It is difficult to get modern humans to understand something when THEIR addiction to its use depends on THEM not understanding it.
This blog is about that very issue. Does technology create an altered immune state in modern humans that lead them open to a myriad of diseases?
Yes.
Did you know that taking exogenous melatonin thins your retina and makes you fatter, yet endogenous melatonin production seems to help us maintain leptin sensitivity? Ask your food gurus and your supplement sellers why this is the case before you ever give them a cent.
Pinealectomy induces circadian arrhythmia and has interesting effects on adipose tissue biology. Did you know that blue light/nnEMF exposure through the eye and skin mimics the physiologic effects of the removal of the pineal gland?
Why is this important in the modern world?
Doctors are IMPOTENT to explain the rise of diabetics who are SKINNY. Those diabetics who are skinny break the rules doctors are taught in med school and residency because educators of doctors have always believed diabetes was only caused by eating too many carbohydrates. What did they not know? Imbibing technology screen blue light and nnEMF is EQUIVALENT to eating carbs 24/7, but the damage it causes to your mitochondria and adipocytes is radically different because of how nnEMF/blue light DEMOLISH melatonin production in the pineal gland. THAT is the key mode of creating a skinny diabetic. Skinny diabetics are also more prone to cataract formation, myopia, and AMD. Did you know that? Have you ever thought to ask yourself why that might be? It is because thin diabetics are hypoxic.
Skinny diabetics are also very hypoxic and they tend to get massive amounts of peripheral artery diseases and intracranial artery diseases that lead to early CABG, stroke, dementia, and melanopsin damage to the retina. This leads to cataracts and AMD too. All these diseases are spiking today. What is the fundamental driver of this process? A chronic mix of low melatonin production, low NO production, and chronic thiamine deficiency acting all at once.
Adipocytes isolated from pinealectomized animals in cite one below all exhibited lower glucose uptake, higher glucose oxidation, and lower de novo lipogenesis. Collectively, these results suggest frank MASSIVE melatonin deficiency, that has long-lasting impacts on adipocyte biology which manifest as failure to conserve energy. You need melatonin to have a normal fat mass. It turns out blue light and nnEMF via CHRONIC overexposure of your eyes/skin can chronically cause you to massively lose fat while becoming a metabolic train wreck and push you to an early death.
Clinicians need to pay attention to different metrics in tech abusers.
In those people we should see an intial period where blue light fattened the patient follwed by some time elapse, when mitochondrial failure occurs. What should we be looking for as clinicians in these patients? Their glucose and insulin levels in the fed and fasted states usually aren not affected, but glucocorticoids on hormone panels are dramatically chronically elevated. Cortisol levels usually become very abnormal and then leptin levels plummet in chronic fashion to a significantly greater degree when the patient screen time is excessive. This is a different picture than one sees in acute leptin resistance.
Why is this mechanism important for clinicians to understand? When leptin levels drop adiocytes atrophy and a very thin phenotype occurs.
The chronic fall in leptin became interesting to me because it should have predicted patients would have an enhanced energy conservation because they were thin, but this isn’t what happened because their fat cells were non functional. Glucocorticoids usually rise at night when the circadian mechanism is intact, after melatonin peaks at 2AM. When the circadian mechanism is chronically disrupted, melatonin levels crater in the night, and this seems to exacerbate the increase in glucocorticoids we see. Blue light and nnEMF also boost insulin levels and blood glucose. Why is this a problem for creation of the skinny diabetic?
Insulin alone has little direct effect on leptin secretion in mammals, but glucocorticoids are super- stimulatory to blood glucose release, and this is enhanced by insulin production. This is often why in history taking we often find skinny diabetics used to be heavier in their life before they became more metabolic derranged. It also explains why skinny diabetic go undiagnosed longer than obese diabetics. Doctors are not taught to look for these signs in normal to slightly elevated BMI cases. This should change in a tech abusing world.
As a result, light, and melatonin as light’s protein proxy, appears to negatively regulate this hormonal axis and boost the anabolic potential of adipocytes. This makes fats cells store fat. It appears the levels of melatonin in the central retinal pathways and the skin hypothalamic axis predict who will go on to become a skinny diabetic. This infographic is critical to understand this very complex light phenomena.
Interestingly, the slide on melatonin creation by AM UVA/UVB exposure points out why both type 1 and 2 diabetics show a latitude effect in their disease course (see below two figures).
WHAT HAPPENS IN THE MATRIX WHEN THIS OCCURS?
NAD+ drops with SIRT1 and UCP-2 mechanism in mitochondrial becomes radically permeable just as RBC become super permeable to lactate as you learned in Hypoxia #5 blog.
WHAT DOES UCP-2 do?
UCP2 controls the carburetor mix of fuel in the matrix, namely, oxygen & hydrogen mix (from stored fat or from hydrogen in foods) in mitochondria and deuterium leakage in a circadian/seasonal manner to stimulate growth & ELF-UV release = broken UCP2 leads to cancer generation. When UCP-2 is leaky eukaryotic cells leak massive quantities of ELF-UV. Functional UCP2 lowers mitochondrial heteroplasmy by controlling how much deuterium enters the TCA cycle to control the anions it moves. UCP2 does this by varying the light released and reduced ROS from cytochrome II to stimulate ketogenic substrates in TCA = based on temperature & light signals from the light environment you choose to live under and not simply by what you eat. This is the food guru fallacy. This also points out why food cannot solve quantum diseases. Think cancer generation.
To achieve optimal ketosis you need to be fasting & be in daily solar red light whilst barefoot grounding to program proton spin = this properly recycles the hydrogens in DNA/RNA backbones to make seasonally responsive to light variations and DNA expression. If you eat carbs out of season or expose yourself to nnEMFs/ALAN over the winter you will increase blood glucose levels and turn off fat burning because of the effects of insulin. UCP function depends on leptin sensitivity = light/dark. Taking exogenous melatonin ACTS to increases blood glucose/AMPk/ and makes UCP-2 non functional. This maybe why skinny diabetics have poor outcomes with cancers, cardiovascular diseases and strokes.
These people will be at risk for many infectious diseases in the modern world.
Today’s parting thought:
The Earth is recovering……….
– Air pollution is slowing down
– Water pollution is clearing up
– Natural wildlife returning home
Coronavirus is Earth’s vaccine
We’re the virus.
Mother Nature is teaching us what karma is when we use parts of the electromagnetic sptrum she told us not to use.
Humans should be mentally preparing themselves for this to be a marathon, not a sprint. Mother Nature has been aiming to beat our asses and now she has them.
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