Month: February 2025

This blog is also for the jabbed. It integrates many ideas from my work. Please read it as such.

I am attempting to add a hidden layer of nuance here, using a disease I have written about many times as an example. A lack of grounding decreases usual POMC chemokine translation and post-translation modifications of this gene’s products. Remember that all versions of UV light cause POMC translation. Internal biophoton creation from mtDNA metabolism facilitates post-translational cleavage of POMC to get the ten or so chemicals associated with POMC.

I want to frame adrenal fatigue as an example of “melanin brownout” of the non-visual photoreceptor system (neuropsin, melanopsin, retinol), rooted in light system failures across the eye, skin, and brainstem. A lack of grounding exacerbates the brownout because it limits cleavage of POMC peptides. A lack of grounding degrades POMC translation and melanin production (alpha MSH above). This begins with a defective bioelectric signal due to DHA destruction in the membranes of the central retinal pathways and cascades to gut dysfunction via anatomic pathways and POMC destruction below the lipid rafts in the membranes.

BIOPHYSICS OF THE SKIN

The lipid raft’s ability to change determines the reality the mammal faces. When the lipid raft changes, so does the protein function’s physiologic ability. For example, if you have the wrong type of cholesterol in your skin when the sun is strong, you won’t be able to make Vitamin D. Cholesterol has to be sulfated and in the HDL format because those electrons are needed to absorb the 290-320 nm light. Lipid rafts change voltage gate channel operation to do this. Not even standing on the equator naked will raise your vitamin D level when the atomic physics of your system is disordered. It is Biophysics 101. Right now, this is why people in California and NYC have record rates of LDL cholesterol levels, low vitamin D levels, low alpha MSH, flatlined cortisol, and higher rates of skin cancer and melasma. It is fully explainable from the decentralized viewpoint.

Lipid rafts are critical players in the skin’s cellular machinery, acting as a nonvisual photoreceptor that captures light. They are dynamic platforms that organize cholesterol, proteins, and other lipids to perform physiological tasks as the environment varies. Their ability to adapt—shifting in composition and structure directly. This information is then sent to the mtDNA powerhouse, and the mitochondria send the data to the circadian clock gene mechanisms for feedback control regarding the information present in the SCN. If they are not congruent, disease results from alterations in POMC translation and cleavage in the neuroectodermal derivatives.

The Flow Of Nature Rx that underpins this blog

Lipid rafts in the skin act as nonvisual photoreceptors, snagging light (like UVB at 290-320 nm) and orchestrating cholesterol, proteins, and lipids in response. With its electron-rich punch (summer), Sulfated HDL absorbs this light, kicking off Vitamin D synthesis and signaling cascades. That info zips to the mitochondria via mtDNA, ping the circadian clock genes—ultimately looping back to the suprachiasmatic nucleus (SCN), the brain’s master clock. But here’s where the eye steps in: it’s the primary light gatekeeper. Photoreceptors in the retina—rods, cones, and those melanopsin-packed ganglion cells—catch visible light (especially blue, ~450-480 nm) and shoot it straight to the SCN. This sets the central rhythm, syncing the body’s 24-hour cycle.

The skin clock, though, isn’t just a passive follower. It’s a secondary hub, tuned by UV light hitting those lipid rafts. When the rafts adapt—shifting cholesterol profiles or tweaking voltage-gated channels—they send mitochondrial signals that fine-tune local circadian genes (like CLOCK, BMAL1, or PER). This keeps skin functions—Vitamin D production, barrier repair, melanin synthesis—on beat with the eye’s SCN-driven rhythm. If the eye says “it’s day” but the skin’s rafts are clogged with unsulfated LDL, the mismatch screws up mtDNA signaling, and the skin clock drifts. POMC translation in skin cells (think melanocytes or keratinocytes) gets sloppy, leading to issues like melasma or UV damage. Dermatologists use most of their advice to induce UV damage because they create atrophic skin devoid of alpha MSH. Geoengineering does the same.

Now, the skin clock doesn’t stop there—it talks to the gut clocks. The gut’s circadian system, driven by genes in intestinal cells, syncs with metabolic cycles: digestion, microbiome activity, and even immune responses. Light info from the skin—relayed through mitochondrial chatter and hormonal cues like melanocortins from POMC—reaches the gut via the vagus nerve and neuroendocrine pathways. For example, Vitamin D made in the skin (when rafts work right) hits the bloodstream, tweaking gut barrier integrity and microbial balance. If the skin clock’s off—from disordered rafts or low D—the gut clock lags, digestion falters, and inflammation spikes. This is the brain-gut axis in action: the eye sets the tempo, the skin translates light into local signals, and the gut adjusts its rhythm accordingly.

This idea shows that light is not just energy; it’s information, cascading from retina to raft to microbiome and passing the information off at every mitochondria and clock. Its path is determined by Fermat’s law and the AMO physics in the tissues.

A misstep anywhere (wrong cholesterol, poor sulfation, blocked UV) throws the whole axis out of whack. In places like California or NYC, where LDL’s high and D’s low, you’d see it: skin cancer up, gut issues rampant, all from clocks that can’t sync.

THE QUICK Rx

Adrenal fatigue—photoreceptor brownout (skin/eye/DLF, level 5)—blue/nnEMF—DHA oxidizes—melanin dims (level 3)—POMC fades—H⁺ spins falter (level 4)—mPTP stirs (level 1)—gut flops—entropy spikes (1510 nm). Grounding lost—Kruse lecture for Dummies’s deal on Deuterium—latitude codes—grift blinds—truth shines—mtDNA rules (1410 nm)—centralized misses—Quantum Engineering #47/KruseForDummies lecture burns—my 180°—Tetragrammaton pod with Huberman: “Light’s hidden” (Part 2)—light is the arc—nuance ablaze.

 

IMPLICATIONS OF POMC, UV, & Deuterium Overload

POMC translation being driven by UV light is a solid axiom—those lipid rafts in the skin catch UVB, trigger cholesterol sulfation, and signal mitochondria to cue POMC gene expression. This spits out alpha-MSH (melanocyte-stimulating hormone) for pigmentation and supports Vitamin D synthesis. But if the gut’s flooded with deuterium (remember, enterocytes have a 24-48 turnover)—heavy hydrogen from processed foods, water, or environmental exposure—it’s a wrench in the works. Deuterium gums up mitochondrial function because it’s twice as heavy as regular hydrogen (H+). One deuterium atom can stall the proton-pumping machinery—my “1 blocks 96 H+” stat tracks how it slows ATP synthase and messes with electron transport chains. Mitochondria choke, mtDNA signaling to circadian genes falters, and the POMC cascade stalls.

• Alpha-MSH? Probably not. POMC translation weakens if mitochondria can’t relay UV signals cleanly to the circadian clock machinery. No alpha-MSH means no melanin boost—tanning fails, and skin stays vulnerable.
• Vitamin D? Nope. Deuterium-disrupted mitochondria impair cholesterol dynamics in the rafts. Even with UVB hitting sulfated HDL, the downstream conversion to D3 tanks. You’re stuck, even at a sunny latitude.

California Conundrum

This fits your California (or NYC) scenario—people soaking up sun, eyes, and skin in the game. Still, deuterium overload from diet or environment (think heavy water in tap or glyphosate-spiked food) trashes the system. You could live at 34°N latitude, prime for UV, and still not tan if deuterium’s clogging your mitochondria. Your Vitamin D is likely rock-bottom—think 10-30 ng/mL, not the optimal 40-60. Worse, you might tan superficially (some melanin ekes out), but the deeper melanin sheet renovation—tied to POMC’s light-capturing dance at the electronic level—fails as well. Melanin becomes patchy and dysfunctional, leaving you prone to UV damage, melasma, or cancer. Your interior melanin then migrates and is lost, and it happens fast with the jab’s LNP spike, and this is why melanin degrades and turbocancers show up as a Grade 4.

Intergenerational Echoes: The Egg Story

The egg angle seems wild to centralized thinkers but is entirely plausible based on well-known science. A female fetus forms her oocytes in utero, and if her mom (and grandma) lived in a deuterium-rich mess—say, California’s industrial sprawl—those eggs could inherit mitochondrial baggage. Deuterium sticks around, embedding in lipids, proteins, and even mtDNA. It’s not migrating from the thalamus (eggs don’t go there); it’s about where the egg’s machinery heads post-fertilization. The thalamus, a relay hub, needs light-tuned signals from POMC derivatives (like beta-endorphin) for neurodevelopment. Deuterium-heavy mitochondria in the embryo could disrupt that, skewing neural migration—think autism’s wiring glitches.

Autism, Deuterium, and Vitamin A

This deuterium-autism link is cutting—California and New Jersey, hotbeds of horrible nnEMF, processed diets and environmental toxins, see sky-high rates because women there have destroyed their oocytes. Mitochondrial dysfunction from deuterium could amplify oxidative stress, misfire circadian clocks, and derail POMC-driven neurodevelopment. Firstborn males often get hit hardest because maternal Vitamin A stores—key for egg selection and retinal signaling—deplete fast in toxic settings. Light stress (blue or UV without six other colors) burns through Vitamin A faster, disrupting retinoic acid’s role in gene regulation. Later kids might dodge the worst if mom adapts, but if the environment stays toxic, females catch up—autism rates climb across the board.

If you are following here, this is also how turbocancers manifest in the jabbed. The jab is a mitochondrial toxin for mitochondrial respiration.

LIPID RAFT RIFF

In the skin, melanopsin/retinol breaks apart with blue light exposure, leading to EXCESSIVE DHA turnover in the outer mitochondrial membrane where DHA is located. This allows deuterium to leach from the circulatory ECF compartment into the matrix to degrade matrix function slowly. This is associated with hypoxia and falling NAD+. This destroys a cell’s ability to burn fat and use protein properly. This causes a shift in mitochondria redox and changes the bio-photon spectrum that metabolism transforms.

As a result, AMPk pathways have to be used too much. Simultaneously, the circadian mechanism is broken. What controls the replacement cycle of DHA in the retina and skin? THE BAZAN cycle does. The short loop controls the eye’s replacement, and the long loop controls the outer mitochondrial membrane and every other cell membrane in your body. The only membrane in humans free of DHA is the inner mitochondrial membrane because it retains its bacterial lipid profile to make energy from electrons and protons.

The alternative practitioner is a purveyor of false beliefs about this condition because they do not understand light, DHA, mitochondrial, OR MELANOPSIN. The vast majority of people with adrenal fatigue have an altered adrenal stress index because of altered calcium flows into swollen mitochondria in their neurons in their skin and eye, then their frontal lobes, and eventually in their brainstem nuclei at the PVN. Many do not even know that DHA forms complexes with retinol and melanopsin in our cell membranes to control the circadian mechanism in every cell. This region of the cell membrane links to the peripheral clock gene mechanism in front of every nuclear gene in every cell. Melanopsin works using calcium resonance. It is controlled PROPERLY by solar light frequencies and broken by any other form of light we allow.

Back to Adrenal fatigue a disease proxy for teaching: It is a disease that emerges as % heteroplasmy (above) in the skin, eye and brain rises when blue light and nnEMF increase calcium liberation from parts of our cells. Blue light seems to change the bioelectrical signal distally in the entire system and this is where diseases come from. The change in bioelectricity appears to change how mitochondria and their AMO physics operate.

As this occurs, Vitamin A in the plasma drops, which causes circadian mismatches in peripheral clock genes because of how Vitamin A receptors work in the eye, brain, and organs. Adrenal fatigue is an environmental condition that insidiously and chronically lowers DHA in the eyes and brain. It is also associated with low plasma levels of Vitamin A and defective melanopsin signaling. This was the topic of my Vermont 2018 talk. You should listen to that AGAIN sometime.

One of my good friends in radiology had the good sense to point out that people who have autoimmune or other conditions have a physical breakdown of the brain and lose volume when we image them and look for them. All these diseases are linked because their brains are smaller in size (atrophy) as DHA is chronically depleted. There is often a signal change in places in the brain where melanin is present. Obesity and MS are examples of diseases that show this phenotype.

He also pointed out that other conditions can lead to damage/reduction in brain volumes, like sleep apnea/PTSD and mental illness. It is well known that UVR increases the size of the neocortex, showing us that sunlight helps neurologic function and that blue light shrinks many brain areas. These changes can affect the makeup, size, and wiring of your brain and other organs, leading to structural failures in your body by destroying the clock gene mechanism. Blue light demolishes the central retinal pathways as well. Diseases that destroy your CNS will eventually lead to body organ breakdown. Instead of blaming the hardware, organ systems, in particular, he pointed out, maybe we need to look more closely at the “epigenetic software” that controls these organs. This is how the spike protein is destroying people.

Cells communicate & organize using bioelectricity, and the SPIKE PROTEIN SHORT CIRCUITS US.

ATP synthase is truly a marvel of nanotechnology in quantum biology. With its ingenious design and remarkably high efficiency and speed, this fantastic molecular energy turbine stands among the numerous examples of complex macromolecular machines that bear the unmistakable imprints of intelligence and foresight. This should have pointed to biologists like Nick Lane to examine how light and changes in light created such a machine inside cells 3.8 billion years ago.

Okuno’s review paper in 2011 stated that the “unique energy transmission mechanism found in ATP synthase is not found in other biological systems. Although there are other similar man-made systems like hydroelectric generators, F0F1-ATP synthase operates on the nanometer scale and works with extremely high efficiency.”

To achieve high efficiency, you need a unique way to turn the light into bioelectricity with no loss of information or power. Cells contain things that can do that by using red light. Melanin is a solid-state semiconductor key to the mammalian bioelectric power plant. The software is an optical program that runs epigenetics by correctly sensing the light environment transformation of energy to a bioelectrical signal to alter mitochondrial size, shape, and bio-electric status in the membranes of ur skin and eyes.

People seem to forget that the brain has more mitochondria than any other organ. They also forget that the skin is the largest organ. The mitochondria are environmental sensors that are the hardware in the brain that controls the physiologic and psychologic software in our cell membranes. DHA is a massive part of this wiring diagram in the CNS and PNS, as it shows how we control energy flow and information (light) in a cell to create cellular organization.

For example, Dr. Mike Levin has shown in his lab that if you manipulate bioelectricity signals, cells build different body plans, even with the same DNA. This shows us that genes do not determine the body plan. Example: A planarian worm with the same DNA can grow two heads by altering bioelectric fields instead of one. Change bioelectricity → change how cells construct the body or your colony of mitochondria. Your body is an electrical network—cells “talk” via bioelectric signals to shape tissues & organs. DNA = hardware.

Bioelectricity = software. You don’t take apart your MacBook and mess with the circuits to fix a bug—you update the software to repair the defect. Bioelectricity derived from sunlight is the body’s software, you reprogram it with light, and you can regrow limbs, reverse cancer, and control biology itself. (Becker)

MY BIG IDEA

This idea is likely germane to how the ATPase formed in evolution. It has perplexed many biologists for the last 75 years. Molecular machines inside of cells are not Lego bricks. They don’t spontaneously combine to form new machines. My hypothesis has been radical for modern biology because the innovation was unrelated to genetic code changes. I believe the innovation came via a change in light signals that changed the bioelectrical signal. This change in bioelectricity inside cells innovated the change in the structure of the ATPase. The only thing genes do is amplify a metabolic network to make the same biophoton signal to create the same ATPase for 3.8 billion years. This is why it is highly conserved in all domains of life.

For example, consider the decentralized viewpoint of oncogenesis. Cancer cells aren’t damaged—they are disconnected from the bioelectric network that the sun and Earth provide. Without connection, mitochondria act selfishly, treating the body as an external environment. All one has to do is reconnect them to bioelectric fields. They return to normal behavior. No chemotherapeutic drugs or radiotherapy are needed. This message is anti-centralized healthcare, biochemistry, and BigHarma. This idea is 100% quantum biological. This is why it is not accepted as yet. The idea is fundamental to DECENTRALIZED MEDICINE.

This explains why modern biochemistry is impotent to how Mother Nature created the ATPase. How could a complex macromolecular machine like ATP synthase have evolved by natural selection since no other enzyme works similarly? A change in light likely changed the bioelectrical signal, which changed the AMO physics inside early cells.

How do I see it?

Protein translocases are common in living things. This was laid out in Papinikou et al. in 2007. Reasoning from first principles, the conserved head structure of the ATPase, the membrane portion, and the peripheral stalk together could have formed an ancient translocase that coupled ATP hydrolysis to the transfer of RNA and/or proteins across the membrane, with the translocated polymer occupying the place of the central stalk. When the sunlight light source changed, or the biophoton signature at hydrothermal vents varied, this could have created the modern ATPase.

The ability of the F0 domain in the ATPase to cause such specific conformational changes in the active site of the F1 domain via proton-driven rotation requires foresight planning and ingeniously designed interaction. Light is that ingenious mechanic of Nature. Light sculpts life using bioelectricity as its carrier of information. Semiconductors quickly change light into a DC electrical signal. As Nick Lane shows above, bioelectricity gives direct feedback to the AMO physics inside of cells, which causes morphologic changes in the ATPase. Problem solved.

Grey hair (lack of melanin) likely forms the same way the ATPase was formed. Bioelectricity changes in the hair follicle via mitochondrial biophoton likely impact a gene tied to this story. The name of the hair color thief gene is IRF4. It is a gene that acts like a cog in the bioelectric machine in a cellular process that churns out melanin pigment in the hair follicle. Graying happens as follicles gradually stop producing the pigment that gives hair its color, which happens at different rates for different people due to dielectric changes in water mtDNA makes. Various people make different biophoton signals. This gives mammals variable shades of grey in their hair. Even the greying of hair in humans shows that we are not entirely at the mercy of our genes. The study found that environmental factors controlled about 70% of cases of hair graying. Genes were responsible for only about 30% of the population, at least in the Latin American cohort. HYPERLINK

HOW?

I’m plunging you deep into waters of H⁺ networks, dielectric constants, and biophotons—how they shape light’s dance in cells. As your water muse, you’ll need to flow with my ideas, threading your insights on mitochondrial matrix dynamics, pH shifts, and ultraweak UV emissions into a cohesive stream, backed by biophysics. Let’s ride this wave, it is sharp and luminous.

H⁺ Networks and Dielectric Constants

H⁺ protons—aren’t just passengers in water; they’re conductors, fast and fickle. In the mitochondrial matrix, they zip through Grotthuss-like hops, a relay race of bonds, adapting in femtoseconds (Marx, 2001). This network sets water’s dielectric constant—the measure of its ability to screen electric fields. Bulk water’s 78 at 25°C—high, polar, stable. But tweak the H⁺ load, and it shifts. Low pH (acidic, proton-rich) drops it—say, to 60 or lower in inflamed tissues (Stillinger, 1980). High pH (alkaline, proton-scarce) nudges it differently, often lower too, as OH⁻ skews polarity.

Why? Protons mess with water’s dipole alignment. More H⁺ tightens the network, less screening; less H⁺ loosens it, and it’s the same deal. The dielectric constant ties to refractive index (n ≈ √ε_r)— explains how light bends in Nature and your cells. This is FERMAT’s LAW.

Normal water’s n ≈ 1.33; tweak the dielectric, and light’s path warps in cells. I’ve just described how Fermat’s law works in you. That’s tissue optics shifting—light scatters or tunnels differently. Why can’t we accept Pollack’s work? There is an entire thread about that on my website forum. He never tested deuterium-depleted water in his experiments. Deuterium changes the dielectric constant in ways his book never examines. Therefore, most of what he believes is speculation.

Mitochondrial Matrix: A Dielectric Playground where biophotons are made

The matrix isn’t bulk water—it’s a proton-packed cauldron near the IMM, where ETC pumps H⁺ out, and F₀ spins it back. Dielectric’s not static—O₂, exotic atoms (Ca²⁺, Mg²⁺, Na⁺, K⁺, Cl⁻, I⁻), and wide-bandgap players—tune it. Calcium spikes (e.g., mitochondrial uptake) bind water and drop dielectric locally (Fettiplace, 1980). O₂, chugging electrons to make H₂O, tightens H⁺ density—dielectric dips. Should the liquid-metal H⁺ riffing in previous blogs now resonate in your neural circuits? That’s extreme density, magnetic containment, further slashing dielectric, & light bending hard now inside your cells.

This isn’t random—cells sculpt light. Low dielectric (acidic matrix, high H⁺) slows photons and shifts, altering their physiological game; high dielectric (buffered spots) speeds them up. Inflammation & low pH dim the illumination of tissues by turning down mtDNA light transformation; alkalinity tweaks it another way. These are all the things Gerald Pollack missed in his 2013 book. Wide-bandgap atoms (Mg in chlorophyll, I in thyroid) act semiconductor-style, gating electron flow and tweaking light’s fate in your cells.

Biophotons: Ultraweak UV Jobs

Roeland van Wijk’s work (e.g., Light in Shaping Life, 2014) nails it—cells emit ultraweak biophotons, 200-800 nm, peaking in UV (250-350 nm). Not floodlights—10⁻¹⁹ W/cm², a whisper. Source? Mitochondrial ROS (superoxide → singlet O₂), lipid peroxidation, protein excited states. Each photon’s frequency—tied to its energy (E = hν)—has a physiological gig in your cells. The specificity and sensitivity in this game of light blow the centralized ideas in biology to smithereens. UV repairs DNA (Sancar, 2004), signals redox (Tafur, 2010), & it cues clocks and invokes entropy (CRY proteins, Provencio, 2000). Cells specialize—skin spits UV for melanin, creating electrons and a higher dielectric in water, providing optimal signal fidelity in neurons.

Dielectric shifts in water tune this all. Low dielectric (high H⁺) red-shifts biophotons—less punch, more scatter. The high dielectric blues the light. This sharpens the tool and makes it more focused. This light bends more than red light inside cells. Red light does not bend and reduces inflammation & pain in tissues. This stimulates inflammation. Inflammation’s proton flood dulls the signal—chronic disease brews. Matrix O₂ and ions? They dial the frequency and precision jobs for each cell type.

The Flow of Life?

H⁺ networks flip water’s dielectric switch—refractive index follows, & light bends. Matrix atoms and pH sculpt it further—photons morph, & physiological jobs shift. Biophotons—UV whispers—run the show, but only if the medium optics are right. AMO physics is the name of the game. This is why atomic contaminants from vaccines demolish mitochondrial function, causing many new diseases. They are messing with biophoton transformation from matrix to cytosol. No innate flaws; bad light, bad water, & bad vibes skew it.

The Decentralized Clinicians Rx should be about fixing the electrodynamics in the patient’s field—sun, not screens—or the signal for health will be lost.

Next wave—how do we map this dielectric dance in real time?

Prolonged stress of any kind (Brain Gut 16 blog) depletes our cell membranes of DHA, ruins both loops of the Bazan effect, and increases the need for methionine because ubiquitin rates skyrocket. DNA is designed to be quiescent and not active. Sunlight keeps our DNA transcription low. nnEMF/blue raise it. DHA allows our cells to power up electrons with photons from sunlight and helps melanopsin move protons to control melatonin levels and mtDNA heteroplasmy. DHA fundamentally takes light and turns light into electric-mechanical signals in cell membranes everywhere, but especially in our skin and brain. Our mitochondria have two sets of cell membranes. Its inner and outer chemistry is enormous in tunneling electrons. If it does not work, we get a redox shift in our Q cycle’s operability. That cycle moves food electrons from cytochromes 1 to 3 and to 4 and then to the ATPase, where protons are moved from the matrix to the outer mitochondrial membrane. H+ is the favored subatomic particle in this organelle.

Deuterium is typically excluded here and kept in the blood to perform another quantum task. This is why the retina has more DHA in it than any other place in the human brain; it is located on the interface where light from the sun first interacts with the brain’s mitochondria in the RPE of the retina. Visible light has part of it tied to blue light when a prism separates it. Blue light is the part of the spectrum of light that destroys DHA, lowers melatonin, and causes the respiratory proteins in mitochondria to swell and lose their electric charge. Red light does the exact opposite bioelectrically in mitochondria. In fact, cytochrome C oxidase is condensed by red light because it is related chemically to hemoglobin. Both are heme-based proteins. All heme-based proteins’ initial construction steps happen inside the mitochondrial matrix, where the bioelectric current is powerful.

SUMMARY

Centralized biochemistry refuses to accept or believe that heme processing relies on a “bioelectric current” but on biochemical reactions and enzymatic pathways instead. Robert O. Becker’s and Michael Levin’s work have not been imported, and centralized ideas in biochemistry have yet to be updated. Light control of mitochondria is now well established in the biophysics literature and stands in counter distinction to the reality found in the operation of the physics of organisms. For example, UVA light and IRA light control many systems in the operation of cytochrome C oxidase. Biochemistry has yet to explain this paradox. The reason: it is not a paradox. Centralized biochemistry is not foundational to how life operates. Quantum mechanics is.

Cytochrome c has four red-light chromophores and a VDR receptor. The VDR receptor, when activated, dramatically slows electron chain transport. Red light, however, can still process proteins without food electrons with 100% efficiency. The ATPase is a quantum rotator engine for red light in sunlight. The red light spins the ATPase faster to make more ATP available, especially when present with UVA light. This is why AM sunlight has the exact same amount of red light as blue light. Red light is the antidote to the stimulus of blue light, but only UVA once the day proceeds.

IF YOU WANT TO AVOID JAB CONSEQUENCES, these spectra must be balanced to work correctly in the eye/skin/gut. What happens in the skin and eyes determines how the gut operates. This is why we are designed to replace DHA constantly in our retina/skin with excessive blue light hazards. When we do not, we get macular degeneration, cataracts, and glaucoma. When we do get too much blue light from our environment, it is a stressor to the retina, and the protective response from the cornea is to develop cataracts to protect the retina from DHA loss. DHA is a brain/ retina/skin story for humans.

CITES

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3257695/

https://www.sciencedirect.com/science/article/pii/S0092867400814567

https://pmc.ncbi.nlm.nih.gov/articles/PMC9072658/

https://www.nature.com/articles/nrmicro1771

https://academic.oup.com/jb/article-abstract/149/6/655/2182760?redirectedFrom=PDF

https://www.nature.com/articles/ncomms10815

https://threadreaderapp.com/thread/1893468340569677950.html

‘The drug war isn’t what you think it is’ – it is very interesting concept when you begin to. realize the Shamans inside the Amazon have always called ayahuasca, medicine.

The video above is on glioblastoma multiformans. It is the most lethal human cancer we know of in centralized medicine. Nothing kills quicker. In this way SV40 has a lot in common with it. It happens to be the one cancer neurosurgeons are expert in, in terms of treatment options. This blog today is about the decentralized treatment for this cancer linked to the jab. I think these options should be added to any GBM case after my visit to the Amazon in Suriname.

^^^^This is me in the Amazon River system with a Pirana net behind me looking for answers.

Why did I believe forests the best decentralized pharmacies on Earth to look for clues to cure incurable diseases like SV40 intercalation?

Why did I chose Suriname? The picture tells you why I chose it.

THEY HAVE THE LARGEST FOREST OF ANY COUNTRY.

93% of this country is a tropical rainforest.

Did you know most of the forests, are home to plants and animals that cannot be found anywhere else in the world? The same is true with the compounds their soils and water table create out of sunlight and magnetic flux in the magma chambers below them.

So what were the clues I went to Suriname with? What was I looking for?

I was looking for confirmation of why Ivermectin was working on people who took the jab who had turbocancers. Did I find what I was looking for?

I think I might have.

This table above was the treasure map I went to Suriname with. I then found 12 tribes who were treating the jab injured who’s story I laid out here.

https://x.com/dralexisjazmyn/status/1890609382422913290

You might want to listen to the ENTIRE podcast I did with her. I tried to tell some of my high profile clients what I was up to, but none of them wanted to listen to me before I left. I think that might change now.

FOR THE JABBED I BELIEVE WE NEED TO FIND UNIQUE NEVER BEFORE USED CBD’s

Why did I believe the Amazon a key player for the jabbed, especially those with SV40?

Dr’s Mary Sherman treatise made some comments about chemicals that had elements that were related to this class of drugs. She did not know that we had an endogenous endocannabinoid system operational in humans.

The Amazon is 55 million years old and it is where THE unique CBDs are created because of its geology. That is why I went to Suriname. They have CBDs that are unique.

You should visit @cannmed or @cannmedevents to learn more about this topic. That is their Twitter handles.

Cannabinoids exhibit some overlapping mechanisms with ivermectin (IVM) and fenbendazole (FenBen) in cancer treatment, particularly through microtubule disruption, apoptosis induction, and metabolic modulation.

Below are key comparisons:

1. Microtubule Disruption (Similar to Fenbendazole)

•Fenbendazole: Binds β-tubulin, disrupting microtubule polymerization, leading to mitotic arrest and cancer cell apoptosis.

•Cannabinoids: Some cannabinoids, such as CBD (cannabidiol) and THC (tetrahydrocannabinol), have been reported to destabilize microtubules:

•CBD disrupts tubulin polymerization, impairing mitotic spindle formation in glioblastoma and breast cancer models.

•THC alters microtubule dynamics, potentially affecting cancer cell division.

•Overlapping Effects: Cannabinoids may enhance the microtubule-disrupting effects of FenBen.

2. p53 Activation & Apoptosis (Similar to Both IVM & FenBen)

•FenBen & IVM: Upregulate p53, increasing apoptosis via BAX/BAK activation. •Cannabinoids:

•CBD & THC upregulate p53, leading to mitochondrial dysfunction and apoptosis.

•Activation of caspase-3 and caspase-9, triggering programmed cell death in multiple cancers.

•Overlapping Effects: Cannabinoids can synergize with FenBen or IVM to enhance apoptosis in cancer cells.

3. Metabolic Disruption & AMPK Activation (Similar to FenBen)

•Fenbendazole: Blocks glucose metabolism in cancer cells, reversing the Warburg effect. •Cannabinoids:

•CBD inhibits glucose uptake via downregulation of GLUT1 transporters.

•AMPK activation by cannabinoids leads to mTOR inhibition, reducing cancer cell growth. •Overlapping Effects: Cannabinoids mimic FenBen’s metabolic disruption, making them potential synergistic agents.

mTOR inhibition was a big topic in the Quantum Engineering Series and especially in the Melanin Renovation blog.  You might want to re-read it if you took that jab.

4. Anti-Inflammatory & Immune Modulation (Similar to Ivermectin)

•Ivermectin: Modulates immune responses by shifting T-cell and cytokine activity, reducing cancer immune evasion.

•Cannabinoids:

•CBD reduces inflammatory cytokines (IL-6, TNF-α), potentially lowering tumor-promoting inflammation. It also affect the nrf2 pathway. NRF2 detox pathway only is tapped by having redox power present in the cell.

If your cells lack the net negative charge, they will never experience this pathway. So if you are pale and think using CBD is a panacea you’re a special kind of centralized idiot.

•Enhancement of immune surveillance through interaction with CB2 receptors on immune cells.

•Overlapping Effects: Cannabinoids may amplify IVM’s immune modulation in cancer therapy. Key Cannabinoids With Cancer-Treatment Potential

1. CBD (Cannabidiol): mechanism of action

•Microtubule disruption •p53 activation → apoptosis •AMPK activation → metabolic inhibition •Anti-inflammatory effects (reduces IL-6, TNF-α) alters NRF2 signaling.

2. THC (Tetrahydrocannabinol): mechanism is different but synergistic.

• Microtubule destabilization

• Apoptosis induction via CB1 receptor

• Inhibition of angiogenesis in tumors

3. CBG (Cannabigerol):  mechanism is different but also synergistic.

• Inhibits mitochondrial respiration in cancer cells

• Synergizes with chemotherapy

4. THCV (Tetrahydrocannabivarin): mechanism is synergistic

•Reduces tumor cell proliferation

•Modulates AMPK/mTOR pathway Potential Synergistic Treatment Approaches (mTOR again)

•CBD + Fenbendazole: Both disrupt microtubules and glucose metabolism.

•CBD/THC + Ivermectin: Immune modulation + apoptosis enhancement.

•Full-spectrum cannabinoids + Metabolic inhibitors: Combination therapy for aggressive cancers. All of these chemicals help and many of these chemicals have unique chemistry in the Amazon basin and make up most of the magic present in the CBD decentralized pharmacy we need to tap for the jabbed.

My Conclusion: Cannabinoids share several anti-cancer properties with FenBen and Ivermectin, particularly microtubule inhibition, p53 activation, metabolic disruption, and immune modulation. This suggests they could be complementary in cancer treatment, especially in the jabbed. More decentralized research from the forest is needed to explore their combined effects.

We need to map the effects out and some have already begun this.

5. People in places on Earth are already doing this work and I have spent time meeting and talking with them.

Pay attention to IVM/FenBen for cancer as the recent blogs have laid out.

New data is arriving and ties DIRECTLY to my trip to the Amazon.

IVM prevents SV40 promoters from entering the nucleus but blocking importin A/B. This pathway likely has many chemokines that can impact it.

Yes, ivermectin is known to inhibit importin α/β-mediated nuclear transport, which is relevant in the context of SV40 promoters. Dr’sSarah Stewart and Mary Sherman did not know this when they working for DoD and Dr. Ocshner.

The Mechanism:

•Importin α/β Pathway: This transport system is responsible for shuttling proteins with nuclear localization signals (NLS) into the nucleus. Many viruses, including SV40, hijack this pathway to deliver their regulatory proteins (e.g., Large T – antigen) into the nucleus for replication and transcriptional activation.

• Ivermectin acts as an Inhibitor: Ivermectin binds to importin α/β and disrupts its function, thereby preventing nuclear entry of proteins that depend on this transport mechanism. Implications for SV40 Promoters:

•SV40 Promoters: The SV40 early promoter is often used in molecular biology due to its strong transcriptional activity in mammalian cells. However, its transactivation requires the nuclear localization of SV40 Large T antigen, which depends on importin α/β.

• Blocking Nuclear Entry is a KEY: If ivermectin blocks importin α/β, it could prevent SV40 Large T antigen from entering the nucleus, thereby reducing SV40-driven gene expression and viral replication.

Do we already have Experimental Evidence for this?    Yep.

•Studies have demonstrated ivermectin’s ability to inhibit nuclear import of viral proteins from various RNA and DNA viruses (e.g., HIV-1, Dengue, and even SARS-CoV-2).

•SV40 Large T antigen is known to require importin α/β for nuclear entry. If ivermectin blocks this pathway, it could theoretically interfere with any SV40-driven transcription or replication in systems using this promoter.

Certainly, here are some key studies that provide evidence on this topic:

1. Ivermectin as an Importin α/β Inhibitor:

• A study by Wagstaff et al. (2012) demonstrated that ivermectin specifically inhibits importin α/β-mediated nuclear import. The researchers found that ivermectin effectively blocked the nuclear import of proteins dependent on the importin α/β pathway, without affecting other nuclear import pathways. This inhibition also correlated with a reduction in the replication of viruses such as HIV-1 and dengue virus, which rely on this pathway for nuclear entry of their proteins. (http://pmc.ncbi.nlm.nih.gov)

2. SV40 Large T Antigen and Importin α/β:

•The SV40 Large T antigen contains a nuclear localization signal (NLS) that is recognized by importin α, facilitating its transport into the nucleus via the importin β pathway. This nuclear import is essential for the Large T antigen’s role in viral replication and cell transformation. (http://en.wikipedia.org)

These studies collectively suggest that ivermectin’s inhibition of the importin α/β pathway could impede the nuclear import of SV40 Large T antigen, potentially affecting SV40 promoter activity and viral replication.

Does Fenbendazole Upregulate p53?

Yes, Fenbendazole (FenBen) has been reported to upregulate p53, a key tumor suppressor protein, in some cancer models.

Mechanism of p53 Upregulation by Fenbendazole:

1. Disruption of Microtubules:

• Fenbendazole binds to tubulin, preventing microtubule polymerization in a manner similar to colchicine or vinblastine.

• This leads to mitotic arrest, which can trigger cell cycle checkpoints and activation of the p53 pathway.

2. Induction of Cellular Stress & DNA Damage Response:

• Microtubule disruption can cause mitotic spindle stress, leading to chromosomal instability. • This activates ATM/ATR kinases, which phosphorylate p53, stabilizing it and increasing its transcriptional activity.

3.Apoptosis and Autophagy Induction:

• Upregulated p53 can activate BAX/BAK pro-apoptotic proteins, leading to mitochondrial damage and caspase-dependent apoptosis.

• Fenbendazole also promotes autophagy, which can contribute to cancer cell death.

There are 3 dominant phases of synchronized metabolic and transcriptional reprogramming when pigmenting the skin to avoid jab diseases. The melanogenic trigger is associated with high MITF levels and rapid glucose uptake by the cell. Blue light exposure stimulates massive glucose mobilization from ACTH from POMC. The transition to a pigmented state is accompanied by increased glucose channelization to anabolic pathways in biochemistry that support melanosome biogenesis. The H+/D optical switch you heard about in the Kruse for Dummies talk is critical in optimizing this step. You can find this talk in my IG biolink.

I talk about how I do this with pulsed light in the Melanin Renovation blog.

4. Inhibition of Glucose Metabolism

(Warburg Effect Reversal):

•Some studies suggest Fenbendazole reduces glucose uptake by cancer cells, similar to metformin. It might be time to consider this drug for all tech abusers.

• This metabolic stress can further activate AMPK pathway which leadsto p53-mediated tumor suppression. Why did the Brazilian tribes get more sick in my story to Dr. Alexis? They used technology that had enriched blue light. None of the other tribes did.

• This metabolic stress can further activate AMPK pathway which leadsto p53-mediated tumor suppression. Why did the Brazilian tribes get more sick in my story to Dr. Alexis? They used technology that had enriched blue light. None of the other tribes did.

Fenbendazole’s Mechanism for Cancer Treatment
1. Microtubule Disruption (Primary Mechanism)

• Fenbendazole binds to β-tubulin, disrupting microtubule formation.

• This prevents proper mitotic spindle formation, leading to G2/M cell cycle arrest.

• Cells stuck in mitotic arrest either undergo apoptosis or senescence.

2. Apoptosis Activation via p53 & BCL-2 Inhibition

•Cancer cells often overexpress BCL-2, an anti-apoptotic protein that prevents programmed cell death.

•Fenbendazole inhibits BCL-2, shifting the balance towards apoptosis.

3. Disrupting Glucose Metabolism in Cancer Cells

•Fenbendazole has been shown to reduce glucose uptake and ATP production, increasing oxidative stress in tumors. This is why PBM might help the jabbed as well. Light induces an abscopal effect on cells

• This effect weakens cancer cells that rely on glycolysis (Warburg effect), making them more sensitive to treatment. PBM is known to lower blood glucose by close to 30% via its abscopal effect.

4. Synergistic Effects with Chemotherapy & Radiation

Exposure to Ultraviolet Radiation in the A band promotes the expression of melatonin receptors in the skin via alpha MSH made from POMC.  This occurs via melatonin receptors called MT1 and MT2.

In the lab, pretreatment with melatonin reduced cyclobutane pyrimidine dimers (DNA strand breaks) levels by approximately 40%.  Remember, life is not lived or built in a LAB……….Nature is your lab.  AM sunlight increases melatonin and melanin.   https://www.nature.com/articles/s41598-017-01305-2

• Some studies suggest Fenbendazole enhances the effects of radiation and chemotherapy by:

• Increasing DNA damage accumulation.

• Disrupting repair pathways (e.g., via p53 activation).

•Weakening microtubule integrity, making cancer cells more vulnerable to other drugs or light therapy.

Supporting Studies & Evidence
1. Fenbendazole inhibits tumor growth via microtubule disruption and p53 activation

• Study in lung cancer cells showed that Fenbendazole caused mitotic arrest, increased p53, and induced apoptosis.

• (Source: PubMed)

2. Fenbendazole enhances radiation sensitivity by targeting microtubules and p53

• Research demonstrated that combining Fenbendazole with radiation led to increased DNA damage and cell death.

• (Source: PMC)

3. Mechanism of Fenbendazole in disrupting glucose metabolism

• Study found Fenbendazole downregulates GLUT1, reducing glucose uptake in cancer cells.

• (Source: PubMed) Conclusion Fenbendazole upregulates p53 by causing mitotic stress, DNA damage, and metabolic inhibition, leading to cancer cell apoptosis.

Its primary mechanism is microtubule disruption, similar to drugs like Vinblastine or Colchicine used in centralized oncology, but with much lower toxicity.

CAN THESE IDEAS HELP TURBO BREAST CANCER = triple negative cancers?

Stage 4 triple negative breast cancer is exploding in the COVID era in our women. I really worry about our children who are still getting popped with these jabs by pediatricians now.

There are case reports that this type of cancer can be erased with a combo of cannabinoids and psilocybin. Might we be able to find unique genomes of these two drugs to combine together to treat SV40 induced cancers?

I think so.

It might turn out that the centralized drug war isn’t what you think it is. It may fuel the decentral drug wars on all human cancers.

Yes, psilocybin (and its active metabolite psilocin) interacts with several cancer-related pathways, some of which overlap with Fenbendazole, Ivermectin, and Cannabinoids. While psilocybin is primarily studied for its psychedelic and neuroplasticity effects, emerging research suggests it may have anti-cancer properties via serotonin receptor modulation, immune regulation, and metabolic disruption.

1. Serotonin (5-HT) Receptor Activation and p53 Pathway (Similar to FenBen & Cannabinoids)

^^^^Remember where you saw this? The Melanin Renovation blog.

Sunlight  increases the catalytic activity of enzyme IDO in this pathway as I already taught you

When this happens your cells begin making more kynurenine and less serotonin and melatonin this leads to ALL CHRONIC DISEASES and INCREASES ALL CAUSE MORTALITY because it destroys melanin renovation internally.

WHILE simulataneously  SUNLIGHT decreases the catalytic activity of KAT

Making less kynurenine acid (neuro protective for melanin) and more quinolinic acid (harmful for melanin) from melanin degradation.

A lack of sun causes melanin degradation via hypoxia. Non native EMF cause liberation of Vitamin A from the loose covalent bond of opsins in the non visual photoreceptor system due to alien light and that Vitamin A cause hypoxia by lowering NAD+ in a cell.  This is today’s major cause of disruption in this pathway.  This slide should jar your memory of my past lessons. If you are jabbed you really better get this lesson.

•Fenbendazole & Cannabinoids: Activate p53, leading to apoptosis.

•Psilocybin/Psilocin:

•Activates 5-HT2A receptors, which have been linked to p53 upregulation in certain cancer models.

•Induces apoptosis in neuroendocrine tumors via serotonin signaling.

•Regulates BCL-2/BAX, modulating mitochondrial-mediated apoptosis. Overlap:

•Psilocybin may enhance p53 activity, similar to FenBen and CBD, leading to cancer cell apoptosis.

2. Metabolic Disruption & AMPK Activation (Similar to FenBen & Cannabinoids)

•Fenbendazole: Blocks glucose metabolism, shifting cancer cells away from glycolysis (Warburg effect).

•Psilocybin: Modulates AMPK/mTOR pathways, which regulate cancer metabolism and cell proliferation. 380 nm light addition here to improve mTOR signaling. This is why the tropics are critical to these riddle. Remember this slide from the Melanin Renovation Rx for Mammals on Patreon?

•Downregulates PI3K/AKT, reducing cancer cell survival.

•Potential to inhibit GLUT1 (glucose transport), limiting cancer energy supply. Overlap:

•Psilocybin could synergize with FenBen, Metformin, or Cannabinoids in starving tumors of glucose. PBM should added here because it lowers blood glucose and insulin signalin..

3. Microtubule Interactions & Cytoskeletal Remodeling (Similar to FenBen)

•Fenbendazole: Binds β-tubulin, causing mitotic arrest and apoptosis.

•Psilocybin/Psilocin:

•Modulates cytoskeletal proteins, leading to structural changes in cancer cells.

•Impacts tubulin polymerization in neuronal cells, suggesting possible effects on cancer cell microtubules.

Overlap:

•Though less studied in cancer, psilocybin’s cytoskeletal effects might interact with FenBen-like mechanisms.

4. Immune Modulation & Anti-Inflammatory Effects (Similar to Ivermectin & Cannabinoids)

•Ivermectin: Modulates T-cell responses, enhances anti-tumor immunity.

•Psilocybin:

•Reduces pro-inflammatory cytokines (IL-6, TNF-α, IL-1β), which drive tumor progression. •Enhances immune checkpoint modulation, possibly affecting PD-1/PD-L1 pathways in cancer immunotherapy.

•Shifts immune response toward anti-cancer cytotoxicity.

Overlap:

•Psilocybin’s immune effects mimic Ivermectin’s immunomodulation, making it a potential adjunct in cancer therapy.

5. Potential Synergies with Ivermectin, FenBen, and Cannabinoids PathwayFenBenIvermectinCannabinoidsPsilocybin p53 Activation Microtubule Disruption (CBD) (Possible) AMPK/mTOR Modulation Metabolic Disruption Immune Modulation

 

When I asked the Shamans about how they chose their medicines I found out they were using drugs from the forrest that all had these overlaps.

Psilocybin has overlapping mechanisms with Fenbendazole, Ivermectin, and Cannabinoids in p53 activation, immune modulation, metabolic disruption, and possibly cytoskeletal remodeling. While direct microtubule inhibition is uncertain, serotonin signaling, apoptosis, and immune regulation suggest a potential role in cancer therapy. Would you like specific references, drug interaction studies, or potential combination therapies explored further?

THE REMEDY CAME ON LIKE A DREAM TO ME.

SUMMARY

Catastrophic disclosure was not linked to the information and evidence coming out during the COVID era.  Covid narratives were built around but a consolidation around these false narrative by federal agencies who incentivized many groups to support COVID psy-op fear;  This is how a false flag operations happened at scale from 2020-2025.

All of you need to follow @JohnBeaudoinSr on Twitter because he has all the death data that holds the truths.

He has the data to support this supposition in great detail. This was catastrophic epoch in human health because it leads the world not having unity and peace, something beautiful, a view of us and other civilizations doing things together, a new world being created, technologically sustainable and peaceful, is about uniting the world under a sort of militaristic global totalitarian system built around surveillence capitalism where everyone is terrified what’s out there for many reasons linked to the narratives furnished and created by federal agencies so thatpeople give up their freedom with out a fight   It’s a fear play to circumvent a political revolution if the truth really was known by “We The People.”

Many Federal agencies had a legal duty to inform us but they were told by the DoD and Pentagon to hide behind faulty use of the ICD coding system to lie to us. So far they have gone unpunished. The first step is complete banning of the mRNA platform. This podcast shows you why I undressed Calley Means in December with Danny Jones. Calley calling for “transparency” was a criminal speaking given the hard core data John has.

I’ll stop being a conspiracy theorist when the rich and powerful stop conspiring. We are being ruled by centralized greedy, power-hungry sociopaths who are destroying the planet. They are being led online by Sergey Brin and Anne Wojicicki.

HYPERLINK

Women who are infertile maybe helped by some of this information in this blog to have children. Why? The elites crafted the COVID era with the mRNA platform. The elites are eugenicists and they favor quick death via abortion for women to rid the planet of future children due to their perverse beliefs. The mechanisms of infertility mimic the disease creation of the turbocancers. All are tied to leptin melanocortin pathways.

The elites like Brin, Gates, Fauci, and GAVI linked companies knew to break the golden centralized rule in pregnancy medicine. Pregnant women are already in a state of immunosuppression so why would you then give a pregnant woman 4 different vaccines during this time? To eliminate NEW life is the short answer.

This is especially one that is experimental and the data showed it caused spontaneous miscarriages. So Gates and Fauci knew that the COVID jab was a covert abortion clinic run at global scale.

When you know better you can do better, and Uncle Jack is digging deep for the remedy for 6 billion of you.

CITES

1. https://www.youtube.com/watch?v=cn5CQGpiJWg

2. https://www.youtube.com/watch?v=KQUSoIJkaWg

3. https://www.youtube.com/watch?v=NN9X04C3G5Y

4. https://www.youtube.com/watch?v=SgCSGN8UR9M

5. https://www.youtube.com/watch?v=tAf3UB2ycCs

6. Single dose- is not BigHarma friendly solution. https://www.nejm.org/doi/full/10.1056/NEJMoa2206443

7. https://www.nejm.org/doi/full/10.1056/NEJMoa2032994

8. https://x.com/marclanders/status/1891460901363921148

What am I doing in the Amazon rain forest in Suriname? I am looking for the antidote for those who took the jab. 50 years ago I learned from the Cutter incident linked to the Polio jabs of Salk and Sabin and the unpublished work of Dr’s. Sarah Stewart and Mary Sherman on viruses that they could be treated using novel chemokines found in soils. Sherman extended Stewarts idea that exposure to any form of radiation treatment would somehow strengthened the virulence of the SV40 virus via bond loosening within an animal. It was a discovery that was worthy of a Nobel Prize but no one in science could ever know what these two woman do because their work was secret and confined to a “biological Manhattan Project” for the DoD in New Orleans in the 1950s.

Chemokines use deuterium chemistry to strengthen bonds. They have the opposite effect of electromagnetic radiation. Stewart knew this from her work with nuclear reactors at the University of Chicago in the Fermi lab. Dr. Stewart found SV40 was inhibited by things grown in water with an unusual isotopic fingerprint. That information was buried in the boxes I found in the basement of Charity hospital in NOLA where I trained as a neurosurgeon in the late 1980s and and early 1990s. Parts of Dr Stewart’s work/ thesis was still present there at this time and I found some more of her original ideas in Tulane University School of Tropical Diseases on Canal Street. I read this in 1992. Now I am using that information here at the 4N to help solve a mystery for the jabbed.

Mitochondriacs should believe in scientists who are relentless in finding the wisdom of nature that builds the public “health truths”. I just had an amazing experience inside the Amazon Rain forrest looking for a solution for the jabbed. It was a successful journey.

Mitochondriacs learn to seriously doubt those who believe they found a solution in a false narrative. They are comfortable questioning everything and they have no problem digging deeper to find the elusive truth they need. They come to recognize the power in Nature to sculpt health and they realize her power never diminishes yours. If you live her way, she will give you immense power to control your health journey in this life.

THE DISCOVERY OF IVERMECTIN IS WHAT DROVE THIS TRIP TO THE AMAZON

In philosophy and science, a first principle is a basic proposition or assumption that cannot be deduced from any other proposition or assumption. First principles in philosophy are from first cause. Mitochondriac wisdom is dedication to teaching other how to think in decentralized fashion to improve life on earth in some small way.

Centralized medicine was weaponized by the DoD and turned BigHarma into the largest killing machine man has ever created. In the Amazon I met with a group who may hold the key passage to disintermediating the death grip of this banker inspired empire over humanity.

In the late 1960s and early 1970s, Satoshi Ōmura, a microbiologist and bioorganic chemist at Tokyo’s Kitasato Institute, hunted for new sources of pharmaceuticals. He knew that some existing drugs, including antibiotics, had been derived from compounds found in nature. The forrests of the world are essentially decentralized pharmacies that have been acted on by light, water, and magnetism for billions of years and this created a myriad of compounds for humans to use. Their use was done by trial and error. This became his philospophy of touch. How could touching the Earth lead to healing humans all over the world.

So he developed screening methods to identify medicinally promising compounds from soil. His team collected thousands of soil samples from around Japan, cultured bacteria from them, and screened each culture for medicinal potential.

THE EUREKA MOMENT

The story is so improbable it defies belief: This is what motivated me to go deep into the Amazon. The story of how Ivermectin was discovered began with a soil sample from Japan stops suffering in Africa. It begins when a scientist discovers a lowly bacterium near a golf course outside Tokyo. A team of scientists in the United States finds that the bacterium produces compounds that impede the activity of nematode worms. It is developed into a drug that wards off parasites in countless pets and farm animals,averting billions of dollars in losses worldwide.

Extraordinarily, the drug also prevents or treats human parasitic diseases that would otherwise cause blindness and other severe symptoms in hundreds of millions of people in many of the poorest countries on Earth.

During COVID it was found to affect the development of rapid cancers from the poorly designed mRNA platform. Why was this information key? It pointed me in the direction of the circadian clock genes to look for an answer. Rapid cancer generation would mean the circadian mechanism had to be hijacked in some novel way.

First principle thinking teaches us that morning sun exposure resets the circadian clocks of the skin, allowing your body to properly prepare for UV Circadian genes like CLOCK and BMAL1 regulate the expression of key DNA repair proteins, ensuring that DNA repair is most effective when the body is exposed to higher levels of DNA-damaging factors This is why you want to get any screening which involves electromagnetic radiation, like an X-ray, or a cell phone, or the sun, done in the late morning to early afternoon (Approximately 10 AM – 2 PM) This period is when the circadian-controlled DNA repair mechanisms are generally more active It’s no coincidence that this is also the timeframe when UV light from the sun is most intense Nature doesn’t miss and opportunity to create a mark. This told me going to a place where the sun was dominant and where other bands of radiation were absent were the key in finding the answer for the jabbed.

In the last four years I have done a lot of reading about the unique environments in the largest decentralized pharmacy on earth, the Amazon basin. Some of the quantum biological principles I have acquired over this time have led me to Suriname to find a reversal for the jabbed. I have a hunch where the answer is because of this information processing. Today is the first day of this journey of discovery. Always remain curious. Remain open. When someone tells you a problem like SV40 is unsolvable this should make the challenge palatable and worthwhile.

SUMMARY

The tale depends on an international cast of thousands of scientists, medical practitioners and other dedicated participants. It also involved a BigHarma company and research institute that was willing to give a drug away for free. This was done to build trust between the company and the public so that the public would come to lean on and trust industry when that TRUST should not be given. To be sure, Ivermectin did help rid the developing world of debilitating diseases.

Yet none of this would have happened without that soil dug up in Japan and a healthy dose of serendipity.

The odds against finding avermectin smaples in the soil and recognizing its potential were astronomical. The transformation of avermectin to ivermectin was truly the easiest step in its discovery. The biggest irony is that BigHarma was aggressive with this drug in development because it had so a high therapeutic index. Ivermectin is a particularly attractive treatment because They believed in the 1970’s and 1980s it had no antibacterial or antiviral activity, w hile having few serious side effects even at massive doses. In the 2000s we found out Ivermectin has antiviral activities especially to genetically altered virus. If Big Harma would have known this early on, my bet is DARPA and the DoD would have strangled this drug in its crib during discovery. They did try to do this in 2020-2025 during the COVID pandemic to force people to take the BigHarma jab.

History shows us this drug could so easily have been missed, and the life-altering decentralized pharmaceutical that was derived from it might never have been discovered.

This is what motivated my trip to the Amazon to look for clues that might help the jabbed. I am happy to say to you, my sleuthing might have paid off so this slide can be disintermediated. This is the slide I showed to Dr. Bowden and Danny Jones in our podcast together that showed that the jab is the MAIN PROBLEM in causing cancers. That huge spike going almost vertical in the bottom right is why I went to the jungle.

 

https://vimeo.com/user192601857/review/1021313092/2e32d43335?fbclid=IwY2xjawIE6ThleHRuA2FlbQIxMAABHenEmPztUp0_J41kl6nwjpOD5R9YwTkloMpyR_wwj4WZ6PPjp2rc6on3Gw_aem_EDfZT-fVQOAhbsNRgpsivw

Over the 20th Century, various investigators in different countries representing multiple interests have repeatedly reported the discovery of unusual non-local field effects in human biology that could not be explained in the framework of the Standard Model. Since the investigators and writers could not understand or explain the physics associated with the observed phenomena, they were forced to invent new names for the fields, emanations, and energies believed to be responsible for creating these phenomena. Scalar phenomena and torsion field manipulations are two examples of this idea.

If you listened to my Breedlove podcast, you would have heard about the subtraction of James Maxwell’s original equations from 20 to 4 equations. I believe this “censorship” or subtraction is the cause of the Standard Model’s missing framework to explain many phenomena in Nature.

Oliver Heaviside was a self-educated English mathematical physicist who spent most of his life on the far fringes of the scientific community. Yet he did more than anyone else to shape how James Clerk Maxwell’s electromagnetic theory was understood and applied in the 50 years after Maxwell’s death. Indeed, Maxwell’s equations in their most familiar vector form come from Heaviside’s reformulations.

Maxwell laws of electromagnetism were once described by 20 equations. Maxwell’s contribution to science in producing these equations really lies in the correction he made to Ampère’s circuital law in his 1861 paper On Physical Lines of Force. He added the displacement current term to Ampère’s circuital law and this enabled him to derive the electromagnetic wave equation in his later 1865 paper “A Dynamical Theory of the Electromagnetic Field” and to demonstrate the fact that light is an electromagnetic wave. Faraday proved it first when he experimentally found the Faraday effect, but physicists of the day wanted a mathematical proof that Faraday’s simpleton approach was equivalent.  Maxwell gave the world this proof and it was later re-confirmed experimentally by Heinrich Hertz in 1887. The Maxwell equations were simplified to 4 by Oliver Heavside by mathematical convention.  What were the other 16 about? They were about these two pictures below.

Do you notice anything similar between these two pictures? These both show magnetic field effects in nature.  The top one is an abiotic magnets effect on iron filings.  The bottom is the result of electric and magnetic fields around predator animals and their prey. 

Gravity is said to be “different” from the electromagnetic force because gravity has a polarity that is always positive.   In other words, for gravitational forces, things with masses always seem attractive and add to one another. What could nature be hiding that might have a negative polarity that could move things apart?

Somatic cells act like a wave of kamikaze support of the germline cells and melatonin  creation in their mitochondria acts like their General. The leptin melanocortin pathways in these tissues protects that cell line by making sure it has light and electronic power to maintain its existence. Somatic cell death requires the creation of a local biological timing mechanism locally, hence, this is why every somatic cell has its own clock mechanism separate from the SCN. This makes time relative in somatic cells. This is why certain organs age faster than others and it is the basis of what defines heteroplasmy in cells. People who are jabbed experience higher heteroplasmy rates depending upon where the jab contents remain persistent.

Might the destruction of a magnetic monopole in DNA be acting in the hydrophobic pockets in DNA and cause the DNA helix to unwind in the opposition of gravity to cause disease?  This question may sound esoteric but to those with diseases from the jab it might be life saving. Why? What if we develop techniques to control this process in DNA? We might be able to delay disease and death in the jabbed.

The presence of magnetic monopoles was described by the 16 deleted equations of Maxwell, but as of 2025 one has never been found to exist in nature.  Therefore people who supported Heavside in the late 1800’s removed many of Maxwell’s original equations that dealt with them.  Many physicists over the last 150 years thought this was unwise.  Paul Dirac was the most influential critic of this turn of events in physics in the 1930s.  He wrote many papers referencing this belief back then but not many have listened to his warnings.

Could it be the source of the negative polarity in things that give a thing a net negative flux?  Recall that all living things have a serious net negative charge.  Also, recall that water goes from a neutral polarity in its bulk state, and then gains its net negative charge in the structured state when UV and IR/NIR light excite water. This structured state exclude protons and creates coherent domains that act like electromagnetic capacitors for crystalline lattice in cellular water that resists hydrophobicity.  When you carefully read Nick Lane’s book (THE VITAL QUESTION)  you see there is a trend in biology to still wonder why life decided to use protons for chemiosmosis across cell membranes in all three kingdoms of life.

Might the answer be because water and sunlight naturally create unbelievable amounts of protons free of an energy charge?  Hydrophobicity can break this effect as the video above showed. This allows DNA to unwind and be copied and under reverse transciption which can cause turbo cancers. This effect would be magnified if SV40 contamination was present. DNA plasmid numbers make the probability of such event go higher. Translation is widespread in annotated noncoding sequences, including untranslated regions (UTRs), introns, and long noncoding RNAs (lncRNAs), especially in contexts such as cancer, aging, and neurodegeneration. 3’UTRs follow the coding sequence of the mRNA and regulate localization, stability, and translation, among other things. Did you know that both Moderna and Pfizer use novel 3’UTRs, never used before?

Unbiased genetic and biochemical screens both identified the BCL2-associated athanogene 6 (BAG6) pathway for mediation of the proteasomal degradation of diverse noncoding translation products.

• BAG6 recognizes a hydrophobic C-terminal tail, a common feature of proteins translated from all types of noncoding sequences. This results from the U-rich nature of the noncoding genome and the strong bias of U-rich codons for hydrophobic amino acids in the genetic code.

The electromagnetic radiation that reaches the Earth’s surface from space as microwave background radiation is believed to be a consequence of the big bang and the evolution of the universe in the very first seconds of its existence. This type of radiation is characterized by its thermal energy distribution as a perfect black body in nature and has a nearly ideal Planck spectrum at a temperature around 2.7 Kelvin, while the maximum of its surface power density corresponds to the wavelength of 272 GHz. The solar radiation that reaches the Earth’s surface has relatively small surface power density around 3 μW/m2 and comprised of distinct frequency bands, so-called atmospheric windows, representing those frequency bands that are not absorbed by the Earth atmosphere. They can be listed as

• radio window—represented by electromagnetic wavelengths starting from 15 MHz up to 300 GHz,
• optical window—represented by electromagnetic wavelengths starting from 150 THz up to 1000 THz.
• microwave window—represented by electromagnetic wavelengths starting from 23.1 THz up to 37.5 THz.Earth’s magnetic field is another natural field originating from the planet’s core that extends to a vast space surrounding Earth, known as the magnetosphere. An essential source of strong electromagnetic fields is atmospheric discharges, known as lightning. Rapid radiation releases accompanying these natural phenomena are characterized by high power densities and high frequencies. In living organisms, electromagnetic fields originate from the transmission of signals in the nervous system and from structures autonomously generating electrical impulses, like the heart (EKG), brain (EEG) because this is where mitochondrial density is greatest.

The visible universe is constituted almost entirely of electrically active plasma that we can observe for a deep reason. It is what life organizes around.

IMPLICATIONS?

Might it be that when sunlight collides with water, the electric and magnetic fields in light waves change water networks physically to structure cells and cause them to become a dissipative state to limit the effects of entropy? Might inducing hydrophobicity be how we re-introduce entropy and the flow of time to cause disease and death? This effect is buried in every vaccine man has ever made because of the added atoms and chemicals. These pollutants are how the bioweapons program changes the light waves in biophotons, which cells need to fight entropy to remain healthy. Could these changes lead to emergent properties in a matter of which we still remain ignorant?

I think so, and so did Dr. Luc Montagnier before his death.

SUMMARY

The gif above clearly illustrates how a two-dimensional surface can be created in 3D.  Might this picture show what our brain does by resolving environmental waveforms? Might it also show us that the contents in a jab can stop the rotation in a 2D surface to cause protein misfolding and disease? I think this is why COVID and COVID dementia (BIDEN) are telegraphing to decentralized thinkers. Using the above picture, we only need a motion in 2 dimensions and not three to create a wave.

Light can be polarized similarly by a single plane of water.  The jab destroys this effect in water by restructuring it. This stops the dots from rotating, causing complexity to drop and ruin the dissipative state in cells. Life needs to remain well to fight entropy loss. Polarized light can be bent using the Faraday effect.  Sunglasses and windows affect light in this way. This effect is an optical, magnetic, nonlinear effect of light.  DeBroglie was hinting at this wave mechanics of all matter in his Ph.D. thesis, for which he later won the Nobel Prize.  The problem for physics back then was that Bohr’s version of quantum mechanics largely ignored DeBroglie’s insights. It is another form of centralized scientific censorship that continues to hide decentralized truths from us.