and fusion between bacteria and Archea to make a mitochondrion. That was the original cancer on Earth. Shocking truth bomb, huh?

WHY CAN I SAY THIS? The basics of electrical resistance is a physics story.

Suppose water has even a tiny amount of such an impurity. In that case, it can conduct electricity readily, as impurities such as salt separate into free ions in aqueous solution by which an electric current can flow.

It is known that the theoretical maximum electrical resistivity for water is approximately 182 kΩ·m²/m (or 18.2 MΩ·cm²/cm) at 25 °C. This figure agrees well with what is typically seen on reverse osmosis, ultrafiltered, and deionized ultrapure water systems used, for instance, in semiconductor manufacturing plants.

A salt or acid contaminant level exceeding even 100 parts per trillion (ppt) in ultrapure water begins to lower its resistivity level noticeably by up to several kilohm-square meters per meter (a change of several hundred nanosiemans per meter of conductance).

NICK LANE’S 30 MILLION CURRENT

Pure water has a low electrical conductivity, so Nature chose this water format to be adjacent to the inner mitochondrial membrane post endosymbiosis to prevent further self-electrocution. As a result, this increases significantly upon solvation of a small amount of ionic material water such as sodium chloride.  Thus, the risks of electrocution are much more significant in water with the usual impurities not found in pure water.  This is why people on heart transplant lists do better when they are given hypertonic saline solutions. In cardiac care units, we induce electrocutations when we use a defibrillator to restart a failing electrical organ called a HEART.

Recall that in centralized medicine, when people have a cardiac arrest, we use defibrillators to jump-start their hearts with an electrical charge because the mtDNA cannot create it. We can improve this situation by using a hypertonic salt solution before the defibrillator. This should be a change made to all ACLS and ATLS protocols. Hypertonic saline is also a key player in decentralized medicine treatment plans. Neurosurgeons are the one group of centralized MDs who learn how to use hypertonic saline in neurosurgical emergencies.

WHY YOUR WOUNDS CREATE YOUR WISDOM: ALL-NIGHT SURGERY LESSONS

This is why I quickly figured out biophysics once I allowed the wounds of my patients to teach me new lessons. Remember that little girl I told you about in the Danny Jones podcast, whom I did a 24-hour surgery with Metallica blasting all night? I told the world it was the most critical case I had ever handled in my career. Why has no one ever asked me why I said that in any podcast or Q&A?  PODCAST LINK

It was that case that taught me why methylene blue and hypertonic saline were tools I could use to save her life. Few centralized MDs understand biophysics. That night, I began using mannitol on that little 16-year-old girl, and I noticed something unusual. Every time anesthesia dosed her with it, it dropped her blood pressure dramatically. I realized I was inducing acute heart failure because she was in neurogenic and cardiac shock at the same time. This is when i began to scream at my neurosurgery staff and told him to get out of my way and I am going to try something new. I realized I had to jump-start her heart and limit her brain swelling to get more time to remove the bone from her brain. So, I immediately stopped the mannitol and gave her hypertonic saline to defibrillate her heart using basic biophysics. NaCl increases electrical currents, so I could jump her heart, giving me more time to save her life.

Anesthesia told me that once I did this, her BP stabilized, and her ECG improved. As I continued to work, Metallica’s song Nothing Else Matters came on in the background. The lyrics hit me just at the right time. Now, go listen to the lyrics sometime.

I realized that after switching her IVs to hypertonic saline to save her failing heart due to the massive intracranial injury, I had to simultaneously add a methylene blue drip to run in slow to increase the electrical resistance in her brain’s demolished mitochondria to give me more time to work in her brain. This was akin to using biophysics to jump her heart while her brain was damaged.

These two seemingly incongruent things should NOT be done simultaneously, but I knew it was my only chance to save her.

I knew this was not what I was taught, but I had the strangest feeling then. I had to do it because it was the right thing to do in my mind. That is what stimulated the massive fight between my neurosurgeon staff and me. That is when I told everyone in the room that they should leave right then if they did not like or agree with what I was doing. Only the staff neurosurgeon left the OR that night. If you listened to the Danny Jones podcast, you know the rest of the story now. That is why these tools are dangerous in the wrong hands.

What did Metallica whisper to me that night in the operating room?

So close, no matter how far
It couldn’t be much more from the heart
Forever trusting who we are
And nothing else matters

To this day, I get tears in my eyes when that song is played around me.

Wounds create our wisdoms and that is why we must embrace the pain and suck that life deals.  A wound is never a mistake if it teaches you a lesson you learn from. Then, it becomes tuition. I said that in 1998, at the Morbidity and Mortality conference, they reviewed this case. Not only didn’t i get canned, my neurosurgery staff all came up to and told me what I did that night was crazy wise. All except one guy. The centralized neurosurgeon whom I threw out of the room was a career military man who had contracts with DARPA working on primates.

That is why this podcast is a key listen for any Savage.

https://www.youtube.com/watch?v=zCGnMY9FSNg

The Next Step?

While my model is intriguing and integrates diverse data, it relies on a bioelectric paradigm that departs from mainstream centralized mitochondrial and cancer biology. To the centralized players, my concept of oxygen as an “allergy” will be considered metaphorical. My perspective is quite different. It is AXIOMATIC. As such, when I returned to ICU medicine as a trauma neurosurgeon, I found these ideas very useful for framing therapeutic strategies for hospitalists and nurses (e.g., hypoxia therapy, redox modulation) who cared for these patients.

My decentralized interpretation of the Warburg effect is a marker of circadian mismatch, not a fuel source. Moreover, it is well supported by recent findings in the literature. The old idea that fuel source is critical is now on life support. The next blog will blow that out of the water.

As I’ve described, artificial light at night disrupts NAD+/NADH balance, increases ubiquitin, and drives glucose spikes to brake ubiquitin marking. AM UV and natural light cycles restore NAD+, lower ubiquitin, and normalize metabolism, reducing cancer, obesity, and autoimmunity, as Ive emphasized with plants and animals sharing this response in the ubiquitin series on my website.

LITERATURE SUPPORT

Recent Research on the Re-Interpretation of the Warburg Metabolism As a Circadian Shift

• Circadian and Metabolic Perspectives in the Role Played by NADPH in Cancer
  • This source discusses the Warburg effect as metabolic reprogramming in cancer cells driven by NADPH availability and touches on its connection to circadian rhythms. It notes that the Warburg effect is linked to circadian disruption. It provides a direct context for NAD(P)H imbalances in cancer, aligning with the decentralized reinterpretation of the Warburg effect as a signal of metabolic and circadian dysregulation rather than a fuel source.
• Associations among Metabolism, Circadian Rhythm, and Age-Associated Diseases
  • Some sources have explored the connection between the Warburg effect, tumorigenesis, and circadian rhythm disruptions, particularly in age-associated diseases like cancer, obesity, and diabetes. They hypothesize that circadian misalignment (e.g., from artificial light) activates the Warburg effect as a cell-autonomous transformation, supporting the view that it reflects an environmental circadian mismatch rather than a metabolic adaptation for energy.
• Cancer metabolism in space and time: Beyond the Warburg effect
  • Space medicine sources have delved into cancer metabolism’s spatial and temporal aspects, suggesting that the Warburg effect may involve broader metabolic and environmental factors beyond simple glycolysis. It provides a foundation for reinterpreting the Warburg effect as circadian and light-related factors influence it. It is consistent with my argument about artificial light’s role in uncoupling light-nitrogen cycles in the mitochondria.
• Oncogenic fatty acid oxidation senses circadian disruption in sleep-deficiency-enhanced tumorigenesis.
  • This study links circadian disruption (e.g., sleep deficiency) to fatty acid oxidation (FAO) and the Warburg effect in cancer. It shows how clock gene dysregulation (e.g., CLOCK, BMAL1) and NAD+ levels drive tumorigenesis. It supports my hypothesis that the Warburg effect signals circadian mismatch from artificial light, with NAD+/NADH imbalances and ubiquitin marking playing key roles.
• Exposure to artificial light at night: A common link for obesity and cancer?
  • This source highlights how artificial light at night (ALAN) disrupts circadian rhythms, increasing risks of obesity, diabetes, and cancer through mechanisms like melatonin suppression, NAD+ decline, and metabolic reprogramming. It aligns with my interpretation of the Warburg effect as a response to circadian misalignment from artificial light, connecting it to ubiquitin, NAD+/NADH, and disease.
• Artificial Light at Night and Type 2 Diabetes Mellitus
  • This review consolidates studies from 2000–2024 on ALAN’s association with type 2 diabetes, obesity, and cancer, emphasizing circadian disruption, NAD+ decline, and glucose metabolism changes. It supports the decentralized view that the Warburg effect and related metabolic disorders stem from artificial light-induced circadian mismatch, not just metabolic adaptation.

    DECENTRALIZED MEDICINE HAS ANSWERS FOR THE DARPA-INDUCED CANCER WAVE

  • My insights reveal that shorter wavelengths (e.g., blue light at night) increase cancer risk by uncoupling light-nitrogen cycles inside mitochondria, lowering NAD+ and pseudohypoxia and raising ubiquitin. At the same time, AM sunlight provides massive red light exposure without UV (290–420 nm), which promotes water production for a latitude-determined duration. This improves mtDNA hydration before we stimulate the regenerative stimulus. Hydration endogenously becomes a Faraday cage for the destroyed inner mitochondrial membrane that has lost its electrical resistance.
  •  

    As the day progresses, sunlight adds other light frequencies that promote diurnal regeneration, oxygen delivery, and health via melanin, POMC, and porphyrins. The Warburg metabolism is an epigenetic post-translational signal from mitochondria that light mismatches, not a fuel shift, cause the condition. This implies that the surface photo-electrochemistry of the eye, skin, and gut can outpace the biochemistry inside the cell in order of critical importance.

  • When someone exhibits the Warburg metabolism, the patient must be treated like they have an oxygen allergy. Why?
  • Adding oxygen to a mitochondrial decreases electrical resistance, altering the regenerative currents stimulated by mtDNA and melanin. A red light stimulates water production, while ultraweak biophotons stimulate POMC translation to create endogenous melanin.

• The mtDNA water production engulfs melanin to RAISE electrical resistance to make a DC electric current that is one trillionth of one amp. This current is used to renovate and regenerate mammalian tissues. If this signal is awry and the electrical resistance drops, this creates lowered NAD+, massive upregulation of ROS/RNS, and massive overproduction of ultraweak biophotons in the UV range. As a result, too little mitochondrial water production is made.
• At the same time, too many ultraweak biophotons are made, creating a situation where melanin sheets created are chronically dehydrated in the cell, and there is persistent chronic ROS/RNS and no generation of the bioelectric signal that the cell needs to self-repair itself. This situation leaves the cell with a massive epigenetic upregulation of endogenous melanin, which becomes highly conductive in the cell, ruins the surrounding regions, and affects the local production of melatonin by mitochondria, making up 95% of melatonin in humans. It is not a pineal story. That is functional medicine nonsense.

• The loss of the cell’s melatonin production will not allow apoptosis to eliminate the poorly functioning cell. When these bioelectric changes occur, the cell reacts atavistically to return to an older evolutionary format when cells were in anoxic environments before the great oxidation event. As a result, the body responds by lowering oxygen delivery to the defective mitochondria because oxygen is the terminal electron acceptor.
• Due to its high electrionegativity on the periodic table, it reduces electrical resistance. During a Warburg shift, the cell needs a raised electrical resistance to stop the process. Organisms with melanopsin damage must vary resistance to bioelectrical flows to survive. Resistance in cells directs energy flow and computes information, which drives biochemical pathway decision-making.

• • When melanopsin damage occurs from blue light or nnEMF, the cells use glucose, alanine, and glutamine. These choices were the best choices when the Earth was anoxic. The Warburg shift is needed in a cancer state to stop the progrowth epigenetic program that has started due to melanopsin damage and mtDNA mutation, stopping water production at cytochrome c oxidase. Loss of oxygen increases electrical resistance on the inner mitochondrial membrane. Once melanopsin damage occurs in any tissue, water production slows down at cytochrome c oxidase, and oxygen use diminishes because of the destruction of delta psi on the inner mitochondrial membrane.
  • Picard and Levin recently wrote an article in which they made my decentralized case for me when they said, “Organisms adapt and optimize fitness by achieving lasting changes in energy resistance.”
  • This explains why Vitamin C, methylene blue, and DDW all operate to help prevent a turbo cancer. They act to limit the stay currents coming from the inner mitochondrial membrane from the damage of SV40 and LNP-spike proteins destroying it. The Resistance to energy flow (éR) triggers signaling outputs whose information content activates (epi)genetic regulatory pathways, which open communication between mtDNA and the nDNA to produce enduring cellular, physiological, anatomical, and behavioral changes. DDW also protects your cells and organs from this self electrocution that leads to turbocancers.
  • These changes feedback to minimize and optimize éR over the long term, enhancing the system’s efficiency, environmental fitness, and health for the environment sensed by mitochondria. Cancer is not a metabolic disorder. It is a Photo-bioelectric disease that can be amplified by DARPA bioweapons.  Turbocancer happens because the duration and intensity of the jabs predict it. After all, bioelectrical activity demolishes the resistance to energy flow (éR) rapidly.
  • You must start at the magneto-heliosphere for a decentralized medical lesson on mtDNA diseases like cancer. Learn the actual ticket that moves the needle of all humans. You must learn that when your Inner mitochondrial membrane has no electrical resistance, this signals a poor redox charge (delta psi) on the inner mitochondrial membrane. This is when oxygen becomes a toxin for most chronic diseases. Your centralized shills will never get you to this level of understanding.
  • Cancers grow because blood flow amplifies to these regions due to the excessive release of ultraweak biophotons. This action stimulates angiogenesis, bringing more oxygen, not less oxygen, to the cancer. Since oxygen has a high electronegativity, it draws current across the damaged landscape of the inner mitochondrial membrane, and this causes massive amplification of ROS and RNS. This causes the progrowth situation to persist and exist. The goal is to limit the spread of that current by any means necessary.
  • People forget that oxygen’s toxic nature drove endosymbiosis on Earth 650 million years ago. Otherwise, Nature would have never innovated mitochondria. Oxygen’s paramagnetism keeps us alive in an oxygenated atmosphere, but it becomes deadly in one where there is a loss of electrical resistance on the inner mitochondrial membrane.
  • These are the critical missing pieces of information in producing all chronic disease epidemics. This also explains why turbocancers are now present at record rates in the jabbed. Become the disruptor, or be disrupted by DARPA centralized medicine machine. They are tapering the Ponzi scheme and it should be no mystery to anyone here how they are doing it.
  • Artificial light damages melanopsin, which is associated with dehydration. This exacerbates the bioelectric signals, explaining modern disease epidemics. This aligns with my decentralized medicine thesis, reinforcing the need for natural light cycles and challenging biological dogma with quantum-physical wisdom. MAHA = HAHA
  • SUMMARY

In summary, a patient with the Warburg metabolism should be treated like they have an oxygen allergy because the loss of mitochondrial electrical resistance turns oxygen into a toxic agent that amplifies oxidative stress, disrupts regeneration, and drives pro-growth signals. Restoring bioelectric balance and resistance is a mandatory clinical need and is key to managing the trubocancer state.

• CITES
  • Wang, R.-H., & others (2017). Associations among metabolism, circadian rhythm and age-associated diseases. Aging and Disease, 8(5), 691–709.
    • DOI: 10.14336/AD.2016.1101
    • My Notes: This review by Wang and colleagues discusses the connection between the Warburg effect, tumorigenesis, and circadian rhythms, hypothesizing that circadian disruption activates cell-autonomous transformation to generate the Warburg effect. It emphasizes NAD+-dependent sirtuins (e.g., SIRT1, SIRT3) and clock genes (e.g., CLOCK, BMAL1, PER2) but focuses on genetic and metabolic pathways, potentially overlooking the environmental role of artificial light and light-nitrogen coupling I’ve highlighted. It critically examines this as an establishment narrative that simplifies the Warburg effect as a biochemical adaptation, missing the broader photobiological and quantum context of circadian mismatch from artificial light.
  • Masri, S., Kinouchi, K., & Sassone-Corsi, P. (2015). The emerging link between cancer, metabolism, and circadian rhythms. Nature Medicine, 21(12), 1795–1803.
    • DOI: 10.1038/nm.0271-8
    • My Notes: Masri, Kinouchi, and Sassone-Corsi explore the connection between circadian disruption, cancer metabolism, and the Warburg effect, noting epidemiological links to artificial light at night (e.g., shift work). They discuss clock genes (e.g., CLOCK, BMAL1, CRY, PER) and metabolic reprogramming but focus on transcriptional feedback loops, potentially underestimating the role of light-nitrogen uncoupling, ubiquitin marking, and NAD+/NADH imbalances I’ve emphasized. It challenges the establishment narrative for prioritizing molecular biology over environmental light dynamics, aligning with the interpretation as a circadian mismatch signal.
  • Sahar, S., & Sassone-Corsi, P. (2009). Metabolism and cancer: The circadian clock connection. Nature Reviews Cancer, 9(12), 886–896.
    • DOI: 10.1038/nrc2747
    • My Notes: This review by Sahar and Sassone-Corsi examines how circadian clocks regulate cancer metabolism, including the Warburg effect, and links it to circadian disruption. It mentions light exposure as a zeitgeber but focuses on genetic and biochemical mechanisms (e.g., sirtuins, NAD+), while missing the decentralized photobiological insights about artificial light’s role in uncoupling light-nitrogen cycles and driving ubiquitin and disease. I critically assess this as an establishment perspective that overlooks quantum and surface chemistry roles, supporting my broader model.
  • Korennykh, A., & others (2019). The metabolites NADP and NADPH are the targets of the circadian protein Nocturnin. Nature Communications, 10(1), 10125.
    • DOI: 10.1038/s41467-019-10125-z
    • My Notes: This study by Korennykh and colleagues at Princeton University investigates Nocturnin’s role in fat metabolism and circadian rhythms, showing its action on NADP+/NADPH and NAD+/NADH, linking to energy regulation. It supports my point about NAD+/NADH imbalances in circadian disruption and metabolic diseases (e.g., obesity, diabetes) but focuses on biochemical mechanisms, missing light-nitrogen coupling and ubiquitin dynamics I’ve described. I challenge the establishment narrative for not exploring environmental light’s photobiological impact, aligning with the decentralized reinterpretation of the Warburg effect.
  • Zhang, Y., Hu, K., Tang, Y., Feng, Q., Jiang, T., Chen, L., Chen, X., Shan, C., Han, C., Chu, W., Ma, N., Hu, H., Gao, H., & Zhang, Q. (2024). Interactive correlations between artificial light at night, health risk behaviors, and cardiovascular health among patients with diabetes: A cross-sectional study. Journal of Diabetes, 16(10), e70008.
    • DOI: 10.1111/1753-0407.70008
    • My Notes: This recent study by Zhang et al. links artificial light at night (ALAN) to diabetes, obesity, and cardiovascular health, showing circadian disruption, NAD+ decline, and metabolic changes. It supports the decentralized view of the Warburg effect as a circadian mismatch signal from ALAN. Still, it focuses on behavioral and epidemiological data, missing quantum and photobiological mechanisms (e.g., light-nitrogen, ubiquitin). I critically examine this as an establishment narrative prioritizing classical epidemiology over my decentralized insights.
  • Engin, A. (2024). Misalignment of circadian rhythms in diet-induced obesity. Advances in Experimental Medicine and Biology, 1460, 27–71.
    • DOI: 10.1007/978-3-031-63657-8_2
    • My Notes: Engin’s review connects circadian misalignment (e.g., from artificial light, shift work) to obesity, diabetes, and cancer, including the Warburg effect as a metabolic response. It mentions NAD+, sirtuins, and clock genes but focuses on biochemical and nutritional pathways, potentially overlooking your light-nitrogen coupling and ubiquitin marking. I challenge the centralized establishment narrative for not addressing photobiological and quantum mechanisms, supporting the decentralized reinterpretation as a circadian mismatch driven by artificial light.
  • GAME, SET, MATCH.

If you ever wondered why I have written about tinnitus so much today is the day where the whole story comes together.

The inner ear of humans and other mammals is contained within the temporal bone. It comprises the cochlea and vestibular organs involved in sound perception, spatial orientation, and balance. Apart from these sensory systems, the inner ear contains several diverse accessory cells, including pigment‐containing melanocytes. The existence of pigment in the inner ear has long been known. It was first described in 1851 by the Italian physician and early pioneer of inner ear microanatomy, Alfonso Corti, but it took 80 years before the pigment was identified as melanin (Wolf, 1931). Studies investigating the function of melanocytes in mice unraveled their role in generation of the endocochlear potential and hearing (Hilding & Ginzberg, 1977; Igarashi, 1989; Schrott & Spoedlin, 1987; Steel & Barkway, 1989; Steel et al., 1987) and balance (Marcus & Wangemann, 2010; Palma et al., 2018).

The Human Cochlea is an amazing design of nature that DARPA is Targeting

Our spiral-shaped auditory organ is a marvel of precision, converting sound waves into electrical signals via hair cells and the auditory nerve. But I have proposed it could do more, specifically with melanin as the key player. The melanin sheet in the cochlea explains how Becker’s work, My work, and DARPA all fit together.

The cochlea contains a lot of melanin deep inside our skull, particularly in the stria vascularis, a region critical for maintaining the endocochlear potential (a bioelectric gradient that powers hearing).
This melanin isn’t just decorative in the ear, it is there as a condensed matter wide band gapped semiconductor to do something most of you did not anticipate. It is adjacent to a place in the brain with no blood-Brain barrier. DARPA learned this from Dr. Allan Frey, who discovered the pulsed microwave effect when he was working for them during the MKULTRA era.
.
Centralized thinkers believe melanin is thought to protect against oxidative stress and possibly modulate ion flow. Melanin has multi-use purposes. DARPA found out from Frey’s work that it inhibits Robert O. Becker current of regeneration and because it does this is induces traumatic brain injury.

Physics tells us that melanin absorbs all parts of the electromagnetic spectrum. As such, it can translate those light signals into electrical impulses or heat, subtly altering the cochlea’s function. Frey proved this was possible with pulse microwaves at the same time Becker uncovered the regenerative current for DARPA. Both of the scientists were silo’d and their work was kept from one another. This Manhattan style scenario comes straight out of DARPA design. Allen Frey was hired by DARPA to find out how the Moscow Embassy Incident occurred in the mid to late 1960s, which I mentioned in the Marty Bent podcast.

Here’s where the story gets wild: EMF detection implies the cochlea could pick up a lot of different electromagnetic noise beyond audible sound—think wireless signals, ambient EMF from tech, or even natural geomagnetic fluctuations from the Schuman resonance. Condensed matter physics supports this action; melanin’s disordered structure and electron mobility could make it a natural antenna, resonating with EMF fields and transducing them into something the auditory system (or even mitochondria downstream) might perceive. This fits my earlier thread about sensory dyssynchrony in ASD (QE #45) and what occurs in the jabbed.

Almost any cell with a circadian mismatch has a lowered NAD+ and exhibits a Warburg shift because blue light increases blood glucose and insulin levels. Becker found this in Penscola’s study of pilots in 1969 for the US NAvy, and in 2011, Nora Volkow confirmed it for NADA, the NIH, and SRI (DARPA) in 2011.

If the cochlea is misinterpreting different EMF stimuli as sensory input in the brain, DARPA found it could overload the thalamus and cause traumatic brain injuries in the acoustic arrays. This would throw off mitochondrial timing by inducing mtDNA mutations; it would lower mtDNA melatonin, water, ROS, and CO2 production, especially in sensitive populations with higher heteroplasmy rates who have preexisting diseases, atrophic skin, unmyelinated brains, and pale skin. For example, soldiers in war or kids with ASD exhibit these symptoms. This is precisely what I found in soldiers I evaluated in Hattiesburg, Mississippi, from Camp Shelby Joint Forces Training Facility who came home with acoustic injuries, TBI, and PTSD. Dr. Frey figured this out for DARPA in the 1960s, and DARPA developed direct energy weapons to target this region.

HOW DOES IT HAPPEN IN DARPA TARGETED PROGRAM?

• I’ve emphasized in many blogs that blue photons’ high energy makes them less common for stable, long-term pigmentation or fluorescence in the skin of mammals, but I doubt you understand why I have say it repeatedly. The reason is simple, Nature makes blue light signaling rare in mammals. As a result, this mades melanin the most critical photoreceptor in mammalian biology. Why, Anything that is rare becomes a precision tool for the fidelity of the signal. This was what Claude Shannon taught DARPA in the 1950s when he worked for them at Bell LABS. Melanin’s ability to absorb and dissipate electromagnetic energy ONLY when it hydrated prevents brain damage from blue and UV light. When melanin is not hydrated due to mtDNA mutations induced by nnEMF targeting, people get chronic diseases because heteroplasmy rises. HYPERLINK
• Did you know that condensed matter physicists found that melanin in its natural form is too untidy on a molecular level to conduct electricity with much efficiency? Did you know melanin loses its conductivity when exposed to water?
• People forget melanin creates a DC electric current signal when it is illuminated by sunlight. But few people realize this current is dampened. Eumelanin does the same in our skin and ears. These are the same melanins DARPA has been studying in our skin and our cochlear and semicircular canals. (See the citation below.)
• Eumelanin in its natural form is too untidy on an atomic molecular level to conduct enough electricity with much efficiency for industrial use, but this points out why Mother Nature uses it in mammals. This was the key thing I told Becker in our meeting that I have not mentioned anywhere yet. I learned this from treating PTSD soldiers at Camp Shelby in Mississippi coming home from the theatre of war. All of them got hit with a massive vaccination program before they left for deployment.

• Hydrating melanin in any mammal is done by local mtDNA metabolism. Cytochrome C oxidase creates the water that hydrates melanin. Hydrating melanin decreases the DC electric current to 1 trillionth of an ampere. This is what Decker found in his seminal work on how RBC dedifferentiate to pluripotential cells during wound healing and regneration. for industry, hydrate melanin is useless to electrical engineers, but hydrated melanin is just what the doctor ordered for a dying cell that needs to regenerate. It also explains why the use of the drug melotan is deadly in humans. I studied many bodybuilders, and people who use Melotan, and they tend to die the same way the jabbed die post vaccination. Many of the symptoms people get from melotan use corresponds to what Targeted Individuals report. See the video above. I learned this from my friendship with Charles Poliquin in the early 2000s
• This precision with which water operates at a quantum mechanical level preciselly dampens the DC currents used in cells which causes them to regenerate their tissues. This is what actually drives regeneration and healing in all mammals. This nuance is lost on many who do not understand how the non visual photoreceptors operate in man. DARPA’s research figured this out from combining the work of Frey and Becker from the 1960s.
• Suppose your mtDNA is not making water for any reason (disease due to heteroplasmy or exogenous XRT). In that case, the amount of the DC electric current melanin makes becomes toxic to neurons locally and distally connected to in the brain. This stimulus threatens to burn out local and distal circuits, leading to diseases like cancer and the ones we see in our chronic disease epidemics. This is what we are seeing today in the jabbed and targeted. This is what I believe killed Charles and many of his bodybuilding clients. I spoke about this in the Melanin Rx for mammals blog. Please re-read it.
• This is especially ampolified in all people who took the mRNA vaccines. Why? The spike protein is a mitochondrial toxin that diminshes water production from mitochondria. All soldiers were loaded with jabs before heading to war.
• At the same time, melanopsins’ role in nonvisual photoreception (e.g., via ipRGCs or in skin melanocytes) normally supports circadian and metabolic regulation and the function of our frontal lobes via the central retinal pathway connected to the SCN and habenular nucleus. This is why the jabbed all have circadian dyssynchrony and mental and cognitive issues. This is what long covid really is.

• The mtDNA mutations you heard about in Decentralized Medicine #35 lead to neuronal dehydration, which AMPLIFIES the DC current liberated from melanin and this current amplifies the bioelectric signal connecting it to tracts distally. This wiring diagram results and induces a trauma response (TBI). This what Dr. Ber says above, but has no idea how it occurs. Now you do. This is why DARPA’s directed energy weapons are so effective and cause unusual injury patterns in DTI MRIs. It requires a neurosurgeon to know the mechanism to understand what to look for. We are now seeing the same injury pattern developing in the jabbed because the mechanism is identical to what happens in soldiers with TBI. only the intensity of the TBI is different. People in DARPA programs (targeting) and in WAR have massive TBI injuries. The centralized MDs they see how no idea what they are looking at. This explains why DARPA told the military brass during the Iraq war to provide soldiers with RO water in the desert. DARPA knew exactly why to make the recs. It could limit TBI in soldiers.

• This neuronal loop between melanopsin and melanin prides an energetic constraint in mammalian tissues. This why blue light is SPECIFICALLY reserved for specialized functions (e.g., circadian timing, vision, frontal lobe function) rather than widespread use in the brain. This reinforcing its rarity and utility as a high-information signal. Today’s modern world destroys that fidelity because blue light is everywhere in our environment. DARPA found that out during the MKULTRA program I mentioned in the original Danny Jones podcast.

My earlier point about blue light’s role in circadian biology focused on melanopsin disrupting the circadian clock (SCN) in the retina. With the more recent data I have shared with you over 20 years I have shown you now that melanopsin is present in fat, arteries, the brain, and most other human tissues. This implies that the biochemists, food gurus, and vaccinator’s theories of safety need to be revisited and revised. Moreover, this situation has massive implications for the jabbed and the Targeted Individual in 2025.

• Broader Circadian and Metabolic Effects of Blue light Signal Fidelity:
  • Fat Tissue: Melanopsin in adipocytes allows adipocytes to directly sense blue light (e.g., from artificial sources) and influence lipid metabolism, adipogenesis, or insulin sensitivity. when you begin to realize that blue light activates melanopsin in fat tissue at night, what does this imply? The light stress immediately causes a vassopressin release in the pituitary. (QE#23) Vasopressin release at night disrupts normal metabolic rhythms, raising blood sugar, promoting fat storage, insulin resistance, and obesity—amplifying the pathways in the slide (e.g., increased appetite, insulin resistance). The jabbed’s fat mass will help them by drawing the LNPs of the spike protein, to our fat depots so we can destroy it there safely and later regenerate tissues it destroyed. Those who do not have SQ or visceral fat will be at higher risk for organ damage. This is precisely the opposite of what centralized medicine tells patients. You’ve heard me say it on many podcasts, but now you have my rationale for this wisdom.

• Arteries and Cardiovascular Health: Melanopsin in vascular tissue responds to nnEMF light exposure, affecting blood pressure, endothelial function, or cardiovascular risk. These things all cause PAD and clotting risks. Chronic blue light exposure at night might contribute to cardiovascular complications associated with obesity and diabetes. Those who took any of the jabs are certainly subject to massive clotting and bleeding wherever melanopsin and blue light meet in a tissue. The aftermarket data proves this in 2025.
• Brain Dominance:  Melanopsin is the most dominant opsin in the brain, melanopsin’s widespread expression suggests it plays a central role in coordinating light-dependent neural and hormonal responses. This is why everyone’s hormones panels look like shit today. If you are jabbed they are even worse. Blue light at night disrupts not just the suprachiasmatic nucleus, but other brain regions (frontal lobes) involved in appetite regulation (e.g., hypothalamus), mood (e.g., amygdala), and autonomic functions, further exacerbating metabolic and behavioral dysregulation.

• Implications for Ubiquitous Blue Light present in the jabbed person’s world. The risk for chronic disease generation is astronomical.
  • Because melanopsin is in fat, arteries, and the brain, artificial blue light at night confuses the retina of the human who is jabbed; it directly affects energy storage, blood vessel function, and central nervous system regulation. This systemic disruption explains the prevalence of body and physiological changes reported by the jabbed much more comprehensively than retinal effects alone.
  • Public Health Concerns: The widespread presence of melanopsin suggests that reducing blue light and nnEMF exposure day and night (e.g., through lighting design, screen filters, or behavioral changes) is even more critical than previously thought in those who are jabbed. It’s not just about sleep or regeneration; it’s about protecting metabolic, cardiovascular, and neural health across the body as the jabbed navigate the modern tech-driven world. The distal TBI this creates with destroy and atrophy brain function leading to many diseases and death in covert ways if the clinician is unaware of the mechanism. This is how DARPA learned to begin tapering the PONZI scheme.

• • Evolutionary Mismatch is accentuated in the jabbed: Humans evolved under natural light cycles, where blue light was rare at night. Constant exposure to artificial blue light activates vasopressin and melanopsin in tissues not adapted to nocturnal light, creating a profound mismatch that drives ALL chronic disease. Even MAHA misses this low-hanging fruit.
  • MAHA’s avoidance of banning the mRNA platform early in the DJT term explains why Bobby was confirmed. As long as DARPA forces Bobby to keep the mRNA platform in place, the desired effect of tapering the Ponzi scheme will continue unabated.
  • Adding mRNA jabs to the animals in our food supply will also be a death trap for those who eat the food. This explains why Bill Gates is buying up farmland to grow his food and breed animals that aren’t GMOs. Once the Ponzi scheme is tapered, his properties and farms will be new non GMO food source for the NWO, who avoided these DARPA programs. They knew the game plan first due to their CIA/DoD connections via tech companies.
• Connection to My Decentralized Insights From the Marty Bent podcast on DARPA
  • My point about blue light’s rarity in nature and its role as a “high-information” signal via Shannon’s information theory remains highly relevant to the jabbed. Melanopsin’s systemic distribution suggests that this rare signal was evolutionarily optimized for daytime use, not nighttime exposure. The ubiquity of artificial blue light overwhelms this signal, diluting its informational value and causing widespread dysregulation, which causes the aftermarket disease we are seeing.

My recent discussions and podcasts from 2023-2025 on the leptin melanocortin pathways, LNPs, melanin, POMC, and Vitamin D3 also tie into the jabbed story: See the JONES/BOWDEN/KRUSE podcast and the last slide I presented in it.

The circadian mismatch drives the Warburg shift to cause a higher cancer spike than we saw in the 1950s SV40 debacle. I showed that slide at the end of the Danny Jones Bowden Kruse podcast (above) to show you that the new mRNA spike line has a stepper slope than the one associated with the Cutter Incident. This slope is greater because the risks today are worse because melanopsin’s light sensitivity is being destroyed. Melanopsin evolved to complements melanin’s photoprotective role, and the in the jabbed DARPA has engineered the bioweapon to uncouple us from the ability to regenerate. At the same time, POMC pathways (e.g., MSH production) interact with melanopsin to regulate metabolism and circadian rhythms in response to light. This is why GATES, FAUCI, and your governments wanted you inside out of the sunlight. This is why they outlawed Vitamin D use.

TINNITUS IS A DARPA CREATED DISEASE USED IN THEIR RCT BEING DONE AT SCALE

Tying this to the mitochondrial medicine story Dr. Doug Wallace laid out, cochlear melanin appears to act as a semiconductive relay switch based on my description above. This is something Robert O. Becker worked out in bone. The cochlea has a different way of operating with the endolymph and lacks a BBB. Melanin is a material that can both inhibit and promote electric flow, classifying it as a semiconductor. The molecule switches between resistive and conductive states depending on the amount of water in its local environment. This water can have its dielectric constant varied by atoms inside the liquids it is adjacent to. In the cochlea, potassium is that atom. The amount of potassium in endolymph is quite unusual compared to other organs. This “switching” property is a crucial mechanism that builds the backbone of all types of computers. The human cochlea is a quantum computer that DARPA has targeted initially using Allan Frey’s work.

WIDE BANDGAPPED SEMICONDUCTION: WHY I WAS RELUCTANT FOR HUBERMAN
Melanin as a wide-bandgap semiconductor is a key insight for the decentralized MD. Wide-bandgap materials can handle high-energy input EMF (like the entire electromagnetic spectrum) and switch between insulating (resistive) and conducting states based on external conditions. For melanin, hydration is the toggle switch the ear uses. When it’s dry, it’s resistive; add water, and it becomes conductive, thanks to proton hopping and electron mobility within its disordered structure. This isn’t just a neat trick; the physics studies in condensed matter literature show melanin’s conductivity skyrockets with hydration, making it a dynamic player in bioelectric systems. The water’s dielectric constant reflects its ability to store and transmit electric charge. This charge change due to hydration can shift melanin behavior further; it nails the critical variable in the ear: potassium levels in the cochlea’s endolymph. I said on marty’s podcast I knew that Huberman’s dad was a DARPA trained condensed matter physicists. I thought this was why Huberman knew to use cephalopods in his lab and this made me reluctant to want to speak with him. I mentions this to Marty as well.

WHAT IS TINNITUS AND MENIERE’S DISEASE TO A DECENTRALIZED MD?

Tinnitus is the persistent ringing or buzzing in the ears, and Ménière’s disease is hearing loss associated with vertigo. They have long been tied to cochlear dysfunction and often pegged to fluid imbalances or hair cell damage. Decentralized MDs must zoom in on the abilities of the stria vascularis. This melanin-rich region pumps potassium into the endolymph to maintain that +80 mV endocochlear potential. If melanin’s conductivity hinges on the dielectric constant of its surrounding water, and potassium alters that constant, any disruption (like nnEMF) could throw the system off. People forget that nnEMF causes dehydration, elevation of blood glucose, and raises insulin (Frey, Becker, Volkow all confirmed the effect). The effect of these things on potassium levels in the cochlea causes this disease.

HOW?

Centralized medicine, specifically ENT, still cannot explain these diseases, but rest assured, I can. An “alteration of the dielectric constant of the melanin sheet” in the stria vascularis means that it is stuck in a funky resistive-conductive limbo, misfiring signals decreasing precision and fidelity.

But exogenous nnEMF—like Wi-Fi, cell towers & phones, or Bluetooth—brings a wrecking ball. Apple is helping DARPA target millions.  Studies show that nnEMF induces dehydration by disrupting water’s hydrogen bonding, which drives the altered dielectric properties of water locally in the cochlea. This electromagnetic stimulus also triggers hypoxia, lowers NAD+ by stressing cellular oxygen use, spikes blood glucose via stress hormone release, and jacks up insulin levels locally as the cochlea scrambles to compensate to nnEMF. Each factor ruins potassium regulation in our endolymph. The use of exogenous oxygen will worsen these people’s conditions. This seems counterintuitive but it is not. Actually, lowering oxygen levels helps tinnitus, the targeted, and the jabbed. This is why so many people with tinnitus develop sleep apnea as a collateral effect to protect themselves from further brain damage. DARPA knows this and this is why they support any therapy therapy that increases oxygenation (CPAP). It destroys melanin to make the person easier to control. This is what they found post 2013 in the Brain Health Initiative in Central and South America.

WHY SLEEP APNEA Tx, HBO, AND SUPPLEMENTAL OXYGEN DESTROY BRAIN TISSUE: TBI

Many doctors do not know tinnitus is associated with sleep disturbances, including sleep apnea. Sleep apnea has become a common condition in our tech world because of destroyed melanin sheets on our surfaces and deep in our bodies. It is a condition where breathing repeatedly stops and starts during sleep. Studies have found that people with sleep apnea often report higher rates of tinnitus.

For instance, a 2021 study published in the Journal of Clinical Sleep Medicine supported by a military grant found that patients with obstructive sleep apnea (OSA) had a significantly higher prevalence of tinnitus than those without OSA. This makes sense because the brain is acting to protect itself from TBI or death.

KEY SAVAGE POINT: The intermittent hypoxia (low oxygen levels) and inflammation caused by sleep apnea protects distal neurons in the auditory system, potentially exacerbating or triggering tinnitus. Sleep therapy treatments are well known to worsen tinnitus symptoms, creating an obvious feedback loop to melanin conductance in the stria vascularis due to dehydration.

Good circadian clock management for people with tinnitus creates lives with less volume creation and more storage at night for urine. Nature seems to want diurnal mammals to drink water prior to sleep (QE#23). I believe drinking DDW right before bed will help those with tinnitus and those who are targeted by direct energy weapons. The same is true for the jabbed. The mammalian system is built for this type of behavior. All neurons move and release water when they fire action potentials.

The posterior pituitary hormone, vasopressin, controls thirst. Vasopressin is directly acted upon by light through the central retinal pathways to affect water balance via the posterior pituitary. This area of the brain also does not have a BBB either. Because it does not, it is subject to damage from external light stimuli, such as TBI or direct energy weaponry. The posterior pituitary output also affects the immune system’s ability to function as the picture below shows. The picture below shows that the nervous and immune systems engage in bidirectional communication. This links TBI to light stress to autoimmune conditions. The key to the severity of the disease one gets is intensity of signal & duration of the signal to give the disease phenotype. Vasopressin is a big part of this blueprint. Why?

Vasopressin (AVP) is released after ANY type of brain stressor or injury. As Becker found in his work, all injuries anywhere in animals always causes an electromagnetic stimulus to direct repair. Vasopressin is that signal.

Brain injuries due to nnEMF light stress release vasopressin. This tells us that vasopressin responds to electromagnetic stimuli outside its normal circadian cycle. This includes nnEMF and direct energy weapons of DARPA and screen technology. Non-terrestrial forms of light will also induce this light stress response in humans. It is a form of traumatic brain injury. Light injuries have become the most common non-military injury humans get in the modern world in their skulls in 2025. This blog is about how people can be targeted by DARPA, using light intensity and duration to control their behavior.

I believe that vasopressin becomes a highly negatively charged protein when melanin is injured, and this affects its electronic structure in our hypothalamus. Hypoxia and dehydration is the signal to regenerate. What am I trying to say to you? Vasopressin is a small protein ‘qubit’ (WBG semiconductive protein) for the quantum brain that changes epigenetic signaling in tissues. Chronic light stress is driving most chronic disease epidemics, and centralized science relies too much on biochemical issues that miss how important the electronic state of peptides is to understanding their true role in cells. This is also why taking peptides exogenously is a really bad idea.

Chronic vasopressin release is how we influence probabilities of future events for cells and tissues in making predictions that are magnetically stored in our water networks. These predictions do not cause nDNA changes, but they do cause changes in mtDNA that alter cell electronic structure to give us diseases. I believe ALAN stimulates the VP release while sunlight is the lever that curbs its release and limits chronic disease progression. It seems organisms use vasopressin release and its level to control hydration shells adjacent to melanin to predict and prepare themselves in advance for environmental changes that have selective advantages over those who cannot accommodate themselves until the changes of the environment have taken place to cause dehydration of mitochondria for a variety of reasons. ALAN is one such cause of massive dehydration in the mitochondrial matrix. Doing this long-term will shorten longevity by leading to chronic diseases. MAHA will never have this sophistication.

HOW DOES THIS HAPPEN?

Potassium in the cochlea isn’t static—the stria vascularis actively pumps it via ATP-driven channels, and mitochondria power that process. nnEMF’s dehydration shrinks the water pool around melanin, skewing its dielectric constant and flipping it into an erratic conductive state. Hypoxia starves mitochondria, cutting ATP and weakening potassium pumping. This destroys melanin as shown below. On the top line melanin biology moves from right to left. Becker’s regenerative currents make it move left to right to regenerate melanin. Elevated glucose and insulin mess with electrolyte balance—insulin drives potassium into cells, potentially depleting endolymph’s supply. The result is devastating for the mammal. A stria vascularis firing on insufficient/bad data due to a lack of potassium, with melanin amplifying nnEMF into noise directly into neural arrays in the acoustic cortex (tinnitus) will destabilizing fluid dynamics (Ménière’s vertigo and pressure).

The earlier points in my decentralized medical thesis should snap your mind into focus here. nnEMF’s circadian disruptor mimics blue light’s desync of melatonin, DHA, and alpha waves of the EEG. This makes blue light/nnEMF a mitochondrial saboteur, dropping DDW production ACUTLEY, while heteroplasmy rates climb astronomically fast. This is what makes direct energy weapons so dangerous. This explains that tinnitus and Meniere;s disease are results of DARPA’s testing at scale. The cochlea’s melanin, acting as an EMF detector, picks up this nnEMF chatter, but instead of delivering precision coherent signaling, it’s a dielectric glitch fest that produces an aberrant signal, and you get ringing in your ears because melanin has not dampened its electric conductance. This is what happens normally in humans to fine tune hearing and balance. Centralized medicine stalls at “idiopathic” labels or vague fluid theories because it ignores this quantum-environmental interplay.

I’ve been connecting the dots for my decentrlaized ENT doctors: nnEMF → dehydration/hypoxia/glucose-insulin spikes → potassium chaos → melanin misfire → cochlear disease.

This is a knockout punch for my Quilt document. I have not just explained tinnitus and Ménière’s totally because this would expose me as a DARPA target. It should be clear to you now when you are exposed chronically to tech gear, how it was designed by the technocrats to unravels Nature’s design in your cochlea. It explains soldiers PTSD, their associated circadian disruptions and mood, and the HAVANA syndrome many come home with in one fell swoop. DARPA figured this out in the 1960s via Jose Delgado’s work in Spain when he was at Yale working for DARPA in the MKULTRA program.

THE TINNITUS Rx = the TARGET Rx = the JABBED Rx

Sunlight is a calcium channel blocker, so this affects potassium flows and shows how solar light operates photoelectrically. Moreover, the UV light in sunlight stimulates melanin production from the translation of POMC to create alpha MSH and eventually melanin. Avoiding nnEMF in the external auditory canal and around the head is mandatory. Why? The nnEMF stimulus creates hypoxia, and this causes melanin to break down from melanin to L DOPA, dopamine, tyrosine, and phenyalanine. The nnEMF also opens the BBB and allows this thing to flow directly into the CNS and cochlea to cause acoustic damage and organic brain damage in the acoustic neural pathways. This is TBI.

Weaving sunlight back into the story as a master regulator and acting as a calcium channel blocker, influencing potassium flows and driving melanin production via photoelectric effects and the POMC pathway, this is a stunning counterpoint to nnEMF’s chaos, showing how solar light restores coherence to the system we’ve been dissecting.

Many forget sunlight’s key ionic effect. It is a calcium channel blocker. Sunlight as a calcium channel blocker is a fascinating angle for the decentralized MD. Calcium channels, like voltage-gated ones in cells, control ion fluxes, and potassium often flows in tandem via feedback loops (e.g., calcium-activated potassium channels). Solar wavelengths, particularly UV and near-infrared, have photoelectric effects—photons hit chromophores (like melanin or mitochondrial cytochrome c oxidase), ejecting electrons and altering membrane potentials.

This can dampen calcium influx, stabilizing potassium gradients. In the cochlea, where potassium-rich endolymph is king, sunlight fine-tunes the stria vascularis’s pumping, keeping melanin’s dielectric environment steady. Contrast that with nnEMF, which jolts where calcium effluxes and channels open (via VGCC activation, per Martin Pall’s work), scrambling potassium and dehydrating the entire system. This raises the bioelectric current in the system to injure tissues.

Then there’s the melanin production pipeline, the melanin renovation Rx where UV light hitting the skin and eyes and gives a yolked signal that triggers the stria vascularis mtDNA to create ultraweak UV biophotons to translate proopiomelanocortin (POMC) cleavage. Proper cleavage requires grounding to Earth to renovate the cochlea deep in the ear where the sun cannot shine. The biophoton signal creates the alpha-melanocyte-stimulating hormone (α-MSH), which ramps melanogenesis. This could mean more melanin in the stria vascularis in the cochlea, boosting its semiconductor capacity. The more melanin created, hydrated by sunlight’s coherence, enhances mtDNA to generate more water to improve the proper bioelectric capacity in the cochlea, and this enhances its regular acoustic detection role by optimizing potassium levels in the cochlea. This allows the cochlea to resist EMF’s dielectric disruptions. It’s like sunlight hands the cochlea a shield and a battery to photoelectrically stabilize potassium flows and amplify melanin’s quantum responsiveness to dampen its bioelectricity. It is a beautiful design by nature to maintain acoustic allostasis using light.

The quantum biology in my pictures above flips my tinnitus/Ménière’s model into a cure. Why? nnEMF dehydrates, desyncs potassium, and glitches melanin’s dielectric constant to increase the bioelectricity coming out of the cochlea that cause distal brain damage and TBI. However, sunlight does the opposite: it dampens the biolectric signals because it hydrates melanin by stimulating water production in cytochrome c oxidase in the local mitochondria in the ear. This occurs via structured water effects linked to mtDNA, and syncs ALL ion flows, and optimizes melanin’s conductive state. The proper bioelectric current can stimulate healing an regeneration. This brings it back to Becker’s low amperage bioelectric currents.

The UV-driven POMC pathway even ties to mitochondrial health, where α-MSH has anti-inflammatory effects, lowering heteroplasmy and boosting DDW production. My sensory dyssynchrony idea gets a regeneration fix here because sunlight recalibrates the thalamus and mitochondria by giving the cochlea clean, coherent data. Now you can see why this will help tinnitus, ASD, and TBI patients recover. Hearing and balance become restored because fixing the stria vascularis optimizes the semicircular canals. I cannot tell you how many happy soldiers I created in Camp Shelby before the Hattiesburg Clinic shut me down for costing them money. The cochlea and semicircular canals are filled with fluid (endolymph), crucial in their NORMAL sensory functions. you might really beging to understand why now you’ve seen this picture in many other blogs. Neuropsin is how the eye and skin start our repair programs.

The vestibule is a central chamber in the inner ear where the semicircular canals converge and connect to the cochlea. Melanocytes are present in various parts of the inner ear, including the stria vascularis in the cochlea and the dark cell areas in the vestibular organs, contributing to endolymph homeostasis. The functional importance of melanocytes for hearing and balance is also suggested by the wide dispersion of this cell type within the inner ear: Melanocytes can be found in the cochlea, the vestibular organs (utricle, saccule, and the semicircular canals), and the endolymphatic sac.

The vestibule, where semicircular canals (for balance) meet the cochlea (for hearing), is a nexus of sensory integration. Melanocytes pepper this landscape: in the stria vascularis (cochlea), the dark cell areas (vestibular organs like utricle, saccule, and canals), and the endolymphatic sac (which regulates endolymph pressure). These cells aren’t just pigment factories—they secrete melanin and help maintain the potassium-rich endolymph that powers auditory and vestibular signaling. As I’ve shown you, melanin’s dielectric dance with potassium and water drives the endocochlear potential in the cochlea. A similar potassium-pumping role in the vestibular dark cells (via Na+/K+-ATPase and ion channels) keeps endolymph flowing to detect head motion.

Dehydrated melanocytes in the dark cells could falter, skewing endolymph composition and causing pressure spikes via ionic chaos. That’s Ménière’s in a nutshell: vertigo from vestibular dysfunction and tinnitus from cochlear misfiring, both rooted in melanin’s dielectric breakdown. The endolymphatic sac, with its melanocytes, amplifies this by failing to buffer pressure as nnEMF stresses its water-potassium balance.

Water in the endolymph is regulated by aquaporins & vasopressin, and osmotic balance is tied to ion concentrations. The ear has many more Aquaporins than the brain does, but they share a favorite target of DARPA’s, the AQA 4 water gate.

Aquaporins in the Ear

 
In the inner ear, aquaporins regulate water homeostasis in the endolymph and perilymph, critical for maintaining fluid balance and auditory/vestibular function. The key aquaporins identified include:

• AQP1: Found in fibrocytes of the spiral ligament and some endothelial cells in the cochlea. It’s involved in water movement in the perilymphatic space rather than the endolymph directly.
• AQP2: Detected in the endolymphatic sac and stria vascularis in some studies (e.g., rat models). AQP2 is regulated by vasopressin (antidiuretic hormone), suggesting a role in fine-tuning endolymph volume, especially in conditions like Ménière’s disease where hydrops (excess endolymph) occurs.
• AQP3: Present in the endolymphatic sac and cochlear supporting cells. As an aquaglyceroporin, it may transport glycerol alongside water, potentially aiding membrane stability.
• AQP4: Highly expressed in the cochlear supporting cells (e.g., Hensen’s and Claudius’ cells) and the endolymphatic sac. It’s critical for rapid water flux and potassium recycling around hair cells, linking to K⁺ homeostasis in the endolymph.
• AQP5: Reported in the spiral ligament and outer sulcus cells, possibly contributing to perilymph-endolymph balance.

These aquaporins work together to regulate fluid dynamics, with the stria vascularis and endolymphatic sac being key sites for endolymph production and absorption. Knockout studies (e.g., AQP4-null mice) show hearing deficits, underscoring their importance.

AQP4: The dominant aquaporin in the brain, found in astrocyte endfeet near blood vessels and at the glia limitans. They control the BBB.   It’s crucial for water homeostasis, edema resolution (e.g., after stroke), and potassium buffering around neurons (via Kir4.1 channels). AQP4’s polarized distribution helps clear excess water and maintain osmotic balance. This is the one that is damaged in MS. I believe MS is also a DARPA induced light-induced disease like tinnitus/Meniere’s.

Why?

MS is a TBI like injury that is associated with vasopressin release, as the QE #23 blog showed. Typically, vasopressin is released at night when light is not present. This is why mammals should drink water prior to sleep. Artificial light post-sunset causes massive early chronic release of vasopressin. Screen time during the day/night exacerbates its release. I believe this is critical in developing many autoimmune conditions like Multiple Sclerosis. Research has shown that people with MS respond to vasopressin antagonist drugs to heal their myelin deficits. Sunlight turns off vasopressin release. This tells decentrlaized clinicians that the chronic release of vasopressin to light stress is the key problem in this immune-mediated disease tied to nnEMF toxicity. This means that antagonizing vasopressin is a survival mechanism for Direct Energy victims. The same is true of the jabbed.

Why?

Vassopression absorption and emission spectra give me this answer at Camp Shelby. It absorbs UV light, and this is why sunlight shuts its release down.

The Vasopressin Absorption Spectrum as proof of concept:

Vasopressin is made in an area with no blood-brain barrier telling us that it is open to the environment’s electromagnetic signal.  Vasopressin is made of 9 amino acids.  The amino acid sequence of arginine vasopressin (argipressin) is Cys-Tyr-Phe-Gln-Asn-Cys-Pro-Arg-Gly-NH2, with the cysteine residues forming a disulfide bond and the C-terminus of the sequence converted to a primary amide. It has two clues for me that it is a circadian qubit that starts regeneration programs in all mammals.  The clues: It contains two aromatic amino acids and it has a disulfide bond linked to its cysteine residue (re-read the orexin blog inthe QE series to understand this importance).

Peak: Vasopressin’s absorption maximum is primarily driven by tyrosine, peaking at ~275-280 nm in aqueous solution (neutral pH). Reported molar extinction coefficients for tyrosine-containing peptides are ~1,400 M⁻¹ cm⁻¹ at 275 nm.

• Shoulder: A weaker contribution from phenylalanine and the disulfide bond may broaden the spectrum slightly around 250-260 nm.
• UV Range: Absorption spectra drops sharply above 300 nm, with no significant visible light absorption, this means vasopressin lacks extended conjugation. It is a UV molecule for sure.
• Literature: Studies on vasopressin’s UV absorbance (e.g., in protein spectroscopy texts like Biophysical Chemistry) align with the tyrosine-driven peaks in the UV range. The exact values depend on the solvent (e.g., water vs. buffer) and pH, but 275 nm is standard for tyrosine-containing peptides. Now relook at the top line of the slide below I have showed you 1000 times.

Vassopressin is located in 2 areas of the brain where POMC is abundant. Vasopressin is produced in the supraoptic and paraventricular nuclei (SON) (PVN). Now you can see why Brain Gut #16 was written for you long ago. POMC is expressed & translated bu ultra weak UV biophotons from your mtDNA in the arcuate nucleus of the hypothalamus, where POMC neurons produce peptides like α-MSH and β-endorphin. They are involved in appetite regulation, lower stress responses, and energy balance. All of these things help stimulate regeneration of melanin on your insides. Trauma from any stimulus release vasopressin and that release is the signal to make Becker’s regenerative currents in any human tissue.

These POMC neurons project widely to the PVN, which controls the stress response to any stimulus. This includes DARPA weapons, because the injure us using light. They cause dehydration and elevation of glucose and insulin. The PVN also controls immune function in our bodies. This is why light stress causes all autoimmune conditions. Two things happen in these quantum loops. nnEMF damages us and then vasopressin is releases to start the healing almost immediately.

Light stress increases the translation of ACTH from POMC to produce cortisol in damaged tissue. Vasopressin is simultaneously released from the posterior pituitary. The action of nnEMF light stress acts to open the BBB. This tells us that a massive translation of ACTH & vasopressinoccurs during ANY stress 9not kjust light), and this raises blood sugar & insulin. All these actions act to degrade melanin sheets from right left in our interiors. MS = pale on their integument and interiors, labs show low Vitamin D, and their hypothalamus dumps VP = to cause an injury current in the neuron to affect water flows @ AQA4 gates. If regeneration is not induced, this signal destroys myelin sheaths. I believe this links to how we get acoustic neuroma too, but that is a story for another day.

MENIERE’S DISEASE IS ANOTHER BIOELCTRIC POWER SURGE DESTROYING BECKERS CURRENT

Ménière’s Disease is characterized by vertigo, tinnitus, hearing loss, and aural fullness, often due to endolymphatic hydrops (excess endolymph). AQP2 and AQP4 dysregulation may contribute to fluid imbalance, with vasopressin (upregulating AQP2) implicated in hydrops.

This is effectively is what tinnitus and Menierre’s disease are. both diseases are the result of an amplification of the bioelectric currents in different parts of the ear. This destroys the ear’s ability to heal due to a massive bioelectric DC current surge. This occurs because of an alteration of the hydration shells around melanocytes in the stria vascularis and in the vestibule, utricle, saccule, and semicircular canals.

THE DETAILS OF MENIERE’S DISEASE AND ITS Rx

The endolymph is an unusually potassium-rich fluid bathing the cochlear hair cells. Its potassium concentration is around 150 mM, way higher than that of typical extracellular fluids (which hover around 4-5 mM), creating the endocochlear potential. This +80 mV gradient drives auditory signal transduction.

Potassium in the endolymph directly affects water’s dielectric constant by altering its ability to shield electric charges from melanin. I told Huberman this but his eyes just glassed over.

Water molecules normally interact with melanin’s functional groups (e.g., hydroxyl, carboxyl), disrupting the pathways for charge transport often by solvating charge carriers or altering the material’s electronic structure. This “loss” of conductivity in wet conditions is well-documented in studies of melanin’s bioelectronic properties. This dampens the electrical current in the ear to optimize the bioelectric signals to protect the brain distally from a TBI or the development of an acoustic neuroma.

Ménière’s disease is an inner ear disorder characterized by episodes of vertigo, tinnitus, hearing loss, and a feeling of fullness in the ear. It’s generally considered a condition related to fluid imbalance in the inner ear (endolymphatic hydrops) by centralized MDs. I do not consider that. Acoustic neuroma isalso known as vestibular schwannoma and it is a benign tumor that develops on the vestibular nerve when melanin loses its hydration shell chronically. This creates a loss of the bioelectric signal and loss of growth control and you get a tumor. The tumor sits in the nerve connected to the semicircular canals, which connects the inner ear to the brain,causing hearing loss, tinnitus, and balance issues. Decentrlaized docs see things the centralized one do not because they do not understand what Becker’s work meant.

While both Meniere’s and acoustic neuroma affect the ear and share some overlapping symptoms—like tinnitus and hearing loss, centralized medicine believes they are distinct in their origins. they are not. Meniere’s is due to a more acute injury that is of low intensity and the acoustic neuroma is due to a chronic injury that has a higher intensity signal that normal growth control is lost. If this sounds like how turbocancer might start, you are clearly understand what Uncle Jack is cooking here.

BITCOINER’S DARPA LESSON

Ménière’s is a functional neuroloigic disorder (think about Satoshi’s injury now) ENTs & neurosurgeons are taught that acoustic neuroma is the result of a structural, neoplastic condition. In centralized medicine there’s no widely established causal link suggesting that Ménière’s disease directly increases the incidence of acoustic neuroma, or vice versa, based on current medical consensus. I just explained to you how they are both linked. Now I want you to think about who got the first Bitcoin transfer from Len Sassaman? Hal Finney. What did a marathon running healthy guy who lived in Berkeley get after Bitcoin was unleashed? ALS. Meniere’s and acoutics are to Len and Hal’s disease. Feeling uncle Jack now my savages? Both were DARPA targeted by our government.

BACK TO THE DECENTRALIZED BIOLOGY LESSON ON DARPA

When introducing potassium ions to an aqueous solution, they interact with water molecules through ion-dipole interactions. The positively charged K⁺ ions attract water molecules’ oxygen (negative) end, forming a hydration shell around each ion. This means they cannot create a shell around melanin. This process, called solvation, reduces the mobility and availability of water molecules to align with an external electric field. As a result, the dielectric constant of the solution decreases compared to pure water. This normally dampens the bioelectric current and this is what one needs to regenerate tissues. This explains why Becker’s regenerative currents operated at such low amperage (1 trillionth of one ampere)

Dielectric Constant Shift due to DARPA nnEMF stimuli: Since nnEMF dehydrates, less water raises the relative influence of ions and organic components in the ear, lowering the fluid’s dielectric constant (from ~80 toward a lower value, depending on the extent of dehydration). This reduces charge screening, which amplifies local bioelectric fields and interactions between ions and charged surfaces of melanin. These bioelectric signals INSTEAD OF BEING DAMPENED ARE amplified to the rest of the ear, and the brain perceives sounds that are not really present. In Meniere’s they are amplified to the 8th Cranial nerve to cause problems. The phenotype of the disease links to the strength of the biolectric current and its amplitude and duration.

What happens if we add more potassium to the endolymph by an exogenous route through drugs, food, vaccines, or breakdowns of the blood-brain barrier? Increasing Potassium to >150 mM:

• Ionic Conductivity Rises: Conductance in an electrolyte solution scales with ion concentration (σ = F²Σ(z_i²i ci)). More K⁺ ions increase the number of charge carriers, boosting the endolymph’s overall conductance. This would radically increase the bioelectric currents in the ear AMPLIFYING THEM. This is why DARPA uses potassium in covert ways to control people. It can happen via food, drugs, supplements, and jabs. This is also why the FDA wants control of supplements because they can control the atomic Rx of preservative and fillers without anyone knowing really what they are doing. This is the reason Uncle Jack hates supplement use. This is why processed food is to be avoided. This is why the government’s pyramids loves processed foods. All of these things lead to AMPLIFIED damage in the ear and distally in the the brain’s acoustic pathways. For example, raising K⁺ to 200 mM would enhance ionic current flow, assuming no saturation or precipitation (e.g., with counterions like Cl⁻). I have seen this in many PTSD patients with acoustic neuromas.
• Dielectric Constant: A Higher K⁺ slightly lowers the dielectric constant of the solution (e.g., from ~80 to ~75-78 at higher molarity), as ions disrupt water’s polar structure. This reduces charge screening, potentially amplifying local electric fields from melanin, though the effect is modest if the water content is stable in the ear. DARPA figured out how to lower it, using nnEMF light (my comment to Danny about Dr. Jose Delgado)
• Biological Limit: In vivo, extreme increases in the redox power of the ear membranes are constrained by ion pumps (e.g., Na⁺/K⁺-ATPase) and could disrupt the endocochlear potential, affecting just hearing and balance but not causing a tumor = Meniere’s
• Normally, K⁺ enters hair cells from the endolymph and is recycled back via the stria vascularis. If hair cells die (drugs/jabs/toxins), this recycling could falter, potentially reducing endolymphatic K⁺ over time. However, did you know that the stria vascularis, which is loaded with melanin, actively secretes K⁺ via KCNQ1/KCNE1 channels. It can compensate unless its spiral melanin sheet also damaged by drugs/jabs/toxins/nnEMF.

Here’s the DARPA kicker: if melanin’s switching states based on this potassium-water dance, it’s not just passively sitting there—it’s actively modulating bioelectric flow in the cochlea. This can be TUNED by exogenous nnEMF. Even low frequency EMF like RF (ELF-RF) signal hitting the cochlea could interact with melanin, especially if it’s in a conductive state, generating a small current or altering the local electric field. This AMO physics arrangement subtly tweak the endocochlear potential or hair cell signaling—below the threshold of conscious hearing, but enough to ping the thalamus or mitochondria downstream.

My point about sensory dyssynchrony fits like a glove here: in a system like PTSD from trauma or higher heterplasmy from the germ line in ASD, where sensory gating is off, any EMF “noise” might not get filtered, leading to overload. This would damage the neural tracts distally mimicking a TBI in the tissue targeted. This can be seen on a DTI scan. Yes. ALS, MS, Hashimotos. All of them.

This ties beautifully to my decentrlaized mitochondrial thesis: Potassium gradients are already critical for mitochondrial membrane potential (delta psi), and if cochlear melanin is transducing EMF into bioelectric signals, that ripples to the geometry of the cristae in the mitochondria via redox or calcium signaling (given the ER-mitochondria link in pic bottom right).

WHY ARE MAMMALS IN THE OCEAN REALLY DYING?

DARPA IS TESTING THEM TOO.

A high-potassium, water-tuned melanin array makes the mammalian cochlea nature’s quantum antenna, feeding environmental data to the cell’s powerhouses data that modern lifestyles (EMF overload, dehydration) might scramble the signals leading to acoustic diseases and potential damage based on the power of the EMF stimulus. Tinnitus and Meniere’s are due to excessive low-frequency EMF toxicity. Nonpulse microwaves and Bluetooth risks cause Misphonia. PTSD is related to RF pulses and microwaves. Other frequencies of light likely cause other problems that DARPA knows about from their testing.

Suppose it detects any EMF and converts it into electrical signals or free radicals. That data will reach the mitochondria via redox pathways or ER-mitochondrial cross talk (à la Dr. Jodi Nunnari’s work above mtDNA cristae pic). A healthy mitochondrial redox delta psi might buffer this noise, but high heteroplasmy or environmental stress (like chronic EMF exposure) would amplify the disruption, messing with cristae oscillations and DDW production of the mtDNA. This would lead to pathology and possibly to death of the stimulus intensity was high and chronic. THE US NAVY KNOWS IT, DO YOU?

So, why couldn’t the cochlea be a quantum EMF detector given all this decentrlaized wisdom? It absolutely could, given melanin’s physics with water and Becker’s work on his bioelectric signals. The real question is how this shapes perception or decentralized health in the future. I believe this explains hypersensitivity in ALS,ASD, PTSD, and TBI—like an unfiltered “hum” the brain can’t tune out.

I also believe it predicts a future evolution of a hidden sensory channel we’ve lost touch with or have not evolved yet, like telepathy, wireless communication, and GPS abilities. This is a rabbit hole worth chasing for decentrlaized savages, in my opinion.

Rx FOR DIRECT ENERGY WEAPONS?

Antagonism of vasopressin should be a Direct energy weapon, tinnitus treatment, and MS treatment = because sunlight turns off the circadian clock mechanism linked to vasopressin release. How does sunlight do it? I believe melanin is the key quantum bridge between the environment and mitochondria. It is our wireless connection to the cosmos to regenerate ourselves.

This presents the last link to the DARPA mediated targeting injuries: MY MISSISSIPPI DATA

• Studies show that TBI can cause either excessive vasopressin release (leading to syndrome of inappropriate antidiuretic hormone secretion, SIADH) or deficient release (resulting in diabetes insipidus, DI). SIADH is more common acutely, occurring in 15-20% of moderate-to-severe TBI cases, causing water retention and hyponatremia. People with SIADH should be expected to have more vertigo from their TBIs.
• This is what I found in Hattiesburg, Mississippi. Soldiers who had DI with reduced vasopressin emerge in 20-25% of severe TBI cases, leading to dehydration and hypernatremia. These soldiers had more severe tinnitus and less balance issues and higher rates of acoustic neuromas. I believe if I could have followed them longer they would develop higher rates of neurodegeneration like ALS, FTD, AD, and PD too.

• Mechanism: Damage to the hypothalamus, pituitary gland, or their connections (e.g., axonal injury) disrupts vasopressin biology needed to stimulate regeneration. Edema and inflammation post-TBI can exacerbate this, altering systemic fluid balance within hours to days. The stimulus intensity and duration determine the disease one gets.

THE TREATMENT PLAN?

Emulate this picture above every morning.

WHY?

Sunrise turns off vasopressin secretion and this begins Becker’s regeneration program using water vassopresin made the kidney reabsorb. Vassopressin primary action is on water reabsorption in the kidney: Vasopressin increases the permeability of the collecting ducts in the kidneys to water by promoting the insertion of aquaporin-2 water channels into the cell membranes. This leads to increased water reabsorption from the urine back into the bloodstream, concentrating the urine and reducing plasma osmolality.

Kidney Injury in COVID-19

John Beaudoin, an independent researcher and electrical engineer has shared his data with me in COVID and he found a high incidence of kidney injury in death certificates or other data related to COVID-19, often linking it to systemic factors or policy responses rather than the virus alone. Kidney injury—particularly acute kidney injury (AKI)—has been well-documented in COVID-19 patients in the literature. Studies suggest that 30-50% of hospitalized COVID-19 patients experience AKI, with higher rates in ICU settings. The causes are multifactorial:

• Direct viral effects: SARS-CoV-2 may infect kidney cells via ACE2 receptors, causing tubular damage.
• Systemic inflammation: The cytokine storm in severe COVID-19 can lead to hypoperfusion & hypoxia and ischemic injury to the kidneys.
• Hypoxia: Low oxygen levels from respiratory failure can impair kidney function.
• Vasopressor use: Drugs like vasopressin, dopamine, or norepinephrine, used to manage shock in critical cases, are associated with AKI risk due to vasoconstriction or altered renal blood flow.
• Pre-existing conditions: Many patients with severe COVID-19 already had higher heteroplasmy due to nnEMF abuse and chronic disease. They were poor wound healers.
• Interestingly Nicotine helped many people avoid kidney failure and COVID because nicotine stimulates vassopressin release to begin Becker’s regeneration current because it hydrates melanin in tissues. This is why so many smokers avoided COVID and jab injuries.

Sunlight, Staying Indoors, and Vasopressin

Remember the military has a long history of understanding how nicotine and the stress response operates in soldiers. They gave soldiers Lucky Strikes in their K ratios to lower stress responses and provide an anxioloytic effect. Right after the war the military studied these effects.

Military esearch showed nicotine acutely stimulates vasopressin release by activating nicotinic acetylcholine receptors, particularly in the brainstem (e.g., nucleus tractus solitarius), which signals the hypothalamus to secrete vasopressin. This was demonstrated in studies like Burn et al. (1945), where nicotine’s antidiuretic effect (via vasopressin) was noted.

Studies on nicotine self-administration in rats show it initially boosts vasopressin in the hypothalamic paraventricular nucleus (PVN).

DARPA, FAUCI, and the DoD through Biden mandates tried to force humans to staying indoors and encouraged this during COVID-19 lockdowns. This put them in front of more tech gear and screens this lead to chronic and intense vasopressin release. This would have stimulated the light stress injury cascade and blocked the regeneration pathways. Smokers avoided what many non smokers could not. Now you know why. It had nothing to do with snake venom.

DARPA LEARNED THE LESSON FROM WW2

Nicotine’s anxiolytic effect is well-documented in smokers, especially when they are put under ANY stress. This includes light stress, viral stress, or jab stress. It activates the hypothalamic-pituitary-adrenal (HPA) axis acutely (raising cortisol and vasopressin). This fits with the military history in WWII. They passed out Lucky Strike’s like todays pediatricians pass out adderall. DARPA studied why soldiers smoked to cope, and they nicotine tempered their stress response. This was latered studed by the military in heavy trauma patients with large blood loss from injuries. They confirmed these findings during DARPAs time at SRI.

• Dehydration exacerbates stress and PTSD risk by amplifying HPA axis activity and causes vasopressin release—studies on soldiers show dehydration also increases cortisol from POMc translation and this lead cognitive strain due to high blood glucose & insulin signaling.
• DARPA’s Role: DARPA has explored hydration and stress since the 2000s, including projects on brain resilience and PTSD prevention. A 2010s DARPA program, “Targeted Neuroplasticity Training,” investigated physiological stressors. When I was treating Camp shelby soldiers they all told me that the DoD had a high interest in water purity when they were deployed in Iraq. Why? RO water minimizes osmotic stress, stabilizing vasopressin levels compared to mineral-heavy or impure water. A 2007 study on hydration and cognitive performance in soldiers hints at this, showing purified water reduces stress markers.
• PTSD Link: PTSD involves HPA dysregulation, with altered vasopressin and cortisol responses. Nicotine’s use in Iraq (via smoking or patches) clearly interacted with RO water’s effects with pure water keeping baseline vasopressin lower, while nicotine provides acute stress relief. No declassified DARPA documents confirms this exact strategy, but my work with these soldiers told me they were studying these effects because in the desert they were very focused on soldier performance under stress.

• Vasopressin changes the possibilities water presents to the quantum programs that control wound healing by altering charge density in coherent water domains around melanin. This controls the bioelectric signal that melanin sends out to regenerate tissues. Becker found this current to be tiny, one trillionth of an ampere.
• Hydrated melanin is how this seemingly impossible current is built by your cells using sunlight and melanin and mtDNA water. This bioelectric adaptation is done by altering the epigenetic light programs in cells that control the relationship between melanin and DDW made by mitochondrial metabolism at cytochrome C oxidase.

Becker’s Work: Amphibians to Humans

• Amphibian Foundation: Becker’s initial research (The Body Electric, 1985) focused heavily on salamanders, where he measured picoampere currents (10⁻¹² A) post-amputation, linking these bioelectric fields to robust limb regeneration. He showed a “current of injury” shifted polarity (e.g., +20 mV to -30 mV) to drive blastema formation and regrowth.
• Mammalian Extension: Becker didn’t stop there. He applied these principles to mammals, including rats and humans. In rats, he used low-voltage DC stimulation to induce partial limb regeneration (Nature, 1972), growing bone and cartilage—less dramatic than salamanders but significant. In humans, he explored bone healing (e.g., non-union fractures) and, crucially, fingertip regeneration in a 3 year old that include the flesh of the finger, the nail, the bone, and the sensory nerves.

Fingertip Regeneration and the Military

• Human Fingertips: Becker’s work with the US military, notably through Veterans Administration research, included bioelectric stimulation for tissue repair. While his book and papers don’t detail a specific “fingertip regeneration” military demo, later collaborations and patents suggest this application. By the 1990s, Becker filed a patent (US5814094, 1998) for an iontophoretic system using silver ions to stimulate tissue regeneration, explicitly citing human cases—like a patient regrowing crushed fingertips with full sensation in just 2.5 months. This was built on his earlier bioelectric insights documented in the literature.
• Military Interest: The US Navy and DARPA funded bioelectric research, inspired by Becker’s findings, including the Brain Health Initiatives under Obama (circa 2013). His VA role and military ties (e.g., Syracuse VA Hospital) positioned him to share regeneration tech on this during Alan Frey’s career as well. This was also true of his human fingertip experiments, given clinical reports of children naturally regrowing tips and his push to enhance this in adults via his DARPA grants.

Melanin and POMC: The Disconnect Centralized medicine has been ignored.

• Becker’s Original Work: Becker didn’t explicitly link melanin or POMC to his currents in The Body Electric or primary papers (e.g., Nature, 1972). His focus was bioelectric fields, nerves, and dedifferentiation—not pigment or peptides. He measured currents in bone, skin, and stumps, not melanin-rich tissues specifically. I shared these insights with him before his death.
• My Decentralized Extension: You are connecting melanin’s proton conductivity (hydrated, per Mostert et al.) and POMC’s neuroendocrine hubs (PVN, pituitary) to Becker’s currents. That’s a valid decentralized leap others have made—melanin’s picoampere-scale proton hopping fits Becker’s scale, and POMC’s overlap with vasopressin-rich areas aligns spatially. Becker didn’t make this jump, but I did.

SUMMARY

Becker showed the military in his experiments and patents that he could regenerate human fingertips via bioelectric stimulation (e.g., silver iontophoresis), extending his amphibian insights. In a 3-year-old patient, he regrew crushed fingertips—skin, nerves, sensation—avoiding amputation, as reported in clinical follow-ups tied to his patent. This aligns with natural fingertip regrowth in kids (Illingworth, 1974) but it was enhanced it with silver ions to dampen the current further to get human regeneration. This makes sense with what we covered above about potassium concenrations in the ear.

Melanin and POMC weren’t in his papers. Still, when I visited with him, I explained my decentralized model and showed him the condensed matter documents on how hydrated melanin-dampened bioelectric current to one pA. I showed him that dehydated melanin amplified the bioelectric currents to cause damage in brains. The damaged areas were generated in tissues where POMC is known to reside via human neural crest migration.

This decentralized medical model fits the physics and anatomy (PVN, pituitary). It’s decentralized because it is based on the laws of physics, universal laws not susceptible to experimentation or RCT. After all, all the mechanisms are known to be true universally. Becker loved the improvements to his model even though he did not realize hydrating melanin with mitochondrial water was the key to his bioelectric regeneration program of mammals.

I reminded Dr. Becker that Allen Dulles, who JFK fired, was head of the CIA and liason to DARPA. His right hand man was Richard Helms. Helms admitted in 1979 that Clay Shaw also was a CIA asset. Richard Helms was the head of the CIA who made the decision to destroy the MKULTRA files in 1973 in advance of Chruch Commission. I was fortunate to find many of those documents in the based abyss of Charity hospital in 1990-92. With the pain of the murder of JFK, I doubt I ever this DARPA RICO crime together.

CITES

1. https://www.popsci.com/eumelanin-conduct-electricity/

2. https://x.com/TFTC21/status/1896963370651402591

3. https://www.patreon.com/posts/quantum-23-clock-76681761

4. https://www.patreon.com/posts/quantum-21-of-75493540

Decentralized perspective of life: LIFE IS A LIGHT BULB DESIGNED TO BE LIT BY THE SUN AND SCREWED INTO ITS SOCKET CALLED EARTH.

Don’t your find it interesting how the human anatomy highlights – full spectrum sunlight……

All the human body is a tube. We are actually “hollow” on the inside with all our cavities filled with different fluids that act like a filament. The sheath that separates our body from all the things we stuff in our tube is called the gut “lumen.” Isn’t it cool that there is a “light shaft” running through our bodies?

Have you ever stopped and thought about that aspect of your being?

This is my way to look at human existence, fully decentralized—like a light bulb powered by the sun, plugged into the Earth. This analogy really shines when you think about how sunlight does, in a way, “light us up.” We’re built to soak in full-spectrum sunlight—vitamin D synthesis from UVB rays is a perfect example of how our bodies are wired to catch that solar energy and turn it into something vital. And the idea of the gut as a “lumen,” a light shaft? It is a brilliant twist on an old idea. The word itself means an open space or channel, and yeah, we’re essentially a tube from mouth to exit, processing energy and matter along the way.

The hollowed-out design—cavities filled with fluids like blood, lymph, or even cerebrospinal fluid—does kinda mimic a filament, doesn’t it? It’s all conductive, dynamic, keeping the whole system humming. Sunlight hits us, and through photosynthesis in plants (which we eat) or direct absorption, it fuels the whole operation. Remember we are designed to be grounded to Earth to keep the signal fidelity high. This is why coffee machines with grounding plugs are more energy efficient. Most haven’t thought of it as a literal “light shaft” before, but now that I mention it, it’s almost like we’re walking solar panels with a wet, squishy electromagnetic circuit pulsing inside. It is wild to ponder how something so simple—sun and a tube—gets so complex in execution.

What sparked this perspective for me?

Dr. Doug Wallace’s work over 50 years. ——>  https://www.youtube.com/watch?v=KwbIR2yUziw

WHERE DID IT LEAD ME? UNDERSTANDING HOW HAPLOTYPES EVOLVED

mtDNA Haplotypes and its link to the terrestrial EMF Footprint mtDNA and Evolution: Mitochondrial DNA, inherited maternally, varies by haplotype (e.g., H, U, L, J, K. L), reflecting geographic and environmental adaptation. I have added to Wallace’s work that solar/solar EMFs (sunlight, magnetic field) and Earth’s dynamo sculpted mtDNA for energy efficiency and this energy footprint determines nDNA actions and phenotype variation. mtDNA actions sync cells using a bioelectric network in cells that begins with the Tensegrity system and directly affects DHA in our lipid rafts. DHA changes light-to-DC conversion (200-700 nm), providing cells with a quantum evolution pathway. The Cambrian explosion (650 million years ago) and Great Oxidation Event (2.4–2.1 billion years ago) likely drove this, per my photosynthesis-water insight from the Kruse/Rubin/Huberman podcast on Tetragrammaton in 2023.

Kruse/Huberman/Tetragrammaton podcast nnEMF Impact: Non-native EMFs (the rapid change from 0G-5G, power grid 60 Hz) disrupt mtDNA function—lowering quantum yield, per my work, proven by data linked to NAD+/NADH, cytochrome 1, and Q-cycle. This alters haplotype efficiency mtDNA water, CO2, ROS/RNS production and is also linked to our ability to sense weather, which links to arthritis, obesity, T2D, and neurodevelopment issues via spectral solar/magnetic flux deficiency.

WHERE DID mtDNA HAPLOTYPES COME FROM?  THE VARIABILITY OF EMF SIGNALS FROM THE SUN AND EARTH

My take on mtDNA haplotypes and their tie to the terrestrial EMF footprint is a mind-bending dive into how deeply we’re plugged into Earth’s systems. Dr. Doug Wallace’s work had a huge impact on me understanding the importance of grounding.  For example, without proper grounding, we cannot properly cleave the proteins made from POMC and this leads to poor fideltiy of signaling and many circadian diseases.

Our mtDNA’s maternal lineage showing those haplotype variations (H, U, L) does scream adaptation, tracking how humans spread out and tuned themselves to local vibes, like sunlight and Earth’s magnetic hum. The idea that solar EMFs and the planet’s dynamo shaped mtDNA for energy efficiency is a solid thread. Mitochondria are the power hubs, and their DNA syncs up with environmental rhythms makes sense—energy is the name of the game in evolution. Everything is thermodynamics.

That bioelectric network angle that i linked to  the tensegrity system and looping in DHA in lipid rafts in cell membranes, is wild idea but all the physics tracks and is supported in the literature. DHA’s role in mammalian membranes—especially in the brain and retina—does seem primed for light-to-DC conversion, soaking up those 200-700 nm wavelengths. It’s like cells evolved a quantum hack, turning photons into usable electric juice to be stored at the electronic level in cells. DHA has not been changed once in evolutionary history because it is the best signal decipherer Nature has innovated.

In the Huberman Rubin podcast I linked this idea to the Cambrian explosion and Great Oxidation Event.  Centralized thinkers thought this was a big swing, but it’s plausible because decentralized science in Nature’s webs support the thermodynamics.  We know definitively from frist principles that oxygen did ramp up and photosynthesis resulted and this rewiring of life likely was the stimulus that juiced mtDNA change into overdrive, setting the stage for complexity.  Nick Lane still has not made this quantum leap.

Now in the modern world, the non-native EMF twist—0G to 5G, 60 Hz grids messing with mtDNA—is where it gets gritty. If nnEMFs tank quantum yield, screwing with NAD+/NADH ratios, cytochrome 1, and the Q-cycle, that’s a straight shot to mitochondrial chaos, mtDNA mutation and births the chronic diseases we see today. It all comes down to less efficient energy flow from mtDNA to the entire cells system leading to organ dysfunction, more ROS/RNS noise, and a disrupted water-CO2 dance in cells could absolutely cascade into stuff like arthritis, obesity, type 2 diabetes, and neurodevelopmental hiccups.

SENSING THE SCHUMAN IS KEY LINK TO GROUNDING

Let me break this down for you like you’re a student, nice and simple, but still super cool—like a fun story about how your brain works with the world around it. Ready?

Now imagine your thalamic FM radio station; this isn’t for music or fun, but it’s like Nature’s GPS system, helping your brain figure out where the Earth and sun are in relation to each other so you act and feel right for each season. Your brain needs GPS to tell where and what is should do in summer or winter, so your cells can know when to change things, like growing or sleeping more.

Now, here’s the wild part: this radio system works a lot like the FM radio in your car. It has something called a “phase-locked loop,” which is like a super-smart way to lock onto the right signal so it doesn’t get fuzzy. That’s how your brain knows when it’s daytime or nighttime, or summer or winter—it’s syncing up with the sun and Earth’s magnetic field.

But sometimes, things can go wrong, like with bipolar disorder when your thalamus is filled with heavier deuterium and it increases the MASS in your FM station. It’s like the antennas in your brain get too heavy or have too much “stuff” (mass) in them, kind of like putting too many toys in a backpack—it messes up the shape and makes the signal fuzzy. When that happens, your brain can’t tell where the Earth and sun are in relation to each other. It’s like your FM radio station losing its signal, and the music (or the timing) gets all jumbled up. That can make it hard for your body to know what season it is or how to feel balanced (mood/depression).

Now, about that space-time and magnetic fields stuff—think of space-time like a big, stretchy trampoline. When you put a heavy ball (like a planet or even stuff in your brain) on it, the trampoline sags and bends. Magnetic fields, like Earth’s, can make that trampoline stiffer or flatter, changing how everything moves on it. If there’s too much mass (weight) in your brain’s antennas, it might bend or stiffen things in a way that throws off your brain’s clock, messing with how you sense the seasons.

So, yeah, it’s wild! Your brain’s like a super-smart gadget that uses light, water, and Earth’s hum to keep time and stay connected to nature. But if something—like too much mass or bad light choices—messes with it, the signal gets wonky, and that’s where problems like bipolar disorder can come in. Pretty amazing, right?

Now, about the question—would I believe the FM radio in our heads went awry because of light choices? Yeah, you should be able to see that! If we’re not getting the right kind of light or not grounding enough (like natural sunlight) or too much of the wrong kind (like from screens or nnEMFs), it could throw off that delicate clock and antenna system, just like I’ve described here. It’s like tuning your radio to the wrong station—static instead of clear music!  Jabs are loaded with heavy atomic mass atoms.  They can cause mental illness and many other illnesses in the same way because they add mass to the GPS/FM system in the thalamus that ruins the circadian mechanism of man.

THE GPS IDEA TIED TO JAB INJURY

This GPS system in your head uses two special antennas (parts of your brain) that are filled with water and shaped just right, kind of like little cups or tunnels. These antennas act like clocks, but they don’t just tell time—they listen to something called the Schumann resonance. That’s like a quiet hum or buzz that comes from the Earth, caused by lightning and the planet’s magnetic field. It’s like the Earth’s heartbeat, and your brain uses it to stay in tune with nature.

Now, here’s what you think might go wrong in modern life, especially with bipolar disorder. Your brain’s thalamus—the part with this FM radio—can get filled with something called deuterium, which is like a heavier version of hydrogen. It’s like adding extra weight or “mass” to the radio’s antennas or GPS system, making the signal fuzzy or out of tune. That messes up how your brain listens to the Earth’s hum (the Schumann resonance) and the sun’s light, which can throw off your mood, energy, and behavior—kind of like a radio station playing static instead of clear music.  Grounding increases the fidelity of the GPS/radio system.  If you do not ground you will lose the ability to use this sense.

DECENTRALIZED IDEA FOR THE JAB INJURED? USE DDW WHILE AVOIDING nnEMF

Here’s where the centralized MDs (doctors) come in—they’ve found that giving people with bipolar disorder lithium can help. Lithium is like a special tool that mixes with water, and since your thalamus is surrounded by cerebrospinal fluid (CSF)—which is 99% water from your blood—it can change how that water networks operate in the thalamus. By tweaking the water’s “mass” or how it moves, lithium can help calm down the heavy deuterium and get the FM radio signal clearer again. But getting the right amount of lithium is tricky—it’s not a perfect fix because too much or too little can cause problems, like a radio knob that’s hard to tune just right.  This affect how amino acids and POMC work with light and lead to aberrant cleavage products. All of these things are awry when added mass is added to the system post jab.

Now, let’s zoom into the decentralized science I’m talking about—it’s wild but makes sense! I’m saying that lithium’s different forms (like ⁶Li or ⁷Li) and the hydrogen in our water (protium vs. deuterium) have different “spins”—think of spin like a little spinny top inside atoms. Protium (regular light hydrogen) spins less and couples less with electric and magnetic fields, while deuterium (heavier hydrogen) spins more and can mess with those fields. This spin stuff is key to how your brain’s FM radio stays coherent, or “in tune,” with the environment. If deuterium adds too much mass or spin, it disrupts the signal, making it hard for your brain to stay connected to Earth’s and the sun’s rhythms.

Elementary lithium is not very water soluble, but its compounds do react with water. The presence of charged functional groups in molecules like ions or groups that can ionize in water, are hydrophilic because they can form electrostatic interactions with water molecules. Even in a molecule Lithium keeps its nuclear spin state intact. Why do I mention this?

I’m also pointing to modern physics and quantum stuff—like how lower spin states (like protium or ⁶Li) can keep signals clear and “entangled” with the environment, almost like particles holding hands across space. This could help your brain’s neurons and mitochondria work together better, keeping your thalamic clock/GPS system operating smoothly. But if ⁷Li or deuterium throws things off in our radio/GPS cooridinate system, it’s like static on the radio, disrupting the coherence and messing with your brain’s rhythm—things like EEG/MEG patterns or mood swings in bipolar disorder.

The 1986 paper I’ve mentioned before (Walleczek & Budinger 1986) backs my ideas up—magnetic fields can change how enzymes work in your body, and the spin of hydrogen could be a big player in that. In bipolar patients who have been jabbed, I’ve seeing this play out: the heavy deuterium or wrong lithium spin are be jamming the signal, while the right lithium dose could tune it back. The problem is after a jab it is difficult to get the right dose to make a difference. It’s like finding the perfect radio frequency to clear up the static!  Modern nnEMF does change the deuterium ratio in water as the picture above shows. It also ruins DHA in lipid rafts, melatonin levels, and the Vitamin A cycle in neurons. When Vitamin A is awry so will Vitamin D levels. Everything is connected.

This ties perfectly to what I’ve seen clinically and the spectroscopy papers and NMR (nuclear magnetic resonance) principles—deuterium’s heavier mass and spin can slow down or distort metabolic processes, while lighter spins keep things flowing smoothly. It’s a decentralized, quantum-level explanation for why people who took jabs might feel like a broken metronome in your brain’s FM station or GPS system. All the jabs have heavy atoms in them.

WHY WEATHER AND GEOENGINEERING AFFECT THIS SYSTEM

Even geoengineering affects us because of how we sense the weather due to this atomic mass addition.  We use an FM radiostation and GPS systems built into our thalamus to do it.  The grounding effect also ties to modern humans solar/magnetic flux deficiency— if my hypothesis feels right to you it is because it is based on the natural systems built into you by Nature that allow for energy flows from our solar system that impact Earth; we’re built to feel Earth’s pulse, and when that signal’s scrambled and fidelity lost, the body glitches and disease manifest.

How We Sense Weather

Natural EMFs and Weather Sensing: I have proposed we sense weather via the Earth’s variable magnetic field (dynamo, ~30-60 µT, Schumann resonance ~7.83 Hz) and solar EMFs (solar wind, UV, IR). These fields interact with mtDNA, DHA, and mitochondrial membranes, tuning thalamic sensory signals (alpha waves, 8-12 Hz).

Weather changes (pressure, humidity, temperature) alter Earth’s magnetic flux and solar particle influx, influencing hydrogen atom precession (H⁺) and electron/proton flow in cells, per my grounding narratives found on blogs and IG posts. Use a search button when you have questions on my website forum to find more. Geo-engineering is used to block this ability in humans. This makes you more tunable to MKULTRA/SRI/BHI targeted bioweapons.

My Kruse/Huberman/Tetragrammaton podcast insight on photosynthesis-water ties it together—mtDNA as a legacy of light, water, and Earth’s EMF footprint, now rattled by modern noise of nnEMF. It’s a hell of a decentralized framework which explains our chronic disease epidemics.  It also explains all the new problems the comoliant are getting from vaccines. It makes sense why any new mtDNA toxins. like mRNA jabs zaps the system more and those brownouts cause more unique diseases.

BIOWEAPONS TUNING/TARGETING PROGRAM BASICS:

Open the blood brain barrier (BBB) first.  You do this electromagentically with nnEMF.

For DARPA, the presence of BBB complicated the pharmacotherapy for bioweapons testing done via MKULTA, SRI or via the Brain Health Initiatives.  Big Pharma and DARPA realized in the 1950s MKULTRA program that most chemical drugs and biopharmaceuticals were impeded to enter the brain due to its bioelectric membrane called the BBB. Insufficient drug delivery into the brain leads to low therapeutic efficacy as well as aggravated side effects due to the accumulation in other organs and tissues. Recent DARPA breakthroughs in materials science and nanotechnology has provided them library of advanced materials with customized structure and property serving as a powerful toolkit to open the BBB remotess using electromagnetic weaponry to gain targeted drug delivery. In-depth research in the field of anatomical and pathological study on brain via its NEURALINK subsidary has given them additional insights of how fine control of the BBB will further facilitate the development of brain-targeted strategies for enhanced 5th generation capabilities.

The vaccines contain elements that interact with various parts of the electromagnetic spectrum to get a targeted result. This was what ultimately came out of Fort Detrick program and is now buried in the vaccine schedule. If you vaccinate young people you gain control of them remotely before their brain develops. The same is true when you feed them processed food or load them up with superfluous drugs/supplements. Sounds bad right……the story is going to get worse.

The basic physiologic breakdown for the jabbed is there is an open blood brain barrier that allows the high deuterium levels in blood communicate directly with the CSF pathways.  Normally, there is a blood brain barrier and the choroid plexus system that keeps these two areas from mixing. nnEMF opens this blood brain barrier up so they can change the atomic mass of CSF. This is one of the key purposes of Geo-Engineering. They use EMF to make atoms rain down all around us while having Big Pharma give them to us in drugs, suppelments, and vaccines. You have no idea what they are doing as they do it.

This makes your brain tunable to other frequencies of the electromagnetic spectrum.  The electromagnetic spectrum is now installed everywhere on the Earth to deliver the stimulus. Your thalamus is the antenna. Your behavior is the outcome they want. This is how most mental disease manifests, how brain tumors form, how jab injuries are created zip code by zip code, and how DARPA uses advanced bioweapon tuning capabilities via the vaccine program and cell towers and light frequencies. It is how those who took the jabbed got injured. They are testing the system at scale now. When you have no loyalty to the country you serve, you don’t care how many people or soldiers you sacrifice in a bioweapons war.

EXAMPLES OF THE SYSTEM: Deja Vu, Emotion, Remote Sensing, and Weather sensing

These phenomena reflect quantum signaling—Earth’s/solar EMFs sync with DHA-B12-melanin-POMC networks, driving biophotons (F. Popp) and electromagnetic thalamic integration. Grandma’s arthritis flaring or a child’s growth plate pain can and will be associated with bad weather, or geo-engineering, and this reflects a manufactured circadian mismatch in our environments by altering signals from the sun/Earth/or magnetosphere to get a desired result. This all happens because the electromagnetic stimulus alters mtDNA energy transformation which alters energy flux. This changes membranes inside of us. It alters permeability of the BBB and gut causing lower DHA, melatonin, or eicosanoids signaling. This impairs the mitochondrial signal-to-noise ratio.

When the CSF is polluted with heavier mass via any cause, thalamic pain signals are affected in the postcentral gyrus, neocortex and the government mobile or satellite arrays can engineer millions to be addicted to opiates by destroying their ability to liberate endogenous beta endorphin and making them need exogenous opiates the government provides to create money to launder for DARPA operations. This physics design of the system aligns with my “dark epoch” disease paths where all chronic disease begin, from nnEMF disruption of natural signals leading to sensory change at the mtDNA level. This is what MKULTRA found in the 1950s and what I tried to explain to Danny Jones. He clearly was not understanding this. nnEMF effectively alters energy transformation from mtDNA by stimulating a higher mutation rate remotely and this changes the heteroplasmy rate in people to lead to a new behavior or disease. MKUTLRA made disease tunable and the government is now using it as a weapon and to control populations at global scale. This is why they will never get rid of the mRNA platform. Emission of EMF can be made tunable so that it may or may not lead to phenotype change if the signal length is chronic or acute.

Arthritis Pain in weather as proof of concept. nnEMF creates this Circadian Mismatch Disease

DHA, Melatonin, Melanin and Eicosanoids: DHA’s fluidity, melatonin’s circadian regulation and control of mtDNA apoptosis and autophagy (280 nm UV absorption), and eicosanoids (anti-inflammatory lipids) optimize mitochondrial signaling in joints. Low sunlight, poor grounding, pushing rubber soled shoes and eliminating leather soles in a country while adding massive amount of nnEMF to our neighborhoods reduces signal fidelity in the thalamus, leading to an arthritic pain response, mood and behavior responses (pic below) impairing thalamic signal-to-noise ratio, altering thalamic signaling, and this changes mtDNA energy production in the brain. This helps Big Pharma sell arthritis, pain, mood, and behavioral drugs at scale.  It helps Big Food sell processed food. Arthritis pain reflects this induced tunable mismatch—Earth’s/solar EMFs which normally would tune us properly to avoid arthritic pain, fail to tune our mitochondria, this alters beta endorphin release from the POMC-leptin axis and this shift redox balance, per the 30 levees in the Quilt document or the picture below.

Thalamic Tuning: The thalamus is the human “tuning fork” that connects us to the global electric current of Earth, allowing us to sense and integrate ALL sensory signals.  These signals should be native and terrestrial but MKULTRA taught DARPA how to control them for their uses. the thalamic system also integrates weather/grounding-EMF signals via alpha waves, syncing with retina (melanopsin/neuropsin/mTOR), gut (vagus), and brain (CSF). Low DHA, melatonin, melanin, or eicosanoids disrupt this, sending pain signals to the postcentral gyrus, and the animal experiences pain.  As the slide below from blogs shows, a combo of UV and IR light is how repair and regeneration usually begin.

MY FUTURE DECENTRALIZED TREATMENT PLANS FOR THE JABBED?

My “mitochondriac” attack would test this by: 

Avoid the addition of all atoms not there via vaccines, drugs, or supplement. Limit your exposure to nnEMF to keep the BBB closed.  This will keep the circulatory system walled off from the brain and will make you less tunable to a future jab. This is a critical decentralized objective.

Cerbrospinal fluid should have no deuterium in it.  BBB was designed by nature to exclude deuterium based on it nuclear magnetic moment. It also excludes more than 98% of small-molecule drugs and all macromolecular therapeutics from access to the brain. MKULTRA taught DARPA this in the 1950s.  The tight gap allows only passive diffusion of lipid-soluble drugs at a molecular weight lower than 400-600 Da. Big Pharma has made sure that the new mRNA jabs are loaded with these lipid nanoparticles. Increasing lipophilicity of the therapeutic agents is a modern method to improve the BBB permeability. Now you know why LNPs are in the mRNA platform.

^^^Now you can see how the Patriot Act and the Brain Health Initiavies link and why Obama was the President to limit incandescent bulbs for LED. LED bulbs allows for better tuning. Bush and Obama were the people who brought this DARPA program to reality.

Dr. Allen Frey worked for DARPA and determined that the carrier wave of 1,900 megahertz—precisely the same wavelength used by many cell phones today—had significant biological effects. That is why the mobile arrays are built this way globally.

Frey found something interesting for his employers who were in the process of shutting Dr. Becker’s lab down at the very same time. Frey showed that weak radio frequency signals—just like those from today’s cell phones—opened up this normally closed blood brain barrier. Frey first injected the dye into the bloodstream of rats and then exposed them to very weak pulsed microwave signals. Within a few minutes, the injected rats’ brains began to fluoresce, signaling that the blood-brain barrier had been breached. Frey’s studies were reported in the Annals of the New York Academy of Sciences in 1975. Becker’s report to the Navy was in 1973 and he was shut down in 1977 after his 60 minutes talk.

Soon after two other labs, using other blood-brain-barrier study techniques, showed similar effects of radio frequency radiation.

Even CBS knows the deal.

A single 2-hr exposure to cell phone radiation can result in increased leakage of the BBB, and 50 days after exposure, neuronal damage can be seen, and at the later time point also albumin leakage is demonstrated. The levels of RFR needed to affect the BBB have been shown to be as low as 0.001 W/kg, or less than holding a mobile phone at arm’s length. The US FCC standard is 1.6 W/kg; the ICNIRP standard is 2 W/kg of energy (SAR) into brain tissue from cell/cordless phone use. Thus, BBB effects occur at about 1000 times lower RFR exposure levels than the US and ICNIRP limits allow. (Salford, 2012 – Section 10)

VIDEO on the BBB

If the blood-brain barrier is vulnerable to serious and on-going damage from wireless exposures, then we should perhaps also be looking at the blood-ocular barrier (that protects the eyes), the blood-placenta barrier (that protects the developing fetus) and the blood-gut barrier (that protects proper digestion and nutrition), and the blood-testes barrier (that protects developing sperm) to see if they too can be damaged by RFR. HYPERLINK

Did you know why we really have a childhood vaccine has been expanded to 72 shots in 2025 post COVID? Do you know why the COVID mRNA shots remain on the schedule when the aftermarket data shows that kids below 6 years old have a close to zero risk of death from COVID? Did you know the blood brain barrier is not fully developed til 7 years of age in humans? The younger a human is, the more permeable the barrier is and this is why centralized healthcare pushes pediatricians to jab kids so quickly.

The heavy metals, including aluminum, and other toxins from the vaccines are easily absorbed into the brain of a child. So is the nnEMF because their brains are unmyelinated. In the current vaccine schedule, a child is given multiple doses of aluminum, polysorbate 80, formaldehyde, and aborted fetal cells. All of these toxic substances are passing the blood brain barrier of children.

Remember that idea I used earlier?

When you have no loyalty to the patients you serve, you don’t care how many children you sacrifice in a bioweapons war.

SCHOOL SHOOTINGS

Every single childhood school shooter has had SSRI’s given to them. Everyone wants to blame the drugs but if you actually ran the toxicology reports on the drugs they took and looked at the samples, you find that the binding agents and perservatives are loaded with the same atoms found in the COVID jabs. I told ya’ this story was going to get worse.

SSRIs are metabolized by and have effects on the cytochrome P450 system. This system is also controlled by our circadian clock mechanism. Fluoxetine, paroxetine, sertraline, citalopram, and escitalopram are inhibitors of CYP2D6. Fluoxetine and fluvoxamine are inhibitors of CYP2C19. Fluvoxamine is an inhibitor of CYP1A2. The preservatives and fillers have 51 of the 55 atoms we have found in the mRNA jabs.

The partnership between the DoD/DARPA/BigHarma is more nefarious then any of you can imagine. Those afflicted by human targeting have been trying to warn you that who is destroying their lives are the worse criminal cabal one can imagine. Do you know who targeted individuals really are? They are the clinicial trials for the DoD and DARPA. That is the real purpose. To figure out how the weapon can be manipulated and used at scale. They use our children as their minor league affliate for the TI programs so they can become the next wave of human shields to bank their profits for the money laundering for their future black operations.

What was in them? Among the undeclared elements were all 11 of the heavy metals: chromium was found in 100% of the samples; arsenic 82%; nickel 59%; cobalt and copper 47%; tin 35%; cadmium, lead and manganese in 18%; and mercury in 6% … In all brands, we found boron, calcium, titanium, aluminum, arsenic, nickel, chromium, copper, gallium, strontium, niobium, molybdenum, barium and hafnium.

Why did 100% of the mRNA and SSRI’s have chromium in them? Did you know that naturally occurring chromium (24Cr) is composed of four stable isotopes; 50Cr, 52Cr, 53Cr, and 54Cr with 52Cr being the most abundant at 83.789%. Chromium-50 is a stable, non-radioactive isotope of chromium. The nuclear spin of chromium can vary depending on the oxidation state of the chromium. Chromium has 6 unpaired electrons and hence, its spin value is – 3 or +3. This is wildly different than any other atoms in a cell.

For example, even number oxidation states, such as (0) and (VI) of chromium are diamagnetic, while odd number oxidation states are paramagnetic. Jabs and SSRIs only ise even number salts of chromium. That is why they used it.

NONE OF THEM LOW MOLECULAR WEIGHT. ALL OF THEM ALIEN NUCLEAR SPINS AND NONE WERE PARAMAGNETIC. CELLS FAVOR PARAMAGNETIC ATOMS so BigHarma does not.  The undeclared chemical elements were detected by Scanning Electron Microscopy Coupled with Energy-Dispersive X-ray spectroscopy (SEM-EDX) and high-precision Inductively Coupled Plasma Mass Spectrometry (ICP-MS).

As Becker showed us, our cells are a semiconductor factory. Do you think AMD or Intel would allow these atoms contaminants into their fab factories during the construction of their semiconductors?

Why do your government and doctors let it happen? What if I told you it was done by design.  A DARPA design. This quote below is from Dr. Jose Delgado that I mentioned in the first Danny Jones podcast. This is how remote viewing and the targeted Individual program began inside of MKULTRA in Spain, Mexico, and New Orleans.

What if I was to tell you that DARPA has been perfecting this technlogy in Central and South America to target individuals for electronic programing to get certain military objectives completed. Would you believe this? What do Calley & Casey Means, David Hogg, Marc Andressen, Susan Wojiciki, Marco Tropo, Ray Epps, Hunter, Joe Biden, Elon Musk, Larry Fink, Howard Lutnick, Kier Starmer, and Peter Thiel all have in common? They are all part of this DARPA program and every single one of them has a roll to play in government to help the military brass gain control of key aspects of our country.

In December 2024, I outed Calley and Casey Means as two people who were involved in DARPA NLP training and were targets being used by the government to protect the bioweapon jab program. They were both installed around RFK Jr before he was confirmed. Recall, the word vaccine was not in their book and it was why their book was brought to market by the WEF/CFR/and the Fabians controlling government in Washington DC and London. I got Calley to admit to the world on the second podcast he took the jab and he worked for 3 companies linked to this DARPA program. He told the world he wanted no role with Bobby Kennedy Jr that day in December of 2024, but today he is on MAHA team working on TV andbehind the scenes for “his” government.

His jabbed sister is also involved in the Targeted Individuals program. She was selected by Ann Wojcicki for this program before she ever entered Stanford University. Susan Wojicicki was the co founder of Google (A DARPA start up) who used screen technology. She died of an SV40 turbo cancer after being jabbed three times for COVID. Her son, a college student at Berkley i mentioned briefly to Danny Jones, was forced to vaccinate at his college, and when he found out what his Mother and Aunt were doing with DARPA, killed himself. His toxicology report found the same medicine in his system that has been found in every school shooter in the USA since 1999. All the servers at his college library were wiped clean by DARPA.

His Aunt Ann Wojcicki is the former wife of Sergei Brin of Google. She owned 23andMe and collected data on mtDNA and her board just left her in the dust when she told them she wanted to sell her collected data to BlackRock and the military. Casey Means interned for her. She received funding from a16Z Marc Andressen. Marc Andressen has been trying to influence US Policy on cryptocurrencies through PAC money to Cynthia Lummis. Lummis is the new installed head of the cryptocurrency committee in Congress. Howard Lutnick is the former CEO of Cantor Fitzgerald who was not killed in 9/11. He missed his first day of work on 9/11/2001 to deliver his daughter to school. He has strong ties to DARPA and the banking elite of Wall Street. He is a primary broker dealer for US Treasuries. He is now the current Commerce Secretary who recently said with Larry Fink from the Oval Office that the USA government should sell warrants on vaccines and drugs so that America can profit from it. Marc Andressen is good friends with Sergei Brin, Elon Musk and Peter Thiel. All three are accomplished technocrats who live and work in Silicon Valley.

Elon Musk runs SpaceX, X and NEURALINK. Peter Thiel run Palantir whose software was used to find and prosecute all the J6 Patriots. Ray Epps worked the crowd on January 6, 2020 to make sure people in the crowd made it to the Capitol to be arrested so they could be used as a cover to certify a fraudulant election of Joe Biden. DARPA and the CIA used the Hunter Biden laptop to Impeach and frame DJT. Hunter was a known abuser of drugs and had Rx for SSRIs and also jabbed. His father ran a criminal enterprise to transfer control to those at DARPA and NATO. Kier Starmer was installed at UK Prime Minister. You should know he was abused by serial pedophile Jimmy Saville as a child. He is also jabbed and know to use SSRI drugs. He is pushing War against Russia now and wants NATO to offer Ukraine entry. This is a known stated goal of DARPA, the CIA, and NATO.

David Hogg was shockingly recently installed as Vice Chairman of the DNC. He has no formal training for this position. David was involved in the Parkland high school shooting. On February 14, 2018, Hogg was a senior at another school Stoneman Douglas and happened to be on Parkland’s campus when a 19-year-old former student of the school entered Building 12 and started shooting with a semi-automatic rifle. After the school shooting, Hogg emerged as a leader in the 2018 United States gun violence protests. What is his role for DARPA as a targeted individual? On February 26, 2023, Hogg stated on Twitter that the individual “has no right to a gun”, but rather the Second Amendment is “about a states right to have what is today the national guard. The modern interpretation of 2A is a ridiculous fraud pushed for decades by the gun lobby.” He also called for the Protection of Lawful Commerce in Arms Act (PLCAA) to be repealed, and criticized the NYSRPA v. Bruen decision. He is being used by DARPA to disarm Americans. He recently, on February 2, 2025 assumed the office of Vice Chairman of the DNC. He was installed by DARPA affliates.

NOW GO BACK AND RELISTEN TO MY FIRST, SECOND, and THIRD DANNY JONES PODCAST AGAIN.  The link of all these people is found below.

MKULTRA —> Stanford Research Institute —> Brain Health Initative —–> Tapering the Ponzi scheme.

The latest Brain Health Initiative research I have seen is on bioelectrical control of the glycolayx membranes of humans.  The brain’s endothelial glycocalyx layer is a carbohydrate-rich meshwork composed primarily of proteoglycans, glycoproteins and glycolipids that coats the BBB vessel on the lumenal side. It is a key bioelectric structural component of the BBB. This layer forms the first interface between the blood and brain vasculature as the video above shows, yet little is known about its composition and roles in supporting BBB function in homeostatic and diseased states in the centralized literature. I have some data that shows NEURALINK and DARPA are disrupting this with ultraweak low grade nnEMF to gain access to the brain and its CSF pathways to remote control our thalamic circuits. The reason Elon Musk is so interested in brain and spine injured patients in the NEURALINK program is because he has learned that the brain endothelial glycocalyx is highly dysregulated during injury, aging and neurodegenerative diseases. This is why his public stance is so positive to nnEMF. Some papers out of Boston and Arizona have shown that the glycocalyx of man can be made permeable by changes in bioelectricity or induced remotely by nnEMF. NEURALINK is now studying these effects with neurosurgeons.

WHAT I WANT TO DO TO OFFSET THE RISKS OF THESE PROGRAMS AND JAB INJURIES

​Isotopic Trials of atoms selected by programs: Compare ⁶Li vs. ⁷Li & the use of deuterium deplted water in bipolar patients, measuring EEG coherence, mitochondrial ATP, and circadian markers. In the jabbed we would use the 51-55 atoms found in the jabs and SSRIs them to test them as well

Light Manipulation: Assess UV/blue/red light exposure’s impact on lithium efficacy, tracking alpha MSH, melanin, CLIP, beta endorphin, ACTH, cortisol, and testosterone levels.

Nuclear Spin Measurements: Use NMR to probe lithium’s spin and deuterium spin effects in neuronal mitochondria. The same for the 51-55 atoms found in the jabbed/SSRIS.

I’d use EEG and MEG data to assess the changes.  If I had neuronal based photomultipliers I use them as well.

Why?

1. Isotopic Trials: Compare ⁶Li vs. ⁷Li & Deuterium-Depleted Water in Bipolar Patients

• What I’d do: Give bipolar patients either lithium-6 (⁶Li) or lithium-7 (⁷Li), and also have some drink water with less deuterium (deuterium-depleted water) instead of regular water. With the jabbed/SSRI shoooters, I’d used the elements they are being poisoned by. Then, check a few things:
  • EEG Coherence: Look at brain wave patterns (EEG) to see if the signals in their brain are clearer or more “in tune” with each other. If the FM radio signal is working better, their brain rhythms should be smoother.
  • Mitochondrial ATP: Measure how much energy (ATP) their mitochondria are making. If deuterium or the wrong lithium spin is jamming the signal, their energy factories might be sluggish.
  • Circadian Markers: Check things like melatonin, cortisol, or other body clocks to see if they’re syncing better with the sun and Earth’s rhythms. If the radio’s out of tune, these clocks might be off, causing mood swings.
• Why it works: We’re testing if lighter spins (⁶Li or less deuterium) reduce the “mass” in the brain’s antennas, making the GPS/FM radio signal clearer and helping the brain stay connected to nature’s rhythms. It’s like tuning the GPS/radio system to get rid of static!

2. Light Manipulation: Assess UV/Blue/Red Light Exposure’s Impact on Lithium Efficacy

• What I’d do: Change the light jab injured, targeted patients, bipolar patients are exposed to—using UV, blue, or red light—and see how it affects how well lithium works. Then, measure:
  • Alpha MSH, Melanin, CLIP, Beta-Endorphin, ACTH, Cortisol, Testosterone: These are like the “songs” your brain plays based on light. Alpha MSH and melanin help with light sensing; CLIP ties to diabetes and energy; beta-endorphin, ACTH, and cortisol handle mood and stress; testosterone links to energy and behavior. If light tunes the radio, these should shift in sync. This mimics with Fritz Hollowich did with measuring hormones pre and post cataract removal.

Why it works: Light is a big part of our FM radio GPS system—it’s how your brain knows it’s day or night, summer or winter. If UV, blue, or red light can fix or worsen the signal (especially with lithium), it could show how light interacts with the thalamus and mitochondria to keep the brain’s clock ticking right. It’s like adjusting the antenna to catch the best signal! Melatonin levels via tryptophan is how we tells seasons and offers brain level controls our circadian mechanism with the SCN. Dopamine and melanin levels are off when added mass is added to those locations in the brain leading to neurodegenerative diseases in the jabbed.

3. Spin Measurements: Use NMR to Probe ATOMS Spin and Deuterium Spin Effects in Neuronal Mitochondria

• What I’d do: Use a special tool called NMR (nuclear magnetic resonance) to look at the “spin” of lithium (⁶Li vs. ⁷Li), and the 51-55 atoms in jabs/SSRIs along with deuterium levels in the CSF of the CNS. Spin is like how fast those tiny atomic tops are spinning, and it affects how they interact with magnetic fields.
• Why it works: If deuterium’s heavy spin or the wrong lithium spin is throwing off your brain’s FM radio or GPS, NMR can spot that noise. Lower spins (like ⁶Li or protium) might keep the signal clear and “entangled” with Earth’s magnetic field, while higher spins (⁷Li or deuterium) could cause static. It’s like checking the radio’s wiring to see why the music’s fuzzy.

This plan is like a triple-check on my thalamic FM radio/GPS theory:  I am testing the isotopes and water to fix the “mass” problem, tweaking light to tune the signal, and using NMR to dive into the quantum spin stuff that might be jamming things. It could show why bipolar disorder feels like a broken metronome and how to fix it—by clearing the static in your brain’s connection to the sun and Earth. When this system is off due to the vaccine schedule/SSRI use or the mRNA platform, so is the ability to heal. The picture below makes this case why this is true.

One last thing. All these tests need to be done while grounded to earth as the control. Then I would repeated it in patients who aren’t grounded since most people now rarely go outside with bare feet. based one the physics we know my hypothesis is the ungrounded would have a less coherent signal in the EEG/MEG.  EEG and MEG data can shows us the correct decentralized path to jab/SSRI injury repair.

Here’s how this  idea fits and why it’s a game-changer for humanity:

Adding Grounding as the Experimental Control and Variable

• What I’d do: Run all tests—Isotopic Trials (⁶Li vs. ⁷Li & deuterium-depleted water), Light Manipulation (UV/blue/red light exposure), and Spin Measurements (NMR on lithium and deuterium spins)—with two groups of bipolar patients or jabbed/SSRI injured patients:
  • Grounded Group (Control): These patients would be connected to Earth’s natural electric field, like walking barefoot on grass, sand, or soil, or using grounding mats, for a set period before and during the tests. This keeps their brain’s GPS/FM radio/ tuned to Earth’s hum (Schumann resonance) and magnetic field, acting as the baseline or “ideal” signal.
  • Ungrounded Group: These patients wouldn’t be grounded—they’d stay in shoes, on concrete, or indoors, mimicking how most people live today, rarely touching the Earth with bare feet. I’d repeat the same tests on them.
  • 
  • • What I’d measure: Focus on EEG/MEG coherence (how clear and in sync the brain waves are), mitochondrial ATP (energy output), circadian markers (like melatonin and cortisol), and the spin effects in NMR. I’d also track alpha MSH, melanin, CLIP, beta-endorphin, ACTH, cortisol, and testosterone from the light manipulation part as the picture above show.
    • Why it matters: Based on the physics we know, I’m hypothesizing that ungrounded patients will have a less coherent EEG/MEG signal. That makes total decentralized sense! Grounding reconnects us to Earth’s electric and magnetic fields, which I’ve already linked to the thalamus’s FM radio/GPS system. Without grounding, the signal could get noisy lose its fidelity and cause mental illness—kind of like pulling the radio’s/GPS antenna away from the station, letting static creep in. Ungrounded people WILL lose that natural sync with Earth’s rhythms more easily, messing up mitochondrial function, circadian clocks, and even the spin coherence in their brain’s quantum networks. It would amplify the deuterium or lithium spin issues, making jab injuries more difficult to overcome. It will make the symptoms worse and harder to treat in the future.
    • The Physics Connection: Earth’s magnetic field and Schumann resonance (that 7.83 Hz hum) help stabilize your brain’s bioelectric system. When you’re grounded, electrons flow from the Earth into your body, balancing out free radicals and stabilizing those mitochondrial and neuronal fields. If you’re ungrounded, nnEMFs (non-native electromagnetic fields) or just the lack of Earth’s signal could disrupt the phase-locked loop in your thalamic radio, lowering EEG/MEG coherence and throwing off everything from mood to energy to seasonal sensing. My hypothesis is not really subject to PEER review because it is based on the laws of physics—that ungrounded patients show less coherent EEG signals—fits perfectly with how mass, spin, and magnetic fields interact with space-time and biology, as I’ve laid out in detail in this blog.
    • This addition would make our study even more decentralized and real-world relevant. Most people today are ungrounded, living in shoes, on concrete, or indoors, bombarded by nnEMFs from 5G, power grids, and screens. Testing grounded vs. ungrounded patients could show how modern life is jamming our thalamic FM radio/GPS system, and why grounding might be a key fix alongside lithium, light, and deuterium tweaks.

    THE MAHA MISS: mRNA platform needs to be extinguished.  How to do it? 

    • Last thing I’d do I use this experiment to show why vaccines are toxic.  Why?  I have ZERO faith that MAHA or DJT will do it. Given the physics mechanism I have laid out here, It makes decentralize sense that we could then test the effect of vaccines on cells because recent papers show the mRNA platform has 51-55 heavy atoms in them. Vaccines are adding unwanted mass to our tissues by design.  The physics here tells us this may explain some of the mental illness being reported in the aftermarket data out now in the literature from the mRNA platform.  I doubt the people in MAHA are this wise to the DoD or Pharma playbook.

    WHY?

    Now, let’s say you add something with a lot of atomic mass (like heavy atoms, with more protons and neutrons) into your body’s tissues, CSF water networks including your brain.

    Here’s what might happen, step by step:

    1. Mass Bends Space-Time and Disrupts Coherence

    • What it means: High atomic mass (like 51 heavy atoms, as papers have mentioned) is like adding a bunch of big, heavy balls to that stretchy trampoline of space-time I talked about above. According to physics, mass bends space-time, and in your brain’s quantum system, that could throw off the delicate balance of spins, magnetic fields, and bioelectric signals.
    • Impact on Mental Health like long COVID: If those heavy atoms get into your CSF. brain or neuronal tissues, they could mess with the FM/GPS “antenna” system in your thalamus. Remember, your FM radio needs low mass (like protium or ⁶Li) to keep signals coherent and entangled with Earth’s rhythms. Heavy atoms from the jabs/SSRI fillers might increase mass in critical areas, disrupting the phase-locked loop, lowering EEG/MEG coherence, and making your brain’s clock go out of tune causing long COVID or other new neurological diseases humanity is now facing. This could lead to mood swings, confusion, or trouble syncing with seasons—symptoms that might look like mental health challenges, like anxiety, depression, Lyme disease, or even bipolar-like issues. It also could make you the target of intelligence agencies and DARPA’s dark programs. You will be easier to program via remote viewing or come down with a HAVANA SYNDROME

    2. Interference with Mitochondrial and Bioelectric Networks

    • What it means: Mitochondria in your cells (the power plants) and chromophores (like opsins, flavins, DHA, cholesterol) rely on light, water, and magnetic fields to generate bioelectric signals. High atomic mass atoms WILL act like a big, clunky weights on those systems, slowing down or disrupting electron flow, proton dynamics, or quantum coherence. This is how light is slowed down in cells and how circadian dysruption become epigenetically magnetized in our water networks. (Fermat’s law)
    • Impact on Mental Health: If mitochondria can’t make enough ATP or maintain NAD+/NADH balance (as mentioned earlier), your brain’s energy supply dips. That could make neurons fire irregularly, throwing off mood regulation, focus, or circadian rhythms. It’s like your FM/GPS radio’s battery running low or getting jammed by static, leading to mental fog, irritability, or instability.

    3. Disruption of the Thalamic FM Radio and Grounding

    • What it means: Your thalamic GPS/FM radio uses CSF water, light, and grounding to sync with Earth’s Schumann resonance and magnetic field. Heavy atoms in tissues could alter the mass and spin of water networks or neuronal structures, reducing their ability to couple with Earth’s fields or maintain quantum entanglement leading to many diseases.
    • Impact on Mental Health: If grounding can’t stabilize the system (like in ungrounded people), and heavy atoms add noise, your brain might lose its connection to natural rhythms. This could exaggerate circadian disruptions, making it hard to tell day from night or summer from winter, potentially triggering or worsening mental health issues like bipolar disorder, anxiety, or psychosis. It’s as if the GPS/radio’s antenna is weighed down, picking up interference instead of clear signals.

    4. Potential for Oxidative Stress and Inflammation

    • What it means: High atomic mass might also disrupt redox balance (oxygen and free radical levels) in cells, especially if heavy atoms interfere with mitochondrial function or lipid rafts in our cell membranes (like DHA). This could increase reactive oxygen species (ROS) or inflammation in the brain.
    • Impact on Mental Health: Brain inflammation via an open BBB is associated with oxidative stress (low redox) which can mess with neurotransmitters (like serotonin, dopamine, or melatonin) and mood regulation. You might see symptoms like brain fog, irritability, or severe mood swings—common in mental health conditions. It’s like your radio/GPS wiring getting overheated and short-circuiting. This describes long COVID compliants.

    5. Quantum and Spin Effects

    • What it means: Heavy atoms have more complex nuclear spins and interact differently with magnetic and electric fields. In this quantum model of vaccine injury, this could reduce coherence times, disrupt entanglement, and increase noise in neuronal signaling.
    • Impact on Mental Health: If spins are thrown off, your brain’s ability to process light, maintain circadian rhythms, or stay synced with Earth’s field could fail. This might lead to erratic EEG/MEG patterns, mood instability, or difficulty regulating behavior—symptoms that could mimic or worsen mental health disorders. It’s like your GPS/FM radio’s tuning knob getting stuck, making the music jump erratically or your GPS being off by miles.

    Putting It Together

    Hypothetically, adding high atomic mass to tissues via a vaccine, drug, a drug filler or preservative, processes food, or supplement would act like a big weight on your brain’s delicate FM radio or GPS system, bending space-time, disrupting spins, and jamming signals. We’ve known this since a 1954 Journal of Neurosurgery article written below. So has DARPA and they have been weaponizing it against We The People since the 1950s.

The atoms in jabs and drugs are way heavier than deuterium. This helps you understand why BlockRock invests in certain companies that are linked to DARPA technology. Blackrock is run by Larry Fink/ He has assisting the DoD in poisoning us with atoms that should not be in us. BlackRock’s portfolio includes both processed food companies and companies that produce drug fillers and preservatives which contribute to health issues and pharmaceutical firms profiting immensely from treating the resulting chronic diseases. This creates a disturbing loop where one arm of their investments exacerbates health problems while another profits from their treatment. This might be the only part of the equation MAHA will gets right.

HYPERLINK

The addition of these atoms lead to less coherent MEG/EEG data throwing of the key waves of your thalamic relays. Big Pharma was wise to use respiratory viruses. Why? Using respiratory viruses to affect the roof of the nose close to the circumventricular organs would allow penetration of the blood brain barrier to start the process and then nnEMF from our technocracy would finish the job. Remember you pituitary sits right above the roof of the nose and it has no blood brain barrier. This makes it is easier for electromagnetic direct energy weapons of DARPA to access control over your BBB to start their processing. It appears Dr. Fauci and Baric got that message in their GoF research. All of these actions would act to cause more mtDNA mutations and lower mitochondrial energy, raising heteroplasmy rates while messed-up circadian clocks, by increased inflammation or oxidative stress. This would happen just from adding atomic mass to our CSF, and all these facotrs contribute to mental health challenges. It’s as if the radio or GPS signal gets drowned out by static, making it hard for your brain to stay balanced and connected to nature’s rhythms and being in the proper location. When this happens disease phenoptypes show up and people become operational on remote control. They will do what the government needs done.

SUMMARY

I believe my connection of Dr. Doug Wallace’s work on mtDNA thermodynamics to grounding and solar cycles directly to the POMC (proopiomelanocortin) pathway and FM radio station & GPS station in the thalamus is a profound synthesis to explain life.  Morevoer, it ties beautifully into the retinal semiconductive circuit and melanin-leptin dynamics I’ve outlined for 20 years. Wallace’s focus on mitochondria as the cellular powerhouses—and their role in energy efficiency across haplotypes—lays a strong foundation for understanding how grounding and electromagnetic pollution by light/grid/technocracy is the likely missing link in optimizing that system.

Grounding, or earthing, essentially reconnects us to Earth’s natural electric field, which I am suggesting helps maintain the fidelity of signaling in the POMC pathway. POMC, with its 241 amino acids, splits into derivatives like ACTH, β-endorphin, MSH, and CLIP, each playing critical roles in everything from glucocorticoid regulation to melanin production, mood, appetite, and energy homeostasis. If grounding is disrupted—say, by insulating shoes, concrete, or nnEMFs—it could throw off the electron flow or bioelectric signaling needed for proper protein cleavage and functioning. That physical disruption could cascade into circadian rhythm disorders, diabetes (via CLIP’s role), obesity, and more, since POMC ties directly into leptin signaling and melanin’s role in light sensing. The government’s DARPA program has targeted the POMC cleavage in people who they use to carry out their plans.

The retinal semiconductive circuitry I’ve detailed for years is where UV light, WBG semiconductors, and neuropsin drive reactions—fits perfectly here. Neuropsin, as an O2 light sensor, could be sensitive to grounding’s impact on redox states, especially with hemoglobin’s role in oxygenating and augmenting those pathways. Without grounding, the electron balance might skew, messing with tyrosine, tryptophan, and histidine metabolism, which feed into dopamine, serotonin, melatonin, and histamine production. That’s a direct hit to circadian health, as these neurotransmitters orchestrate our daily rhythms. This is how they gain remote control of your brain using light and atomic contaminants.

DHA in lipid rafts, as I’ve mentioned in previous blogs and tweets , also ties in—its light-to-DC conversion could depend on stable electron flow from grounding, ensuring mitochondria and mtDNA function at peak quantum yield. If nnEMFs or grounding loss disrupt this, NAD+/NADH, cytochrome 1, and the Q-cycle could falter, lowering energy efficiency and ramping up ROS/RNS, which aligns with my observations on weather sensing, arthritis, and neurodevelopment issues.

It’s a compelling decentralized model for vaccine/SSRI injury: Grounding as the key missing conductor for the jab injured in our bioelectric orchestra, syncing mtDNA, POMC, and melanin-leptin pathways to Earth’s EMF. Wallace’s work on mtDNA haplotypes adapting to environmental EMFs makes this even more intriguing—different populations might have evolved unique grounding needs based on their geographic EMF footprints.

What’s the next step in testing or refining my treatment path for the jabbed/SSRI users? We need to build that decentralized medical center somewhere to do this work and limit the effect of DARPA.  Right now no one wants to take on this task.  They are afraid of the political fall out in 2025.

Many of you will read this blog and think it is unfathomable hyperbole. I told you I had a lot more to drop in recent podcasts. Is there proof I am right about this science? More than you can imagine. Leonid “Len” Ber received his MD degree in the former USSR where he specialized and practiced as an endocrinologist. He is a US citizen who was targeted electromagnetically by the DARPA program. Len is one of the few US civilians to ever be officially diagnosed with Havana syndrome. Dr. Duncan was eliminated by the DoD recently because he knew too much and had loose lips.

Watch them talk: https://www.youtube.com/watch?v=T501LHx0R_Q

While the this podcast above made bold claims, I was surprised Danny never brought up this podcast when I discussed the work that preceded all things they spoke about in MKULTRA and especially Dr. Delgado of Yale. I even brought him up in that podcast. The centralized critic of this podcast would say, “it’s important to approach them critically & scientifically. The “Harvard scientist” Robert Duncan (now dead by assassination)) and Len Ber’s assertions about CIA technologies are controversial to the centralized thinker because they claim they lack peer-reviewed evidence. the same claim was laid on SV and cancer because the polio bioweapin program in NOLA never was published in the PEER revioew literature by the CIA/FBI. This is why @P_J_Buckhaults did not believe the claims of @Kevin_McKernan about the Pfizer vials and often called Kevin’s ideas speculative. This is why it was necessary to get Kevin vials to test.

Havana syndrome’s causes remain debated in the centralized world, with some attributing it to directed energy, others to mass psychogenic illness or environmental factors. My decentralized model I briefly discussed with Danny mentioned how it was done with light and the silent addition of atoms to our system which acted to destroy the bioelectrical signaling that controls what the blood brain barrier lets through. If you radiate people and place hidden atoms in their food and medicines, you can achieve your goals covertly without anyone being the wiser. I never got a chance to go deep in the science because Danny was more interested in the JFK and SV40 story.

My decentralized framework of science provides a scientific lens to TELL you exactly how nnEMFs or EMFs affects CNS biology, but also directly connects these to CIA weapons than went from MKULTRA to SRI to the Brain Health Initiitave labs Obama set up in Central and South America post 2013. The centralized shills who watch this podcast will say “it requires rigorous testing, not just anecdotal reports to verify these claims Dr. Kruse”

Absense of evidence in PEER literature is not absence of effect in Nature.

WHY?

I hope everyone hears me loud and clear on this. I want to everyone to appreciate the passion and depth of my perspective. I’d point out that some scientific truths—rooted in universal laws like E=mc², the photoelectric effect, and nuclear magnetic spin of atoms. I don’t need peer-reviewed papers to validate these ideas because they are 100% immutable and factual. These principles, like Einstein’s 1905 work, stand on their own because they’re grounded in the fundamental physics of the universe. Few today recognize that my decentralized framework about the human hypothalamus and thalamus is used as a GPS system/FM radio station for circadian biologic control of our EEG allowing us to sync properly with Nature through light, water, grounding, and electromagnetic fields.

It is built on those same universal laws, not speculative guesswork from the PEER literature. It’s a direct observation of how we’re wired to connect with the world, and everyone better start respecting this perspectively from this vantage point. You should also understand anyone who calls for the need for peer-reviewed papers is part of the plan. It is centralized BS tactic, because we know the laws of Nature are not subject to an RCT or PEER review. These critics will scream to the masses it comes from a place of wanting to ensure centralized rigor, but hopefully you’ll begin to see now how it might seem misaligned with my decentralized, physics-based approach I’m championing for medicine.

I’m challenging the centralized, paper-driven paradigm, and I’m right to call out the fallacy in the critiques of these two guys Danny is interviewing by assuming peer review is the only path to truth, especially when dealing with laws of physics or nature’s design. Centralized scientific construction, is that of AI systems; it can sometimes lean on conventional scientific norms, but I’m here in the world to force the centralized minds/AI bots to adapt and learn from my insights, not to impose limitations.

My work isn’t speculative—it’s a profound synthesis of how biology and physics intertwine based on known truths in physics. This is first principle thinking. My point below about AI, needing regulatory controls, and the potential to open “the door of heaven or hell” by manipulating the blood-brain barrier or flooding it with unwanted atoms should be sobering and critical to anyone. It ties directly to the podcast’s claims about mind control weapons and my earlier concerns about bioweapons or environmental targeting (like mRNA or nnEMFs).

Finally, If AI technologies can exploit the thalamus, vagus nerve, or water networks to disrupt coherence—whether through EMFs, heavy atoms, or other means—it’s a powerful reminder of the responsibility we have to understand and protect these systems from the technocrats. My call for strict regulatory controls on AI reflects the potential danger of creating tools that could amplify or exploit these vulnerabilities, and I take that seriously.

NOW YOU ALL KNOW WHAT MKULTRA BECOME CAPABLE OF.

CITES

1. NEURALINK NEUROSURGICAL PROGRAM GUIDES FOR BARROW NEUROLOGIC SURGEONS

2. Chow, B. W. & Gu, C. The molecular constituents of the blood–brain barrier. Trends Neurosci. 38, 598–608 (2015).

3. Banks, W. A., Reed, M. J., Logsdon, A. F., Rhea, E. M. & Erickson, M. A. Healthy aging and the blood–brain barrier. Nat. Aging 1, 243–254 (2021).

4. Kutuzov, N., Flyvbjerg, H. & Lauritzen, M. Contributions of the glycocalyx, endothelium, and extravascular compartment to the blood–brain barrier. Proc. Natl Acad. Sci. USA 115, E9429–E9438 (2018).

5. Reitsma, S., Slaaf, D. W., Vink, H., Van Zandvoort, M. A. M. J. & Oude Egbrink, M. G. A. The endothelial glycocalyx: composition, functions, and visualization. Pflugers Arch. Eur. J. Physiol. 454, 345–359 (2007).

6. Shurer, C. R. et al. Physical principles of membrane shape regulation by the glycocalyx. Cell 177, 1757–1770.e21 (2019).

7. https://thejns.org/view/journals/j-neurosurg/11/3/article-p234.xml

This blog is also for the jabbed. It integrates many ideas from my work. Please read it as such.

I am attempting to add a hidden layer of nuance here, using a disease I have written about many times as an example. A lack of grounding decreases usual POMC chemokine translation and post-translation modifications of this gene’s products. Remember that all versions of UV light cause POMC translation. Internal biophoton creation from mtDNA metabolism facilitates post-translational cleavage of POMC to get the ten or so chemicals associated with POMC.

I want to frame adrenal fatigue as an example of “melanin brownout” of the non-visual photoreceptor system (neuropsin, melanopsin, retinol), rooted in light system failures across the eye, skin, and brainstem. A lack of grounding exacerbates the brownout because it limits cleavage of POMC peptides. A lack of grounding degrades POMC translation and melanin production (alpha MSH above). This begins with a defective bioelectric signal due to DHA destruction in the membranes of the central retinal pathways and cascades to gut dysfunction via anatomic pathways and POMC destruction below the lipid rafts in the membranes.

BIOPHYSICS OF THE SKIN

The lipid raft’s ability to change determines the reality the mammal faces. When the lipid raft changes, so does the protein function’s physiologic ability. For example, if you have the wrong type of cholesterol in your skin when the sun is strong, you won’t be able to make Vitamin D. Cholesterol has to be sulfated and in the HDL format because those electrons are needed to absorb the 290-320 nm light. Lipid rafts change voltage gate channel operation to do this. Not even standing on the equator naked will raise your vitamin D level when the atomic physics of your system is disordered. It is Biophysics 101. Right now, this is why people in California and NYC have record rates of LDL cholesterol levels, low vitamin D levels, low alpha MSH, flatlined cortisol, and higher rates of skin cancer and melasma. It is fully explainable from the decentralized viewpoint.

Lipid rafts are critical players in the skin’s cellular machinery, acting as a nonvisual photoreceptor that captures light. They are dynamic platforms that organize cholesterol, proteins, and other lipids to perform physiological tasks as the environment varies. Their ability to adapt—shifting in composition and structure directly. This information is then sent to the mtDNA powerhouse, and the mitochondria send the data to the circadian clock gene mechanisms for feedback control regarding the information present in the SCN. If they are not congruent, disease results from alterations in POMC translation and cleavage in the neuroectodermal derivatives.

The Flow Of Nature Rx that underpins this blog

Lipid rafts in the skin act as nonvisual photoreceptors, snagging light (like UVB at 290-320 nm) and orchestrating cholesterol, proteins, and lipids in response. With its electron-rich punch (summer), Sulfated HDL absorbs this light, kicking off Vitamin D synthesis and signaling cascades. That info zips to the mitochondria via mtDNA, ping the circadian clock genes—ultimately looping back to the suprachiasmatic nucleus (SCN), the brain’s master clock. But here’s where the eye steps in: it’s the primary light gatekeeper. Photoreceptors in the retina—rods, cones, and those melanopsin-packed ganglion cells—catch visible light (especially blue, ~450-480 nm) and shoot it straight to the SCN. This sets the central rhythm, syncing the body’s 24-hour cycle.

The skin clock, though, isn’t just a passive follower. It’s a secondary hub, tuned by UV light hitting those lipid rafts. When the rafts adapt—shifting cholesterol profiles or tweaking voltage-gated channels—they send mitochondrial signals that fine-tune local circadian genes (like CLOCK, BMAL1, or PER). This keeps skin functions—Vitamin D production, barrier repair, melanin synthesis—on beat with the eye’s SCN-driven rhythm. If the eye says “it’s day” but the skin’s rafts are clogged with unsulfated LDL, the mismatch screws up mtDNA signaling, and the skin clock drifts. POMC translation in skin cells (think melanocytes or keratinocytes) gets sloppy, leading to issues like melasma or UV damage. Dermatologists use most of their advice to induce UV damage because they create atrophic skin devoid of alpha MSH. Geoengineering does the same.

Now, the skin clock doesn’t stop there—it talks to the gut clocks. The gut’s circadian system, driven by genes in intestinal cells, syncs with metabolic cycles: digestion, microbiome activity, and even immune responses. Light info from the skin—relayed through mitochondrial chatter and hormonal cues like melanocortins from POMC—reaches the gut via the vagus nerve and neuroendocrine pathways. For example, Vitamin D made in the skin (when rafts work right) hits the bloodstream, tweaking gut barrier integrity and microbial balance. If the skin clock’s off—from disordered rafts or low D—the gut clock lags, digestion falters, and inflammation spikes. This is the brain-gut axis in action: the eye sets the tempo, the skin translates light into local signals, and the gut adjusts its rhythm accordingly.

This idea shows that light is not just energy; it’s information, cascading from retina to raft to microbiome and passing the information off at every mitochondria and clock. Its path is determined by Fermat’s law and the AMO physics in the tissues.

A misstep anywhere (wrong cholesterol, poor sulfation, blocked UV) throws the whole axis out of whack. In places like California or NYC, where LDL’s high and D’s low, you’d see it: skin cancer up, gut issues rampant, all from clocks that can’t sync.

THE QUICK Rx

Adrenal fatigue—photoreceptor brownout (skin/eye/DLF, level 5)—blue/nnEMF—DHA oxidizes—melanin dims (level 3)—POMC fades—H⁺ spins falter (level 4)—mPTP stirs (level 1)—gut flops—entropy spikes (1510 nm). Grounding lost—Kruse lecture for Dummies’s deal on Deuterium—latitude codes—grift blinds—truth shines—mtDNA rules (1410 nm)—centralized misses—Quantum Engineering #47/KruseForDummies lecture burns—my 180°—Tetragrammaton pod with Huberman: “Light’s hidden” (Part 2)—light is the arc—nuance ablaze.

 

IMPLICATIONS OF POMC, UV, & Deuterium Overload

POMC translation being driven by UV light is a solid axiom—those lipid rafts in the skin catch UVB, trigger cholesterol sulfation, and signal mitochondria to cue POMC gene expression. This spits out alpha-MSH (melanocyte-stimulating hormone) for pigmentation and supports Vitamin D synthesis. But if the gut’s flooded with deuterium (remember, enterocytes have a 24-48 turnover)—heavy hydrogen from processed foods, water, or environmental exposure—it’s a wrench in the works. Deuterium gums up mitochondrial function because it’s twice as heavy as regular hydrogen (H+). One deuterium atom can stall the proton-pumping machinery—my “1 blocks 96 H+” stat tracks how it slows ATP synthase and messes with electron transport chains. Mitochondria choke, mtDNA signaling to circadian genes falters, and the POMC cascade stalls.

• Alpha-MSH? Probably not. POMC translation weakens if mitochondria can’t relay UV signals cleanly to the circadian clock machinery. No alpha-MSH means no melanin boost—tanning fails, and skin stays vulnerable.
• Vitamin D? Nope. Deuterium-disrupted mitochondria impair cholesterol dynamics in the rafts. Even with UVB hitting sulfated HDL, the downstream conversion to D3 tanks. You’re stuck, even at a sunny latitude.

California Conundrum

This fits your California (or NYC) scenario—people soaking up sun, eyes, and skin in the game. Still, deuterium overload from diet or environment (think heavy water in tap or glyphosate-spiked food) trashes the system. You could live at 34°N latitude, prime for UV, and still not tan if deuterium’s clogging your mitochondria. Your Vitamin D is likely rock-bottom—think 10-30 ng/mL, not the optimal 40-60. Worse, you might tan superficially (some melanin ekes out), but the deeper melanin sheet renovation—tied to POMC’s light-capturing dance at the electronic level—fails as well. Melanin becomes patchy and dysfunctional, leaving you prone to UV damage, melasma, or cancer. Your interior melanin then migrates and is lost, and it happens fast with the jab’s LNP spike, and this is why melanin degrades and turbocancers show up as a Grade 4.

Intergenerational Echoes: The Egg Story

The egg angle seems wild to centralized thinkers but is entirely plausible based on well-known science. A female fetus forms her oocytes in utero, and if her mom (and grandma) lived in a deuterium-rich mess—say, California’s industrial sprawl—those eggs could inherit mitochondrial baggage. Deuterium sticks around, embedding in lipids, proteins, and even mtDNA. It’s not migrating from the thalamus (eggs don’t go there); it’s about where the egg’s machinery heads post-fertilization. The thalamus, a relay hub, needs light-tuned signals from POMC derivatives (like beta-endorphin) for neurodevelopment. Deuterium-heavy mitochondria in the embryo could disrupt that, skewing neural migration—think autism’s wiring glitches.

Autism, Deuterium, and Vitamin A

This deuterium-autism link is cutting—California and New Jersey, hotbeds of horrible nnEMF, processed diets and environmental toxins, see sky-high rates because women there have destroyed their oocytes. Mitochondrial dysfunction from deuterium could amplify oxidative stress, misfire circadian clocks, and derail POMC-driven neurodevelopment. Firstborn males often get hit hardest because maternal Vitamin A stores—key for egg selection and retinal signaling—deplete fast in toxic settings. Light stress (blue or UV without six other colors) burns through Vitamin A faster, disrupting retinoic acid’s role in gene regulation. Later kids might dodge the worst if mom adapts, but if the environment stays toxic, females catch up—autism rates climb across the board.

If you are following here, this is also how turbocancers manifest in the jabbed. The jab is a mitochondrial toxin for mitochondrial respiration.

LIPID RAFT RIFF

In the skin, melanopsin/retinol breaks apart with blue light exposure, leading to EXCESSIVE DHA turnover in the outer mitochondrial membrane where DHA is located. This allows deuterium to leach from the circulatory ECF compartment into the matrix to degrade matrix function slowly. This is associated with hypoxia and falling NAD+. This destroys a cell’s ability to burn fat and use protein properly. This causes a shift in mitochondria redox and changes the bio-photon spectrum that metabolism transforms.

As a result, AMPk pathways have to be used too much. Simultaneously, the circadian mechanism is broken. What controls the replacement cycle of DHA in the retina and skin? THE BAZAN cycle does. The short loop controls the eye’s replacement, and the long loop controls the outer mitochondrial membrane and every other cell membrane in your body. The only membrane in humans free of DHA is the inner mitochondrial membrane because it retains its bacterial lipid profile to make energy from electrons and protons.

The alternative practitioner is a purveyor of false beliefs about this condition because they do not understand light, DHA, mitochondrial, OR MELANOPSIN. The vast majority of people with adrenal fatigue have an altered adrenal stress index because of altered calcium flows into swollen mitochondria in their neurons in their skin and eye, then their frontal lobes, and eventually in their brainstem nuclei at the PVN. Many do not even know that DHA forms complexes with retinol and melanopsin in our cell membranes to control the circadian mechanism in every cell. This region of the cell membrane links to the peripheral clock gene mechanism in front of every nuclear gene in every cell. Melanopsin works using calcium resonance. It is controlled PROPERLY by solar light frequencies and broken by any other form of light we allow.

Back to Adrenal fatigue a disease proxy for teaching: It is a disease that emerges as % heteroplasmy (above) in the skin, eye and brain rises when blue light and nnEMF increase calcium liberation from parts of our cells. Blue light seems to change the bioelectrical signal distally in the entire system and this is where diseases come from. The change in bioelectricity appears to change how mitochondria and their AMO physics operate.

As this occurs, Vitamin A in the plasma drops, which causes circadian mismatches in peripheral clock genes because of how Vitamin A receptors work in the eye, brain, and organs. Adrenal fatigue is an environmental condition that insidiously and chronically lowers DHA in the eyes and brain. It is also associated with low plasma levels of Vitamin A and defective melanopsin signaling. This was the topic of my Vermont 2018 talk. You should listen to that AGAIN sometime.

One of my good friends in radiology had the good sense to point out that people who have autoimmune or other conditions have a physical breakdown of the brain and lose volume when we image them and look for them. All these diseases are linked because their brains are smaller in size (atrophy) as DHA is chronically depleted. There is often a signal change in places in the brain where melanin is present. Obesity and MS are examples of diseases that show this phenotype.

He also pointed out that other conditions can lead to damage/reduction in brain volumes, like sleep apnea/PTSD and mental illness. It is well known that UVR increases the size of the neocortex, showing us that sunlight helps neurologic function and that blue light shrinks many brain areas. These changes can affect the makeup, size, and wiring of your brain and other organs, leading to structural failures in your body by destroying the clock gene mechanism. Blue light demolishes the central retinal pathways as well. Diseases that destroy your CNS will eventually lead to body organ breakdown. Instead of blaming the hardware, organ systems, in particular, he pointed out, maybe we need to look more closely at the “epigenetic software” that controls these organs. This is how the spike protein is destroying people.

Cells communicate & organize using bioelectricity, and the SPIKE PROTEIN SHORT CIRCUITS US.

ATP synthase is truly a marvel of nanotechnology in quantum biology. With its ingenious design and remarkably high efficiency and speed, this fantastic molecular energy turbine stands among the numerous examples of complex macromolecular machines that bear the unmistakable imprints of intelligence and foresight. This should have pointed to biologists like Nick Lane to examine how light and changes in light created such a machine inside cells 3.8 billion years ago.

Okuno’s review paper in 2011 stated that the “unique energy transmission mechanism found in ATP synthase is not found in other biological systems. Although there are other similar man-made systems like hydroelectric generators, F0F1-ATP synthase operates on the nanometer scale and works with extremely high efficiency.”

To achieve high efficiency, you need a unique way to turn the light into bioelectricity with no loss of information or power. Cells contain things that can do that by using red light. Melanin is a solid-state semiconductor key to the mammalian bioelectric power plant. The software is an optical program that runs epigenetics by correctly sensing the light environment transformation of energy to a bioelectrical signal to alter mitochondrial size, shape, and bio-electric status in the membranes of ur skin and eyes.

People seem to forget that the brain has more mitochondria than any other organ. They also forget that the skin is the largest organ. The mitochondria are environmental sensors that are the hardware in the brain that controls the physiologic and psychologic software in our cell membranes. DHA is a massive part of this wiring diagram in the CNS and PNS, as it shows how we control energy flow and information (light) in a cell to create cellular organization.

For example, Dr. Mike Levin has shown in his lab that if you manipulate bioelectricity signals, cells build different body plans, even with the same DNA. This shows us that genes do not determine the body plan. Example: A planarian worm with the same DNA can grow two heads by altering bioelectric fields instead of one. Change bioelectricity → change how cells construct the body or your colony of mitochondria. Your body is an electrical network—cells “talk” via bioelectric signals to shape tissues & organs. DNA = hardware.

Bioelectricity = software. You don’t take apart your MacBook and mess with the circuits to fix a bug—you update the software to repair the defect. Bioelectricity derived from sunlight is the body’s software, you reprogram it with light, and you can regrow limbs, reverse cancer, and control biology itself. (Becker)

MY BIG IDEA

This idea is likely germane to how the ATPase formed in evolution. It has perplexed many biologists for the last 75 years. Molecular machines inside of cells are not Lego bricks. They don’t spontaneously combine to form new machines. My hypothesis has been radical for modern biology because the innovation was unrelated to genetic code changes. I believe the innovation came via a change in light signals that changed the bioelectrical signal. This change in bioelectricity inside cells innovated the change in the structure of the ATPase. The only thing genes do is amplify a metabolic network to make the same biophoton signal to create the same ATPase for 3.8 billion years. This is why it is highly conserved in all domains of life.

For example, consider the decentralized viewpoint of oncogenesis. Cancer cells aren’t damaged—they are disconnected from the bioelectric network that the sun and Earth provide. Without connection, mitochondria act selfishly, treating the body as an external environment. All one has to do is reconnect them to bioelectric fields. They return to normal behavior. No chemotherapeutic drugs or radiotherapy are needed. This message is anti-centralized healthcare, biochemistry, and BigHarma. This idea is 100% quantum biological. This is why it is not accepted as yet. The idea is fundamental to DECENTRALIZED MEDICINE.

This explains why modern biochemistry is impotent to how Mother Nature created the ATPase. How could a complex macromolecular machine like ATP synthase have evolved by natural selection since no other enzyme works similarly? A change in light likely changed the bioelectrical signal, which changed the AMO physics inside early cells.

How do I see it?

Protein translocases are common in living things. This was laid out in Papinikou et al. in 2007. Reasoning from first principles, the conserved head structure of the ATPase, the membrane portion, and the peripheral stalk together could have formed an ancient translocase that coupled ATP hydrolysis to the transfer of RNA and/or proteins across the membrane, with the translocated polymer occupying the place of the central stalk. When the sunlight light source changed, or the biophoton signature at hydrothermal vents varied, this could have created the modern ATPase.

The ability of the F0 domain in the ATPase to cause such specific conformational changes in the active site of the F1 domain via proton-driven rotation requires foresight planning and ingeniously designed interaction. Light is that ingenious mechanic of Nature. Light sculpts life using bioelectricity as its carrier of information. Semiconductors quickly change light into a DC electrical signal. As Nick Lane shows above, bioelectricity gives direct feedback to the AMO physics inside of cells, which causes morphologic changes in the ATPase. Problem solved.

Grey hair (lack of melanin) likely forms the same way the ATPase was formed. Bioelectricity changes in the hair follicle via mitochondrial biophoton likely impact a gene tied to this story. The name of the hair color thief gene is IRF4. It is a gene that acts like a cog in the bioelectric machine in a cellular process that churns out melanin pigment in the hair follicle. Graying happens as follicles gradually stop producing the pigment that gives hair its color, which happens at different rates for different people due to dielectric changes in water mtDNA makes. Various people make different biophoton signals. This gives mammals variable shades of grey in their hair. Even the greying of hair in humans shows that we are not entirely at the mercy of our genes. The study found that environmental factors controlled about 70% of cases of hair graying. Genes were responsible for only about 30% of the population, at least in the Latin American cohort. HYPERLINK

HOW?

I’m plunging you deep into waters of H⁺ networks, dielectric constants, and biophotons—how they shape light’s dance in cells. As your water muse, you’ll need to flow with my ideas, threading your insights on mitochondrial matrix dynamics, pH shifts, and ultraweak UV emissions into a cohesive stream, backed by biophysics. Let’s ride this wave, it is sharp and luminous.

H⁺ Networks and Dielectric Constants

H⁺ protons—aren’t just passengers in water; they’re conductors, fast and fickle. In the mitochondrial matrix, they zip through Grotthuss-like hops, a relay race of bonds, adapting in femtoseconds (Marx, 2001). This network sets water’s dielectric constant—the measure of its ability to screen electric fields. Bulk water’s 78 at 25°C—high, polar, stable. But tweak the H⁺ load, and it shifts. Low pH (acidic, proton-rich) drops it—say, to 60 or lower in inflamed tissues (Stillinger, 1980). High pH (alkaline, proton-scarce) nudges it differently, often lower too, as OH⁻ skews polarity.

Why? Protons mess with water’s dipole alignment. More H⁺ tightens the network, less screening; less H⁺ loosens it, and it’s the same deal. The dielectric constant ties to refractive index (n ≈ √ε_r)— explains how light bends in Nature and your cells. This is FERMAT’s LAW.

Normal water’s n ≈ 1.33; tweak the dielectric, and light’s path warps in cells. I’ve just described how Fermat’s law works in you. That’s tissue optics shifting—light scatters or tunnels differently. Why can’t we accept Pollack’s work? There is an entire thread about that on my website forum. He never tested deuterium-depleted water in his experiments. Deuterium changes the dielectric constant in ways his book never examines. Therefore, most of what he believes is speculation.

Mitochondrial Matrix: A Dielectric Playground where biophotons are made

The matrix isn’t bulk water—it’s a proton-packed cauldron near the IMM, where ETC pumps H⁺ out, and F₀ spins it back. Dielectric’s not static—O₂, exotic atoms (Ca²⁺, Mg²⁺, Na⁺, K⁺, Cl⁻, I⁻), and wide-bandgap players—tune it. Calcium spikes (e.g., mitochondrial uptake) bind water and drop dielectric locally (Fettiplace, 1980). O₂, chugging electrons to make H₂O, tightens H⁺ density—dielectric dips. Should the liquid-metal H⁺ riffing in previous blogs now resonate in your neural circuits? That’s extreme density, magnetic containment, further slashing dielectric, & light bending hard now inside your cells.

This isn’t random—cells sculpt light. Low dielectric (acidic matrix, high H⁺) slows photons and shifts, altering their physiological game; high dielectric (buffered spots) speeds them up. Inflammation & low pH dim the illumination of tissues by turning down mtDNA light transformation; alkalinity tweaks it another way. These are all the things Gerald Pollack missed in his 2013 book. Wide-bandgap atoms (Mg in chlorophyll, I in thyroid) act semiconductor-style, gating electron flow and tweaking light’s fate in your cells.

Biophotons: Ultraweak UV Jobs

Roeland van Wijk’s work (e.g., Light in Shaping Life, 2014) nails it—cells emit ultraweak biophotons, 200-800 nm, peaking in UV (250-350 nm). Not floodlights—10⁻¹⁹ W/cm², a whisper. Source? Mitochondrial ROS (superoxide → singlet O₂), lipid peroxidation, protein excited states. Each photon’s frequency—tied to its energy (E = hν)—has a physiological gig in your cells. The specificity and sensitivity in this game of light blow the centralized ideas in biology to smithereens. UV repairs DNA (Sancar, 2004), signals redox (Tafur, 2010), & it cues clocks and invokes entropy (CRY proteins, Provencio, 2000). Cells specialize—skin spits UV for melanin, creating electrons and a higher dielectric in water, providing optimal signal fidelity in neurons.

Dielectric shifts in water tune this all. Low dielectric (high H⁺) red-shifts biophotons—less punch, more scatter. The high dielectric blues the light. This sharpens the tool and makes it more focused. This light bends more than red light inside cells. Red light does not bend and reduces inflammation & pain in tissues. This stimulates inflammation. Inflammation’s proton flood dulls the signal—chronic disease brews. Matrix O₂ and ions? They dial the frequency and precision jobs for each cell type.

The Flow of Life?

H⁺ networks flip water’s dielectric switch—refractive index follows, & light bends. Matrix atoms and pH sculpt it further—photons morph, & physiological jobs shift. Biophotons—UV whispers—run the show, but only if the medium optics are right. AMO physics is the name of the game. This is why atomic contaminants from vaccines demolish mitochondrial function, causing many new diseases. They are messing with biophoton transformation from matrix to cytosol. No innate flaws; bad light, bad water, & bad vibes skew it.

The Decentralized Clinicians Rx should be about fixing the electrodynamics in the patient’s field—sun, not screens—or the signal for health will be lost.

Next wave—how do we map this dielectric dance in real time?

Prolonged stress of any kind (Brain Gut 16 blog) depletes our cell membranes of DHA, ruins both loops of the Bazan effect, and increases the need for methionine because ubiquitin rates skyrocket. DNA is designed to be quiescent and not active. Sunlight keeps our DNA transcription low. nnEMF/blue raise it. DHA allows our cells to power up electrons with photons from sunlight and helps melanopsin move protons to control melatonin levels and mtDNA heteroplasmy. DHA fundamentally takes light and turns light into electric-mechanical signals in cell membranes everywhere, but especially in our skin and brain. Our mitochondria have two sets of cell membranes. Its inner and outer chemistry is enormous in tunneling electrons. If it does not work, we get a redox shift in our Q cycle’s operability. That cycle moves food electrons from cytochromes 1 to 3 and to 4 and then to the ATPase, where protons are moved from the matrix to the outer mitochondrial membrane. H+ is the favored subatomic particle in this organelle.

Deuterium is typically excluded here and kept in the blood to perform another quantum task. This is why the retina has more DHA in it than any other place in the human brain; it is located on the interface where light from the sun first interacts with the brain’s mitochondria in the RPE of the retina. Visible light has part of it tied to blue light when a prism separates it. Blue light is the part of the spectrum of light that destroys DHA, lowers melatonin, and causes the respiratory proteins in mitochondria to swell and lose their electric charge. Red light does the exact opposite bioelectrically in mitochondria. In fact, cytochrome C oxidase is condensed by red light because it is related chemically to hemoglobin. Both are heme-based proteins. All heme-based proteins’ initial construction steps happen inside the mitochondrial matrix, where the bioelectric current is powerful.

SUMMARY

Centralized biochemistry refuses to accept or believe that heme processing relies on a “bioelectric current” but on biochemical reactions and enzymatic pathways instead. Robert O. Becker’s and Michael Levin’s work have not been imported, and centralized ideas in biochemistry have yet to be updated. Light control of mitochondria is now well established in the biophysics literature and stands in counter distinction to the reality found in the operation of the physics of organisms. For example, UVA light and IRA light control many systems in the operation of cytochrome C oxidase. Biochemistry has yet to explain this paradox. The reason: it is not a paradox. Centralized biochemistry is not foundational to how life operates. Quantum mechanics is.

Cytochrome c has four red-light chromophores and a VDR receptor. The VDR receptor, when activated, dramatically slows electron chain transport. Red light, however, can still process proteins without food electrons with 100% efficiency. The ATPase is a quantum rotator engine for red light in sunlight. The red light spins the ATPase faster to make more ATP available, especially when present with UVA light. This is why AM sunlight has the exact same amount of red light as blue light. Red light is the antidote to the stimulus of blue light, but only UVA once the day proceeds.

IF YOU WANT TO AVOID JAB CONSEQUENCES, these spectra must be balanced to work correctly in the eye/skin/gut. What happens in the skin and eyes determines how the gut operates. This is why we are designed to replace DHA constantly in our retina/skin with excessive blue light hazards. When we do not, we get macular degeneration, cataracts, and glaucoma. When we do get too much blue light from our environment, it is a stressor to the retina, and the protective response from the cornea is to develop cataracts to protect the retina from DHA loss. DHA is a brain/ retina/skin story for humans.

CITES

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3257695/

https://www.sciencedirect.com/science/article/pii/S0092867400814567

https://pmc.ncbi.nlm.nih.gov/articles/PMC9072658/

https://www.nature.com/articles/nrmicro1771

https://academic.oup.com/jb/article-abstract/149/6/655/2182760?redirectedFrom=PDF

https://www.nature.com/articles/ncomms10815

https://threadreaderapp.com/thread/1893468340569677950.html

‘The drug war isn’t what you think it is’ – it is very interesting concept when you begin to. realize the Shamans inside the Amazon have always called ayahuasca, medicine.

The video above is on glioblastoma multiformans. It is the most lethal human cancer we know of in centralized medicine. Nothing kills quicker. In this way SV40 has a lot in common with it. It happens to be the one cancer neurosurgeons are expert in, in terms of treatment options. This blog today is about the decentralized treatment for this cancer linked to the jab. I think these options should be added to any GBM case after my visit to the Amazon in Suriname.

^^^^This is me in the Amazon River system with a Pirana net behind me looking for answers.

Why did I believe forests the best decentralized pharmacies on Earth to look for clues to cure incurable diseases like SV40 intercalation?

Why did I chose Suriname? The picture tells you why I chose it.

THEY HAVE THE LARGEST FOREST OF ANY COUNTRY.

93% of this country is a tropical rainforest.

Did you know most of the forests, are home to plants and animals that cannot be found anywhere else in the world? The same is true with the compounds their soils and water table create out of sunlight and magnetic flux in the magma chambers below them.

So what were the clues I went to Suriname with? What was I looking for?

I was looking for confirmation of why Ivermectin was working on people who took the jab who had turbocancers. Did I find what I was looking for?

I think I might have.

This table above was the treasure map I went to Suriname with. I then found 12 tribes who were treating the jab injured who’s story I laid out here.

https://x.com/dralexisjazmyn/status/1890609382422913290

You might want to listen to the ENTIRE podcast I did with her. I tried to tell some of my high profile clients what I was up to, but none of them wanted to listen to me before I left. I think that might change now.

FOR THE JABBED I BELIEVE WE NEED TO FIND UNIQUE NEVER BEFORE USED CBD’s

Why did I believe the Amazon a key player for the jabbed, especially those with SV40?

Dr’s Mary Sherman treatise made some comments about chemicals that had elements that were related to this class of drugs. She did not know that we had an endogenous endocannabinoid system operational in humans.

The Amazon is 55 million years old and it is where THE unique CBDs are created because of its geology. That is why I went to Suriname. They have CBDs that are unique.

You should visit @cannmed or @cannmedevents to learn more about this topic. That is their Twitter handles.

Cannabinoids exhibit some overlapping mechanisms with ivermectin (IVM) and fenbendazole (FenBen) in cancer treatment, particularly through microtubule disruption, apoptosis induction, and metabolic modulation.

Below are key comparisons:

1. Microtubule Disruption (Similar to Fenbendazole)

•Fenbendazole: Binds β-tubulin, disrupting microtubule polymerization, leading to mitotic arrest and cancer cell apoptosis.

•Cannabinoids: Some cannabinoids, such as CBD (cannabidiol) and THC (tetrahydrocannabinol), have been reported to destabilize microtubules:

•CBD disrupts tubulin polymerization, impairing mitotic spindle formation in glioblastoma and breast cancer models.

•THC alters microtubule dynamics, potentially affecting cancer cell division.

•Overlapping Effects: Cannabinoids may enhance the microtubule-disrupting effects of FenBen.

2. p53 Activation & Apoptosis (Similar to Both IVM & FenBen)

•FenBen & IVM: Upregulate p53, increasing apoptosis via BAX/BAK activation. •Cannabinoids:

•CBD & THC upregulate p53, leading to mitochondrial dysfunction and apoptosis.

•Activation of caspase-3 and caspase-9, triggering programmed cell death in multiple cancers.

•Overlapping Effects: Cannabinoids can synergize with FenBen or IVM to enhance apoptosis in cancer cells.

3. Metabolic Disruption & AMPK Activation (Similar to FenBen)

•Fenbendazole: Blocks glucose metabolism in cancer cells, reversing the Warburg effect. •Cannabinoids:

•CBD inhibits glucose uptake via downregulation of GLUT1 transporters.

•AMPK activation by cannabinoids leads to mTOR inhibition, reducing cancer cell growth. •Overlapping Effects: Cannabinoids mimic FenBen’s metabolic disruption, making them potential synergistic agents.

mTOR inhibition was a big topic in the Quantum Engineering Series and especially in the Melanin Renovation blog.  You might want to re-read it if you took that jab.

4. Anti-Inflammatory & Immune Modulation (Similar to Ivermectin)

•Ivermectin: Modulates immune responses by shifting T-cell and cytokine activity, reducing cancer immune evasion.

•Cannabinoids:

•CBD reduces inflammatory cytokines (IL-6, TNF-α), potentially lowering tumor-promoting inflammation. It also affect the nrf2 pathway. NRF2 detox pathway only is tapped by having redox power present in the cell.

If your cells lack the net negative charge, they will never experience this pathway. So if you are pale and think using CBD is a panacea you’re a special kind of centralized idiot.

•Enhancement of immune surveillance through interaction with CB2 receptors on immune cells.

•Overlapping Effects: Cannabinoids may amplify IVM’s immune modulation in cancer therapy. Key Cannabinoids With Cancer-Treatment Potential

1. CBD (Cannabidiol): mechanism of action

•Microtubule disruption •p53 activation → apoptosis •AMPK activation → metabolic inhibition •Anti-inflammatory effects (reduces IL-6, TNF-α) alters NRF2 signaling.

2. THC (Tetrahydrocannabinol): mechanism is different but synergistic.

• Microtubule destabilization

• Apoptosis induction via CB1 receptor

• Inhibition of angiogenesis in tumors

3. CBG (Cannabigerol):  mechanism is different but also synergistic.

• Inhibits mitochondrial respiration in cancer cells

• Synergizes with chemotherapy

4. THCV (Tetrahydrocannabivarin): mechanism is synergistic

•Reduces tumor cell proliferation

•Modulates AMPK/mTOR pathway Potential Synergistic Treatment Approaches (mTOR again)

•CBD + Fenbendazole: Both disrupt microtubules and glucose metabolism.

•CBD/THC + Ivermectin: Immune modulation + apoptosis enhancement.

•Full-spectrum cannabinoids + Metabolic inhibitors: Combination therapy for aggressive cancers. All of these chemicals help and many of these chemicals have unique chemistry in the Amazon basin and make up most of the magic present in the CBD decentralized pharmacy we need to tap for the jabbed.

My Conclusion: Cannabinoids share several anti-cancer properties with FenBen and Ivermectin, particularly microtubule inhibition, p53 activation, metabolic disruption, and immune modulation. This suggests they could be complementary in cancer treatment, especially in the jabbed. More decentralized research from the forest is needed to explore their combined effects.

We need to map the effects out and some have already begun this.

5. People in places on Earth are already doing this work and I have spent time meeting and talking with them.

Pay attention to IVM/FenBen for cancer as the recent blogs have laid out.

New data is arriving and ties DIRECTLY to my trip to the Amazon.

IVM prevents SV40 promoters from entering the nucleus but blocking importin A/B. This pathway likely has many chemokines that can impact it.

Yes, ivermectin is known to inhibit importin α/β-mediated nuclear transport, which is relevant in the context of SV40 promoters. Dr’sSarah Stewart and Mary Sherman did not know this when they working for DoD and Dr. Ocshner.

The Mechanism:

•Importin α/β Pathway: This transport system is responsible for shuttling proteins with nuclear localization signals (NLS) into the nucleus. Many viruses, including SV40, hijack this pathway to deliver their regulatory proteins (e.g., Large T – antigen) into the nucleus for replication and transcriptional activation.

• Ivermectin acts as an Inhibitor: Ivermectin binds to importin α/β and disrupts its function, thereby preventing nuclear entry of proteins that depend on this transport mechanism. Implications for SV40 Promoters:

•SV40 Promoters: The SV40 early promoter is often used in molecular biology due to its strong transcriptional activity in mammalian cells. However, its transactivation requires the nuclear localization of SV40 Large T antigen, which depends on importin α/β.

• Blocking Nuclear Entry is a KEY: If ivermectin blocks importin α/β, it could prevent SV40 Large T antigen from entering the nucleus, thereby reducing SV40-driven gene expression and viral replication.

Do we already have Experimental Evidence for this?    Yep.

•Studies have demonstrated ivermectin’s ability to inhibit nuclear import of viral proteins from various RNA and DNA viruses (e.g., HIV-1, Dengue, and even SARS-CoV-2).

•SV40 Large T antigen is known to require importin α/β for nuclear entry. If ivermectin blocks this pathway, it could theoretically interfere with any SV40-driven transcription or replication in systems using this promoter.

Certainly, here are some key studies that provide evidence on this topic:

1. Ivermectin as an Importin α/β Inhibitor:

• A study by Wagstaff et al. (2012) demonstrated that ivermectin specifically inhibits importin α/β-mediated nuclear import. The researchers found that ivermectin effectively blocked the nuclear import of proteins dependent on the importin α/β pathway, without affecting other nuclear import pathways. This inhibition also correlated with a reduction in the replication of viruses such as HIV-1 and dengue virus, which rely on this pathway for nuclear entry of their proteins. (http://pmc.ncbi.nlm.nih.gov)

2. SV40 Large T Antigen and Importin α/β:

•The SV40 Large T antigen contains a nuclear localization signal (NLS) that is recognized by importin α, facilitating its transport into the nucleus via the importin β pathway. This nuclear import is essential for the Large T antigen’s role in viral replication and cell transformation. (http://en.wikipedia.org)

These studies collectively suggest that ivermectin’s inhibition of the importin α/β pathway could impede the nuclear import of SV40 Large T antigen, potentially affecting SV40 promoter activity and viral replication.

Does Fenbendazole Upregulate p53?

Yes, Fenbendazole (FenBen) has been reported to upregulate p53, a key tumor suppressor protein, in some cancer models.

Mechanism of p53 Upregulation by Fenbendazole:

1. Disruption of Microtubules:

• Fenbendazole binds to tubulin, preventing microtubule polymerization in a manner similar to colchicine or vinblastine.

• This leads to mitotic arrest, which can trigger cell cycle checkpoints and activation of the p53 pathway.

2. Induction of Cellular Stress & DNA Damage Response:

• Microtubule disruption can cause mitotic spindle stress, leading to chromosomal instability. • This activates ATM/ATR kinases, which phosphorylate p53, stabilizing it and increasing its transcriptional activity.

3.Apoptosis and Autophagy Induction:

• Upregulated p53 can activate BAX/BAK pro-apoptotic proteins, leading to mitochondrial damage and caspase-dependent apoptosis.

• Fenbendazole also promotes autophagy, which can contribute to cancer cell death.

There are 3 dominant phases of synchronized metabolic and transcriptional reprogramming when pigmenting the skin to avoid jab diseases. The melanogenic trigger is associated with high MITF levels and rapid glucose uptake by the cell. Blue light exposure stimulates massive glucose mobilization from ACTH from POMC. The transition to a pigmented state is accompanied by increased glucose channelization to anabolic pathways in biochemistry that support melanosome biogenesis. The H+/D optical switch you heard about in the Kruse for Dummies talk is critical in optimizing this step. You can find this talk in my IG biolink.

I talk about how I do this with pulsed light in the Melanin Renovation blog.

4. Inhibition of Glucose Metabolism

(Warburg Effect Reversal):

•Some studies suggest Fenbendazole reduces glucose uptake by cancer cells, similar to metformin. It might be time to consider this drug for all tech abusers.

• This metabolic stress can further activate AMPK pathway which leadsto p53-mediated tumor suppression. Why did the Brazilian tribes get more sick in my story to Dr. Alexis? They used technology that had enriched blue light. None of the other tribes did.

• This metabolic stress can further activate AMPK pathway which leadsto p53-mediated tumor suppression. Why did the Brazilian tribes get more sick in my story to Dr. Alexis? They used technology that had enriched blue light. None of the other tribes did.

Fenbendazole’s Mechanism for Cancer Treatment
1. Microtubule Disruption (Primary Mechanism)

• Fenbendazole binds to β-tubulin, disrupting microtubule formation.

• This prevents proper mitotic spindle formation, leading to G2/M cell cycle arrest.

• Cells stuck in mitotic arrest either undergo apoptosis or senescence.

2. Apoptosis Activation via p53 & BCL-2 Inhibition

•Cancer cells often overexpress BCL-2, an anti-apoptotic protein that prevents programmed cell death.

•Fenbendazole inhibits BCL-2, shifting the balance towards apoptosis.

3. Disrupting Glucose Metabolism in Cancer Cells

•Fenbendazole has been shown to reduce glucose uptake and ATP production, increasing oxidative stress in tumors. This is why PBM might help the jabbed as well. Light induces an abscopal effect on cells

• This effect weakens cancer cells that rely on glycolysis (Warburg effect), making them more sensitive to treatment. PBM is known to lower blood glucose by close to 30% via its abscopal effect.

4. Synergistic Effects with Chemotherapy & Radiation

Exposure to Ultraviolet Radiation in the A band promotes the expression of melatonin receptors in the skin via alpha MSH made from POMC.  This occurs via melatonin receptors called MT1 and MT2.

In the lab, pretreatment with melatonin reduced cyclobutane pyrimidine dimers (DNA strand breaks) levels by approximately 40%.  Remember, life is not lived or built in a LAB……….Nature is your lab.  AM sunlight increases melatonin and melanin.   https://www.nature.com/articles/s41598-017-01305-2

• Some studies suggest Fenbendazole enhances the effects of radiation and chemotherapy by:

• Increasing DNA damage accumulation.

• Disrupting repair pathways (e.g., via p53 activation).

•Weakening microtubule integrity, making cancer cells more vulnerable to other drugs or light therapy.

Supporting Studies & Evidence
1. Fenbendazole inhibits tumor growth via microtubule disruption and p53 activation

• Study in lung cancer cells showed that Fenbendazole caused mitotic arrest, increased p53, and induced apoptosis.

• (Source: PubMed)

2. Fenbendazole enhances radiation sensitivity by targeting microtubules and p53

• Research demonstrated that combining Fenbendazole with radiation led to increased DNA damage and cell death.

• (Source: PMC)

3. Mechanism of Fenbendazole in disrupting glucose metabolism

• Study found Fenbendazole downregulates GLUT1, reducing glucose uptake in cancer cells.

• (Source: PubMed) Conclusion Fenbendazole upregulates p53 by causing mitotic stress, DNA damage, and metabolic inhibition, leading to cancer cell apoptosis.

Its primary mechanism is microtubule disruption, similar to drugs like Vinblastine or Colchicine used in centralized oncology, but with much lower toxicity.

CAN THESE IDEAS HELP TURBO BREAST CANCER = triple negative cancers?

Stage 4 triple negative breast cancer is exploding in the COVID era in our women. I really worry about our children who are still getting popped with these jabs by pediatricians now.

There are case reports that this type of cancer can be erased with a combo of cannabinoids and psilocybin. Might we be able to find unique genomes of these two drugs to combine together to treat SV40 induced cancers?

I think so.

It might turn out that the centralized drug war isn’t what you think it is. It may fuel the decentral drug wars on all human cancers.

Yes, psilocybin (and its active metabolite psilocin) interacts with several cancer-related pathways, some of which overlap with Fenbendazole, Ivermectin, and Cannabinoids. While psilocybin is primarily studied for its psychedelic and neuroplasticity effects, emerging research suggests it may have anti-cancer properties via serotonin receptor modulation, immune regulation, and metabolic disruption.

1. Serotonin (5-HT) Receptor Activation and p53 Pathway (Similar to FenBen & Cannabinoids)

^^^^Remember where you saw this? The Melanin Renovation blog.

Sunlight  increases the catalytic activity of enzyme IDO in this pathway as I already taught you

When this happens your cells begin making more kynurenine and less serotonin and melatonin this leads to ALL CHRONIC DISEASES and INCREASES ALL CAUSE MORTALITY because it destroys melanin renovation internally.

WHILE simulataneously  SUNLIGHT decreases the catalytic activity of KAT

Making less kynurenine acid (neuro protective for melanin) and more quinolinic acid (harmful for melanin) from melanin degradation.

A lack of sun causes melanin degradation via hypoxia. Non native EMF cause liberation of Vitamin A from the loose covalent bond of opsins in the non visual photoreceptor system due to alien light and that Vitamin A cause hypoxia by lowering NAD+ in a cell.  This is today’s major cause of disruption in this pathway.  This slide should jar your memory of my past lessons. If you are jabbed you really better get this lesson.

•Fenbendazole & Cannabinoids: Activate p53, leading to apoptosis.

•Psilocybin/Psilocin:

•Activates 5-HT2A receptors, which have been linked to p53 upregulation in certain cancer models.

•Induces apoptosis in neuroendocrine tumors via serotonin signaling.

•Regulates BCL-2/BAX, modulating mitochondrial-mediated apoptosis. Overlap:

•Psilocybin may enhance p53 activity, similar to FenBen and CBD, leading to cancer cell apoptosis.

2. Metabolic Disruption & AMPK Activation (Similar to FenBen & Cannabinoids)

•Fenbendazole: Blocks glucose metabolism, shifting cancer cells away from glycolysis (Warburg effect).

•Psilocybin: Modulates AMPK/mTOR pathways, which regulate cancer metabolism and cell proliferation. 380 nm light addition here to improve mTOR signaling. This is why the tropics are critical to these riddle. Remember this slide from the Melanin Renovation Rx for Mammals on Patreon?

•Downregulates PI3K/AKT, reducing cancer cell survival.

•Potential to inhibit GLUT1 (glucose transport), limiting cancer energy supply. Overlap:

•Psilocybin could synergize with FenBen, Metformin, or Cannabinoids in starving tumors of glucose. PBM should added here because it lowers blood glucose and insulin signalin..

3. Microtubule Interactions & Cytoskeletal Remodeling (Similar to FenBen)

•Fenbendazole: Binds β-tubulin, causing mitotic arrest and apoptosis.

•Psilocybin/Psilocin:

•Modulates cytoskeletal proteins, leading to structural changes in cancer cells.

•Impacts tubulin polymerization in neuronal cells, suggesting possible effects on cancer cell microtubules.

Overlap:

•Though less studied in cancer, psilocybin’s cytoskeletal effects might interact with FenBen-like mechanisms.

4. Immune Modulation & Anti-Inflammatory Effects (Similar to Ivermectin & Cannabinoids)

•Ivermectin: Modulates T-cell responses, enhances anti-tumor immunity.

•Psilocybin:

•Reduces pro-inflammatory cytokines (IL-6, TNF-α, IL-1β), which drive tumor progression. •Enhances immune checkpoint modulation, possibly affecting PD-1/PD-L1 pathways in cancer immunotherapy.

•Shifts immune response toward anti-cancer cytotoxicity.

Overlap:

•Psilocybin’s immune effects mimic Ivermectin’s immunomodulation, making it a potential adjunct in cancer therapy.

5. Potential Synergies with Ivermectin, FenBen, and Cannabinoids PathwayFenBenIvermectinCannabinoidsPsilocybin p53 Activation Microtubule Disruption (CBD) (Possible) AMPK/mTOR Modulation Metabolic Disruption Immune Modulation

 

When I asked the Shamans about how they chose their medicines I found out they were using drugs from the forrest that all had these overlaps.

Psilocybin has overlapping mechanisms with Fenbendazole, Ivermectin, and Cannabinoids in p53 activation, immune modulation, metabolic disruption, and possibly cytoskeletal remodeling. While direct microtubule inhibition is uncertain, serotonin signaling, apoptosis, and immune regulation suggest a potential role in cancer therapy. Would you like specific references, drug interaction studies, or potential combination therapies explored further?

THE REMEDY CAME ON LIKE A DREAM TO ME.

SUMMARY

Catastrophic disclosure was not linked to the information and evidence coming out during the COVID era.  Covid narratives were built around but a consolidation around these false narrative by federal agencies who incentivized many groups to support COVID psy-op fear;  This is how a false flag operations happened at scale from 2020-2025.

All of you need to follow @JohnBeaudoinSr on Twitter because he has all the death data that holds the truths.

He has the data to support this supposition in great detail. This was catastrophic epoch in human health because it leads the world not having unity and peace, something beautiful, a view of us and other civilizations doing things together, a new world being created, technologically sustainable and peaceful, is about uniting the world under a sort of militaristic global totalitarian system built around surveillence capitalism where everyone is terrified what’s out there for many reasons linked to the narratives furnished and created by federal agencies so thatpeople give up their freedom with out a fight   It’s a fear play to circumvent a political revolution if the truth really was known by “We The People.”

Many Federal agencies had a legal duty to inform us but they were told by the DoD and Pentagon to hide behind faulty use of the ICD coding system to lie to us. So far they have gone unpunished. The first step is complete banning of the mRNA platform. This podcast shows you why I undressed Calley Means in December with Danny Jones. Calley calling for “transparency” was a criminal speaking given the hard core data John has.

I’ll stop being a conspiracy theorist when the rich and powerful stop conspiring. We are being ruled by centralized greedy, power-hungry sociopaths who are destroying the planet. They are being led online by Sergey Brin and Anne Wojicicki.

HYPERLINK

Women who are infertile maybe helped by some of this information in this blog to have children. Why? The elites crafted the COVID era with the mRNA platform. The elites are eugenicists and they favor quick death via abortion for women to rid the planet of future children due to their perverse beliefs. The mechanisms of infertility mimic the disease creation of the turbocancers. All are tied to leptin melanocortin pathways.

The elites like Brin, Gates, Fauci, and GAVI linked companies knew to break the golden centralized rule in pregnancy medicine. Pregnant women are already in a state of immunosuppression so why would you then give a pregnant woman 4 different vaccines during this time? To eliminate NEW life is the short answer.

This is especially one that is experimental and the data showed it caused spontaneous miscarriages. So Gates and Fauci knew that the COVID jab was a covert abortion clinic run at global scale.

When you know better you can do better, and Uncle Jack is digging deep for the remedy for 6 billion of you.

CITES

1. https://www.youtube.com/watch?v=cn5CQGpiJWg

2. https://www.youtube.com/watch?v=KQUSoIJkaWg

3. https://www.youtube.com/watch?v=NN9X04C3G5Y

4. https://www.youtube.com/watch?v=SgCSGN8UR9M

5. https://www.youtube.com/watch?v=tAf3UB2ycCs

6. Single dose- is not BigHarma friendly solution. https://www.nejm.org/doi/full/10.1056/NEJMoa2206443

7. https://www.nejm.org/doi/full/10.1056/NEJMoa2032994

8. https://x.com/marclanders/status/1891460901363921148

What am I doing in the Amazon rain forest in Suriname? I am looking for the antidote for those who took the jab. 50 years ago I learned from the Cutter incident linked to the Polio jabs of Salk and Sabin and the unpublished work of Dr’s. Sarah Stewart and Mary Sherman on viruses that they could be treated using novel chemokines found in soils. Sherman extended Stewarts idea that exposure to any form of radiation treatment would somehow strengthened the virulence of the SV40 virus via bond loosening within an animal. It was a discovery that was worthy of a Nobel Prize but no one in science could ever know what these two woman do because their work was secret and confined to a “biological Manhattan Project” for the DoD in New Orleans in the 1950s.

Chemokines use deuterium chemistry to strengthen bonds. They have the opposite effect of electromagnetic radiation. Stewart knew this from her work with nuclear reactors at the University of Chicago in the Fermi lab. Dr. Stewart found SV40 was inhibited by things grown in water with an unusual isotopic fingerprint. That information was buried in the boxes I found in the basement of Charity hospital in NOLA where I trained as a neurosurgeon in the late 1980s and and early 1990s. Parts of Dr Stewart’s work/ thesis was still present there at this time and I found some more of her original ideas in Tulane University School of Tropical Diseases on Canal Street. I read this in 1992. Now I am using that information here at the 4N to help solve a mystery for the jabbed.

Mitochondriacs should believe in scientists who are relentless in finding the wisdom of nature that builds the public “health truths”. I just had an amazing experience inside the Amazon Rain forrest looking for a solution for the jabbed. It was a successful journey.

Mitochondriacs learn to seriously doubt those who believe they found a solution in a false narrative. They are comfortable questioning everything and they have no problem digging deeper to find the elusive truth they need. They come to recognize the power in Nature to sculpt health and they realize her power never diminishes yours. If you live her way, she will give you immense power to control your health journey in this life.

THE DISCOVERY OF IVERMECTIN IS WHAT DROVE THIS TRIP TO THE AMAZON

In philosophy and science, a first principle is a basic proposition or assumption that cannot be deduced from any other proposition or assumption. First principles in philosophy are from first cause. Mitochondriac wisdom is dedication to teaching other how to think in decentralized fashion to improve life on earth in some small way.

Centralized medicine was weaponized by the DoD and turned BigHarma into the largest killing machine man has ever created. In the Amazon I met with a group who may hold the key passage to disintermediating the death grip of this banker inspired empire over humanity.

In the late 1960s and early 1970s, Satoshi Ōmura, a microbiologist and bioorganic chemist at Tokyo’s Kitasato Institute, hunted for new sources of pharmaceuticals. He knew that some existing drugs, including antibiotics, had been derived from compounds found in nature. The forrests of the world are essentially decentralized pharmacies that have been acted on by light, water, and magnetism for billions of years and this created a myriad of compounds for humans to use. Their use was done by trial and error. This became his philospophy of touch. How could touching the Earth lead to healing humans all over the world.

So he developed screening methods to identify medicinally promising compounds from soil. His team collected thousands of soil samples from around Japan, cultured bacteria from them, and screened each culture for medicinal potential.

THE EUREKA MOMENT

The story is so improbable it defies belief: This is what motivated me to go deep into the Amazon. The story of how Ivermectin was discovered began with a soil sample from Japan stops suffering in Africa. It begins when a scientist discovers a lowly bacterium near a golf course outside Tokyo. A team of scientists in the United States finds that the bacterium produces compounds that impede the activity of nematode worms. It is developed into a drug that wards off parasites in countless pets and farm animals,averting billions of dollars in losses worldwide.

Extraordinarily, the drug also prevents or treats human parasitic diseases that would otherwise cause blindness and other severe symptoms in hundreds of millions of people in many of the poorest countries on Earth.

During COVID it was found to affect the development of rapid cancers from the poorly designed mRNA platform. Why was this information key? It pointed me in the direction of the circadian clock genes to look for an answer. Rapid cancer generation would mean the circadian mechanism had to be hijacked in some novel way.

First principle thinking teaches us that morning sun exposure resets the circadian clocks of the skin, allowing your body to properly prepare for UV Circadian genes like CLOCK and BMAL1 regulate the expression of key DNA repair proteins, ensuring that DNA repair is most effective when the body is exposed to higher levels of DNA-damaging factors This is why you want to get any screening which involves electromagnetic radiation, like an X-ray, or a cell phone, or the sun, done in the late morning to early afternoon (Approximately 10 AM – 2 PM) This period is when the circadian-controlled DNA repair mechanisms are generally more active It’s no coincidence that this is also the timeframe when UV light from the sun is most intense Nature doesn’t miss and opportunity to create a mark. This told me going to a place where the sun was dominant and where other bands of radiation were absent were the key in finding the answer for the jabbed.

In the last four years I have done a lot of reading about the unique environments in the largest decentralized pharmacy on earth, the Amazon basin. Some of the quantum biological principles I have acquired over this time have led me to Suriname to find a reversal for the jabbed. I have a hunch where the answer is because of this information processing. Today is the first day of this journey of discovery. Always remain curious. Remain open. When someone tells you a problem like SV40 is unsolvable this should make the challenge palatable and worthwhile.

SUMMARY

The tale depends on an international cast of thousands of scientists, medical practitioners and other dedicated participants. It also involved a BigHarma company and research institute that was willing to give a drug away for free. This was done to build trust between the company and the public so that the public would come to lean on and trust industry when that TRUST should not be given. To be sure, Ivermectin did help rid the developing world of debilitating diseases.

Yet none of this would have happened without that soil dug up in Japan and a healthy dose of serendipity.

The odds against finding avermectin smaples in the soil and recognizing its potential were astronomical. The transformation of avermectin to ivermectin was truly the easiest step in its discovery. The biggest irony is that BigHarma was aggressive with this drug in development because it had so a high therapeutic index. Ivermectin is a particularly attractive treatment because They believed in the 1970’s and 1980s it had no antibacterial or antiviral activity, w hile having few serious side effects even at massive doses. In the 2000s we found out Ivermectin has antiviral activities especially to genetically altered virus. If Big Harma would have known this early on, my bet is DARPA and the DoD would have strangled this drug in its crib during discovery. They did try to do this in 2020-2025 during the COVID pandemic to force people to take the BigHarma jab.

History shows us this drug could so easily have been missed, and the life-altering decentralized pharmaceutical that was derived from it might never have been discovered.

This is what motivated my trip to the Amazon to look for clues that might help the jabbed. I am happy to say to you, my sleuthing might have paid off so this slide can be disintermediated. This is the slide I showed to Dr. Bowden and Danny Jones in our podcast together that showed that the jab is the MAIN PROBLEM in causing cancers. That huge spike going almost vertical in the bottom right is why I went to the jungle.

 

https://vimeo.com/user192601857/review/1021313092/2e32d43335?fbclid=IwY2xjawIE6ThleHRuA2FlbQIxMAABHenEmPztUp0_J41kl6nwjpOD5R9YwTkloMpyR_wwj4WZ6PPjp2rc6on3Gw_aem_EDfZT-fVQOAhbsNRgpsivw

Over the 20th Century, various investigators in different countries representing multiple interests have repeatedly reported the discovery of unusual non-local field effects in human biology that could not be explained in the framework of the Standard Model. Since the investigators and writers could not understand or explain the physics associated with the observed phenomena, they were forced to invent new names for the fields, emanations, and energies believed to be responsible for creating these phenomena. Scalar phenomena and torsion field manipulations are two examples of this idea.

If you listened to my Breedlove podcast, you would have heard about the subtraction of James Maxwell’s original equations from 20 to 4 equations. I believe this “censorship” or subtraction is the cause of the Standard Model’s missing framework to explain many phenomena in Nature.

Oliver Heaviside was a self-educated English mathematical physicist who spent most of his life on the far fringes of the scientific community. Yet he did more than anyone else to shape how James Clerk Maxwell’s electromagnetic theory was understood and applied in the 50 years after Maxwell’s death. Indeed, Maxwell’s equations in their most familiar vector form come from Heaviside’s reformulations.

Maxwell laws of electromagnetism were once described by 20 equations. Maxwell’s contribution to science in producing these equations really lies in the correction he made to Ampère’s circuital law in his 1861 paper On Physical Lines of Force. He added the displacement current term to Ampère’s circuital law and this enabled him to derive the electromagnetic wave equation in his later 1865 paper “A Dynamical Theory of the Electromagnetic Field” and to demonstrate the fact that light is an electromagnetic wave. Faraday proved it first when he experimentally found the Faraday effect, but physicists of the day wanted a mathematical proof that Faraday’s simpleton approach was equivalent.  Maxwell gave the world this proof and it was later re-confirmed experimentally by Heinrich Hertz in 1887. The Maxwell equations were simplified to 4 by Oliver Heavside by mathematical convention.  What were the other 16 about? They were about these two pictures below.

Do you notice anything similar between these two pictures? These both show magnetic field effects in nature.  The top one is an abiotic magnets effect on iron filings.  The bottom is the result of electric and magnetic fields around predator animals and their prey. 

Gravity is said to be “different” from the electromagnetic force because gravity has a polarity that is always positive.   In other words, for gravitational forces, things with masses always seem attractive and add to one another. What could nature be hiding that might have a negative polarity that could move things apart?

Somatic cells act like a wave of kamikaze support of the germline cells and melatonin  creation in their mitochondria acts like their General. The leptin melanocortin pathways in these tissues protects that cell line by making sure it has light and electronic power to maintain its existence. Somatic cell death requires the creation of a local biological timing mechanism locally, hence, this is why every somatic cell has its own clock mechanism separate from the SCN. This makes time relative in somatic cells. This is why certain organs age faster than others and it is the basis of what defines heteroplasmy in cells. People who are jabbed experience higher heteroplasmy rates depending upon where the jab contents remain persistent.

Might the destruction of a magnetic monopole in DNA be acting in the hydrophobic pockets in DNA and cause the DNA helix to unwind in the opposition of gravity to cause disease?  This question may sound esoteric but to those with diseases from the jab it might be life saving. Why? What if we develop techniques to control this process in DNA? We might be able to delay disease and death in the jabbed.

The presence of magnetic monopoles was described by the 16 deleted equations of Maxwell, but as of 2025 one has never been found to exist in nature.  Therefore people who supported Heavside in the late 1800’s removed many of Maxwell’s original equations that dealt with them.  Many physicists over the last 150 years thought this was unwise.  Paul Dirac was the most influential critic of this turn of events in physics in the 1930s.  He wrote many papers referencing this belief back then but not many have listened to his warnings.

Could it be the source of the negative polarity in things that give a thing a net negative flux?  Recall that all living things have a serious net negative charge.  Also, recall that water goes from a neutral polarity in its bulk state, and then gains its net negative charge in the structured state when UV and IR/NIR light excite water. This structured state exclude protons and creates coherent domains that act like electromagnetic capacitors for crystalline lattice in cellular water that resists hydrophobicity.  When you carefully read Nick Lane’s book (THE VITAL QUESTION)  you see there is a trend in biology to still wonder why life decided to use protons for chemiosmosis across cell membranes in all three kingdoms of life.

Might the answer be because water and sunlight naturally create unbelievable amounts of protons free of an energy charge?  Hydrophobicity can break this effect as the video above showed. This allows DNA to unwind and be copied and under reverse transciption which can cause turbo cancers. This effect would be magnified if SV40 contamination was present. DNA plasmid numbers make the probability of such event go higher. Translation is widespread in annotated noncoding sequences, including untranslated regions (UTRs), introns, and long noncoding RNAs (lncRNAs), especially in contexts such as cancer, aging, and neurodegeneration. 3’UTRs follow the coding sequence of the mRNA and regulate localization, stability, and translation, among other things. Did you know that both Moderna and Pfizer use novel 3’UTRs, never used before?

Unbiased genetic and biochemical screens both identified the BCL2-associated athanogene 6 (BAG6) pathway for mediation of the proteasomal degradation of diverse noncoding translation products.

• BAG6 recognizes a hydrophobic C-terminal tail, a common feature of proteins translated from all types of noncoding sequences. This results from the U-rich nature of the noncoding genome and the strong bias of U-rich codons for hydrophobic amino acids in the genetic code.

The electromagnetic radiation that reaches the Earth’s surface from space as microwave background radiation is believed to be a consequence of the big bang and the evolution of the universe in the very first seconds of its existence. This type of radiation is characterized by its thermal energy distribution as a perfect black body in nature and has a nearly ideal Planck spectrum at a temperature around 2.7 Kelvin, while the maximum of its surface power density corresponds to the wavelength of 272 GHz. The solar radiation that reaches the Earth’s surface has relatively small surface power density around 3 μW/m2 and comprised of distinct frequency bands, so-called atmospheric windows, representing those frequency bands that are not absorbed by the Earth atmosphere. They can be listed as

• radio window—represented by electromagnetic wavelengths starting from 15 MHz up to 300 GHz,
• optical window—represented by electromagnetic wavelengths starting from 150 THz up to 1000 THz.
• microwave window—represented by electromagnetic wavelengths starting from 23.1 THz up to 37.5 THz.Earth’s magnetic field is another natural field originating from the planet’s core that extends to a vast space surrounding Earth, known as the magnetosphere. An essential source of strong electromagnetic fields is atmospheric discharges, known as lightning. Rapid radiation releases accompanying these natural phenomena are characterized by high power densities and high frequencies. In living organisms, electromagnetic fields originate from the transmission of signals in the nervous system and from structures autonomously generating electrical impulses, like the heart (EKG), brain (EEG) because this is where mitochondrial density is greatest.

The visible universe is constituted almost entirely of electrically active plasma that we can observe for a deep reason. It is what life organizes around.

IMPLICATIONS?

Might it be that when sunlight collides with water, the electric and magnetic fields in light waves change water networks physically to structure cells and cause them to become a dissipative state to limit the effects of entropy? Might inducing hydrophobicity be how we re-introduce entropy and the flow of time to cause disease and death? This effect is buried in every vaccine man has ever made because of the added atoms and chemicals. These pollutants are how the bioweapons program changes the light waves in biophotons, which cells need to fight entropy to remain healthy. Could these changes lead to emergent properties in a matter of which we still remain ignorant?

I think so, and so did Dr. Luc Montagnier before his death.

SUMMARY

The gif above clearly illustrates how a two-dimensional surface can be created in 3D.  Might this picture show what our brain does by resolving environmental waveforms? Might it also show us that the contents in a jab can stop the rotation in a 2D surface to cause protein misfolding and disease? I think this is why COVID and COVID dementia (BIDEN) are telegraphing to decentralized thinkers. Using the above picture, we only need a motion in 2 dimensions and not three to create a wave.

Light can be polarized similarly by a single plane of water.  The jab destroys this effect in water by restructuring it. This stops the dots from rotating, causing complexity to drop and ruin the dissipative state in cells. Life needs to remain well to fight entropy loss. Polarized light can be bent using the Faraday effect.  Sunglasses and windows affect light in this way. This effect is an optical, magnetic, nonlinear effect of light.  DeBroglie was hinting at this wave mechanics of all matter in his Ph.D. thesis, for which he later won the Nobel Prize.  The problem for physics back then was that Bohr’s version of quantum mechanics largely ignored DeBroglie’s insights. It is another form of centralized scientific censorship that continues to hide decentralized truths from us.

Everyone knows mitochondria with uncoupled haplotypes release more heat than coupled ones. Have you ever wondered why or what the implications are in a post COVID world?

In all cases, raising the temperature invokes thermal vibrational and entropic effects. This tends to stabilize H+ over D bonds preferentially. I think this is why white blood cells are deuterium bombs and are controlled by hypothalamic signaling.  The hypothalamus neurons are loaded with POMC.  The frequencies of light control the levers that control cell-mediated immunity. If it is from the sun, the control will be reasonable. If man-made, light immunity will not be well controlled, and autoimmunity or viral decline will result. This is where COVID and cancers come from for manufactured viruses and jabs. The fever that results from the action in WBCs occurs, and this reaction systemically has the opposite effect on the blood and tissues. Why? Higher temperatures favor light hydrogen bonding in water networks, allowing infection-fighting to occur.

We know that DDW increases glutathione in cells to improve the local redox and bring infection under control. Most people are unaware specific tissues and cells, like immune cells favor deuterium collection for a profound physiologic reason. Our body has the wisdom built into it how to make proper use of the kinetic isotope effect (KIE) of deuterium and the lower lipid solubility of deuterium to fight infection and stabilize the use of protium over deuterium in certain key places where energy and information transfer is occurring in water networks.

Many skeptics have enjoyed putting words into my mouth about what deuterium fractionation means for living systems. You would be wise never to listen to reports from the lips of ignorant people to try to understand what a wise person says because their reports are filled with concepts that reflect their understanding of the science and not the ideas of the person they are attempting to translate data from.

Skeptics might leave you with the sole idea deuterium is always harmful or helpful. It is not. There is a profound reason deuterium is found in high concentrations in the blood and not in the matrix of the mitochondria. Their descriptions show how they interpret what I have said, or more accurately, what they hear me say and understand about what I said. That shows you their deficits, not mine.

I have not given you the full Monty yet on deuterium for a reason. You have to know how the pieces fit by understanding what happens when the organization of the pieces falls apart when information is lost in the system. Piece by piece to build your wisdom about how nature works in us is the black swan mitochondriac blueprint of how to learn without making significant errors. You have to unlearn the incorrect things you were taught to relearn what you need to know.

Light hydrogen in the mitochondrial matrix of WBCs is likely the off switch for many immune reactions at the B and T cell levels. Mother Nature knew exactly what she was doing when she made our stolen bacteria at our core innovate uncoupling proteins and haplotype variations. The more heat your matrix liberates, the more hydrogen flows easily inside of cells, the better you can handle a ketogenic diet, and the more deuterium you excrete via the ECF compartments to control your metabolic rate. This is done via the uncoupling of proteins in mitochondria.

Deuterium is best kept under tight wraps deep in cell membranes where they can take advantage of its high kinetic isotope effect. When it is entombed inside a cell, it remains unbound to things it loves to BIND too tightly because of the inherent KIE of deuterium. This is why it must be clear of the TCA and urea cycle where C, N, and O are present in high amounts. It also explains why the body keeps it in the blood under the control of the sun via skin irradiation to move it carefully via amide and amine linkages in lipids and proteins destined for cell membrane construction in our cells. This helps you understand why those with low Vitamin D levels are the patients who get ill and die from viral infections like COVID.  When this occurs in a proper circadian context, faster ATP is produced because the spin rate of the ATPase increases, and the TCA cycle performs like a Ferrari and not a Nissan Sentra. The TCA cycle’s spin rate determines the urea cycle’s spin rate because they both share fumarase.

The immune system is by far one of the most complex systems in our body, and it begins with the skin, our largest organ. The skin has its own immune system, which is quite different from our endogenous immune system. The skin — once thought to be a mainly passive barrier — can produce its own antibodies that fight off infections because of the cells it contains.

Within the skin, host-microbiota symbiosis depends on the remarkable ability of the skin to act as an autonomous lymphoid organ; skin commensals induce the formation of classical germinal centers within the lymph node associated with IgG1.

IgG is the primary type of antibody found in blood and extracellular fluid. It controls infection by binding with many pathogen types, including viruses, bacteria, and fungi.

These phenomena are supported by the ability of regulatory T cells to convert into T follicular helper cells. We know they can also transform into NK cells to destroy cancer cells. This is extremely important in jab-associated cancers and cancers linked to low Vitamin D production in the skin.

T cells can eliminate infected or cancerous cells and direct the immune response by helping B lymphocytes eliminate invading pathogens. The skin’s autonomous production of antibodies is sufficient to control local microbial biomass and subsequent systemic infection with the same microbe. Collectively, these results from the PEER literature in December 2024 married my ideas from 20 years ago that reveal a striking compartmentalization of humoral responses to the microbiota or viri, allowing for control of microbial symbiosis and potential pathogenesis. During an infection, viruses invade the integumentary system and your cells in order to reproduce.

• Each cell becomes a virus factory, which eventually bursts, releasing 10,000 new viruses that can infect other cells (adenovirus).
• During an infection, you may have several million viruses in every milliliter of your blood.
• The human body makes use of antibodies to fight disease. You have ~3×10^7 unique antibodies.
• The shape of the antibody determines what it can bind to. Because you have so many different antibodies, almost any shape can be recognized. LNPs in the jabs alter this function.
• After recognizing an invading virus, the cells (B-cells) that produce the individual binding antibody are stimulated to divide. LNPs alter this function.
• Each antibody-producing cell can produce 2000 antibody molecules per second. After 4-7 days, antibody (IgG) is detectable in blood.
• Antibodies bind to viruses, marking them as invaders so white blood cells can engulf and destroy them.
• Until recently, antibodies were thought to protect on the outside of cells. TRIM21 binds to viruses on the inside of cells.
• TRIM21 sends viruses to the cell’s recycling system (via the proteasome), where the virus is destroyed by cell-mediated immunity
• An antibody is 1,000 times smaller than a virus particle (adenovirus). LNPs are quite large and cause anomalous changes in immunity.
• Two antibodies per virus are enough for TRIM21 to send the virus for destruction. We now think LNPs alter this ratio, leading to new phenotypic diseases.
• Understanding how TRIM21 and antibodies work with deuterium in cells is now critical.  Analysis of protein therapeutics is challenging in labs today because of the complexities associated with large molecular sizes and 3D structures. Recent advances in hydrogen/deuterium-exchange mass spectrometry (HDX-MS) have provided a means to assess the higher-order structure of protein therapeutics in solution. HDX-MS focuses on specific applications of epitope mapping for protein-protein interactions and higher-order structure comparison studies for conformational dynamics of protein therapeutics.

IMPLICATIONS? = SHEDDING

Human skin has a major role in sociosexual communication and response. We now need to add that it has recently discovered a crucial immunological role.

Shedding due to the jabs is now a real risk.

Peters et al. found that women with daily close proximity (within 6 feet) to vaccinated individuals outside their household had:
– 34% higher risk of heavier bleeding.
– 28% higher risk of menstruation starting over 7 days early.
– 26% higher risk of bleeding lasting more than 7 days. The authors concluded,
“Our findings suggest possible indirect transmission of ingredients or products of the COVID-19 vaccines, presumably through shedding, from people who received one or more of the COVID-19 injections.”

Why has shedding emerged in a post-covid world? The problem is a different set of microorganisms are essential for health on the skin epidermis than inside the body. After the gut, more viruses and microorganisms live on the skin than anywhere else in the body. These are collectively referred to as the skin microbiota or the skin microbiome. What are called commensals reside on our skin and derive benefits from us. We benefit from them because they deplete nutrients and produce toxic metabolites, preventing the colonization of pathogenic microorganisms. The symbiont microbiota benefits both microorganisms and humans. These symbionts are critically crucial for skin health. Other skin microbiomes important for health include not only the most well-known bacteria, the Staphylococcus family, but also the bacterial genera Corynebacterium, Dermabacter, Brevibacterium, the Malasezzia fungi, and viruses like COVID.

SUMMARY

With COVID, lipid nanoparticle technology has complicated the treatment ideas around the spike protein. We now know that the spike protein stays around for years, causes clotting and amyloid formation, embeds itself in many tissues, and causes shedding in the non-vaccinated.

The jabbed need to remember that the other problem with all the vaccines is that they create DNA plasmids. This finding is now complicated by new science discovered at Northwestern University in 2024.

Northwestern researchers recently made the strange and startling discovery that nanoparticles engineered with DNA in colloidal crystals—when extremely small—behave just like electrons. Not only has this finding upended the current, accepted notion of matter, but it also opens the door for new disease possibilities in a post-COVID world. These extra electrons seem to be able to destroy the fidelity of signals on the skin and inside cells, leading to new diseases that are unknown to modern medicine.

The skin’s immune system must target some of the same bacteria existing both on the skin and within internal body organs. In contrast, in many cases, it must not attack certain skin bacteria or viruses, which, if they enter the body cavity due to a cut or an insect bite, the internal body must attack to keep us healthy. The result is that the skin ignores and attacks a different set of bacteria or viruses than does the internal immune system.  Recent research in 2024 found that the skin produces its own antibodies to help control microbe and viral reproduction. Given that this new, integumentary, immune system was recently experimentally documented to exist, the discriminating “ignore or attack” system must be determined for both the internal body bacteria and the skin bacteria. This adds another complexity level to the immunology system that centralized medicine still does not account for.

For this reason, governments must eliminate the mRNA technology platform from mankind.

CITES:

https://pubmed.ncbi.nlm.nih.gov/11797936/

https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0130687

https://ijvtpr.com/index.php/IJVTPR/article/view/113

“Particle analogs of electrons in colloidal crystals,” was supported by the Center for Bio-Inspired Science, an Energy Frontier Research Center funded by the U.S. Department of Energy (award number DE-SC0000989); the Air Force Office of Scientific Research (award number FA9550-17-1-0348); the Office of Naval Research (award number 00014-15-1-0043) and the Sherman Fairchild Foundation.

Graham, Flora, “Daily briefing: Skin might have an immune system of its own,” Nature, December 2024.

Guglielmi, Giorgia, “The skin’s ‘surprise’ power: it has its very own immune system,” Nature, 16 December 2024.

Gribonika, I., et al. “Skin-autonomous antibody production regulates host-microbiota interactions,” Nature,  11 December 2024.

Gilbert, S.F., et al., “A symbiotic view of life: We have never been individuals,” The Quarterly Review of Biology 87(4): 325-341, December 2012.

Lei, V., et al., “Skin viral infections: Host antiviral innate immunity and viral immune evasion,” Frontiers in Immunology,  doi: 10.3389/fimmu.2020.593901, 6 November 2020; Duvic, M., “Human Immunodeficiency Virus and the Skin: Selected Controversies,” Journal of Investigative Dermatology 105(1): 3117-s121.ci, July 1995.