I apologize to the readers in advance. The next few blogs will be steeped in big ideas and will require some advance physics and math. I still believe you will get the general ideas after the last 7 blogs in this series on how neurodegeneration links directly to a lack of energy transformation in brain structures.
The Photobiological Recursive Loop Sets The Tone for What It Means To Be Awake
Introduction: The Quantum Dance of Light and Life
Life evolved under the sun’s full spectrum, harnessing light as a fundamental driver of cellular function. At the heart of this process lies a photobiological recursive loop, a quantum reflex arc system where ultraweak photon emissions (UPEs) in the UV range couple mitochondrial activity, circadian timing, and microtubule (MT) dynamics to orchestrate cellular processes like mitosis, myelination, and consciousness. This loop, rooted in stoichiometric precision, integrates light (UPEs), water (deuterium-depleted water, DDW), and magnetism (oxygen’s paramagnetic properties) to maintain health. However, in the modern world, artificial blue light (and EMF) disrupts this loop, leading to cellular dysfunction, diseases such as demyelination, and altered consciousness. Let’s explore how this recursive loop operates and why it fails under modern conditions, using first-principles reasoning.
The Photobiological Recursive Loop: A First-Principles Breakdown
The recursive loop is a self-reinforcing cycle where UPEs act as quantum signals, linking mitochondria, MTs, and circadian rhythms. Let’s build this theory from the ground up:
- UPE Generation in Mitochondria:
- Fundamental Mechanism: Mitochondria, the cell’s powerhouses, produce ATP via oxidative phosphorylation (OXPHOS). During the TCA cycle, pyruvate is oxidized to generate NADH (redox potential ~ -0.32 V vs. SHE), which donates electrons to the electron transport chain (ETC). This creates a proton gradient across the inner mitochondrial membrane (IMM), with a potential (Δψ) of ~150–180 mV, driving ATP synthesis.
- Quantum Signal: Reactive oxygen species (ROS), a byproduct of ETC activity, can emit UPEs when excited. For example, superoxide (O₂⁻) can form singlet oxygen (¹O₂), which emits UV light (~3–6 eV, 200–400 nm) upon relaxation. Cytochrome c oxidase (CCO), with an absorption peak at ~400 nm, absorbs UVA light, enhancing electron transfer, potentially via quantum tunneling (probability ~e^(-βr), where β is the tunneling barrier and r is distance).
The Integration (Tweets 4 and 7)
Tweet 4: https://x.com/DrJackKruse/status/1613299267703308289
Claim: “The non-linear optical effect in cells is directly tied to the amount of EZ water around the mitochondrial membranes. Mitochondria are the key source of UPE in cells. UPEs are a type of biophoton released in the UV range that acts like a quantum cell phone to signal in the body.”
- Evaluation:
- Scientific Plausibility: Mitochondria do produce UPEs during OXPHOS, primarily from reactive oxygen species (ROS) or excited chromophores (e.g., heme, flavins), with emissions in the UV-Vis range. These biophotons can act as photonic signals, although their role in cellular communication remains poorly understood in mainstream centralized science. It is well established in the biophysics literature. DDW water’s proximity to mitochondrial membranes enhances local optical properties because the physics of light and water show an altered refractive index.
- Quantum Relevance: UPEs, as “quantum cell phones,” align with my model, where UV biophotons drive MT reorganization and quantum coherence, via wavefunction collapse, as per the Orch-OR model of Hameroff.
- Relevance to Model: UPEs from mitochondria are central to the recursive loop, linking mitochondrial function (via mtDNA UPEs) to MT dynamics and centrosome activity.
- Tweet 7: https://x.com/DrJackKruse/status/1613300571741487104
Claim: “The mitochondrial matrix is filled with EZ water. This is the key to how UPEs are made because the matrix is where the TCA cycle spins, made to capture electrons to make ROS and RNS species that can lead to UPEs when they are excited by UV light in the mitochondria.”
- Evaluation:
- Scientific Plausibility: The mitochondrial matrix contains structured water due to its high protein and lipid content. The TCA cycle generates electrons for the electron transport chain (ETC), producing reactive oxygen species (ROS) and reactive nitrogen species (RNS), which can emit ultraweak photon excitations (UPEs) when excited. UV light in mitochondria (e.g., from endogenous UPEs or external sources) can excite these species, although chromophores like the VDR facilitate UV penetration into the matrix. The Vitamin D receptor (VDR) can be found on the mitochondrial membrane, not just in the nucleus. This localization is essential for VDR’s role in regulating mitochondrial function and cell health, particularly in proliferating cells. It protects them from a chronic endosymbiosis we know as cancer. Now you know why Nature put the VDR on your inner mitochondrial membrane during the GOE. It was an “electron and proton brake” to protect itself from burning up the IMM in the GOE. It was also Nature’s best chemotherapy.
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Without this brake, organ damage can also occur. This organ, the kidney was the most damaged organ with the LNP of the jabs. https://www.sciencedirect.com/science/article/pii/S2213231724000387
- Relevance: UPE production in the matrix supports my model, which posits that mitochondrial redox reactions generate quantum signals for MT dynamics not only in the brain but also throughout the body, transferring light information that guides physiological function.
- Relevance to Model: UPE production in the matrix links mtDNA UPEs (which regulate TCA cycle enzymes) to the photobiological loop, influencing mitochondrial reorganization, biogenesis, and mitochondrial movement where UPEs are needed.
- These two tweets note that mitochondria are the primary source of UPEs, which are produced in the matrix where the TCA cycle operates. UVA light absorbed by chromophores like hemoglobin (https://x.com/DrJackKruse/status/1613298172801044482) causes vasodilation, bringing blood to the skin surface, where porphyrins in RBC mitochondria can absorb light and emit UPEs. Blood is also well known to emit UPEs.
- Evaluation:
UPEs as Quantum Signals:
Fundamental Mechanism: UPEs, as UV biophotons, carry quantum information through quantum coherence or entanglement. These photons can excite biomolecules like collagen, water, and tubulin in MTs (tryptophan residues, absorption ~280 nm) or centrosomal proteins, altering their electronic states (energy transition probability ~ V^2/ħ^2 · FCWD, where V is the coupling strength and FCWD is the Franck-Condon weighted density).
Recursive Feedback: Excited tubulin enhances MT polymerization (probability =
BIOPHYSICS OF LEPTIN RESISTANCE
When the recursive loop is not functional for any reason, what do we refer to this as in my decentralized photo-bioelectric thesis? Leptin resistance.
WHAT DOES IT IMPLY? You see the absorption spectra associated with leptin? 220 nm light. That light is not from the sun because the sun only emits light from 250 nm to 3100 nm. Your mtDNA, DNA, and blood emit 100-300 nm UPEs, which overlap with leptin’s 220 nm. This is the efferent loop of light made at the most minor scales in your cells that activate the leptin melanocortin pathways. Without that light being made, you are leptin resistant.
If you use and abuse tech, then you are nnEMF toxic = leptin resistant. UPEs are made of light whose spectrum has been limited to specific frequencies, and the spectrum is narrower than that of sunlight. This makes UPEs more laser-like. Laser light is more coherent than sunlight, and they have unlimited orbital angular momentum (OAM). OAM is what I taught you about in the previous blog series.
Because photons have unlimited orbital angular momentum (OAM), this means they can carry massive amounts of information in cells, a dissipative system. Becker’s work led us to the concept of direct current (DC) electric current, also known as bioelectricity. Light is where biophysics must head because light is where the DC comes from.
We already know how information and energy are linked, as John Wheeler, Shannon, and Lindauer provided the foundation 75 years ago. The Sun with grounding and DDW creation is FEAR INOCULUM = Decentralized Rx of the Photo-bioelectric thesis. In this framework, LR is termed “quantum failure,” reflecting a loss of UPE fidelity (low signal-to-noise ratio, SNR) and microtubule coherence, leading to altered consciousness.
Linkage of Wheeler, Landauer, and Shannon Principles to UPEs and OAM
Wheeler’s “it from bit” principle posits that physical reality emerges from information, where energy and information are fundamentally equivalent (Wheeler, 1989). Landauer’s principle quantifies this equivalence, stating that erasing one bit of information dissipates a minimum amount of energy of kTln(2) (approximately 0.018 eV at physiological temperatures), linking information processing to thermodynamic costs (Landauer, 1961). Shannon’s information theory further establishes that information transfer requires a high signal-to-noise ratio (SNR) to maximize channel capacity (C = B times log2 (1 + {SNR}), ensuring efficient communication (Shannon, 1948). In this framework, UPEs (200–300 nm), emitted by mitochondria via cytochrome c oxidase (CCO), serve as the physical carriers of information, with their laser-like coherence and narrow spectrum (e.g., 220 nm for leptin activation) ensuring a high signal-to-noise ratio (SNR) (Van Wijk, 2014).
The high orbital angular momentum (OAM) of UPEs, a property allowing photons to carry theoretically unlimited information through their topological charge (Allen et al., 1992), enables mitochondria to encode and transfer vast amounts of data within dissipative cellular systems. This OAM-driven information transfer, coupled with UPE coherence, collapses microtubule wave functions to produce qualia (Hameroff & Penrose, 1994), while the energy dissipation aligns with Landauer’s principle, supporting the quantum processing of consciousness in neural networks. Note what I said about this in 2017.
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- Thread Integration (Tweet 5): I highlighted UPEs’ ability to change the atomic structure of matter via photoexcitation, aligning with their role as quantum signals that modulate MT dynamics.
Microtubule Dynamics and Mitosis:
Fundamental Mechanism: MTs, composed of α/β-tubulin dimers, exhibit dynamic instability. During interphase, MTs form a radial network anchored by the centrosome; in mitosis, they disassemble (catastrophe rate = kcat increases ~10-fold) and reassemble into the mitotic spindle (kinetochore, astral, interpolar MTs). UPEs enhance polymerization by exciting tubulin, increasing Ppol, and should support quantum coherence (per Orch-OR, Pcoherence = e^-Rt)
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- Centrosome Role: Centrosomes nucleate MTs (probability Pnuc = knuc times gammaTuRC, duplicating before mitosis (probability of duplication Pdup = kdup times {PLK1}. UPEs clearly act as a quantum checkpoint, triggering duplication. UV light is the stimulus from UPEs
Tweet 6: https://x.com/DrJackKruse/status/1613299900279848964
Claim: “Since blood is in every tissue in the body, this implies the entire body can be affected by UPE light energy in the blood. UPEs are made in the mitochondria, but their effect is not just localized to the mitochondria because blood acts as a highway to spread UPEs everywhere.”
- Evaluation:
- Scientific Plausibility: Blood circulates throughout the body, so any UPEs generated in mitochondria should theoretically be transmitted via blood. Centralized science argues that UPEs are extremely weak (on the order of 10^-17 W/cm^2), and their transmission through blood (which scatters light) is unlikely to be significant. Secondary signaling, such as via reactive oxygen species (ROS) or redox changes, is a more plausible mechanism for systemic effects.
- Decentralized Science laughs at this assertion. Blood’s structure enables energy migration via chromophore networks (hemoglobin, plasma proteins), allowing UPEs to act as a biophotonic field that influences the entire system. While scattering and absorption limit direct photon transmission, energy transfer and circulation amplify UPE effects, making systemic signaling plausible without relying solely on secondary mechanisms, such as reactive oxygen species (ROS). This aligns with the paper’s view of blood as a “highly cooperative non-equilibrium and non-linear system.” The biophysics literature shows otherwise. Biophoton research in blood reveals its decentralized properties with UPEs, June 2003, Indian Journal of Experimental Biology 41(5):473-82
Quantum Relevance: UPEs spreading via blood should act as a quantum signal network based on the paper above, directly influencing MT dynamics and consciousness across tissues. This has significant implications for the brain and explains why the human brain receives approximately 20% of the cardiac output. We need it for our species’ version of consciousness.
Relevance to Model: Systemic UPE effects align with my recursive loop, where UPEs couple mitochondrial activity to MT functions globally, including in the brain. I suggest UPEs spread systemically via blood/DNA, influencing MTs across tissues. Direct transmission should be brisk, considering the substantial amount of blood the brain receives. In contrast, secondary signaling (e.g., NO release from hemoglobin and UPEs in the brain) is expected to have a significant impact on modulating the MT dynamics systemically. Now to tie it all together for you.
Circadian Timing via Rev-erbα/β:
- Fundamental Mechanism: Sunlight’s UVA and blue components (via melanopsin, absorption ~480 nm) entrain the suprachiasmatic nucleus (SCN), regulating nuclear clock genes (CLOCK, BMAL1). Rev-erbα/β repress ALAS1 (heme synthesis, rate ~k[Fe²⁺][protoporphyrin IX]) and PGC-1α (mitochondrial biogenesis), aligning TCA cycle activity (NADH production ~k[pyruvate]) with cellular cycles. The probability of circadian alignment is:
- Thread Integration: Tweet 11: https://x.com/DrJackKruse/status/1613302102415278087
Claim: “This is the key step in how sunlight controls the circadian timing in cells via the TCA cycle. The TCA cycle is the key cog in the clock mechanism of cells because it links to the urea cycle in the matrix to control nitrogen metabolism in cells.” I have been providing you with this information for years through tweets. The tweets were disconnected from this thesis, but I provided you with numerous clues over 20 years
- First-Principles Thinking:
- Sunlight and Circadian Timing: Sunlight’s UV and blue components (via melanopsin in the retina) entrain the suprachiasmatic nucleus (SCN), which regulates nuclear clock genes (e.g., CLOCK, BMAL1). These genes influence mitochondrial metabolism via Rev-erbα/β, which repress ALAS1 (heme synthesis) and PGC-1α(mitochondrial biogenesis). That ferrodoxin lesson I gave comes in handy now. The pieces should be manifesting in your eyes now.
- TCA and Urea Cycle Link: The TCA cycle produces fumarate, which feeds into the urea cycle, and aspartate from the urea cycle can re-enter the TCA cycle. This regulates nitrogen metabolism (e.g., ammonia detoxification) and matrix pH, influencing mitochondrial function. Excessive ammonia levels affect consciousness and cognition. This is why those with liver and kidney disease cannot think well.
- Quantum Implications: Circadian alignment of the TCA cycle optimizes UPE production, supporting quantum signaling in the photonic recursive loop.
- Mitochondrial Function and mtDNA UPEs:
Fundamental Mechanism: mtDNA upstream promoter elements (UPEs) regulate OXPHOS gene expression (e.g., MT-CO1 for CCO), ensuring heme and Fe-S cluster availability (probability =
- This drives ATP production (rate ~k[Δψ][ADP]) and UPE emission, supporting the recursive loop physiology at the core of my decentralized thesis.
Thread Integration: Tweet 9: https://x.com/DrJackKruse/status/1613301472136921093
Claim: “When UPEs are absorbed by EZ water, they can also lead to a change in the molecular structure of water by changing the H-bonding network to control the mitochondrial membrane potential (MMP) that drives ATP production in the cell.”
- Evaluation Of My Madness:
Scientific Plausibility: The absorption of UPE by water has been definitively shown to alter hydrogen bonding, suggesting that significant structural changes are likely. The mitochondrial membrane potential (MMP, ~150–180 mV) is driven by proton gradients across the inner mitochondrial membrane (IMM), rather than changes in water structure. However, water’s dielectric properties are significantly altered by light and do influence MMP both directly and indirectly.
- Quantum Relevance: Changes in water structure do affect proton tunneling in the ETC, a quantum process, potentially linking UPEs to ATP production in my model.
Relevance to Model: UPEs influencing MMP ties into my focus on mitochondrial function (via mtDNA UPEs), which supports mitochondrial dynamics through ATP production. These lessons were the first ones I gave on Patreon in 2017.
- Tweet 10: https://x.com/DrJackKruse/status/1613301745769119745
Claim: “The EZ water harvests the light energy from UPEs in the matrix, and then it is transferred to the inner mitochondrial membrane (IMM) where the ATPase sits to make ATP from sunlight via the TCA cycle.”
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- Evaluation:
Scientific Plausibility: UPE energy transfer to the IMM is plausible in a quantum context, as biophotons should excite chromophores like cytochrome c oxidase (CCO), which has four red light absorption spectra in the electron transport chain (ETC). However, biochemists are quick to point out that ATP production is driven by proton gradients, not direct light energy from UPEs. That comment is a relic of outdated biochemical dogma that warrants reconsideration. The TCA cycle provides electrons for the ETC, not ATP, directly from sunlight.
- Decentralized reality suggests otherwise. Light prevails over the proton-motive force ideas of Mitchell. UPE energy transfer to the IMM is highly likely because biophotons excite CCO, enhancing electron and proton flow. Light directly modulates ATP production via this process:
NO Inhibition and NIR Rescue: NO binds to CCO, stopping ATP production, and NIR light dissociates NO, restoring it.
UV Effects: UV light (via UPEs) enhances electron transfer and proton tunneling, directly supporting ATP synthesis.
VDR Role: UV-mediated 1,25D activates IMM VDR, optimizing ETC function.
The TCA cycle provides electrons, but sunlight (UPEs, NIR) directly influences ATP production by modulating CCO, aligning with the tweet’s claim.
Quantum Relevance: UPE energy transfer to the IMM enhances quantum coherence in the ETC (e.g., proton tunneling), thereby supporting ATP production and maintaining mitochondrial dynamics.
Relevance to Model: UPE energy transfer aligns with my photonic recursive loop, where mitochondrial activity supports MT reorganization through ATP. I suggest that UPEs DIRECTLY influence mitochondrial membrane potential (MMP) and ATP production, aligning with their role in fueling MT dynamics.
- Decentralized reality suggests otherwise. Light prevails over the proton-motive force ideas of Mitchell. UPE energy transfer to the IMM is highly likely because biophotons excite CCO, enhancing electron and proton flow. Light directly modulates ATP production via this process:
- Evaluation:
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- Evaluation Of My Madness:
- Evaluation:
Inter-Mitochondrial Junctions (IMJs):
- Fundamental Mechanism: IMJs, stabilized by mitofusins, facilitate mitochondrial transport along MTs (via kinesin, velocity ~1 μm/s), ensuring localized ATP/ROS delivery to centrosomes. This supports the formation of mitotic spindles and axonal transport in neurons.
Thread Integration Tweet 18: https://x.com/DrJackKruse/status/1613305418586931201
- Claim: “The non-linear optical effect of UPEs in the blood also can control the flow of blood in the body because EZ water in the blood can change the zeta potential of RBCs to control blood flow.”
- Evaluation:
Scientific Plausibility: Zeta potential (surface charge) of RBCs influences blood flow by affecting RBC aggregation and viscosity. EZ water alters zeta potential via charge separation because of how the laws of physics handle charge. Charge is a conserved physical quantity in quantum field theory (QFT). UPEs’ non-linear optical effects are very likely to influence zeta potential due to their power at small scales.
- Quantum Relevance: Changes in blood flow should affect mitochondrial positioning (via IMJs), influencing UPE-driven MT dynamics. The paper above, which utilizes blood photons, supports this claim.
- Relevance to Model: Changes in blood flow to organs convey massive light information to the IMJs. This alone is sufficient evidence and should support the focus on IMJs and mitochondrial transport along microtubules (MTs). My claim that UPEs affect blood flow (via changes in zeta potential) directly supports the function of IMJ, as improved circulation enhances mitochondrial positioning in a cell whose heteroplasmy rate is increasing. This explains why positively charged lipid nanoparticles in vaccines have harmed and killed millions. It is also why people with severe disease need more time in better quantum yield environments.
- Evaluation:
- Claim: “The non-linear optical effect of UPEs in the blood also can control the flow of blood in the body because EZ water in the blood can change the zeta potential of RBCs to control blood flow.”
- Why the Recursive Photonic Loop Operates as It Does
The recursive photonic loop evolved to harness light’s quantum properties for cellular precision:
- Stoichiometric Precision: Light (UVA UPEs), water (DDW in CSF), and magnetism (oxygen’s paramagnetic switch, μ ~ 2.8 μ_B) form a balanced system. UVA absorbed by hemoglobin (energy ~3.1–4 eV) generates UPEs, which couple to tubulin (energy transition ~4.4 eV), enhancing MT coherence. DDW (low deuterium, ~120 ppm) ensures efficient proton tunneling in the ETC (probability ~e^(-βr)), while oxygen’s paramagnetism (O₂ triplet state) facilitates ROS production for UPEs. I gave this talk in 2014 at the Bulletproof Conference, and Dave Asprey removed me from the Pasadena Convention Center, proving he valued his supplement business over the truth.
Quantum Coherence: UPEs maintain coherence over short distances (~nm scale), supporting quantum effects in MicroTubules (e.g., vibrational modes, frequency ~10^12 Hz) and mitochondria (e.g., electron tunneling in CCO). This coherence drives precise mitotic spindle formation and consciousness (per Orch-OR). UPE quality and character light affect everything about humans.
- Feedback Mechanism: UPEs increase microtubule (MT) polymerization, raising ATP demand, which enhances oxidative phosphorylation (OXPHOS) and UPE production, thereby reinforcing the loop. Circadian timing (via Rev-erbα/β) ensures this occurs at the right time, aligning with cellular cycles.
- Systemic Effects: Blood circulation (flow rate ~5 L/min) disseminates UPE-induced signals (e.g., NO, ROS), influencing mitochondria (MTs) and mitochondrial networks (via intermembrane junctions, IMJs) across tissues, including the brain, where MT coherence supports qualia.
- Modern Disruption by Artificial Blue Light
Artificial blue light (400–500 nm) prevalent in modern environments (screens, LEDs) disrupts the recursive loop at multiple levels:
- Melatonin Suppression: Blue light (energy ~2.7 eV) inhibits melatonin synthesis (redox potential ~0.5 V) via SCN signaling, reducing antioxidant protection. This increases ROS, lowering UPE production (PUPE).
- Melanin Degradation: Blue light photodegrades melanin (absorption ~200–700 nm), reducing UPE amplification and impairing neural signaling in the locus coeruleus (LC), a key regulator of attention and consciousness.
- Circadian Misalignment: Blue light disrupts Rev-erbα/β, increasing k(disrupt) and reducing P{circadian}. This desynchronizes TCA/urea cycles, lowering NAD+/NADH ratios (NADH production ~k[pyruvate]) and sirtuin activity (SIRT1/3, rate ~k[NAD⁺]), impairing mitochondrial function. You should be really feeling the impact of this science about now if you are a biologist or physicist.
- Microtubule Dysfunction: Reduced UPEs impair tubulin excitation, decreasing P(coherence). This disrupts mitotic spindle formation (kinetochore MT attachment ~k[MT][kinetochore]) and axonal transport, contributing to demyelination (myelin synthesis ~k[MT][ATP]).
- Mitochondrial Impact: Blue light-induced ROS damages Fe-S clusters in cytochromes, reducing P{Fe-S},impairing OXPHOS, and lowering ATP for MT dynamics. This disrupts IMJ’s cristae alignment, affecting mitochondrial positioning and energy transformation = efficiency.
- Consciousness: Impaired MT coherence alters qualia, reducing first-principles thinking (cognitive clarity ~f(P{coherence}, blinding centralized science to quantum failures (low UPEs, MT incoherence).
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- Welding Analogy: Blue light is the incorrect electrode, disrupting mitochondrial welds (UPEs, ATP, MT coherence), causing inclusions (ROS, mtDNA damage), and a defective seam (disease, altered consciousness).
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- Stoichiometric Precision: Light (UVA UPEs), water (DDW in CSF), and magnetism (oxygen’s paramagnetic switch, μ ~ 2.8 μ_B) form a balanced system. UVA absorbed by hemoglobin (energy ~3.1–4 eV) generates UPEs, which couple to tubulin (energy transition ~4.4 eV), enhancing MT coherence. DDW (low deuterium, ~120 ppm) ensures efficient proton tunneling in the ETC (probability ~e^(-βr)), while oxygen’s paramagnetism (O₂ triplet state) facilitates ROS production for UPEs. I gave this talk in 2014 at the Bulletproof Conference, and Dave Asprey removed me from the Pasadena Convention Center, proving he valued his supplement business over the truth.
My Unified Decentralized Viewpoint
The photobiological recursive loop operates as a quantum reflex arc system, having evolved since the Great Oxidation Event (GOE), during which hemoglobin adapted to harness UV light for aerobic metabolism. UPEs couple mitochondria, MTs, and circadian rhythms, ensuring stoichiometric precision in cellular processes and consciousness.
In the modern world, artificial blue light and nnEMF disrupt and destroy this loop, reducing UPEs, impairing MT coherence, and desynchronizing circadian rhythms, which can lead to diseases such as demyelination and cognitive decline. Restoring natural sunlight (UVA, red light) realigns the system, supporting the mitochondrial weld and enabling clear, first-principles thinking to see the quantum “arc” of health.
SUMMARY
This post integrates first-principles reasoning with the thread’s claims, explaining how the photobiological recursive loop uses UPEs to couple mitochondrial function, MT dynamics, and circadian timing. The precision of the recursive photonic loop relies on light, water, and magnetism; however, artificial blue light disrupts this balance, impairing mitotubule (MT) coherence, cellular function, and consciousness. Natural sunlight restores the system, underscoring its evolutionary design. Now that the ground work is set, on to the HARD PROBLEM OF CONSCIOUSNESS.
STRAP IN. UNCLE JACK IS SLAYING THE BIGGEST PROBLEMS IN SCIENCE IN THIS SERIES.
CITES
All Tweets referenced are below and in the slides.
