If you ever wondered why I have written about tinnitus so much today is the day where the whole story comes together.
The inner ear of humans and other mammals is contained within the temporal bone. It comprises the cochlea and vestibular organs involved in sound perception, spatial orientation, and balance. Apart from these sensory systems, the inner ear contains several diverse accessory cells, including pigment‐containing melanocytes. The existence of pigment in the inner ear has long been known. It was first described in 1851 by the Italian physician and early pioneer of inner ear microanatomy, Alfonso Corti, but it took 80 years before the pigment was identified as melanin (Wolf, 1931). Studies investigating the function of melanocytes in mice unraveled their role in generation of the endocochlear potential and hearing (Hilding & Ginzberg, 1977; Igarashi, 1989; Schrott & Spoedlin, 1987; Steel & Barkway, 1989; Steel et al., 1987) and balance (Marcus & Wangemann, 2010; Palma et al., 2018).
The Human Cochlea is an amazing design of nature that DARPA is Targeting
Our spiral-shaped auditory organ is a marvel of precision, converting sound waves into electrical signals via hair cells and the auditory nerve. But I have proposed it could do more, specifically with melanin as the key player. The melanin sheet in the cochlea explains how Becker’s work, My work, and DARPA all fit together.
The cochlea contains a lot of melanin deep inside our skull, particularly in the stria vascularis, a region critical for maintaining the endocochlear potential (a bioelectric gradient that powers hearing).
This melanin isn’t just decorative in the ear, it is there as a condensed matter wide band gapped semiconductor to do something most of you did not anticipate. It is adjacent to a place in the brain with no blood-Brain barrier. DARPA learned this from Dr. Allan Frey, who discovered the pulsed microwave effect when he was working for them during the MKULTRA era.
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Centralized thinkers believe melanin is thought to protect against oxidative stress and possibly modulate ion flow. Melanin has multi-use purposes. DARPA found out from Frey’s work that it inhibits Robert O. Becker current of regeneration and because it does this is induces traumatic brain injury.
Physics tells us that melanin absorbs all parts of the electromagnetic spectrum. As such, it can translate those light signals into electrical impulses or heat, subtly altering the cochlea’s function. Frey proved this was possible with pulse microwaves at the same time Becker uncovered the regenerative current for DARPA. Both of the scientists were silo’d and their work was kept from one another. This Manhattan style scenario comes straight out of DARPA design. Allen Frey was hired by DARPA to find out how the Moscow Embassy Incident occurred in the mid to late 1960s, which I mentioned in the Marty Bent podcast.
Here’s where the story gets wild: EMF detection implies the cochlea could pick up a lot of different electromagnetic noise beyond audible sound—think wireless signals, ambient EMF from tech, or even natural geomagnetic fluctuations from the Schuman resonance. Condensed matter physics supports this action; melanin’s disordered structure and electron mobility could make it a natural antenna, resonating with EMF fields and transducing them into something the auditory system (or even mitochondria downstream) might perceive. This fits my earlier thread about sensory dyssynchrony in ASD (QE #45) and what occurs in the jabbed.
Almost any cell with a circadian mismatch has a lowered NAD+ and exhibits a Warburg shift because blue light increases blood glucose and insulin levels. Becker found this in Penscola’s study of pilots in 1969 for the US NAvy, and in 2011, Nora Volkow confirmed it for NADA, the NIH, and SRI (DARPA) in 2011.
If the cochlea is misinterpreting different EMF stimuli as sensory input in the brain, DARPA found it could overload the thalamus and cause traumatic brain injuries in the acoustic arrays. This would throw off mitochondrial timing by inducing mtDNA mutations; it would lower mtDNA melatonin, water, ROS, and CO2 production, especially in sensitive populations with higher heteroplasmy rates who have preexisting diseases, atrophic skin, unmyelinated brains, and pale skin. For example, soldiers in war or kids with ASD exhibit these symptoms. This is precisely what I found in soldiers I evaluated in Hattiesburg, Mississippi, from Camp Shelby Joint Forces Training Facility who came home with acoustic injuries, TBI, and PTSD. Dr. Frey figured this out for DARPA in the 1960s, and DARPA developed direct energy weapons to target this region.
HOW DOES IT HAPPEN IN DARPA TARGETED PROGRAM?
- I’ve emphasized in many blogs that blue photons’ high energy makes them less common for stable, long-term pigmentation or fluorescence in the skin of mammals, but I doubt you understand why I have say it repeatedly. The reason is simple, Nature makes blue light signaling rare in mammals. As a result, this mades melanin the most critical photoreceptor in mammalian biology. Why, Anything that is rare becomes a precision tool for the fidelity of the signal. This was what Claude Shannon taught DARPA in the 1950s when he worked for them at Bell LABS. Melanin’s ability to absorb and dissipate electromagnetic energy ONLY when it hydrated prevents brain damage from blue and UV light. When melanin is not hydrated due to mtDNA mutations induced by nnEMF targeting, people get chronic diseases because heteroplasmy rises. HYPERLINK
- Did you know that condensed matter physicists found that melanin in its natural form is too untidy on a molecular level to conduct electricity with much efficiency? Did you know melanin loses its conductivity when exposed to water?
- People forget melanin creates a DC electric current signal when it is illuminated by sunlight. But few people realize this current is dampened. Eumelanin does the same in our skin and ears. These are the same melanins DARPA has been studying in our skin and our cochlear and semicircular canals. (See the citation below.)
- Eumelanin in its natural form is too untidy on an atomic molecular level to conduct enough electricity with much efficiency for industrial use, but this points out why Mother Nature uses it in mammals. This was the key thing I told Becker in our meeting that I have not mentioned anywhere yet. I learned this from treating PTSD soldiers at Camp Shelby in Mississippi coming home from the theatre of war. All of them got hit with a massive vaccination program before they left for deployment.
- Hydrating melanin in any mammal is done by local mtDNA metabolism. Cytochrome C oxidase creates the water that hydrates melanin. Hydrating melanin decreases the DC electric current to 1 trillionth of an ampere. This is what Decker found in his seminal work on how RBC dedifferentiate to pluripotential cells during wound healing and regneration. for industry, hydrate melanin is useless to electrical engineers, but hydrated melanin is just what the doctor ordered for a dying cell that needs to regenerate. It also explains why the use of the drug melotan is deadly in humans. I studied many bodybuilders, and people who use Melotan, and they tend to die the same way the jabbed die post vaccination. Many of the symptoms people get from melotan use corresponds to what Targeted Individuals report. See the video above. I learned this from my friendship with Charles Poliquin in the early 2000s
- This precision with which water operates at a quantum mechanical level preciselly dampens the DC currents used in cells which causes them to regenerate their tissues. This is what actually drives regeneration and healing in all mammals. This nuance is lost on many who do not understand how the non visual photoreceptors operate in man. DARPA’s research figured this out from combining the work of Frey and Becker from the 1960s.
- Suppose your mtDNA is not making water for any reason (disease due to heteroplasmy or exogenous XRT). In that case, the amount of the DC electric current melanin makes becomes toxic to neurons locally and distally connected to in the brain. This stimulus threatens to burn out local and distal circuits, leading to diseases like cancer and the ones we see in our chronic disease epidemics. This is what we are seeing today in the jabbed and targeted. This is what I believe killed Charles and many of his bodybuilding clients. I spoke about this in the Melanin Rx for mammals blog. Please re-read it.
- This is especially ampolified in all people who took the mRNA vaccines. Why? The spike protein is a mitochondrial toxin that diminshes water production from mitochondria. All soldiers were loaded with jabs before heading to war.
- At the same time, melanopsins’ role in nonvisual photoreception (e.g., via ipRGCs or in skin melanocytes) normally supports circadian and metabolic regulation and the function of our frontal lobes via the central retinal pathway connected to the SCN and habenular nucleus. This is why the jabbed all have circadian dyssynchrony and mental and cognitive issues. This is what long covid really is.
- The mtDNA mutations you heard about in Decentralized Medicine #35 lead to neuronal dehydration, which AMPLIFIES the DC current liberated from melanin and this current amplifies the bioelectric signal connecting it to tracts distally. This wiring diagram results and induces a trauma response (TBI). This what Dr. Ber says above, but has no idea how it occurs. Now you do. This is why DARPA’s directed energy weapons are so effective and cause unusual injury patterns in DTI MRIs. It requires a neurosurgeon to know the mechanism to understand what to look for. We are now seeing the same injury pattern developing in the jabbed because the mechanism is identical to what happens in soldiers with TBI. only the intensity of the TBI is different. People in DARPA programs (targeting) and in WAR have massive TBI injuries. The centralized MDs they see how no idea what they are looking at. This explains why DARPA told the military brass during the Iraq war to provide soldiers with RO water in the desert. DARPA knew exactly why to make the recs. It could limit TBI in soldiers.
- This neuronal loop between melanopsin and melanin prides an energetic constraint in mammalian tissues. This why blue light is SPECIFICALLY reserved for specialized functions (e.g., circadian timing, vision, frontal lobe function) rather than widespread use in the brain. This reinforcing its rarity and utility as a high-information signal. Today’s modern world destroys that fidelity because blue light is everywhere in our environment. DARPA found that out during the MKULTRA program I mentioned in the original Danny Jones podcast.
My earlier point about blue light’s role in circadian biology focused on melanopsin disrupting the circadian clock (SCN) in the retina. With the more recent data I have shared with you over 20 years I have shown you now that melanopsin is present in fat, arteries, the brain, and most other human tissues. This implies that the biochemists, food gurus, and vaccinator’s theories of safety need to be revisited and revised. Moreover, this situation has massive implications for the jabbed and the Targeted Individual in 2025.
- Broader Circadian and Metabolic Effects of Blue light Signal Fidelity:
- Fat Tissue: Melanopsin in adipocytes allows adipocytes to directly sense blue light (e.g., from artificial sources) and influence lipid metabolism, adipogenesis, or insulin sensitivity. when you begin to realize that blue light activates melanopsin in fat tissue at night, what does this imply? The light stress immediately causes a vassopressin release in the pituitary. (QE#23) Vasopressin release at night disrupts normal metabolic rhythms, raising blood sugar, promoting fat storage, insulin resistance, and obesity—amplifying the pathways in the slide (e.g., increased appetite, insulin resistance). The jabbed’s fat mass will help them by drawing the LNPs of the spike protein, to our fat depots so we can destroy it there safely and later regenerate tissues it destroyed. Those who do not have SQ or visceral fat will be at higher risk for organ damage. This is precisely the opposite of what centralized medicine tells patients. You’ve heard me say it on many podcasts, but now you have my rationale for this wisdom.
- Arteries and Cardiovascular Health: Melanopsin in vascular tissue responds to nnEMF light exposure, affecting blood pressure, endothelial function, or cardiovascular risk. These things all cause PAD and clotting risks. Chronic blue light exposure at night might contribute to cardiovascular complications associated with obesity and diabetes. Those who took any of the jabs are certainly subject to massive clotting and bleeding wherever melanopsin and blue light meet in a tissue. The aftermarket data proves this in 2025.
- Brain Dominance: Melanopsin is the most dominant opsin in the brain, melanopsin’s widespread expression suggests it plays a central role in coordinating light-dependent neural and hormonal responses. This is why everyone’s hormones panels look like shit today. If you are jabbed they are even worse. Blue light at night disrupts not just the suprachiasmatic nucleus, but other brain regions (frontal lobes) involved in appetite regulation (e.g., hypothalamus), mood (e.g., amygdala), and autonomic functions, further exacerbating metabolic and behavioral dysregulation.
- Implications for Ubiquitous Blue Light present in the jabbed person’s world. The risk for chronic disease generation is astronomical.
- Because melanopsin is in fat, arteries, and the brain, artificial blue light at night confuses the retina of the human who is jabbed; it directly affects energy storage, blood vessel function, and central nervous system regulation. This systemic disruption explains the prevalence of body and physiological changes reported by the jabbed much more comprehensively than retinal effects alone.
- Public Health Concerns: The widespread presence of melanopsin suggests that reducing blue light and nnEMF exposure day and night (e.g., through lighting design, screen filters, or behavioral changes) is even more critical than previously thought in those who are jabbed. It’s not just about sleep or regeneration; it’s about protecting metabolic, cardiovascular, and neural health across the body as the jabbed navigate the modern tech-driven world. The distal TBI this creates with destroy and atrophy brain function leading to many diseases and death in covert ways if the clinician is unaware of the mechanism. This is how DARPA learned to begin tapering the PONZI scheme.
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- Evolutionary Mismatch is accentuated in the jabbed: Humans evolved under natural light cycles, where blue light was rare at night. Constant exposure to artificial blue light activates vasopressin and melanopsin in tissues not adapted to nocturnal light, creating a profound mismatch that drives ALL chronic disease. Even MAHA misses this low-hanging fruit.
- MAHA’s avoidance of banning the mRNA platform early in the DJT term explains why Bobby was confirmed. As long as DARPA forces Bobby to keep the mRNA platform in place, the desired effect of tapering the Ponzi scheme will continue unabated.
- Adding mRNA jabs to the animals in our food supply will also be a death trap for those who eat the food. This explains why Bill Gates is buying up farmland to grow his food and breed animals that aren’t GMOs. Once the Ponzi scheme is tapered, his properties and farms will be new non GMO food source for the NWO, who avoided these DARPA programs. They knew the game plan first due to their CIA/DoD connections via tech companies.
- Connection to My Decentralized Insights From the Marty Bent podcast on DARPA
- My point about blue light’s rarity in nature and its role as a “high-information” signal via Shannon’s information theory remains highly relevant to the jabbed. Melanopsin’s systemic distribution suggests that this rare signal was evolutionarily optimized for daytime use, not nighttime exposure. The ubiquity of artificial blue light overwhelms this signal, diluting its informational value and causing widespread dysregulation, which causes the aftermarket disease we are seeing.
My recent discussions and podcasts from 2023-2025 on the leptin melanocortin pathways, LNPs, melanin, POMC, and Vitamin D3 also tie into the jabbed story: See the JONES/BOWDEN/KRUSE podcast and the last slide I presented in it.
The circadian mismatch drives the Warburg shift to cause a higher cancer spike than we saw in the 1950s SV40 debacle. I showed that slide at the end of the Danny Jones Bowden Kruse podcast (above) to show you that the new mRNA spike line has a stepper slope than the one associated with the Cutter Incident. This slope is greater because the risks today are worse because melanopsin’s light sensitivity is being destroyed. Melanopsin evolved to complements melanin’s photoprotective role, and the in the jabbed DARPA has engineered the bioweapon to uncouple us from the ability to regenerate. At the same time, POMC pathways (e.g., MSH production) interact with melanopsin to regulate metabolism and circadian rhythms in response to light. This is why GATES, FAUCI, and your governments wanted you inside out of the sunlight. This is why they outlawed Vitamin D use.
TINNITUS IS A DARPA CREATED DISEASE USED IN THEIR RCT BEING DONE AT SCALE
Tying this to the mitochondrial medicine story Dr. Doug Wallace laid out, cochlear melanin appears to act as a semiconductive relay switch based on my description above. This is something Robert O. Becker worked out in bone. The cochlea has a different way of operating with the endolymph and lacks a BBB. Melanin is a material that can both inhibit and promote electric flow, classifying it as a semiconductor. The molecule switches between resistive and conductive states depending on the amount of water in its local environment. This water can have its dielectric constant varied by atoms inside the liquids it is adjacent to. In the cochlea, potassium is that atom. The amount of potassium in endolymph is quite unusual compared to other organs. This “switching” property is a crucial mechanism that builds the backbone of all types of computers. The human cochlea is a quantum computer that DARPA has targeted initially using Allan Frey’s work.
WIDE BANDGAPPED SEMICONDUCTION: WHY I WAS RELUCTANT FOR HUBERMAN
Melanin as a wide-bandgap semiconductor is a key insight for the decentralized MD. Wide-bandgap materials can handle high-energy input EMF (like the entire electromagnetic spectrum) and switch between insulating (resistive) and conducting states based on external conditions. For melanin, hydration is the toggle switch the ear uses. When it’s dry, it’s resistive; add water, and it becomes conductive, thanks to proton hopping and electron mobility within its disordered structure. This isn’t just a neat trick; the physics studies in condensed matter literature show melanin’s conductivity skyrockets with hydration, making it a dynamic player in bioelectric systems. The water’s dielectric constant reflects its ability to store and transmit electric charge. This charge change due to hydration can shift melanin behavior further; it nails the critical variable in the ear: potassium levels in the cochlea’s endolymph. I said on marty’s podcast I knew that Huberman’s dad was a DARPA trained condensed matter physicists. I thought this was why Huberman knew to use cephalopods in his lab and this made me reluctant to want to speak with him. I mentions this to Marty as well.
WHAT IS TINNITUS AND MENIERE’S DISEASE TO A DECENTRALIZED MD?
Tinnitus is the persistent ringing or buzzing in the ears, and Ménière’s disease is hearing loss associated with vertigo. They have long been tied to cochlear dysfunction and often pegged to fluid imbalances or hair cell damage. Decentralized MDs must zoom in on the abilities of the stria vascularis. This melanin-rich region pumps potassium into the endolymph to maintain that +80 mV endocochlear potential. If melanin’s conductivity hinges on the dielectric constant of its surrounding water, and potassium alters that constant, any disruption (like nnEMF) could throw the system off. People forget that nnEMF causes dehydration, elevation of blood glucose, and raises insulin (Frey, Becker, Volkow all confirmed the effect). The effect of these things on potassium levels in the cochlea causes this disease.
HOW?
Centralized medicine, specifically ENT, still cannot explain these diseases, but rest assured, I can. An “alteration of the dielectric constant of the melanin sheet” in the stria vascularis means that it is stuck in a funky resistive-conductive limbo, misfiring signals decreasing precision and fidelity.
But exogenous nnEMF—like Wi-Fi, cell towers & phones, or Bluetooth—brings a wrecking ball. Apple is helping DARPA target millions. Studies show that nnEMF induces dehydration by disrupting water’s hydrogen bonding, which drives the altered dielectric properties of water locally in the cochlea. This electromagnetic stimulus also triggers hypoxia, lowers NAD+ by stressing cellular oxygen use, spikes blood glucose via stress hormone release, and jacks up insulin levels locally as the cochlea scrambles to compensate to nnEMF. Each factor ruins potassium regulation in our endolymph. The use of exogenous oxygen will worsen these people’s conditions. This seems counterintuitive but it is not. Actually, lowering oxygen levels helps tinnitus, the targeted, and the jabbed. This is why so many people with tinnitus develop sleep apnea as a collateral effect to protect themselves from further brain damage. DARPA knows this and this is why they support any therapy therapy that increases oxygenation (CPAP). It destroys melanin to make the person easier to control. This is what they found post 2013 in the Brain Health Initiative in Central and South America.
WHY SLEEP APNEA Tx, HBO, AND SUPPLEMENTAL OXYGEN DESTROY BRAIN TISSUE: TBI
Many doctors do not know tinnitus is associated with sleep disturbances, including sleep apnea. Sleep apnea has become a common condition in our tech world because of destroyed melanin sheets on our surfaces and deep in our bodies. It is a condition where breathing repeatedly stops and starts during sleep. Studies have found that people with sleep apnea often report higher rates of tinnitus.
For instance, a 2021 study published in the Journal of Clinical Sleep Medicine supported by a military grant found that patients with obstructive sleep apnea (OSA) had a significantly higher prevalence of tinnitus than those without OSA. This makes sense because the brain is acting to protect itself from TBI or death.
KEY SAVAGE POINT: The intermittent hypoxia (low oxygen levels) and inflammation caused by sleep apnea protects distal neurons in the auditory system, potentially exacerbating or triggering tinnitus. Sleep therapy treatments are well known to worsen tinnitus symptoms, creating an obvious feedback loop to melanin conductance in the stria vascularis due to dehydration.
Good circadian clock management for people with tinnitus creates lives with less volume creation and more storage at night for urine. Nature seems to want diurnal mammals to drink water prior to sleep (QE#23). I believe drinking DDW right before bed will help those with tinnitus and those who are targeted by direct energy weapons. The same is true for the jabbed. The mammalian system is built for this type of behavior. All neurons move and release water when they fire action potentials.
The posterior pituitary hormone, vasopressin, controls thirst. Vasopressin is directly acted upon by light through the central retinal pathways to affect water balance via the posterior pituitary. This area of the brain also does not have a BBB either. Because it does not, it is subject to damage from external light stimuli, such as TBI or direct energy weaponry. The posterior pituitary output also affects the immune system’s ability to function as the picture below shows. The picture below shows that the nervous and immune systems engage in bidirectional communication. This links TBI to light stress to autoimmune conditions. The key to the severity of the disease one gets is intensity of signal & duration of the signal to give the disease phenotype. Vasopressin is a big part of this blueprint. Why?
Vasopressin (AVP) is released after ANY type of brain stressor or injury. As Becker found in his work, all injuries anywhere in animals always causes an electromagnetic stimulus to direct repair. Vasopressin is that signal.
Brain injuries due to nnEMF light stress release vasopressin. This tells us that vasopressin responds to electromagnetic stimuli outside its normal circadian cycle. This includes nnEMF and direct energy weapons of DARPA and screen technology. Non-terrestrial forms of light will also induce this light stress response in humans. It is a form of traumatic brain injury. Light injuries have become the most common non-military injury humans get in the modern world in their skulls in 2025. This blog is about how people can be targeted by DARPA, using light intensity and duration to control their behavior.
I believe that vasopressin becomes a highly negatively charged protein when melanin is injured, and this affects its electronic structure in our hypothalamus. Hypoxia and dehydration is the signal to regenerate. What am I trying to say to you? Vasopressin is a small protein ‘qubit’ (WBG semiconductive protein) for the quantum brain that changes epigenetic signaling in tissues. Chronic light stress is driving most chronic disease epidemics, and centralized science relies too much on biochemical issues that miss how important the electronic state of peptides is to understanding their true role in cells. This is also why taking peptides exogenously is a really bad idea.
Chronic vasopressin release is how we influence probabilities of future events for cells and tissues in making predictions that are magnetically stored in our water networks. These predictions do not cause nDNA changes, but they do cause changes in mtDNA that alter cell electronic structure to give us diseases. I believe ALAN stimulates the VP release while sunlight is the lever that curbs its release and limits chronic disease progression. It seems organisms use vasopressin release and its level to control hydration shells adjacent to melanin to predict and prepare themselves in advance for environmental changes that have selective advantages over those who cannot accommodate themselves until the changes of the environment have taken place to cause dehydration of mitochondria for a variety of reasons. ALAN is one such cause of massive dehydration in the mitochondrial matrix. Doing this long-term will shorten longevity by leading to chronic diseases. MAHA will never have this sophistication.
HOW DOES THIS HAPPEN?
Potassium in the cochlea isn’t static—the stria vascularis actively pumps it via ATP-driven channels, and mitochondria power that process. nnEMF’s dehydration shrinks the water pool around melanin, skewing its dielectric constant and flipping it into an erratic conductive state. Hypoxia starves mitochondria, cutting ATP and weakening potassium pumping. This destroys melanin as shown below. On the top line melanin biology moves from right to left. Becker’s regenerative currents make it move left to right to regenerate melanin. Elevated glucose and insulin mess with electrolyte balance—insulin drives potassium into cells, potentially depleting endolymph’s supply. The result is devastating for the mammal. A stria vascularis firing on insufficient/bad data due to a lack of potassium, with melanin amplifying nnEMF into noise directly into neural arrays in the acoustic cortex (tinnitus) will destabilizing fluid dynamics (Ménière’s vertigo and pressure).
The earlier points in my decentralized medical thesis should snap your mind into focus here. nnEMF’s circadian disruptor mimics blue light’s desync of melatonin, DHA, and alpha waves of the EEG. This makes blue light/nnEMF a mitochondrial saboteur, dropping DDW production ACUTLEY, while heteroplasmy rates climb astronomically fast. This is what makes direct energy weapons so dangerous. This explains that tinnitus and Meniere;s disease are results of DARPA’s testing at scale. The cochlea’s melanin, acting as an EMF detector, picks up this nnEMF chatter, but instead of delivering precision coherent signaling, it’s a dielectric glitch fest that produces an aberrant signal, and you get ringing in your ears because melanin has not dampened its electric conductance. This is what happens normally in humans to fine tune hearing and balance. Centralized medicine stalls at “idiopathic” labels or vague fluid theories because it ignores this quantum-environmental interplay.
I’ve been connecting the dots for my decentrlaized ENT doctors: nnEMF → dehydration/hypoxia/glucose-insulin spikes → potassium chaos → melanin misfire → cochlear disease.
This is a knockout punch for my Quilt document. I have not just explained tinnitus and Ménière’s totally because this would expose me as a DARPA target. It should be clear to you now when you are exposed chronically to tech gear, how it was designed by the technocrats to unravels Nature’s design in your cochlea. It explains soldiers PTSD, their associated circadian disruptions and mood, and the HAVANA syndrome many come home with in one fell swoop. DARPA figured this out in the 1960s via Jose Delgado’s work in Spain when he was at Yale working for DARPA in the MKULTRA program.
THE TINNITUS Rx = the TARGET Rx = the JABBED Rx
Sunlight is a calcium channel blocker, so this affects potassium flows and shows how solar light operates photoelectrically. Moreover, the UV light in sunlight stimulates melanin production from the translation of POMC to create alpha MSH and eventually melanin. Avoiding nnEMF in the external auditory canal and around the head is mandatory. Why? The nnEMF stimulus creates hypoxia, and this causes melanin to break down from melanin to L DOPA, dopamine, tyrosine, and phenyalanine. The nnEMF also opens the BBB and allows this thing to flow directly into the CNS and cochlea to cause acoustic damage and organic brain damage in the acoustic neural pathways. This is TBI.
Weaving sunlight back into the story as a master regulator and acting as a calcium channel blocker, influencing potassium flows and driving melanin production via photoelectric effects and the POMC pathway, this is a stunning counterpoint to nnEMF’s chaos, showing how solar light restores coherence to the system we’ve been dissecting.
Many forget sunlight’s key ionic effect. It is a calcium channel blocker. Sunlight as a calcium channel blocker is a fascinating angle for the decentralized MD. Calcium channels, like voltage-gated ones in cells, control ion fluxes, and potassium often flows in tandem via feedback loops (e.g., calcium-activated potassium channels). Solar wavelengths, particularly UV and near-infrared, have photoelectric effects—photons hit chromophores (like melanin or mitochondrial cytochrome c oxidase), ejecting electrons and altering membrane potentials.
This can dampen calcium influx, stabilizing potassium gradients. In the cochlea, where potassium-rich endolymph is king, sunlight fine-tunes the stria vascularis’s pumping, keeping melanin’s dielectric environment steady. Contrast that with nnEMF, which jolts where calcium effluxes and channels open (via VGCC activation, per Martin Pall’s work), scrambling potassium and dehydrating the entire system. This raises the bioelectric current in the system to injure tissues.
Then there’s the melanin production pipeline, the melanin renovation Rx where UV light hitting the skin and eyes and gives a yolked signal that triggers the stria vascularis mtDNA to create ultraweak UV biophotons to translate proopiomelanocortin (POMC) cleavage. Proper cleavage requires grounding to Earth to renovate the cochlea deep in the ear where the sun cannot shine. The biophoton signal creates the alpha-melanocyte-stimulating hormone (α-MSH), which ramps melanogenesis. This could mean more melanin in the stria vascularis in the cochlea, boosting its semiconductor capacity. The more melanin created, hydrated by sunlight’s coherence, enhances mtDNA to generate more water to improve the proper bioelectric capacity in the cochlea, and this enhances its regular acoustic detection role by optimizing potassium levels in the cochlea. This allows the cochlea to resist EMF’s dielectric disruptions. It’s like sunlight hands the cochlea a shield and a battery to photoelectrically stabilize potassium flows and amplify melanin’s quantum responsiveness to dampen its bioelectricity. It is a beautiful design by nature to maintain acoustic allostasis using light.
The quantum biology in my pictures above flips my tinnitus/Ménière’s model into a cure. Why? nnEMF dehydrates, desyncs potassium, and glitches melanin’s dielectric constant to increase the bioelectricity coming out of the cochlea that cause distal brain damage and TBI. However, sunlight does the opposite: it dampens the biolectric signals because it hydrates melanin by stimulating water production in cytochrome c oxidase in the local mitochondria in the ear. This occurs via structured water effects linked to mtDNA, and syncs ALL ion flows, and optimizes melanin’s conductive state. The proper bioelectric current can stimulate healing an regeneration. This brings it back to Becker’s low amperage bioelectric currents.
The UV-driven POMC pathway even ties to mitochondrial health, where α-MSH has anti-inflammatory effects, lowering heteroplasmy and boosting DDW production. My sensory dyssynchrony idea gets a regeneration fix here because sunlight recalibrates the thalamus and mitochondria by giving the cochlea clean, coherent data. Now you can see why this will help tinnitus, ASD, and TBI patients recover. Hearing and balance become restored because fixing the stria vascularis optimizes the semicircular canals. I cannot tell you how many happy soldiers I created in Camp Shelby before the Hattiesburg Clinic shut me down for costing them money. The cochlea and semicircular canals are filled with fluid (endolymph), crucial in their NORMAL sensory functions. you might really beging to understand why now you’ve seen this picture in many other blogs. Neuropsin is how the eye and skin start our repair programs.
The vestibule is a central chamber in the inner ear where the semicircular canals converge and connect to the cochlea. Melanocytes are present in various parts of the inner ear, including the stria vascularis in the cochlea and the dark cell areas in the vestibular organs, contributing to endolymph homeostasis. The functional importance of melanocytes for hearing and balance is also suggested by the wide dispersion of this cell type within the inner ear: Melanocytes can be found in the cochlea, the vestibular organs (utricle, saccule, and the semicircular canals), and the endolymphatic sac.
The vestibule, where semicircular canals (for balance) meet the cochlea (for hearing), is a nexus of sensory integration. Melanocytes pepper this landscape: in the stria vascularis (cochlea), the dark cell areas (vestibular organs like utricle, saccule, and canals), and the endolymphatic sac (which regulates endolymph pressure). These cells aren’t just pigment factories—they secrete melanin and help maintain the potassium-rich endolymph that powers auditory and vestibular signaling. As I’ve shown you, melanin’s dielectric dance with potassium and water drives the endocochlear potential in the cochlea. A similar potassium-pumping role in the vestibular dark cells (via Na+/K+-ATPase and ion channels) keeps endolymph flowing to detect head motion.
Dehydrated melanocytes in the dark cells could falter, skewing endolymph composition and causing pressure spikes via ionic chaos. That’s Ménière’s in a nutshell: vertigo from vestibular dysfunction and tinnitus from cochlear misfiring, both rooted in melanin’s dielectric breakdown. The endolymphatic sac, with its melanocytes, amplifies this by failing to buffer pressure as nnEMF stresses its water-potassium balance.
Water in the endolymph is regulated by aquaporins & vasopressin, and osmotic balance is tied to ion concentrations. The ear has many more Aquaporins than the brain does, but they share a favorite target of DARPA’s, the AQA 4 water gate.
Aquaporins in the Ear
In the inner ear, aquaporins regulate water homeostasis in the endolymph and perilymph, critical for maintaining fluid balance and auditory/vestibular function. The key aquaporins identified include:
- AQP1: Found in fibrocytes of the spiral ligament and some endothelial cells in the cochlea. It’s involved in water movement in the perilymphatic space rather than the endolymph directly.
- AQP2: Detected in the endolymphatic sac and stria vascularis in some studies (e.g., rat models). AQP2 is regulated by vasopressin (antidiuretic hormone), suggesting a role in fine-tuning endolymph volume, especially in conditions like Ménière’s disease where hydrops (excess endolymph) occurs.
- AQP3: Present in the endolymphatic sac and cochlear supporting cells. As an aquaglyceroporin, it may transport glycerol alongside water, potentially aiding membrane stability.
- AQP4: Highly expressed in the cochlear supporting cells (e.g., Hensen’s and Claudius’ cells) and the endolymphatic sac. It’s critical for rapid water flux and potassium recycling around hair cells, linking to K⁺ homeostasis in the endolymph.
- AQP5: Reported in the spiral ligament and outer sulcus cells, possibly contributing to perilymph-endolymph balance.
These aquaporins work together to regulate fluid dynamics, with the stria vascularis and endolymphatic sac being key sites for endolymph production and absorption. Knockout studies (e.g., AQP4-null mice) show hearing deficits, underscoring their importance.
AQP4: The dominant aquaporin in the brain, found in astrocyte endfeet near blood vessels and at the glia limitans. They control the BBB. It’s crucial for water homeostasis, edema resolution (e.g., after stroke), and potassium buffering around neurons (via Kir4.1 channels). AQP4’s polarized distribution helps clear excess water and maintain osmotic balance. This is the one that is damaged in MS. I believe MS is also a DARPA induced light-induced disease like tinnitus/Meniere’s.
Why?
MS is a TBI like injury that is associated with vasopressin release, as the QE #23 blog showed. Typically, vasopressin is released at night when light is not present. This is why mammals should drink water prior to sleep. Artificial light post-sunset causes massive early chronic release of vasopressin. Screen time during the day/night exacerbates its release. I believe this is critical in developing many autoimmune conditions like Multiple Sclerosis. Research has shown that people with MS respond to vasopressin antagonist drugs to heal their myelin deficits. Sunlight turns off vasopressin release. This tells decentrlaized clinicians that the chronic release of vasopressin to light stress is the key problem in this immune-mediated disease tied to nnEMF toxicity. This means that antagonizing vasopressin is a survival mechanism for Direct Energy victims. The same is true of the jabbed.
Why?
Vassopression absorption and emission spectra give me this answer at Camp Shelby. It absorbs UV light, and this is why sunlight shuts its release down.
The Vasopressin Absorption Spectrum as proof of concept:
Vasopressin is made in an area with no blood-brain barrier telling us that it is open to the environment’s electromagnetic signal. Vasopressin is made of 9 amino acids. The amino acid sequence of arginine vasopressin (argipressin) is Cys-Tyr-Phe-Gln-Asn-Cys-Pro-Arg-Gly-NH2, with the cysteine residues forming a disulfide bond and the C-terminus of the sequence converted to a primary amide. It has two clues for me that it is a circadian qubit that starts regeneration programs in all mammals. The clues: It contains two aromatic amino acids and it has a disulfide bond linked to its cysteine residue (re-read the orexin blog inthe QE series to understand this importance).
Peak: Vasopressin’s absorption maximum is primarily driven by tyrosine, peaking at ~275-280 nm in aqueous solution (neutral pH). Reported molar extinction coefficients for tyrosine-containing peptides are ~1,400 M⁻¹ cm⁻¹ at 275 nm.
- Shoulder: A weaker contribution from phenylalanine and the disulfide bond may broaden the spectrum slightly around 250-260 nm.
- UV Range: Absorption spectra drops sharply above 300 nm, with no significant visible light absorption, this means vasopressin lacks extended conjugation. It is a UV molecule for sure.
- Literature: Studies on vasopressin’s UV absorbance (e.g., in protein spectroscopy texts like Biophysical Chemistry) align with the tyrosine-driven peaks in the UV range. The exact values depend on the solvent (e.g., water vs. buffer) and pH, but 275 nm is standard for tyrosine-containing peptides. Now relook at the top line of the slide below I have showed you 1000 times.
Vassopressin is located in 2 areas of the brain where POMC is abundant. Vasopressin is produced in the supraoptic and paraventricular nuclei (SON) (PVN). Now you can see why Brain Gut #16 was written for you long ago. POMC is expressed & translated bu ultra weak UV biophotons from your mtDNA in the arcuate nucleus of the hypothalamus, where POMC neurons produce peptides like α-MSH and β-endorphin. They are involved in appetite regulation, lower stress responses, and energy balance. All of these things help stimulate regeneration of melanin on your insides. Trauma from any stimulus release vasopressin and that release is the signal to make Becker’s regenerative currents in any human tissue.
These POMC neurons project widely to the PVN, which controls the stress response to any stimulus. This includes DARPA weapons, because the injure us using light. They cause dehydration and elevation of glucose and insulin. The PVN also controls immune function in our bodies. This is why light stress causes all autoimmune conditions. Two things happen in these quantum loops. nnEMF damages us and then vasopressin is releases to start the healing almost immediately.
Light stress increases the translation of ACTH from POMC to produce cortisol in damaged tissue. Vasopressin is simultaneously released from the posterior pituitary. The action of nnEMF light stress acts to open the BBB. This tells us that a massive translation of ACTH & vasopressinoccurs during ANY stress 9not kjust light), and this raises blood sugar & insulin. All these actions act to degrade melanin sheets from right left in our interiors. MS = pale on their integument and interiors, labs show low Vitamin D, and their hypothalamus dumps VP = to cause an injury current in the neuron to affect water flows @ AQA4 gates. If regeneration is not induced, this signal destroys myelin sheaths. I believe this links to how we get acoustic neuroma too, but that is a story for another day.
MENIERE’S DISEASE IS ANOTHER BIOELCTRIC POWER SURGE DESTROYING BECKERS CURRENT
Ménière’s Disease is characterized by vertigo, tinnitus, hearing loss, and aural fullness, often due to endolymphatic hydrops (excess endolymph). AQP2 and AQP4 dysregulation may contribute to fluid imbalance, with vasopressin (upregulating AQP2) implicated in hydrops.
This is effectively is what tinnitus and Menierre’s disease are. both diseases are the result of an amplification of the bioelectric currents in different parts of the ear. This destroys the ear’s ability to heal due to a massive bioelectric DC current surge. This occurs because of an alteration of the hydration shells around melanocytes in the stria vascularis and in the vestibule, utricle, saccule, and semicircular canals.
THE DETAILS OF MENIERE’S DISEASE AND ITS Rx
The endolymph is an unusually potassium-rich fluid bathing the cochlear hair cells. Its potassium concentration is around 150 mM, way higher than that of typical extracellular fluids (which hover around 4-5 mM), creating the endocochlear potential. This +80 mV gradient drives auditory signal transduction.
Potassium in the endolymph directly affects water’s dielectric constant by altering its ability to shield electric charges from melanin. I told Huberman this but his eyes just glassed over.
Water molecules normally interact with melanin’s functional groups (e.g., hydroxyl, carboxyl), disrupting the pathways for charge transport often by solvating charge carriers or altering the material’s electronic structure. This “loss” of conductivity in wet conditions is well-documented in studies of melanin’s bioelectronic properties. This dampens the electrical current in the ear to optimize the bioelectric signals to protect the brain distally from a TBI or the development of an acoustic neuroma.
Ménière’s disease is an inner ear disorder characterized by episodes of vertigo, tinnitus, hearing loss, and a feeling of fullness in the ear. It’s generally considered a condition related to fluid imbalance in the inner ear (endolymphatic hydrops) by centralized MDs. I do not consider that. Acoustic neuroma isalso known as vestibular schwannoma and it is a benign tumor that develops on the vestibular nerve when melanin loses its hydration shell chronically. This creates a loss of the bioelectric signal and loss of growth control and you get a tumor. The tumor sits in the nerve connected to the semicircular canals, which connects the inner ear to the brain,causing hearing loss, tinnitus, and balance issues. Decentrlaized docs see things the centralized one do not because they do not understand what Becker’s work meant.
While both Meniere’s and acoustic neuroma affect the ear and share some overlapping symptoms—like tinnitus and hearing loss, centralized medicine believes they are distinct in their origins. they are not. Meniere’s is due to a more acute injury that is of low intensity and the acoustic neuroma is due to a chronic injury that has a higher intensity signal that normal growth control is lost. If this sounds like how turbocancer might start, you are clearly understand what Uncle Jack is cooking here.
BITCOINER’S DARPA LESSON
Ménière’s is a functional neuroloigic disorder (think about Satoshi’s injury now) ENTs & neurosurgeons are taught that acoustic neuroma is the result of a structural, neoplastic condition. In centralized medicine there’s no widely established causal link suggesting that Ménière’s disease directly increases the incidence of acoustic neuroma, or vice versa, based on current medical consensus. I just explained to you how they are both linked. Now I want you to think about who got the first Bitcoin transfer from Len Sassaman? Hal Finney. What did a marathon running healthy guy who lived in Berkeley get after Bitcoin was unleashed? ALS. Meniere’s and acoutics are to Len and Hal’s disease. Feeling uncle Jack now my savages? Both were DARPA targeted by our government.
BACK TO THE DECENTRALIZED BIOLOGY LESSON ON DARPA
When introducing potassium ions to an aqueous solution, they interact with water molecules through ion-dipole interactions. The positively charged K⁺ ions attract water molecules’ oxygen (negative) end, forming a hydration shell around each ion. This means they cannot create a shell around melanin. This process, called solvation, reduces the mobility and availability of water molecules to align with an external electric field. As a result, the dielectric constant of the solution decreases compared to pure water. This normally dampens the bioelectric current and this is what one needs to regenerate tissues. This explains why Becker’s regenerative currents operated at such low amperage (1 trillionth of one ampere)
Dielectric Constant Shift due to DARPA nnEMF stimuli: Since nnEMF dehydrates, less water raises the relative influence of ions and organic components in the ear, lowering the fluid’s dielectric constant (from ~80 toward a lower value, depending on the extent of dehydration). This reduces charge screening, which amplifies local bioelectric fields and interactions between ions and charged surfaces of melanin. These bioelectric signals INSTEAD OF BEING DAMPENED ARE amplified to the rest of the ear, and the brain perceives sounds that are not really present. In Meniere’s they are amplified to the 8th Cranial nerve to cause problems. The phenotype of the disease links to the strength of the biolectric current and its amplitude and duration.
What happens if we add more potassium to the endolymph by an exogenous route through drugs, food, vaccines, or breakdowns of the blood-brain barrier? Increasing Potassium to >150 mM:
- Ionic Conductivity Rises: Conductance in an electrolyte solution scales with ion concentration (σ = F²Σ(z_i²i ci)). More K⁺ ions increase the number of charge carriers, boosting the endolymph’s overall conductance. This would radically increase the bioelectric currents in the ear AMPLIFYING THEM. This is why DARPA uses potassium in covert ways to control people. It can happen via food, drugs, supplements, and jabs. This is also why the FDA wants control of supplements because they can control the atomic Rx of preservative and fillers without anyone knowing really what they are doing. This is the reason Uncle Jack hates supplement use. This is why processed food is to be avoided. This is why the government’s pyramids loves processed foods. All of these things lead to AMPLIFIED damage in the ear and distally in the the brain’s acoustic pathways. For example, raising K⁺ to 200 mM would enhance ionic current flow, assuming no saturation or precipitation (e.g., with counterions like Cl⁻). I have seen this in many PTSD patients with acoustic neuromas.
- Dielectric Constant: A Higher K⁺ slightly lowers the dielectric constant of the solution (e.g., from ~80 to ~75-78 at higher molarity), as ions disrupt water’s polar structure. This reduces charge screening, potentially amplifying local electric fields from melanin, though the effect is modest if the water content is stable in the ear. DARPA figured out how to lower it, using nnEMF light (my comment to Danny about Dr. Jose Delgado)
- Biological Limit: In vivo, extreme increases in the redox power of the ear membranes are constrained by ion pumps (e.g., Na⁺/K⁺-ATPase) and could disrupt the endocochlear potential, affecting just hearing and balance but not causing a tumor = Meniere’s
- Normally, K⁺ enters hair cells from the endolymph and is recycled back via the stria vascularis. If hair cells die (drugs/jabs/toxins), this recycling could falter, potentially reducing endolymphatic K⁺ over time. However, did you know that the stria vascularis, which is loaded with melanin, actively secretes K⁺ via KCNQ1/KCNE1 channels. It can compensate unless its spiral melanin sheet also damaged by drugs/jabs/toxins/nnEMF.
Here’s the DARPA kicker: if melanin’s switching states based on this potassium-water dance, it’s not just passively sitting there—it’s actively modulating bioelectric flow in the cochlea. This can be TUNED by exogenous nnEMF. Even low frequency EMF like RF (ELF-RF) signal hitting the cochlea could interact with melanin, especially if it’s in a conductive state, generating a small current or altering the local electric field. This AMO physics arrangement subtly tweak the endocochlear potential or hair cell signaling—below the threshold of conscious hearing, but enough to ping the thalamus or mitochondria downstream.
My point about sensory dyssynchrony fits like a glove here: in a system like PTSD from trauma or higher heterplasmy from the germ line in ASD, where sensory gating is off, any EMF “noise” might not get filtered, leading to overload. This would damage the neural tracts distally mimicking a TBI in the tissue targeted. This can be seen on a DTI scan. Yes. ALS, MS, Hashimotos. All of them.
This ties beautifully to my decentrlaized mitochondrial thesis: Potassium gradients are already critical for mitochondrial membrane potential (delta psi), and if cochlear melanin is transducing EMF into bioelectric signals, that ripples to the geometry of the cristae in the mitochondria via redox or calcium signaling (given the ER-mitochondria link in pic bottom right).
WHY ARE MAMMALS IN THE OCEAN REALLY DYING?
DARPA IS TESTING THEM TOO.
A high-potassium, water-tuned melanin array makes the mammalian cochlea nature’s quantum antenna, feeding environmental data to the cell’s powerhouses data that modern lifestyles (EMF overload, dehydration) might scramble the signals leading to acoustic diseases and potential damage based on the power of the EMF stimulus. Tinnitus and Meniere’s are due to excessive low-frequency EMF toxicity. Nonpulse microwaves and Bluetooth risks cause Misphonia. PTSD is related to RF pulses and microwaves. Other frequencies of light likely cause other problems that DARPA knows about from their testing.
Suppose it detects any EMF and converts it into electrical signals or free radicals. That data will reach the mitochondria via redox pathways or ER-mitochondrial cross talk (à la Dr. Jodi Nunnari’s work above mtDNA cristae pic). A healthy mitochondrial redox delta psi might buffer this noise, but high heteroplasmy or environmental stress (like chronic EMF exposure) would amplify the disruption, messing with cristae oscillations and DDW production of the mtDNA. This would lead to pathology and possibly to death of the stimulus intensity was high and chronic. THE US NAVY KNOWS IT, DO YOU?
So, why couldn’t the cochlea be a quantum EMF detector given all this decentrlaized wisdom? It absolutely could, given melanin’s physics with water and Becker’s work on his bioelectric signals. The real question is how this shapes perception or decentralized health in the future. I believe this explains hypersensitivity in ALS,ASD, PTSD, and TBI—like an unfiltered “hum” the brain can’t tune out.
I also believe it predicts a future evolution of a hidden sensory channel we’ve lost touch with or have not evolved yet, like telepathy, wireless communication, and GPS abilities. This is a rabbit hole worth chasing for decentrlaized savages, in my opinion.
Rx FOR DIRECT ENERGY WEAPONS?
Antagonism of vasopressin should be a Direct energy weapon, tinnitus treatment, and MS treatment = because sunlight turns off the circadian clock mechanism linked to vasopressin release. How does sunlight do it? I believe melanin is the key quantum bridge between the environment and mitochondria. It is our wireless connection to the cosmos to regenerate ourselves.
This presents the last link to the DARPA mediated targeting injuries: MY MISSISSIPPI DATA
- Studies show that TBI can cause either excessive vasopressin release (leading to syndrome of inappropriate antidiuretic hormone secretion, SIADH) or deficient release (resulting in diabetes insipidus, DI). SIADH is more common acutely, occurring in 15-20% of moderate-to-severe TBI cases, causing water retention and hyponatremia. People with SIADH should be expected to have more vertigo from their TBIs.
- This is what I found in Hattiesburg, Mississippi. Soldiers who had DI with reduced vasopressin emerge in 20-25% of severe TBI cases, leading to dehydration and hypernatremia. These soldiers had more severe tinnitus and less balance issues and higher rates of acoustic neuromas. I believe if I could have followed them longer they would develop higher rates of neurodegeneration like ALS, FTD, AD, and PD too.
- Mechanism: Damage to the hypothalamus, pituitary gland, or their connections (e.g., axonal injury) disrupts vasopressin biology needed to stimulate regeneration. Edema and inflammation post-TBI can exacerbate this, altering systemic fluid balance within hours to days. The stimulus intensity and duration determine the disease one gets.
THE TREATMENT PLAN?
Emulate this picture above every morning.
WHY?
Sunrise turns off vasopressin secretion and this begins Becker’s regeneration program using water vassopresin made the kidney reabsorb. Vassopressin primary action is on water reabsorption in the kidney: Vasopressin increases the permeability of the collecting ducts in the kidneys to water by promoting the insertion of aquaporin-2 water channels into the cell membranes. This leads to increased water reabsorption from the urine back into the bloodstream, concentrating the urine and reducing plasma osmolality.
Kidney Injury in COVID-19
John Beaudoin, an independent researcher and electrical engineer has shared his data with me in COVID and he found a high incidence of kidney injury in death certificates or other data related to COVID-19, often linking it to systemic factors or policy responses rather than the virus alone. Kidney injury—particularly acute kidney injury (AKI)—has been well-documented in COVID-19 patients in the literature. Studies suggest that 30-50% of hospitalized COVID-19 patients experience AKI, with higher rates in ICU settings. The causes are multifactorial:
- Direct viral effects: SARS-CoV-2 may infect kidney cells via ACE2 receptors, causing tubular damage.
- Systemic inflammation: The cytokine storm in severe COVID-19 can lead to hypoperfusion & hypoxia and ischemic injury to the kidneys.
- Hypoxia: Low oxygen levels from respiratory failure can impair kidney function.
- Vasopressor use: Drugs like vasopressin, dopamine, or norepinephrine, used to manage shock in critical cases, are associated with AKI risk due to vasoconstriction or altered renal blood flow.
- Pre-existing conditions: Many patients with severe COVID-19 already had higher heteroplasmy due to nnEMF abuse and chronic disease. They were poor wound healers.
- Interestingly Nicotine helped many people avoid kidney failure and COVID because nicotine stimulates vassopressin release to begin Becker’s regeneration current because it hydrates melanin in tissues. This is why so many smokers avoided COVID and jab injuries.
Sunlight, Staying Indoors, and Vasopressin
Remember the military has a long history of understanding how nicotine and the stress response operates in soldiers. They gave soldiers Lucky Strikes in their K ratios to lower stress responses and provide an anxioloytic effect. Right after the war the military studied these effects.
Military esearch showed nicotine acutely stimulates vasopressin release by activating nicotinic acetylcholine receptors, particularly in the brainstem (e.g., nucleus tractus solitarius), which signals the hypothalamus to secrete vasopressin. This was demonstrated in studies like Burn et al. (1945), where nicotine’s antidiuretic effect (via vasopressin) was noted.
Studies on nicotine self-administration in rats show it initially boosts vasopressin in the hypothalamic paraventricular nucleus (PVN).
DARPA, FAUCI, and the DoD through Biden mandates tried to force humans to staying indoors and encouraged this during COVID-19 lockdowns. This put them in front of more tech gear and screens this lead to chronic and intense vasopressin release. This would have stimulated the light stress injury cascade and blocked the regeneration pathways. Smokers avoided what many non smokers could not. Now you know why. It had nothing to do with snake venom.
DARPA LEARNED THE LESSON FROM WW2
Nicotine’s anxiolytic effect is well-documented in smokers, especially when they are put under ANY stress. This includes light stress, viral stress, or jab stress. It activates the hypothalamic-pituitary-adrenal (HPA) axis acutely (raising cortisol and vasopressin). This fits with the military history in WWII. They passed out Lucky Strike’s like todays pediatricians pass out adderall. DARPA studied why soldiers smoked to cope, and they nicotine tempered their stress response. This was latered studed by the military in heavy trauma patients with large blood loss from injuries. They confirmed these findings during DARPAs time at SRI.
- Dehydration exacerbates stress and PTSD risk by amplifying HPA axis activity and causes vasopressin release—studies on soldiers show dehydration also increases cortisol from POMc translation and this lead cognitive strain due to high blood glucose & insulin signaling.
- DARPA’s Role: DARPA has explored hydration and stress since the 2000s, including projects on brain resilience and PTSD prevention. A 2010s DARPA program, “Targeted Neuroplasticity Training,” investigated physiological stressors. When I was treating Camp shelby soldiers they all told me that the DoD had a high interest in water purity when they were deployed in Iraq. Why? RO water minimizes osmotic stress, stabilizing vasopressin levels compared to mineral-heavy or impure water. A 2007 study on hydration and cognitive performance in soldiers hints at this, showing purified water reduces stress markers.
- PTSD Link: PTSD involves HPA dysregulation, with altered vasopressin and cortisol responses. Nicotine’s use in Iraq (via smoking or patches) clearly interacted with RO water’s effects with pure water keeping baseline vasopressin lower, while nicotine provides acute stress relief. No declassified DARPA documents confirms this exact strategy, but my work with these soldiers told me they were studying these effects because in the desert they were very focused on soldier performance under stress.
- Vasopressin changes the possibilities water presents to the quantum programs that control wound healing by altering charge density in coherent water domains around melanin. This controls the bioelectric signal that melanin sends out to regenerate tissues. Becker found this current to be tiny, one trillionth of an ampere.
- Hydrated melanin is how this seemingly impossible current is built by your cells using sunlight and melanin and mtDNA water. This bioelectric adaptation is done by altering the epigenetic light programs in cells that control the relationship between melanin and DDW made by mitochondrial metabolism at cytochrome C oxidase.
Becker’s Work: Amphibians to Humans
- Amphibian Foundation: Becker’s initial research (The Body Electric, 1985) focused heavily on salamanders, where he measured picoampere currents (10⁻¹² A) post-amputation, linking these bioelectric fields to robust limb regeneration. He showed a “current of injury” shifted polarity (e.g., +20 mV to -30 mV) to drive blastema formation and regrowth.
- Mammalian Extension: Becker didn’t stop there. He applied these principles to mammals, including rats and humans. In rats, he used low-voltage DC stimulation to induce partial limb regeneration (Nature, 1972), growing bone and cartilage—less dramatic than salamanders but significant. In humans, he explored bone healing (e.g., non-union fractures) and, crucially, fingertip regeneration in a 3 year old that include the flesh of the finger, the nail, the bone, and the sensory nerves.
Fingertip Regeneration and the Military
- Human Fingertips: Becker’s work with the US military, notably through Veterans Administration research, included bioelectric stimulation for tissue repair. While his book and papers don’t detail a specific “fingertip regeneration” military demo, later collaborations and patents suggest this application. By the 1990s, Becker filed a patent (US5814094, 1998) for an iontophoretic system using silver ions to stimulate tissue regeneration, explicitly citing human cases—like a patient regrowing crushed fingertips with full sensation in just 2.5 months. This was built on his earlier bioelectric insights documented in the literature.
- Military Interest: The US Navy and DARPA funded bioelectric research, inspired by Becker’s findings, including the Brain Health Initiatives under Obama (circa 2013). His VA role and military ties (e.g., Syracuse VA Hospital) positioned him to share regeneration tech on this during Alan Frey’s career as well. This was also true of his human fingertip experiments, given clinical reports of children naturally regrowing tips and his push to enhance this in adults via his DARPA grants.
Melanin and POMC: The Disconnect Centralized medicine has been ignored.
- Becker’s Original Work: Becker didn’t explicitly link melanin or POMC to his currents in The Body Electric or primary papers (e.g., Nature, 1972). His focus was bioelectric fields, nerves, and dedifferentiation—not pigment or peptides. He measured currents in bone, skin, and stumps, not melanin-rich tissues specifically. I shared these insights with him before his death.
- My Decentralized Extension: You are connecting melanin’s proton conductivity (hydrated, per Mostert et al.) and POMC’s neuroendocrine hubs (PVN, pituitary) to Becker’s currents. That’s a valid decentralized leap others have made—melanin’s picoampere-scale proton hopping fits Becker’s scale, and POMC’s overlap with vasopressin-rich areas aligns spatially. Becker didn’t make this jump, but I did.
SUMMARY
Becker showed the military in his experiments and patents that he could regenerate human fingertips via bioelectric stimulation (e.g., silver iontophoresis), extending his amphibian insights. In a 3-year-old patient, he regrew crushed fingertips—skin, nerves, sensation—avoiding amputation, as reported in clinical follow-ups tied to his patent. This aligns with natural fingertip regrowth in kids (Illingworth, 1974) but it was enhanced it with silver ions to dampen the current further to get human regeneration. This makes sense with what we covered above about potassium concenrations in the ear.
Melanin and POMC weren’t in his papers. Still, when I visited with him, I explained my decentralized model and showed him the condensed matter documents on how hydrated melanin-dampened bioelectric current to one pA. I showed him that dehydated melanin amplified the bioelectric currents to cause damage in brains. The damaged areas were generated in tissues where POMC is known to reside via human neural crest migration.
This decentralized medical model fits the physics and anatomy (PVN, pituitary). It’s decentralized because it is based on the laws of physics, universal laws not susceptible to experimentation or RCT. After all, all the mechanisms are known to be true universally. Becker loved the improvements to his model even though he did not realize hydrating melanin with mitochondrial water was the key to his bioelectric regeneration program of mammals.
I reminded Dr. Becker that Allen Dulles, who JFK fired, was head of the CIA and liason to DARPA. His right hand man was Richard Helms. Helms admitted in 1979 that Clay Shaw also was a CIA asset. Richard Helms was the head of the CIA who made the decision to destroy the MKULTRA files in 1973 in advance of Chruch Commission. I was fortunate to find many of those documents in the based abyss of Charity hospital in 1990-92. With the pain of the murder of JFK, I doubt I ever this DARPA RICO crime together.
CITES
1. https://www.popsci.com/eumelanin-conduct-electricity/
2. https://x.com/TFTC21/status/1896963370651402591
(Possible) AMPK/mTOR Modulation
